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Books on the topic 'Muscle regeneration'

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Author: Grafiati
Published: 4 June 2021
Last updated: 25 May 2024

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1

Stefano, Schiaffino, and Partridge Terence, eds. Skeletal muscle repair and regeneration. Dordrecht: Springer, 2008.

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2

Kyba, Michael, ed. Skeletal Muscle Regeneration in the Mouse. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3810-0.

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3

DeStefano, Rob. Muscle medicine: The revolutionary approach to maintaining, strengthening, and repairing your muscles and joints. New York: Fireside, 2009.

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4

B, Christ, Čihák Radomír, and European Anatomical Congress (7th : 1984 : Innsbruck, Austria), eds. Development and regeneration of skeletal muscles: Symposium held on occasion of the 7th European Anatomical Congress in Innsbruck, September 3, 1984. Basel: Karger, 1986.

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5

1895-1964, Blatz William Emet, and Kilborn Leslie G. 1895-1967, eds. Studies in the regeneration of denervated mammaliam muscle. Ottawa: J. de L. Taché, 1994.

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6

1935-, Oberpriller John O., Oberpriller Jean C. 1942-, Mauro Alexander, Rockefeller University, Cornell University Medical College, and Rosenfeld Heart Foundation, eds. The Development and regenerative potential of cardiac muscle. Chur: Harwood Academic Publishers, 1991.

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7

C, Claycomb William, Di Nardo Paolo, and New York Academy of Sciences., eds. Cardiac growth and regeneration. New York, N.Y: New York Academy of Sciences, 1995.

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8

White, Jason, and Gayle Smythe, eds. Growth Factors and Cytokines in Skeletal Muscle Development, Growth, Regeneration and Disease. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-27511-6.

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9

1953-, Angelov D. N., ed. Axonal branching and recovery of coordinated muscle activity after transection of the facial nerve in adult rats. Berlin: Springer, 2005.

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10

Coulthard, Rosalind Jane. The roles of motoneurons and their muscle targets in synaptogenesis during regeneration of a foreign transplant. Ottawa: National Library of Canada, 1998.

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11

Childers, Martin K., ed. Regenerative Medicine for Degenerative Muscle Diseases. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3228-3.

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12

Reinhard, Dengler, ed. The Motor unit: Physiology, diseases, regeneration. Munich ; Baltimore: Urban & Schwarzenberg, 1990.

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13

Muscle Homeostasis and Regeneration. MDPI, 2020. http://dx.doi.org/10.3390/books978-3-03943-437-4.

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14

Skeletal Muscle Repair and Regeneration. Dordrecht: Springer Netherlands, 2008. http://dx.doi.org/10.1007/978-1-4020-6768-6.

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15

Partridge, Terence, and Stefano Schiaffino. Skeletal Muscle Repair and Regeneration. Springer, 2010.

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16

Partridge, Terence, and Stefano Schiaffino. Skeletal Muscle Repair and Regeneration. Springer, 2008.

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17

Muscle Cells: Development, Disorders and Regeneration. Nova Biomedical, 2013.

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18

(Editor), R. Cihak, ed. Development and Regeneration of Skeletal Muscles (Bibliotheca Anatomica). S. Karger AG (Switzerland), 1986.

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19

Pinheiro, Carlos Hermano J., and Lucas Guimarães-Ferreira, eds. Frontiers in Skeletal Muscle Wasting, Regeneration and Stem Cells. Frontiers Media SA, 2016. http://dx.doi.org/10.3389/978-2-88919-832-0.

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20

Kyba, Michael. Skeletal Muscle Regeneration in the Mouse: Methods and Protocols. Springer New York, 2016.

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21

Skeletal Muscle Regeneration in the Mouse: Methods and Protocols. Springer, 2018.

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22

Merlini, Luciano, Cesare Faldini, and Paolo Bonaldo, eds. Muscle-Tendon-Innervation Unit: Degeneration and Aging - Pathophysiological and Regeneration Mechanisms. Frontiers Media SA, 2017. http://dx.doi.org/10.3389/978-2-88945-103-6.

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23

Sambu, Sammy K. The automation of metrics useful in evaluating the regeneration of skeletal muscle. 2008, 2008.

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24

White, Jason, and Gayle Smythe. Growth Factors and Cytokines in Skeletal Muscle Development, Growth, Regeneration and Disease. Springer, 2018.

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25

White, Jason, and Gayle Smythe. Growth Factors and Cytokines in Skeletal Muscle Development, Growth, Regeneration and Disease. Springer International Publishing AG, 2016.

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26

White, Jason, and Gayle Smythe. Growth Factors and Cytokines in Skeletal Muscle Development, Growth, Regeneration and Disease. Springer London, Limited, 2016.

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27

Kuiken, Todd A., Aimee E. Schultz Feuser, and Ann K. Barlow. Targeted Muscle Reinnervation: A Neural Interface for Artificial Limbs. Taylor & Francis Group, 2013.

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28

Kuiken, Todd A., Aimee E. Schultz Feuser, and Ann K. Barlow. Targeted Muscle Reinnervation: A Neural Interface for Artificial Limbs. Taylor & Francis Group, 2013.

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29

Kuiken, Todd A., Aimee E. Schultz Feuser, and Ann K. Barlow. Targeted Muscle Reinnervation: A Neural Interface for Artificial Limbs. Taylor & Francis Group, 2017.

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30

Kuiken, Todd A., Aimee E. Schultz Feuser, and Ann K. Barlow. Targeted Muscle Reinnervation: A Neural Interface for Artificial Limbs. Taylor & Francis Group, 2013.

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31

Muñoz-Cánoves, Pura, Jaime J. Carvajal, Adolfo Lopez de Munain, and Ander Zeta, eds. Role of Stem Cells in Skeletal Muscle Development, Regeneration, Repair, Aging and Disease. Frontiers Media SA, 2016. http://dx.doi.org/10.3389/978-2-88919-866-5.

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32

Wong, Anna. Regeneration and transformation of the claw closer muscle in the snapping shrimp, "Alpheus heterochelis". 1987.

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33

Marco, Crescenzi, ed. Reactivation of the cell cycle in terminally differentiated cells. Georgetown, Tex: Landes Bioscience, 2002.

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34

Angelov, D. N., O. Guntinas-Lichius, K. Wewetzer, W. F. Neiss, and M. Streppel. Axonal Branching and Recovery of Coordinated Muscle Activity after Transsection of the Facial Nerve in Adult Rats (Advances in Anatomy, Embryology and Cell Biology). Springer, 2005.

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35

Salvatori, Daniela, Harsha D. Devalla, and Robert Passier. Cells to repair the infarcted myocardium. Edited by José Maria Pérez-Pomares, Robert G. Kelly, Maurice van den Hoff, José Luis de la Pompa, David Sedmera, Cristina Basso, and Deborah Henderson. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757269.003.0030.

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The adult mammalian heart has poor regenerative capacity. Loss of functional cardiomyocytes following myocardial infarction leads to the replacement of functional muscle by scar tissue. This has a detrimental effect on cardiac function and may lead to heart failure. Potential regeneration of severe cardiac damage would require replacement of dead and damaged cardiomyocytes by transplantation, recruitment of endogenous progenitor cells, or induction of cardiomyocyte proliferation. For more than a decade, clinical trials to ameliorate the injured heart have been under way. However, after evaluation of the outcome of these trials it is evident that the beneficial effects of these cell-based transplantations are only marginal, and beneficial effects, if any, are not caused by regeneration of cardiomyocytes. In recent years, alternative approaches and various cell sources have been studied and suggested for cardiac repair. Recent advances in these cell-based therapies or strategies to activate endogenous cardiac repair are discussed.
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36

Crescenzi, Marco. Reactivation of the Cell Cycle in Terminally Differentiated Cells (Molecular Biology Intelligence Unit, 17). Springer, 2003.

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37

Tjärnlund, Anna, and Ingrid E. Lundberg. Diagnostic and classification criteria. Edited by Hector Chinoy and Robert Cooper. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198754121.003.0002.

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Diagnosis of idiopathic inflammatory myopathies (IIM) is based on clinical features such as subacute progress of symmetrical weakness of proximal muscle and muscle fatigue, in combination with laboratory confirmation of myopathy, including elevated muscle enzyme levels in serum and histological demonstration of skeletal muscle inflammation, as well as fibre regeneration and degeneration in muscle biopsies. Several classification criteria for IIM have historically been proposed. New classification criteria for IIM have been developed, and are based on real patient data from adult and juvenile IIM cases worldwide. These criteria provide a probability of having IIM with defined cut-off values for categorizing ‘possible’, ‘probable’, and ‘definite’ IIM. Autoantibodies in IIM are becoming increasingly important for diagnosis and classification, and newly identified autoantibodies specific for inclusion body myositis may provide a future diagnostic tool.
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38

The use of TENS for non-painful conditions. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199673278.003.0010.

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Peripheral nerves consist of afferent and efferent neurones with different functions. TENS can be used to excite somatic efferents to influence the activity of skeletal muscle, and autonomic efferents to influence the activity of smooth muscle, cardiac muscle, and glands. There are physiological rationale to support the use of TENS to manage various non-painful conditions. Clinical experience suggests TENS is often beneficial. The purpose of this chapter is to describe the mechanism of action, clinical use and clinical efficacy for TENS when used to manage non-painful conditions. The chapter covers the effects of TENS on the autonomic nervous system, circulatory system, tissue regeneration, and psychomotor conditions. It also considers the use of TENS for incontinence, constipation, ileus and gastrointestinal discomfort, post-surgical symptoms, and antiemesis.
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39

Childers, Martin K. Regenerative Medicine for Degenerative Muscle Diseases. Humana, 2015.

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40

Childers, Martin K. Regenerative Medicine for Degenerative Muscle Diseases. Humana, 2019.

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41

Childers, Martin K. Regenerative Medicine for Degenerative Muscle Diseases. Humana Press, 2015.

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42

Clarke, Andrew. Temperature and reaction rate. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780199551668.003.0007.

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All other things being equal, physiological reaction rate increases roughly exponentially with temperature. Organisms that have adapted over evolutionary time to live at different temperatures can have enzyme variants that exhibit similar kinetics at the temperatures to which they have adapted to operate. Within species whose distribution covers a range of temperatures, there may be differential expression of enzyme variants with different kinetics across the distribution. Enzymes adapted to different optimum temperatures differ in their amino acid sequence and thermal stability. The Gibbs energy of activation tends to be slightly lower in enzyme variants adapted to lower temperatures, but the big change is a decrease in the enthalpy of activation, with a corresponding change in the entropy of activation, both associated with a more open, flexible structure. Despite evolutionary adjustments to individual enzymes involved in intermediary metabolism (ATP regeneration), many whole-organism processes operate faster in tropical ectotherms compared with temperate or polar ectotherms. Examples include locomotion (muscle power output), ATP regeneration (mitochondrial function), nervous conduction and growth.
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43

A, Ewing William, Herschdorfer Nathalie, and Blaser Jean-Christophe, eds. reGeneration: 50 photographers of tomorrow, 2005-2025. London: Thames & Hudson, 2005.

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44

Shiffman, Melvin A., and Dominik Duscher. Regenerative Medicine and Plastic Surgery: Skin and Soft Tissue, Bone, Cartilage, Muscle, Tendon and Nerves. Springer, 2019.

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45

Rosenblatt, Jonathan David. Myosin isozymes and light chains in regenerating skeletal muscle following chronic, low frequency, electrical stimulation. 1987.

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46

Shiffman, Melvin A., and Dominik Duscher. Regenerative Medicine and Plastic Surgery: Skin and Soft Tissue, Bone, Cartilage, Muscle, Tendon and Nerves. Springer International Publishing AG, 2020.

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