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Relevant bibliographies by topics / Neural transplantation; Parkinson's disease

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Academic literature on the topic 'Neural transplantation; Parkinson's disease'

Author: Grafiati

Published: 4 June 2021

Last updated: 3 February 2022

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Journal articles on the topic "Neural transplantation; Parkinson's disease"

1

Mehta, V., J. Spears, and I. Mendez. "Neural Transplantation in Parkinson's Disease." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 24, no. 04 (November 1997): 292–301. http://dx.doi.org/10.1017/s0317167100032959.

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ABSTRACT:Parkinson's disease is a neurodegenerative disorder that affects about 1% of Canadians between the ages of fifty and seventy. The medical management for these patients consists of drug therapy that is initially effective but has limited long term benefits and does not alter the progressive course of the disease. The recalcitrance of longstanding Parkinson's disease to medical management has prompted the use of alternative surgical therapies. Many neurosurgical procedures have been utilized in order to improve the disabling symptoms these patients harbour. Although most of the current procedures involve making destructive lesions within various basal ganglia nuclei, neural transplantation attempts to reconstitute the normal nigrostriatal pathway and restore striatal dopamine. The initial success of neural transplantation in the rodent and primate parkinsonian models has led to its clinical application in the treatment of parkinsonian patients. Currently, well over one hundred patients throughout the world have been grafted with fetal tissue in an effort to ameliorate their parkinsonian symptoms. Although the results of neural transplantation in clinical trials are promising, a number of issues need to be resolved before this technology can become a standard treatment option. This review focuses on the current status of neural transplantation in Parkinson's disease within the context of other surgical therapies in current use.

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Lindvall, Olle. "Neural Transplantation." Cell Transplantation 4, no. 4 (July 1995): 393–400. http://dx.doi.org/10.1177/096368979500400410.

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Cell transplantation is now being explored as a new therapeutic strategy to restore function in the diseased human central nervous system. Neural grafts show long-term survival and function in patients with Parkinson's disease but the symptomatic relief needs to be increased. Cell transplantation seems justified in patients with Huntington's disease and, at a later stage, possibly also in demyelinating disorders. The further development in this research field will require systematic studies in animal experiments but also well-designed clinical trials in small groups of patients.

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Shimizu, K., M. Yamada, Y. Matsui, K. Tamura, S. Moriuchi, and H. Mogami. "Neural Transplantation in Mouse Parkinson's Disease." Stereotactic and Functional Neurosurgery 54, no. 1-8 (1990): 353–57. http://dx.doi.org/10.1159/000100234.

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Hagell, Peter, Paola Piccini, Anders Björklund, Patrik Brundin, Stig Rehncrona, Håkan Widner, Lesley Crabb, et al. "Dyskinesias following neural transplantation in Parkinson's disease." Nature Neuroscience 5, no. 7 (June 3, 2002): 627–28. http://dx.doi.org/10.1038/nn863.

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Borlongan, Cesario V., and Paul R. Sanberg. "Neural transplantation for treatment of Parkinson's disease." Drug Discovery Today 7, no. 12 (June 2002): 674–82. http://dx.doi.org/10.1016/s1359-6446(02)02297-3.

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Kupsch, Andreas, and Wolfgang H. Oertel. "Neural transplantation, trophic factors and Parkinson's disease." Life Sciences 55, no. 25-26 (January 1994): 2083–95. http://dx.doi.org/10.1016/0024-3205(94)00389-0.

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Freed, William J. "Neural Transplantation: Prospects for Clinical use." Cell Transplantation 2, no. 1 (January 1993): 13–31. http://dx.doi.org/10.1177/096368979300200105.

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Neural transplantation has been extensively applied in Parkinson's disease, including numerous clinical studies, studies in animal models, and related basic research on cell biology. There is evidence that the clinical trials of both adrenal medulla transplantation and fetal substantia nigra transplantation have produced a detectable clinical effect, although it is not yet clear whether the clinical benefit is sufficient to justify a more widespread application of these procedures. Studies of long-term outcome and quantitative tests are important in assaying the degree of benefit produced by transplantation procedures in Parkinson's disease and for developing improved and refined procedures. Other disease-related applications of neural transplantation are beginning to be developed. These include Huntington's disease, chronic pain, epilepsy, spinal cord injury, and perhaps even demyelinating diseases and cortical ischemic injury. Although most of these applications lie in the future, it is not too soon to begin to consider the scientific justification that should be required for initiation of human clinical trials.

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Madrazo, Ignacio, Rebecca Franco-Bourland, Maricarmen Aguilera, Feggy Ostrosky-Solis, Carlos Cuevas, Hugo Castrejón, Eduardo Magalloón, and Mario Madrazo. "Development of Human Neural Transplantation." Neurosurgery 29, no. 2 (August 1, 1991): 165–77. http://dx.doi.org/10.1227/00006123-199108000-00001.

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Abstract The possibility of altering the course of Parkinson's disease by brain grafting is slowly becoming a reality through the efforts of many research groups worldwide. It has been shown that this procedure, as performed in high-level medical research centers, usually produces no permanent adverse effects and can effectively ameliorate parkinsonian signs in certain patients. This progress has served to reinforce our commitment to develop neural transplantation into an effective therapy to treat such a devastating neurodegenerative disease. We have summarized the most important events that have shaped the initial phase of this research. In the course of the last 4 years, considerable knowledge has been gained in the clinical neurosciences regarding the real potential of various brain grafting procedures in treating Parkinson's disease, their shortcomings, and their usefulness in carefully selected patients. There is still no consensus regarding the various fundamental aspects of human brain grafting in Parkinson's disease. Questions concerning surgical technique, candidate selection, the optimal brain regions for implantation, the optimal tissue for implantation, and the real usefulness of brain grafting must be addressed. The importance of the quality of adrenal medulla fragments for grafting, the requirement for immunosuppressors in fetal brain grafting, and the optimal fetal age and the amount of donor tissue for effective grafting are additional areas of concern. The potential of xenografting, preserved tissues, and genetically engineered cells for human brain grafting remain unanswered. The development of human neural transplantation is the responsibility and privilege of neurosurgery.

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9

Rosenfeld, J. V., T. J. Kilpatrick, and P. F. Bartlett. "Neural transplantation for Parkinson's disease: a critical appraisal." Australian and New Zealand Journal of Medicine 21, no. 4 (August 1991): 477–84. http://dx.doi.org/10.1111/j.1445-5994.1991.tb01357.x.

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Björklund, Anders, Stephen B. Dunnett, Patrik Brundin, A. Jon Stoessl, Curt R. Freed, Robert E. Breeze, Marc Levivier, Marc Peschanski, Lorenz Studer, and Roger Barker. "Neural transplantation for the treatment of Parkinson's disease." Lancet Neurology 2, no. 7 (July 2003): 437–45. http://dx.doi.org/10.1016/s1474-4422(03)00442-3.

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Dissertations / Theses on the topic "Neural transplantation; Parkinson's disease"

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Baker, K. Adam. "Neural transplantation in the rat model of Parkinson's disease." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape4/PQDD_0021/MQ57270.pdf.

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Åkerud, Peter. "GDNF family ligands and neural stem cells in Parkinson's disease /." Stockholm : [Karolinska Univ. Press], 2001. http://diss.kib.ki.se/2001/91-7349-042-3/.

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Hurelbrink, Carrie Brienne. "Optimisation of neural transplantation for Parkinson's and Huntington's diseases." Thesis, University of Cambridge, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.431504.

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Kuan, Wei-Li. "Strategies to improve neural transplantation in Parkinson's disease using fetal ventral mesencephalic tissue." Thesis, University of Cambridge, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.613243.

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Love, Rebecca Margaret. "Improving the survival of embryonic dopaminergic neurons." Thesis, University of Oxford, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.343277.

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Armstrong, Richard James Ernest. "Cell replacement therapy through transplantation of expanded neural precursor cells : experiments in animal models of Parkinson's and Huntington's diseases." Thesis, University of Cambridge, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.431572.

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Ziemba, Kristine S. "TARGETING AXON GROWTH FROM NEURONS TRANSPLANTEDINTO THE CENTRAL NERVOUS SYSTEM." UKnowledge, 2007. http://uknowledge.uky.edu/gradschool_diss/497.

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Damage to the adult mammalian central nervous system (CNS), either by traumatic injury or disease, usually results in permanent sensory and/or motor deficits. Regeneration of neural circuits is limited both by the lack of growthpromoting molecules and by the presence of growth-inhibitory molecules in the mature brain and spinal cord. The research described here examines the therapeutic potential of viral vectors and neuronal transplants to reconstruct damaged neural pathways in the CNS. Experimental neural transplantation techniques often fall short of expectations because of limited transplant survival and insufficient neurite outgrowth to repair connections and induce behavioral recovery. These shortcomings are addressed in the current studies by virus-mediated expression of cell-specific neurotrophic and guidance molecules in the host brain prior to cell transplantation. The initial proof-of-principle studies show that viral vectors can be used to create axon-guidance pathways in the adult mammalian brain. With such pathways in place, subsequent transplantation of neurons leads to longdistance, targeted outgrowth of neurites. Application of this technique to a rat model of Parkinsons disease demonstrates that circuit reconstruction leads to functional recovery. For this study, rats were lesioned on one side of their brain with 6-hydroxydopamine to produce a hemiparkinsonian state. The motor deficit was confirmed by amphetamine-induced rotation testing and spontaneous motor asymmetry testing. The rats were then divided into experimental groups to receive lentivirus injections along a path between the substantia nigra (SN) and the striatum to express glial cell-line derived neurotrophic factor (GDNF), GDNF family receptor alpha-1 (GFR1), netrin-1 or green fluorescent protein (GFP, control). One group received combination injections of lenti-GDNF and lenti-GFR1. One week after virus injections, animals received transplants of embryonic midbrain dopaminergic neurons into their SNs. They were tested for motor asymmetry every two weeks for a total of eight weeks and then brain tissue was harvested for immunohistochemical analysis. Results demonstrate that virus-induced expression of GDNF and GFR1 supports growth of dopaminergic fibers from cells transplanted into the SN all the way to the striatum, and these animals have a significant reduction in both drug-induced and spontaneous motor asymmetry.

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Droguerre, Marine. "Thérapie cellulaire de la maladie de Parkinson : transplantation intranigrale vs intrastriatale." Thesis, Poitiers, 2015. http://www.theses.fr/2015POIT2320/document.

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La maladie de Parkinson (MP) est une pathologie neurodégénérative associée principalement à une perte progressive de neurones dopaminergiques de la substance noire (SN) conduisant à une diminution de dopamine au niveau du striatum. Une des approches thérapeutiques expérimentales de la MP est la greffe de neurones dopaminergiques, non pas au niveau de la SN, mais directement dans la région cible, le striatum, et ceux avec des résultats variables. Dans cette étude, nous avons comparé en détail la récupération fonctionnelle suite à la transplantation de mésencéphale ventral (MV) fœtal provenant de souris exprimant la GFP sous le contrôle du promoteur de la tyrosine hydroxylase soit au niveau de la SN soit dans le striatum de souris adultes lésées unilatéralement à la 6-hydroxydopamine. Les conséquences anatomiques et fonctionnelles ont été analysées par des approches comportementales, électrophysiologiques et immunohistochimiques. Nos résultats montrent que les neurones greffés dans les deux emplacements envoient des projections vers le striatum. De plus, les deux types de greffes induisent une amélioration significative de la motricité ainsi que de l'activité des neurones du striatum. Toutefois, seule la greffe intranigrale a permis la restauration de la motricité fine des membres antérieurs et un retour à une excitabilité des neurones striataux à l’état basal
Parkinson’s disease (PD) is a neurodegenerative disorder associated with a progressive loss of dopaminergic (DA) neurons in the substantia nigra (SN), leading to a loss of dopamine in the striatum. One of the experimental therapeutic approaches in PD is the graft of DA neurons not in their ontogenic site, the SN, but directly into the target region, the striatum and those leads to variable results. In this study, we have analyzed in detail the functional outcome of fetal VM tissue expressing GFP under the control of a tyrosine hydroxylase promoter grafts placed either into the SN or striatum in unilaterally 6-hydroxydopamine lesioned adult mice. Anatomical and functional outcome were analyzed using behavioral, electrophysiological and immunohistochemical approaches. Our results show that transplanted neurons in both locations can survive and re-innervate the striatum. Furthermore, both grafts locations significantly restored motor performance and induced the recovery of striatal firing properties. However, only intranigral transplantation allows recovery of fine motor skills of previous members and efficiently normalized cortico-striatal responses

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9

Scholz, Torben [Verfasser], and Guido [Akademischer Betreuer] Nikkhah. "Development of dopaminergic neurons from neural stem cells and transplantation into a neonatal and adult animal model of Parkinson’s disease = Entwicklung dopaminerger Neurone aus neuralen Stammzellen und Transplantation in ein neonatales und adultes Modell der Parkinsonschen Krankheit." Freiburg : Universität, 2011. http://d-nb.info/112346460X/34.

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Mo, Mimi Shin Ning. "Neural vulnerability in models of Parkinson's disease." Thesis, University of Oxford, 2007. http://ora.ox.ac.uk/objects/uuid:ac82e1c1-5d9f-473f-97ac-fcb70b2587ca.

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Parkinson's disease (PD) is a neurodegenerative disorder with no known cure. This thesis explores the degenerative process in two neurotoxin-based models, the 6-hydroxydopamine and the chronic 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine(MPTP)/probenecid mouse models, to yield important information about the pathogenesis of PD. Neuronal survival patterns in Parkinsonian patients and animals are heterogeneous. More dopaminergic neurons are lost from the ventral tier of the substantia nigra (SN) than from the dorsal tier or the adjacent ventral tegmental area, possibly due to differential expression of the calcium-binding protein, calbindin D28K. Brain sections were processed for tyrosine hydroxylase (TH) and calbindin (CB) immunocytochemistry to distinguish the dopaminergic subpopulations. I show that more TH+/CB- and TH-/CB+ than TH+/CB+ neurons are lost in both models, suggesting that CB confers some degree of protection for dopaminergic neurons. With respect to connectivity, I show that both TH+ and CB+ neurons receive striatal and dorsal raphe inputs. I investigated the possibility of a progressive loss in midbrain neurons by prolonging the post-lesion survival period. In both models, there is an irreversible neuronal cell loss of TH+, CB+ and TH+/CB+ neurons but the effects of survival time and lesion treatments differ for the three neuronal types. The lesions also appear to be toxic to GABAergic neurons. I explore whether, once neurodegeneration has started, neurons can be rescued by pharmacological intervention. Salicylic acid appears both to reduce microglial activation and significantly improve TH+, but not CB+ or TH+/CB+ neuronal survival. PD appears multifactorial in origin and may involve complex interactions between genetic and environmental influences. I show that a xenobiotic-metabolising enzyme, arylamine N-acetyltransferase may fulfil a neuroprotective role in the SN by limiting the environmental risks. Taken together, this study provides a body of information on two different mouse PD models and highlights possible genetic predispositions to PD neuropathology.

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Books on the topic "Neural transplantation; Parkinson's disease"

1

Freed, Curt. Healing the brain: A doctor's controversial quest for a cell therapy to cure Parkinson's disease. New York: Times Books/Henry Holt, 2002.

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Freed, Curt. Healing the brain: A doctor's controversial quest for a cell therapy to cure Parkinson's disease. New York: Times Books/Henry Holt, 2002.

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3

Dong, Jing-fei. Morphological and biochemical characterization of human second trimester foetal dopaminergic neurones and identification of factors influencing their survival and preservation in vitro: A study related to clinical neural transplantation for Parkinson's disease. Wolverhampton: University of Wolverhampton, 1993.

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Triarhou, Lazaros C. Dopaminergic Neuron Transplantation in the Weaver Mouse Model of Parkinson’s Disease. Boston, MA: Springer US, 2002. http://dx.doi.org/10.1007/978-1-4615-0699-7.

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Green, C. J. Current status of fetal tissue transplantation in Parkinson's disease. Vancouver, B.C: B.C. Office of Health Technology Assessment, Centre for Health Services & Policy Research, University of British Columbia, 1994.

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Chadwick, Derek J., and Jamie A. Goode, eds. Neural Transplantation in Neurodegenerative Disease: Current Status and New Directions. Chichester, UK: John Wiley & Sons, Ltd, 2000. http://dx.doi.org/10.1002/0470870834.

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Maneuf, Yannick P. M. Modulation of neural transmission in the basal ganglia: Implications for the treatmentof Parkinson's disease. Manchester: University of Manchester, 1994.

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Jon, Palfreman, ed. The case of the frozen addicts. New York: Pantheon Books, 1995.

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Committee on the Use of Animals in Research (U.S.), National Academy of Sciences (U.S.), and Institute of Medicine (U.S.), eds. Science, medicine, and animals. Washington, D.C: National Academy Press, 1991.

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Triarhou, Lazaros C. Dopaminergic Neuron Transplantation in the Weaver Mouse Model of Parkinson's Disease. Springer, 2012.

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Book chapters on the topic "Neural transplantation; Parkinson's disease"

1

Freeman, T. B., A. E. Willing, T. Zigova, P. R. Sanberg, and R. A. Hauser. "Neural Transplantation in Parkinson's Disease." In Progress in Neurological Surgery, 331–38. Basel: KARGER, 2000. http://dx.doi.org/10.1159/000062061.

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Lindvall, Olle. "Neural Transplantation in Parkinson's Disease." In Neural Transplantation in Neurodegenerative Disease: Current Status and New Directions, 110–28. Chichester, UK: John Wiley & Sons, Ltd, 2008. http://dx.doi.org/10.1002/0470870834.ch7.

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Kohsaka, S. "Immunological aspects of neural transplantation." In Parkinson’s Disease. From Clinical Aspects to Molecular Basis, 119–27. Vienna: Springer Vienna, 1991. http://dx.doi.org/10.1007/978-3-7091-9146-0_12.

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Ridet, Jean-Luc, Nicole Déglon, and Patrick Aebischer. "Gene Transfer Techniques for the Delivery of GDNF in Parkinson's Disease." In Neural Transplantation in Neurodegenerative Disease: Current Status and New Directions, 202–19. Chichester, UK: John Wiley & Sons, Ltd, 2008. http://dx.doi.org/10.1002/0470870834.ch13.

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Bohn, Martha C., Bronwen Connor, Dorothy A. Kozlowski, and M. Hasan Mohajeri. "Gene Transfer for Neuroprotection in Animal Models of Parkinson's Disease and Amyotrophic Lateral Sclerosis." In Neural Transplantation in Neurodegenerative Disease: Current Status and New Directions, 70–93. Chichester, UK: John Wiley & Sons, Ltd, 2008. http://dx.doi.org/10.1002/0470870834.ch5.

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Isacson, Ole, Peyman Pakzaban, and Wendy R. Galpern. "Transplanting Fetal Neural Xenogeneic Cells in Parkinson’s and Huntington’s Disease Models." In Cell Transplantation for Neurological Disorders, 189–210. Totowa, NJ: Humana Press, 1998. http://dx.doi.org/10.1007/978-1-59259-476-4_10.

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Pardal, Ricardo, and José López-Barneo. "Neural Stem Cells and Transplantation Studies in Parkinson’s Disease." In Advances in Experimental Medicine and Biology, 206–16. New York, NY: Springer US, 2012. http://dx.doi.org/10.1007/978-1-4614-2098-9_14.

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Motono, Makoto, Asuka Morizane, and Jun Takahashi. "Induction and Transplantation of Neural Stem Cells from Human Pluripotent Stem Cells in Experimental Models (Rat) of Parkinson's disease." In Neural Stem Cell Assays, 189–95. Hoboken, NJ, USA: John Wiley & Sons, Inc, 2015. http://dx.doi.org/10.1002/9781118308295.ch19.

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Bjugstad, Kimberly B., and John R. Sladek. "Neural Transplantation in the Nonhuman Primate Model of Parkinson’s Disease." In Cell Therapy, Stem Cells, and Brain Repair, 61–82. Totowa, NJ: Humana Press, 2006. http://dx.doi.org/10.1007/978-1-59745-147-5_3.

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Sugaya, Kiminobu, Tingyu Qu, Hojoong M. Kim, and Christopher L. Brannen. "Human Neural Stem Cell Transplantation Improves Cognitive Function of Aged Brain." In Mapping the Progress of Alzheimer’s and Parkinson’s Disease, 229–34. Boston, MA: Springer US, 2002. http://dx.doi.org/10.1007/978-0-306-47593-1_38.

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Conference papers on the topic "Neural transplantation; Parkinson's disease"

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Wang, Haijuna, Ye Luo, Ziying Liu, and Chunxi Man. "Experimental study on Neural stem cell transplantation therapy modified by the gene of tyrosine hydroxylase and Glial cell line derived neurotrophic factor for Parkinson's disease rat model." In International Conference on Medical Engineering and Bioinformatics. Southampton, UK: WIT Press, 2014. http://dx.doi.org/10.2495/meb140821.

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Bai, Qifan, Tianyu Shen, Baoteng Xu, Qian Yu, Huijun Zhang, Chengjie Mao, Chunfeng Liu, and Shouyan Wang. "Quantification of the motor symptoms of Parkinson's disease." In 2017 8th International IEEE/EMBS Conference on Neural Engineering (NER). IEEE, 2017. http://dx.doi.org/10.1109/ner.2017.8008297.

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Prashanth, R., S. Dutta Roy, S. Ghosh, and Pravat K. Mandal. "Shape features as biomarkers in early Parkinson's disease." In 2013 6th International IEEE/EMBS Conference on Neural Engineering (NER). IEEE, 2013. http://dx.doi.org/10.1109/ner.2013.6695985.

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Zhang, Xiaobo, Yan Yang, Hao Wang, Shangming Ning, and Heng Wang. "Deep Neural Networks with Broad Views for Parkinson's Disease Screening." In 2019 IEEE International Conference on Bioinformatics and Biomedicine (BIBM). IEEE, 2019. http://dx.doi.org/10.1109/bibm47256.2019.8983000.

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Bakar, Z. A., N. M. Tahir, and I. M. Yassin. "Classification of Parkinson's disease based on Multilayer Perceptrons Neural Network." In its Applications (CSPA). IEEE, 2010. http://dx.doi.org/10.1109/cspa.2010.5545301.

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Salimi-Badr, Armin, and Mohammad Hashemi. "A Neural-Based Approach to Aid Early Parkinson's Disease Diagnosis." In 2020 11th International Conference on Information and Knowledge Technology (IKT). IEEE, 2020. http://dx.doi.org/10.1109/ikt51791.2020.9345635.

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Li, Liang, Qian Yu, Baoteng Xu, Qifan Bai, Yunpeng Zhang, Huijun Zhang, Chengjie Mao, Chunfeng Liu, Tianyu Shen, and Shouyan Wang. "Multi-sensor wearable devices for movement monitoring in Parkinson's disease." In 2017 8th International IEEE/EMBS Conference on Neural Engineering (NER). IEEE, 2017. http://dx.doi.org/10.1109/ner.2017.8008294.

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Maziewski, P., P. Suchomski, B. Kostek, and A. Czyzewski. "An intuitive graphical user interface for the Parkinson's Disease patients." In 2009 4th International IEEE/EMBS Conference on Neural Engineering (NER). IEEE, 2009. http://dx.doi.org/10.1109/ner.2009.5109223.

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Shaban, Mohamed. "Automated Screening of Parkinson's Disease Using Deep Learning Based Electroencephalography." In 2021 10th International IEEE/EMBS Conference on Neural Engineering (NER). IEEE, 2021. http://dx.doi.org/10.1109/ner49283.2021.9441065.

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Kraipeerapun, Pawalai, and Somkid Amornsamankul. "Using stacked generalization and complementary neural networks to predict Parkinson's disease." In The 2015 11th International Conference on Natural Computation. IEEE, 2015. http://dx.doi.org/10.1109/icnc.2015.7378178.

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Reports on the topic "Neural transplantation; Parkinson's disease"

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Isacson, Ole. Knock-Out and Transgenic Strategies to Improve Neural Transplantation Therapy for Parkinson's Disease. Fort Belvoir, VA: Defense Technical Information Center, July 2001. http://dx.doi.org/10.21236/ada396461.

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