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Relevant bibliographies by topics / Myasthenic

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Academic literature on the topic 'Myasthenic'

Author: Grafiati

Published: 4 June 2021

Last updated: 17 February 2022

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Journal articles on the topic "Myasthenic"

1

van Lunteren, Erik, Michelle Moyer, and Henry J. Kaminski. "Adverse effects of myasthenia gravis on rat phrenic diaphragm contractile performance." Journal of Applied Physiology 97, no. 3 (2004): 895–901. http://dx.doi.org/10.1152/japplphysiol.01266.2003.

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Myasthenia gravis has variable effects on the respiratory system, ranging from no abnormalities to life-threatening respiratory failure. Studies characterized diaphragm muscle contractile performance in rat autoimmune myasthenia gravis. Rats received monoclonal antibody that recognizes acetylcholine receptor determinants (or inactive antibody); 3 days later, phrenic nerve and diaphragm were studied in vitro. Myasthenic rats segregated into two groups, those with normal vs. impaired limb muscle function when tested in intact animals (“mild” and “severe” myasthenic). Baseline diaphragm twitch fo

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Saraiva, Paulo A. P., José Lamartine de Assis, and Paulo E. Marchiori. "Evaluation of the respiratory function in myasthenia gravis: an important tool for clinical feature and diagnosis of the disease." Arquivos de Neuro-Psiquiatria 54, no. 4 (1996): 601–7. http://dx.doi.org/10.1590/s0004-282x1996000400009.

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Myasthenic gravis may affect both inspiratory and expiratory muscles. Respiratory involvement occurred in almost all patients with myasthenia gravis in all clinical forms of the disease: 332 lung function tests done in 324 myasthenic patients without respiratory symptoms (age 34.6 ± 18.3 years) were examined. Lung volumes analysis showed that all the patients of both sexes with generalized or ocular myasthenia gravis showed "myasthenic pattern". Male patients with "ocular" form only presented the "myasthenic pattern" with lung impairment and had, from the lung function point of view, a more be

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Kruglyakov, N. M., D. G. Levitova, G. I. Bagzhanov, et al. "Application of the Technique of Extracorporeal Membrane Oxygenation in a Patient With Respiratory Distress Syndrome Associated With Myasthenia Gravis." Russian Sklifosovsky Journal "Emergency Medical Care" 10, no. 2 (2021): 393–400. http://dx.doi.org/10.23934/2223-9022-2021-10-2-393-400.

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Myasthenia gravis is an autoimmune neuromuscular disease characterized by pathologically rapid fatigue of striated muscles [1]. The main symptom of myasthenia gravis is the presence of pathological muscle weakness with involvement of the ocular, bulbar and skeletal muscles in the pathological process. The provoking factors for the development of myasthenia gravis can be infectious diseases, surgery, drugs [2, 3]. The main danger is represented by myasthenic and cholinergic crises, which are characterized by a severe course and high mortality; therefore, the problems of treating myasthenia grav

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Schroeter, Michael, Günther Thayssen, and Julia Kaiser. "Myasthenia Gravis – Exacerbation and Crisis." Neurology International Open 02, no. 01 (2018): E10—E15. http://dx.doi.org/10.1055/s-0043-118441.

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AbstractMyasthenic exacerbation and crisis are most critical incidences in myasthenia gravis. Even nowadays myasthenic crisis is a life-threatening condition, with a lethality of 2–3%. We review means of avoiding myasthenic exacerbation and crisis, elaborate on red flags and how to establish highly-active therapy in a timely manner. This includes the reasonable use of cholinesterase inhibitors, immunoadsorption or plasma exchange, as well as immunoglobulins and steroids. Immunosuppressive agents and monoclonal antibody therapy add to the therapeutic options.Intensive care of myasthenic patient

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Job, Anand, R. Raman, and C. Gnanamuthu. "Laryngeal stridor in myasthenia gravis." Journal of Laryngology & Otology 106, no. 7 (1992): 633–34. http://dx.doi.org/10.1017/s0022215100120390.

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AbstractTwo patients with laryngeal stridor secondary to myasthenia gravis are reported. The cause of bilateral abductor weakness in myasthenia is discussed; anticipation of the stridor in myasthenic patients is highlighted.

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Anand, Uttara Swati, Stalin Viswanathan, and Jayanthi Arulneyam. "Pulmonary Edema in Myasthenic Crisis." Case Reports in Critical Care 2013 (2013): 1–3. http://dx.doi.org/10.1155/2013/863620.

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We report a previously asymptomatic 50-year-old lady who came with myasthenic crisis as initial presentation of myasthenia gravis. She developed pulmonary edema following intravenous immunoglobulin administration and had ischemic changes in ECG and left ventricular dysfunction on echocardiography. She improved with diuretics, dobutamine, and fluid restriction alone. This is the first report in English-language medical literature describing the association between myasthenic crisis and likely takotsubo cardiomyopathy-related pulmonary edema following intravenous immunoglobulin administration.

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Itoh, Hironori, Keizo Shibata, Masahiro Yoshida, and Ken Yamamoto. "Neuromuscular Monitoring at the Orbicularis Oculi May Overestimate the Blockade in Myasthenic Patients." Anesthesiology 93, no. 5 (2000): 1194–97. http://dx.doi.org/10.1097/00000542-200011000-00010.

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Background In most publications about myasthenia, monitoring neuromuscular blockade during anesthesia is recommended. In healthy patients, the relation of blockade between muscles has been established, but there is little information about the relation in myasthenic patients. Our objective was to investigate whether the relation between the orbicularis oculi and adductor pollicis muscles is the same in healthy patients and myasthenic patients. Methods After anesthesia was induced with 4-6 mg/kg thiopental and 2 microg/kg fentanyl, followed by 2% sevoflurane and 60% nitrous oxide in oxygen, 10

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Zaloum, A., JR Falet, A. Elkrief, and C. Chalk. "P.022 Myasthenia gravis following dabrafenib and trametinib for metastatic melanoma." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 46, s1 (2019): S19. http://dx.doi.org/10.1017/cjn.2019.122.

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Background: Inhibitors of BRAF and MEK, enzymes in the mitogen-activated protein kinase (MAPK) pathway, are now widely used in the treatment of metastatic melanoma. We report a case of acetylcholine receptor (AChR) antibody-positive myasthenia gravis developing after exposure to dabrafenib, a BRAF inhibitor, and trametinib, a MEK inhibitor. Methods: A 68-year-old man presented with dysarthria, dysphagia, cough, dyspnea, and fever. Examination revealed fatigable ptosis and proximal muscle weakness. He had started dabrafenib and trametinib for metastatic melanoma two weeks prior. He was diagnose

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Souza, Paulo Victor Sgobbi de, Gabriel Novaes de Rezende Batistella, Valéria Cavalcante Lino, Wladimir Bocca Vieira de Rezende Pinto, Marcelo Annes, and Acary Souza Bulle Oliveira. "Clinical and genetic basis of congenital myasthenic syndromes." Arquivos de Neuro-Psiquiatria 74, no. 9 (2016): 750–60. http://dx.doi.org/10.1590/0004-282x20160106.

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ABSTRACT Neuromuscular junction disorders represent a wide group of neurological diseases characterized by weakness, fatigability and variable degrees of appendicular, ocular and bulbar musculature involvement. Its main group of disorders includes autoimmune conditions, such as autoimmune acquired myasthenia gravis and Lambert-Eaton syndrome. However, an important group of diseases include congenital myasthenic syndromes with a genetic and sometimes hereditary basis that resemble and mimick many of the classic myasthenia neurological manifestations, but also have different presentations, which

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Benatar, M. "Myasthenia Gravis and Myasthenic Disorders." Neurology 81, no. 1 (2013): 99. http://dx.doi.org/10.1212/wnl.0b013e318297ef4d.

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Dissertations / Theses on the topic "Myasthenic"

1

Carr, A. S. "An epidemiological study of myasthenia gravis and congenital myasthenic syndromes in Northern Ireland." Thesis, Queen's University Belfast, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.546021.

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Abdelgany, Amr. "Gene therapy for congenital myasthenic syndromes." Thesis, University of Oxford, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.441062.

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Finlayson, Sarah E. "Congenital myasthenic syndromes : current diagnostic and therapeutic aspects." Thesis, University of Oxford, 2014. http://ora.ox.ac.uk/objects/uuid:5e08ab86-8a20-48b3-86b9-683eb8b2c6e4.

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Zoltowska, Katarzyna Marta. "Novel pathogenic mechanisms of myasthenic disorders and potential therapeutic approaches." Thesis, University of Oxford, 2014. http://ora.ox.ac.uk/objects/uuid:e817f50a-0318-4944-bf67-773af523c4c3.

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Congenital myasthenic syndrome (CMS) and myasthenia gravis (MG) are, respectively, inherited or autoimmunological disorders caused by aberrant neuromuscular transmission, which manifests as fatiguable muscle weakness. A novel subtype of CMS, resulting from mutations in GFPT1 and characterised by a limb girdle pattern of muscle weakness, has been described. The gene encodes L glutamine:D fructose-6-phosphate amidotransferase 1 (GFAT1) – a key rate limiting enzyme in the hexosamine biosynthetic pathway, providing building blocks for glycosylation of proteins and lipids. The research focused on t

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Cheung, Jonathan Yu. "Pathogenic mechanisms of RAPSN mutations in congenital myasthenic syndromes." Thesis, University of Oxford, 2015. https://ora.ox.ac.uk/objects/uuid:c343ca03-563e-4b4a-9e35-aac09bfc5ea7.

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Rapsyn is essential for clustering acetylcholine receptors (AChR) at the neuromuscular junction (NMJ). Congenital myasthenic syndrome (CMS) due to RAPSN mutations compromises neuromuscular transmission through a deficiency of AChR at the NMJ. RAPSN-CMS is autosomal recessive, and most patients harbour a common mutation p.Asn88Lys. A definitive genetic diagnosis for patients who do not carry the p.Asn88Lys allele can be challenging. In this thesis 10 novel variants in RAPSN are shown to impair AChR clustering in vitro, and are thus defined as pathogenic. The properties of RAPSN mutations p.Val4

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Issop, Yasmin. "A GFPT1 deficient mouse model of congenital myasthenic syndrome." Thesis, University of Newcastle upon Tyne, 2017. http://hdl.handle.net/10443/3902.

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Congenital myasthenic syndromes (CMS) are inherited disorders characterised by fatigable muscle weakness resulting from impaired transmission at the neuromuscular junction (NMJ). CMS occur due to mutations in genes encoding proteins responsible for maintaining the structure and function of the NMJ. Glutamine-fructose-6-phosphate transaminase 1 (GFPT1) is the rate-limiting enzyme in the hexosamine biosynthetic pathway which yields precursors required for protein and lipid glycosylation. Mutations in GFPT1 and genes downstream of this pathway are pathogenic for CMS. One hypothesis is that hypogl

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Chaouch, Amina. "The clinical and genetic characteristics of congenital myasthenic syndromes." Thesis, University of Newcastle upon Tyne, 2014. http://hdl.handle.net/10443/2748.

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Congenital myasthenic syndromes (CMS) are inherited disorders in which the safety margin of the neuromuscular transmission is compromised. The clinical hallmark of CMS is fatigable weakness. To date, nineteen genes have been implicated in causing this disorder, with most mutations located in postsynaptic proteins. Nevertheless, a great proportion of patients remains with no molecular diagnosis and cannot therefore access optimum therapy. In this thesis, each topic is summarized in one chapter that corresponds to one or part of a selected journal publication, or a book chapter. Chapter 1 is ded

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Childs, Lisbeth Ann. "The effects of myasthenic serum on skeletal muscle cells in culture." Thesis, University of Bath, 1985. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.484407.

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Rat muscle cells were grown in culture for use as an experimental model in which to study the myolytic effects of myasthenic serum in vitro. Use was made of a procedure which depends upon the selective uptake of tritium-labelled carnitine by cultured myotubes, loss of which can be monitored following cytolytic damage. The studies demonstrated that heat-inactivated myasthenic serum samples caused myotube-specific lysis in a manner that was dependent on the addition of complement. The concentration and activity of the complement source was shown to be a major factor in detecting myotoxicity. Usi

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Nichols, Philip Paul. "Transcriptional regulation of the human nicotinic acetylcholine receptor." Thesis, University of Oxford, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.326016.

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Pinto, Ashwin. "Specificity of autoantibodies in Lambert-Eaton myasthenic syndrome for neuronal calcium channels." Thesis, University of Oxford, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.342539.

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Books on the topic "Myasthenic"

1

Jones, Dominic. Antibodies against the voltage-gated calcium channel in Lambert-Eaton myasthenic syndrome. Oxford Brookes University, 2000.

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2

Baets, M. H. De, 1950-, Oosterhuis, Henricus Josephus Gerardus Hubertus., and Toyka K. V. 1945-, eds. Myasthenia gravis. Karger, 1988.

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3

Christadoss, Premkumar, ed. Myasthenia Gravis. Springer Netherlands, 2000. http://dx.doi.org/10.1007/978-94-011-4060-7.

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Giovanni, Giacalone. Contributo della pranoterapia nella myasthenia gravis =: Pranotherapy help in the myasthenia gravis. Edizioni G. & G., 1986.

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service), SpringerLink (Online, ed. Myasthenia Gravis and Related Disorders. Humana Press, 2009.

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Kaminski, Henry J. Myasthenia Gravis and Related Disorders. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-156-7.

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Kaminski, Henry J., ed. Myasthenia Gravis and Related Disorders. Humana Press, 2003. http://dx.doi.org/10.1007/978-1-59259-341-5.

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Kaminski, Henry J., and Linda L. Kusner, eds. Myasthenia Gravis and Related Disorders. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-73585-6.

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Kaminski, Henry J. Myasthenia Gravis and Related Disorders. Humana Press, 2002. http://dx.doi.org/10.1385/1592593410.

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Giacalone, Giovanni. Pranotherapy help in the myasthenia gravis. [s.n.], 1986.

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Book chapters on the topic "Myasthenic"

1

Swash, Michael, and Martin S. Schwartz. "Myasthenia Gravis and Other Myasthenic Syndromes." In Neuromuscular Diseases. Springer London, 1997. http://dx.doi.org/10.1007/978-1-4471-3834-1_12.

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2

Swash, Michael, and Martin Schwartz. "Myasthenia Gravis and Other Myasthenic Syndromes." In Neuromuscular Diseases. Springer London, 1988. http://dx.doi.org/10.1007/978-1-4471-3526-5_12.

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3

Oette, Mark, Marvin J. Stone, Hendrik P. N. Scholl, et al. "Myasthenic Syndrome." In Encyclopedia of Molecular Mechanisms of Disease. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_9280.

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Janjua, Nazli, and Stephan A. Mayer. "Myasthenic Crisis." In Critical Care Neurology and Neurosurgery. Humana Press, 2004. http://dx.doi.org/10.1007/978-1-59259-660-7_26.

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Bensard, Denis D., Philip F. Stahel, Jorge Cerdá, et al. "Myasthenic Crisis." In Encyclopedia of Intensive Care Medicine. Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-00418-6_1910.

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Toyka, Klaus V., Wolfgang Müllges, and Daniel F. Hanley. "Myasthenia gravis and Lambert-Eaton Myasthenic Syndrome." In Neurocritical Care. Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-87602-8_71.

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Muley, Suraj A., and Christopher M. Gomez. "Congenital Myasthenic Syndromes." In Myasthenia Gravis and Related Disorders. Humana Press, 2003. http://dx.doi.org/10.1007/978-1-59259-341-5_14.

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Beeson, David. "Congenital Myasthenic Syndromes." In Myasthenia Gravis and Related Disorders. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-73585-6_16.

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Kaminski, Henry J., and Robert L. Ruff. "The Myasthenic Syndromes." In Molecular Biology of Membrane Transport Disorders. Springer US, 1996. http://dx.doi.org/10.1007/978-1-4613-1143-0_28.

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Engel, A. G., K. Ohno, and A. A. Stans. "Congenital Myasthenic Syndromes." In Monographs in Clinical Neuroscience. KARGER, 2000. http://dx.doi.org/10.1159/000061631.

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Conference papers on the topic "Myasthenic"

1

Pickens, A. W., and M. C. Miles. "Nivolumab Toxicity: Myasthenic Crisis and Myocarditis." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a4837.

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Ashhurst, Jasmine, Rami Haddad, and Rob Zielinski. "062 Pembrolizumab induced lambert-eaton myasthenic syndrome." In ANZAN Annual Scientific Meeting 2021 Abstracts. BMJ Publishing Group Ltd, 2021. http://dx.doi.org/10.1136/bmjno-2021-anzan.62.

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Lochmüller, H. "Update on inherited neuromuscular transmission disorders (congenital myasthenic syndromes)." In 24. Kongress des Medizinisch-Wissenschaftlichen Beirates der Deutschen Gesellschaft für Muskelkranke (DGM) e.V. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1684994.

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Rizzo, A. N., A. Gupta, P. Hume, and T. M. Bull. "Lambert Eaton Myasthenic Syndrome Presenting as Hypoventilation-Induced Right Heart Dysfunction." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a3768.

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Henrich, Maria, Burcin Ceylan, Adela Marina, Angela Abicht, Heike Kölbel, and Ulrike Schara. "P 409. The Noonan’s Syndrome as a Differential Diagnosis of a Congenital Myasthenic Syndrome." In Abstracts of the 44th Annual Meeting of the Society for Neuropediatrics. Georg Thieme Verlag KG, 2018. http://dx.doi.org/10.1055/s-0038-1675966.

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Moreno, FJ García, MS Pernia Lopez, and M. Sanjurjo Saez. "3PC-027 Amifampridine and pyridostigmine hard capsules for treatment of congenital myasthenic syndromes: a case report." In Abstract Book, 23rd EAHP Congress, 21st–23rd March 2018, Gothenburg, Sweden. British Medical Journal Publishing Group, 2018. http://dx.doi.org/10.1136/ejhpharm-2018-eahpconf.79.

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Wu, D., D. Condit, and J. C. Nascimento. "Pembrolizumab Induced Myasthenia Gravis." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a2408.

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Malladi, S., A. Aburahma, L. Rashdan, and K. Ranabhat. "The Many Faces of Myasthenia Gravis." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a2296.

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Liang, T., M. I. Boulos, B. J. Murray, S. Krishnan, H. Katzberg, and K. Umapathy. "Detection of myasthenia gravis using electrooculography signals." In 2016 38th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2016. http://dx.doi.org/10.1109/embc.2016.7590845.

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Ceylan, Kenan Can, Seyda Ors Kaya, Ozgur Ozturk, Yasar Zorlu, and Muhtesem Gedizlioglu. "Minimally Invasive Thymectomy in Non-Thymomatous Myasthenia Gravis Cases." In ERS International Congress 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/1393003.congress-2017.pa2056.

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Reports on the topic "Myasthenic"

1

Asenova, Asya, and Veneta Bojinova. Juvenile Myasthenia Gravis - Clinical Course and Serologic Tests. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, 2020. http://dx.doi.org/10.7546/crabs.2020.12.17.

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Xie, Rongfang, Liting Liu, Ruiqi Wang, and Chunhua Huang. Traditional Chinese medicine for Myasthenia gravis: study protocol for a network meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2020. http://dx.doi.org/10.37766/inplasy2020.6.0049.

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Huang, Chunhua, Rongfang Xie, Liting Liu, and Ruiqi Wang. Efficacy and safety of Buzhong-Yiqi Decoction for Myasthenia gravis：study protocol for a systematic review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2020. http://dx.doi.org/10.37766/inplasy2020.5.0080.

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Peng, Yuxuan, Lu Li, Yuting Pan, et al. External treatment of Traditional Chinese Medicine for myasthenia gravis: a protocol for systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2021. http://dx.doi.org/10.37766/inplasy2021.1.0083.

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Li, Lu, Jiachun Lu, BiBo Lu, et al. Tonifying Spleen and Replenishing Kidney method of Traditional Chinese Medicine for myasthenia gravis: a protocol for systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2021. http://dx.doi.org/10.37766/inplasy2021.1.0097.

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Surgery to remove the thymus gland improves weakness for people with myasthenia gravis. National Institute for Health Research, 2016. http://dx.doi.org/10.3310/signal-000310.

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