Relevant bibliographies by topics / Neurobiology

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Neurobiology'

Author: Grafiati
Published: 4 June 2021
Last updated: 29 July 2024

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Journal articles on the topic "Neurobiology"

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STRAUSFELD, N. J. "Spider Neurobiology: Neurobiology of Arachnids." Science 231, no. 4745 (March 28, 1986): 1610. http://dx.doi.org/10.1126/science.231.4745.1610.

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Christensen, Bruce K., and Robert M. Bilder. "Dual Cytoarchitectonic Trends: An Evolutionary Model of Frontal Lobe Functioning and Its Application to Psychopathology." Canadian Journal of Psychiatry 45, no. 3 (April 2000): 247–56. http://dx.doi.org/10.1177/070674370004500303.

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Objective: To introduce and discuss an evolutionary model of frontal lobe functioning (the dual cytoarchitectonic trends theory [DTT]) and its application to understanding the neurobiology of schizophrenia and anxiety disorders. Method: An introduction to the DTT with respect to neural architecture, connectivity, and function is presented. In addition, neurobiologic, neuropathologic, clinical, and cognitive research supporting the application of this model to schizophrenia and anxiety disorders is reviewed. Results: Traditional neuropsychologic models of acquired brain damage have been limited in their ability to explain frontal lobe dysfunction and its consequences in relation to psychopathology. The DTT offers an appropriately general neural-systems framework that may be better able to account for the diversity of symptoms, widespread neuropathology, and developmental abnormalities that are associated with most forms of psychopathology. Conclusions: Research investigating the neurobiology of psychopathology would benefit from adopting models of brain dysfunction that are consistent with neurodevelopmental pathology and evolution. Such efforts would likely lead to a greater understanding of neurobiologic mechanisms and, ultimately, better treatment strategies.
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Wilding, J., P. Widdowson, and G. Williams. "Neurobiology." British Medical Bulletin 53, no. 2 (January 1, 1997): 286–306. http://dx.doi.org/10.1093/oxfordjournals.bmb.a011614.

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Lumsden, Andrew, Susan Chapman, Stefan Jungbluth, Esther Bell, Ana Coutinho, Moheb Costandi, Niels Adams, et al. "Neurobiology." Current Opinion in Neurobiology 10, no. 3 (June 2000): 275–86. http://dx.doi.org/10.1016/s0959-4388(00)00102-1.

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Lumsden, Andrew, Susan Chapman, Ana Coutinho, Jonathan Gilthorpe, Aida Halilagic, Frank Shubert, Richard Wingate, et al. "Neurobiology." Current Opinion in Neurobiology 10, no. 4 (August 2000): 423–32. http://dx.doi.org/10.1016/s0959-4388(00)00106-9.

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Lumsden, Andrew, Susan Chapman, Frank Schubert, Leah Toole, Mark Mayford, Stephan Hamann, Paul J. Reber, et al. "Neurobiology." Current Opinion in Neurobiology 11, no. 1 (February 2001): 1–9. http://dx.doi.org/10.1016/s0959-4388(00)00185-9.

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Lumsden, Andrew, Ana Coutinho, Jamilé Hazan, Frank Schubert, Mark Mayford, Stephan Hamann, Paul J. Reber, et al. "Neurobiology." Current Opinion in Neurobiology 11, no. 2 (April 2001): 135–43. http://dx.doi.org/10.1016/s0959-4388(00)00187-2.

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Lumsden, Andrew, Laura Andreae, Jonathan Gilthorpe, Sally Lowell, Frank Schubert, Stephan Hamann, Paul J. Reber, et al. "Neurobiology." Current Opinion in Neurobiology 11, no. 3 (June 2001): 259–66. http://dx.doi.org/10.1016/s0959-4388(00)00205-1.

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Price, J. "Neurobiology." Current Opinion in Neurobiology 11, no. 4 (August 1, 2001): 395–404. http://dx.doi.org/10.1016/s0959-4388(00)00224-5.

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Price, Jack, Alcino J. Silva, Steven A. Kushner, Paul J. Reber, Michael Häusser, John N. Wood, David S. Bredt, et al. "Neurobiology." Current Opinion in Neurobiology 11, no. 6 (December 2001): 643–50. http://dx.doi.org/10.1016/s0959-4388(01)00262-8.

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Dissertations / Theses on the topic "Neurobiology"

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Vílchez, Acosta Alba del Valle. "Analysis of Reelin function in brain development and in adult neurogenesis = Análisis de la función de Reelina en el desarrollo del cerebro y la neurogénesis adulta." Doctoral thesis, Universitat de Barcelona, 2019. http://hdl.handle.net/10803/668316.

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Reelin is a large extracellular matrix glycoprotein with a crucial role both during brain development, where it is key for neuronal migration and for the formation of the layered structure of cerebral cortex and cerebellum, and in the adulthood, where it is involved in adult synaptic plasticity, including neurogenesis in the dentate gyrus and dendritogenesis amongst other processes. Reelin acts through the binding to its canonical receptors (apolipoprotein E receptor 2, ApoER2; and very low density lipoprotein receptor, VLDLR) which trigger a complex signaling cascade involving numerous kinases and the adaptor protein Dab1. At the embryonic stage, Reelin is expressed mainly by Cajal-Retzius cells on the developing brain whereas at perinatal stages its expression gradually disappears from Cajal-Retzius cells and starts to be expressed by GABAergic interneurons of the cortex and hippocampus. In the neocortex, postmitotic neurons migrate in an ordered sequence that determines the normal “inside-out” layer formation. The malpositioning of cortical neurons is a result of abnormal migration and could cause severe layering malformations with functional consequences related with neurodevelopmental diseases such as Schizophrenia, Autism and Epilepsy. In this context, one of the most studied models has been the reeler mouse which presents a characteristic phenotype caused by an autosomal mutation in the Rln gene. The reeler mouse presents several morphological defects including a failed pre-plate splitting that causes a roughly inverted neuronal layering in the cortex, mispositioning of pyramidal neurons as well as granular cells on the dentate gyrus and profound cerebellar hypoplasia. However the study of the effects of Reelin signaling in the adult brain is difficult in the reeler mouse model due to the failed migration and mispositioning during development. Thus, to unravel the function of Reelin at different developmental stages (from embryonic to adult) as well as to gain insight in the potential distinct contribution of Reelin from different cell-types, we have generated three Reelin deficient conditional transgenic lines which allow us to ubiquitously delete Reelin in a temporally-controlled manner (Cre fR/fR) or selectively remove Reelin from Cajal-Retzius cells (CR fR/fR) or GABAergic interneurons (Gad fR/fR). Analysis of the cortical organization using layer-specific markers reveals that, unlike the reeler mouse, none of our transgenic lines shows the characteristic inversion of cortical layers. Moreover, our data strongly indicates that Reelin from Cajal-Retzius cells is important for the typical inside-out laminar cortical development but seems to be dispensable for pre-plate splitting. Furthermore, our results suggest that the absence of Reelin during early postnatal and adult stages seems to impact on the well-defined laminar structure of the cortex, leading to an invasion of layer I by late-born neurons from layer II-III. Regarding the hippocampus, our results suggest, on the one hand, a differential contribution of Reelin expressed by Cajal-Retzius cells and by GABAergic interneurons in the formation of the laminar structures of the hippocampus. On the other hand, temporally-controlled removal of Reelin at postnatal stages demonstrates that it is essential for the correct formation of the hippocampus whereas in the adult seems to be key for several aspects of hippocampus neurogenesis, including neuronal positioning in the dentate gyrus and dendritic orientation at different maturation stages of adult new-born granule cells. Finally, our findings also support the importance of Reelin expression for proper Purkinje cell migration, but not for granule cell disposition in the cerebellum at early postnatal and adult stages. Taken altogether, our results suggest a causal relation between the absence of Reelin and structural alterations in the hippocampus, cortex and cerebellum, either at developments stages or adult stages.
Reelina es una glicoproteína extracelular de matriz esencial para la regulación de los procesos de migración neuronal y posicionamiento de las neuronas corticales durante el desarrollo del encéfalo. Durante la embriogénesis, Reelina es producida por las células Cajal-Retzius de la superficie de la corteza en desarrollo. En este estadío, las neuronas postmitóticas migran de forma ordenada originando una estructura laminar en seis capas, en las cuáles las neuronas más jóvenes se sitúan en las capas más externas. La pérdida de Reelina durante el desarrollo comporta fallos en la migración de las neuronas, provocando a su vez grandes alteraciones en la estructuración de la corteza que contribuyen a la patogénesis de muchos trastornos neurológicos como el autismo, la epilepsia, la esquizofrenia, o el trastorno bipolar. En este contexto, uno de los fenotipos más estudiado es el del ratón mutante de Reelina, reeler, que presenta una estructura cortical alterada con las capas invertidas. Sin embargo, dado que la expresión de Reelina durante el desarrollo ocurre a edades embrionarias muy tempranas, es difícil estudiar el efecto de su pérdida en este tipo de mutantes a edades más tardías, en los que los primeros efectos de su pérdida son tan profundos. Todo ello evidencia la necesidad de desarrollar otro tipo de modelos en los que la pérdida de Reelina sea más gradual o selectiva. En estadíos perinatales y en el cerebro adulto Reelina es expresada principalmente por interneuronas gabaérgicas, donde presumiblemente Reelina controla funciones de formación de sinapsis y mantenimiento de la plasticidad sináptica de las neuronas del córtex y del hipocampo. Nuestros resultados muestran un fenotipo diferencial para cada uno de nuestros mutantes, sugiriendo un papel diferente de Reelina expresada por cada tipo celular, o en función del estadío en el cuál la deleccionamos. En concreto, hemos visto que la función de Reelina es imprescindible para la correcta laminación del córtex, para la formación del hipocampo, y para el correcto posicionamiento de las células Purkinje del cerebelo. Además la pérdida de Reelina en estadíos adultos comporta fallos en la neurogénesis de la zona subventricular del giro dentado.
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Rudén, Ludvig. "Neurobiology of opioid addiction." Thesis, Högskolan i Skövde, Institutionen för biovetenskap, 2018. http://urn.kb.se/resolve?urn=urn:nbn:se:his:diva-15735.

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Since the use of opioids started to emerge for analgesic reasons in the 19th century with the synthetization of morphine, opioids have been studied rigorously to better understand its effects on the brain. This thesis shows that both the analgesic effects and the reinforcing effects of opioids are mediated by the same receptor, the mu opioid receptor (MOR). MOR activity has been correlated to both primary and secondary reinforcers and should be considered to cause positive reinforcement together with increases in dopamine transmission for all drugs of abuse, and not only in relation to opioids. Opioid tolerance, dependence and even addiction are to some extent thought to relate to opioids’ acute effect of cyclic adenosine monophosphate (cAMP) superactivation. Based upon these findings, the allostasis theory of addiction is considered to be the most suitable in defining opioid addiction. The theory claims that the mesolimbic dopamine system becomes sensitized, increasing the attractiveness of opioids. This while counteradaptation increases the pleasurable tolerance of opioids, encouraging the user to increase its intake for the same initial reward. Furthermore the theory claims that cAMP superactivation is causing an unfolding effect of neurobiological and neurochemical expressions which leads to the disorder of addiction. cAMP superactivation is mediating the negatively reinforcing aspects of opioid addiction together with changes to corticotropin-releasing factor (CRF) in the brain stress system, such as the hypothalamic-pituitary-adrenal (HPA) axis and the extended amygdala.
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Balia, Maddalena. "Etude de la connectivité GABAergique des précurseurs d’oligodendrocytes durant le développement cortical." Thesis, Sorbonne Paris Cité, 2016. http://www.theses.fr/2016USPCB106/document.

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Pas de résumé
Oligodendrocyte precursors cells (OPCs) are a major pool of progenitors during early development, but also persist in the adult. In 2000, a major dogma in neuroscience was broken when the existence of bona fide synapses between neurons and OPCs, a non-neuronal cell in the brain, was demonstrated. Now, it is known that OPCs can be contacted either by glutamatergic or GABAergic synapses in grey and white matter regions. However, the role of these synapses, especially of GABAergic synapses, in OPC physiology is still unclear. Our team previously demonstrated that synaptic inputs received by OPCs in the deep layer of the somatosensory cortex are primarly GABAergic. It shows that these synapses are transient, reaching a peak of connectivity in the second postnatal week (2PNW) and disappearing in the fourth postnatal week (4PNW). Nevertheless, a different mode of GABAergic transmission still persists in the 4PNW in a form of an extrasynaptic transmission relying solely on GABA spillover from nearby neurons. In the first study of this thesis, I demonstrated that the developmental switch of transmission from synaptic to extrasynaptic between GABAergic interneurons and OPCs is accompanied by molecular changes in the subunit composition of the GABAA receptors (GABAARs) of OPCs. These changes are mainly characterized by the downregulation of the γ2 and the α5 subunits between the 2PNW and the 4PNW. Interestingly, the γ2 subunit, known as a hallmark of synaptic GABAARs in neurons, is downregulated in concomitance with the loss of synaptic inputs of cortical OPCs. Pharmacology specific for γ2 showed that the switch of transmission starts at the end of the 2PNW, with a gradual loss of sensitivity to diazepam, and a decrease in the amplitude of the miniature GABAergic evoked events. The leading hypotheses regarding the role of OPC synapses include proliferation and differentiation of OPCs as well as myelination. However, none has been formally demonstrated. Since γ2 is expressed exclusively at synaptic sites in OPCs, I targeted this subunit to inactivate γ2-mediated synapses and unravel their fonction in OPCs during postnatal development. The inducible deletion of γ2 in OPCs decreased by more than a half their GABAergic synaptic activity during the 2PNW, indicating that this model constitutes a suitable tool to inactivate cortical GABAergic OPC synapses. Contrary to initial hypotheses, including those of my team, we did not observe any change in proliferation, differentiation or developmental myelination pattern of the somatosensory cortex in the knockout mouse. In addition, two-photon calcium imaging allowed us to demonstrate that evoked γ2- mediated synaptic signaling does not involved calcium signaling. Nevertheless, we observed a decrease in the number of OPCs at P30 in the knockout mouse, suggesting that these synapses regulate the self-maintenance capacity of OPCs rather than oligodendrogenesis or myelination. To confirm that the reduction of OPC density is caused specifically by the inactivation of γ2-mediated synapses, we examined the proportion of recombinant OPCs and OLs, and used non-recombinant cells as internal controls. We observed a significant 32% decrease of recombined OPCs in the knockout at P30 that was not compensated by recombinant OLs or non-recombinant cells. Hence, postsynaptic γ2-mediated GABAARs play a role in adjusting OPC density during postnatal development. In conclusion, during my thesis I have demonstrated that the postnatal switch of transmission from synaptic in the 2PNW to extrasynaptic at the 4PNW between interneurons and OPCs is accompanied by the down-regulation of the γ2 subunit of GABAARs in cortical OPCs. Impairing the γ2-mediated synaptic GABAergic signaling in OPCs did not result in drastic changes in the proliferation of differentiation of these cells. Instead, our results rather indicate a role in the density homeostasis of OPC population during cortical development
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Campbell, Thomas. "The Neurobiology of Intertemporal Choice." Thesis, University of Oxford, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.491330.

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Intertemporal choices are both common and important and yet the neurobiology of intertemporal choice is poorly understood. The work in this thesis contributes to a growing literature on the neurobiology of intertemporal choice. In particular I have investigated the contributions of the orbitofrontal cortex (OFC) and hippocampus (HPC) to intertemporal choice through the creation of novel intertemporal choice behavioral tasks. Firstly I reported that lesions of the HPC, but not the OFC, cauSe impulsive choice in a non-spatial T-maze based behavioral task. Secondly I described the creation and validation of a spatial, T-maze based intertemporal choice task for mice. This task was then used to investigate the contributions of the dopaminergic and serotonergic systems to intertemporal choice in mice. These experiments suggested that the dopaminergic system, but not the serotonergic system, is important in intertemporal choice. I then examined the contributions of the mouse OFC and HPC to intertemporal choice using an operant intertemporal choice task for mic.e. In this task, lesions of the HPC, but not the OFC, cause an increase in self- . controlled choice. Finally I reported a series of modelling experiments exploring the adaptiveness of self-control in foraging. These experiments called into question an influential theory suggesting that interspecific differences i~ metabolic rates helped .drive the evolution of impulsive strategies.. ' The behavioral tasks developed in this thesis may be used to further our understanding of the neurobiology of intertemporal choice and in particular the genetic basis of intertemporal choice.
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Nandi, L. Reema S. N. "The developmental neurobiology of opioids." Thesis, University College London (University of London), 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.413159.

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Egorova, Natalia. "Neurobiology of speech act processing." Thesis, University of Cambridge, 2013. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.648313.

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Sierra, Siegert Mauricio. "Depersonalization : from phenomenology to neurobiology." Thesis, University of Cambridge, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.621369.

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Johnson, Miranda Diane. "Neurobiology of activity-based anorexia." Tallahassee, Fla. : Florida State University, 2009. http://purl.fcla.edu/fsu/lib/digcoll/undergraduate/honors-theses/244564.

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Atherton, Christine J. "The neurobiology of object constancy." Thesis, Bangor University, 2005. https://research.bangor.ac.uk/portal/en/theses/the-neurobiology-of-object-constancy(3f31a74c-3acb-42f2-8941-967e61ad8bac).html.

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`Object constancy' is the name given to the brain's ability to overcome the myriad environmental obstacles to visual perception and produce a stable, consistent internal representation of object shape. Changes in object orientation represent one such confound. It can be inferred from the time taken to recognise misoriented objects that we encode specific object views based on our experience of those objects and their typical orientations ('viewpoint-dependent recognition'). Such studies also suggest that we may recognise certain objects in a manner that is not dependent on their orientation ('viewpoint-invariant recognition'). Further studies indicate that the time to resolve two angularly disparate shapes (`mental rotation') increases as a function of their angular disparity. It is hypothesised, based on these findings, that viewpoint-dependent recognition and mental rotation share a common mechanism for transforming the global stimulus percept into alignment, but that viewpointinvariant recognition is achieved by some other, non-transformational means. This thesis presents studies that examine the cortical correlates of viewpoint-dependent and viewpointinvariant object recognition using novel objects to eliminate the confounding effects of prior experience. It also presents a study that directly compares the cortical correlates of mental rotation, viewpoint-dependent and viewpoint-invariant recognition. Further comparison of these object constancy processes is then made using electrophysiological markers of visuospatial transformation. The findings of these studies indicate that viewpoint-dependent recognition and mental rotation recruit a bilateral parietal-premotor network for the manipulation of global stimulus percepts, hypothesised to be the same mechanism as that used for physical object manipulation and prehension. Viewpoint-invariant recognition does not appear to recruit such a mechanism, and this process appears to be less expensive in terms of cognitive resources than transformational object constancy mechanisms. Thus, implementation of a viewpoint-invariant mechanism to recognise misoriented objects is preferable, but may not be possible where stimulus features are few or ambiguous. In recognising misoriented objects, viewpoint-dependent and viewpoint-invariant mechanisms initially proceed in parallel, but successful recognition of object invariant features may be sufficient to terminate the viewpoint-dependent mechanism.
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Schmitz, Nicole. "The neurobiology of autistic spectrum disorder." Thesis, King's College London (University of London), 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.406247.

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Books on the topic "Neurobiology"

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Gilles, Raymond, and Jacques Balthazart, eds. Neurobiology. Berlin, Heidelberg: Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-87599-1.

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Torre, Vincent, and Franco Conti, eds. Neurobiology. Boston, MA: Springer US, 1996. http://dx.doi.org/10.1007/978-1-4615-5899-6.

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Open University. SD206 Course Team, ed. Neurobiology. Milton Keynes: Open University Press, 1992.

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Dworkin, Sebastian, ed. Neurobiology. New York, NY: Springer US, 2024. http://dx.doi.org/10.1007/978-1-0716-3585-8.

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1949-, Robinson David, ed. Neurobiology. Berlin: Springer in association with the Open University, 1998.

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Smith, C. U. M. Elements of molecular neurobiology. Chichester: Wiley, 1989.

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Bianchi, Lynne. Developmental Neurobiology. New York, NY: Garland Science, Taylor & Francis Group, LLC,: Garland Science, 2017. http://dx.doi.org/10.1201/9781351189477.

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Rao, Mahendra S., and Marcus Jacobson†, eds. Developmental Neurobiology. Boston, MA: Springer US, 2005. http://dx.doi.org/10.1007/0-387-28117-7.

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Heinemann, Steve, and James Patrick, eds. Molecular Neurobiology. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4615-7488-0.

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Banghart, Matthew R., ed. Chemical Neurobiology. Totowa, NJ: Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-345-9.

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Book chapters on the topic "Neurobiology"

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Punzo, Fred. "Neurobiology." In The Biology of Camel-Spiders, 45–69. Boston, MA: Springer US, 1998. http://dx.doi.org/10.1007/978-1-4615-5727-2_3.

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Bunge, Mario, and Rubén Ardila. "Neurobiology." In Philosophy of Psychology, 139–65. New York, NY: Springer New York, 1987. http://dx.doi.org/10.1007/978-1-4612-4696-1_7.

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Blom, Jan Dirk. "Neurobiology." In Alice in Wonderland Syndrome, 137–66. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-18609-8_5.

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Anagnostou, Evdokia, Deepali Mankad, Joshua Diehl, Catherine Lord, Sarah Butler, Andrea McDuffie, Lisa Shull, et al. "Neurobiology." In Encyclopedia of Autism Spectrum Disorders, 2014. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-1698-3_100928.

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Ferguson, Lorena A., and Stephanie L. Leal. "Neurobiology." In Lifestyle Psychiatry, 111–31. Boca Raton: CRC Press, 2023. http://dx.doi.org/10.1201/b22810-12.

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Clausen, Torben, José Luis Trejo, Mark P. Mattson, Alexis M. Stranahan, Joanna Erion, Rosa Maria Bruno, Stefano Taddei, and Melinda M. Manore. "Neurobiology." In Encyclopedia of Exercise Medicine in Health and Disease, 636. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_2749.

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Schmahl, C. "Neurobiology." In Trauma Sequelae, 89–106. Berlin, Heidelberg: Springer Berlin Heidelberg, 2022. http://dx.doi.org/10.1007/978-3-662-64057-9_6.

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Schmahl, C. "Neurobiology." In Trauma Sequelae, 89–106. Berlin, Heidelberg: Springer Berlin Heidelberg, 2022. http://dx.doi.org/10.1007/978-3-662-64057-9_6.

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Schmahl, C. "Neurobiology." In Trauma Sequelae, 89–106. Berlin, Heidelberg: Springer Berlin Heidelberg, 2022. http://dx.doi.org/10.1007/978-3-662-64057-9_6.

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Snyder, Solomon H. "Molecular Neurobiology." In Molecular Neurobiology, 3–8. Totowa, NJ: Humana Press, 1988. http://dx.doi.org/10.1007/978-1-4612-4604-6_2.

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Conference papers on the topic "Neurobiology"

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Zeringue, Henry C. "Microtechnologies for Neurobiology." In ASME 2010 First Global Congress on NanoEngineering for Medicine and Biology. ASMEDC, 2010. http://dx.doi.org/10.1115/nemb2010-13341.

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Oscillatory activity in cortical networks is thought to provide the foundation for many high-level processes including working memory and attention. It has been shown that spatial information propagation delay and connectivity density can determine the innate properties of local network activity. The initial formation of neuronal networks in the central nervous system occurs due to the interaction of the genetic programming of the cells and the presentation of external molecular cues. The activity-driven refinement that occurs later, giving rise to the highly complex networks within the brain, are dependent on the initial anatomical formation and structural connectivity which occurs without external activity cues. We describe technologies used to (1) modulate the genetic programming of neurons and (2) precisely control temporal and spatial presentation of environmental cues in vitro. We are exploring the ability to define simple oscillatory networks using these experimental techniques.
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Bateman, Chris, and Lennart E. Nacke. "The neurobiology of play." In the International Academic Conference. New York, New York, USA: ACM Press, 2010. http://dx.doi.org/10.1145/1920778.1920780.

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Kyselova, A., and E. Chernishova. "NEUROBIOLOGY OF POSTTRAUMATIC STRESS DISORDER." In Scientific discoveries: projects, strategies and development. European Scientific Platform, 2019. http://dx.doi.org/10.36074/25.10.2019.v2.13.

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Jiang, Yaoguang, and Michael Platt. "The neurobiology of strategic competition." In 2022 Conference on Cognitive Computational Neuroscience. San Francisco, California, USA: Cognitive Computational Neuroscience, 2022. http://dx.doi.org/10.32470/ccn.2022.1270-0.

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Fromherz, Peter. "Microelectronics meets Molecular and Neurobiology." In 2002 International Conference on Solid State Devices and Materials. The Japan Society of Applied Physics, 2002. http://dx.doi.org/10.7567/ssdm.2002.pl-3.

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DOLAN, RAY J. "THE NEUROBIOLOGY OF EMOTION AND MOOD." In Proceedings of the International School of Biocybernetics. WORLD SCIENTIFIC, 2002. http://dx.doi.org/10.1142/9789812776563_0008.

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De Valois, Karen K. "Spatial vision based upon color differences." In Computational Vision Based on Neurobiology, edited by Teri B. Lawton. SPIE, 1994. http://dx.doi.org/10.1117/12.171146.

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Stevenson, Scott B., Clifton M. Schor, and Lawrence K. Cormack. "Disparity tuning of cyclopean visual mechanisms." In Computational Vision Based on Neurobiology, edited by Teri B. Lawton. SPIE, 1994. http://dx.doi.org/10.1117/12.171145.

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Oram, Michael W., and David I. Perrett. "Neural processing of biological motion in the macaque temporal cortex." In Computational Vision Based on Neurobiology, edited by Teri B. Lawton. SPIE, 1994. http://dx.doi.org/10.1117/12.171138.

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Bonds, A. B. "Coding of inhibition in visual cortical spike streams." In Computational Vision Based on Neurobiology, edited by Teri B. Lawton. SPIE, 1994. http://dx.doi.org/10.1117/12.171134.

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Reports on the topic "Neurobiology"

1

Copenhagen, David. Retinal Neurobiology and Visual Processing. Fort Belvoir, VA: Defense Technical Information Center, October 1996. http://dx.doi.org/10.21236/ada325859.

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Lucas, Ashley. Neurobiology of Seasonal Life-history Transitions. Portland State University Library, January 2000. http://dx.doi.org/10.15760/etd.2508.

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McGaugh, James L., Gary Lynch, and Norman M. Weinberger. Conference on the Neurobiology of Learning and Memory (3rd). Fort Belvoir, VA: Defense Technical Information Center, September 1988. http://dx.doi.org/10.21236/ada201631.

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Germain, Anne. Neurobiology of Sleep and Sleep Treatment Response in PTSD. Fort Belvoir, VA: Defense Technical Information Center, October 2009. http://dx.doi.org/10.21236/ada525916.

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Gebhardt, Stefan. The neurobiology of depression and the dilemma of pain treatment. Science Repository Oü, October 2018. http://dx.doi.org/10.31487/j.nnb.2018.10.007.

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Germain, Anne. Neurobiology of Sleep and Sleep Treatments in PTSD (NOS-STIP). Fort Belvoir, VA: Defense Technical Information Center, October 2010. http://dx.doi.org/10.21236/ada537223.

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Henderson, Brian E. ,. M. D. Development of Structural Neurobiology and Genomics Programs in the Neurogenetic Institute. Office of Scientific and Technical Information (OSTI), November 2006. http://dx.doi.org/10.2172/894898.

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Silman, Israel. XIth International Symposium on Cholinergic Mechanisms - Function and Dysfunction & 2nd Misrahi Symposium on Neurobiology. Fort Belvoir, VA: Defense Technical Information Center, July 2002. http://dx.doi.org/10.21236/ada410482.

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McGaugh, James L., Gary Lynch, Norman M. Weinberger, and Larry R. Squire. Conference on the Neurobiology of Learning and Memory (4th) Held in Irvine, California on 17-20 October 1990. Fort Belvoir, VA: Defense Technical Information Center, February 1992. http://dx.doi.org/10.21236/ada247174.

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Iasevoli, Felice, Camilla Avagliano, Luigi D'Ambrosio, Annarita Barone, Mariateresa Ciccarelli, Giuseppe De Simone, Benedetta Mazza, Licia Vellucci, and Andrea de Bartolomeis. Dopamine dynamics and neurobiology of non-response to antipsychotics, relevance for Treatment Resistant Schizophrenia. A systematic review and critical appraisal. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, February 2023. http://dx.doi.org/10.37766/inplasy2023.2.0104.

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You might also be interested in the extended bibliographies on the topic 'Neurobiology' for particular source types:
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