Relevant bibliographies by topics / Myocardial Infarctio

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Myocardial Infarctio'

Author: Grafiati
Published: 9 March 2023
Last updated: 31 July 2025

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Journal articles on the topic "Myocardial Infarctio"

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Oleynikov, V. E., A. A. Chernova, E. V. Averyanova, and A. V. Kulyutsin. "Pathogenesis and clinical significance of reperfusion arrhythmias in acute myocardial infarctio." Russian Journal of Cardiology 29, no. 3S (2024): 5958. http://dx.doi.org/10.15829/1560-4071-2024-5958.

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The problem of ischemia-reperfusion injury, in particular, reperfusion arrhythmias, has remained controversial for many years. To date, there are no one frame of mind on the clinical and prognostic significance of tachyarrhythmias in the acute period of myocardial infarction. In addition, data on the incidence of reperfusion arrhythmias and related mortality are very contradictory. The review summarizes current concepts and results of studies devoted to the study of the clinical role of reperfusion arrhythmias. Their pathogenesis, structure, and impact on the long-term prognosis of patients ar
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Shriki, J. E., K. Surti, A. Farvid, J. S. Shinbane, and P. M. Colletti. "Quantitative Evaluation of the Amount of Delayed Myocardial Enhancement as a Predictor of Systolic Dysfunction." Open Cardiovascular Medicine Journal 3, no. 1 (2009): 35–38. http://dx.doi.org/10.2174/1874192400903010035.

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30 patients with delayed contrast enhancement in patterns suggestive of myocardial infarctions were reviewed. Infarct mass was quantitatively measured using short axis images obtained in the delayed phase of gadopentetate administration. Left ventricular mass and ejection fraction were measured using short axis, steady state free precession images. A relationship is drawn between increased mass of infarction and decreased left ventricular ejection fraction. For each gram of infarct, there is a 0.5 % reduction in ejection fraction (EF = 50 - (0.48 x gm infarcted myocardium); r2= 0.49). For each
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O'Regan, Declan P., Rizwan Ahmed, Clare Neuwirth, et al. "Cardiac MRI of myocardial salvage at the peri-infarct border zones after primary coronary intervention." American Journal of Physiology-Heart and Circulatory Physiology 297, no. 1 (2009): H340—H346. http://dx.doi.org/10.1152/ajpheart.00011.2009.

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The purpose of this study was to use cardiac MRI to define the morphology of the reversibly injured peri-infarct border zone in patients treated with primary percutaneous coronary intervention (PPCI) for acute ST elevation myocardial infarction. In 15 patients, T2-weighted myocardial edema imaging was used to identify the ischemic bed or area at risk (AAR), and late gadolinium enhancement imaging was used to measure infarct size. Images were coregistered, and the boundaries of edema and necrosis were defined using an edge-detection methodology. We observed that infarction always involved the s
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Carlsson, M., P. C. Ursell, D. Saloner, and M. Saeed. "Multidetector computed tomography for characterization of calcium deposits in reperfused myocardial infarction." Acta Radiologica 50, no. 4 (2009): 396–405. http://dx.doi.org/10.1080/02841850902756540.

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Background: Calcium overload is a major cause of reperfusion myocardial injury. Multidetector computed tomography (MDCT) has been previously used in visualizing coronary artery calcium, but not calcium deposits in reperfused infarction. Purpose: To assess the ability of MDCT to 1) noninvasively visualize and characterize calcium deposits in reperfused infarcts, and 2) monitor regional wall swelling, regional systolic wall thickening, and infarct resorption. Material and Methods: Reperfused myocardial infarcts were created in seven pigs by 2-hour occlusion of the left anterior descending corona
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Nilsson, S., G. Wikström, A. Ericsson, M. Wikström, A. Waldenström, and A. Hemmingsson. "MR Imaging of Gadolinium-DTPA-BMA-Enhanced Reperfused and Nonreperfused Porcine Myocardial Infarction." Acta Radiologica 36, no. 4-6 (1995): 633–40. http://dx.doi.org/10.1177/028418519503600465.

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To investigate whether Gd-DTPA-BMA-enhanced MR imaging permits differentiation between reperfused and nonreperfused myocardial infarction, myocardial infarction was induced in 12 domestic pigs. In 6 pigs, Gd-DTPA-BMA, 0.3 mmol/kg b.w. was administered i.v. 60 min after the occlusion. In 6 other pigs, the infarctions were reperfused 80 min after the occlusion, followed by injection of Gd-DTPA-BMA after 20 min of reperfusion. Radiolabeled microspheres were used to confirm zero-flow during the occlusion period and reperfusion in the infarcted myocardium. All pigs were killed 20 min after injectio
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Ristic-Andjelkov, Andjelka, Branislav Baskot, Milorad Damjanovic, and Sasa Rafajlovski. "Ischemic preconditioning." Vojnosanitetski pregled 62, no. 1 (2005): 73–77. http://dx.doi.org/10.2298/vsp0501073r.

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Background. Ischemic preconditioning is a phenomenon during which myocardium, subjected to brief episodes of ischemia followed by reperfusion, tolerates better the subsequent, more prolonged episode of this ischemia, thus reducing the infarction size substantially. Case report. Two patients with acute left anterior descendent artery occlusion received fibrinolytic therapy (alteplase) within 6 hours of the onset of chest pain, but developed myocardial infarctions of different sizes. The first patient, without the history of preinfarction angina, developed large anterior infarct, because there w
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Spath, Nick B., Trisha Singh, Giorgos Papanastasiou, et al. "Assessment of stunned and viable myocardium using manganese-enhanced MRI." Open Heart 8, no. 1 (2021): e001646. http://dx.doi.org/10.1136/openhrt-2021-001646.

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ObjectiveIn a proof-of-concept study, to quantify myocardial viability in patients with acute myocardial infarction using manganese-enhanced MRI (MEMRI), a measure of intracellular calcium handling.MethodsHealthy volunteers (n=20) and patients with ST-elevation myocardial infarction (n=20) underwent late gadolinium enhancement (LGE) using gadobutrol and MEMRI using manganese dipyridoxyl diphosphate. Patients were scanned ≤7 days after reperfusion and rescanned after 3 months. Differential manganese uptake was described using a two-compartment model.ResultsAfter manganese administration, health
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Song, Yi-Sun, Hyun-Woo Joo, In-Hwa Park, et al. "Transplanted Human Amniotic Epithelial Cells Secrete Paracrine Proangiogenic Cytokines in Rat Model of Myocardial Infarctio." Cell Transplantation 24, no. 10 (2015): 2055–64. http://dx.doi.org/10.3727/096368914x685609.

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Bo, Yang, and Li Min. "GW24-e0030 A multi-nationality study on gender differences in anxiety early after acute myocardial infarctio." Heart 99, Suppl 3 (2013): A259.2—A259. http://dx.doi.org/10.1136/heartjnl-2013-304613.730.

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PETTERSEN, K. I. "When Time is Precious--Time Lags as Indicators of Quality of Care in Acute Myocardial Infarctio." International Journal for Quality in Health Care 7, no. 1 (1995): 3–10. http://dx.doi.org/10.1093/intqhc/7.1.3.

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Dissertations / Theses on the topic "Myocardial Infarctio"

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HANTIKAINEN, ESSI MARJATTA. "DIETARY NON ENZYMATIC ANTIOXIDANT CAPACITY AND THE RISK OF CARDIOVASCULAR DISEASES – AN EPIDEMIOLOGICAL APPROACH." Doctoral thesis, Università degli Studi di Milano-Bicocca, 2020. http://hdl.handle.net/10281/263728.

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ABSTRACT Cardiovascular diseases are the leading cause of premature death and disability in the world. A diet containing high amounts of plant-based foods has been associated with a reduced risk of cardiovascular diseases and the beneficial effect has been attributed to the antioxidants found in the foods. However, findings from randomized controlled trials on the role of antioxidant supplementation have been disappointing, reporting null results or even harmful effects. It has been suggested that antioxidants interact with each other to promote cardiovascular health. Therefore, the Non Enzyma
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Murphy, Megan K. "Fibrin microthreads promote stem cell growth for localized delivery in regenerative therapy." Worcester, Mass. : Worcester Polytechnic Institute, 2008. http://www.wpi.edu/Pubs/ETD/Available/etd-090208-143505/.

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Löwbeer, Christian. "Cardiac troponin T in clinical and experimental studies /." Stockholm, 2007. http://diss.kib.ki.se/2007/978-91-7357-426-6/.

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Surányi, Pál. "Relaxation rate-based magnetic resonance imaging quantification of myocardial infarction." Thesis, Birmingham, Ala. : University of Alabama at Birmingham, 2007. https://www.mhsl.uab.edu/dt/2007r/suranyi.pdf.

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Elhdere, Souada Ahmed. "Illness cognitions in myocardial infarction." Thesis, University of Surrey, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.548363.

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Williams, John. "Marker proteins in myocardial infarction." Thesis, University of Ulster, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.359319.

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Ruparelia, Neil. "Monocytes in acute myocardial infarction." Thesis, University of Oxford, 2013. http://ora.ox.ac.uk/objects/uuid:02ad6ebd-a8c2-4cb6-a1f7-0cdf8cec59ed.

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Acute myocardial infarction (AMI) results in the activation of the innate immune system with monocytes playing critical roles in both the initial inflammation following myocardial ischaemia and subsequent recovery. Monocytes are a heterogeneous cell population and observations from experimental models demonstrate that immediately following myocardial injury, classical inflammatory monocytes, which are highly phagocytic, are recruited to ischaemic myocardium from the bone marrow and spleen and peak at 48 hours. This is followed by the recruitment of non-classical monocytes that are involved in
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Bouhidel, Jalaleddinne Omar. "Etude de la cardioprotection contre l'infarctus du myocarde au cours de l'obésité expérimentale." Thesis, Paris Est, 2010. http://www.theses.fr/2010PEST0034.

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L'infarctus du myocarde (IDM) est l'une des principales causes de morbi-mortalité dans les pays développés et ce malgré l'amélioration enregistrée ces dernières années dans sa prise en charge thérapeutique. L'obésité est classée comme étant un facteur de risque majeur pour les maladies coronaires par l'American Heart Association, et concerne 14,5 % de la population française (enquête ObEpi-Roche/INSERM, 2009). En utilisant un modèle expérimental d'obésité, la souris ob/ob génétiquement dépourvue en leptine, l'objectif du présent travail de thèse a été d'étudier l'efficacité de stratégies cardi
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Buchanan, Lynne M. "Psychophysiological recovery after acute myocardial infarction /." Thesis, Connect to this title online; UW restricted, 1989. http://hdl.handle.net/1773/7244.

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Dawson, Lynn Gail. "Coping behaviours in myocardial infarction rehabilitation." Thesis, University of British Columbia, 1986. http://hdl.handle.net/2429/25722.

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This study was designed to discover the coping behaviours used by patients six to twelve months following a myocardial infarction (MI). The conceptualization of coping behaviours was based on the UBC Model for Nursing which directed the researcher to examine coping behaviours used to meet the patients' basic human needs. The specific research question was, "What new or already established coping behaviours have patients utilized after an MI in an attempt to satisfy their basic human needs?" Seven participants who had experienced an MI six to twelve months previously, were recruited from car
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Books on the topic "Myocardial Infarctio"

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1931-, Frohlich Edward D., ed. Preventive aspects of coronary heart disease. Davis, 1990.

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J, Gersh Bernard, and Rahimtoola Shahbudin H, eds. Acute myocardial infarction. 2nd ed. Chapman & Hall, 1996.

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David, McCall, ed. Acute myocardial infarction. Churchill Livingstone, 1991.

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J, Gersh Bernard, and Rahimtoola Shahbudin H, eds. Acute myocardial infarction. Elsevier, 1991.

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G, Nayler Winifred, Parratt James R, and International Society for Heart Research. European Section. Meeting., eds. Myocardial response to acute injury. Macmillan Academic and Professional, 1991.

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R, Parratt James, and Nayler W. G. 1930-, eds. Myocardial response to acute injury. Macmillan Press, 1992.

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Blocker, William P. Rehabilitation after myocardial infarction. CIBA-Geigy, 1986.

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E, Bush David, United States. Agency for Healthcare Research and Quality., and Johns Hopkins University. Evidence-based Practice Center., eds. Post-myocardial infarction depression. Agency for Healthcare Research and Quality, 2005.

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NHS Centre for Reviews & Dissemination., ed. Aspirin and myocardial infarction. NHS Centre for Reviews and Dissemination, University of York, 1995.

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E, Manson JoAnn, ed. Prevention of myocardial infarction. Oxford University Press, 1996.

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Book chapters on the topic "Myocardial Infarctio"

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Becker, Christoph R. "Myocardial Infarction." In Integrated Cardiothoracic Imaging with MDCT. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-72387-5_17.

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Selvester, Ronald H., David G. Strauss, and Galen S. Wagner. "Myocardial Infarction." In Electrocardiology. Springer London, 2011. http://dx.doi.org/10.1007/978-0-85729-874-4_4.

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Seaver, Robert L. "Myocardial Infarction." In When Doctors Get Sick. Springer US, 1988. http://dx.doi.org/10.1007/978-1-4899-2001-0_4.

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Roth, Elliot J. "Myocardial Infarction." In Encyclopedia of Clinical Neuropsychology. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-57111-9_2192.

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Saclarides, Theodore J. "Myocardial Infarction." In Common Surgical Diseases. Springer New York, 1998. http://dx.doi.org/10.1007/978-1-4757-2945-0_24.

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Liu, Peijun, Yining Wang, and Zheng-yu Jin. "Myocardial Infarction." In Cardiac CT. Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-5305-9_2.

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Roth, Elliot J. "Myocardial Infarction." In Encyclopedia of Clinical Neuropsychology. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-56782-2_2192-2.

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Ko, Hanjo. "Myocardial Infarction." In Anesthesiology. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-50141-3_2.

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Blake, Thomas M. "Myocardial Infarction." In The Practice of Electrocardiography. Humana Press, 1994. http://dx.doi.org/10.1007/978-1-4612-0291-2_11.

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Miura, Tetsuji, Derek M. Yellon, and James M. Downey. "Myocardial Infarction." In Physiology and Pathophysiology of the Heart. Springer US, 1989. http://dx.doi.org/10.1007/978-1-4613-0873-7_47.

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Conference papers on the topic "Myocardial Infarctio"

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Damit, Dayang Suhaida Awang, A’Isyah Nadiyah Hilmi, Siti Noraini Sulaiman, Muhammad Khusairi Osman, Noor Khairiah A. Karim, and Nor Adni Mat Leh. "Impact of Class Labeling on Myocardium Segmentation using Cascaded Deep Learning for Improved Myocardial Infarction Segmentation." In 2024 IEEE 14th International Conference on Control System, Computing and Engineering (ICCSCE). IEEE, 2024. http://dx.doi.org/10.1109/iccsce61582.2024.10696767.

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El Omary, Sara, Souad Lahrache, and Rajae El Ouazzani. "Exploring Machine Learning Algorithms for Myocardial Infarction Diagnosis." In 2024 International Conference on Intelligent Systems and Computer Vision (ISCV). IEEE, 2024. http://dx.doi.org/10.1109/iscv60512.2024.10620080.

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Khan, Md Rahat Kader, Tamador Mohaidat, and Kasem Khalil. "Predicting Acute Myocardial Infarction Using Machine Learning Algorithms." In 2024 IEEE 2nd International Conference on Artificial Intelligence, Blockchain, and Internet of Things (AIBThings). IEEE, 2024. https://doi.org/10.1109/aibthings63359.2024.10863008.

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Discher, Dennis, and Adam Engler. "Mesenchymal Stem Cell Injection After Myocardial Infarction Improves Myocardial Compliance." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176754.

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Cellular therapy for myocardial injury has improved ventricular function in both animal and clinical studies, though the mechanism of benefit is unclear. This study was undertaken to examine the effects of cellular injection after infarction on myocardial elasticity. Coronary artery ligation of Lewis rats was followed by direct injection of human mesenchymal stem cells (MSC) into the acutely ischemic myocardium. Two weeks post-infarct, myocardial elasticity was mapped by atomic force microscopy. MSC-injected hearts near the infarct region were two-fold stiffer than myocardium from non-infarcte
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Goktepe, Serdar, Joseph P. Ulerich, and Ellen Kuhl. "How to Treat the Loss of Beat: Modeling and Simulation of Ventricular Growth and Remodeling and Novel Post-Infarction Therapies." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-193159.

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Heart disease is the primary cause of death in industrialized nations. In 2007 alone, an estimated 79 million adults in the U.S., one in three, had one or more types of cardiovascular disease, generating health care costs in excess of $430 billion. A leading cause of congestive heart failure is myocardial infarction. Within the first few hours after the infarct, a complex cascade of events is initiated in the myocardium manifesting itself clinically in disproportionate thinning and dilation of the infarct region accompanied by distortion in form and function of the entire heart, figure 1. As r
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Fomovsky, Gregory M., and Jeffrey W. Holmes. "Evolution of Scar Mechanical Properties During Myocardial Infarct Healing in Rat." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176422.

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The mechanics of healing myocardial infarcts are an important determinant of post-infarction left ventricular (LV) function and remodeling. Large animal infarct models are well studied; healing infarct scars have been shown to be mechanically and structurally anisotropic [1], and this anisotropy may help preserve LV function during some stages of healing [2]. At the same time, it has been suggested that the rat model of myocardial infarction is more similar to humans in the range of infarct sizes and observed LV dysfunction [3]. However, in the rat model, infarct mechanics and their effect on
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Richardson, William J., and Jeffrey W. Holmes. "Do Infarcts Really Expand or Compact? Relationship Between Changing Material Properties and Apparent Infarct Remodeling." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14411.

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Myocardial infarction (MI) is a leading cause of mortality and morbidity with over 600,000 new Americans suffering an MI each year [1]. Following infarction, damaged muscle is gradually replaced by collagenous scar tissue, while undamaged (remote) myocytes remodel due to altered load. Remodeling of both the infarcted and remote myocardium are important determinants of cardiac function and the risk of progression to heart failure.
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Zhang, Song, John A. Crow, Robert C. Cooper, et al. "Detection of Myocardial Fiber Disruption in Artificial Lesions With 3D DT-MRI Tract Models." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-193121.

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In the United States, it is estimated that in 2008 approximately 1.2 million people will suffer a new or recurrent myocardial infarction. In 2005, the latest full year for which statistics are available, 16 million Americans (7.3% of the population) had some form of coronary heart disease. Loss of myocardium as a result of myocardial infarction increases wall stress locally and globally and triggers adaptive responses at the molecular, cellular, and tissue levels. These adaptive responses can lead to left ventricular dilation and congestive heart failure. Accurate non-invasive evaluation of my
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Zhang, Pei, Tieluo Li, Katrina Williams, et al. "Analysis of Infarct Size on Myocardial Infarction Remodeling." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53117.

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In the United States, over one million patients sustain left ventricular (LV) injury after myocardial infarction (MI). LV remodeling is an adaptive process of hypertrophy that includes infarct expansion, reduced contractility and LV dilation. Progressive enlargement of non-ischemic, hypocontractile myocardium in the adjacent zone (AZ) following the transmural MI has been identified clinically, which contributes to the development post-MI cardiomyopathy in patients. Till now, how the early regional biomechanical and cellular changes, particularly in the AZ, relate to LV remodeling process remai
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Masithulela, Fulufhelo. "Analysis of Passive Filling With Fibrotic Myocardial Infarction." In ASME 2015 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2015. http://dx.doi.org/10.1115/imece2015-50003.

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Cardiovascular diseases account for one third of all deaths worldwide, more than 33% of which are related to ischemic heart disease, involving a myocardial infarction (MI). Following myocardial infarction, the injured region and ventricle undergo structural changes which are thought to be caused by elevated stresses and reduction of strains in the infarcted wall. The fibrotic phase is defined as the period when the amount of new collagen and number of fibroblasts rapidly increase in the infarcted tissue. We studied through finite element analysis the mechanics of the infarcted and remodeling r
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Reports on the topic "Myocardial Infarctio"

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Li, Xiao, Fayang Ling, Wenchuan Qi, et al. Preclinical Evidence of Acupuncture on infarction size of Myocardial ischemia: A Systematic Review and Meta-Analysis of Animal Studies. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.6.0044.

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Review question / Objective: Whether acupuncture is effective for infarction size on myocardial ischemia rat models. Condition being studied: Myocardial ischemia is a typical pathological condition of coronary heart disease (CHD), which has been a global issue with high incidence and mortality. Myocardial infarction caused by myocardial ischemia leads to cardiac dysfunction, and the size of myocardial infarction also determines the recovery and prognosis of cardiac function. Acupuncture, a long history of traditional Chinese medicine, is widely used to treat symptoms like thoracalgia and palpi
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Dejong, Marla J., Kyungeh An, Candace C. Cherrington, and Debra K. Moser. Predictors of Symptom Appraisal for Patients with Acute Myocardial Infarction. Defense Technical Information Center, 2004. http://dx.doi.org/10.21236/ada427523.

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Crowley, James P., C. R. Valeri, and Joseph Chazan. Myocardial Infarction and Transfusion Requirements in Transfusion Dependent Anemic Patients. Defense Technical Information Center, 1990. http://dx.doi.org/10.21236/ada360239.

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Naydenov, Stefan, Nikolay Runev, Emil Manov, Nadya Naydenova, Mikhail Matveev, and Plamen Krastev. Diagnostic Potential of Signal-Averaged Orthogonal Electrocardiography in Acute Myocardial Infarction. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, 2021. http://dx.doi.org/10.7546/crabs.2021.02.16.

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Liu, Sihan, Shuo Han, Yingzi Lin, and Yingzhe Jin. The obesity paradox in patients with ST-segment elevation myocardial infarction. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2021. http://dx.doi.org/10.37766/inplasy2021.6.0015.

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Li, Peng, Na jia, Bing Liu, and Qing He. Effect of cardiac shock wave therapy on adverse cardiovascular event for patients with coronary artery disease: an updated systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.1.0103.

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Review question / Objective: We have previously demonstrated that cardiac shock wave therapy (CSWT) effectively improves myocardial perfusion in patients with coronary artery disease (CAD). In this study, we want to address whether CSWT could decrease the risk of adverse cardiovascular events in CAD patients unsuitable for revascularization. Eligibility criteria: Trials are considered eligible if they meet these criteria: (1) patients included are diagnosed as refractory angina or ischemic heart failure; (2) the study i a randomized controlled trial (RCT) or a prospective cohort study; (3) int
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Zhang, John Q. Post-Myocardial Infarction and Exercise Training on Myosin Heavy Chain and Cardiac Function. Science Repository, 2019. http://dx.doi.org/10.31487/j.jicoa.2019.01.08.

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Yin, Ruoyun, Fan Zhang, Zhaoya Fan, Lei Tang, and Yuan Yang. Association between Abacavir Use With Myocardial Infarction: A systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2020. http://dx.doi.org/10.37766/inplasy2020.10.0054.

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De Jong, Marla J. A Cross-Sectional Examination of Changes in Anxiety Early After Acute Myocardial Infarction. Defense Technical Information Center, 2003. http://dx.doi.org/10.21236/ada416443.

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Sánchez Fernández, Eduardo, and Maria D. Mauricio. Histone Deacetylase Inhibitors as a Promising Treatment Against Myocardial Infarction: A Systematic Review. Fundación Avanza, 2025. https://doi.org/10.60096/fundacionavanza/11552025.

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