Academic literature on the topic 'Mutagenesis'
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Journal articles on the topic "Mutagenesis"
Gupta, Neha, Megha Malu, and Aparajita Beera. "Mutagenesis." International Journal of Oral Care & Research 5, no. 3 (2017): 252–56. http://dx.doi.org/10.5005/jp-journals-10051-0109.
Full textNatarajan, A. T. "Mutagenesis." Cytogenetic and Genome Research 81, no. 2 (1998): 159–64. http://dx.doi.org/10.1159/000015017.
Full textSymonds, Neville. "Anticipatory mutagenesis?" Nature 337, no. 6203 (January 1989): 119–20. http://dx.doi.org/10.1038/337119a0.
Full textNovak, Kris. "Meningococcal mutagenesis." Nature Biotechnology 18, no. 11 (November 2000): 1129. http://dx.doi.org/10.1038/81060.
Full textWeitzman, Jonathan. "Minos mutagenesis." Genome Biology 1 (2000): spotlight—20001128–01. http://dx.doi.org/10.1186/gb-spotlight-20001128-01.
Full textWeitzman, Jonathan B. "Mouse mutagenesis." Genome Biology 3 (2002): spotlight—20020226–01. http://dx.doi.org/10.1186/gb-spotlight-20020226-01.
Full textMurli, Sumati, and Graham C. Walker. "SOS mutagenesis." Current Opinion in Genetics & Development 3, no. 5 (October 1993): 719–25. http://dx.doi.org/10.1016/s0959-437x(05)80089-9.
Full textVainio, Harri. "Environmental mutagenesis." Molecular Medicine Today 2, no. 9 (September 1996): 370–71. http://dx.doi.org/10.1016/s1357-4310(96)80010-4.
Full textAntalis, T. "Serpin mutagenesis." Methods 32, no. 2 (February 2004): 130–40. http://dx.doi.org/10.1016/s1046-2023(03)00204-4.
Full textFawcett, H. H. "Environmental mutagenesis." Journal of Hazardous Materials 10, no. 1 (February 1985): 152. http://dx.doi.org/10.1016/0304-3894(85)80013-3.
Full textDissertations / Theses on the topic "Mutagenesis"
Martin, Stephen Lewis. "Novel methods for mutagenesis." Thesis, University of Oxford, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.302789.
Full textEnnis, Don Gregory. "Genetics of SOS mutagenesis." Diss., The University of Arizona, 1988. http://hdl.handle.net/10150/184602.
Full textPorto, Marília de Paula [UNESP]. "Ação moduladora do citral e eugenol em eventos genéticos em magrófagos murinos in vitro." Universidade Estadual Paulista (UNESP), 2012. http://hdl.handle.net/11449/92460.
Full textConselho Nacional de Desenvolvimento Científico e Tecnológico (CNPq)
Coordenação de Aperfeiçoamento de Pessoal de Nível Superior (CAPES)
Fundação de Amparo à Pesquisa do Estado de São Paulo (FAPESP)
Devido a propriedades terapêuticas, várias plantas e seus constituintes químicos vêm sendo muitas vezes utilizados como o primeiro recurso para o tratamento de diversas doenças. Nesse contexto, compostos isolados de plantas têm sido alvos de inúmeros estudos que avaliam, além da atividade, seus possíveis mecanismos de ação. Dentre os compostos com potencial quimioprotetor, o citral e o eugenol merecem atenção devido suas estruturas químicas de monoterpeno e de composto fenólico, respectivamente, e por seus potenciais anti-inflamatório, antiparasitário e antioxidante. Considerando que mutação no DNA pode ser a primeira etapa de várias doenças, e que lesões induzidas nessa molécula podem ser prevenidas ou moduladas por compostos naturais, este estudo objetivou avaliar, pelo teste do cometa, o potencial genotóxico do citral (25, 50 e 100 Tg/mL) e do eugenol (0,31, 0,62, 1,24 e 2,48 Tg/mL), após diferentes tempos de tratamento (6, 10, 24 e 30 h) e, também, seus possíveis efeitos moduladores sobre danos induzidos no DNA pela doxorrubicina (DOX), em diferentes protocolos de tratamento (pré, pós e simultaneamente à DOX) e momentos de análise (12, 24 e 36 h), em macrófagos peritoneais de camundongos. Além disso, foi avaliado o potencial toxicogenômico do citral e do eugenol por meio da modulação da expressão dos genes NF-kB1, COX-2 e TNF-α (relacionados a processos inflamatórios) em macrófagos ativados ou não por lipopolissacarideo de bactéria (LPS). Os resultados mostraram que ambos os compostos apresentaram potencial genotóxico. No caso do citral, a genotoxicidade foi observada para as duas maiores concentrações, mas apenas no tempo de 6h; para o eugenol, o aumento de danos no DNA foi detectado para todas as concentrações, em pelo menos um momento de análise. Com relação ao potencial...
Because of the therapeutic properties, several plants and their chemical constituents have been used for treatment of various diseases. Therefore, isolated compounds from plants have been targets of numerous studies that evaluate their activity and mechanisms of action. Among compounds with chemopreventive potential, citral and eugenol have gain attention because of their chemical structures, monoterpene and phenol,respectively, and for their anti-inflammatory, antioxidant and antiparasitic potentials. Since DNA mutation is the first step for some diseases, and since the lesions induced in this molecule can be prevented or modulated by natural compounds, aim of the present study was first to evaluate the genotoxic potential of citral (25, 50 and 100 Tg/mL) and eugenol (0.31, 0.62, 1.24 and 2.48 Tg/mL) at different times after exposure (6, 10, 24 and 30 h), and then, their ability to modulate DNA damage induced by doxorubicin (DOX) at different treatment protocols (pre, post and simultaneous with DOX) and times (12, 24 and 36 h) in mice peritoneal macrophages. In addition, the toxicogenomic potential of citral and eugenol by modulating the expression of NF-KB1, COX-2 and TNF-α (related to inflammatory processes) genes in macrophages activated or not by bacterial lipopolysaccharide (LPS) was also investigated. The results showed that both compounds have genotoxic potential. In the case of citral, genotoxicity was observed for the two major concentrations, but only 6h after the exposure. For eugenol, increased DNA damage was detected for all concentrations, in at least one moment of analysis. Related to the antigenotoxicity, both citral and eugenol presented protective effects at different concentrations and treatment protocols, and the more effective activities were detected at simultaneous and pre-treatment... (Complete abstract click electronic access below)
Porto, Marília de Paula. "Ação moduladora do citral e eugenol em eventos genéticos em magrófagos murinos in vitro /." Botucatu : [s.n.], 2012. http://hdl.handle.net/11449/92460.
Full textCoorientador: Glenda Nicioli da Silva
Banca: Luís Fernando Barbisan
Banca: Denise Crispim Tavares
Resumo: Devido a propriedades terapêuticas, várias plantas e seus constituintes químicos vêm sendo muitas vezes utilizados como o primeiro recurso para o tratamento de diversas doenças. Nesse contexto, compostos isolados de plantas têm sido alvos de inúmeros estudos que avaliam, além da atividade, seus possíveis mecanismos de ação. Dentre os compostos com potencial quimioprotetor, o citral e o eugenol merecem atenção devido suas estruturas químicas de monoterpeno e de composto fenólico, respectivamente, e por seus potenciais anti-inflamatório, antiparasitário e antioxidante. Considerando que mutação no DNA pode ser a primeira etapa de várias doenças, e que lesões induzidas nessa molécula podem ser prevenidas ou moduladas por compostos naturais, este estudo objetivou avaliar, pelo teste do cometa, o potencial genotóxico do citral (25, 50 e 100 Tg/mL) e do eugenol (0,31, 0,62, 1,24 e 2,48 Tg/mL), após diferentes tempos de tratamento (6, 10, 24 e 30 h) e, também, seus possíveis efeitos moduladores sobre danos induzidos no DNA pela doxorrubicina (DOX), em diferentes protocolos de tratamento (pré, pós e simultaneamente à DOX) e momentos de análise (12, 24 e 36 h), em macrófagos peritoneais de camundongos. Além disso, foi avaliado o potencial toxicogenômico do citral e do eugenol por meio da modulação da expressão dos genes NF-kB1, COX-2 e TNF-α (relacionados a processos inflamatórios) em macrófagos ativados ou não por lipopolissacarideo de bactéria (LPS). Os resultados mostraram que ambos os compostos apresentaram potencial genotóxico. No caso do citral, a genotoxicidade foi observada para as duas maiores concentrações, mas apenas no tempo de 6h; para o eugenol, o aumento de danos no DNA foi detectado para todas as concentrações, em pelo menos um momento de análise. Com relação ao potencial... (Resumo completo, clicar acesso eletrônico abaixo)
Abstract: Because of the therapeutic properties, several plants and their chemical constituents have been used for treatment of various diseases. Therefore, isolated compounds from plants have been targets of numerous studies that evaluate their activity and mechanisms of action. Among compounds with chemopreventive potential, citral and eugenol have gain attention because of their chemical structures, monoterpene and phenol,respectively, and for their anti-inflammatory, antioxidant and antiparasitic potentials. Since DNA mutation is the first step for some diseases, and since the lesions induced in this molecule can be prevented or modulated by natural compounds, aim of the present study was first to evaluate the genotoxic potential of citral (25, 50 and 100 Tg/mL) and eugenol (0.31, 0.62, 1.24 and 2.48 Tg/mL) at different times after exposure (6, 10, 24 and 30 h), and then, their ability to modulate DNA damage induced by doxorubicin (DOX) at different treatment protocols (pre, post and simultaneous with DOX) and times (12, 24 and 36 h) in mice peritoneal macrophages. In addition, the toxicogenomic potential of citral and eugenol by modulating the expression of NF-KB1, COX-2 and TNF-α (related to inflammatory processes) genes in macrophages activated or not by bacterial lipopolysaccharide (LPS) was also investigated. The results showed that both compounds have genotoxic potential. In the case of citral, genotoxicity was observed for the two major concentrations, but only 6h after the exposure. For eugenol, increased DNA damage was detected for all concentrations, in at least one moment of analysis. Related to the antigenotoxicity, both citral and eugenol presented protective effects at different concentrations and treatment protocols, and the more effective activities were detected at simultaneous and pre-treatment... (Complete abstract click electronic access below)
Mestre
Silva, Ana Carolina Buzinari da [UNESP]. "Análise de uma biblioteca de mutantes de Xanthomonas citri subsp. citri quanto à patogenicidade." Universidade Estadual Paulista (UNESP), 2014. http://hdl.handle.net/11449/121954.
Full textO estudo da interação planta-patógeno é de grande importância para o entendimento do cancro cítrico, justificando assim a busca por genes que estejam ligados a patogenicidade e virulência em Xanthomonas citri subsp. citri (Xac), agente causal dessa doença. Neste estudo, foi realizada mutagênese aleatória por inserção do transposon EZ-Tn5 in vitro no genoma da Xac. Obteve-se 8000 mutantes onde 292 foram conduzidos em ensaio experimental in planta. Cinco mutantes expressaram sintomatologia alterada, dois com ausência total de sintomas e três com leve hiperplasia. A análise da sequência dos genes onde se inseriu o transposon indicam mutações nos genes purF, yapH, oar, um gene que codifica uma proteína hipotética (XAC 0196), e na região entre os genes pobB (XAC0362) e glpR (XAC0361). As análises de curvas de crescimento bacteriano in planta demonstraram que, exceto o gene purF, todos os demais podem ser genes envolvidos na patogenicidade de Xac. Dois destes, yapH e oar são descritos como relacionados à adesividade bacteriana, evidenciando que a interferência nesse processo exerce influência direta no sucesso da infecção de Xac. Destaca-se também a importância da identificação de uma proteína hipotética, já que essa apresentou sintomatologia atenuada quando ocorreu a inserção do transposon
The study of plant-pathogen interaction is very important to citrus canker understanding, justifying the search for virulence and pathogenicity related genes in Xanthomonas citri subsp. citri (Xac), causal agent of this disease. In this study, a random mutagenesis by Tn5 transposon insertion into Xac’s genome was performed. Eight thousand mutants were produced and 292 mutants were tested in planta. From those, five mutants expressed altered symptomatology, two showed complete absence of symptoms and three reduced hyperplasia. Gene sequences analysis where transposon was inserted, indicated mutations in purF, yapH and oar genes, in a region that codes for a hypothetical protein (XAC0196), and in a region between pobB (XAC0362) and glpR (XAC0361) genes. Analysis of bacteria growth curve in planta showed that, except for purF gene, all the others genes may be involved in Xac pathogenicity. Two of these genes, yapH and oar, are described as bacterial adhesion related genes, highlighting that interference in this process has direct influence in the Xac infection success. The importance of hypothetical protein identification is emphasized, since it presented attenuated symptomatology when mutated
Terrazas, Peterson Menezes. "Estudo do potencial genotóxico da Gutiferona A em diferentes células de camundongos in vitro /." Botucatu, 2013. http://hdl.handle.net/11449/108542.
Full textBanca: Maria Aparecida Marin Morales
Banca: Cláudia Aparecida Rainho
Resumo: Garcinia achachairu (GAC) é uma planta de origem boliviana que vem sendo utilizada na medicina popular para o tratamento de distúrbios gástricos, reumatismo, inflamações e como cicatrizante. A caracterização fitoquímica do extrato desta planta revelou que, uma benzofenona, a Gutiferona A (GA), é um dos seus compostos majoritários, que segundo estudos recentes, apresenta importante atividade antioxidante e antimicrobiana. Considerando o interesse em se aprofundar as análises do potencial farmacológico da GA e a inexistência de estudos que avaliem a sua toxicidade genética, o presente estudo foi elaborado visando investigar o potencial genotóxico e mutagênico da GA em diferentes células de camundongos in vivo, utilizando alguns dos testes tradicionais na área de mutagênese, como o Ensaio Cometa (EC) para a verificação da genotoxicidade e o Teste do Micronúcleo (TM) para a verificação da mutagenicidade. O experimento foi conduzido com camundongos Suíços albinos machos (Mus musculus) de 12 semanas, divididos em cinco grupos, constituídos cada um por seis animais. O grupo controle negativo recebeu, via gavagem, 0,3 mL de DMSO 1%. O grupo controle positivo, recebeu intraperitonealmente, 80 mg/kg de doxorrubicina. Os grupos tratados receberam, via gavagem, 0,3 mL da GA nas doses de 15, 30 e 60 mg/kg. Para a avaliação da genotoxicidade foi coletado sangue da veia caudal dos camundongos (4 e 24 horas após o tratamento), células do fígado, medula óssea, cérebro e testículos (coletadas 24 horas após o tratamento). Para a avaliação da mutagenicidade, foram coletadas células da medula óssea 24 horas após o tratamento. A citotoxicidade foi avaliada pela contagem de 200 eritrócitos policromáticos (PCE) e normocromáticos (NCE) e determinação de sua razão (PCE/NCE). Na amostra de sangue de 4h, analisadas pelo EC, os resultados obtidos mostraram que nas doses de 30 mg/kg e 60 mg/Kg. A análise ...
Abstract: Garcinia achachairu (GAC) is a native plant from Bolivia that has been used in folk medicine for the treatment of gastric disorders, rheumatism, inflammation and as a healing. The phytochemical characterization of this plant extract revealed that the benzophenone guttiferone A (GA) is one of its major compounds, which according to recent studies, has important antioxidant and antimicrobial activity. Considering the interest in deepening the analysis of the pharmacological potential of GA and the lack of studies assessing its genetic toxicity, the present study was designed in order to investigate the genotoxic and mutagenic effects of GA in different cells of mice in vivo, using some of the traditional tests in the mutagenesis area, the Comet Assay (CA) for genotoxicity evaluation and the Micronucleus Test (MT) for the mutagenicity assessment. The experiment was conducted in Swiss albino male mice (Mus musculus) with 12 weeks, divided into five groups with six animals each. The negative control group received, by oral gavage, 0.3 mL of 1% DMSO. The positive control group received, intraperitoneally, 80 mg/Kg of doxorubicin. The treated groups received 0.3 ml of GA at 15, 30 and 60 mg/kg, by gavage. For the genotoxicity evaluation, blood was collected from the tail vein of the mice (4 and 24 hours after treatment), and liver, bone marrow, brain and testicular cells were collected 24 hours after treatment. For the mutagenicity assessment, bone marrow cells were collected 24 hours after treatment. Cytotoxicity was assessed by scoring 200 consecutive polychromatic (PCE) and normochromatic (NCE) erythrocytes and their ratio (PCE/NCE) determined. For the 4 h blood sample, the results with GA at doses of 30 and 60 mg/kg showed that was a statistically significant increase in DNA damage in comparison to the negative control. For the 24 h blood sample, only 60 mg/kg dose showed significant genotoxicity. The analysis of ther ...
Mestre
Silva, Ana Carolina Buzinari da. "Análise de uma biblioteca de mutantes de Xanthomonas citri subsp. citri quanto à patogenicidade /." Jaboticabal, 2014. http://hdl.handle.net/11449/121954.
Full textCoorientador: Jesus Aparecido Ferro
Banca: Flavia Maria de Souza Carvalho
Banca: Fabrício José Jaciani
Resumo: O estudo da interação planta-patógeno é de grande importância para o entendimento do cancro cítrico, justificando assim a busca por genes que estejam ligados a patogenicidade e virulência em Xanthomonas citri subsp. citri (Xac), agente causal dessa doença. Neste estudo, foi realizada mutagênese aleatória por inserção do transposon EZ-Tn5 in vitro no genoma da Xac. Obteve-se 8000 mutantes onde 292 foram conduzidos em ensaio experimental in planta. Cinco mutantes expressaram sintomatologia alterada, dois com ausência total de sintomas e três com leve hiperplasia. A análise da sequência dos genes onde se inseriu o transposon indicam mutações nos genes purF, yapH, oar, um gene que codifica uma proteína hipotética (XAC 0196), e na região entre os genes pobB (XAC0362) e glpR (XAC0361). As análises de curvas de crescimento bacteriano in planta demonstraram que, exceto o gene purF, todos os demais podem ser genes envolvidos na patogenicidade de Xac. Dois destes, yapH e oar são descritos como relacionados à adesividade bacteriana, evidenciando que a interferência nesse processo exerce influência direta no sucesso da infecção de Xac. Destaca-se também a importância da identificação de uma proteína hipotética, já que essa apresentou sintomatologia atenuada quando ocorreu a inserção do transposon
Abstract: The study of plant-pathogen interaction is very important to citrus canker understanding, justifying the search for virulence and pathogenicity related genes in Xanthomonas citri subsp. citri (Xac), causal agent of this disease. In this study, a random mutagenesis by Tn5 transposon insertion into Xac's genome was performed. Eight thousand mutants were produced and 292 mutants were tested in planta. From those, five mutants expressed altered symptomatology, two showed complete absence of symptoms and three reduced hyperplasia. Gene sequences analysis where transposon was inserted, indicated mutations in purF, yapH and oar genes, in a region that codes for a hypothetical protein (XAC0196), and in a region between pobB (XAC0362) and glpR (XAC0361) genes. Analysis of bacteria growth curve in planta showed that, except for purF gene, all the others genes may be involved in Xac pathogenicity. Two of these genes, yapH and oar, are described as bacterial adhesion related genes, highlighting that interference in this process has direct influence in the Xac infection success. The importance of hypothetical protein identification is emphasized, since it presented attenuated symptomatology when mutated
Mestre
Baeza, Centurión Pablo 1989. "Understanding alternative splicing using deep mutagenesis." Doctoral thesis, Universitat Pompeu Fabra, 2020. http://hdl.handle.net/10803/668749.
Full textEl empalme alternativo es un proceso de la expresión génica en eucariontes en el que los intrones del transcrito se eliminan, dejando únicamente exones para formar un RNAm maduro. Para estudiar los efectos de mutaciones en este proceso, diseñamos una librería de mutantes con las 12 mutaciones (y todas sus combinaciones) que surgieron a lo largo de la evolución enhumanos de un exón alternativo: el exón 6 de FAS. Esto nos permitió estudiar los efectos de cada mutación en miles de contextos genéticos distintos. Descubrimos que la misma mutación puede tener efectos muy diferentes en el empalme de un exón dependiendo de los niveles de inclusión del mismo. Los mayores efectos se observan en exones con niveles intermedios de inclusión, mientras que los menores efectos ocurren en exones con niveles de inclusión muy altos o muy bajos. Tras mutagenizar dos exones constitutivos, confirmamos que, con la excepción de mutaciones en los sitios de empalme, es poco probable que una mutación afecte la inclusión de dichos exones. Dado que el empalme alternativo es un proceso se encuentra alterado en muchas enfermedades genéticas humanas, pusimos nuestros resultados en un contexto más práctico al plantearnos qué probabilidad hay de que una mutación al azar sea capaz de alterar dicho proceso. Ya que la gran mayoría de exones humanos tienen altos niveles de inclusión, concluimos que es poco probable que una mutación escogida al azar sea capaz de alterar los niveles de inclusión de algún exón. De hecho, esto sólo es probable en el caso de mutaciones en los sitios de empalme o de aquellas que afecten la inclusión de un exón alternativo.
Brown, Jeremy Stuart. "Signature tagged-mutagenesis of aspergillus fumigatus." Thesis, Imperial College London, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.322287.
Full textSeaman, Jonathan. "Signature-tagged mutagenesis in Rhizobium leguminosarum." Thesis, University of Reading, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.499374.
Full textBooks on the topic "Mutagenesis"
Mutagenesis. New York: TOR, 1993.
Find full textPruett-Miller, Shondra M., ed. Chromosomal Mutagenesis. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-1862-1.
Full textDavis, Gregory D., and Kevin J. Kayser, eds. Chromosomal Mutagenesis. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-232-8.
Full textPruett-Miller, Shondra M. Chromosomal mutagenesis. New York: Humana Press, 2015.
Find full textKumar, Nitish, ed. Plant Mutagenesis. Cham: Springer Nature Switzerland, 2024. http://dx.doi.org/10.1007/978-3-031-50729-8.
Full textD, Davis Gregory, and Kayser Kevin J, eds. Chromosomal mutagenesis. Totowa, N.J: Humana Press, 2008.
Find full textH, Phillips D., and Venitt S, eds. Environmental mutagenesis. Oxford: Bios Scientific Publishers, 1995.
Find full textReeves, Andrew, ed. In Vitro Mutagenesis. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-6472-7.
Full textRicke, Steven C., Si Hong Park, and Morgan L. Davis, eds. Microbial Transposon Mutagenesis. New York, NY: Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4939-9570-7.
Full textMittelman, David, ed. Stress-Induced Mutagenesis. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-6280-4.
Full textBook chapters on the topic "Mutagenesis"
Lázaro, Ester. "Mutagenesis." In Encyclopedia of Astrobiology, 1101–2. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-11274-4_1035.
Full textLázaro, Ester. "Mutagenesis." In Encyclopedia of Astrobiology, 1650–51. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-662-44185-5_1035.
Full textNeuffer, M. G. "Mutagenesis." In The Maize Handbook, 212–19. New York, NY: Springer New York, 1994. http://dx.doi.org/10.1007/978-1-4612-2694-9_23.
Full textSpencer, John F. T., Dorothy M. Spencer, and I. J. Bruce. "Mutagenesis." In Yeast Genetics, 30–58. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-73356-7_7.
Full textCrueger, Anneliese. "Mutagenesis." In Biotechnology, 4–45. Weinheim, Germany: Wiley-VCH Verlag GmbH, 2008. http://dx.doi.org/10.1002/9783527620838.ch1.
Full textLabigne, Agnés, and Peter J. Jenks. "Mutagenesis." In Helicobacter pylori, 335–44. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555818005.ch30.
Full textClark, M. S., and W. J. Wall. "Mutagenesis." In Chromosomes, 147–75. Dordrecht: Springer Netherlands, 1996. http://dx.doi.org/10.1007/978-94-009-0073-8_6.
Full textLázaro, Ester. "Mutagenesis." In Encyclopedia of Astrobiology, 1. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-27833-4_1035-3.
Full textLázaro, Ester. "Mutagenesis." In Encyclopedia of Astrobiology, 2036. Berlin, Heidelberg: Springer Berlin Heidelberg, 2023. http://dx.doi.org/10.1007/978-3-662-65093-6_1035.
Full textYamane-Ohnuki, Naoko, Kazuya Yamano, and Mitsuo Satoh. "Biallelic Gene Knockouts in Chinese Hamster Ovary Cells." In Chromosomal Mutagenesis, 1–16. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-232-8_1.
Full textConference papers on the topic "Mutagenesis"
TRICOIRE, LUDOVIC, KEISUKE TSUZUKI, and BERTRAND LAMBOLEZ. "AEQUORIN BIOLUMINESCENCE DISSECTED BY RANDOM MUTAGENESIS." In Chemistry, Biology and Applications. WORLD SCIENTIFIC, 2007. http://dx.doi.org/10.1142/9789812770196_0039.
Full textLytle, C. D., P. G. Carney, R. P. Felten, H. F. Bushar, and R. C. Straight. "Mutagenesis of herpesvirus by different photodynamic treatments." In ICALEO® ‘88: Proceedings of the Laser Research in Medicine, Dentistry & Surgery Conference. Laser Institute of America, 1988. http://dx.doi.org/10.2351/1.5057964.
Full textMcDonald, Isabel K., Stephen C. Holmes, Karen J. Young, Joseph S. Vyle, Timothy J. Pickering, and Jane A. Grasby. "Functional group mutagenesis of the hairpin ribozyme." In XIth Symposium on Chemistry of Nucleic Acid Components. Prague: Institute of Organic Chemistry and Biochemistry, Academy of Sciences of the Czech Republic, 1999. http://dx.doi.org/10.1135/css199902306.
Full textDomingo, Esteban. "LETHAL MUTAGENESIS 2019: A SEQUENCE SPACE ODYSSEY." In Viruses: Discovering Big in Small. TORUS PRESS, 2019. http://dx.doi.org/10.30826/viruses-2019-03.
Full textLi, Ming. "Highly efficient site-specific mutagenesis in malaria mosquitoes." In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.115516.
Full textWang, Lei, Lisha Kuang, Young-Ok Son, John Andrew Hitron, Pratheeshkumar Poyil, Zhuo Zhang, Jia Luo, Zhigang Wang, and Xianglin Shi. "Abstract 5360: Ethanol enhances arsenic-induced mutagenesis in colon." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-5360.
Full textRahmat, Rashida. "Strain Mutagenesis of Stenotrophomonas maltophilia for Higher Exopolysaccharide Production." In IBRAS 2021 INTERNATIONAL CONFERENCE ON BIOLOGICAL RESEARCH AND APPLIED SCIENCE. Juw, 2021. http://dx.doi.org/10.37962/ibras/2021/81-82.
Full textYuwei, Dong, Miao Jingzhi, Tang Shirong, Chen Shanglong, and Wu Yonghua. "Chemical Mutagenesis Breeding of Protoplast of Ammonia-oxidizing Bacteria." In 2015 AASRI International Conference on Circuits and Systems (CAS 2015). Paris, France: Atlantis Press, 2015. http://dx.doi.org/10.2991/cas-15.2015.22.
Full textZhao, Lihong, and Hongjun Sun. "Breeding of high dye-decolorization strain by UV mutagenesis." In 2011 International Conference on Consumer Electronics, Communications and Networks (CECNet). IEEE, 2011. http://dx.doi.org/10.1109/cecnet.2011.5769338.
Full textTISI, LAURENCE, CHRISTOPHER LOWE, and JAMES MURRAY. "MUTAGENESIS OF SOLVENT-EXPOSED HYDROPHOBIC RESIDUES IN FIREFLY LUCIFERASE." In Proceedings of the 11th International Symposium. WORLD SCIENTIFIC, 2001. http://dx.doi.org/10.1142/9789812811158_0047.
Full textReports on the topic "Mutagenesis"
Andrews, Paul W., and Leslie Hill. AS52/GPT Mammalian Mutagenesis Assay. Fort Belvoir, VA: Defense Technical Information Center, May 1996. http://dx.doi.org/10.21236/ada597200.
Full textKale, Purushottam. JP8 Induced Mutagenesis and Hormesis. Fort Belvoir, VA: Defense Technical Information Center, March 2008. http://dx.doi.org/10.21236/ada479392.
Full textWalker, Graham C. Final report [DNA Repair and Mutagenesis - 1999]. Office of Scientific and Technical Information (OSTI), May 2001. http://dx.doi.org/10.2172/807345.
Full textEaton-Rye, Dr., Julian, and Gaozhong Shen. Specific mutagenesis of a chlorophyll-binding protein. Progress report. Office of Scientific and Technical Information (OSTI), January 1990. http://dx.doi.org/10.2172/5701773.
Full textSun, LuZhe. Effect of Estrogen on Mutagenesis in Human Mammary Epithelial Cells. Fort Belvoir, VA: Defense Technical Information Center, June 2005. http://dx.doi.org/10.21236/ada443767.
Full textThilly, W. G. Comparative mutagenesis of human cells in vivo and in vitro. Office of Scientific and Technical Information (OSTI), May 1992. http://dx.doi.org/10.2172/10152396.
Full textAuthor, Not Given. Analysis of cyanobacterial photosystem 2 genes by cloning and mutagenesis. Office of Scientific and Technical Information (OSTI), January 1989. http://dx.doi.org/10.2172/5079285.
Full textThilly, W. G. (Comparative) mutagenesis of human cells in vivo and in vitro. Office of Scientific and Technical Information (OSTI), January 1989. http://dx.doi.org/10.2172/7235709.
Full textThilly, W. G. Comparative mutagenesis of human cells in vivo and in vitro. Office of Scientific and Technical Information (OSTI), January 1990. http://dx.doi.org/10.2172/7279262.
Full textThilly, W. G. Comparative mutagenesis of human cells in vivo and in vitro. Office of Scientific and Technical Information (OSTI), May 1992. http://dx.doi.org/10.2172/5115937.
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