Relevant bibliographies by topics / Neonatal rat spinal cord

Academic literature on the topic 'Neonatal rat spinal cord'

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Published: 14 March 2023

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Journal articles on the topic "Neonatal rat spinal cord"

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Walker, Suellen M., B. David Westin, Ronald Deumens, Marjorie Grafe, and Tony L. Yaksh. "Effects of Intrathecal Ketamine in the Neonatal Rat." Anesthesiology 113, no. 1 (July 1, 2010): 147–59. http://dx.doi.org/10.1097/aln.0b013e3181dcd71c.

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Background Systemic ketamine can trigger apoptosis in the brain of rodents and primates during susceptible developmental periods. Clinically, spinally administered ketamine may improve the duration or quality of analgesia in children. Ketamine-induced spinal cord toxicity has been reported in adult animals but has not been systematically studied in early development. Methods In anesthetized rat pups, intrathecal ketamine was administered by lumbar percutaneous injection. Changes in mechanical withdrawal threshold evaluated dose-dependent antinociceptive and carrageenan-induced antihyperalgesic effects in rat pups at postnatal day (P) 3 and 21. After intrathecal injection of ketamine at P3, 7, or 21, spinal cords were examined for apoptosis (Fluoro-Jade C and activated caspase-3), histopathologic change, and glial responses (ionized calcium-binding adapter molecule 1 and glial fibrillary acid protein). After maximal doses of ketamine or saline at P3 or P21, sensory thresholds and gait analysis were evaluated at P35. Results Intrathecal injection of 3 mg/kg ketamine at P3 and 15 mg/kg at P21 reverses carrageenan-induced hyperalgesia. Baseline neuronal apoptosis in the spinal cord was greater at P3 than P7, predominantly in the dorsal horn. Intrathecal injection of 3-10 mg/kg ketamine in P3 pups (but not 15 mg/kg at P21) acutely increased apoptosis and microglial activation in the spinal cord and altered spinal function (reduced mechanical withdrawal threshold and altered static gait parameters) at P35. Conclusions Because acute pathology and long-term behavioral change occurred in the same dose range as antihyperalgesic effects, the therapeutic ratio of intrathecal ketamine is less than one in the neonatal rat. This measure facilitates comparison of the relative safety of spinally administered analgesic agents.
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Fok-Seang, J., and RH Miller. "Astrocyte precursors in neonatal rat spinal cord cultures." Journal of Neuroscience 12, no. 7 (July 1, 1992): 2751–64. http://dx.doi.org/10.1523/jneurosci.12-07-02751.1992.

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Miller, R. H., and V. Szigeti. "Clonal analysis of astrocyte diversity in neonatal rat spinal cord cultures." Development 113, no. 1 (September 1, 1991): 353–62. http://dx.doi.org/10.1242/dev.113.1.353.

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Within the mammalian CNS, astrocytes appear to be a heterogeneous class of cells. To assay the number of distinct types of astrocytes in the rat spinal cord, cell lineage and phenotypic analyses were carried out on cultures from newborn rat spinal cord and five distinct types of astrocytes were observed. Proliferating precursors for each class of astrocyte were isolated by low density culture and shown to give rise to 5 distinct and morphologically homogeneous clusters of GFAP + astrocytes. Immunocytochemical analysis with antibodies A2B5 and Ran-2, which identify different glial lineages in optic nerve cultures, demonstrated that many clusters included both A2B5+ and A2B5- cells. Similarly, many clusters also possessed a mixture of Ran-2+ and Ran-2-cells, suggesting that in spinal cord cultures, in contrast to optic nerve cultures, expression of these antigens is regulated by individual cells rather than by cell lineage. Single-cell cloning studies, revealed that the abundance and proliferative capacity of individual astrocyte precursors differed depending on the type of astrocyte. To assay the effects of a complex cellular environment on the composition of astrocyte clones, lineage analysis was performed in complete spinal cord cultures using a replication deficient retrovirus. Although similar morphologically homogeneous clones of cells to those seen with single-cell clones were observed, the proliferative capacity and relative abundance of the distinct astrocyte precursors differed from that seen in single-cell cloning studies. Together these observations suggest that in spinal cord, gliogenesis is considerably more complex than in the optic nerve and that cultures of newborn rat spinal cord contain multiple, distinct populations of astrocytes.
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Peng, Yuan Bo, Qing Dong Ling, M. A. Ruda, and Daniel R. Kenshalo. "Electrophysiological Changes in Adult Rat Dorsal Horn Neurons After Neonatal Peripheral Inflammation." Journal of Neurophysiology 90, no. 1 (July 2003): 73–80. http://dx.doi.org/10.1152/jn.01019.2002.

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Neonatal peripheral inflammation has been shown to produce profound anatomical changes in the dorsal horn of adult rats. In this study, we explored whether parallel physiological changes exist. Neonatal rats were injected with complete Freund's adjuvant (CFA) into the left hind paw. At 8–10 wk of age, single dorsal horn neurons were recorded in response to graded intensities of mechanical stimuli delivered to the receptive field. In addition, cord dorsum potentials, produced by electrical stimuli delivered to the left sciatic nerve at 2.5× threshold, were recorded bilaterally from L2 to S3. There were significant increases in background activity and responses to brush and pinch in neonatal rats that were treated with CFA, as compared with control rats. Further analysis showed similar significant changes when dorsal horn neurons were categorized into wide dynamic range (WDR), high-threshold (HT), and low-threshold (LT) groups. The receptive field was significantly larger in neonatally treated rats as compared with control rats. Additionally, there was a significant increase in the response to a 49°C heat stimulus in neonatally treated rats as compared with control rats. There was also a trend for the amplitudes of N1, N2, and P waves of the cord dorsum potential to increase and latencies to decrease in neonatally treated rats, but no significant differences were detected between different levels of the spinal cord (L2 to S3). These data further support the notion that anatomical and physiological plasticity changes occurred in the spinal cord following early neonatal CFA treatment.
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Sugaya, K., and W. C. De Groat. "Micturition reflexes in the in vitro neonatal rat brain stem-spinal cord-bladder preparation." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 266, no. 3 (March 1, 1994): R658—R667. http://dx.doi.org/10.1152/ajpregu.1994.266.3.r658.

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An in vitro neonatal (1-7 day) rat brain stem-spinal cord-bladder (BSB) preparation was used to examine the central control of micturition. Isovolumetric bladder contractions occurred spontaneously or were induced by electrical stimulation of the ventrolateral brain stem, spinal cord, bladder wall (ES-BW), or by perineal tactile stimulation (PS). Transection of the spinal cord at the L1 segment increased the amplitude of ES-BW- and PS-evoked contractions, and subsequent removal of the spinal cord further increased spontaneous and ES-BW-evoked contractions but abolished PS-evoked contractions. Hexamethonium (1 mM), a ganglionic blocking agent, mimicked the effect of cord extirpation. Tetrodotoxin (1 microM) blocked ES-BW- and PS-evoked contractions but enhanced spontaneous contractions. Bicuculline methiodide (10-50 microM), a gamma-aminobutyric acid A receptor antagonist, increased the amplitude of spontaneous, ES-BW- and PS-evoked contractions. These results indicate that PS-evoked contractions are mediated by spinal reflex pathways, whereas spontaneous and ES-BW-evoked contractions that are elicited by peripheral mechanisms are subject to a tonic inhibition dependent on an efferent outflow from the spinal cord. PS-evoked micturition is also subject to inhibitory modulation arising from sites rostral to the lumbosacral spinal cord. Although electrical stimulation of bulbospinal excitatory pathways can initiate bladder contractions in the neonatal rat, these pathways do not appear to have an important role in controlling micturition during the first postnatal week.
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Ma, Junxuan, Vaibhav Patil, Abhay Pandit, Leo R. Quinlan, David P. Finn, Sibylle Grad, Mauro Alini, and Marianna Peroglio. "In Vitro Model to Investigate Communication between Dorsal Root Ganglion and Spinal Cord Glia." International Journal of Molecular Sciences 22, no. 18 (September 8, 2021): 9725. http://dx.doi.org/10.3390/ijms22189725.

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Chronic discogenic back pain is associated with increased inflammatory cytokine levels that can influence the proximal peripheral nervous system, namely the dorsal root ganglion (DRG). However, transition to chronic pain is widely thought to involve glial activation in the spinal cord. In this study, an in vitro model was used to evaluate the communication between DRG and spinal cord glia. Primary neonatal rat DRG cells were treated with/without inflammatory cytokines (TNF-α, IL-1β, and IL-6). The conditioned media were collected at two time points (12 and 24 h) and applied to spinal cord mixed glial culture (MGC) for 24 h. Adult bovine DRG and spinal cord cell cultures were also tested, as an alternative large animal model, and results were compared with the neonatal rat findings. Compared with untreated DRG-conditioned medium, the second cytokine-treated DRG-conditioned medium (following medium change, thus containing solely DRG-derived molecules) elevated CD11b expression and calcium signal in neonatal rat microglia and enhanced Iba1 expression in adult bovine microglia. Cytokine treatment induced a DRG-mediated microgliosis. The described in vitro model allows the use of cells from large species and may represent an alternative to animal pain models (3R principles).
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Greer, J. J., Z. al-Zubaidy, and J. E. Carter. "Thyrotropin-releasing hormone stimulates perinatal rat respiration in vitro." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 271, no. 5 (November 1, 1996): R1160—R1164. http://dx.doi.org/10.1152/ajpregu.1996.271.5.r1160.

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In the present study, we test whether thyrotropin-releasing hormone (TRH) stimulates respiratory frequency in perinatal rats by acting at regions of the medulla responsible for respiratory rhythmogenesis, the pre-Botzinger complex. We also test whether TRH stimulates respiration in the fetal rat at a time shortly after the inception of respiratory rhythmogenesis [embryonic days (E) 17-18]. Two in vitro experimental models were utilized: the isolated brain stem-spinal cord preparation from fetal (E17-E18) and neonatal [postnatal days (P) 0-2] rats and the medullary slice preparation isolated from neonatal rats (P1-P2). Bath application of TRH caused a dose-dependent, reversible increase (maximum increase approximately 60%) in the frequency of respiratory rhythmic neural discharge generated by brain stem-spinal cord [half-maximal effective concentration (EC50) approximately 9 nM] and medullary slice (EC50 approximately 2.5 nM) neonatal rat preparations. Pressure injection of TRH unilaterally into the region of the pre-Botzinger complex of the neonatal medullary slice caused an approximately 28% increase in the frequency of respiratory discharge. Application of TRH to the medium bathing fetal rat brain stem-spinal cord preparations caused an approximately threefold increase in respiratory discharge frequency. We conclude that TRH stimulates respiratory discharge frequency from the time near inception of respiratory motor discharge and acts directly at the pre-Botzinger complex.
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Pulford, Bruce E., Andrea R. Mihajlov, Howard O. Nornes, and L. Ray Whalen. "Effects of Cultured Adrenal Chromaffin Cell Implants on Hindlimb Reflexes of the6-OHDA Lesioned Rat." Journal of Neural Transplantation and Plasticity 5, no. 2 (1994): 89–102. http://dx.doi.org/10.1155/np.1994.89.

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The effects of implantation of cultured adrenal medullary cells on the recovery of neurotransmitter specific reflex activity were studied in the rat spinal cord using electrophysiological testing methods. Cell suspensions of cultured neonatal adrenal medullary chromaffin (AM) cells (which produce catecholamines), or Schwann (Sc) cells (controls) were implanted into the lumbar region of the spinal cord 2 weeks after catecholamine (CA) denervation by intracisternal injection of 6-hydroxydopamine (6-OHDA). All cells were taken from 7 day neonates and cultured for 10 days in the presence of nerve growth factor (NGF). Three months after implantation, the extent of implant-associated recovery of reflex activity was determined by measuring electromyogram (EMG) activity and force associated with the long latency component of the hindlimb withdrawal reflex (which is CA modulated). After the electrophysiological testing, rats were anesthetized, and the spinal cords were rapidly removed and frozen. Spinal cords were sectioned longitudinally, and implanted cells were visualized using glyoxylic acid techniques. Labelled sections were examined to determine cell survival. Results indicate that 1) chromaffin cells survive for 3 months in the segments of the cord into which they have been implanted and 2) rats implanted with AM cells have significantly more forceful withdrawal reflexes than those that received Sc cells or received no implant after lesioning.
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Maclean, Jason N., Kristine C. Cowley, and Brian J. Schmidt. "NMDA Receptor-Mediated Oscillatory Activity in the Neonatal Rat Spinal Cord Is Serotonin Dependent." Journal of Neurophysiology 79, no. 5 (May 1, 1998): 2804–8. http://dx.doi.org/10.1152/jn.1998.79.5.2804.

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MacLean, Jason N., Kristine C. Cowley, and Brian J. Schmidt. NMDA receptor-mediated oscillatory activity in the neonatal rat spinal cord is serotonin dependent. J. Neurophysiol. 79: 2804–2808, 1998. The effect of serotonin (5-HT) receptor blockade on rhythmic network activity and on N-methyl-d-aspartate (NMDA) receptor-induced membrane voltage oscillations was examined using an in vitro neonatal rat spinal cord preparation. Pharmacologically induced rhythmic hindlimb activity, monitored via flexor and extensor electroneurograms or ventral root recordings, was abolished by 5-HT receptor antagonists. Intrinsic motoneuronal voltage oscillations, induced by NMDA in the presence of tetrodotoxin (TTX), either were abolished completely or transformed to long-lasting voltage shifts by 5-HT receptor antagonists. Conversely, 5-HT application facilitated the expression of NMDA-receptor–mediated rhythmic voltage oscillations. The results suggest that an interplay between 5-HT and NMDA receptor actions may be critical for the production of rhythmic motor behavior in the mammalian spinal cord, both at the network and single cell level.
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Kremer, E., and A. Lev-Tov. "GABA-Receptor–Independent Dorsal Root Afferents Depolarization in the Neonatal Rat Spinal Cord." Journal of Neurophysiology 79, no. 5 (May 1, 1998): 2581–92. http://dx.doi.org/10.1152/jn.1998.79.5.2581.

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Kremer, E. and A. Lev-Tov. GABA-receptor–independent dorsal root afferents depolarization in the neonatal rat spinal cord. J. Neurophysiol. 79: 2581–2592, 1998. Dorsal root afferent depolarization and antidromic firing were studied in isolated spinal cords of neonatal rats. Spontaneous firing accompanied by occasional bursts could be recorded from most dorsal roots in the majority of the cords. The afferent bursts were enhanced after elevation of the extracellular potassium concentration ([K+]e) by 1–2 mM. More substantial afferent bursts were produced when the cords were isolated with intact brain stems. Rhythmic afferent bursts could be recorded from dorsal roots in some of the cords during motor rhythm induced by bath-applied serotonin and N-methyl-d-aspartate (NMDA). Bilaterally synchronous afferent bursts were produced in pairs of dorsal roots after replacing the NaCl in the perfusate with sodium-2-hydroxyethansulfonate or after application of the γ-aminobutyric acid-A (GABAA) receptor antagonist bicuculline with or without serotonin (5-HT) and NMDA. Antidromic afferent bursts also could be elicited under these conditions by stimulation of adjacent dorsal roots, ventrolateral funiculus axons, or ventral white commissural (VWC) fibers. The antidromic bursts were superimposed on prolonged dorsal root potentials (DRPs) and accompanied by a prolonged increase in intraspinal afferent excitability. Surgical manipulations of the cord revealed that afferent firing in the presence of bicuculline persisted in the hemicords after hemisection and still was observed after removal of their ventral horns. Cutting the VWC throughout its length did not perturb the bilateral synchronicity of the discharge. These findings suggest that the activity of dorsal horn neurons is sufficient to produce the discharge and that the bilateral synchronicity can be maintained by cross connectivity that is relayed from side to side dorsal to the VWC. Antagonists of GABAB, 5-HT2/5-HT1C, or glutamate metabotropic group II and III receptors could not abolish afferent depolarization in the presence of bicuculline. Depolarization comparable in amplitude to DRPs, could be produced in tetrodotoxin-treated cords by elevation of [K+]e to the levels reported to develop in the neonatal rat spinal cord in response to dorsal root stimulation. A mechanism involving potassium transients produced by neuronal activity therefore is suggested to be the major cause of the GABA-independent afferent depolarization reported in our study. Possible implications of potassium transients in the developing and the adult mammalian spinal cord are discussed.
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Dissertations / Theses on the topic "Neonatal rat spinal cord"

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Cina, Cima. "Distribution of locomotor-labelled neurons in the neonatal rat thoracolumbar spinal cord." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp04/mq23253.pdf.

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Jones, Philip Leslie St John. "Structure-activity studies of novel compounds acting at metabotropic excitatory amino acid receptors in neonatal rat spinal motoneurons." Thesis, University of Bristol, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.385687.

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Richard, Levine, Richard Vaillancourt, and Ralph Fregosi. "Evaluation of the Brainstem Spinal Cord Preparation in the Neonatal Rat as a Model for Prenatal Nicotine Exposure." The University of Arizona, 2012. http://hdl.handle.net/10150/614504.

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Class of 2012 Abstract
Specific Aims: The goal of this project was to evaluate the use of a preparation of the brainstem and spinal cord of neonatal rats that has been widely used for observing and quantifying central nervous activity, as well as the response to pharmacological manipulation. To achieve this, we specifically aimed to remove the intact brainstem and spinal cord of newborn rats, and develop a preparation that would maintain physiological function and allow for recording of electrical activity. Methods: Multiple dissections were performed on neonatal rats. Conditions during the dissections were controlled to maintain physiological function. Once removed, the intact brainstem and spinal cord was placed in a preparation that allowed for manipulation and access to nerve rootlets. Finally, glass suction electrodes were used to record electrical activity directly from the nerve rootlets. Once recorded, the data were stored on a hard drive for further analysis. Main Results: We were successful in isolating the intact brainstem and spinal cord in neonatal rats while maintaining physiological conditions and nervous activity. The preparation allowed for easy access to nerve roots as well as customization for different experiments. We were also successful in recording nerve activity in the preparation and collection of data for use in future experiments Conclusions: We conclude that the brainstem spinal cord preparation described in this study is a valuable tool that allows for recording and analysis of nerve activity, and specifically for measurement of respiratory motor output. This is a preparation that can be used in a variety of experiments that attempt to observe or quantify the activity of central nerve cells and allows for pharmacological interventions that could be applied in various experiments.
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Hase, Takao. "Locomotor performance of the rat after neonatal repairing of spinal cord injuries : Quantitative assessment and electromyographic study." Kyoto University, 2002. http://hdl.handle.net/2433/149710.

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Levine, Richard. "Evaluation of the Brainstem Spinal Cord Preparation in the Neonatal Rat as a Model for Prenatal Nicotine Exposure." The University of Arizona, 2012. http://hdl.handle.net/10150/623649.

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Class of 2012 Abstract
Specific Aims: The goal of this project was to evaluate the use of a preparation of the brainstem and spinal cord of neonatal rats that has been widely used for observing and quantifying central nervous activity, as well as the response to pharmacological manipulation. To achieve this, we specifically aimed to remove the intact brainstem and spinal cord of newborn rats, and develop a preparation that would maintain physiological function and allow for recording of electrical activity. Methods: Multiple dissections were performed on neonatal rats. Conditions during the dissections were controlled to maintain physiological function. Once removed, the intact brainstem and spinal cord was placed in a preparation that allowed for manipulation and access to nerve rootlets. Finally, glass suction electrodes were used to record electrical activity directly from the nerve rootlets. Once recorded, the data were stored on a hard drive for further analysis. Main Results: We were successful in isolating the intact brainstem and spinal cord in neonatal rats while maintaining physiological conditions and nervous activity. The preparation allowed for easy access to nerve roots as well as customization for different experiments. We were also successful in recording nerve activity in the preparation and collection of data for use in future experiments Conclusions: We conclude that the brainstem spinal cord preparation described in this study is a valuable tool that allows for recording and analysis of nerve activity, and specifically for measurement of respiratory motor output. This is a preparation that can be used in a variety of experiments that attempt to observe or quantify the activity of central nerve cells and allows for pharmacological interventions that could be applied in various experiments.
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Miller, Jacqueline Chantal. "Pharmacological characterisation and the immunohistochemical localisation of glutamate receptor subtypes in the lumbar region of the neonatal rat spinal cord." Thesis, University of Bristol, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.247861.

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More, Julia Carrie-Ann. "Pharmacological characterisation of kainate receptors in the neonatal rat spinal cord using novel antagonists based on the natural product willardine." Thesis, University of Bristol, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.396711.

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Plenderleith, M. B. "The effects of neonatal capsaicin treatment on the functional properties of sensory neurones in the dorsal horn of the rat spinal cord." Thesis, University of Bristol, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.356742.

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Altas, Melanie. "Spinal cord transplants in a rat model of spinal cord injury." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape7/PQDD_0021/MQ49305.pdf.

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Krenz, Natalie. "Plasticity in the rat spinal cord following spinal cord transection, contribution to autonomic dysreflexia." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape7/PQDD_0008/NQ40268.pdf.

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Books on the topic "Neonatal rat spinal cord"

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Pyner, Susan. Organisation of sympathetic preganglionic neurones in the upper thoracic spinal cord in the adult and neonate rat. Birmingham: University of Birmingham, 1992.

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Oudega, Martin, Egbert A. J. F. Lakke, Enrico Marani, and Raph T. W. M. Thomeer. Development of the Rat Spinal Cord: Immuno- and Enzyme Histochemical Approaches. Berlin, Heidelberg: Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-78474-3.

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Lewis, David Ian. Chemical transmission in the sympathetic nuclei of the rat spinal cord. Birmingham: University of Birmingham, 1988.

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1956-, Oudega Martin, ed. Development of the rat spinal cord: Immuno- and enzyme histochemical approaches. Berlin: Springer-Verlag, 1993.

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Spanswick, David. Chemical neurotransmission in the rat hippocampus and spinal cord in vitro. Birmingham: University of Birmingham, 1991.

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Park, Eugene. Characterization of changes in ampa receptor subunit expression in spinal cord white matter following acute compression spinal cord injury in the rat. Ottawa: National Library of Canada, 2002.

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Silva, Gabriel A. Metabotropic glutamate receptor expression in rat spinal cord astrocytes in vitro and in situ. Ottawa: National Library of Canada = Bibliothèque nationale du Canada, 1999.

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The projections to the spinal cord of the rat during development: A time-table of descent. Berlin: Springer, 1997.

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Lakke, E. A. J. F. The Projections to the Spinal Cord of the Rat During Development: A Timetable of Descent. Berlin, Heidelberg: Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-642-60601-4.

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Enescu, Cristina. Methods of enhancing mechanical properties of hydrogel tubes used as nerve guidance channels in rat spinal cord injury. Ottawa: National Library of Canada, 2003.

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Book chapters on the topic "Neonatal rat spinal cord"

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Seybold, Virginia S., and Lia G. Abrahams. "Primary Cultures of Neonatal Rat Spinal Cord." In Pain Research, 203–13. Totowa, NJ: Humana Press, 2004. http://dx.doi.org/10.1385/1-59259-770-x:067.

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Cazalets, Jean-René. "Organization of the Spinal Locomotor Network in Neonatal Rat." In Neurobiology of Spinal Cord Injury, 89–111. Totowa, NJ: Humana Press, 2000. http://dx.doi.org/10.1007/978-1-59259-200-5_4.

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Cazalets, J. R. "Dual Control of Central Pattern Generators: Neonatal Rat Spinal Cord In Vitro." In Neural Control of Movement, 187–94. Boston, MA: Springer US, 1995. http://dx.doi.org/10.1007/978-1-4615-1985-0_24.

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Arata, Akiko, and Morimitsu Fujii. "Catecholaminergic Modulation of the Respiratory Rhythm Generator in the Isolated Brainstem—Spinal Cord Preparation from Neonatal Rat." In Integration in Respiratory Control, 83–87. New York, NY: Springer New York, 2008. http://dx.doi.org/10.1007/978-0-387-73693-8_14.

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Okada, Y., S. Yokota, Y. Shinozaki, R. Aoyama, Y. Yasui, M. Ishiguro, and Y. Oku. "Anatomical Architecture and Responses to Acidosis of a Novel Respiratory Neuron Group in the High Cervical Spinal Cord (HCRG) of the Neonatal Rat." In Advances in Experimental Medicine and Biology, 387–94. Dordrecht: Springer Netherlands, 2009. http://dx.doi.org/10.1007/978-90-481-2259-2_44.

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Rajaram, Veena, and Veena Rajaram. "Brain and Spinal Cord." In Color Atlas of Fetal and Neonatal Histology, 285–320. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4614-0019-6_27.

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Sakuraba, Shigeki, Yuki Hosokawa, Yuki Kaku, Junzo Takeda, and Shun-ichi Kuwana. "Laudanosine has No Effects on Respiratory Activity but Induces Non-Respiratory Excitement Activity in Isolated Brainstem-Spinal Cord Preparation of Neonatal Rats." In Advances in Experimental Medicine and Biology, 177–80. New York, NY: Springer New York, 2009. http://dx.doi.org/10.1007/978-1-4419-5692-7_35.

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Claus, D., W. Weitbrecht, and B. Neundörfer. "Pentobarbital: The Influence on Somatosensory Conduction in the Rat." In Spinal Cord Monitoring, 90–94. Berlin, Heidelberg: Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70687-5_11.

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Shannon, Patrick. "Brain and Spinal Cord." In Color Atlas of Human Fetal and Neonatal Histology, 291–310. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-11425-1_29.

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Palladini, G., and B. Caronti. "Regeneration in the axotomized cord: influence of cyclosporine A and neonatal immune desensitization in mammals." In Spinal Cord Monitoring, 157–80. Vienna: Springer Vienna, 1998. http://dx.doi.org/10.1007/978-3-7091-6464-8_7.

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Conference papers on the topic "Neonatal rat spinal cord"

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Brieu, N., E. Beaumont, and F. Lesage. "Intrinsic Optical Imaging Of The Rat Lumbar Spinal Cord." In Biomedical Optics. Washington, D.C.: OSA, 2008. http://dx.doi.org/10.1364/biomed.2008.bmd7.

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Ochoa-Gutierrez, Victor J., Pavan C. Konda, Sara Motaghian, Julien Reboud, Jonathan M. Cooper, and Andrew R. Harvey. "Multi-spectral vascular oximetry of rat dorsal spinal cord." In Optics and Biophotonics in Low-Resource Settings VI, edited by David Levitz and Aydogan Ozcan. SPIE, 2020. http://dx.doi.org/10.1117/12.2558281.

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Fiford, Rodney J., and Lynne E. Bilston. "Strain Distribution and Relaxation Behaviour of Rat Spinal Cord." In ASME 1998 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 1998. http://dx.doi.org/10.1115/imece1998-0123.

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Abstract Uniaxial testing of rat spinal cord was performed to investigate the viscoelastic behaviour of spinal cord in tension. Stress relaxation and stress-strain data is presented, and strain distribution within the material is investigated using video analysis and surface markings.
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Olby, Natasha J., and W. F. Blakemore. "Photochemically induced spinal ischaemia: a model of spinal cord trauma in the rat." In Photonics West '95, edited by R. Rox Anderson, Graham M. Watson, Rudolf W. Steiner, Douglas E. Johnson, Stanley M. Shapshay, Michail M. Pankratov, George S. Abela, et al. SPIE, 1995. http://dx.doi.org/10.1117/12.209061.

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Fung, E. K., D. Weinzimmer, S. Strittmatter, Yiyun Huang, and R. E. Carson. "Segmentation of rat spinal cord in PET using spatiotemporal information." In 2010 IEEE Nuclear Science Symposium and Medical Imaging Conference (2010 NSS/MIC). IEEE, 2010. http://dx.doi.org/10.1109/nssmic.2010.5874483.

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Chen, Xiaoming, Garrett W. Astary, Thomas H. Mareci, and Malisa Sarntinoranont. "In Vivo Contrast-Enhanced MR Imaging for Direct Infusion Into Rat Peripheral Nerve." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192919.

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Direct infusion of therapeutic agents into the spinal cord provides a promising way to treat traumatic injury and intrinsic diseases of the spinal cord, which may cause paralysis and other neurological deficits. Direct infusion into the spinal cord involves complex invasive surgery since the spinal cord is well protected by the vertebral bone. Instead, infusion directly into peripheral nerves is of interest since it provides a remote delivery site to the spinal cord, requiring less invasive surgery and reducing the risk of spinal cord injury during surgery. It may also allow targeting of specific neurons at nerve root entry. Previous studies have shown [1, 2] that transport in peripheral nerves is anisotropic with a preferred direction parallel to the fiber tracts. A large-scale longitudinal spread of molecular agents may be obtained and spread of molecular agents into the spinal cord may be possible.
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Raczkowska, Marlena N., Wendy Y. X. Peh, Yuni Teh, Monzurul Alam, Shih-Cheng Yen, and Nitish V. Thakor. "Closed-Loop Bladder Neuromodulation Therapy in Spinal Cord Injury Rat Model." In 2019 9th International IEEE/EMBS Conference on Neural Engineering (NER). IEEE, 2019. http://dx.doi.org/10.1109/ner.2019.8717001.

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Dejneka, A., V. Zablotskii, O. Lunov, and S. Kubinova. "Magnetically targeted stem cell delivery in spinal cord injury: Rat model." In 2017 IEEE International Magnetics Conference (INTERMAG). IEEE, 2017. http://dx.doi.org/10.1109/intmag.2017.8007875.

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Prasad, A., and M. Sahin. "Chronic recordings from the rat spinal cord descending tracts with microwires." In 2011 33rd Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2011. http://dx.doi.org/10.1109/iembs.2011.6090821.

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Chen, Xiaoming, Garrett W. Astary, Thomas H. Mareci, and Malisa Sarntinoranont. "In Vivo Characterization of Transport Anisotropy in Rat Spinal Cord Using Diffusion Tensor Imaging." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192898.

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Biotransport in nervous tissues is complicated by the existence of neural fibers. These axonal fibers result in inhomogeneous and anisotropic extracellular transport, which complicates the prediction of local drug delivery such as convection-enhanced delivery [1]. Previous studies by our group [4] have shown that by using diffusion tensor imaging (DTI) [2, 3], anisotropic transport in rat spinal cord can be modeled using computational models, and consequently extracellular flows which influence drug transport can be well predicted. In previous studies, DTI-based models used data from an excised and fixed rat spinal cord. In the current study, we extend our DTI study to in vivo measures, and report the in vivo characterization of transport anisotropy in rat spinal cord. The MR imaging method is presented and the DTI data is discussed.
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Reports on the topic "Neonatal rat spinal cord"

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Benton, Bernard J., John H. McDonough, Thomas A. Koviak, and Tsung-Ming A. Shih. Time-Course Effects of GA, GB, GD, GF and VX on Spinal Cord Cholinesterase and Acetylcholine Levels in Six Discrete Areas of the Rat Brain. Fort Belvoir, VA: Defense Technical Information Center, January 2001. http://dx.doi.org/10.21236/ada396059.

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