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Books on the topic 'Neural crest cells'

Author: Grafiati

Published: 4 June 2021

Last updated: 22 June 2024

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1

Schwarz, Quenten, and Sophie Wiszniak, eds. Neural Crest Cells. New York, NY: Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4939-9412-0.

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2

Hall, Brian K., ed. The Neural Crest and Neural Crest Cells in Vertebrate Development and Evolution. Boston, MA: Springer US, 2009. http://dx.doi.org/10.1007/978-0-387-09846-3.

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3

Maya, Sieber-Blum, ed. Neurotrophins and the neural crest. Boca Raton, Fla: CRC Press, 1999.

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4

Neural Crest Cells. Elsevier, 2014. http://dx.doi.org/10.1016/c2012-0-00698-9.

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5

Medeiros, Daniel Meulemans, Brian Frank Eames, and Igor Adameyko. Evolving Neural Crest Cells. Taylor & Francis Group, 2020.

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6

Sieber-Blum, Maya. Neural Crest Stem Cells. WORLD SCIENTIFIC, 2011. http://dx.doi.org/10.1142/8127.

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7

Medeiros, Daniel Meulemans, Brian Frank Eames, and Igor Adameyko. Evolving Neural Crest Cells. Taylor & Francis Group, 2020.

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8

Medeiros, Daniel Meulemans, Brian Frank Eames, and Igor Adameyko. Evolving Neural Crest Cells. Taylor & Francis Group, 2020.

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9

Medeiros, Daniel Meulemans, Brian Frank Eames, and Igor Adameyko. Evolving Neural Crest Cells. Taylor & Francis Group, 2022.

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10

Medeiros, Daniel Meulemans, Brian Frank Eames, and Igor Adameyko. Evolving Neural Crest Cells. Taylor & Francis Group, 2020.

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11

Neural Crest Cells: Methods and Protocols. Springer New York, 2019.

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12

Schwarz, Quenten, and Sophie Wiszniak. Neural Crest Cells: Methods and Protocols. Springer New York, 2020.

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13

Hall, Brian K. The Neural Crest and Neural Crest Cells in Vertebrate Development and Evolution. Springer, 2010.

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14

Neural Crest Stem Cells Breakthroughs And Applications. World Scientific Publishing Company, 2012.

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15

Neural Crest Cells: Evolution, Development and Disease. Elsevier Science & Technology Books, 2013.

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16

(Editor), B. Kramer, and B. B. Rawdon (Editor), eds. Embryos, Endocrine Cells and the Neural Crest. Witwatersrand University Press, 1995.

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17

Trainor, Paul. Neural Crest Cells: Evolution, Development and Disease. Elsevier Science & Technology Books, 2013.

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18

Medeiros, Daniel Meulemans. Origin and Evolution of Neural Crest Cells. Taylor & Francis Group, 2020.

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19

Dyer, Laura A., and Margaret L. Kirby. The role of the neural crest in cardiac development. Edited by José Maria Pérez-Pomares, Robert G. Kelly, Maurice van den Hoff, José Luis de la Pompa, David Sedmera, Cristina Basso, and Deborah Henderson. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757269.003.0019.

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The cardiac neural crest (CNC) plays pivotal roles in numerous steps of cardiac development. Every aspect of the CNC cell’s lifespan is highly orchestrated, from its induction in the dorsal neural tube to its migration to its differentiation at its final destination. During migration, CNC cells are affected by their environment and simultaneously modulate the extra-cellular milieu through which they migrate. In the pharyngeal arches, CNC cells repattern the originally symmetrical arch arteries, producing the great arteries. Because the cardiac neural crest is essential for many aspects of heart development, it is unsurprising that human CNC-related syndromes have severe phenotypes. This chapter describes how CNC cells are formed and contribute to their final destinations. Essential signalling pathways are presented in the context of CNC development, and CNC-related syndromes are included to highlight this population’s broad importance during development.

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20

Hall, Brian K. The Neural Crest and Neural Crest Cells in Vertebrate Development and Evolution (Springer series in solid-state sciences). Springer, 2008.

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21

Carpenter, Ellen Malinin. An analysis of the migration, location, and distribution of neural crest-derived Schwann cells in chick embryos. 1988.

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22

Pomeranz, Howard David. Formation of the enteric nervous system: Derivation of precursors from both sacral and vagal levels of the neural crest, pathways to the bowel followed by emigrés from the crest, and the ability of enteric crest-derived cells to interact with laminin. 1991.

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23

Kalcheim, Chaya, and Nicole Le Douarin. The Neural Crest (Developmental and Cell Biology Series). 2nd ed. Cambridge University Press, 1999.

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24

Rammah, Mayyasa, Francesca Rochais, and Robert G. Kelly. Incorporation of myocardial progenitors at the arterial pole of the heart. Edited by José Maria Pérez-Pomares, Robert G. Kelly, Maurice van den Hoff, José Luis de la Pompa, David Sedmera, Cristina Basso, and Deborah Henderson. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757269.003.0007.

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The arterial pole of the heart is a hotspot for life-threatening forms of congenital heart defects (CHDs). It is formed by progressive addition of myocardium from epithelial progenitor cells in the second heart field (SHF). SHF cells contribute successively to the right ventricle and proximal and distal outflow tract myocardial walls which, after neural crest influx and cardiac septation, give rise to myocardium at the base of the aorta and pulmonary trunk. SHF cells are characterized by continued proliferation and differentiation delay controlled by an array of transcriptional regulators and signalling pathways which define the SHF progenitor cell niche in pharyngeal mesoderm. Failure of normal SHF deployment leads to a shortened outflow tract and failure of ventriculo-arterial alignment, resulting in a spectrum of conotruncal CHD. We discuss the origins of the SHF in cardiopharyngeal mesoderm and focus on the mechanisms driving SHF deployment, summarizing current understanding of critical signalling pathways and transcription factors.

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25

Trainor, Paul. Neural Crest and Placodes. Elsevier Science & Technology Books, 2015.

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26

Trainor, Paul. Neural Crest and Placodes. Elsevier Science & Technology Books, 2015.

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27

Brown, Christina, and Meredith Kato. Neuroblastoma. Edited by Kirk Lalwani, Ira Todd Cohen, Ellen Y. Choi, and Vidya T. Raman. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190685157.003.0033.

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Neuroblastoma is a solid tumor that arises from neural crest cells. It is a common cancer of childhood, accounting for 7.8% of all childhood malignancy. It affects primarily infants and toddlers with older children carrying a worse prognosis. While surgery is a primary treatment modality, these tumors are not well encapsulated and tend to invade adjacent structures making resection more difficult. As such, patients often come to the operating room after neoadjuvant chemotherapy and the surgeries can be long with large blood losses. Anesthesia for the resection for neuroblastoma must take into account the effect of ongoing treatment on the patients as well as the surgical challenges inherent in this disease.

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28

Armstrong, Bruce K., Claire M. Vajdic, and Anne E. Cust. Melanoma. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0057.

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Melanoma is a cancer of melanocytes, cells that produce the brown-black skin pigment melanin. Melanocytes originate in cells of the neural crest and migrate during embryogenesis, principally to the epidermis, eyes, and some mucous membranes (mouth, nose, esophagus, anus, genitourinary organs, and conjunctiva). Cutaneous melanoma afflicts mainly fair-skinned people of European origin, among whom sun exposure is the major cause. Five-year relative survival can exceed 90%. Invasive cutaneous melanoma in US whites occurs mostly on the trunk (34%), and upper limbs and shoulders (26%). Melanoma incidence rates have been increasing predominantly in European-origin populations. Ultraviolet (UV) radiation, from the sun or artificial tanning devices, probably both initiates and promotes melanoma. Nevi are markers of increased melanoma risk and direct precursors in some cases; nevus-prone people may require only modest sun exposure to initiate melanoma. Other risk factors include family history and sun sensitivity.

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29

Beltran, Ralph J. Pheochromocytoma. Edited by Kirk Lalwani, Ira Todd Cohen, Ellen Y. Choi, and Vidya T. Raman. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190685157.003.0044.

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Pheochromocytoma is a rare type of neoplasm diagnosed in children. It originates in the adrenal gland and is different from paragangliomas which arise outside the adrenals. Both types of tumors arise from neural crest cells and lead to signs and symptoms related to hypersecretion of catecholamines. Related symptoms include hypertension, tachycardia, episodic headache, sweating, and abdominal pain. These tumors may be associated with multiple endocrine type 2 syndrome, multiple endocrine neoplasia, and von Hippel-Lindau disease, among other hereditary conditions. Pheochromocytomas can be malignant in nature but may be indistinguishable from benign disease histologically and functionally. When malignant, they present with regional invasion or distant metastasis. Conditions presenting as sympathetic overactivity in pediatric patients can resemble pheochromocytoma (i.e., panic disorder, amphetamine consumption).

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30

Zaffran, Stéphane. Cardiac growth II: Cardiomyocyte polarization. Edited by José Maria Pérez-Pomares, Robert G. Kelly, Maurice van den Hoff, José Luis de la Pompa, David Sedmera, Cristina Basso, and Deborah Henderson. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757269.003.0010.

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During vertebrate embryogenesis, the planar cell polarity (PCP) signalling pathway is responsible for cell movements essential for convergent extension during gastrulation, neural tube closure, neural crest cell migration, and heart morphogenesis. In the heart, the non-canonical Wnt/PCP pathway regulates cell polarity, cell shape, and cell dynamics during formation of the cardiac crescent and deployment of second heart field cardiac progenitors to the poles of the heart tube. PCP signalling is also essential for the establishment of left–right patterning in the early embryo. This chapter reviews our current understanding of PCP signalling in heart morphogenesis and how it affects the pathogenesis of congenital heart diseases.

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31

Neural crest cell lineage restricts skeletal muscle progenitor cell differentiation through Neuregulin1-ErbB3 signaling. Developmental Cell, 2011.

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32

Craniofacial Development: The Tissue and Molecular Interactions That Control Development of the Head (Advances in Anatomy, Embryology and Cell Biology). Springer, 2004.

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