Journal articles: 'Myoclonies'

1

Borg, M. "Myoclonies." EMC - Neurologie 2, no. 2 (January 2005): 1–19. http://dx.doi.org/10.1016/s0246-0378(05)21708-6.

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Borg, M. "Myoclonies." EMC - Neurologie 9, no. 3 (July 2012): 1–21. http://dx.doi.org/10.1016/s0246-0378(12)57572-x.

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Borg, M. "Myoclonies." EMC - Neurologie 2, no. 2 (May 2005): 105–32. http://dx.doi.org/10.1016/j.emcn.2004.10.004.

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Auvin, S., P. Derambure, F. Cassim, and L. Vallée. "Myoclonies et myoclonies épileptiques : orientation diagnostique et connaissances physiopathologiques." Revue Neurologique 164, no. 1 (January 2008): 3–11. http://dx.doi.org/10.1016/j.neurol.2007.12.001.

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Rey, M. "Sommeil et myoclonies." Médecine du Sommeil 3, no. 10 (December 2006): 50–52. http://dx.doi.org/10.1016/s1769-4493(06)70087-5.

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6

Genton, P., M. Bureau, C. Dravet, and R. Guerrini. "Les myoclonies épileptiques." Neurophysiologie Clinique/Clinical Neurophysiology 28, no. 4 (September 1998): 374–75. http://dx.doi.org/10.1016/s0987-7053(98)80134-2.

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Cassim, F., P. Derambure, JL Bourriez, L. Defebvre, A. Destée, and JD Guieu. "Myoclonies négatives d’origine corticale." Neurophysiologie Clinique/Clinical Neurophysiology 28, no. 4 (September 1998): 375. http://dx.doi.org/10.1016/s0987-7053(98)80135-4.

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Touchon, J., M. Miloudi, W. Camu, and F. Tarodo. "Myoclonies d’action observées chez l’enfant." Neurophysiologie Clinique/Clinical Neurophysiology 28, no. 4 (September 1998): 376–77. http://dx.doi.org/10.1016/s0987-7053(98)80138-x.

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9

Goossens, D., M. Guatterie, M. Barat, and M. de Séze. "Myoclonies vélo-pharyngées et dysphagie." Annales de Réadaptation et de Médecine Physique 47, no. 1 (February 2004): 13–19. http://dx.doi.org/10.1016/j.annrmp.2003.07.002.

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10

Vanrenterghem, B., E. Bakhache, R. Azar, and J. B. Campagne. "Myoclonies et agitation psychomotrice sous péfloxacine." Réanimation Urgences 5, no. 2 (January 1996): 117–18. http://dx.doi.org/10.1016/s1164-6756(96)80162-9.

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Vercueil, L., and J. Krieger. "Les myoclonies chez l’adulte : démarche diagnostique." Neurophysiologie Clinique/Clinical Neurophysiology 31, no. 1 (February 2001): 3–17. http://dx.doi.org/10.1016/s0987-7053(00)00244-6.

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12

Lucas, C., G. Soetaert, D. Leys, and H. Petit. "Myoclonies Au Cours D’Un Traitement Par Buflomedil." Acta Clinica Belgica 44, no. 5 (January 1989): 360–61. http://dx.doi.org/10.1080/17843286.1989.11718042.

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13

Defebvre, L. "Pathologies extrapyramidales : apport de l’étude des myoclonies." Revue Neurologique 163, no. 11 (November 2007): 1115–21. http://dx.doi.org/10.1016/s0035-3787(07)74188-7.

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14

Viallet, F., L. Vercueil, D. Gayraud, B. Bonnefoi-Kyriacou, and L. Renie. "Mouvements anormaux secondaires (dystonies, myoclonies, tremblements, dyskinésies)." EMC - Neurologie 2, no. 4 (January 2005): 1–15. http://dx.doi.org/10.1016/s0246-0378(05)40185-2.

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Viallet, F., L. Vercueil, D. Gayraud, B. Bonnefoi, and L. Renie. "Mouvements anormaux secondaires (dystonies, myoclonies, tremblements, dyskinésies)." EMC - Neurologie 9, no. 2 (April 2012): 1–16. http://dx.doi.org/10.1016/s0246-0378(12)57571-8.

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16

Jomir, L., S. Sirvain, S. Bouly, J. F. Garrido, and O. Hallé. "Syndrome opsoclonus-myoclonies : à propos d’un cas." La Revue de Médecine Interne 30, no. 11 (November 2009): 980–81. http://dx.doi.org/10.1016/j.revmed.2008.11.016.

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Viallet, F., L. Vercueil, D. Gayraud, B. Bonnefoi-Kyriacou, and L. Renie. "Mouvements anormaux secondaires (dystonies, myoclonies, tremblements, dyskinésies)." EMC - Neurologie 2, no. 4 (November 2005): 430–52. http://dx.doi.org/10.1016/j.emcn.2005.07.002.

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18

Leclerc, C., and E. Vauguet. "Myoclonies induites par le sévoflurane chez l'enfant." Annales Françaises d'Anesthésie et de Réanimation 25, no. 8 (August 2006): 903. http://dx.doi.org/10.1016/j.annfar.2006.06.007.

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19

Imounan, Fatima, Lahbouje Siham, Naamane Taoufik, Raoud Abdelhak, and Daoudi Naima. "Myoclonies propriospinales suite à une prise de quinolones." Revue Neurologique 173 (March 2017): S149. http://dx.doi.org/10.1016/j.neurol.2017.01.273.

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20

Gross, Marie-Laure, Anne Doé De Maindreville, Maria Del Mar Amador, David Grabli, Anne Thiriaux, Marie-José Vidailhet, and Emmanuel Flamand-Roze. "Myoclonies d’action et protéinurie modérée isolée : penser SCARB2." Revue Neurologique 174 (April 2018): S125. http://dx.doi.org/10.1016/j.neurol.2018.01.284.

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21

Durand, J. M., H. Telle, V. Taramasco, N. Guiles, and J. Soubeyrand. "Syndrome confusionnel, myoclonies et traitement par la Péfloxacine." La Revue de Médecine Interne 13, no. 7 (December 1992): S550. http://dx.doi.org/10.1016/s0248-8663(05)81129-4.

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22

Vilchez, JJ, A. Lago, JA Burguera, R. Yaya, and V. Casanova. "Chlormethiazole in the Management of the Opsoclonus-Myoclonus Syndrome." Journal of International Medical Research 22, no. 1 (January 1994): 55–62. http://dx.doi.org/10.1177/030006059402200107.

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Two instances of successful treatment of the rare ocular dyskinesia, opsoclonus, with chlormethiazole are reported. A 65-year-old woman had the opsoclonus-myoclonus syndrome associated with carcinoma of the breast; her myoclonia and opsoclonus did not respond to intravenous diazepam or phenytoin. Treatment with intravenous chlormethiazole resulted in rapid control of her myoclonic attacks, followed by slower but complete resolution of the opsoclonus. Following control of the acute symptoms the patient was transferred to an oral chlormethiazole maintenance dose which was further reduced and subsequently discontinued after 5 months, when the patient's overall clinical status had improved. A 53-year-old man with opsoclonia, myoclonia, ataxia and encephalopathy, not associated with neoplasia, was given immunosuppressor drugs to establish basal control, and oral chlormethiazole for symptomatic treatment. Almost immediately after the initial dose of chlormethiazole the patient became more orientated; he was sedated and the agitation and myoclonic fits were brought under control quite quickly. The opsoclonus responded progressively and was completely resolved after a few days. The initial oral dose of chlormethiazole was gradually reduced and was discontinued after 5–6 months. Chlormethiazole was well tolerated; it may have an important role in the management of the rare opsoclonus-myoclonus syndrome.

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23

Gardiol, Lamy, and Humbert. "Myoclonies chez une personne très âgée: une cause fréquente et réversible." Praxis 99, no. 5 (March 1, 2010): 315–18. http://dx.doi.org/10.1024/1661-8157/a000055.

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Nous rapportons le cas d'une personne âgée de 98 ans hospitalisée pour des myoclonies généralisées. Le laboratoire montre une hypocalcémie ainsi qu'une vitamine D abaissée. Nous posons donc le diagnostic de troubles neuromusculaires sur hypocalcémie dans un contexte d'hypovitaminose D. Les symptômes s'amendent rapidement après substitution intraveineuse puis orale. Ce cas illustre l'importance du bilan électrolytique, surtout chez la personne âgée, lors de manifestations neuromusculaires.

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24

Rucar, Alice, Oana Catar, Anne Sophie Renard, and Christophe Verny. "Des myoclonies non épileptiques révélant une myélinolyse centro pontine." Revue Neurologique 173 (March 2017): S150. http://dx.doi.org/10.1016/j.neurol.2017.01.275.

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25

Drouet, A., and J. Valance. "Myoclonies de repos et d'action induites par la pipérazine." La Revue de Médecine Interne 15, no. 5 (January 1994): 364–65. http://dx.doi.org/10.1016/s0248-8663(05)81448-1.

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26

Stephan-Carlier, A., L. le Chapelain, I. Arnoud, J. M. Beis, and J. Paysant. "Myoclonies spinales segmentaires chez un patient tétraplégique d’origine traumatique." Annals of Physical and Rehabilitation Medicine 56 (October 2013): e77. http://dx.doi.org/10.1016/j.rehab.2013.07.434.

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27

Yahdi, V., N. Ech-Cherif El Kettani, S. Lytim, M. Fikri, M. Yahyaoui, M. R. El Hassani, and M. Jiddane. "Myoclonies et syndrome démentiel chez une patiente de 60ans." Feuillets de Radiologie 53, no. 6 (December 2013): 370–73. http://dx.doi.org/10.1016/j.frad.2013.10.002.

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Yahdi, V., N. Ech-Cherif El Kettani, S. Lytim, M. Fikri, M. Yahyaoui, M. R. El Hassani, and M. Jiddane. "Myoclonies et syndrome démentiel chez une patiente de 60ans." Feuillets de Radiologie 53, no. 6 (December 2013): 393. http://dx.doi.org/10.1016/j.frad.2013.10.004.

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29

Gayraud, D., B. Bonnefoi, A. Roux, and F. Viallet. "Mouvements anormaux secondaires (dystonies, chorées/ballisme, myoclonies, tremblements, dyskinésies)." EMC - Neurologie 44, no. 1 (January 2021): 1–17. https://doi.org/10.1016/s0246-0378(20)42810-6.

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30

Capovilla, Giuseppe, Guido Rubboli, Francesca Beccaria, Maria Elena Lorenzetti, Alessandra Montagnini, Cristina Resi, Elena Gardella, Antonio Gambardella, Antonino Romeo, and Carlo Alberto Tassinari. "A clinical spectrum of the myoclonic manifestations associated with typical absences in childhood absence epilepsy. A video‐polygraphic study." Epileptic Disorders 3, no. 2 (June 2001): 57–61. http://dx.doi.org/10.1684/j.1950-6945.2001.tb00471.x.

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ABSTRACT We investigated the electroclinical features of 12 patients with childhood absence epilepsy (CAE), presenting with typical absence seizures associated with myoclonic manifestations of the face or neck. All patients underwent repeated and prolonged split‐screen video‐polygraphic EEG recordings. The polygraphic recordings and clinical correlations of the absence seizures were analysed. All patients presented with multi‐quotidian, typical absence seizures. During the absences, the patients could show mild, rhythmic, myoclonic jerks involving facial areas (eyebrows, nostrils, perioral region, chin) or neck muscles (sternocleidomastoideus), with the same frequency as the spike‐wave complexes. Polygraphic tracings demonstrated that the myoclonias were correlated to the spike component. Clinically, all patients showed a benign course, with complete seizure control under antiepileptic treatment. In the follow‐up, 7 patients withdrew from treatment without relapse. We conclude that all our patients showed an electroclinical picture consistent with CAE. The occurrence of myoclonic manifestations of the face or neck associated with the absences did not influence the benign course of their disease. The electroclinical features observed in our group of patients differentiates our cases both from epilepsy with myoclonic absences and from absences with perioral myoclonia ( with Video ).

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31

Mezouar, N., A. Not, C. Dussaule, P. Chretien, F. Archambaud, C. Denier, and P. Masnou. "Myoclonies focales révélant une encéphalite limbique à anticorps anti-LGI1." Revue Neurologique 170 (April 2014): A143—A144. http://dx.doi.org/10.1016/j.neurol.2014.01.384.

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32

Hennebicq, S., G. Dawlat, S. Franck, C. De Maeseneire, and Y. Bouckaert. "Opsoclonus myoclonus syndrome in a covid 19 patient - case report." Revue Medicale de Bruxelles 44, no. 1 (2023): 44–46. http://dx.doi.org/10.30637/2023.21-033.

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33

Tortuyaux, R., L. Defebvre, G. Baille, and C. Simonin. "Encéphalomyélite progressive avec rigidité et myoclonies compliquée d’un coup de chaleur." Pratique Neurologique - FMC 9, no. 1 (February 2018): 42–47. http://dx.doi.org/10.1016/j.praneu.2018.01.006.

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34

Aissaoui, Y., L. Belyamani, A. El Wali, S. M. Idrissi Hajjouji, M. Atmani, and N. Drissi Kamili. "Prévention des myoclonies induites par l'étomidate en utilisant une priming dose." Annales Françaises d'Anesthésie et de Réanimation 25, no. 10 (October 2006): 1041–45. http://dx.doi.org/10.1016/j.annfar.2006.07.079.

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35

Wirth, Thomas, Charlotte Kaeuffer, Jean-Baptiste Chanson, Mathilde Renaud, Mathieu Anheim, Francis Schneider, and Christine Tranchant. "Encéphalomyélite progressive avec rigidité et myoclonies, un diagnostic difficile en réanimation médicale." Revue Neurologique 173 (March 2017): S147. http://dx.doi.org/10.1016/j.neurol.2017.01.268.

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36

McLeod, GA, and MC Ng. "P.011 Not just for babies: positive rolandic sharp waves in adult post-hypoxic myoclonus." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 45, s2 (June 2018): S18. http://dx.doi.org/10.1017/cjn.2018.113.

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Background: Post-hypoxic myoclonus is broadly divided into myoclonic status epilepticus (MSE) and Lance-Adams syndrome (LAS), where diagnosis depends on clinical and electroencephalographic (EEG) findings. Positive rolandic sharp waves (PRS) are a classic EEG finding in pre-term infants with white matter necrosis, but they are not known to be epileptogenic and have never been described in adults. Methods: We report a unique case of PRS correlated with myoclonic seizures in a post-hypoxic adult patient. Results: Shortly after cardiac arrest, a 21-year-old woman developed multifocal post-hypoxic myoclonus. Early development of myoclonus suggested MSE, but her EEG findings were atypical for MSE; initially, the only notable feature on EEG were subtle PRS. LAS did not fit the clinical picture or EEG findings. As myoclonus persisted over the following weeks, PRS evolved on EEG into positive rolandic predominant generalized polyspike-wave complexes that became definitively time-locked to each myoclonic jerk. PRS were diagnosed as epileptogenic and frequent myoclonic jerks were diagnosed as continuous myoclonic seizure. Myoclonus resolved to medication and mental status returned to baseline. Conclusions: We report for the first time that PRS can appear in adult patients and be epileptogenic, and produce a non-classical variant of post-hypoxic myoclonus that carries good prognosis.

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37

Szabo, A., P. Pichit, M. Baulac, and S. Dupont. "Myoclonies atypiques sévères répondant rapidement à l’immunothérapie : l’encéphalite limbique à anticorps anti-LGI1." Pratique Neurologique - FMC 2, no. 4 (December 2011): 268–72. http://dx.doi.org/10.1016/j.praneu.2011.09.005.

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38

Van Der Veen, Sterre, Marina A. J. Tijssen, and Samuel F. Berkovic. "The Muddle of Myoclonus." Neurology: Clinical Practice 13, no. 5 (August 31, 2023): e200187. http://dx.doi.org/10.1212/cpj.0000000000200187.

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Myoclonus is often approached in different ways by epileptologists and movement disorder specialists, leading to confusion in the literature. Multiplicity and inconsistency over the past 2 centuries resulted in a lack of precision and ambiguity of the terminology. We show that this is a current problem in which one phenomenon has been described with many terms and vice versa. Of more importance, we discuss the conceptualization of myoclonus from perspectives of both fields and focus on the borderland that exists, especially in the spectrum of cortical and epileptic myoclonus. By giving 2 examples, we illustrate the conundrum: the spectrum of progressive myoclonus epilepsies and progressive myoclonic ataxias and “cortical tremor” observed in familial cortical myoclonic tremor with epilepsy or familial adult myoclonic epilepsy. We attempt to facilitate to bridge these subspecialties and form the base for a uniform understanding to take this issue forward toward future classifications, discussions, and scientific research.

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39

Obara, Koji, Tsuyoshi Imota, Shigeo Mamiya, and Itaru Toyoshima. "The Effectiveness of Perampanel for Myoclonic Seizures in Down Syndrome with Isodicentric Chromosome 21." Case Reports in Neurology 12, no. 3 (September 10, 2020): 270–75. http://dx.doi.org/10.1159/000508357.

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Epileptic seizures are common in the elderly Down syndrome population. We encountered a patient with Down syndrome in whom karyotyping showed the rare isodicentric chromosome 21 and who suffered from myoclonic seizures. A 52-year-old woman with Down syndrome experienced sudden onset of drowsiness and frequent myoclonic jerks in the upper body. Video-EEG recordings demonstrated generalized polyspike-wave discharges consistent with myoclonic jerks, which were exacerbated by photo-stimulation. Her myoclonus completely resolved with perampanel administration. Perampanel was effective for myoclonic seizures in our patient. We suggest that perampanel is an option as first-line therapy for epilepsy and myoclonus in elderly Down syndrome patients.

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40

Krauss, Gregory L., and Gregory C. Mathews. "Similarities in Mechanisms and Treatments for Epileptic and Nonepileptic Myoclonus." Epilepsy Currents 3, no. 1 (January 2003): 19–21. http://dx.doi.org/10.1111/j.1535-7597.2003.03107.x.

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Myoclonus is a disordered movement that may be an ictal phenomenon or may be due to various injuries in brain and spinal cord motor structures. Many epileptic and nonepileptic myoclonic conditions are associated with abnormalities in inhibitory neurotransmission. γ-Aminobutyric acid type A (GABAA)-receptor antagonists may trigger myoclonus. Several antiepilepsy drugs (AEDs) effective against myoclonic seizures [valproic acid (VPA), clonazepam (CZP), levetiracetam (LEV)] enhance GABAergic neurotransmission and improve myoclonic movement disorders. Together these associations suggest links between episodic disorders involving synchronous cortical discharges (seizures) and hyperkinetic movement disorders.

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41

Liron, L., M. Chambost, P. Depierre, D. Peillon, and C. Combe. "Efficacité de l'acide valproïque sur les myoclonies d'action postanoxiques (syndrome de Lance et Adams)." Annales Françaises d'Anesthésie et de Réanimation 17, no. 10 (January 1998): 1247–49. http://dx.doi.org/10.1016/s0750-7658(99)80033-1.

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42

Naquet, R., and C. Batini. "Stimulation lumineuse intermittente et myoclonies. Rôles respectifs du tronc cérébral et des réseaux corticothalamiques." Neurophysiologie Clinique/Clinical Neurophysiology 28, no. 4 (September 1998): 360. http://dx.doi.org/10.1016/s0987-7053(98)80006-3.

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43

Boyez, E., and P. Malherbe. "Curarisation en traitement symptomatique d'appoint des myoclonies induites par une intoxication par le chloralose." Annales Françaises d'Anesthésie et de Réanimation 23, no. 4 (April 2004): 361–63. http://dx.doi.org/10.1016/j.annfar.2003.11.020.

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44

Capelle, Hans-Holger, Johannes C. Wöhrle, Ralf Weigel, Eva Grips, Hans-Jörg Bäzner, and Joachim K. Krauss. "Propriospinal myoclonus due to cervical disc herniation." Journal of Neurosurgery: Spine 2, no. 5 (May 2005): 608–11. http://dx.doi.org/10.3171/spi.2005.2.5.0608.

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✓ Propriospinal myoclonus is a rare form of spinal myoclonus. In most cases the cause has remained unclear. Secondary propriospinal myoclonus has been described secondary to various disorders including trauma, tumor, and infection. Thus far, propriospinal myoclonus caused by cervical disc herniation has not been reported. In the present report, the authors describe the case of a 53-year-old man who presented with radicular symptoms of the right C-6 nerve root and myoclonic twitches predominantly affecting the abdominal muscles but spreading to adjacent muscles. The spread was triggered and enforced by certain movements. Magnetic resonance imaging studies revealed a C-6 nerve root compression at the C5–6 level on the right side but no cervical myelopathy. Electromyography studies confirmed the diagnosis of propriospinal myoclonus. After discectomy and cage-augmented fusion via an anterior approach, the myoclonic movement disorder gradually subsided. To the authors' knowledge, this is the first report on successful treatment of propriospinal myoclonus by spinal disc surgery.

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45

Di Bonaventura, Carlo, Jinane Fattouch, Francesco Mari, Gabriella Egeo, Anna Elisabetta Vaudano, Massimiliano Prencipe, Mario Manfredi, and Anna Teresa Giallonardo. "Clinical experience with levetiracetam in idiopathic generalized epilepsy according to different syndrome subtypes." Epileptic Disorders 7, no. 3 (September 2005): 231–35. http://dx.doi.org/10.1684/j.1950-6945.2005.tb00127.x.

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Clinical experience in open‐label studies and anectodal reports suggest that levetiracetam is effective in generalized epilepsy. In this open‐label prospective study, 19 patients (3 men, 16 women) affected by idiopathic generalized epilepsy were followed for at least 6 months following the introduction of levetiracetam. Patients were categorized according to syndrome subtype: juvenile myoclonic epilepsy (8), juvenile absence epilepsy (5), childhood absence epilepsy (4), and eyelid myoclonia with absences (2). Eleven patients received levetiracetam as monotherapy, eight as add‐on therapy. Effectiveness was demonstrated in 18 patients: 13 became seizure‐free (five juvenile myoclonic epilepsy, five juvenile absence epilepsy, three childhood absence epilepsy), and five achieved significant reductions in seizure frequency (three juvenile myoclonic epilepsy, one childhood absence epilepsy, one eyelid myoclonia with absences). Only one patient experienced no change in seizure frequency (eyelid myoclonia with absences). Clinical improvement was accompanied by EEG abnormality suppression or reduction. Levetiracetam was well tolerated; no patient reported side‐effects.

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46

Mann, M. W., B. Gueguen, S. Guillou, E. Debrand, and C. Soufflet. "Chess‐playing epilepsy: a case report with video‐EEG and back averaging." Epileptic Disorders 6, no. 4 (December 2004): 293–96. http://dx.doi.org/10.1684/j.1950-6945.2004.tb00079.x.

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A patient suffering from juvenile myoclonic epilepsy experienced myoclonic jerks, fairly regularly, while playing chess. The myoclonus appeared particularly when he had to plan his strategy, to choose between two solutions or while raising the arm to move a chess figure. Video‐EEG‐polygraphy was performed, with back averaging of the myoclonus registered during a chess match and during neuropsychological testing with Kohs cubes. The EEG spike wave complexes were localised in the fronto‐central region. [Published with video sequences]

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47

Kandearachchi, K. A. M. S. P., A. G. H. Menike, K. G. Madawala, H. H. Sarathchandra, N. L. S. H. Jayawardana, H. R. R. Kulasiri, and D. I. J. Samarasekara. "Spinal Myoclonus a Rare Complication Following Administration of Spinal Anaeasthesia – A Case Report." Sri Lankan Journal of Anaesthesiology 32, no. 1 (January 30, 2024): 74–78. http://dx.doi.org/10.4038/slja.v32i1.9251.

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Spinal myoclonus is a rare complication following spinal anesthesia. Transient myoclonic jerks are referred as spinal myoclonus and it is diagnosed by exclusion. This case report describes a successful management of spinal myoclonus after spinal anesthesia.A 48 year old, woman who had undergone incision and drainage under spinal anesthesia, developed truncal myoclonus 45 minutes following induction of anesthesia. It was effectively alleviated through administration of IV midazolam.

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48

Onder, Halil. "A Case of Myoclonic Epilepsy Presenting with Status Epilepticus in an Elderly Male Patient." Journal of Neurosciences in Rural Practice 11, no. 03 (June 16, 2020): 463–66. http://dx.doi.org/10.1055/s-0040-1709348.

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AbstractMyoclonic epilepsy in the form of status epilepticus is an extremely rare reported presentation. Herein, we describe an 87-year old male patient presenting with abrupt-onset rhythmic myoclonic jerks that were evaluated as synchronous positive and negative myoclonus. Further etiological investigations revealed that the myoclonus was associated with ictogenesis, and appropriate antiepileptic treatment provided total cessation of the movements. To my knowledge, this is a unique case of myoclonic status epilepticus in an elderly patient, which may have been associated with a cerebrovascular disease. In the presentation of this case, we will review the related literature and discuss some considerations to explain the pathophysiology of epileptic myoclonic movements and the possible role of pontine lesions.

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49

Shishido, Ayumi, Hiroshi Yamaguchi, Yusuke Ishida, and Tamaki Ohashi. "Prolonged Febrile Myoclonus in an Infant." Journal of Pediatric Epilepsy 09, no. 01 (March 2020): 022–24. http://dx.doi.org/10.1055/s-0040-1709654.

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AbstractFebrile myoclonus is considered a benign symptom of myoclonic jerks associated with fever. Although cases have been sporadically reported, it is a poorly characterized phenomenon. In this study, we reported the case of a previously healthy 20-month-old boy who developed atypically prolonged myoclonic jerks, appearing more than 100 times over a period of 3 days. Blood cultures were negative, and his electroencephalogram was normal. The patient was discharged with a diagnosis of febrile myoclonus and was followed up for several months. His brain magnetic resonance imaging and brain development were also normal. Physicians must understand the benign clinical course of febrile myoclonus in order to avoid unnecessary procedures such as lumbar puncture.

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Dooley, J. M., and J. D. Hayden. "Benign Febrile Myoclonus in Childhood." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 31, no. 4 (November 2004): 504–5. http://dx.doi.org/10.1017/s0317167100003711.

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Abstract:

Background:Myoclonus is often associated with progressive myoclonic epilepsy or neurodegenerative conditions. Febrile myoclonus is a benign phenomenon, which has only been reported previously in one child.Methods:The clinical features of three children with fever-induced myoclonus are described.Results:Fever-induced myoclonus is characterized by frequent myoclonus, which resolves with resolution of the fever in otherwise healthy children.Conclusion:Recognition of fever-induced myoclonus as a benign phenomenon may prevent unnecessary investigations and interventions.Conclusion:Le fait de reconnaître le myoclonus induit par l'hyperthermie comme un phÉnomène bÉnin peut Éviter des investigations et des interventions inutiles.

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Journal articles on the topic 'Myoclonies'

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