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Relevant bibliographies by topics / Neuroendocrinology

Academic literature on the topic 'Neuroendocrinology'

Author: Grafiati

Published: 4 June 2021

Last updated: 30 July 2024

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Journal articles on the topic "Neuroendocrinology"

1

Simard, Marie F., Harold E. Carlson, and Shereen Ezzat. "Neuroendocrinology." Neurosurgical Focus 16, no. 4 (April 2004): 1. http://dx.doi.org/10.3171/foc.2004.16.4.1.

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Kohler, Peter O. "Neuroendocrinology." Current Opinion in Endocrinology and Diabetes 1, no. 1 (January 1994): 71–78. http://dx.doi.org/10.1097/00060793-199400010-00015.

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Smith, M. Sysan. "Neuroendocrinology." Current Opinion in Endocrinology and Diabetes 2, no. 2 (April 1995): 145–47. http://dx.doi.org/10.1097/00060793-199504000-00012.

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Smith, M. Susan. "Neuroendocrinology." Current Opinion in Endocrinology and Diabetes 3, no. 2 (April 1996): 147–48. http://dx.doi.org/10.1097/00060793-199604000-00012.

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&NA;. "Neuroendocrinology." Current Opinion in Endocrinology and Diabetes 3, no. 2 (April 1996): 845. http://dx.doi.org/10.1097/00060793-199604000-00021.

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&NA;. "Neuroendocrinology." Current Opinion in Endocrinology and Diabetes 4, no. 2 (April 1997): B72. http://dx.doi.org/10.1097/00060793-199704000-00019.

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Cameron, Judy L. "Neuroendocrinology." Current Opinion in Endocrinology and Diabetes 5, no. 1 (February 1998): 49–51. http://dx.doi.org/10.1097/00060793-199802000-00009.

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Cameron, Judy L. "Neuroendocrinology." Current Opinion in Endocrinology & Diabetes 6, no. 1 (February 1999): 1. http://dx.doi.org/10.1097/00060793-199902000-00001.

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Kaiser, Ursula. "Neuroendocrinology." Current Opinion in Endocrinology & Diabetes 7, no. 3 (June 2000): 151–52. http://dx.doi.org/10.1097/00060793-200006000-00008.

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Cookson, John. "Neuroendocrinology." International Clinical Psychopharmacology 3, no. 1 (January 1988): 88–89. http://dx.doi.org/10.1097/00004850-198801000-00009.

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Dissertations / Theses on the topic "Neuroendocrinology"

1

Ghuman, Sarvpreet Singh. "Neuroendocrinology of stress and reproduction interaction." Thesis, University of Liverpool, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.403220.

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Claudiano, Gustavo da Silva [UNESP]. "Aspectos da fisiopatologia da sepse em Piaractus mesopotamicus induzida por Aeromonas hydrophila." Universidade Estadual Paulista (UNESP), 2015. http://hdl.handle.net/11449/128025.

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Made available in DSpace on 2015-10-06T13:02:55Z (GMT). No. of bitstreams: 0 Previous issue date: 2015-06-30. Added 1 bitstream(s) on 2015-10-06T13:19:18Z : No. of bitstreams: 1 000849627.pdf: 553636 bytes, checksum: 5dfc24590b221b9994b1d94923ed02d2 (MD5)
A patogenia da sepse envolve múltiplas inter-relações na dinâmica entre os diversos componentes relacionados ao hospedeiro e ao patógeno, com altas taxas de mortalidade em várias espécies animais. Embora seja uma das principais causas de mortalidade de peixes, há escassez de literatura sobre seus fenômenos fisiopatológicos e em seus mecanismos de modulação. Assim, neste trabalho, objetivou-se estudar os aspectos fisiopatológicos da evolução da reposta séptica induzida por Aeromonas hydrophila em Piaractus mesopotamicus - pacus. O inóculo utilizada foi determinada pela DL50 (DL50-96h) e estimada em 1,78 x 109. A sepse foi induzida pela inoculação na cavidade celomática e seguiram-se as coletas de sangue nos tempos pré-determinados de 1, 3, 6 e 9 horas após indução e o grupo controle. Verificou-se rápido aumento da cortisolemia com inibição da absorção de glicose, seguido de hipocortisolemia e hiperglicemia. Os hormônios da tireoide, apresentando diminuição imediata de suas concentrações séricas T3 e T4. Este último apresentou aumento 6 HPI. As alterações hormonais induzidas pela sepse desencadearam alterações nas vias metabólicas com aumento do catabolismo protéico e lipídico, utilização da via da glicólise anaeróbica transitória e lesão hepática. O leucograma demonstrou leucopenia e trombocitopenia seguido de cessar da quimiotaxia dos leucócitos após 6 HPI e graves alterações morfológicas em leucócitos e eritrócitos. As variáveis do sistema imune inato apresentaram aumento da produção de EROs 3 HPI, seguido de diminuição, sem alteração da concentração da lisozima sérica e progressivo aumento do ALS e AAB. Após inoculação ip. de A. hydrophila, foram evidenciados sinais clínicos de aeromonose, bacteremia crescente e sobrevida de 57,14 % depois 36 HPI
The pathogenesis of sepsis involves multiple interrelationships in the dynamics between various components related to the host and the pathogen, with high mortality rates in several animal species. Although it is a major cause of fish deaths, there is lack of literature about its physiopathology and the mechanisms of modulation. This work aimed to study the physiopathological aspects of the evolution of the septic response induced by Aeromonas hydrophila in Piaractus mesopotamicus - pacu. The dose was determined by the LD50 (LD50-96h) and estimated in 1,78 x 109. Sepsis was induced by the administration in the coelomic cavity and was followed by the collection of blood in the pre-determined times of 1, 3, 6, and 9 hours after induction and more control. There was a rapid increase of cortisol concentration in plasma, inhibition of glucose uptake, followed by hypocortisolemia and hyperglycemia. Thyroid hormones have immediate reduction of serum concentration of T3 and T4. The latter showed an increase after 6 HPI. Hormonal changes induced by sepsis-triggered changes in metabolic pathways enhancing protein and lipid catabolism, use of transient anaerobic glycolysis via and liver damage. The leucocyte count showed leukopenia and thrombopenia followed by cessation of leukocyte migration after 6 HPI and severe morphological changes in leukocytes and erythrocytes. The innate immune variables showed increased production of ROS after 3 HPI, followed by reduction without changing the concentration of serum lysozyme and progressive increase in SHA and ABA. After ip inoculation of A. hydrophila in pacu, clinical signs were evident of Aeromonas infection, growing bacteremia and survival of 57,14 % after 36 HPI

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Claudiano, Gustavo da Silva. "Aspectos da fisiopatologia da sepse em Piaractus mesopotamicus induzida por Aeromonas hydrophila /." Jaboticabal, 2015. http://hdl.handle.net/11449/128025.

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Orientador: Flávio Ruas de Moraes
Coorientador: Julieta Rodini Egracia de Moraes
Coorientador: Cleni Mara Marzocchi-Machado
Banca: Aureo Evangelista Santana
Banca: Rogério Salvador
Banca: Luiz Lehmann Coutinho
Banca: Antonio Vicente Mundim
Resumo: A patogenia da sepse envolve múltiplas inter-relações na dinâmica entre os diversos componentes relacionados ao hospedeiro e ao patógeno, com altas taxas de mortalidade em várias espécies animais. Embora seja uma das principais causas de mortalidade de peixes, há escassez de literatura sobre seus fenômenos fisiopatológicos e em seus mecanismos de modulação. Assim, neste trabalho, objetivou-se estudar os aspectos fisiopatológicos da evolução da reposta séptica induzida por Aeromonas hydrophila em Piaractus mesopotamicus - pacus. O inóculo utilizada foi determinada pela DL50 (DL50-96h) e estimada em 1,78 x 109. A sepse foi induzida pela inoculação na cavidade celomática e seguiram-se as coletas de sangue nos tempos pré-determinados de 1, 3, 6 e 9 horas após indução e o grupo controle. Verificou-se rápido aumento da cortisolemia com inibição da absorção de glicose, seguido de hipocortisolemia e hiperglicemia. Os hormônios da tireoide, apresentando diminuição imediata de suas concentrações séricas T3 e T4. Este último apresentou aumento 6 HPI. As alterações hormonais induzidas pela sepse desencadearam alterações nas vias metabólicas com aumento do catabolismo protéico e lipídico, utilização da via da glicólise anaeróbica transitória e lesão hepática. O leucograma demonstrou leucopenia e trombocitopenia seguido de cessar da quimiotaxia dos leucócitos após 6 HPI e graves alterações morfológicas em leucócitos e eritrócitos. As variáveis do sistema imune inato apresentaram aumento da produção de EROs 3 HPI, seguido de diminuição, sem alteração da concentração da lisozima sérica e progressivo aumento do ALS e AAB. Após inoculação ip. de A. hydrophila, foram evidenciados sinais clínicos de aeromonose, bacteremia crescente e sobrevida de 57,14 % depois 36 HPI
Abstract: The pathogenesis of sepsis involves multiple interrelationships in the dynamics between various components related to the host and the pathogen, with high mortality rates in several animal species. Although it is a major cause of fish deaths, there is lack of literature about its physiopathology and the mechanisms of modulation. This work aimed to study the physiopathological aspects of the evolution of the septic response induced by Aeromonas hydrophila in Piaractus mesopotamicus - pacu. The dose was determined by the LD50 (LD50-96h) and estimated in 1,78 x 109. Sepsis was induced by the administration in the coelomic cavity and was followed by the collection of blood in the pre-determined times of 1, 3, 6, and 9 hours after induction and more control. There was a rapid increase of cortisol concentration in plasma, inhibition of glucose uptake, followed by hypocortisolemia and hyperglycemia. Thyroid hormones have immediate reduction of serum concentration of T3 and T4. The latter showed an increase after 6 HPI. Hormonal changes induced by sepsis-triggered changes in metabolic pathways enhancing protein and lipid catabolism, use of transient anaerobic glycolysis via and liver damage. The leucocyte count showed leukopenia and thrombopenia followed by cessation of leukocyte migration after 6 HPI and severe morphological changes in leukocytes and erythrocytes. The innate immune variables showed increased production of ROS after 3 HPI, followed by reduction without changing the concentration of serum lysozyme and progressive increase in SHA and ABA. After ip inoculation of A. hydrophila in pacu, clinical signs were evident of Aeromonas infection, growing bacteremia and survival of 57,14 % after 36 HPI
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4

Honaramooz, Ali. "Neuroendocrinology of gonadotrophin secretion in prepubertal heifers." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp03/NQ37889.pdf.

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Burmeister, Sabrina Suzanne. "Behavioral neuroendocrinology of the green treefrog (Hyla cinerea) /." Full text (PDF) from UMI/Dissertation Abstracts International, 2001. http://wwwlib.umi.com/cr/utexas/fullcit?p3008291.

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Gutzler, Stephanie. "The Neuroendocrinology of Seasonal Aggression in Female Syrian Hamsters." Digital Archive @ GSU, 2009. http://digitalarchive.gsu.edu/biology_diss/61.

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Aggression is a feature of many clinical disorders including autism, Alzheimer’s disease, bipolar disorder, and schizophrenia. The available treatment options act to prevent impulsive aggression through modulation of GABAergic and dopaminergic pathways which come with metabolic and dyskinetic side effects. The mechanism underlying aggressive motivation, however, has not been elucidated. In addition, previous studies have been heavily biased towards males of various species. Mimicking changes in day length, or photoperiod, in the laboratory is a natural manipulation used to examine seasonal changes in aggressive behavior in many species. In response to the reduction in the duration of light exposure, animals undergo gonadal regression and become reproductively quiescent. During this non-breeding season in male photoperiod-responsive animals, aggressive behavior increases significantly. Although studies have shown offensive aggression remains elevated in female rodents, seasonal regulation of this behavior in females has not been thoroughly studied. The neuropeptide arginine-vasopressin (AVP) has been implicated in the facilitation of aggressive behavior in male rodents and fishes; therefore, it is useful to examine AVP as a modulator of seasonal aggression in females. Because the actions of AVP in female social behavior may be hormonally-dependent, we investigated the hormonal mechanisms that regulate the expression of AVP receptors and the behavioral actions of AVP on aggression. In addition to changes in gonadal steroid hormones during the non-breeding season, we identified photoperiod-dependent alterations in adrenal hormone secretion as AVP plays a role in regulation of hypothalamic-pituitary-adrenal axis (HPA) activity and anxiety-like behaviors in animal models.

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Sunnasy, Dharmadeho. "Stress related changes in urinary biogenic amines in humans." Thesis, University of Greenwich, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.307885.

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Herbison, Allan E. "The medical preoptic gaba system and its role in regulating luteinising hormone secretion." Thesis, University of Cambridge, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.385415.

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Bornstein, Stefan R. "From Neuroendocrinology to Neuroimmunomodulation – A Tribute to Prof. Dr. Samuel McCann." Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2014. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-135755.

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One of the leading experts in the field of Neuroendocrinology and Neuroimmunmodulation, Samuel Mac Donald McCann, known by all his friends as ‘Don’, passed away in 2007. This article pays tribute to his outstanding scientific contribution and a glimpse on his fascinating personality. A member of the National Academy of Sciences of the United States and pioneer in the field of neuroendocrine regulation, he identified numerous hormones and peptides and set the stage for basic concepts in physiology and clinical medicine
Dieser Beitrag ist mit Zustimmung des Rechteinhabers aufgrund einer (DFG-geförderten) Allianz- bzw. Nationallizenz frei zugänglich

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McCann, John Patrick. "Pulmonary neuroendocrinology in health and disease : an immunocytochemical and radioimmunoassay study." Thesis, Queen's University Belfast, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.259343.

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Books on the topic "Neuroendocrinology"

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Lovejoy, David A. Neuroendocrinology. Chichester, UK: John Wiley & Sons, Ltd, 2005. http://dx.doi.org/10.1002/0470027878.

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Korf, Horst-Werner, and Klaus-Henning Usadel, eds. Neuroendocrinology. Berlin, Heidelberg: Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-642-60915-2.

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L, Barrow Daniel, Selman Warren R, and Congress of Neurological Surgeons, eds. Neuroendocrinology. Baltimore: Williams & Wilkins, 1992.

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Anne, Klibanski, ed. Neuroendocrinology. Philadelphia: Saunders, 2001.

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J, Everitt Barry, and Lightman Stafford L, eds. Neuroendocrinology. Oxford: Blackwell Scientific, 1986.

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B, Nemeroff Charles, ed. Neuroendocrinology. Boca Raton: CRC Press, 1992.

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Sandro, Loche, ed. Pediatric neuroendocrinology. Basel: Karger, 2010.

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Murphy, David, and Harold Gainer, eds. Molecular Neuroendocrinology. Chichester, UK: John Wiley & Sons, Ltd, 2016. http://dx.doi.org/10.1002/9781118760369.

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MacGregor, Duncan J., and Gareth Leng, eds. Computational Neuroendocrinology. Chichester, UK: John Wiley & Sons, Ltd, 2016. http://dx.doi.org/10.1002/9781119159438.

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Wray, Susan, and Seth Blackshaw, eds. Developmental Neuroendocrinology. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-40002-6.

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Book chapters on the topic "Neuroendocrinology"

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Krieger, Dorothy T. "Neuroendocrinology." In Contemporary Endocrinology, 1–26. Boston, MA: Springer US, 1985. http://dx.doi.org/10.1007/978-1-4684-4817-7_1.

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Huijbregts, Lukas, Lucie Chevrier, and Nicolas de Roux. "Neuroendocrinology." In Yearbook of Pediatric Endocrinology 2012, 1–11. Basel: S. KARGER AG, 2012. http://dx.doi.org/10.1159/000341200.

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Villanueva, Carine, Lukas Huihjbregts, and Nicolas de Roux. "Neuroendocrinology." In Yearbook of Pediatric Endocrinology 2010, 1–11. Basel: KARGER, 2010. http://dx.doi.org/10.1159/000320573.

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de Roux, Nicolas. "Neuroendocrinology." In Yearbook of Pediatric Endocrinology 2008, 3–10. Basel: KARGER, 2008. http://dx.doi.org/10.1159/000158553.

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Chevrier, Lucie, Brooke Tata, and Nicolas De Roux. "Neuroendocrinology." In Yearbook of Pediatric Endocrinology 2013, 1–12. Basel: S. KARGER AG, 2013. http://dx.doi.org/10.1159/000353828.

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Chevrier, Lucie, Lukas Huijbregts, Carine Villanueva, and Nicolas De Roux. "Neuroendocrinology." In Yearbook of Pediatric Endocrinology 2011, 1–11. Basel: KARGER, 2011. http://dx.doi.org/10.1159/000330482.

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Huijbregts, Lukas, Carine Villanueva, and Nicolas de Roux. "Neuroendocrinology." In Yearbook of Pediatric Endocrinology, 1–12. Basel: KARGER, 2009. http://dx.doi.org/10.1159/000239683.

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Craft, Suzanne, and John Newcomer. "Cognitive Neuroendocrinology." In Medical Neuropsychology, 5–24. Boston, MA: Springer US, 2001. http://dx.doi.org/10.1007/978-1-4615-1287-5_2.

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Oksche, A. "Ernst and Berta Scharrer - Pioneers in Neuroendocrinology." In Neuroendocrinology, 1–4. Berlin, Heidelberg: Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-642-60915-2_1.

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Schultz, C., D. Koppers, E. Braak, and H. Braak. "Neurofibrillary Degeneration in Hypophysiotrophic Nuclei of the Aging Human Hypothalamus." In Neuroendocrinology, 115–26. Berlin, Heidelberg: Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-642-60915-2_10.

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Conference papers on the topic "Neuroendocrinology"

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Kim, Donghun Andy. "Neuroendocrinology in the blacklegged tick,Ixodes scapularis." In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.93888.

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Orchard, Ian. "Advances in insect neuroendocrinology/physiology through genomics and peptidomics: Diuresis inRhodnius prolixus." In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.91458.

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Cruz, Suelem Pereira da, Samia Insaurriaga Jundi, Gildo da Cruz Lopes, Luciano Antonio Marcolino, and Mônica Gomes de Almeida. "Adenoma hipofisário não funcionante na gravidez." In 45º Congresso da SGORJ XXIV Trocando Ideias. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/jbg-0368-1416-20211311108.

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Introdução: O adenoma hipofisário não funcionante é um tumor benigno, geralmente de crescimento lento, sem evidência de hipersecreção hormonal. Apesar de incomum, esse tipo de lesão pode surgir durante a gestação. Em geral é assintomático. Quando presentes, as manifestações clínicas mais comuns estão relacionadas ao efeito de massa causado pelo tumor, como alterações visuais e cefaleia. Relato de caso: Primigesta de 27 anos, com 30 semanas de gestação, foi encaminhada ao hospital terciário por redução da acuidade visual e cefaleia retro-orbitária iniciadas durante a gestação. Ressonância nuclear magnética de crânio revelou formação expansiva lobulada na sela túrcica, medindo 29x30x19 mm. A lesão tinha extensão suprabasilar, rechaçando o quiasma óptico e tocando a carótida intracraniana esquerda, compatível com macroadenoma hipofisário e possível sangramento. A função hipofisária era normal, e a hipótese diagnóstica era de adenoma hipofisário não secretor. A avaliação fetal revelou peso abaixo do esperado para a idade gestacional, sem outras alterações. Iniciou-se corticoterapia para a maturação pulmonar fetal. Com 31 semanas de gestação, foi realizada neurocirurgia transesfenoidal para a ressecção do tumor. A paciente foi medicada com hidrocortisona durante a indução anestésica e por 24 horas após a cirurgia, para evitar a insuficiência das glândulas suprarrenais. A cirurgia evoluiu sem intercorrências. No pós-operatório, a paciente desenvolveu diabetes insípido, que foi tratado com desmopressina por dois dias. A paciente apresentou melhora significativa da acuidade visual e da cefaleia, recebeu alta hospitalar e seguiu em acompanhamento no pré-natal e ambulatório de neuroendocrinologia. O parto ocorreu com 38 semanas e 6 dias por cesariana, sob indicação obstétrica (apresentação pélvica), com nascimento de bebê pesando 2830 g, Apgar 8 e 9 (primeiro e quinto minuto, respectivamente), sem alterações aparentes. Puerpério fisiológico. Conclusão: O tratamento de primeira linha do adenoma hipofisário não funcionante sintomático, mesmo durante a gestação, é a ressecção cirúrgica. Algumas fontes sugerem tratamento conservador, com interrupção da gestação pré-termo e abordagem posterior. Porém há risco de evolução para apoplexia hipofisária e comprometimento da gestação. Além disso, sem abordagem precoce, é possível que o tumor cresça e gere lesões irreversíveis nas estruturas adjacentes, resultando em sequelas e redução da qualidade de vida da paciente. Por outro lado, a cirurgia durante a gestação também está associada a risco de resultados perinatais adversos. A complicação cirúrgica mais comum é o diabetes insípido transitório. Raramente outras complicações como hidrocefalia, meningite, deficiências hormonais e morte podem ocorrer. No caso apresentado, o tratamento foi instituído assim que o diagnóstico foi feito, pelo grave comprometimento materno. Não houve complicações graves, com bons resultados para o binômio mãe-feto.

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