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Books on the topic 'Myositi'

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Author: Grafiati

Published: 9 March 2023

Last updated: 10 March 2023

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1

Aggarwal, Rohit, and Chester V. Oddis, eds. Managing Myositis. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-15820-0.

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2

J, Staiger C., ed. Actin: A dynamic framework for multiple plant cell functions. Dordrecht: Kluwer Academic Publishers, 2000.

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3

Coluccio, Lynne M., ed. Myosins. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-38062-5.

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4

Coluccio, Lynne M. Myosins. Dordrecht: Springer Netherlands, 2008. http://dx.doi.org/10.1007/978-1-4020-6519-4.

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5

Melica, Elisa. Myosotis. Nardò (LE), i.e. Lecce: Besa Editrice, 2000.

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6

Myosins. 2nd ed. Oxford: Oxford University Press, 1999.

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7

Betina, Begong-Bodoli. Myosotis: Roman. Saint-Louis, Sénégal: Xamal, 1997.

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8

Hương, Dương Thu. Myosotis: Roman. Arles: Picquier, 2003.

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9

Askanas, Valerie, and W. King Engel. Muscle aging: Inclusion-body myositis and myopathies. Oxford: Wiley-Blackwell, 2012.

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10

1933-, Sugi Haruo, and Pollack Gerald H, eds. Mechanism of myofilament sliding in muscle contraction. New York: Plenum Press, 1993.

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11

Pénide, Dominique. La pension Myosotis. Castelnau-le-Lez: Climats, 2000.

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12

Kim, Kyŏng. Myossi Maekchujŏm. Sŏul: Songsong Ch'aekpang, 2020.

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13

Askanas, Valerie, and W. King Engel, eds. Muscle Aging, Inclusion-Body Myositis and Myopathies. Oxford, UK: Wiley-Blackwell, 2012. http://dx.doi.org/10.1002/9781444398311.

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14

The inflammatory myopathies. Dordrecht: Humana Press, 2009.

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15

C, Dalakas Marinos, ed. Polymyositis and dermatomyositis. Boston: Butterworths, 1988.

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16

Obrestad, Tor. Mimosa, myosotis, rosmarin: Dikt. [Oslo]: Cappelen, 1994.

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17

Boukhrissa, Naı̈la. Myosotis: Odeur du délice. Alger: ENAG éditions, 2019.

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18

Parker, James N., and Philip M. Parker. The official patient's sourcebook on inclusion body myositis. Edited by Icon Group International Inc. San Diego, Calif: Icon Health Publications, 2002.

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19

Sellers, James R. Motor proteins 2: myosin. London: Academic Press, 1995.

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20

L, Mastaglia Frank, ed. Inflammatory diseases of muscle. Oxford: Blackwell Scientific, 1988.

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21

Schultheiss, H. P., and Michel Noutsias. Inflammatory cardiomyopathy (DCMi): Pathogenesis and therapy. Basel: Birkhäuser, 2010.

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22

M, Parker Philip, and Parker James N, eds. Official patient's sourcebook on polymyositis: A revised and updated directory for the internet age. San Diego CA: Icon Group Int'l., Inc., 2004.

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23

Crest, Emmanuel. Myosotis, ou, Le Noël de l'herboriste. [Paris]: Griot, 1991.

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24

A, Cross R., and Kendrick-Jones J, eds. Motor proteins: A volume based on the EMBO Workshop, Cambridge, September 1990. Cambridge [England]: Company of Biologists, 1991.

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25

Myosotis ou Le nuancier de Gérard de Nerval. Nantes: Ed. du Petit véhicule, 2006.

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26

Odartchenko, Cécile. Myosotis, ou, Le nuancier de Gérard de Nerval: Essai. Nantes: Petit véhicule, 2006.

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27

Syrový, Ivo. Kontraktilní bílkoviny a funkční požadavky svalu. Praha: Academia, 1985.

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28

I promise I'll pay attention: Living with a seemingly invisible, progressively debilitating disease. Mustang, Oklahoma: Tate Publishing and Enterprises, LLC, 2016.

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29

Thomas, D. D. Molecular Interactions of Actin: Actin-Myosin Interaction and Actin-Based Regulation. Berlin, Heidelberg: Springer Berlin Heidelberg, 2002.

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30

Cooper, Robert G., and Hector Chinoy. Myositis. Oxford University Press, 2018.

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31

Boros, Christina, K. Nistala, and L. R. Wedderburn. Juvenile myositis. Edited by Hector Chinoy and Robert Cooper. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198754121.003.0010.

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Idiopathic inflammatory myopathies (IIM) of childhood are rare serious disorders, of which the most common is juvenile dermatomyositis (JDM). It is recognized increasingly that, even within JDM, there is significant heterogeneity. Recently defined biomarkers, such as the myositis-specific autoantibodies (MSA) have been valuable to define juvenile IIM subgroups and may provide novel methods of classification. Although randomized controlled trials in JDM are challenging, an increasing consensus-based body of evidence is being used to drive standardization of treatment and care, with new drugs for severe cases being increasingly tested. Significant differences between adults and children with dermatomyositis (DM) exist, and are reviewed in this chapter. These differences emphasize the importance of specialists in the area of transition of young people who have had JDM to receive their care from a team of health professionals with comprehensive knowledge of JDM, as well as transitional care issues.

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32

(Editor), C. J. Staiger, F. Baluska (Editor), D. Volkmann (Editor), and P. Barlow (Editor), eds. Actin: A Dynamic Framework for Multiple Plant Cell Functions (Developments in Plant and Soil Sciences). Springer, 2000.

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33

Machado, Pedro M. Inclusion body myositis. Edited by Hector Chinoy and Robert Cooper. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198754121.003.0011.

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Sporadic inclusion body myositis (IBM) is an acquired muscle disorder associated with ageing, for which there is no effective treatment. It is characterized by a typical early clinical phenotype with (often asymmetric) weakness of the knee extensors and finger flexors, potential involvement of pharyngeal and upper-oesophageal muscles (which may contribute to malnutrition and aspiration), and progressive and slow deterioration, which may lead to severe disability and loss of quality of life. Muscle biopsy shows chronic myopathic features, lymphocytic infiltration with invasion of non-necrotic fibres, rimmed vacuoles, mitochondrial changes, and pathological accumulation of proteins in the muscle tissue. It remains uncertain whether IBM is primarily an immune-mediated inflammatory myopathy or a degenerative myopathy with an associated inflammatory component. This chapter will describe the clinical features, natural history, investigations, current pathogenic concepts, outcome measures, and therapeutic approaches in IBM. Despite recent clues, in many respects IBM remains an unsolved mystery.

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34

Lilleker, James B. Advances in Myositis. Scientific Research Publishing, 2022.

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35

Liégard, Lisa. Myosotis. Lulu Press, Inc., 2008.

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36

Huong, Duong Thu. Myosotis. Philippe Picquier, 2001.

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37

Coluccio, Lynne M. Myosins. Springer, 2008.

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38

Diana, Enzo. Myosotis. Independently Published, 2022.

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39

Ashour, Noor. Myosotis. Independently Published, 2021.

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40

Valerie, Askanas, Serratrice Georges, and Engel W. King, eds. Inclusion-body myositis and myopathies. Cambridge: Cambridge University Press, 1997.

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41

Chinoy, Hector, and Robert G. Cooper, eds. Myositis (Oxford Rheumatology Library). Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198754121.001.0001.

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Myositis, or the idiopathic inflammatory myopathies (IMM) are a group of rare autoimmune diseases of considerable health significance. Features include muscle weakness, raised skeletal muscle enzymes, and characteristic histopathological changes. IIM can be split into polymyositis (PM), dermatomyositis (DM), and inclusion body myositis (IBM). If diagnosed promptly PM and DM can be treated with immunosuppression, but IBM currently cannot be treated. Summarizing the current understanding of the epidemiology, genetic and environmental risk factors, and clinical features, this handbook gives practical strategies for laboratory investigations and treatment paradigms. Future strategies and ongoing trials are also discussed.

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42

Kagen, Lawrence J. Myositis & Myopathies: Raynauld's Phenomenon. Rapid Science Publishers, 1994.

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43

Broadbent, David. Myosin: Biosynthesis, Classes and Function. Nova Science Publishers, Incorporated, 2018.

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44

Schmidt, Jens. Extramuscular complications occurring in myositis. Edited by Hector Chinoy and Robert Cooper. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198754121.003.0004.

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Extra-muscular complications affecting patients suffering from the idiopathic inflammatory myopathies (IIM) are common, and appear in recognizable patterns affecting the skin, lungs, joints, oesophagus or heart, although these complications rarely all occur simultaneously. During the initial presentation of symptoms, involvement of organs other than muscle can aid the confirmation of the correct IIM subtype. Extra-muscular manifestations can be severe and life-threatening, e.g. with respiratory or cardiac involvement. Escalations of immunosuppression and other treatment modalities will likely be required in such cases since standard immunosuppression usually is not sufficient for an effective treatment of e.g. interstitial lung disease. IIM patients should therefore be regularly checked for extra-muscular manifestations, and management altered as appropriate.

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45

Oddis, Chester V., and Rohit Aggarwal. Managing Myositis: A Practical Guide. Springer International Publishing AG, 2020.

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46

Aggarwal, Rohit, and Chester Oddis. Managing Myositis: A Practical Guide. Springer International Publishing AG, 2019.

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47

Kilpatrick, James R. Coping with a Myositis Disease. Kilpatrick Publishing Company, 2000.

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48

Adrian, Judith Gwinn. Nancer the Dancer: Myositis and Me. HenschelHAUS Publishing, Inc., 2020.

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49

Adrian, Judith Gwinn. Nancer the Dancer: Myositis and Me. HenschelHAUS Publishing, Inc., 2020.

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50

Myositis: Facts, Feelings and Future Hopes. Thoughtful Publications, 2003.

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