Academic literature on the topic 'Nasal olfactory tissues'
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Journal articles on the topic "Nasal olfactory tissues"
Muluk, Nuray Bayar. "Olfactory functions in Behçet’s disease: A review." Romanian Journal of Rhinology 8, no. 32 (October 1, 2018): 213–17. http://dx.doi.org/10.2478/rjr-2018-0023.
Full textOlson, M. J., J. L. Martin, A. C. LaRosa, A. N. Brady, and L. R. Pohl. "Immunohistochemical localization of carboxylesterase in the nasal mucosa of rats." Journal of Histochemistry & Cytochemistry 41, no. 2 (February 1993): 307–11. http://dx.doi.org/10.1177/41.2.8419465.
Full textJung, Su Young, Dong Choon Park, Sung Su Kim, and Seung Geun Yeo. "Expression, Distribution and Role of Aquaporins in Various Rhinologic Conditions." International Journal of Molecular Sciences 21, no. 16 (August 14, 2020): 5853. http://dx.doi.org/10.3390/ijms21165853.
Full textGrubb, B. R., T. D. Rogers, R. C. Boucher, and L. E. Ostrowski. "Ion transport across CF and normal murine olfactory and ciliated epithelium." American Journal of Physiology-Cell Physiology 296, no. 6 (June 2009): C1301—C1309. http://dx.doi.org/10.1152/ajpcell.00578.2008.
Full textKincaid, Anthony E., and Jason C. Bartz. "The Nasal Cavity Is a Route for Prion Infection in Hamsters." Journal of Virology 81, no. 9 (February 14, 2007): 4482–91. http://dx.doi.org/10.1128/jvi.02649-06.
Full textPahrudin Arrozi, Aslina, Daijiro Yanagisawa, Tomoko Kato, Hiroyasu Akatsu, Yoshio Hashizume, Daita Kaneda, and Ikuo Tooyama. "Nasal Extracts from Patients with Alzheimer’s Disease Induce Tau Aggregates in a Cellular Model of Tau Propagation." Journal of Alzheimer's Disease Reports 5, no. 1 (April 6, 2021): 263–74. http://dx.doi.org/10.3233/adr-210298.
Full textDeJoia, Crista, Brian Moreaux, Kimberly O'Connell, and Richard A. Bessen. "Prion Infection of Oral and Nasal Mucosa." Journal of Virology 80, no. 9 (May 1, 2006): 4546–56. http://dx.doi.org/10.1128/jvi.80.9.4546-4556.2006.
Full textTAMURA, Hiro-omi, Yuki HARADA, Atsushi MIYAWAKI, Katsuhiko MIKOSHIBA, and Michio MATSUI. "Molecular cloning and expression of a cDNA encoding an olfactory-specific mouse phenol sulphotransferase." Biochemical Journal 331, no. 3 (May 1, 1998): 953–58. http://dx.doi.org/10.1042/bj3310953.
Full textCalderon-Garciduenas, Lilian, Robert R. Maronpot, Ricardo Torres-Jardon, Carlos Henriquez-Roldan, Robert Schoonhoven, Hilda Acuna-Ayala, Anna Villarreal-Calderon, et al. "DNA Damage in Nasal and Brain Tissues of Canines Exposed to Air Pollutants Is Associated with Evidence of Chronic Brain Inflammation and Neurodegeneration." Toxicologic Pathology 31, no. 5 (August 2003): 524–38. http://dx.doi.org/10.1080/01926230390226645.
Full textCarboni, Anthony A., Kay J. Cullen, and William G. Lavelle. "The Effects of Zinc on the Olfactory Neuroepithelium and Olfactory Bulbs of the Sprague-Dawley Rat after oral Administration of Zinc-Gluconate Trihydrate." American Journal of Rhinology 20, no. 3 (May 2006): 262–68. http://dx.doi.org/10.2500/ajr.2006.20.2854.
Full textDissertations / Theses on the topic "Nasal olfactory tissues"
Lu, Jike Faculty of Medicine UNSW. "Transplantation of nasal olfactory tissues into transected spinal cord of adult rats." Awarded by:University of New South Wales, 2000. http://handle.unsw.edu.au/1959.4/17798.
Full textFranzén, Anna. "Tissue-Selective Activation and Toxicity of Substituted Dichlorobenzenes : Studies on the Mechanism of Cell Death in the Olfactory Mucosa." Doctoral thesis, Uppsala University, Department of Pharmaceutical Biosciences, 2005. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-6161.
Full textThe nasal passages are constantly exposed to both air- and bloodborne foreign compounds. In particular, the olfactory mucosa is demonstrated to be susceptible to a variety of drugs and chemicals. In this thesis, mechanisms involved in tissue-selective toxicity in the olfactory mucosa of rodents have been investigated using the olfactory toxicant 2,6-dichlorophenyl methylsulphone (2,6-diClPh-MeSO2) as a model compound. Comparative studies were performed with the non-toxic 2,5-dichlorophenyl methylsulphone (2,5-diClPh-MeSO2) and the reasons for the strikingly different toxicity were investigated.
A strong bioactivation and protein adduction of 2,6-diClPh-MeSO2 in olfactory microsomes and S9-fractions of rodents was demonstrated. In contrast, no significant metabolic activation of 2,5-diClPh-MeSO2 was observed and the bioactivation in the liver for both chlorinated isomers was negligible. In vitro studies with recombinant yeast cell microsomes expressing mouse cytochrome P450 2A5 (CYP2A5) demonstrated a metabolic activation of 2,6-diClPh-MeSO2. The 2,6-diClPh-MeSO2-induced lesions and CYP2A5 expression preferentially occurred in Bowman’s glands and sustentacular cells of the olfactory mucosa. A significant depletion of glutathione (GSH) in the olfactory mucosa was demonstrated in vivo, while no changes were observed in the liver. There was a rapid induction of the endoplasmic reticulum (ER)-specific chaperone Grp78, activation of the ER-specific caspase-12 and the downstream caspase-3 in the Bowman’s glands. Electron microscopy revealed swelling of ER and mitochondria and a lost integrity of the Bowman’s glands.
Based on these results, the proposed mechanism for 2,6-diClPh-MeSO2-induced toxicity in the olfactory mucosa is bioactivation by CYP2A5 into a reactive intermediate causing protein adduction and GSH-depletion. This is initiating a sequence of downstream events of ER-stress, changes in ion homeostasis, ultrastructural organelle disruption and apoptotic signalling. In spite of the initial apoptotic signals, the terminal phase of apoptosis seemed to be blocked and necrotic features occurred. The predominant expression of CYP2A5 in the olfactory mucosa is proposed to play a key role for the tissue- and cell-specific toxicity induced by 2,6-diClPh-MeSO2.
Lu, Jike. "Transplantation of nasal olfactory tissues into transected spinal cord of adult rats /." 2000. http://www.library.unsw.edu.au/~thesis/adt-NUN/public/adt-NUN20010622.112434/index.html.
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