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Books on the topic 'Myogenic'

To see the other types of publications on this topic, follow the link: Myogenic.

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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1

Murofushi, Toshihisa, and Kimitaka Kaga. Vestibular Evoked Myogenic Potential. Tokyo: Springer Japan, 2009. http://dx.doi.org/10.1007/978-4-431-85908-6.

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2

Barlas, Panagiotis. An investigation of the effects of acupuncture upon experimentally-induced myogenic pain. [s.l: The Author], 1997.

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3

Yamaguchi, Terence P. The regulation of myogenic determination and differentiation by growth factors and oncogenes. Ottawa: National Library of Canada, 1990.

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4

Tai, Helen H. Inhibition of myogenic differentiation in Harvey ras-transfected BCb3sH1 muscle cells. Ottawa: National Library of Canada, 1990.

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5

Miller, Mathew Gordon. Integrin-Linked Kinase 1 (ILK1) is necessary for myogenic differentiation in rat L6 myoblasts. Ottawa: National Library of Canada, 2000.

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6

Osepchook, Claire C. Expression patterns of muscle growth factors and myogenic regulatory factors in response to undernutrition in ovine skeletal muscle. Ottawa: National Library of Canada, 2002.

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7

Myers, Bre Lynn. Vestibular learning manual. San Diego: Plural Pub., 2011.

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8

Vestibular Evoked Myogenic Potential. Springer, 2008.

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9

Kaga, Kimitaka, and Toshihisa Murofushi. Vestibular Evoked Myogenic Potential: Its Basics and Clinical Applications. Springer, 2010.

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10

Endlich, Karlhans, and Rodger Loutzenhiser. Tubuloglomerular feedback, renal autoregulation, and renal protection. Edited by Neil Turner. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0209.

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Vascular tone of glomerular blood vessels is controlled dynamically in response to a number of stimuli of which tubuloglomerular feedback and blood flow (and glomerular filtration rate) autoregulation are the most prominent. Both tubuloglomerular feedback- and myogenic-mediated pre-glomerular vasoconstriction are important in the response to reduced pressure. The renal myogenic mechanism, which has the potential to adjust steady-state tone in response to the oscillating systolic pressure signal, additionally plays an essential role in protecting the kidney from the damaging effects of hypertension.

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11

Gugino, Laverne D., Rafael Romero, Marcella Rameriz, Marc E. Richardson, and Linda S. Aglio. TMS in the perioperative period. Edited by Charles M. Epstein, Eric M. Wassermann, and Ulf Ziemann. Oxford University Press, 2012. http://dx.doi.org/10.1093/oxfordhb/9780198568926.013.0020.

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Two stimulation approaches developed for selectively exciting descending motor pathways are, transcranial electrical (TES) and transcranial magnetic (TMS) stimulation. This article highlights the comparison between electrical and magnetic transcranial stimulation. Magnetic stimulation is relatively painless; therefore it is the more preferred technique. The article reviews the use of TMS for monitoring the functional integrity of the descending motor systems during surgery and discusses the potential role of TMS in the preoperative period for conscious patients planning to undergo neurosurgical procedures involving the cerebral cortex. Selective monitoring of spinal cord motor function involves acquisition of TMS-induced epidural and/or myogenic responses. As patients are generally given anesthesia before spinal cord surgeries, this article discusses the effect of general anesthetic agents on the myogenic responses.

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12

Fairbank, Jeremy. Neuromuscular scoliosis. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199550647.003.003012.

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♦ All children with neurogenic or myogenic conditions are at risk of developing scoliosis♦ The more severe the involvement, particularly in those who cannot stand, the more likely is the curve to progress♦ Surgery is the only effective way of treating these curves, and in general the earlier the better.

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13

Gross, Michael Karl. Thymidine kinase mRNA and protein levels during myogenic with drawal from the cell cycle: Identification of an mRNA-independent regulatory mechanism. 1988.

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14

Ramdass, Ranjit. Neurophysiology in the assessment of inflammatory myopathies. Edited by Hector Chinoy and Robert Cooper. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198754121.003.0015.

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Clinical neurophysiology (electrodiagnosis) includes the assessment of peripheral nerves by electrical stimulation (nerve conduction studies, NCS) and needle examination of muscles (electromyography, EMG). Electrodiagnostic assessment is a functional extension of clinical examination into the laboratory. It plays an important role in the investigation of a patient suspected of having myositis, providing valuable information regarding peripheral nerve, neuromuscular junction and muscle functions, to better characterize clinical syndromes. NCS can establish the presence and quantify the severity of a primary or co-existing peripheral neuropathy, while EMG examination can help discriminate between primary myogenic and primary neurogenic disorders. EMG is potentially more sensitive than clinical examination, as abnormalities can be detected in muscles apparently unaffected on clinical examination. Additionally, a number of muscles can be sampled to help target an optimal muscle biopsy site. Neurophysiology can also assist in monitoring treatment responses and detecting emerging problems, such steroid myopathy or drug-induced neuropathy.

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15

Blaser, Annika Reintam, and Adam M. Deane. Normal physiology of the gastrointestinal system. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0172.

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The gastrointestinal (GI) system is responsible for digestion and absorption, but also has important endocrine, immune and barrier functions. Additionally, the GI system plays a major role in fluid, electrolyte and acid-base balance. The GI system is regulated by complex myogenic, neural and humoral mechanisms, and, in health, these are affected by the presence of luminal nutrient, thereby modulating function of the GI system. Accordingly, GI function varies depending on whether a person is fasted or in the postprandial state. Adequate fasting and postprandial perfusion, motility and exocrine secretion are required for ‘normal’ functioning. The protective mechanisms of the GI system consist of physical (intact gut mucosa), non-immune (gastric acid, intestinal mucin, bile and peristalsis) and immune (gut-associated lymphoid tissue, GALT) elements. Disruption of GI protection is a putative mechanism underlying the development of multiple-organ dysfunction syndrome. Maintenance of GI function is increasingly recognised as an important factor underlying survival in critical illness.

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