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Books on the topic 'Neoplastic lesion'

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Author: Grafiati

Published: 9 March 2023

Last updated: 10 March 2023

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1

Frith, C. H. Color atlas of neoplastic and non-neoplastic lesions in aging mice. Amsterdam: Elsevier, 1988.

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2

Frith, C. H. Incidence of neoplastic and nonneoplastic lesions in several strains of mice. Little Rock, AR: Toxicology Pathology Associates, 1987.

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3

Chevanne, Marta, and Riccardo Caldini. Immagini di Istopatologia. Florence: Firenze University Press, 2007. http://dx.doi.org/10.36253/978-88-5518-023-8.

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This collection of images of Histopathology is the fruit of the authors' thirty years' experience in the performance of practical exercises in General Pathology. It is aimed at students attending lessons of General Pathology on the Degree Courses in Medical Surgery and Biological Sciences. It does not aspire either to be complete from the point of view of the various organic pathologies, or to replace direct and personal observation of the histological preparations through the microscope, but is rather intended as an aid to students preparing for the exam. It does not include the rudiments of cytology and microscopic anatomy, which it is assumed have already been mastered by those approaching General Histopathology, nor are histopathological phenomena systematically addressed, for which the reader is referred to textbooks on General Pathology. The 44 preparations presented here have been grouped in line with the main arguments of General Pathology: Cellular Degeneration, Inflammation, Neoplasia both benign and malign, and Vascular Pathology. They have been selected for their didactic significance and the simplicity and clarity of the lesions present, without taking into account the information to be derived from the clinical case history. The images of the preparations, in which the best possible quality of reproduction has been sought, are presented in progressive enlargements and are accompanied by brief descriptions comprising the explanations essential for identification of the characteristic aspects of the elementary lesion, as well as any eventual defects in the preparations themselves. Effectively, the objective of the work is to enable the student to exercise his understanding of the images. For this reason the casuistics included is as essential as possible, and the method of presentation utilised is designed to avoid mere visual memorisation, stimulating first analysis and then synthesis, and the development of individual logical skills so as to indicate whether aspects of cellular pathology, inflammation or neoplasia are present.

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4

Konrad, Müller-Hermelink Hans, Neumann H. -G, and Dekant W, eds. Risk and progression factors in carcinogenesis. Berlin: Springer, 1997.

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5

Laureno, Robert. Symmetry. Edited by Robert Laureno. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190607166.003.0005.

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This chapter examines the causes of symmetric neurologic lesions in toxic and metabolic disease, trauma, neoplastic disease, vascular disease, and infectious disease. The importance of symmetry in diagnosis is emphasized. Symmetry can be a problem for the neurologist; especially on computed tomography (CT) scans, symmetric disease can be difficult to notice on the background of symmetric brain anatomy because the eye more quickly detects a one-sided lesion or multifocal asymmetric lesions than symmetric lesions. Symmetry of brain lesions is important to the clinician because it suggests a toxic, metabolic, or other chemically based cause. Diseases with symmetric pathology but unknown cause probably are chemically based; the clue of symmetry gives investigators an opportunity to pursue various avenues of research.

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6

Wheaton, Michael, Dustin Nowacek, and Zachary London. Radiculopathy and Plexopathy. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0125.

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Disorders of the nerve roots and neural plexi present with pain, numbness, or weakness in the neck, back, or extremities. Although the history and physical examination provide essential diagnostic information, imaging and electrodiagnostic studies may further aid in localizing and characterizing the underlying lesion. Causes of radiculopathy include intervertebral disc herniation, spondylosis, spinal synovial cysts, infection, metastatic disease, hematoma, or infiltrative disease. The brachial and lumbosacral plexi are susceptible to trauma, structural anomalies, neoplastic infiltration, and inflammatory processes. Management of these disorders is directed at treating the underlying cause, alleviating pain, and focused physical rehabilitation.

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7

Neoplastic Lesions Of The Skin. Demos Medical Publishing, 2013.

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8

Plaza, José, and Victor Prieto. Neoplastic Lesions of the Skin. Springer Publishing Company, Incorporated, 2013.

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9

Plaza, José, Saul Suster, and Victor G. Prieto. Demos Surgical Pathology Guides: Neoplastic Lesions of the Skin. Springer Publishing Company, Incorporated, 2013.

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10

Dean, Michael, and Karobi Moitra. Biology of Neoplasia. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0002.

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The term “cancer” encompasses a large heterogeneous group of diseases that involve uncontrolled cell growth, division, and survival, culminating in local invasion and/or distant metastases. Cancer is fundamentally a genetic disease at the cellular level. Tumors occur because clones of abnormal cells acquire multiple lesions in DNA, nearly always involving mutations, chromosomal rearrangements, and extensive alteration of the epigenome. Up to 10% of cancers also involve inherited germline mutations that are moderately to highly penetrant. Cancers begin as localized growths or premalignant lesions that may regress or disappear spontaneously, or progress to a malignant primary tumor. The somatic changes that drive abnormal growth involve activating mutations of specific oncogenes, inactivation of tumor suppressor genes, and/or disruption of epigenetic controls. The latter can result from methylation or the modification of histones and other proteins that affect the remodeling of chromosomes. Numerous non-inherited factors can cause cancer by accelerating these events.

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11

Misulis, Karl E., and E. Lee Murray. Cranial Neuropathies. Edited by Karl E. Misulis and E. Lee Murray. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190259419.003.0008.

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Single cranial neuropathies are much more common than multiple cranial neuropathies but both are discussed in this chapter. The most common single cranial neuropathies are facial, abducens, oculomotor, and optic. Multiple cranial neuropathies can develop for a variety of reasons, but among the most common are neoplastic meningitis, vasculitis, diabetes, and intrinsic brainstem lesions.

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12

Bates, David. Brainstem syndromes. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199204854.003.02409_update_001.

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Case History—A 78 yr old woman presenting with brainstem signs following a seizure.Brainstem syndromes typically cause ipsilateral cranial nerve lesions and contralateral long tract signs. They are commonly due to brainstem ischaemia, but can also be caused by neoplasia, demyelination, infective and hamartomatous lesions. Imaging, ideally with MRI rather than CT, is obligatory and only then—and possibly following other investigations to identify systemic abnormality or cerebrospinal fluid changes—can appropriate therapy be introduced....

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13

Nagpal, Ameet, and Brad Wisler. Thoracic Spinal Stenosis. Edited by Mehul J. Desai. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199350940.003.0011.

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Thoracic spinal stenosis is an uncommon pathologic condition of the spine. This chapter reviews its etiology, epidemiology, anatomic features, symptoms, diagnosis, and treatment. Four of the main causes of thoracic spinal stenosis are ossification of the ligamentum flavum, ossification of the posterior longitudinal ligament, thoracic disc herniation, and thoracic spondylosis. Even rarer secondary causes include generalized skeletal disorders, metabolic and endocrine disorders of the spine, neoplastic lesions, and vascular malformations. The chapter presents a brief review of the currently available surgical techniques. An updated review is provided of the literature on non-surgical management of the disease, mainly interventional pain management.

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14

Hatef, Jeffrey, and Russell R. Lonser. Hemangioblastoma. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190696696.003.0007.

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Hemangioblastomas are benign central nervous system tumors that are found primarily (99%) in the cerebellum, brainstem, and spinal cord. They can occur sporadically (67% of cases) or in the context of the familial neoplasia syndrome, von Hippel-Lindau disease (VHL; 33%). These lesions often remain quiescent or grow in a saltatory pattern. When these tumors cause signs or symptoms, the signs or symptoms are often associated with peritumoral cyst formation. Whether the tumor occurs sporadically or in the context of VHL, complete resection is the treatment of choice when necessary. This chapter describes the clinical, imaging, and treatment features of this neoplasm.

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15

Frisch, Morten. Penile Cancer. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0055.

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Penile cancers are rare primary malignancies located on the glans, foreskin, or shaft of the penis, excluding the urethra. The vast majority of penile cancers are epithelial tumors representing histological subtypes of squamous cell carcinoma (SCC). Most penile SCCs are believed to develop through pre-invasive lesions known as penile intraepithelial neoplasia and penile carcinoma in situ. They account for 0.1%–0.3% of all incident cancers (excluding non-melanoma skin cancers) in the United States and other developed countries and up to 1% of all cancers in some countries in sub-Saharan Africa. Penile cancers are rare in men younger than 40 years, and are typically diagnosed among men above age 60. The two most important risk factors are pathological phimosis and infection with high-risk types of human papillomaviruses (HPV), both of which are preventable conditions.

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16

Ladds, Philip. Pathology of Australian Native Wildlife. CSIRO Publishing, 2009. http://dx.doi.org/10.1071/9780643097933.

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Pathology of Australian Native Wildlife brings together in one volume available information on the pathology of Australian native vertebrate wildlife, excluding fish. It provides rapid access to documented information on diseases in Australian wildlife, domiciled either in Australia or overseas. The book comprises 45 chapters, each detailing pathological changes caused by specific pathogens including viruses, bacteria, fungi, protozoa, helminths and ectoparasites, and other injurious agents and conditions such as toxins and neoplasia affecting terrestrial and marine mammals, birds, reptiles and amphibians. Although the aim is to describe morphological (gross and microscopic) changes, the author also indicates history and clinical signs, thus providing guidance as to which lesions should be specifically searched for, and what ancillary testing might be needed to confirm a diagnosis. Illustrated throughout with colour photographs, this will be the essential reference for veterinary pathologists and clinicians, as well as wildlife researchers, zoos, wildlife parks, environmentalists, conservationists and students. Awarded a 2010 Whitley Certificate of Commendation for Zoological Resource.

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17

Neary, John, and Neil Turner. The patient with haematuria. Edited by Neil Turner. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0046.

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Haematuria is a common presenting feature of diseases of the kidney or the renal tract. It is also common in screening tests, single dipstick tests being positive in perhaps 5% of individuals. Age and whether the blood is visible (macroscopic) or non-visible (microscopic) impact largely on whether the explanation is likely to be broadly urological or nephrological. Origins are most commonly simple or urological. Macroscopic bleeding is rare in renal disease, and urine colour is then usually more rather smoky than red except when there is very acute inflammation. The chief urological causes are neoplasia, infection, stones, and trauma. Some traditionally medical conditions may cause simple bleeding; examples include cystic kidney diseases, papillary necrosis and macro- or microvascular ischaemic lesions. The major concern to nephrologists is that even non-visible haematuria may be a pointer to inflammatory or destructive glomerular processes. The presence of casts or dysmorphic red cells is a pointer to glomerular disease; more important in clinical practice are the three other key markers of renal disease: proteinuria, renal impairment in the absence of urinary tract obstruction, and hypertension. In the general population, microscopic haematuria does associate with a long-term increased risk of end-stage renal failure, so after negative investigations, occasional long-term checks are indicated. The case for population screening for haematuria appears weak.

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