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Published: 4 June 2021
Last updated: 20 February 2023

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1

Intellectual functions and the brain: An historical perspective. Seattle: Hogrefe & Huber Publishers, 1992.

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2

Walker, Matthew P. The Role of Sleep in Neurocognitive Function. Oxford University Press, 2012. http://dx.doi.org/10.1093/oxfordhb/9780195376203.013.0007.

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3

Castellanos, Irina, David B. Pisoni, William G. Kronenberger, and Jessica Beer. Neurocognitive Function in Deaf Children With Cochlear Implants. Edited by Marc Marschark and Patricia Elizabeth Spencer. Oxford University Press, 2015. http://dx.doi.org/10.1093/oxfordhb/9780190241414.013.17.

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4

Heffernan, Thomas, ed. The Impact of Active and Passive Smoking Upon Health and Neurocognitive Function. Frontiers Media SA, 2016. http://dx.doi.org/10.3389/978-2-88919-977-8.

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5

Kronenberger, William G., and David B. Pisoni. Neurocognitive Functioning in Deaf Children with Cochlear Implants. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190880545.003.0016.

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Cochlear implantation restores some attributes of hearing and spoken language to prelingually deaf children. However, reduced access to auditory and spoken-language experiences for children with cochlear implants can alter the development of downstream neurocognitive functions such as sequential processing and self-regulatory language skills, which are critical building blocks for executive functioning. Executive functioning is the active regulation of cognitive, behavioral, and emotional processes in the service of planned, organized, controlled, goal-driven behavior. This chapter presents findings from two primary lines of research on the development of executive functioning in prelingually deaf, early implanted children with cochlear implants. The first is identification of specific executive function domains that are at risk for delay in children with cochlear implants compared to hearing children. The second is reciprocal influences of executive function and spoken-language skills throughout development in children and adolescents with cochlear implants.
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6

Drane, Daniel L., and Dona E. C. Locke. Mechanisms of Possible Neurocognitive Dysfunction. Edited by Barbara A. Dworetzky and Gaston C. Baslet. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190265045.003.0005.

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This chapter covers what is known about the possible mechanisms of neurocognitive dysfunction in patients with psychogenic nonepileptic seizures (PNES). It begins with a review of all research examining possible cognitive deficits in this population. Cognitive research in PNES is often obscured by noise created by a host of comorbid conditions (e.g., depression, post-traumatic stress disorder, chronic pain) and associated issues (e.g., effects of medications and psychological processes that can compromise attention or broader cognition). More recent studies employing performance validity tests raise the possibility that studies finding broad cognitive problems in PNES may be highlighting a more transient phenomenon secondary to these comorbid or secondary factors. Such dysfunction would likely improve with successful management of PNES symptomatology, yet the effects of even transient variability likely compromises daily function until these issues are resolved. Future research must combine the use of neuropsychological testing, performance validity measures, psychological theory, neuroimaging analysis, and a thorough understanding of brain–behavior relationships to address whether there is a focal neuropathological syndrome associated with PNES.
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7

Newman, Jennifer, and Charles R. Marmar. Executive Function in Post-Traumatic Stress Disorder. Edited by Charles B. Nemeroff and Charles R. Marmar. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190259440.003.0015.

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This chapter discusses the role of executive function in post-traumatic stress disorder (PTSD), which is far from fully understood. Deficits are subtle and findings are often inconsistent. Impairments have been related to worsening of psychological symptoms, functioning, and quality of life. They can also negatively impact treatment. Functional imaging shows that neurocognitive deficits in PTSD may be related to an imbalance in brain connectivity, where emotion processing is enhanced and control is reduced. Structural findings show abnormalities in brain regions involved in higher-level functions. However, findings are often discrepant. Factors related to these inconclusive results are considered, including developmental course, premorbid functioning, and comorbidities such as traumatic brain injury, depression, substance use, attention deficit hyperactivity disorder, health behaviors, and medical concerns. Treatment implications, limitations of this work, and future directions are presented. The aim of future research is to advance scientific understanding of PTSD, neurocognitive impairments, and related conditions, with the goal of improving outcomes for those who encounter trauma.
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8

Eliason, Michelle. Neurocognitive Workbook: An Interactive Solution to Manage Memory & Executive Function Changes with Long-Term Occupational Therapy. Buffalo Occupational Therapy, 2022.

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9

Alan, Frazer, Molinoff Perry B, and Winokur Andrew 1944-, eds. Biological bases of brain function and disease. New York: Raven Press, 1994.

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10

Rucci, Jennifer M., and Robert E. Feinstein. Neurocognitive Disorders and Mental Disorders Due to Another Medical Condition. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199326075.003.0005.

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The defining feature of neurocognitive disorders is a decline in cognitive functioning. Patients suffering from delirium experience an acute change in mental status, fluctuating levels of consciousness, and an inability to acquire new information. Patients with major neurocognitive disorder experience significant cognitive decline in complex attention, executive function, learning and memory, language, perceptual-motor, and social cognition. The chapter also discusses mental disorders due to another medical condition. These patients can experience psychotic, mood, or anxious symptoms or a personality change; their intellectual functioning usually remains intact. A patient presenting with a first episode of psychiatric symptoms and no prior psychiatric history should be evaluated for an acute medical etiology causing the psychiatric symptoms, particularly if he or she is over 40 years of age. Anticholinesterase inhibitors (donepezil, galantamine, and rivastigmine) may slow the rate of cognitive decline in Alzheimer’s disease, and the combination of an anticholinesterase inhibitor and memantine may be more effective than either medication alone.
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11

Piccinini, Gualtiero. Neurocognitive Mechanisms. Oxford University Press, 2020. http://dx.doi.org/10.1093/oso/9780198866282.001.0001.

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This book provides the foundations for a neurocomputational explanation of cognition based on contemporary cognitive neuroscience. An ontologically egalitarian account of composition and realization, according to which all levels are equally real, is defended. Multiple realizability and mechanisms are explicated in light of this ontologically egalitarian framework. A goal-contribution account of teleological functions is defended, and so is a mechanistic version of functionalism. This provides the foundation for a mechanistic account of computation, which in turn clarifies the ways in which the computational theory of cognition is a multilevel mechanistic theory supported by contemporary cognitive neuroscience. The book argues that cognition is computational at least in a generic sense. The computational theory of cognition is defended from standard objections yet a priori arguments for the computational theory of cognition are rebutted. The book contends that the typical vehicles of neural computations are representations and that, contrary to the received view, neural representations are observable and manipulable in the laboratory. The book also contends that neural computations are neither digital nor analog; instead, neural computations are sui generis. The book concludes by investigating the relation between computation and consciousness, suggesting that consciousness may have a functional yet not wholly computational nature.
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12

Domhoff, G. William. The Emergence of Dreaming. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190673420.001.0001.

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This book presents a new neurocognitive theory of dreams that documents the similarities of dreaming to waking thought, demonstrates that personal psychological meaning can be found in a majority of dream reports, has a strong developmental dimension based on excellent longitudinal and cross-sectional studies carried out in sleep labs with children ages 3–15, locates the neural substrate for dreaming in the same brain network active during mind-wandering and daydreaming, and marshals the evidence that shows it is very unlikely that dreaming has any adaptive function. These claims are based on five different sets of descriptive empirical findings that were developed between the late 1950s and the first sixteen years of the twenty-first century. All of these findings were unanticipated by scientific dream researchers and then resisted to varying degrees by dream theorists for a variety of reasons. The first five chapters spell out the theory and the evidence for it without any discussion or criticism of past theories. The next two chapters present detailed criticisms of two major alternative theories. The penultimate chapter presents evidence that it is very unlikely that dreaming has any adaptive function in the evolutionary sense of the term, although humans have invented uses for dreams in religious and healing rituals. In that regard, dreaming has an emergent function in culture that was invented in the course of history due to human cognitive capacities. The final chapter presents a general agenda for future research using new methodologies to test all of the neurocognitive hypotheses.
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13

Nie, Pei Huey, and David L. Sultzer. Treatments for Neurocognitive Disorders. Oxford University Press, 2015. http://dx.doi.org/10.1093/med:psych/9780199342211.003.0026.

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Dementia, or neurocognitive disorders, refers to a number of clinical syndromes originating in brain pathology and characterized by cognitive deficits and functional impairment. This chapter provides an update on treatment options in addition to a brief summary of dementia types and an overview of the diagnostic criteria for cognitive disorders. The diagnosis of dementia is ultimately a clinical one and includes a multidimensional perspective; as such, treatment requires a comprehensive approach. This chapter addresses two aspects of the treatment of neurocognitive disorders: pharmacological interventions that can temporarily slow the decline of cognitive deficits and the management of behavioral and psychological symptoms (neuropsychiatric symptoms) associated with dementia syndromes. The chapter also reviews disease-modifying treatments in development that may beneficially alter the course of disease, or reduce or prevent symptom expression in those at risk.
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14

Harder, Louise, and Atul Malhotra. Pathophysiology and therapeutic strategy for sleep disturbance in the ICU. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0225.

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Robust data have shown that sleep disruption and inadequate sleep duration in the general population impact neurocognitive function and produce cardiometabolic sequelae. Despite widespread recognition of the importance of sleep as an essential homeostatic function, there are relatively few data regarding the importance of sleep in critically-ill patients. Obstructive sleep apnoea is a common respiratory condition that is prevalent in the ICU and can be particularly problematic pre-intubation, post-extubation, and in the peri-operative setting. Considerable discussion regarding the impact of sleep versus sedation has occurred, with some insights emerging from improvements in our understanding of basic neurobiology. Sleep disturbance may also have an impact in critically-ill mechanically-ventilated patients by contributing to the development of delirium, which is associated with poor outcomes. However, further data are required to determine the ideal strategy to optimize sleep in the ICU and whether such strategies will in turn improve hard outcomes of critically-ill patients.
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15

Woldstad, Christopher, Michael Boska, and Howard E. Gendelman. Neurological Complications of HIV in The Central Nervous System. Edited by Mary Ann Cohen, Jack M. Gorman, Jeffrey M. Jacobson, Paul Volberding, and Scott Letendre. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199392742.003.0026.

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This chapter serves to highlight both the research advances made in understanding the effects of HIV on the nervous system and what lies ahead. Particular focus is given to both the effects HIV can play on the nervous system at the molecular and cellular levels and the comorbid conditions that affect neural function. Attention is also given to specific biomarkers to be used for increasing the effectiveness and availability of therapies. The pathogenesis of HIV-associated neurocognitive disorders (HAND) is comparable to that of several other neurodegenerative disorders, and their mechanistic similarities are also discussed in detail. With the introduction of antiretroviral therapy the life expectancy of persons with HIV has increased, with a concomitant decrease in the incidence of severe dementia. There has been a remarkable improvement in cognitive function with almost a complete reversal of associated symptoms of disease. Past and present disease manifestations and the implications for treatment are outlined in the chapter.
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16

Domhoff, G. William. The Neurocognitive Theory of Dreaming. The MIT Press, 2022. http://dx.doi.org/10.7551/mitpress/14679.001.0001.

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A comprehensive neurocognitive theory of dreaming based on the theories, methodologies, and findings of cognitive neuroscience and the psychological sciences. G. William Domhoff's neurocognitive theory of dreaming is the only theory of dreaming that makes full use of the new neuroimaging findings on all forms of spontaneous thought and shows how well they explain the results of rigorous quantitative studies of dream content. Domhoff identifies five separate issues—neural substrates, cognitive processes, the psychological meaning of dream content, evolutionarily adaptive functions, and historically invented cultural uses—and then explores how they are intertwined. He also discusses the degree to which there is symbolism in dreams, the development of dreaming in children, and the relative frequency of emotions in the dreams of children and adults. During dreaming, the neural substrates that support waking sensory input, task-oriented thinking, and movement are relatively deactivated. Domhoff presents the conditions that have to be fulfilled before dreaming can occur spontaneously. He describes the specific cognitive processes supported by the neural substrate of dreaming and then looks at dream reports of research participants. The “why” of dreaming, he says, may be the most counterintuitive outcome of empirical dream research. Though the question is usually framed in terms of adaptation, there is no positive evidence for an adaptive theory of dreaming. Research by anthropologists, historians, and comparative religion scholars, however, suggests that dreaming has psychological and cultural uses, with the most important of these found in religious ceremonies and healing practices. Finally, he offers suggestions for how future dream studies might take advantage of new technologies, including smart phones.
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17

Hall, Peter A., Geoffrey T. Fong, and Cassandra J. Lowe. Affective Dynamics in Temporal Self-Regulation Theory. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190499037.003.0006.

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Affective experiences are part of our everyday life, but do they influence health-related decisions and behaviors in a systematic way? Temporal self-regulation theory (TST) posits that health behaviors are a joint function of neurobiologically rooted executive control processes, prepotency, and intentions. The relative weights of these in turn depend largely on the ecological context in which the behaviors are being performed. On the surface, then, TST is a model of health behavior that relies predominantly on social-cognitive and neurocognitive constructs to explain health behavior trajectories. For this reason, it appears to not deal directly with the topic of affect in general, and emotion more specifically. However, there are several facets of the TST model that involve these processes, or are heavily influenced by them. This chapter discusses each of the primary points of intersection between affective processes and constructs within TST.
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18

Greven, Corina U., Jennifer S. Richards, and Jan K. Buitelaar. Sex differences in ADHD. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198739258.003.0016.

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This chapter reviews sex differences in ADHD, focusing on differences in prevalence, comorbidity, and impairment, and discusses potential mechanisms underlying these differences. ADHD is more common in males than females (sex ratio ~3:1). Males with ADHD show greater comorbidity with comorbid externalizing (conduct) problems, while females with ADHD show internalizing problems. Females with ADHD may experience greater subjective impairment than males with ADHD. Referral and diagnostic issues, relating to sex-specific display of ADHD symptoms (more overt and disruptive in males, more subtle in females), underdiagnosis, or misdiagnosis in girls, as well as biases due to informant source, likely contribute to sex differences in ADHD. Potential biological mechanisms include endocrine factors (testosterone, glucocorticoids, and hypothalamic–pituitary–adrenal axis activation differences), aetiological sex differences (sex-chromosome genes), sex differences in neurocognitive functioning, and differences in brain structure and function. The chapter provides an outlook for future research and clinical implications.
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19

Alcantud-Marín, Francisco, Fernanda López-Ramón, Esperanza Navarro-Pardo, Veronica Moreno-Campos, and Yurena Alonso-Esteban. Handbook of Research on Neurocognitive Development of Executive Functions and Implications for Intervention. IGI Global, 2022.

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20

Alcantud-Marín, Francisco, Fernanda López-Ramón, Esperanza Navarro-Pardo, Veronica Moreno-Campos, and Yurena Alonso-Esteban. Handbook of Research on Neurocognitive Development of Executive Functions and Implications for Intervention. IGI Global, 2022.

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21

Alcantud-Marín, Francisco, Laxmi Paudel, Nadeepa Wickramage, Annalease Gibson, and Michael Rogers. Handbook of Research on Neurocognitive Development of Executive Functions and Implications for Intervention. IGI Global, 2021.

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22

Alcantud-Marín, Francisco, Fernanda López-Ramón, Esperanza Navarro-Pardo, Veronica Moreno-Campos, and Yurena Alonso-Esteban. Handbook of Research on Neurocognitive Development of Executive Functions and Implications for Intervention. IGI Global, 2022.

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23

Torrent, Carla, Caterina del Mar Bonnin, and Anabel Martinez-Arán. Functional remediation therapy for bipolar disorder. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198748625.003.0014.

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Impairment in functional outcome is commonly observed even when patients are euthymic and includes multiple areas of functioning. Many factors such as sociodemographic, clinical, pharmacological, and neurocognitive variables have been associated with functional impairment. The term ‘functional remediation’ has been coined to define an innovative strategy aimed at targeting the critical factors for full psychosocial adjustment and functional recovery in the context of psychoses and more specifically bipolar disorders. Functional remediation involves not only neurocognitive techniques and training but also psychoeducation on cognition-related issues and problem-solving within an ecological framework. The inclusion of context processing, performance monitoring, encoding, and manipulation of the information as well as fostering compensatory strategies must be included in the functional remediation intervention. In this regard, real-world problems affecting daily functioning are used for bipolar disorder to be transferred to daily practice.
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24

Robertson, Dominic. Your Brain Is Not Broken: Neurodiversity, and Ways of Improving Neurocognitive Functions in Autistic Individuals. Independently Published, 2022.

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25

Scholey, Andrew, and Con Kerry Kenneth Stough. Advances in Natural Medicines, Nutraceuticals and Neurocognition. Taylor & Francis Group, 2013.

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26

Scholey, Andrew, and Con Kerry Kenneth Stough. Advances in Natural Medicines Nutraceuticals and Neurocognition. Taylor & Francis Group, 2019.

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27

Scholey, Andrew, and Con Stough. Advances in Natural Medicines, Nutraceuticals and Neurocognition. Taylor & Francis Group, 2013.

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28

Scholey, Andrew, and Con Kerry Kenneth Stough. Advances in Natural Medicines, Nutraceuticals and Neurocognition. Taylor & Francis Group, 2013.

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29

Scholey, Andrew, and Con Kerry Kenneth Stough. Advances in Natural Medicines, Nutraceuticals and Neurocognition. Taylor & Francis Group, 2013.

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30

Smallwood, Jonathan, Daniel Margulies, Boris C. Bernhardt, and Elizabeth Jefferies. Investigating the Elements of Thought. Edited by Kalina Christoff and Kieran C. R. Fox. Oxford University Press, 2018. http://dx.doi.org/10.1093/oxfordhb/9780190464745.013.34.

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Spontaneous thoughts come in a large variety of different forms, varying in their experiential content as well as the functional outcomes with which they are associated. This chapter describes a component process architecture for spontaneous thought in which different types of experience arise through the combinations of different underlying neurocognitive processes. These underlying elements of cognition are not specific to spontaneous thought, since many, if not all, of these neurocognitive processes can be engaged when participants perform an externally directed task. We consider neurocognitive evidence that shows how this component process architecture provides explanatory value for accounts of spontaneous thought since it provides a mechanism that captures both the complex variety of spontaneous experiences that characterize the human condition, as well as the different functional outcomes that these different experiences are associated with.
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31

O'Callaghan, Claire, and Muireann Irish. Candidate Mechanisms of Spontaneous Cognition as Revealed by Dementia Syndromes. Edited by Kalina Christoff and Kieran C. R. Fox. Oxford University Press, 2018. http://dx.doi.org/10.1093/oxfordhb/9780190464745.013.6.

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The capacity to engage in spontaneous self-generated thought is fundamental to the human experience, yet surprisingly little is known regarding the neurocognitive mechanisms that support this complex ability. Dementia syndromes offer a unique opportunity to study how the breakdown of large-scale functional brain networks impacts spontaneous cognition. Indeed, many of the characteristic cognitive changes in dementia reflect the breakdown of foundational processes essential for discrete aspects of self-generated thought. This chapter discusses how disease-specific alterations in memory-based/construction and mentalizing processes likely disrupt specific aspects of spontaneous, self-generated thought. In doing so, it provides a comprehensive overview of the neurocognitive architecture of spontaneous cognition, paying specific attention to how this sophisticated endeavor is compromised in dementia.
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32

Hagoort, Peter, ed. Human Language. The MIT Press, 2019. http://dx.doi.org/10.7551/mitpress/10841.001.0001.

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A unique overview of the human language faculty at all levels of organization. Language is not only one of the most complex cognitive functions that we command, it is also the aspect of the mind that makes us uniquely human. Research suggests that the human brain exhibits a language readiness not found in the brains of other species. This volume brings together contributions from a range of fields to examine humans' language capacity from multiple perspectives, analyzing it at genetic, neurobiological, psychological, and linguistic levels. In recent decades, advances in computational modeling, neuroimaging, and genetic sequencing have made possible new approaches to the study of language, and the contributors draw on these developments. The book examines cognitive architectures, investigating the functional organization of the major language skills; learning and development trajectories, summarizing the current understanding of the steps and neurocognitive mechanisms in language processing; evolutionary and other preconditions for communication by means of natural language; computational tools for modeling language; cognitive neuroscientific methods that allow observations of the human brain in action, including fMRI, EEG/MEG, and others; the neural infrastructure of language capacity; the genome's role in building and maintaining the language-ready brain; and insights from studying such language-relevant behaviors in nonhuman animals as birdsong and primate vocalization. Section editorsChristian F. Beckmann, Carel ten Cate, Simon E. Fisher, Peter Hagoort, Evan Kidd, Stephen C. Levinson, James M. McQueen, Antje S. Meyer, David Poeppel, Caroline F. Rowland, Constance Scharff, Ivan Toni, Willem Zuidema
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33

Goodkin, Karl, David M. Stoff, Dilip V. Jeste, and Maria J. Marquine. Older Age and HIV. Edited by Mary Ann Cohen, Jack M. Gorman, Jeffrey M. Jacobson, Paul Volberding, and Scott Letendre. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199392742.003.0036.

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This chapter addresses the growing problem of aging and HIV infection throughout the world. Emphasis is placed on conceptualization such as the development of phenotypes within the population of older persons with HIV. The phenotypes include (1) disability, (2) frailty/vulnerability, (3) comorbid conditions, (4) cognitive aging, (5) premature or accelerated aging, and (6) successful aging. Older age and illness progression is addressed with respect to frailty or vulnerability to progression and the development of a dysfunctional, disabled status in activities of daily living. The issue of older age and HIV-associated neurocognitive disorder is discussed, and the research related to pattern of cognitive impairment, including dementia, is reviewed. The chapter also differentiates characteristics of accelerated aging from characteristics of successful aging. Integration of gerontology with HIV medicine and HIV psychiatry can be accomplished through focusing future study on optimization of functional status and quality of life in aging with HIV.
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34

Doyle, Lauren R., and Sarah N. Mattson. Behavioral Teratogenic Effects of Alcohol: Focus on Neurobehavioral Disorder Associated With Prenatal Alcohol Exposure. Edited by Thomas H. Ollendick, Susan W. White, and Bradley A. White. Oxford University Press, 2018. http://dx.doi.org/10.1093/oxfordhb/9780190634841.013.39.

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Prenatal exposure to teratogens may alter fetal development and significantly impact later life. Perhaps the best known teratogen is alcohol; prenatal alcohol exposure causes a broad range of effects that can cause lifelong impairment. Of greatest significance are the functional impairments in behavior and cognition. Recognition of these impairments led to the inclusion of the neurobehavioral disorder associated with prenatal alcohol exposure (ND-PAE) in the fifth edition of the Diagnostic and Statistical Manual of Mental Disorders under “conditions for further study.” This proposed diagnosis captures the significant neurodevelopmental and mental health impacts associated with prenatal alcohol exposure and requires impairment in neurocognitive functioning, self-regulation, and adaptive functioning. This chapter reviews clinical impacts of prenatal alcohol exposure, with particular focus on ND-PAE. Methods for comprehensively assessing fetal alcohol spectrum disorders, specifically ND-PAE, are discussed as well as preliminary evidence for implementing effective interventions with these individuals.
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35

Bacio, Guadalupe A., Ty Brumback, and Sandra A. Brown. Alcohol and Youth. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190676001.003.0011.

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Adolescence is a period of intense transition and change, from social and emotional changes with increased independence from family to physical changes associated with the onset of puberty. The onset of involvement with alcohol and drugs emerges simultaneously with these interrelated biological, cognitive, affective, and social changes. This chapter considers how developmental processes and emerging domains of functioning impact alcohol consumption in adolescence and presents examples of two lines of research that use integrative models to explore these relationships: the National Consortium on Alcohol and NeuroDevelopment in Adolescence, a longitudinal study designed to examine the developmental impact of alcohol and other drug use on neuroanatomy, neurocognition, and behavior; and Project Options, a voluntary, high school-based intervention aimed at reducing dangerous alcohol use. The chapter concludes with a discussion of research questions for future study, highlighting the central function of technological, behavioral, biomedical, and data analytic advances in these efforts.
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36

Kaufmann, Liane, Karin Kucian, and Michael von Aster. Development of the numerical brain. Edited by Roi Cohen Kadosh and Ann Dowker. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199642342.013.008.

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This article focuses on typical trajectories of numerical cognition from infancy all the way through to adulthood (please note that atypical pathways of numerical cognition will be dealt in‘Brain Correlates of Numerical Disabilities’). Despite the fact that developmental imaging studies are still scarce to date there is converging evidence that (1) neural signatures of non-verbal number processing may be observed already in infants; and (2) developmental changes in neural responsivity are characterized by increasing functional specialization of number-relevant frontoparietal brain regions. It has been suggested that age and competence-related modulations of brain activity manifest as an anterior-posterior shift. On the one hand, the recruitment of supporting frontal brain regions decreases, while on the other hand, reliance on number-relevant (fronto-)parietal neural networks increases. Overall, our understanding of the neurocognitive underpinnings of numerical development grew considerably during the last decade. Future research is expected to benefit substantially from the fast technological advances enabling researchers to gain more fine-grained insights into the spatial and temporal dynamics of the neural signatures underlying numerical development.
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37

Freeman, Margaret H. The Poem as Icon. Oxford University Press, 2020. http://dx.doi.org/10.1093/oso/9780190080419.001.0001.

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The objective in this book is to show how poetry enables us cognitively to aesthetically access, experience, and identify with the visible and invisible “being” of reality, with art as one cognitive expression of the aesthetic faculty, science another. Just as scientific knowledge of reality is achieved through physically exploring the far reaches of the visible and invisible worlds, so is poetic experience achieved through iconically simulating in semblance the “being” of reality that integrates both self and world in participatory unity. “Being” here should not be understood as the existence of material substance, but as the essence of all that is, both visible and invisible, material and immaterial, a life force in continuous flux and change. The book explores cognition as the sensory-motor-emotive-conceptual processes of “minding” and the aesthetic faculty as the processes of attention, imagination, memory, discrimination, expertise, and judgment that underlie all human cognition, including the arts and the sciences. Drawing from research such as blending and neurocognition in interdisciplinary cognitive literary studies, the book attempts to resolve long-standing questions about the function of poetry. Accepting the premise that poetry is its own artistic reason for being, it introduces the major elements—semblance, metaphor, schema, and affect—that constitute a poem as icon in motivating a poet’s intension and a respondent’s engagement. In so doing the book makes the case that a poem is a potential icon of the felt reality of being and shows that poetic iconicity provides a means for evaluating great poetry and an explanation for its endurance.
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38

Jakobsson, Jan. Anaesthesia for day-stay surgery. Edited by Philip M. Hopkins. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199642045.003.0068.

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Day-stay surgery is becoming increasingly common the world over. There are several benefits of avoiding in-hospital care. Early ambulation reduces the risk for thromboembolic events, facilitates wound healing, and avoiding admission reduces the risk for hospital-related infection. Additionally, the risk of neurocognitive side-effects can be avoided by returning the elderly patient to their home environment. Day-stay anaesthesia calls for adequate and structured preoperative assessment and patient evaluation, and the potential risk associated with surgery and anaesthesia should be assessed on an individual basis. Need for preoperative testing should be based on functional status of the patient and preoperative medical history but even the surgical procedure should be taken into account. Preoperative fasting should be in accordance with modern guidelines, refraining from food for 6 hours and fluids for 2 hours prior to induction in low-risk patients. Preventive analgesia and prophylaxis of postoperative nausea and vomiting (PONV) should be administered preoperatively. Local anaesthesia should be administered prior to incision, constituting part of multimodal analgesia. The multimodal analgesia strategy should also include paracetamol and a non-steroidal anti-inflammatory drug in order to reduce the noxious stimulus from the surgical field. Third-generation inhaled anaesthetics or a propofol-based maintenance are both feasible alternatives. Titrating depth of anaesthesia by using an EEG-based depth of anaesthesia monitor may facilitate the recovery process. The laryngeal mask airway has become commonly used and has several advantages. Ultrasound-guided peripheral blocks may facilitate the early postoperative course by reducing pain and avoiding the use of opiates. Perineural catheters may be an option for prolongation of the block following painful orthopaedic procedures but a strict protocol and follow-up must be secured. Not only pain but even nausea and vomiting should be prevented, and therefore risk stratification, for example by the Apfel score, and PONV prophylaxis in accordance with the risk score is strongly recommended. Early ambulation should be encouraged postoperatively. Safe discharge should include an escort who also remains at home during the first postoperative night. Analgesics should be provided and be readily available for self-care when the patient comes home. Pain medication should include an opioid; however, the benefit versus risk must be assessed on an individual basis. Patients should also be instructed about a rescue return-to-hospital plan. Quality of care should include follow-up and analysis of clinical practice, and institution of methods to improve quality should be enforced for the benefit of the ambulatory surgical patient.
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