Relevant bibliographies by topics / Neuroglia

Academic literature on the topic 'Neuroglia'

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Published: 4 June 2021
Last updated: 31 July 2024

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Journal articles on the topic "Neuroglia"

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Salkov, V. N., D. N. Voronkov, and V. S. Sukhorukov. "Quantitative changes in ferritin-containing glia in the structures of the substantia nigra in aging and Parkinson’s disease." CLINICAL AND EXPERIMENTAL MORPHOLOGY 13, no. 2 (2024): 20–25. http://dx.doi.org/10.31088/cem2024.13.2.20-25.

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Introduction. Iron accumulates in the substantia nigra (SN) in aging and Parkinson’s disease (PD). However, there is a distinct lack of information about the changes in the metabolism of ferritin–an iron-binding protein in nigral cells–in aging and PD. The aim of the study was to quantify the changes in the number of H- and L-ferritin glia in the SN structures in aging and PD. Materials and methods. We examined autopsies of PD patients (5 cases), mature and elderly people (6 cases), as well as senile people (5 cases). Immunohistochemistry and light microscopy were used to study the location of H- and L-ferritin chains in the SN structures. The density of H– and L–ferritin-containing neuroglia was determined with computer morphometry. Results. In all cases, ferritin was accumulated predominantly in the reticular part of the SN in unpigmented neurons and neuroglial cells. The density of H– and L–ferritin-containing neuroglia in the SN of PD patients and senile people was significantly higher compared to that in mature and elderly people. The same differences between the groups of PD patients and elderly people were found only for the density of H–ferritin-containing neuroglia. Conclusion. The differences revealed between the age groups in the density of H– and L–ferritin-containing neuroglia characterize their increase with age and correspond to the accumulation of iron in the SN during aging. The differences revealed with the same parameters between patients with PD and mature, elderly, and senile patients characterize the imbalance of iron accumulation and oxidation processes in ferritin-containing glial cells of patients with PD. Keywords: aging, Parkinson’s disease, substantia nigra, immunohistochemistry, morphometric, ferritin, neuroglia
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Miller, R. H. "Neuroglia." Neurology 48, no. 2 (February 1, 1997): 560. http://dx.doi.org/10.1212/wnl.48.2.560-a.

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Min, Kyung-Whan. "Neuroglia." JAMA: The Journal of the American Medical Association 276, no. 10 (September 11, 1996): 837. http://dx.doi.org/10.1001/jama.1996.03540100081038.

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Smolič, Tina, Robert Zorec, and Nina Vardjan. "Pathophysiology of Lipid Droplets in Neuroglia." Antioxidants 11, no. 1 (December 23, 2021): 22. http://dx.doi.org/10.3390/antiox11010022.

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In recent years, increasing evidence regarding the functional importance of lipid droplets (LDs), cytoplasmic storage organelles in the central nervous system (CNS), has emerged. Although not abundantly present in the CNS under normal conditions in adulthood, LDs accumulate in the CNS during development and aging, as well as in some neurologic disorders. LDs are actively involved in cellular lipid turnover and stress response. By regulating the storage of excess fatty acids, cholesterol, and ceramides in addition to their subsequent release in response to cell needs and/or environmental stressors, LDs are involved in energy production, in the synthesis of membranes and signaling molecules, and in the protection of cells against lipotoxicity and free radicals. Accumulation of LDs in the CNS appears predominantly in neuroglia (astrocytes, microglia, oligodendrocytes, ependymal cells), which provide trophic, metabolic, and immune support to neuronal networks. Here we review the most recent findings on the characteristics and functions of LDs in neuroglia, focusing on astrocytes, the key homeostasis-providing cells in the CNS. We discuss the molecular mechanisms affecting LD turnover in neuroglia under stress and how this may protect neural cell function. We also highlight the role (and potential contribution) of neuroglial LDs in aging and in neurologic disorders.
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Chvátal, Alexandr, and Alexei Verkhratsky. "An Early History of Neuroglial Research: Personalities." Neuroglia 1, no. 1 (August 16, 2018): 245–81. http://dx.doi.org/10.3390/neuroglia1010016.

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Neuroscience, like most other divisions of natural philosophy, emerged in the Hellenistic world following the first experimental discoveries of the nerves connecting the brain with the body. The first fundamental doctrine on brain function highlighted the role for a specific substance, pneuma, which appeared as a substrate for brain function and, being transported through the hollow nerves, operated the peripheral organs. A paradigm shift occurred in 17th century when brain function was relocated to the grey matter. Beginning from the end of the 18th century, the existence of active and passive portions of the nervous tissue were postulated. The passive part of the nervous tissue has been further conceptualised by Rudolf Virchow, who introduced the notion of neuroglia as a connective tissue of the brain and the spinal cord. During the second half of the 19th century, the cellular architecture of the brain was been extensively studied, which led to an in-depth morphological characterisation of multiple cell types, including a detailed description of the neuroglia. Here, we present the views and discoveries of the main personalities of early neuroglial research.
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Verkhratsky, Alexei, and Vladimir Parpura. "Introduction to Neuroglia." Colloquium Series on Neuroglia in Biology and Medicine: From Physiology to Disease 1, no. 1 (February 24, 2014): 1–74. http://dx.doi.org/10.4199/c00102ed1v01y201401ngl001.

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Stout, Randy F., and Navin Pokala. "Neuroglia inC. elegans." Colloquium Series on Neuroglia in Biology and Medicine: From Physiology to Disease 5, no. 1 (February 27, 2018): i—56. http://dx.doi.org/10.4199/c00160ed1v01y201712ngl011.

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Jellinger, K. A. "Neuroglia, 2nd edn." European Journal of Neurology 18, no. 12 (July 28, 2011): e154-e154. http://dx.doi.org/10.1111/j.1468-1331.2011.03492.x.

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Somjen, G. G. "The Neuroglia Mystery." Science 260, no. 5116 (June 25, 1993): 1984–85. http://dx.doi.org/10.1126/science.260.5116.1984.

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Heneka, Michael T., José J. Rodríguez, and Alexei Verkhratsky. "Neuroglia in neurodegeneration." Brain Research Reviews 63, no. 1-2 (May 2010): 189–211. http://dx.doi.org/10.1016/j.brainresrev.2009.11.004.

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Dissertations / Theses on the topic "Neuroglia"

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Edwards, James Roy. "Modelling Chemical Communication in Neuroglia." Thesis, The University of Sydney, 2007. http://hdl.handle.net/2123/2184.

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In vivo many forms of glia utilise both intercellular and extracellular pathways in the form of IP3 permeable gap junctions and cytoplasmic ATP diffusion to produce calcium waves. We introduce a model of ATP and Ca2+ waves in clusters of glial cells in which both pathways are included. Through demonstrations of its capacity to replicate the results of existing theoretical models of individual pathways and to simulate experimental observations of retinal glia the validity of the model is confirmed. Characteristics of the waves resulting from the inclusion of both pathways are identified and described.
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Edwards, James Roy. "Modelling Chemical Communication in Neuroglia." University of Sydney, 2007. http://hdl.handle.net/2123/2184.

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Master of Science
In vivo many forms of glia utilise both intercellular and extracellular pathways in the form of IP3 permeable gap junctions and cytoplasmic ATP diffusion to produce calcium waves. We introduce a model of ATP and Ca2+ waves in clusters of glial cells in which both pathways are included. Through demonstrations of its capacity to replicate the results of existing theoretical models of individual pathways and to simulate experimental observations of retinal glia the validity of the model is confirmed. Characteristics of the waves resulting from the inclusion of both pathways are identified and described.
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Tomac, Andreas C. "Glial cell line-derived neurotrophic factor : expression patterns, neuronal transport, regulation, effects and receptor dependence /." Stockholm, 1998. http://diss.kib.ki.se/search/diss.se.cfm?19980618toma.

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Fung, Chun-kit. "In vitro and in vivo studies of skin-derived Schwann cells in nerve regeneration." Click to view the E-thesis via HKUTO, 2010. http://sunzi.lib.hku.hk/hkuto/record/B43936027.

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Гринцова, Наталія Борисівна, Наталия Борисовна Гринцова, and Nataliia Borysivna Hryntsova. "Реакція нейроглії кори мозочка за умов впливу на організм сульфатів міді, цинку та заліза." Thesis, Сумський державний університет, 2015. http://essuir.sumdu.edu.ua/handle/123456789/42239.

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Jennings, Alison Ruth. "Oligodendrocyte progenitor cells : from experimental remyelination to multiple sclerosis." University of Western Australia. School of Surgery and Pathology, 2007. http://theses.library.uwa.edu.au/adt-WU2008.0047.

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In experimental models of demyelination such as cat optic nerve injected with antibody to galactocerebroside, stepwise and ultimately full repair occurs, starting with recruitment of oligodendrocyte progenitor cells (OP) from surrounding tissue and culminating in remyelination by young competent oligodendrocytes. Endogenous repair of demyelination can also occur in the adult human central nervous system, as evidenced by remyelinated shadow plaques in MS, but ultimately fails in this disease, leading to areas of chronic demyelination where surviving axons are both dysfunctional in terms of conduction and vulnerable to ongoing damage. In order to meaningfully investigate this failure of remyelination in the human situation, an essential prerequisite is to be able to reliably identify the neuroglial cells, and in particular, oligodendrocyte lineage cells, involved in the repair pathway in situ in post mortem tissue. While some marker antigens have been shown to remain demonstrable despite autolytic change and through differing fixation levels, others are far more sensitive and only reliable in freshly obtained tissue with light fixation. For instance, the surface antigens NG2 and PDGFαR, which have been widely used in experimental studies as a marker for OP both in vivo and in vitro, have been shown to be adversely affected by both fixation and autolysis. To this end, the cat optic nerve demyelination model, in which the reparative oligodendrocyte lineage stages have been antigenically defined, was extended to normal optic nerve including lightly fixed tissue. Here, NG2, PDGFαR and the oligodendrocyte lineage transcription factors Olig1 and Olig2 were able to be demonstrated and then correlated with the existing antigenic phenotypes. Subsequently, normal human optic nerve, optimised for both morphological preservation and antigen retention, was used to develop an in vivo staining profile for all neuroglia including OP, that was then applied to conventionally prepared, normal and MS tissue. It was found that, with careful attention to technical parameters such as post mortem interval and details of fixation, OP and other stages of the remyelinating oligodendrocyte lineage could be identified in such material, resulting in meaningful insight into the repair status of the three MS samples studied.
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Yang, Luping. "Molecular mechanisms of TAR-independent regulation by HIV-1 tat in central nervous system-derived glial cells." Diss., Georgia Institute of Technology, 1996. http://hdl.handle.net/1853/25338.

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楊鐸輝 and Tok-fai Vincent Yeung. "Aspects of the biological interactions between natriuretic peptides and cultured glial cells." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1997. http://hub.hku.hk/bib/B31981665.

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Leung, Ho-yan, and 梁可昕. "A study of membrane-bound neuregulin in mediating fate commitment of Schwann cell-like cells." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2013. http://hdl.handle.net/10722/193482.

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Central nervous system injuries often lead to devastating consequences due to an unfavourable environment created after the injury. Current treatments have yet to address the environment for improved prospects of functional recovery. Transplantation of Schwann cells into the lesion site could in part address the issue, promoting nerve regeneration and enhancing functional recovery. Bone marrow stromal cells (BMSCs) promise to be a viable, autologous source for Schwann cell derivation. Fate-committed Schwann cells derived from BMSCs through co-culture with purified dorsal root ganglia (DRG) neurons suggest that the DRG neurons present juxtacrine cues that direct commitment to the Schwann cell fate. We hypothesize that Neuregulin 1 type III (NRG1(III)) is one such juxtacrine cue to which BMSC-derived Schwann cell-like cells (SCLC) respond in the switch to fate commitment. In this study, NRG1(III) was found to be expressed on freshly isolated DRG neurons and that SCLCs expressed both the ErbB2 and 3 receptors. Western blot analysis for phosphorylated Akt and MAPK provided indicators of downstream signalling of NRG1/ErbB complexes. We then tested if both the soluble and membrane bound forms of NRG1 mediate SCLC differentiation towards fate commitment. In contrast to the membrane-bound form on DRG neurons, soluble NRG1 failed to direct the SCLCs towards the Schwann cell fate. HEK293T cells that stably overexpress NRG1(III) were generated and tested as a neuronal surrogate that presents NRG1(III) on the cell surface. In a 5-day co-culture system with HEK293TNrg1(III) cells, SCLCs were found to develop elongated processes, acquiring either unipolar or bipolar morphology that resembles that of Schwann cells. Screening for marker expression by RT-PCR suggested that at this stage of morphological transition, SCLCs were not yet committed to the Schwann cell fate. The co-culture system will be pursued to find ex vivo conditions that direct differentiation of BMSC-derived SCLCs to fate-committed Schwann cells.
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Biochemistry
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Sitnikov, Sergey. "Activity dependent neuron-glia interactions in health and disease." Thesis, University of Cambridge, 2015. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.708663.

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Books on the topic "Neuroglia"

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R, Ransom Bruce, ed. Neuroglia. 3rd ed. New York: Oxford University Press, 2013.

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Helmut, Kettenmann, and Ransom Bruce R, eds. Neuroglia. 2nd ed. New York: Oxford University Press, 2005.

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Helmut, Kettenmann, and Ransom Bruce R, eds. Neuroglia. New York: Oxford University Press, 1995.

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Verkhratsky, Alexei, Margaret S. Ho, Robert Zorec, and Vladimir Parpura, eds. Neuroglia in Neurodegenerative Diseases. Singapore: Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-9913-8.

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Parpura, Vladimir, and Alexei Verkhratsky, eds. Pathological Potential of Neuroglia. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-0974-2.

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de Vellis, Jean. Neuroglia in the Aging Brain. New Jersey: Humana Press, 2001. http://dx.doi.org/10.1385/1592591051.

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1957-, Castellano Bernardo, González Berta 1955-, and Nieto-Sampedro Manuel 1944-, eds. Understanding glial cells. Boston: Kluwer Academic, 1998.

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Bloom, Ona. Cells of the nervous system. Philadelphia: Chelsea House Publishers, 2005.

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Reichenbach, Andreas. Müller cells in the healthy and diseased retina. New York: Springer, 2010.

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Michael, Aschner, and Kimelberg Harold K, eds. The role of glia in neurotoxicity. Boca Raton: CRC Press, 1996.

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Book chapters on the topic "Neuroglia"

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Sun, Dong. "Neuroglia." In Encyclopedia of Clinical Neuropsychology, 2405–9. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-57111-9_341.

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y Cajal, Santiago Ramón. "Neuroglia." In Texture of the Nervous System of Man and the Vertebrates, 205–23. Vienna: Springer Vienna, 1999. http://dx.doi.org/10.1007/978-3-7091-6435-8_8.

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Sun, Dong. "Neuroglia." In Encyclopedia of Clinical Neuropsychology, 1–5. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-56782-2_341-3.

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Sun, Dong. "Neuroglia." In Encyclopedia of Clinical Neuropsychology, 1746–49. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-0-387-79948-3_341.

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Verkhratsky, Alexei, Margaret S. Ho, and Vladimir Parpura. "Evolution of Neuroglia." In Neuroglia in Neurodegenerative Diseases, 15–44. Singapore: Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-9913-8_2.

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Verkhratsky, Alexei, Robert Zorec, Jose Julio Rodriguez-Arellano, and Vladimir Parpura. "Neuroglia in Ageing." In Neuroglia in Neurodegenerative Diseases, 181–97. Singapore: Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-9913-8_8.

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Kettenmann, Helmut, and Alex Verkhratsky. "Glial Cells: Neuroglia." In Neuroscience in the 21st Century, 547–78. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3474-4_19.

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Kettenmann, Helmut, and Alexei Verkhratsky. "Glial Cells: Neuroglia." In Neuroscience in the 21st Century, 825–60. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-88832-9_19.

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Kettenmann, Helmut, and Alexei Verkhratsky. "Glial Cells: Neuroglia." In Neuroscience in the 21st Century, 1–36. New York, NY: Springer New York, 2021. http://dx.doi.org/10.1007/978-1-4614-6434-1_19-3.

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Verkhratsky, Alexei, Margaret S. Ho, Robert Zorec, and Vladimir Parpura. "The Concept of Neuroglia." In Neuroglia in Neurodegenerative Diseases, 1–13. Singapore: Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-9913-8_1.

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Conference papers on the topic "Neuroglia"

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Anggraeni, Nilam, Kristanti Wanito Wigati, I. Lukitra Wardani, and Lilik Herawati. "High-Calorie Diet Reduces Neuroglia Count." In Surabaya International Physiology Seminar. SCITEPRESS - Science and Technology Publications, 2017. http://dx.doi.org/10.5220/0007335001690173.

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Conte, Giorgia. "Nanoglial interfaces as neuroglia biosensor and biomodulator." In Emerging Imaging and Sensing Technologies for Security and Defence VII, edited by Andrea Camposeo, Maria Farsari, Luana Persano, Lynda E. Busse, John G. Rarity, Paul M. Alsing, Michael L. Fanto, et al. SPIE, 2022. http://dx.doi.org/10.1117/12.2656311.

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Pompa, Marcello, Gennaro Tartarisco, Simona Panunzi, Laura D'Orsi, Alessandro Borri, and Andrea De Gaetano. "A Neuronal Circuit Simulation Highlights the Role of Neuroglia in Modulating Information Transmission." In 2023 IEEE International Conference on Systems, Man, and Cybernetics (SMC). IEEE, 2023. http://dx.doi.org/10.1109/smc53992.2023.10394557.

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Wu, Xuehai, John G. Georgiadis, and Assimina A. Pelegri. "Brain White Matter Model of Orthotropic Viscoelastic Properties in Frequency Domain." In ASME 2019 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2019. http://dx.doi.org/10.1115/imece2019-12182.

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Abstract Finite element analysis is used to study brain axonal injury and develop Brain White Matter (BWM) models while accounting for both the strain magnitude and the strain rate. These models are becoming more sophisticated and complicated due to the complex nature of the BMW composite structure with different material properties for each constituent phase. State-of-the-art studies, focus on employing techniques that combine information about the local axonal directionality in different areas of the brain with diagnostic tools such as Diffusion-Weighted Magnetic Resonance Imaging (Diffusion-MRI). The diffusion-MRI data offers localization and orientation information of axonal tracks which are analyzed in finite element models to simulate virtual loading scenarios. Here, a BMW biphasic material model comprised of axons and neuroglia is considered. The model’s architectural anisotropy represented by a multitude of axonal orientations, that depend on specific brain regions, adds to its complexity. During this effort, we develop a finite element method to merge micro-scale Representative Volume Elements (RVEs) with orthotropic frequency domain viscoelasticity to an integrated macro-scale BWM finite element model, which incorporates local axonal orientation. Previous studies of this group focused on building RVEs that combined different volume fractions of axons and neuroglia and simulating their anisotropic viscoelastic properties. Via the proposed model, we can assign material properties and local architecture on each element based on the information from the orientation of the axonal traces. Consecutively, a BWM finite element model is derived with fully defined both material properties and material orientation. The frequency-domain dynamic response of the BMW model is analyzed to simulate larger scale diagnostic modalities such as MRI and MRE.
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Haghiri, Saeed, Arash Ahmadi, Moslem Nouri, and Moslem Heidarpur. "An investigation on neuroglial interaction effect on Izhikevich neuron behaviour." In 2014 22nd Iranian Conference on Electrical Engineering (ICEE). IEEE, 2014. http://dx.doi.org/10.1109/iraniancee.2014.6999509.

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Salah, Samia, M'hamed Hadj sadok, and Abderrezak Guessoum. "Development of the new Neuroglial approach: Application on singularly perturbed systems." In 2013 World Congress on Computer and Information Technology (WCCIT). IEEE, 2013. http://dx.doi.org/10.1109/wccit.2013.6618682.

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"Neurogrid: Emulating a Million Neurons in the Cortex." In Conference Proceedings. Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2006. http://dx.doi.org/10.1109/iembs.2006.260925.

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Shivers, Michael Quincy. "Abstract 3323: Anticancer activity of medicinal plant and neuroglioma." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-3323.

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Kolosov, M. S., D. E. Bragin, A. Kohany, and Anatoly B. Uzdensky. "Neuroglial relationships in the crayfish stretch receptor under photodynamic injury: changes in the nuclear morphology." In SPIE Proceedings, edited by Valery V. Tuchin. SPIE, 2003. http://dx.doi.org/10.1117/12.518872.

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Baskaran, Avinash, Sujata Basyal, Brendon C. Allen, and Chad G. Rose. "NeuroGAIN: Neuromechanical Generative Demand Forecasting Toward Optimal Control of Soft Hand Exoskeletons." In 2024 International Symposium on Medical Robotics (ISMR). IEEE, 2024. http://dx.doi.org/10.1109/ismr63436.2024.10585782.

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