Relevant bibliographies by topics / Muscle oxygenation

Academic literature on the topic 'Muscle oxygenation'

Author: Grafiati
Published: 4 June 2021
Last updated: 15 June 2024

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Journal articles on the topic "Muscle oxygenation"

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Chuang, Ming-Lung, I.-Feng Lin, and Meng-Jer Hsieh. "More Impaired Dynamic Ventilatory Muscle Oxygenation in Congestive Heart Failure than in Chronic Obstructive Pulmonary Disease." Journal of Clinical Medicine 8, no. 10 (October 7, 2019): 1641. http://dx.doi.org/10.3390/jcm8101641.

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Patients with chronic obstructive pulmonary disease (COPD) and congestive heart failure (CHF) often have dyspnea. Despite differences in primary organ derangement and similarities in secondary skeletal muscle changes, both patient groups have prominent functional impairment. With similar daily exercise performance in patients with CHF and COPD, we hypothesized that patients with CHF would have worse ventilatory muscle oxygenation than patients with COPD. This study aimed to compare differences in tissue oxygenation and blood capacity between ventilatory muscles and leg muscles and between the two patient groups. Demographic data, lung function, and maximal cardiopulmonary exercise tests were performed in 134 subjects without acute illnesses. Muscle oxygenation and blood capacity were measured using frequency-domain near-infrared spectroscopy (fd-NIRS). We enrolled normal subjects and patients with COPD and CHF. The two patient groups were matched by oxygen-cost diagram scores, New York Heart Association functional classification scores, and modified Medical Research Council scores. COPD was defined as forced expired volume in one second and forced expired vital capacity ratio ≤0.7. CHF was defined as stable heart failure with an ejection fraction ≤49%. The healthy subjects were defined as those with no obvious history of chronic disease. Age, body mass index, cigarette consumption, lung function, and exercise capacity were different across the three groups. Muscle oxygenation and blood capacity were adjusted accordingly. Leg muscles had higher deoxygenation (HHb) and oxygenation (HbO2) and lower oxygen saturation (SmO2) than ventilatory muscles in all participants. The SmO2 of leg muscles was lower than that of ventilatory muscles because SmO2 was calculated as HbO2/(HHb+HbO2), and the HHb of leg muscles was relatively higher than the HbO2 of leg muscles. The healthy subjects had higher SmO2, the patients with COPD had higher HHb, and the patients with CHF had lower HbO2 in both muscle groups throughout the tests. The patients with CHF had lower SmO2 of ventilatory muscles than the patients with COPD at peak exercise (p < 0.01). We conclud that fd-NIRS can be used to discriminate tissue oxygenation of different musculatures and disease entities. More studies on interventions on ventilatory muscle oxygenation in patients with CHF and COPD are warranted.
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Theodorou, Anastasios A., Panagiotis T. Zinelis, Vassiliki J. Malliou, Panagiotis N. Chatzinikolaou, Nikos V. Margaritelis, Dimitris Mandalidis, Nickos D. Geladas, and Vassilis Paschalis. "Acute L-Citrulline Supplementation Increases Nitric Oxide Bioavailability but Not Inspiratory Muscle Oxygenation and Respiratory Performance." Nutrients 13, no. 10 (September 22, 2021): 3311. http://dx.doi.org/10.3390/nu13103311.

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The present study aimed to investigate whether acute L-citrulline supplementation would affect inspiratory muscle oxygenation and respiratory performance. Twelve healthy males received 6 g of L-citrulline or placebo in a double-blind crossover design. Pulmonary function (i.e., forced expired volume in 1 s, forced vital capacity and their ratio), maximal inspiratory pressure (MIP), fractional exhaled nitric oxide (NO•), and sternocleidomastoid muscle oxygenation were measured at baseline, one hour post supplementation, and after an incremental resistive breathing protocol to task failure of the respiratory muscles. The resistive breathing task consisted of 30 inspirations at 70% and 80% of MIP followed by continuous inspirations at 90% of MIP until task failure. Sternocleidomastoid muscle oxygenation was assessed using near-infrared spectroscopy. One-hour post-L-citrulline supplementation, exhaled NO• was significantly increased (19.2%; p < 0.05), and this increase was preserved until the end of the resistive breathing (16.4%; p < 0.05). In contrast, no difference was observed in the placebo condition. Pulmonary function and MIP were not affected by the L-citrulline supplementation. During resistive breathing, sternocleidomastoid muscle oxygenation was significantly reduced, with no difference noted between the two supplementation conditions. In conclusion, a single ingestion of 6 g L-citrulline increased NO• bioavailability but not the respiratory performance and inspiratory muscle oxygenation.
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Messere, Alessandro, Gianluca Ceravolo, Walter Franco, Daniela Maffiodo, Carlo Ferraresi, and Silvestro Roatta. "Increased tissue oxygenation explains the attenuation of hyperemia upon repetitive pneumatic compression of the lower leg." Journal of Applied Physiology 123, no. 6 (December 1, 2017): 1451–60. http://dx.doi.org/10.1152/japplphysiol.00511.2017.

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The rapid hyperemia evoked by muscle compression is short lived and was recently shown to undergo a rapid decrease even in spite of continuing mechanical stimulation. The present study aims at investigating the mechanisms underlying this attenuation, which include local metabolic mechanisms, desensitization of mechanosensitive pathways, and reduced efficacy of the muscle pump. In 10 healthy subjects, short sequences of mechanical compressions ( n = 3–6; 150 mmHg) of the lower leg were delivered at different interstimulus intervals (ranging from 20 to 160 s) through a customized pneumatic device. Hemodynamic monitoring included near-infrared spectroscopy, detecting tissue oxygenation and blood volume in calf muscles, and simultaneous echo-Doppler measurement of arterial (superficial femoral artery) and venous (femoral vein) blood flow. The results indicate that 1) a long-lasting (>100 s) increase in local tissue oxygenation follows compression-induced hyperemia, 2) compression-induced hyperemia exhibits different patterns of attenuation depending on the interstimulus interval, 3) the amplitude of the hyperemia is not correlated with the amount of blood volume displaced by the compression, and 4) the extent of attenuation negatively correlates with tissue oxygenation ( r = −0,78, P < 0.05). Increased tissue oxygenation appears to be the key factor for the attenuation of hyperemia upon repetitive compressive stimulation. Tissue oxygenation monitoring is suggested as a useful integration in medical treatments aimed at improving local circulation by repetitive tissue compression. NEW & NOTEWORTHY This study shows that 1) the hyperemia induced by muscle compression produces a long-lasting increase in tissue oxygenation, 2) the hyperemia produced by subsequent muscle compressions exhibits different patterns of attenuation at different interstimulus intervals, and 3) the extent of attenuation of the compression-induced hyperemia is proportional to the level of oxygenation achieved in the tissue. The results support the concept that tissue oxygenation is a key variable in blood flow regulation.
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Mangone, Laura A., Beth A. Taylor, Robert Schmelzer, Sung Gi Noh, Michael C. White, Oh Sung Kwon, and Paul D. Thompson. "Skeletal muscle mitochondrial capacity in patients with statin-associated muscle symptoms (SAMS)." Open Heart 11, no. 1 (February 2024): e002551. http://dx.doi.org/10.1136/openhrt-2023-002551.

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ObjectiveThe objective of this article is to evaluate near-infrared spectroscopy (NIRS), a non-invasive technique to assess tissue oxygenation and mitochondrial function, as a diagnostic tool for statin-associated muscle symptoms (SAMS).MethodsWe verified SAMS in 39 statin-treated patients (23 women) using a double-blind, placebo-controlled, cross-over protocol. Subjects with suspected SAMS were randomised to simvastatin 20 mg/day or placebo for 8 weeks, followed by a 4-week no treatment period and then assigned to the alternative treatment, either simvastatin or placebo. Tissue oxygenation was measured before and after each statin or placebo treatment using NIRS during handgrip exercise at increasing intensities of maximal voluntary contraction (MVC).Results44% (n=17) of patients were confirmed as having SAMS (11 women) because they reported discomfort only during simvastatin treatment. There were no significant differences in percent change in tissue oxygenation in placebo versus statin at all % MVCs in all subjects. The percent change in tissue oxygenation also did not differ significantly between confirmed and unconfirmed SAMS subjects on statin (−2.4% vs −2.4%, respectively) or placebo treatment (−1.1% vs −9%, respectively). The percent change in tissue oxygenation was reduced after placebo therapy in unconfirmed SAMS subjects (−10.2%) (p≤0.01) suggesting potential measurement variability.ConclusionsNIRS in the forearm cannot differentiate between confirmed and unconfirmed SAMS, but further research is needed to assess the usability of NIRS as a diagnostic tool for SAMS.Trial registration numberNCT03653663.
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Ahmadi, Sirous, Peter J. Sinclair, and Glen M. Davis. "Muscle oxygenation following concentric exercise." Isokinetics and Exercise Science 15, no. 4 (November 19, 2007): 309–19. http://dx.doi.org/10.3233/ies-2007-0288.

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Ahmadi, Sirous, Peter J. Sinclair, and Glen M. Davis. "Muscle Oxygenation after Downhill Walking- Induced Muscle Soreness." Medicine & Science in Sports & Exercise 39, Supplement (May 2007): S39. http://dx.doi.org/10.1249/01.mss.0000273041.68107.42.

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Ufland, Pierre, Thomas Lapole, Said Ahmaidi, and Martin Buchheit. "Muscle force recovery in relation to muscle oxygenation." Clinical Physiology and Functional Imaging 32, no. 5 (April 24, 2012): 380–87. http://dx.doi.org/10.1111/j.1475-097x.2012.01141.x.

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Neuschwander, T. B., B. R. Macias, A. R. Hargens, and Q. Zhang. "Mild External Compression of the Leg Increases Skin and Muscle Microvascular Blood Flow and Muscle Oxygenation during Simulated Venous Hypertension." ISRN Vascular Medicine 2012 (December 10, 2012): 1–6. http://dx.doi.org/10.5402/2012/930913.

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We studied the effect of mild external leg compression on both skin and muscle microvascular flow, and muscle oxygenation in the leg of healthy subjects during simulated venous hypertension. Skin and muscle microvascular blood flows were measured using photoplethysmography (PPG), and muscle oxygenation was measured using near-infrared spectroscopy (NIRS). Both PPG and NIRS probes were placed over the anterior compartment of the right leg in 8 healthy subjects. Measurements were taken under three experimental conditions: external leg compression (40 mmHg); simulated venous hypertension (65 mmHg thigh cuff); external leg compression during simulated venous hypertension. Muscle oxygenation was measured only under external leg compression during simulated venous hypertension. Simulated venous hypertension decreased skin and muscle microvascular blood flows from 100% (baseline) to 35.8±2.9% and 31.9±1.3% (P<0.001), respectively. External leg compression during simulated venous hypertension caused 2-fold increases in both skin and muscle microvascular blood flows compared to simulated venous hypertension (P<0.001). Similarly, external leg compression during simulated venous hypertension significantly restored muscle oxygenation by 23±7% compared to its baseline (P<0.05). Our results demonstrate that mild external leg compression counteracts the decreases in skin microvascular flow, muscle microvascular flow, and muscle oxygenation induced by simulated venous hypertension in the leg.
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Theodorakopoulou, Marieta, Andreas Zafeiridis, Konstantina Dipla, Danai Faitatzidou, Aggelos Koutlas, Fotini Iatridi, Artemios Karagiannidis, et al. "EFFECTS OF PROTEINURIA ON MUSCLE OXYGENATION AND MICROVASCULAR REACTIVITY IN PATIENTS WITH PRE-DIALYSIS CKD: A POST-HOC ANALYSIS." Journal of Hypertension 42, Suppl 1 (May 2024): e206. http://dx.doi.org/10.1097/01.hjh.0001021532.72701.f2.

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Objective: Vascular dysfunction is a hallmark of CKD. Previous studies showed an impaired microvascular reactivity in the skeletal muscles, which deteriorates in advanced CKD stages. This analysis aims to examine the impact of proteinuria on skeletal muscle oxygenation and microvascular reactivity at rest, during an occlusion-reperfusion maneuver, and during exercise in patients with pre-dialysis CKD. Design and method: 66 patients with CKD stage 2-4 were included in this post-hoc analysis; 24-h urine samples were used for evaluation of proteinuria. Continuous measurement of muscle oxygenation [tissue saturation index (TSI%)] via near-infrared-spectroscopy at rest, during occlusion-reperfusion and during a 3-min handgrip exercise (at 35% of maximal-voluntary-contraction). Results: The study groups were similar in terms of age (proteinuric vs nonproteinuric: 68.4±10.6 vs 67.2±10.8; p=0.676), eGFR (41.3±18.9 vs 46.3±14.8; p=0.248) and BMI (28.4±4.9 vs 28.1±4.8; p=0.803). Resting muscle oxygenation did not differ between study groups (proteinuric vs nonproteinuric: 63,35±4,09 vs 62,34±3,21; p=0.280). During occlusion, proteinuric CKD patients had marginally lower TSI occlusion magnitude (25,77±7,87 vs 29,95±10,34; p=0.074) but no difference in occlusion slope (-0,09±.,03 vs -,10±.,04; p=0.134). During reperfusion, the TSI reperfusion slopes were significantly lower in proteinuric CKD (slope to max 1,03±.,45 vs 1,39±.0,69; p=0.035 and 10-sec slope: 1,34±.,63 vs 1,92±.,75; p=0.002); hyperemic response was numerically lower in proteinuric CKD (7,13±.4,27 vs 8,89±.4,68; p=0.131). Finally, during exercise no differences were detected in the average muscle oxygenation between-group (10,76±6,05 vs 10,65±5,39; p=0.943). Conclusions: Although no differences in resting muscle oxygenation were detected, CKD patients with proteinuria showed more impaired skeletal muscle oxidative capacity (as suggested by lower TSI magnidute during occlusion), and miscrovascular reactivity during reperfusion.
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Games, Kenneth E., JoEllen M. Sefton, and Alan E. Wilson. "Whole-Body Vibration and Blood Flow and Muscle Oxygenation: A Meta-Analysis." Journal of Athletic Training 50, no. 5 (May 1, 2015): 542–49. http://dx.doi.org/10.4085/1062-6050-50.2.09.

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Context The use and popularity of whole-body vibration (WBV) has increased in recent years, but there is a lack of consensus in the literature about the effectiveness of the treatment. Objective To quantitatively examine the effects of WBV on muscle oxygenation and peripheral blood flow in healthy adults. Data Sources We searched Web of Science and PubMed databases and reference lists from relevant articles using the key terms whole body vibration, whole-body vibration, WBV, blood flow, peripheral blood flow, oxygenation, muscle oxygenation, circulation, circulatory, near infrared spectroscopy, NIRS, and power Doppler. Key terms were searched using single word and combination searches. No date range was specified. Study Selection Criteria for inclusion were (1) use of a commercially available WBV device, (2) a human research model, (3) a pre-WBV condition and at least 1 WBV experimental condition, and (4) reporting of unstandardized means and standard deviations of muscle oxygenation or peripheral blood flow. Data Extraction Means, standard deviations, and sample sizes were extracted from the text, tables, and figures of included studies. A total of 35 and 90 data points were extracted for the muscle-oxygenation and blood-flow meta-analyses, respectively. Data for each meta-analysis were combined and analyzed using meta-analysis software. Weighted, random-effects meta-analyses using the Hedges g metric were completed for muscle oxygenation and blood flow. We then conducted follow-up analyses using the moderator variables of vibration type, vibration time, vibration frequency, measurement location, and sample type. Data Synthesis We found 18 potential articles. Further examination yielded 10 studies meeting the inclusion criteria. Whole-body vibration was shown to positively influence peripheral blood flow. Additionally, the moderators of vibration type and frequency altered the influence of WBV on blood flow. Overall, WBV did not alter muscle oxygenation; however, when the measurement site was considered, muscle oxygenation increased or decreased depending on the location. Conclusions Acute bouts of WBV increase peripheral blood flow but do not alter skeletal muscle oxygenation. Vibration type appears to be the most important factor influencing both muscle oxygenation and peripheral blood flow.
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Dissertations / Theses on the topic "Muscle oxygenation"

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Ahmadi, Sirous. "Monitoring muscle oxygenation and myoelectric activity after damage-inducing exercise." Thesis, The University of Sydney, 2007. http://hdl.handle.net/2123/2240.

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In this thesis, three experiments were conducted to monitor: (i) muscle oxygenation and electromyographic activity of the biceps brachii after exercise-induced muscle damage (ii) muscle oxygenation after downhill walking-induced muscle damage, and, (iii) muscle oxygenation following a bout of vigorous concentric exercise. Maximal eccentric exercise (EE) of biceps brachii resulted in significantly increased mean resting oxygen saturation and decreased deoxyhaemoglobin. During isometric contractions at 50% and 80% of subjects’ maximum voluntary torque (MVT), oxygen desaturation and resaturation kinetics and volume were significantly decreased after EE, and these declines were significantly prevalent over the following 6 days. Additionally, a significant shift in median frequency intercept (measured by electromyography; EMG) towards lower frequencies was observed during isometric contractions at both 50% and 80% MVT after EE in the exercised arm. After an exhaustive session of downhill walking, another form of EE, resting total haemoglobin and oxyhaemoglobin decreased. Furthermore, during isometric contractions at 30%, 50% and 80% of MVT, prolonged and significant increases were observed in oxygen desaturation and resaturation kinetics and volumes after ambulatory EE. In contrast to the two EE experiments, concentric contractions did not evoke any prolonged changes in muscle oxygenation. Collectively, the findings of this thesis revealed significant and prolonged changes in muscle oxygenation at rest and during exercise, following sessions of strenuous eccentric exercise. Although not clear, the possible mechanism responsible for the changes in muscle oxygenation after EE could be increased resting muscle oxygen utilization due to probable muscle damage and a subsequent requirement of energy demanding repair processes. Concentric exercise resulted in fatigue, but it did not affect muscle oxygenation. Although a prolonged reduction in EMG median frequency intercept was observed after EE, this was not closely time-associated with the biochemical, anthropometric or functional markers of muscle damage.
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Ahmadi, Sirous. "Monitoring muscle oxygenation and myoelectric activity after damage-inducing exercise." University of Sydney, 2007. http://hdl.handle.net/2123/2240.

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Doctor of Philosophy
In this thesis, three experiments were conducted to monitor: (i) muscle oxygenation and electromyographic activity of the biceps brachii after exercise-induced muscle damage (ii) muscle oxygenation after downhill walking-induced muscle damage, and, (iii) muscle oxygenation following a bout of vigorous concentric exercise. Maximal eccentric exercise (EE) of biceps brachii resulted in significantly increased mean resting oxygen saturation and decreased deoxyhaemoglobin. During isometric contractions at 50% and 80% of subjects’ maximum voluntary torque (MVT), oxygen desaturation and resaturation kinetics and volume were significantly decreased after EE, and these declines were significantly prevalent over the following 6 days. Additionally, a significant shift in median frequency intercept (measured by electromyography; EMG) towards lower frequencies was observed during isometric contractions at both 50% and 80% MVT after EE in the exercised arm. After an exhaustive session of downhill walking, another form of EE, resting total haemoglobin and oxyhaemoglobin decreased. Furthermore, during isometric contractions at 30%, 50% and 80% of MVT, prolonged and significant increases were observed in oxygen desaturation and resaturation kinetics and volumes after ambulatory EE. In contrast to the two EE experiments, concentric contractions did not evoke any prolonged changes in muscle oxygenation. Collectively, the findings of this thesis revealed significant and prolonged changes in muscle oxygenation at rest and during exercise, following sessions of strenuous eccentric exercise. Although not clear, the possible mechanism responsible for the changes in muscle oxygenation after EE could be increased resting muscle oxygen utilization due to probable muscle damage and a subsequent requirement of energy demanding repair processes. Concentric exercise resulted in fatigue, but it did not affect muscle oxygenation. Although a prolonged reduction in EMG median frequency intercept was observed after EE, this was not closely time-associated with the biochemical, anthropometric or functional markers of muscle damage.
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Vallet, Benoît. "Reactivite vasculaire et oxygenation tissulaire." Lille 2, 1994. http://www.theses.fr/1994LIL2P265.

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Hernández, Andrés Gladden L. Bruce. "Muscle oxygenation does not affect the prior exercise effect." Auburn, Ala., 2009. http://hdl.handle.net/10415/1837.

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Ufland, Pierre. "Muscle oxygenation and capacity to repeat high-intensity exercises." Amiens, 2012. http://www.theses.fr/2012AMIE0018.

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Monitoring non-invasively muscle oxygenation (mO2), inferred from oxygenated haemoglobin (Hb)/myoglobin (Mb) and deoxygenated Hb/Mb measures, has emerged at the end of the 80s with the develoment of near-infrared spectroscopy (NIRS). Today, NIRS devices are portable and make possible to continuously and remotely monitor the balance between oxygen (O2) delivery to working muscles and muscle O2 uptake in cpillary beds of the investigated muscle. For instance, while a high muscle O2 uptake ability in generaly associated with performance improvement, a low muscle O2 uptake ability may be associated with delayed metabolic recovery between successive efforts. The aims of this thesis were to examine the methodological considerations on the use of NIRS to asses mO2 and muscle O2 uptake (mVO2) (studies 1 and 2) and to consider the eventual relashionship that can exist between mO2 and physical performance during repeated high-intensity exercices (studies 2, 4 and 5). We first investigated the reliability and the sensitivity of the post exercise mO2 (study 1). We found a moderate level of reliability, which was independent of exercise intensity during an occlusion-free recovery condition. Conversely, when the recovery of NIRS-derived mVO2 was considered, measures were also moderately reliable but exercise-intensity dependent. In a subsequent study (studie 2), we examined the influence of changes of direction (COD) on mVO2. We observed a greaterm VO2 with COD (i. E. , at similar absolute running speeds, 20-m shuttle runs were associated with higher mVO2 than straight-line runs). These results suggested that mVO2 is also sensitive to running modality (i. E. , COD). In the second part of the thesis, we first examined the role of mO2 recovery in muscular force recovery during repeated-maximal effort (study 3). The results confirmed that mO2 recovery may play a moderate role on the maximal force production. Afterward, we investigated the influence of training background on mVO2 recovery post-exercise (study 4). We found that an endurance training background is associated with faster mVO2 recovery compared with a sprint training background, which was itself related to better repeated-sprint ability. Finally, in a longitudinal study (study 5), we examined the chronic changes on post-sprints mO2 after an aerobic training period. The improvement in repeated-sprint ability after training was related to the acceleration of the mO2 post-sprints, suggesting again that muscle aerobic function might play a role in the (metabolic) between-sprints recovery
Il est généralement accepté qu'une plus grande capacité de consommation d'O2 musculaire est associée à une meilleure performance aérobie. A l'inverse, une faible capacité de consommation d'O2 musculaire est également associée à une récupération inter-effort réduite/ralentie. A la fin des années 80, le suivi non invasif de l'oxygénation musculaire (mO2), déduite par la mesure de l'hémoglobine ([Hb]) / myoglobine ([Mb]) saturé en oxygène (O2) et de l'[Hb]/[Mb] dépourvu d'O2, a émergé dans de nombreuses études avec l'inclusion de la spectroscopie du proche infrarouge (NIRS). Le fait que la NIRS soit devenue portable rend libre l'évaluation continue de la balance entre apport d'O2 aux muscles en action et consommation musculaire d'O2 des lits capillaires du muscle considéré. Dans cette thèse, nous nous sommes attachés à examiner les considérations méthodologiques de l'utilisation de la NIRS pour évaluer mO2 et consommation d’oxygène musculaire (mVO2) (Etudes 1 et 2). Les études 2, 4 et 5 ont étudié les relations éventuelles qui peuvent exister entre la mO2 et la performance lors d'exercices répétés à haute intensité. Initialement nous avons étudié la reproductibilité et la sensibilité de mO2 post-exercice (Etude 1). Un niveau modéré de reproductibilité, ainsi qu'une indépendance des relevés en fonction de l'intensité d'exercice lors de la récupération sans occlusion ont été constatés. Par contre, lorsque la récupération de la mVO2 était observée, il fut relevé que les mesures étaient, certes modérément reproductibles à l’instar de mO2 post-exercice mais à contrario dépendantes de l'intensité d'exercice. Dans une étude subséquente (Etude 2), nous avons inspecté l'influence des changements de direction (COD) sur la mVO2. Nous avons ainsi observé une plus grande mVO2 avec COD : pour des vitesses similaires absolues de course, les courses en navette sur 20 m étaient associées à de plus grandes mVO2 que lors des courses en ligne droite. Ces résultats suggéraient que mVO2 déjà sensible à l’intensité de l’exercice, l’était aussi à la modalité de course (avec ou sans COD). En second lieu, le rôle de la mO2 sur le paramètre de récupération de force musculaire durant des efforts maximaux répétés a été examiné (Etude 3). Les résultats ont confirmé que la récupération de mO2 peut jouer un rôle modéré dans la production de force maximale. Suite à cette étude, nous avons souhaité évaluer l'influence du type d'entraînement sur la récupération de mVO2 (Etude 4). Ainsi, la comparaison entre entraînement en endurance et entraînement au sprint révèle une meilleure récupération de mVO2 dans le groupe endurance. Cette accélération de la récupération de mVO2 était liée à une meilleure capacité à répéter des sprints. L’entraînement agit donc sur le paramètre de récupération de mV2. En dernière partie, nous avons inspecté dans une étude longitudinale (Etude 5) les effets d’une période d'entraînement aérobie sur la mO2 post-sprints. L'amélioration de la capacité à répéter des sprints a été associée à l'accélération de la mO2 post-sprints, ce qui confirme que la fonction musculaire aérobie pourrait jouer un rôle au niveau dans la récupération métabolique entre les sprints
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Ferreira, Leonardo Franklin. "Dynamics of muscle blood flow, O[subscript2] uptake and muscle microvascular oxygenation during exercise." Diss., Manhattan, Kan. : Kansas State University, 2006. http://hdl.handle.net/2097/201.

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Dias, Wilson Vinicius. "Adaptations du métabolisme musculaire en réponse à l’exercice et à une supplémentation en antioxydants chez des patients atteints de Dystrophie Fascioscapulohumérale." Thesis, Montpellier, 2015. http://www.theses.fr/2015MONTT030.

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La dystrophie FacioScapuloHumérale (FSHD), décrite pour la première fois en 1885 par Landouzy Dejerine, est la première dystrophie musculaire de l’adulte en France affectant entre 4000 et 5000 personnes. La destruction progressive des fibres musculaires entraîne une atrophie et une faiblesse musculaires s’aggravant progressivement, avec cependant une grande variabilité intra-familiale du degré des atteintes. Une caractéristique de l’atteinte musculaire est généralement son asymétrie. Les premières manifestations concernent souvent les muscles du visage, les muscles de l’omoplate et des muscles perihuméraux. En progressant la pathologie va toucher d’autres territoires musculaires. Dans environ 10 à 15 % des cas, à un stade évolué, les patients sont contraints d'utiliser un fauteuil roulant. En dépit d’avancées majeures dans la compréhension du locus morbide, les mécanismes exacts responsables des défauts musculaires de la FSHD ne sont toujours pas compris et il n’existe aucune thérapie. Toutefois, il existe de plus en plus de données qui permettent une implication probable du stress oxydant dans cette pathologie. L’hypothèse selon laquelle les réponses antioxydantes sont altérées dans la FSHD s’appuie sur des dérégulations d’enzymes impliqués dans le stress oxydant. Une étude prospective réalisée sur des patients FSHD et des volontaires sains nous a ainsi permis de mettre en évidence une corrélation entre le stress oxydant systémique et musculaire et leurs déficits fonctionnels musculaires. Ces données nous ont conduit à la mise en place d’un essai clinique randomisé, contrôlé, en double aveugle contre placébo, visant à évaluer les effets d’une supplémentation en antioxydants chez 54 patients atteints de FSHD pendant 17 semaines. Cet essai a ainsi permis de montrer une augmentation significative de la force et l’endurance des quadriceps corrélée à une diminution du stress oxydant et une augmentation des défenses antioxydantes chez les patients atteints de FSHD. De nombreuses caractéristiques de la FSHD pourraient être causées et/ou exacerbées par des perturbations de la production des espèces radicalaires ou une réponse non adaptée à cette production. Aussi le premier objectif de ma thèse est de mener une étude comparative des profils d’oxygénation par spectroscopie dans le proche infrarouge de patients atteints de FSHD et sains. Le second objectif est d’évaluer l’effet de la supplémentation en antioxydant sur le volume des quadriceps par IRM et leur qualité musculaire déterminée par le ratio Force/Volume musculaire du quadriceps et d’évaluer les corrélations entre ces variables, la force et le stress oxydant. Les données obtenues ont permis de montrer une réduction de la capacité oxydative lors d’une contraction isométrique volontaire des quadriceps et ont permis d’étudier l’effet de la supplémentation sur les volumes et la qualité musculaire des quadriceps. Ces augmentations sont associées non seulement à une augmentation de la force des quadriceps mais aussi à une diminution du stress oxydant et une augmentation des défenses antioxydantes. L’ensemble de ces données montrent que le stress oxydant pourrait jouer un rôle important dans la FSHD et qu’une approche antioxydante semble adaptée à cette pathologie. Des analyses plus fines sur l’action des espèces réactives de l’oxygène (ROS) et leurs sources pourraient contribuer à une meilleure compréhension des bases physiopathologiques de la FSHD
Facioscapulohumeral muscular dystrophy (FSHD), first described in 1885 by Landouzy Dejerine, is the most common inherited skeletal muscle disease of adult life affecting 4000 to 5000 persons in France. Progressive evolution of the disease leads to progressive weakness and atrophy of muscle fibers associated to a wide variability. The pattern of muscle weakness is often asymmetrical and the rate and extent of progression may vary considerably with sudden periods of unexplained rapid disease progression. This muscle disorder is characterized by progressive muscle weakness, beginning with facial muscles and the shoulder girdle, followed by the pelvic girdle and the muscles of the lower extremities. In 10 to 15% of cases, patients need to use a wheelchair. Despite major progress in the understanding of the genetic basis of FSHD, the exact mechanisms that lead to FSHD defects are not completely understood and no curative treatment is available. However, there is growing evidence that oxidative stress may contribute to FSHD pathology. The hypothesis that oxidative stress responses might be specifically altered in FSHD is supported by the deregulation of enzymes involved in oxidative stress.A prospective study realized with FSHD patients and healthy subjects unrevealed the correlation between systemic and muscular oxidative stress and functional muscle defects. Based on these data, we organized a randomized, double-blind, placebo-controlled pilot clinical trial in order to evaluate the effects of 17 weeks antioxidant supplementation in 54 FSHD patients. This clinical trial demonstrates a significant increase in muscle force and quadriceps endurance correlated to a decrease in oxidative stress and an increase in antioxidant defense in FSHD patientsFurthermore, many FSHD features may be caused or exacerbated by perturbations in the production of free radicals or inappropriate response to such stressors. Therefore the first objective was planned to investigate muscle oxygenation patterns during and after a MVCQ by near-infrared diffuse optical spectroscopy (NIRS). The second objective is to evaluate the effect of antioxidant supplementation on quadriceps volumes by IRM and determine the muscle quality using Strength/ Volume ratio of quadriceps muscles and correlate this variables with force and oxidative stress parameters.The major findings of this study show a significant decrease in oxidative capacity during voluntary isometric contraction in quadriceps and demonstrate the effect of supplementation on muscle volume and quality. Indeed, vitamin E, vitamin C, zinc and selenium supplementation improves muscle volume and quality of both quadriceps by enhancing the antioxidant defences and reducing oxidative stress.This increase are associated to increase in strength and decrease in oxidative stress and increase in antioxidant defences. Taken together, we show that oxidative stress plays an important role in FSHD and that an anti-oxidant strategy adapted to the FSHD-specific “oxidative stress” may be a relevant therapeutic approach for these patients. Further analyses of ROS production and sources could contribute to a better understanding of the pathophysiological mechanisms implicated in FSHD
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Greenbaum, Adam Raphael. "A microelectrode study of skeletal muscle oxygenation and perfusion during rest and electrical stimulation." Thesis, Imperial College London, 1995. http://hdl.handle.net/10044/1/7744.

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Germain, Geneviève. "Effect of hyperbaric oxygen therapy on exercise-induced muscle injury." Thesis, McGill University, 2002. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=29504.

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The purpose of this study was to examine the effects of HBO2 therapy on exercise-induced muscle damage. Subjects (n = 16 university student volunteers) were randomly divided into an experimental group that received HBO2 therapy and a control group that did not receive any treatments. HBO2 treatments consisted of 5 sessions of breathing 95% oxygen at 2.5 atm abs for 100 min. Temporary muscle soreness was created using a single-leg eccentric exercise task involving the quadriceps femoris. Over the next 14 days, measurements were obtained on muscle soreness, leg circumference, quadriceps peak torque, quadriceps average power, fatigue and plasma creative kinase. After eccentric exercise, plasma CK levels and perceived muscle soreness were elevated but were not different between HBO2 and control groups. HBO2 therapy did not alter leg circumference, quadriceps peak torque, average power or fatigue compared to the control group. The data indicated that five HBO2 treatments did not speed recovery following eccentric exercise that induced temporary muscle damage.
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Dascombe, Benjamin James, and b. dascombe@cqu edu au. "Pulmonary Oxygen Uptake and Muscle Oxygenation Responses to Exercise in Well-Trained Young and Middle-Aged Cyclists." Central Queensland University. Department of Health and Human Performance, 2007. http://library-resources.cqu.edu.au./thesis/adt-QCQU/public/adt-QCQU20070719.105750.

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This thesis details four consecutive research investigations which were designed to examine the effect of age on the pulmonary oxygen uptake (VO2)and muscle oxygenation (mOxy) responses to exercise in well-trained cyclists. (Abridged)
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Books on the topic "Muscle oxygenation"

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Colwell, Cynthia M. Researching Music Therapy in Medical Settings. Edited by Jane Edwards. Oxford University Press, 2015. http://dx.doi.org/10.1093/oxfordhb/9780199639755.013.16.

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Research has indicated that music therapy is effective in hospital contexts for managing pain, reducing anxiety, ameliorating social isolation, slowing the impact of cognitive or developmental regression or delays, expressing emotions, and altering physiological responses as medically needed. Music can impact physiological responses including heart rate, blood pressure, pulse oxygenation, pain indicators, respiration, muscle tension, cardiac output, and immunologic function. Participation in music therapy interventions can improve treatment adherence, reduce deleterious symptoms of diseases and effects of medical procedures, and generally enhance quality of life in an unfamiliar and potentially unappealing environment. This chapter will describe a sample of how music therapists have conducted research in medical contexts and will present ways in which such research can be planned and undertaken.
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Kreit, John W. Patient–Ventilator Interactions and Asynchrony. Edited by John W. Kreit. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190670085.003.0011.

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Patient–Ventilator Interactions and Asynchrony describes what happens when the patient and the ventilator do not work together in an effective, coordinated manner. Effective mechanical ventilation requires the synchronized function of two pumps: The mechanical ventilator is governed by the settings chosen by the clinician; the patient’s respiratory system is controlled by groups of neurons in the brain stem. Ideally, the ventilator simply augments and amplifies the activity of the respiratory system. Asynchrony between the ventilator and the patient reduces patient comfort, increases work of breathing, predisposes to respiratory muscle fatigue, and may even impair oxygenation and ventilation. The chapter describes the causes and consequences of patient–ventilator asynchrony during ventilator triggering and the inspiratory phase of the respiratory cycle and explains how to adjust ventilator settings to improve patient comfort and reduce the work of breathing.
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Gattinon, Luciano, and Eleonora Carlesso. Acute respiratory failure and acute respiratory distress syndrome. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0064.

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Respiratory failure (RF) is defined as the acute or chronic impairment of respiratory system function to maintain normal oxygen and CO2 values when breathing room air. ‘Oxygenation failure’ occurs when O2 partial pressure (PaO2) value is lower than the normal predicted values for age and altitude and may be due to ventilation/perfusion mismatch or low oxygen concentration in the inspired air. In contrast, ‘ventilatory failure’ primarily involves CO2 elimination, with arterial CO2 partial pressure (PaCO2) higher than 45 mmHg. The most common causes are exacerbation of chronic obstructive pulmonary disease (COPD), asthma, and neuromuscular fatigue, leading to dyspnoea, tachypnoea, tachycardia, use of accessory muscles of respiration, and altered consciousness. History and arterial blood gas analysis is the easiest way to assess the nature of acute RF and treatment should solve the baseline pathology. In severe cases mechanical ventilation is necessary as a ‘buying time’ therapy. The acute hypoxemic RF arising from widespread diffuse injury to the alveolar-capillary membrane is termed Acute Respiratory Distress Syndrome (ARDS), which is the clinical and radiographic manifestation of acute pulmonary inflammatory states.
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Gattinon, Luciano, and Eleonora Carlesso. Acute respiratory failure and acute respiratory distress syndrome. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199687039.003.0064_update_001.

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Respiratory failure (RF) is defined as the acute or chronic impairment of respiratory system function to maintain normal oxygen and CO2 values when breathing room air. ‘Oxygenation failure’ occurs when O2 partial pressure (PaO2) value is lower than the normal predicted values for age and altitude and may be due to ventilation/perfusion mismatch or low oxygen concentration in the inspired air. In contrast, ‘ventilatory failure’ primarily involves CO2 elimination, with arterial CO2 partial pressure (PaCO2) higher than 45 mmHg. The most common causes are exacerbation of chronic obstructive pulmonary disease (COPD), asthma, and neuromuscular fatigue, leading to dyspnoea, tachypnoea, tachycardia, use of accessory muscles of respiration, and altered consciousness. History and arterial blood gas analysis is the easiest way to assess the nature of acute RF and treatment should solve the baseline pathology. In severe cases mechanical ventilation is necessary as a ‘buying time’ therapy. The acute hypoxemic RF arising from widespread diffuse injury to the alveolar-capillary membrane is termed Acute Respiratory Distress Syndrome (ARDS), which is the clinical and radiographic manifestation of acute pulmonary inflammatory states.
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Book chapters on the topic "Muscle oxygenation"

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Lund, Niels. "Skeletal and Cardiac Muscle Oxygenation." In Advances in Experimental Medicine and Biology, 37–43. Boston, MA: Springer US, 1985. http://dx.doi.org/10.1007/978-1-4684-3291-6_3.

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Barlow, Clyde H., Emily Bailey, Katherine A. Kelly, and Jeffrey J. Kelly. "Reflectance Measurement of Heart Muscle Oxygenation." In Oxygen Transport to Tissue XX, 487–500. Boston, MA: Springer US, 1998. http://dx.doi.org/10.1007/978-1-4615-4863-8_59.

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Jöbsis-VanderVliet, Frans F., and Paul D. Jöbsis. "Intracellular Oxygenation and Redox Status in Muscle." In Advances in Experimental Medicine and Biology, 233. Boston, MA: Springer US, 1994. http://dx.doi.org/10.1007/978-1-4615-1875-4_38.

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Weinzirl, J., M. Wolf, M. Nelle, P. Heusser, and U. Wolf. "Colored Light and Brain and Muscle Oxygenation." In Oxygen Transport to Tissue XXXIII, 33–36. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4614-1566-4_5.

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Bredle, D. L., C. K. Chapler, and S. M. Cain. "Central Reflex Effects of Hypoxia on Muscle Oxygenation." In Oxygen Transport to Tissue XI, 681–85. Boston, MA: Springer US, 1989. http://dx.doi.org/10.1007/978-1-4684-5643-1_75.

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Steimers, A., M. Vafiadou, G. Koukourakis, D. Geraskin, P. Neary, and M. Kohl-Bareis. "Muscle Oxygenation During Running Assessed by Broad Band NIRS." In Advances in Experimental Medicine and Biology, 41–47. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3023-4_5.

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Hoofd, Louis, and Hans Degens. "Statistical Treatment of Oxygenation-Related Data in Muscle Tissue." In Oxygen Transport to Tissue XXXV, 137–42. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-7411-1_19.

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Wakasugi, Tatsushi, Yuki Uchiyama, and Shinichiro Morishita. "Skeletal Muscle Oxygenation in Patients with Malignant Hematopoietic Disease." In Physical Therapy and Research in Patients with Cancer, 495–512. Singapore: Springer Nature Singapore, 2022. http://dx.doi.org/10.1007/978-981-19-6710-8_22.

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King, Cheryl E. "Muscle Oxygenation and Performance during Low Level Carbon Monoxide Exposure." In Advances in Experimental Medicine and Biology, 533–40. Boston, MA: Springer US, 1990. http://dx.doi.org/10.1007/978-1-4684-8181-5_60.

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Hoofd, Louis, and Hans Degens. "The Influence Of Flow Redistribution On Working Rat Muscle Oxygenation." In Advances in Experimental Medicine and Biology, 55–60. Boston, MA: Springer US, 2009. http://dx.doi.org/10.1007/978-0-387-85998-9_9.

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Conference papers on the topic "Muscle oxygenation"

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Bretonneau, Quentin, Claire de Bisschop, and Aurélien Pichon. "Intercostal muscle oxygenation during expiratory load breathing." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa3359.

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Zhang, Li, and Gaoqing Song. "Influence on muscle oxygenation to EMG parameters at different skeletal muscle contraction." In Saratov Fall Meeting 2009, edited by Valery V. Tuchin and Elina A. Genina. SPIE, 2009. http://dx.doi.org/10.1117/12.850983.

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Van Hollebeke, Marine, Diego Poddighe, Beatrix Clerckx, Greet Hermans, Rik Gosselink, Daniel Langer, and Zafeiris Louvaris. "Inspiratory muscle training improves muscle oxygenation and efficiency of extradiaphragmatic inspiratory muscles in difficult to wean (DTW) patients." In ERS International Congress 2021 abstracts. European Respiratory Society, 2021. http://dx.doi.org/10.1183/13993003.congress-2021.oa4228.

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Geraskin, Dmitri, Petra Platen, Julia Franke, and Matthias Kohl-Bareis. "Algorithms for Muscle Oxygenation Monitoring corrected for Adipose Tissue Thickness." In European Conference on Biomedical Optics. Washington, D.C.: OSA, 2007. http://dx.doi.org/10.1364/ecbo.2007.6629_25.

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Geraskin, Dmitri, Petra Platen, Julia Franke, and Matthias Kohl-Bareis. "Algorithms for Muscle Oxygenation Monitoring corrected for Adipose Tissue Thickness." In Biomedical Topical Meeting. Washington, D.C.: OSA, 2006. http://dx.doi.org/10.1364/bio.2006.me65.

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Geraskin, Dmitri, Petra Platen, Julia Franke, and Matthias Kohl-Bareis. "Algorithms for Muscle Oxygenation Monitoring corrected for Adipose Tissue Thickness." In Biomedical Topical Meeting. Washington, D.C.: OSA, 2006. http://dx.doi.org/10.1364/bio.2006.me78.

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Geraskin, Dmitri, Petra Platen, Julia Franke, and Matthias Kohl-Bareis. "Algorithms for muscle oxygenation monitoring corrected for adipose tissue thickness." In European Conference on Biomedical Optics, edited by Brian W. Pogue and Rinaldo Cubeddu. SPIE, 2007. http://dx.doi.org/10.1117/12.727854.

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Schenkman, Kenneth A., Wayne A. Ciesielski, Eileen M. Bulger, David J. Carlbom, Jeremy M. Shaver, Dana M. Fisk, Kellie L. Sheehan, et al. "Noninvasive Measurement Of Muscle Oxygenation Identifies Shock In Trauma Patients." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a3775.

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Vallet, Benoit, Jacques Mangalaboyi, Patrick Menager, Scott E. Curtis, Claude Chopin, and Serge R. Mordon. "Changes in muscle tissue oxygenation assessed by near-infrared spectroscopy (NIRS)." In International Symposium on Biomedical Optics Europe '94, edited by Rinaldo Cubeddu, Renato Marchesini, Serge R. Mordon, Katarina Svanberg, Herbert H. Rinneberg, and Georges A. Wagnieres. SPIE, 1995. http://dx.doi.org/10.1117/12.198734.

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Lima, Fabiano, Cosme Buzzachera, Isis Grigoletto, Juliana Uzeloto, Juliano Gabardo, Dionei Ramos, Carlos Augusto Camillo, and Ercy Ramos. "Muscle oxygenation and hemodynamics after submaximal exercise in different COPD severities." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa1192.

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Reports on the topic "Muscle oxygenation"

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Holdsworth, Clark, Steven Copp, Tadakatsu Inagaki, Daniel Hirai, Scott Ferguson, Gabrielle Sims, Michael White, David Poole, and Timothy Musch. Chronic (-)-epicatechin administration does not affect contracting skeletal muscle microvascular oxygenation. Peeref, May 2022. http://dx.doi.org/10.54985/peeref.2206p3750191.

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