Relevant bibliographies by topics / Neuromuscular development

Academic literature on the topic 'Neuromuscular development'

Author: Grafiati
Published: 10 December 2022
Last updated: 28 January 2023

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Journal articles on the topic "Neuromuscular development"

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Orlando, Lianna. "Neuromuscular synapse development." Trends in Neurosciences 24, no. 7 (July 2001): 373. http://dx.doi.org/10.1016/s0166-2236(00)01911-1.

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KELLY, A. M., and N. A. RUBINSTEIN. "Development of neuromuscular specialization." Medicine & Science in Sports & Exercise 18, no. 3 (June 1986): 292–98. http://dx.doi.org/10.1249/00005768-198606000-00007.

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Miller, Geoffrey. "Neuromuscular development and disease." Neuromuscular Disorders 3, no. 1 (January 1993): 90. http://dx.doi.org/10.1016/0960-8966(93)90050-t.

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Russman, B. S. "Neuromuscular Diseases During Development." Archives of Neurology 55, no. 6 (June 1, 1998): 879. http://dx.doi.org/10.1001/archneur.55.6.879.

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Kleinman, Ronald E. "Complementary Feeding and Neuromuscular Development." Pediatrics 106, Supplement_4 (November 1, 2000): 1279. http://dx.doi.org/10.1542/peds.106.s4.1279a.

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Campagna, Jason A. "Development of the Neuromuscular Junction." International Anesthesiology Clinics 44, no. 2 (May 2006): 1–20. http://dx.doi.org/10.1097/00004311-200604420-00003.

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Witzemann, Veit. "Development of the neuromuscular junction." Cell and Tissue Research 326, no. 2 (July 4, 2006): 263–71. http://dx.doi.org/10.1007/s00441-006-0237-x.

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Aydin, Onur, Austin P. Passaro, Mohamed Elhebeary, Gelson J. Pagan-Diaz, Anthony Fan, Sittinon Nuethong, Rashid Bashir, Steven L. Stice, and M. Taher A. Saif. "Development of 3D neuromuscular bioactuators." APL Bioengineering 4, no. 1 (March 1, 2020): 016107. http://dx.doi.org/10.1063/1.5134477.

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Jennings, Charles G. B., and Steven J. Burden. "Development of the neuromuscular synapse." Current Opinion in Neurobiology 3, no. 1 (February 1993): 75–81. http://dx.doi.org/10.1016/0959-4388(93)90038-z.

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Collins, Brandon W., Gregory E. P. Pearcey, Natasha C. M. Buckle, Kevin E. Power, and Duane C. Button. "Neuromuscular fatigue during repeated sprint exercise: underlying physiology and methodological considerations." Applied Physiology, Nutrition, and Metabolism 43, no. 11 (November 2018): 1166–75. http://dx.doi.org/10.1139/apnm-2018-0080.

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Neuromuscular fatigue occurs when an individual’s capacity to produce force or power is impaired. Repeated sprint exercise requires an individual to physically exert themselves at near-maximal to maximal capacity for multiple short-duration bouts, is extremely taxing on the neuromuscular system, and consequently leads to the rapid development of neuromuscular fatigue. During repeated sprint exercise the development of neuromuscular fatigue is underlined by a combination of central and peripheral fatigue. However, there are a number of methodological considerations that complicate the quantification of the development of neuromuscular fatigue. The main goal of this review is to synthesize the results from recent investigations on the development of neuromuscular fatigue during repeated sprint exercise. Hence, we summarize the overall development of neuromuscular fatigue, explain how recovery time may alter the development of neuromuscular fatigue, outline the contributions of peripheral and central fatigue to neuromuscular fatigue, and provide some methodological considerations for quantifying neuromuscular fatigue during repeated sprint exercise.
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Dissertations / Theses on the topic "Neuromuscular development"

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Ribchester, Richard R. "Development and plasticity of neuromuscular innervation." Thesis, University of Edinburgh, 2005. http://hdl.handle.net/1842/29963.

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The thesis presents contributions to the field of neuromuscular synaptic plasticity. Synaptic remodelling brings about changes in convergence and divergence in many different parts of the nervous system during development.  Neuromuscular junctions have proved to be accessible synapses in which to describe and explain the mechanisms. During development, muscle fibres initially receive convergent, polyneuronal innervation (π) by axons arising from different motor neurones. The characteristic mononeuronal innervation (µ) pattern of adult muscle is achieved by synapse elimination, a process of weakening of synaptic strength followed by withdrawal of synaptic boutons, until all but one of the motor neuron inputs to an endplate is lost. Similar hyperinnervation and elimination occur in adult muscle after nerve injury, collateral sprouting and regeneration. These processes are strongly influenced by activity, apparently in accordance with Hebbian rules of synaptic plasticity. But how decisive is activity in ultimately determining the pattern of neuromuscular connectivity? The amount of sprouting is increased and the rate of synapse elimination is decreased when muscle activity is blocked. Sprouts regress and synapse elimination resumes when muscles are stimulated, or once normal activity is restored. Selectively blocking or restoring activity in some motor neurones but not others supplying a π-junction gives a competitive advantage to the more active neuromuscular synapses. However, activity is not sufficient to effect synapse elimination because many muscle fibres retain π-junctions after activity resumes following a period of paralysis. Nor is activity strictly necessary, because – paradoxically – synapse elimination continues at some motor endplates even when muscles are completely paralysed. Competition for neurotrophic factors may play an important role in determining the outcome of synapse elimination, but factors intrinsic to the motor neurone, perhaps involving the selective trafficking of maintenance factors along specific axon collaterals, appear to be important also. In each motor neurone, synapses are eliminated or strengthened asynchronously.
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Lee, Chi Wai. "Development of the presynaptic nerve terminal during neuromuscular synaptogenesis /." View abstract or full-text, 2005. http://library.ust.hk/cgi/db/thesis.pl?BIOL%202005%20LEE.

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Currie, Douglas A. "Neuromuscular development in the adult abdomen of Drosophila melanogaster." Thesis, University of Cambridge, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.239201.

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Benatar, Michael G. "Presynaptic function in development and disease at the neuromuscular junction." Thesis, University of Oxford, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.388959.

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Teriakidis, Adrianna. "Intra-neuronal influences on development of the mammalian neuromuscular junction." Thesis, University of Edinburgh, 2010. http://hdl.handle.net/1842/25245.

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During development of the nervous system an excess number of synapses are formed, most of which are subsequently pruned, resulting in functional neural networks. The precise mechanisms that determine which synapses are formed and which synapses are maintained are not thoroughly understood. The aim of this thesis is to investigate the intra-neuronal constraints and influences on synapse formation and elimination during development. In the first part of the thesis I investigated intra-neuronal influences on synapse elimination. Synapse elimination is known to occur at polyneuronally innervated neuromuscular junctions through competition, leading to mononeuronally innervated muscle fibres. However, whether synapse elimination ever occurs in the absence of competition, leading to muscle fibres becoming denervated, has not been resolved. The data presented in this thesis suggest that only large motor units undergo a reduction in motor unit size in the absence of competition. Using the Rasmussen and Willshaw (1993) version of the Dual Constraint Model I show that these data are consistent with the prediction that synapses will be eliminated from muscle fibres when a neuron's resources become stretched, for instance as a result of the normal growth of the animal. Larger motor units, which innervate more synapses, will thus be more vulnerable to the extra demand put upon them by the growth of each synapse. The model predicts that synapse elimination in the absence of competition should occur at least over the first 6 months of life and not only during the first two postnatal weeks, when most polyneuronal innervation is normally eliminated. In the second part of the thesis, I investigated intra-neuronal influences on synapse formation. Specifically I tested the hypothesis that each branch of a motor neuron forms synapses randomly and independently of other branches. If true there should be instances where two branches from the same neuron initially innervate the same endplate (sibling branch convergence). Sibling branch convergence was experimentally investigated in both regenerating and developing motor neurons. The evidence suggests that sibling branches can converge on the same muscle fibre and that they can competitively eliminate each other. However, it appears that convergence does not occur at the frequency that would be expected, suggesting that branches from the same motor neuron do not form synapses independently of each other. At present there are limitations in imaging immature networks due to the spatial resolution limit of light microscopy. The last part of this thesis explores thin serial sectioning and reconstruction as a possible technique for increasing resolution in the z-axis. This technique has potential to be developed but I show that at present it does not provide sufficient resolution to discriminate between developing motor axons in neonatal lumbrical muscles.
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Broadie, Kendal Scot. "Development of the neuromuscular junction in the embryo of Drosophila melanogaster." Thesis, University of Cambridge, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.309336.

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Landgraf, Matthias. "Mechanisms underlying the development of neuromuscular connectivity in the Drosophila embryo." Thesis, University of Cambridge, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.627075.

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Pujari, Amit Narahar. "Development and evaluation of vibration apparatus and method for neuromuscular stimulation." Thesis, University of Aberdeen, 2016. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=231217.

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Vibration stimulation has been used as a tool to relieve muscle pain and spasm in physical therapy for many years. However recently, vibration, mainly Whole Body Vibration (WBV), has been increasingly studied and used as an exercise intervention in sports and rehabilitation. Although the physiological mechanisms which guide the body's response to this exercise modality are relatively poorly understood, evidence indicates that vibration can enhance muscle strength, power, and flexibility as well as increase bone mineral density in the general population. Evidence also suggests that the neuromuscular response to vibration stimulation depends on muscle length, stretch level (contraction) along with the vibration characteristics. One way to alter muscle length and contraction levels while receiving vibration is to superimpose the stimulation on graded isometric contraction. However, current WBV device designs cannot facilitate the delivery of vibration stimulation superimposed on graded isometric voluntary contraction. The aim of this PhD project was twofold, firstly to develop and evaluate a prototype WBV device which enables the delivery of vibration stimulation that can be superimposed on graded isometric contraction and secondly, to assess the neuromuscular responses to vibration superimposed on graded isometric contractions in lower limbs using this device. Due to the novelty of the device design and the method of the delivery, this study initially investigated the effects of different vibration frequencies and amplitudes combined with various effort levels on neuromuscular responses in lower limbs. The results of this study confirm that isometric contraction superimposed on vibration stimulation induce enhanced neuromuscular activity in the lower limbs. The results also confirm that although the neuromuscular responses to vibration depend on multiple factors the main determinants seem to be the vibration frequency, amplitude and muscle contraction /forc The results also confirm that although the neuromuscular responses to vibration depend on multiple factors the main determinants seem to be the vibration frequency, amplitude and muscle contraction /force level. Another limitation of most existing vibration devices is that they are not capable of delivering frequency of the vibration independent of amplitude and vice versa. Further, the evidence suggests that vibration amplitude can play an important role in neuromuscular response to vibration, especially when superimposed with graded contraction/force levels. To address the above limitation, the second aim of this PhD project was to develop and evaluate a prototype miniature upper limb vibration device capable of delivering precise and independent vibration frequency and amplitude stimulation. The miniature upper limb vibration (ULV) device with piezo actuators developed for this thesis, enables precise vibration stimulation to be delivered in a seated position with graded voluntary contraction superimposed. The neuromuscular responses to vibration superimposed on graded isometric contractions in upper limbs were also assessed by investigating the fatiguing effects of superimposed vibration stimulation using this newly developed device. This study is the first to investigate and compare the fatiguing effects of superimposed vibration stimulation pre and postvibration exercise in upper limbs. The results of this study confirm that isometric contraction superimposed on vibration stimulation lead to increased fatigue levels and neuromuscular activity in upper limbs. The results also indicate that post-vibration treatment the muscles display enhanced force generation capability associated with lower fatigue levels. In summary, two (WBV and ULV) novel vibration exercise devices were successfully developed and evaluated for this thesis. The results of the studies on these devices confirm that vibration stimulation superimposed on graded isometric contraction can induce higher neuromuscular activity compared to isometric contraction alone in both upper and lower limbs. However the effects of vibration frequency, amplitude and contraction/force levels seem to differ between the upper and lower limbs.
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Côté, Patrice D. "Dystroglycan function in development and neuromuscular disease : a study by gene targeting." Thesis, McGill University, 2001. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=36900.

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The dystrophin associated protein (DAP) complex has been implicated in such basic physiological processes as the maintenance of cellular homeostasis, the assembly of basement membranes (BM), and synaptogenesis. The dystroglycans, alpha and beta, constitute the functional core of the DAP complex since they establish the transmembrane link between dystrophin, whose mutations lead to Duchenne (DMD) or Becker (BMD) muscular dystrophy, and the extracellular matrix (ECM). The alpha and beta subunits of dystroglycan result from posttranslational processing of a single propeptide encoded by DAG1. No clinical cases or animal models have been identified with spontaneous mutations in that gene. Therefore, to gain insight to the function of dystroglycans I have used gene targeting to disrupt the coding sequence of Dag1 in murine embryonic stem (ES) cells. We find that dystroglycans are critical in the early stages of development and that a null mutation in Dag1 results in embryonic lethality at embryonic day (E) 6.5 because of a disruption in Reichert's BM. In culture however, the absence of dystroglycan in ES cells targeted for both Dag1 alleles does not obviously hinder their developmental potential. Consequently, I reasoned that it might be possible to circumvent the early lethality observed in 'classical' knockout mice, by injecting Dag1-null ES cells into wild-type blastocysts to generate chimeric mice only partially devoid of dystroglycan. Several chimeric mice developed to maturity and Dag1 -null ES cells were found to contribute extensively to the hindlimb musculature thus allowing the analysis of dystroglycan depleted muscles. These muscles are severely dystrophic, have low levels of dystrophin and a disrupted residual DAP complex, but have apparently normal BMs. Chimeric muscles also have disrupted neuromuscular junctions. In culture, myotubes derived from Dag1-null ES cells form clusters of acetylcholine receptors (AChRs) but these occupy a surface area three time
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Clark, David Rodney. "Neuromuscular assessment of trunk muscle function in loaded, free barbell back squat : implications for development of trunk stability in dynamic athletic activity." Thesis, University of Stirling, 2018. http://hdl.handle.net/1893/28080.

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Traditional core stability training was developed as a method of treating and preventing back pain. It was however, seamlessly applied to healthy and athletic populations without scientific evidence supporting its efficacy. Traditional core stability focussed on isolating and training the anatomical region between the pelvis and diaphragm, using isometric or low load exercises to enhance spinal stability. Scientific research challenged this approach for healthy function and athletic performance, resulting in a more functional anatomical definition, which included pelvic and shoulder girdles. Hence, a revised definition of dynamic trunk stability; the efficient coordination, transfer and resistance by the trunk, of force and power generated by upper and lower appendicular skeletal extremities during all human movement. This led to an integrated exercise training approach to dynamic trunk stability. Although early evidence suggested loaded compound exercises preformed upright, in particular back squat, were effective in activating and developing trunk muscles, evidence was inconclusive. Accordingly, the aims of this PhD were to investigate neuromuscular trunk function in loaded, free barbell back squat to understand training implications for trunk stability in dynamic athletic activity. Five research studies were conducted; 4 are published and 1 is being prepared for re-submission. The literature review revealed evidence that back squat was an effective method of activating trunk stabilzers and showed that these muscles were load sensitive (study 1). A survey of practitioners reported an understanding and appreciation of the challenge against core stability training for athletic populations. Furthermore, perceptions were aligned with growing evidence for dynamic and functional trunk stability training (study 2). A test-retest neuromuscular study established interday reliability and sensitivity of electromyographical measurement of trunk muscle activity in squats (study 3). Trunk muscle activation in back squat was higher than hack squat at the same relative, but lower absolute loads (study 4). Trunk muscle activation was lower in squats and bodyweight jumps in the strong compared to weak group (study 5). Furthermore, activation of the trunk muscles increased in each 30o segment of squat descent and was highest in first 30o segment of ascent for all loads (study 5). In conclusion, this series of studies confirmed acute effect of squats on trunk stabilizers and demonstrated that external load increases activation in these muscles. Parallel squat depth is important in optimizing trunk muscle activation. Finally, high levels of squat strength result in lower trunk muscle activation in loaded squats and explosive jumps.
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Books on the topic "Neuromuscular development"

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Fondazione Pierfranco e Luisa Mariani. Postgraduate Course. Neuromuscular diseases during development. London: John Libbey, 1997.

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David, Evered, and Whelan Julie, eds. Plasticity of the neuromuscular system. Chichester: Wiley, 1988.

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McGraw, Myrtle B. The neuromuscular maturation of the human infant. London: Mac Keith Press, 1989.

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Budnik, Vivian. The fly neuromuscular junction: Structure and function. 2nd ed. San Diego, Calif: Elsevier/Academic Press, 2006.

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Vivian, Budnik, and Ruiz-Cãnada Catalina, eds. The Fly neuromuscular junction: Structure and function. 2nd ed. San Diego, Calif: Elsevier/Academic Press, 2006.

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W, Winlow, and McCrohan C. R, eds. Growth and plasticity of neural connections. Manchester, UK: Manchester University Press, 1987.

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Standring, Janette. The development and evaluation of a muscle strength and functional assessment protocol foruse in the management of patients with neuromuscular diseases. Manchester: University of Manchester, 1994.

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Yi, Su-rang. Noe sonsang hwanja ŭi undong mabi chaehwal chʻiryo pʻŭrogŭraem i naejang toen hyudaeyong sinʼgyŏng kŭnyuk chŏnʼgi chagŭkki ŭi kaebal mit sangpʻumhwa =: Development of pre-programmed portable neuromuscular electrical stimulator (PPP-NMES) for rehabilitation of motor dysfunction due to brain injury. [Seoul]: Pogŏn Pokchibu, 2007.

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Meade, Vickie. Partners in movement: A family-centered approach to pediatric kinesiology. San Antonio, Tx: Therapy Skill Builders, 1998.

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Takao, Kumazawa, Kruger Lawrence, and Mizumura Kazue, eds. The polymodal receptor: A gateway to pathological pain. Amsterdam: Elsevier, 1996.

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Book chapters on the topic "Neuromuscular development"

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Minns, Robert A. "Neuromotor Development and Examination." In Children's Neuromuscular Disorders, 9–26. London: Springer London, 2011. http://dx.doi.org/10.1007/978-0-85729-552-1_2.

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Heanue, Tiffany A., and Alan J. Burns. "Development of the Enteric Neuromuscular System." In Pediatric Neurogastroenterology, 9–19. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-43268-7_2.

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Fox, Michael A. "Development of the Vertebrate Neuromuscular Junction." In The Sticky Synapse, 39–84. New York, NY: Springer New York, 2009. http://dx.doi.org/10.1007/978-0-387-92708-4_3.

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Heanue, Tiffany A., and Alan J. Burns. "Development of the Enteric Neuromuscular System." In Pediatric Neurogastroenterology, 9–21. Totowa, NJ: Humana Press, 2012. http://dx.doi.org/10.1007/978-1-60761-709-9_2.

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Fahim, M. A. "Neuromuscular Remodelling During Development and Aging." In Biomedical and Life Physics, 505–6. Wiesbaden: Vieweg+Teubner Verlag, 1996. http://dx.doi.org/10.1007/978-3-322-85017-1_51.

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Markovic, Filip, and Elyanne M. Ratcliffe. "Development of the Enteric Neuromuscular System." In Pediatric Neurogastroenterology, 11–19. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-15229-0_2.

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Vrbová, G. "The Role of Activity in the Development of the Mammalian Motor Unit." In Electrical Stimulation and Neuromuscular Disorders, 3–11. Berlin, Heidelberg: Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-71337-8_1.

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Cracco, Cecilia, and Alessandro Vercelli. "Androgen-Dependent Plasticity of a Neuromuscular System." In Development of the Central Nervous System in Vertebrates, 205–16. Boston, MA: Springer US, 1992. http://dx.doi.org/10.1007/978-1-4615-3018-3_15.

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Gordon, Tessa, Linda Bambrick, and Roberto Orozco. "Comparison of Injury and Development in the Neuromuscular System." In Novartis Foundation Symposia, 210–26. Chichester, UK: John Wiley & Sons, Ltd., 2007. http://dx.doi.org/10.1002/9780470513675.ch13.

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Van Epps, Heather, and Yishi Jin. "Development of the Drosophila and C. Elegans Neuromuscular Junctions." In Molecular Mechanisms of Synaptogenesis, 43–65. Boston, MA: Springer US, 2006. http://dx.doi.org/10.1007/978-0-387-32562-0_4.

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Conference papers on the topic "Neuromuscular development"

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Schepelmann, Alexander, Hartmut Geyer, and Michael Taylor. "Development of a Testbed for Robotic Neuromuscular Controllers." In Robotics: Science and Systems 2012. Robotics: Science and Systems Foundation, 2012. http://dx.doi.org/10.15607/rss.2012.viii.049.

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Camarillo–Gómez, Karla A., Gerardo I. Pérez-Soto, and Luis A. Torres-Rico. "Development of a Lower Limb Orthosis to Form Neuromuscular Patterning." In ASME 2014 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. American Society of Mechanical Engineers, 2014. http://dx.doi.org/10.1115/detc2014-35398.

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In this paper, a lower limb orthosis is proposed to form the human gait neuromuscular patterns in patients with myelomeningocele. The orthosis has two lower limbs of 2–DOF each which reduces the motion of the hip and knee to the sagittal plane. The orthosis are assembled in a back support which also supports the patients weight. The control system for the orthosis allows to reproduce in a repetitive, controlled and autonomous way the human gait cycle at different velocities according to the patient requirements; so that, the neuromuscular patterning can be supervised by a therapist. The development of these orthosis seeks to improve the quality of life of those infants with myelomenigocele and to introduce a lower cost Mexican technology with Mexican anthropometric dimensions.
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Ordean, Mircea-Nicolae. "Procedures For Recover Ciphosis -Comparison Between Neuromuscular Electrostimulation And Auditive Feedback-." In ERD 2017 - Education, Reflection, Development, Fourth Edition. Cognitive-Crcs, 2018. http://dx.doi.org/10.15405/epsbs.2018.06.83.

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Sanghyeop Lee and Changhwan Kim. "Development of a robot-aided neuromuscular rehabilitation method using perturbing forces." In 2013 44th International Symposium on Robotics (ISR). IEEE, 2013. http://dx.doi.org/10.1109/isr.2013.6695681.

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Subhash, K. M., P. N. Pournami, and Paul K. Joseph. "Census transform based feature extraction of EMG signals for neuromuscular disease classification." In 2017 IEEE 15th Student Conference on Research and Development (SCOReD). IEEE, 2017. http://dx.doi.org/10.1109/scored.2017.8305432.

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Omairi, Saleh, Antonios Matsakas, Silvia Torelli, and Ketan Patel. "Muscle-specific Expression of Erry in the Myostatin Null Background Leads to the Development of Hypertrophied Oxidative Muscle." In Congenital Dystrophies - Neuromuscular Disorders Precision Medicine: Genomics to Care and Cure. Hamad bin Khalifa University Press (HBKU Press), 2020. http://dx.doi.org/10.5339/qproc.2020.nmd.26.

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Obuchi, Minami, and Ryu Kato. "Development of Hand-Assistance Device using Hand-Joint Orthosis and Neuromuscular Electrical Stimulation." In 2021 43rd Annual International Conference of the IEEE Engineering in Medicine & Biology Society (EMBC). IEEE, 2021. http://dx.doi.org/10.1109/embc46164.2021.9630540.

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Al-dosari, Aldana, Nadin Younes, and Gheyath Nasrallah. "Ecotoxicological assessment of two surfactant on the emryonic development." In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2021. http://dx.doi.org/10.29117/quarfe.2021.0149.

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In this study, zebrafish (Danio rerio) embryos was served as a model for marine fauna to determine if there is any potential of organ-specific toxicity (neuromuscular, hepatic, cytotoxic, and cardiac) caused by Silicone-Q-22 and Ploy-Q-47. as both surfactants are considered eco-friendly corrosion inhibitors. The calculated LC50 of Silicon-Q-22 and Poly-Q-47 was 22.36 and 8.28 mg/L, respectively. At NOEC both surfactants had resulted in teratogenic defects and cardiotoxicity, but only poly Q-47 resulted in neurotoxicity.
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Mehrabi, Naser, and John McPhee. "Steering Feel Improvement for Different Driver Types Using Model-Based Control." In ASME 2014 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. American Society of Mechanical Engineers, 2014. http://dx.doi.org/10.1115/detc2014-34521.

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A realistic driver model can support the development of new steering technologies by reducing the time-consuming trial and error process of designing products. A neuromuscular driver model, by offering physiologically realistic steering maneuvers can provide insights into the task performance and energy consumption of the driver, including fatigue and muscle co-contraction. Here, two muscles are used in a simplified neuromuscular driver model. To study the effect of driver’s characteristics such as age, gender and physical ability on steering, the muscle parameters are adjusted to represent a particular population. Then, this modified driver model is used to to tune the Electric Power Steering (EPS) assist curves for that particular population.
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Kulkarni, Vedant, Jon Davids, and Anita Bagley. "Smartphone App to Enable Community-based Surveillance for Neuromuscular Hip Dysplasia in Children with Cerebral Palsy: Development and Application." In Selection of Abstracts From NCE 2016. American Academy of Pediatrics, 2018. http://dx.doi.org/10.1542/peds.141.1_meetingabstract.3.

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