Relevant bibliographies by topics / Muscle contraction

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Muscle contraction'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 August 2024

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Journal articles on the topic "Muscle contraction"

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Conley, Kevin E., and Stan L. Lindstedt. "Energy-saving mechanisms in muscle: the minimization strategy." Journal of Experimental Biology 205, no. 15 (August 1, 2002): 2175–81. http://dx.doi.org/10.1242/jeb.205.15.2175.

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SUMMARYMany mechanisms reduce the cost of muscle activity. Here, we describe a set of specializations that reduce the cost of contraction in the high-frequency twitches that are used by a wide variety of animals for either sound production or flight. Minimizing the cost of these contractions means that cellular ATP production can meet ATP demand and sustain the high contractile rate. Two classes of specialization are found that minimize the contractile cost. The first class reduces the muscle work required per contraction. Light appendages such as rattles, insect limbs and membranous wings that require little work for movement are used in high-frequency contractions. The second set of specializations involves processes that minimize energy use. High-frequency muscles tend to have a lower cross-bridge content, fewer attached cross-bridges and shorter length changes per contraction. The result is low muscle-specific forces (stress), small length changes (strain) and rapid contraction times that suggest that these muscles push the lower limit of contractile function. The consequence of function at this lower extreme of contraction is to minimize the contractile cost of high-frequency muscles. Thus, specializations that permit rapid contractions at a low rate of ATP use per twitch are the basis of a minimization strategy for energy saving in muscles contracting at high frequency.
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Hogan, Michael C., Erica Ingham, and S. Sadi Kurdak. "Contraction duration affects metabolic energy cost and fatigue in skeletal muscle." American Journal of Physiology-Endocrinology and Metabolism 274, no. 3 (March 1, 1998): E397—E402. http://dx.doi.org/10.1152/ajpendo.1998.274.3.e397.

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It has been suggested that during a skeletal muscle contraction the metabolic energy cost at the onset may be greater than the energy cost related to holding steady-state force. The purpose of the present study was to investigate the effect of contraction duration on the metabolic energy cost and fatigue process in fully perfused contracting muscle in situ. Canine gastrocnemius muscle ( n = 6) was isolated, and two contractile periods (3 min of isometric, tetanic contractions with 45-min rest between) were conducted by each muscle in a balanced order design. The two contractile periods had stimulation patterns that resulted in a 1:3 contraction-to-rest ratio, with the difference in the two contractile periods being in the duration of each contraction: short duration 0.25-s stimulation/0.75-s rest vs. long duration 1-s stimulation/3-s rest. These stimulation patterns resulted in the same total time of stimulation, number of stimulation pulses, and total time in contraction for each 3-min period. Muscle O2 uptake, the fall in developed force (fatigue), the O2 cost of developed force, and the estimated total energy cost (ATP utilization) of developed force were significantly greater ( P < 0.05) with contractions of short duration. Lactate efflux from the working muscle and muscle lactate concentration were significantly greater with contractions of short duration, such that the calculated energy derived from glycolysis was three times greater in this condition. These results demonstrate that contraction duration can significantly affect both the aerobic and anaerobic metabolic energy cost and fatigue in contracting muscle. In addition, it is likely that the greater rate of fatigue with more rapid contractions was a result of elevated glycolytic production of lactic acid.
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Thomas, G. D., J. Hansen, and R. G. Victor. "Inhibition of alpha 2-adrenergic vasoconstriction during contraction of glycolytic, not oxidative, rat hindlimb muscle." American Journal of Physiology-Heart and Circulatory Physiology 266, no. 3 (March 1, 1994): H920—H929. http://dx.doi.org/10.1152/ajpheart.1994.266.3.h920.

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Previous studies have produced conflicting evidence as to whether sympathetic vasoconstriction is impaired in active skeletal muscle. Because alpha 2-, not alpha 1-, adrenergic vasoconstriction is attenuated by mild acidosis, we hypothesized that alpha 2-mediated sympathetic vasoconstriction would be attenuated in contracting glycolytic muscle, which produces more acidosis than oxidative muscle. We compared effects of lumbar sympathetic nerve stimulation and alpha-adrenergic agonists on arterial pressure, femoral blood flow, and force output during contractions of oxidative or glycolytic muscles in anesthetized rats. We found that 1) sympathetic vasoconstriction was preserved during contractions of oxidative soleus muscle and during low-intensity contractions of glycolytic gastrocnemiusplantaris muscles but was abolished during maximal contractions of these glycolytic muscles; 2) this sympatholytic effect was caused by impaired alpha 2-, not alpha 1-, vasoconstriction; and 3) the increased muscle blood flow resulting from a combination of impaired vasconstriction and increased arterial pressure was paralleled by increased force of gastrocnemius-plantaris muscle contraction. Thus contraction-induced impairment of alpha 2-vasoconstriction can augment muscle blood flow and muscle contraction, but the degree of impairment depends on fiber type and intensity of muscle contraction.
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Todd, Gabrielle, Janet L. Taylor, Jane E. Butler, Peter G. Martin, Robert B. Gorman, and Simon C. Gandevia. "Use of motor cortex stimulation to measure simultaneously the changes in dynamic muscle properties and voluntary activation in human muscles." Journal of Applied Physiology 102, no. 5 (May 2007): 1756–66. http://dx.doi.org/10.1152/japplphysiol.00962.2006.

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Force responses to transcranial magnetic stimulation of motor cortex (TMS) during exercise provide information about voluntary activation and contractile properties of the muscle. Here, TMS-generated twitches and muscle relaxation during the TMS-evoked silent period were measured in fresh, heated, and fatigued muscle. Subjects performed isometric contractions of elbow flexors in two studies. Torque and EMG were recorded from elbow flexor and extensor muscles. One study ( n = 6) measured muscle contraction times and relaxation rates during brief maximal and submaximal contractions in fresh and fatigued muscle. Another study ( n = 7) aimed to 1) assess the reproducibility of muscle contractile properties during brief voluntary contractions in fresh muscle, 2) validate the technique for contractile properties in passively heated muscle, and 3) apply the technique to study contractile properties during sustained maximal voluntary contractions. In both studies, muscle contractile properties during voluntary contractions were compared with the resting twitch evoked by motor nerve stimulation. Measurement of muscle contractile properties during voluntary contractions is reproducible in fresh muscle and reveals faster and slower muscle relaxation rates in heated and fatigued muscle, respectively. The technique is more sensitive to altered muscle state than the traditional motor nerve resting twitch. Use of TMS during sustained maximal contractions reveals slowing of muscle contraction and relaxation with different time courses and a decline in voluntary activation. Voluntary output from the motor cortex becomes insufficient to maintain complete activation of muscle, although slowing of muscle contraction and relaxation indicates that lower motor unit firing rates are required for fusion of force.
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Günther, Michael, Oliver Röhrle, Daniel F. B. Haeufle, and Syn Schmitt. "Spreading out Muscle Mass within a Hill-Type Model: A Computer Simulation Study." Computational and Mathematical Methods in Medicine 2012 (2012): 1–13. http://dx.doi.org/10.1155/2012/848630.

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It is state of the art that muscle contraction dynamics is adequately described by a hyperbolic relation between muscle force and contraction velocity (Hill relation), thereby neglecting muscle internal mass inertia (first-order dynamics). Accordingly, the vast majority of modelling approaches also neglect muscle internal inertia. Assuming that such first-order contraction dynamics yet interacts with muscle internal mass distribution, this study investigates two questions: (i) what is the time scale on which the muscle responds to a force step? (ii) How does this response scale with muscle design parameters? Thereto, we simulated accelerated contractions of alternating sequences of Hill-type contractile elements and point masses. We found that in a typical small muscle the force levels off after about 0.2 ms, contraction velocity after about 0.5 ms. In an upscaled version representing bigger mammals' muscles, the force levels off after about 20 ms, and the theoretically expected maximum contraction velocity is not reached. We conclude (i) that it may be indispensable to introduce second-order contributions into muscle models to understand high-frequency muscle responses, particularly in bigger muscles. Additionally, (ii) constructing more elaborate measuring devices seems to be worthwhile to distinguish viscoelastic and inertia properties in rapid contractile responses of muscles.
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Kato, Tatsuya, Atsushi Sasaki, Hikaru Yokoyama, Matija Milosevic, and Kimitaka Nakazawa. "Effects of neuromuscular electrical stimulation and voluntary commands on the spinal reflex excitability of remote limb muscles." Experimental Brain Research 237, no. 12 (October 10, 2019): 3195–205. http://dx.doi.org/10.1007/s00221-019-05660-6.

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Abstract It is well known that contracting the upper limbs can affect spinal reflexes of the lower limb muscle, via intraneuronal networks within the central nervous system. However, it remains unknown whether neuromuscular electrical stimulation (NMES), which can generate muscle contractions without central commands from the cortex, can also play a role in such inter-limb facilitation. Therefore, the objective of this study was to compare the effects of unilateral upper limb contractions using NMES and voluntary unilateral upper limb contractions on the inter-limb spinal reflex facilitation in the lower limb muscles. Spinal reflex excitability was assessed using transcutaneous spinal cord stimulation (tSCS) to elicit responses bilaterally in multiple lower limb muscles, including ankle and thigh muscles. Five interventions were applied on the right wrist flexors for 70 s: (1) sensory-level NMES; (2) motor-level NMES; (3) voluntary contraction; (4) voluntary contraction and sensory-level NMES; (5) voluntary contraction and motor-level NMES. Results showed that spinal reflex excitability of ankle muscles was facilitated bilaterally during voluntary contraction of the upper limb unilaterally and that voluntary contraction with motor-level NMES had similar effects as just contracting voluntarily. Meanwhile, motor-level NMES facilitated contralateral thigh muscles, and sensory-level NMES had no effect. Overall, our results suggest that inter-limb facilitation effect of spinal reflex excitability in lower limb muscles depends, to a larger extent, on the presence of the central commands from the cortex during voluntary contractions. However, peripheral input generated by muscle contractions using NMES might have effects on the spinal reflex excitability of inter-limb muscles via spinal intraneuronal networks.
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Stebbins, C. L., O. A. Carretero, T. Mindroiu, and J. C. Longhurst. "Bradykinin release from contracting skeletal muscle of the cat." Journal of Applied Physiology 69, no. 4 (October 1, 1990): 1225–30. http://dx.doi.org/10.1152/jappl.1990.69.4.1225.

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Results of previous studies from our laboratory suggest that bradykinin has a role in the exercise pressor reflex elicited by static muscle contraction. The purpose of this study was to quantify the release of bradykinin from contracting skeletal muscle. In 18 cats, blood samples were withdrawn directly from the venous effluent of the triceps surae muscles immediately before and after 30 s of static contraction producing peak muscle tensions of 33, 50, and 100% of maximum electrically stimulated contraction. Contractions producing muscle tensions of 50 and 100% of maximum increased muscle venous bradykinin levels by 27 +/- 9 and 19 +/- 10 pg/ml, respectively. Conversely, 33% maximum contraction did not alter muscle venous bradykinin concentrations. However, when captopril was administered to slow the degradation of bradykinin, muscle venous bradykinin increased from 68 +/- 15 pg/ml at rest to 106 +/- 18 after contractions of 33% of maximum. When muscle ischemia was induced by 2 min of arterial occlusion before and during 30 s of 33% of maximum contraction, muscle venous bradykinin increased by 15 +/- 5 pg/ml. In addition, contraction-induced changes in muscle venous pH and lactate strongly correlated with bradykinin concentrations (r = 0.80 and 0.83, respectively). These data demonstrate that static contraction of relatively high intensity evokes the release of bradykinin from skeletal muscle and that ischemia, decreased pH, and increased lactate are strongly correlated with this release.
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Hogan, Michael C., Bruno Grassi, Michele Samaja, Creed M. Stary, and L. B. Gladden. "Effect of contraction frequency on the contractile and noncontractile phases of muscle venous blood flow." Journal of Applied Physiology 95, no. 3 (September 2003): 1139–44. http://dx.doi.org/10.1152/japplphysiol.00226.2003.

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The purpose of this study was to test the hypothesis that increasing muscle contraction frequency, which alters the duty cycle and metabolic rate, would increase the contribution of the contractile phase to mean venous blood flow in isolated skeletal muscle during rhythmic contractions. Canine gastrocnemius muscle ( n = 5) was isolated, and 3-min stimulation periods of isometric, tetanic contractions were elicited sequentially at rates of 0.25, 0.33, and 0.5 contractions/s. The O2 uptake, tension-time integral, and mean venous blood flow increased significantly ( P < 0.05) with each contraction frequency. Venous blood flow during both the contractile (106 ± 6, 139 ± 8, and 145 ± 8 ml·100 g-1·min-1) and noncontractile phases (64 ± 3, 78 ± 4, and 91 ± 5 ml·100 g-1·min-1) increased with contraction frequency. Although developed force and duration of the contractile phase were never significantly different for a single contraction during the three contraction frequencies, the amount of blood expelled from the muscle during an individual contraction increased significantly with contraction frequency (0.24 ± 0.03, 0.32 ± 0.02, and 0.36 ± 0.03 ml·N-1·min-1, respectively). This increased blood expulsion per contraction, coupled with the decreased time in the noncontractile phase as contraction frequency increased, resulted in the contractile phase contribution to mean venous blood flow becoming significantly greater (21 ± 4, 30 ± 4, and 38 ± 6%) as contraction frequency increased. These results demonstrate that the percent contribution of the muscle contractile phase to mean venous blood flow becomes significantly greater as contraction frequency (and thereby duty cycle and metabolic rate) increases and that this is in part due to increased blood expulsion per contraction.
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Hespel, P., and E. A. Richter. "Mechanism linking glycogen concentration and glycogenolytic rate in perfused contracting rat skeletal muscle." Biochemical Journal 284, no. 3 (June 15, 1992): 777–80. http://dx.doi.org/10.1042/bj2840777.

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The influence of differences in glycogen concentration on glycogen breakdown and on phosphorylase activity was investigated in perfused contracting rat skeletal muscle. The rats were preconditioned by a combination of swimming exercise and diet (carbohydrate-free or carbohydrate-rich) in order to obtain four sub-groups of rats with varying resting muscle glycogen concentrations (range 10-60 mumol/g wet wt.). Pre-contraction muscle glycogen concentration was closely positively correlated with glycogen breakdown over 15 min of intermittent short tetanic contractions (r = 0.75; P less than 0.001; n = 56) at the same tension development and oxygen uptake. Additional studies in supercompensated and glycogen-depleted hindquarters during electrical stimulation for 20 s or 2 min revealed that the difference in glycogenolytic rate was found at the beginning rather than at the end of the contraction period. Phosphorylase alpha activity was approximately twice as high (P less than 0.001) in supercompensated muscles as in glycogen-depleted muscles after 20 s as well as after 2 min of contractions. It is concluded that glycogen concentration is an important determinant of phosphorylase activity in contracting skeletal muscle, and probably via this mechanism a regulator of glycogenolytic rate during muscle contraction.
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Herdmann, J., P. Enck, P. Zacchi-Deutschbein, and U. Ostermann. "Speed and pressure characteristics of external anal sphincter contractions." American Journal of Physiology-Gastrointestinal and Liver Physiology 269, no. 2 (August 1, 1995): G225—G231. http://dx.doi.org/10.1152/ajpgi.1995.269.2.g225.

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The principle of isochronism reflects constant contraction time for varying strengths of muscle contraction. This principle was studied for the innervation of the pelvic floor in humans using motor-evoked potentials (MEPs) and evoked pressure curves (EPCs) from the external anal sphincter muscle (EAS). MEPs and EPCs were simultaneously recorded after transcranial magnetic stimulation of the motor cortex. Voluntary contractions were also studied. Contraction times of the EAS were significantly longer in voluntary contractions (mean, 237 ms) than in EPCs (mean, 90 ms). Depending on either mode of contraction, contraction times varied only slightly despite a wide range of contraction strengths. It is shown that the contractile behavior of the EAS is a function of slow- and fast-twitch muscle fiber distribution and that the principle of isochronism governs motor performance not only of limb muscles but also of the EAS. There exists a unique optimal working range of each muscle to meet its individual function. Disturbance of this principle results in a less efficient contraction with either inappropriate basic tone or disturbed reflex activation in the EAS. Both are possible causes of incontinence.
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Dissertations / Theses on the topic "Muscle contraction"

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Murtada, Sae-Il. "Smooth muscle modeling activation and contraction of contractile units in smooth muscle /." Licentiate thesis, Stockholm : Skolan för teknikvetenskap, Kungliga Tekniska högskolan, 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:kth:diva-11349.

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Baker, Brent A. "Characterization of skeletal muscle performance and morphology following acute and chronic mechanical loading paradigms." Morgantown, W. Va. : [West Virginia University Libraries], 2007. https://eidr.wvu.edu/etd/documentdata.eTD?documentid=5325.

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Thesis (Ph. D.)--West Virginia University, 2007.
Title from document title page. Document formatted into pages; contains xii, 270 p. : ill. (some col.). Includes abstract. Includes bibliographical references.
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Pasquet, Benjamin. "Etude de la spécificité de la commande motrice et de sa régulation pendant différents types de contractions musculaires." Doctoral thesis, Universite Libre de Bruxelles, 2009. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/210280.

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Le but de cette dissertation doctorale était de mieux comprendre les mécanismes de contrôle tant centraux que périphériques qui sont à l’origine de la régulation neuromusculaire lors de mouvement impliquant des contractions de type excentrique. Lors d’une première étude réalisée sur le muscle jambier antérieur, nous avons montré qu’un exercice utilisant des contractions excentriques présentait une meilleure résistance à la fatigue que lorsque des contractions concentriques étaient impliquées puisque celui-ci conduit à une moindre diminution du couple de force et de l’activité électromyographique. L’absence de fatigue nerveuse centrale et l’observation d’un comportement spécifique du couple de force et de l’activité électromyographique lors de ces épreuves de fatigue semblait traduire la mise en jeu de processus périphériques différents. La plus grande fatigue observée lors de l’épreuve concentrique suggérait une activation plus importante que pour l’épreuve excentrique, dont les conséquences métaboliques renforcent les altérations du couplage excitation-contraction. Dans un second temps, nous avons étudié l’effet des modifications de longueur de fascicule du muscle jambier antérieur sur le comportement spécifique des unités motrices (ordre, fréquence et seuil de recrutement) lors de contractions isométriques. Nous avons ensuite analysé le comportement d’unités motrices selon les différentes modalités de contractions (concentrique vs. excentrique) sur ce même muscle. Pour y répondre, différentes techniques d’analyse ont été utilisées dont l’enregistrement électromyographique intramusculaire et l’ultrasonographie. Enfin, nous avons cherché à analyser l’évolution des différents mécanismes de régulation d’origine périphérique et /ou central susceptible de modifier l’excitabilité du pool de motoneurone lors de contractions concentriques et excentriques. Pour y répondre, les modulations d’une part, du réflexe de Hoffmann (réflexe H) par stimulation électrique et d’autre part, celles du potentiel moteur évoqué (MEP) par stimulation magnétique transcorticale, ont été investiguées. Ces réponses ont été enregistrées à différents angles de la plage articulaires étudiée lors des contractions concentriques et excentriques, ainsi qu’aux deux extrémités angulaires lors de contraction isométriques. Notre travail indique que l’ordre de recrutement des unités motrices entre les contractions concentriques et excentriques étant identique, le système nerveux n’utilise qu’une seule et même stratégie d’activation liée à la taille des motoneurones impliqués dans ces deux types de contractions. En outre, les contractions excentriques lorsqu’elles sont réalisées à vitesse constante, sont associées à une modulation spécifique de la fréquence de décharge des unités motrices. Ce comportement diffère de celui observé lors de contractions concentriques, malgré une modification linéaire et similaire de la longueur des fascicules et du couple de force au cours de ces deux tâches. Les modulations du recrutement des unités motrices semblent davantage dépendre de la longueur musculaire tandis que les modulations de fréquence prédominent pendant les contractions en raccourcissement. Ce comportement spécifique semble dépendant de mécanismes de régulation principalement localisés au niveau spinal. Ainsi, le degré d’inhibition des afférences fusoriales affectant le pool de motoneurones du muscle tibial antérieur lors de sollicitations actives du muscle, dépend davantage de l’angle articulaire et donc de la longueur du muscle plutôt que du mode de contraction. Lors de sollicitations isométriques, le retour sensoriel Ia est principalement contrôlé au niveau présynaptique en fonction de la longueur du muscle. Lors de sollicitations concentriques et excentriques, ces mécanismes présynaptiques réguleraient l'excitabilité spinale de manière similaire entre les deux modes. Néanmoins, bien que l'inhibition présynaptique soit probablement plus marquée lors des sollicitations excentriques, ce mode de contraction semble également régulé par des mécanismes d'inhibition intervenant au niveau postsynaptique tel que l'inhibition récurrente de Renshaw. Ce mécanisme localisé au niveau postsynaptique permettrait de réguler la fréquence de pulsation des unités motrices lors de sollicitations excentriques dans le but le faciliter l'exécution du mouvement. L'originalité de notre travail a été d’étudier le comportement d’une même unité dans les deux modes de contractions alors que la méthode d’analyse généralement adoptée consistait à comparer des populations d’unités motrices entre-elles. De plus, les changements de la longueur du muscle au cours du mouvement ainsi que les vitesses de raccourcissement ou d'allongement ont été estimés à partir de la mesure directe de la longueur des fascicules musculaires. Cette dernière présente l’avantage de fournir une information de longueur et de vitesse sur la portion de muscle à partir de laquelle les enregistrements d’unités motrices ont été obtenus. Enfin, étant donné les modulations possibles tant au niveau spinal que supraspinal des mécanismes nerveux mis en jeu, il semblait important d’analyser celles-ci pendant le mouvement et aux différents angles investigués. Cette précision méthodologique a permis d'élargir la discussion concernant les possibles modifications de la balance "excitation-inhibition" lors de sollicitations excentriques, qui, jusqu’à présent, n'avaient été analysées que pour un angle articulaire donné.
Doctorat en Sciences de la motricité
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Yeung, Wai Ella, and 楊慧. "Eccentric contraction-induced injury in mammalian skeletal muscle." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2003. http://hub.hku.hk/bib/B29750313.

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Monteiro, André Antonio. "Blood flow change in human masseter muscle elicited by voluntary isometric contraction." Stockholm : Kongl. Carolinska Medico Chirurgiska Institutet, 1990. http://catalog.hathitrust.org/api/volumes/oclc/21700760.html.

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Self, Brian P. "A control model of muscle contraction." Thesis, This resource online, 1991. http://scholar.lib.vt.edu/theses/available/etd-03022010-020135/.

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Lou, Fang. "A study of the contractile properties of vertebrate skeletal muscle with special reference to the force-velocity relationship and the cellular mechanisms of muscle fatigue /." Lund : Dept. of Pharmacology, University of Lund, 1994. http://books.google.com/books?id=zO9qAAAAMAAJ.

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Kawano, Yoji, Takeshi Yoshimura, and Kozo Kaibuchi. "Smooth muscle contraction by small GTPase Rho." Nagoya University School of Medicine, 2002. http://hdl.handle.net/2237/5374.

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Wadsworth, R. M. "Regulation of contraction of arterial smooth muscle." Thesis, University of Strathclyde, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.248764.

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Griffiths, R. H. ugh. "Modelling the Regulation of Skeletal Muscle Contraction." Thesis, University of Kent, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.499839.

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Books on the topic "Muscle contraction"

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Bagshaw, Clive R. Muscle Contraction. Dordrecht: Springer Netherlands, 1993. http://dx.doi.org/10.1007/978-94-015-6839-5.

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Rassier, Dilson E. Muscle biophysics: From molecules to cells. New York: Springer, 2010.

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1945-, Squire John, ed. Molecular mechanisms in muscular contraction. London: Macmillan, 1989.

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1945-, Squire John, ed. Molecular mechanisms in muscular contraction. Basingstoke: Macmillan, 1990.

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Rüegg, Johann Caspar. Calcium in Muscle Contraction. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-77560-4.

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Pollack, Gerald H. Muscles & molecules: Uncovering the principles of biological motion. Seattle, Wash: Ebner & Sons Publishers, 1990.

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Muscle, Symposium (2004 Tokyo Japan). Sliding filament mechanism in muscle contraction: Fifty years of research. New York: Springer, 2005.

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Muscle Symposium (2004 Tokyo, Japan). Sliding filament mechanism in muscle contraction: Fifty years of research. New York: Springer, 2005.

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1945-, Squire John, ed. Molecular mechanisms in muscular contraction. Boca Raton, Fla: CRC Press, 1990.

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1933-, Sugi Haruo, and Fujihara Kagaku Zaidan, eds. Molecular and cellular aspects of muscle contraction. New York: Kluwer Academic/Plenum Publishers, 2003.

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Book chapters on the topic "Muscle contraction"

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Bagshaw, Clive R. "Muscle cells." In Muscle Contraction, 21–32. Dordrecht: Springer Netherlands, 1993. http://dx.doi.org/10.1007/978-94-015-6839-5_3.

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Bagshaw, Clive R. "Introduction." In Muscle Contraction, 1–3. Dordrecht: Springer Netherlands, 1993. http://dx.doi.org/10.1007/978-94-015-6839-5_1.

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Bagshaw, Clive R. "Problems and prospects." In Muscle Contraction, 136–44. Dordrecht: Springer Netherlands, 1993. http://dx.doi.org/10.1007/978-94-015-6839-5_10.

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Bagshaw, Clive R. "Gross anatomy and physiology." In Muscle Contraction, 4–20. Dordrecht: Springer Netherlands, 1993. http://dx.doi.org/10.1007/978-94-015-6839-5_2.

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Bagshaw, Clive R. "Contractile proteins." In Muscle Contraction, 33–57. Dordrecht: Springer Netherlands, 1993. http://dx.doi.org/10.1007/978-94-015-6839-5_4.

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Bagshaw, Clive R. "Mechanism of ATP hydrolysis." In Muscle Contraction, 58–70. Dordrecht: Springer Netherlands, 1993. http://dx.doi.org/10.1007/978-94-015-6839-5_5.

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Bagshaw, Clive R. "Crossbridge structure and function." In Muscle Contraction, 71–94. Dordrecht: Springer Netherlands, 1993. http://dx.doi.org/10.1007/978-94-015-6839-5_6.

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Bagshaw, Clive R. "Mechanochemical coupling." In Muscle Contraction, 95–113. Dordrecht: Springer Netherlands, 1993. http://dx.doi.org/10.1007/978-94-015-6839-5_7.

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Bagshaw, Clive R. "Molecular basis of regulation." In Muscle Contraction, 114–25. Dordrecht: Springer Netherlands, 1993. http://dx.doi.org/10.1007/978-94-015-6839-5_8.

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Bagshaw, Clive R. "In vitro motility assays." In Muscle Contraction, 126–35. Dordrecht: Springer Netherlands, 1993. http://dx.doi.org/10.1007/978-94-015-6839-5_9.

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Conference papers on the topic "Muscle contraction"

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Chaves, Juliana N., Hani C. Yehia, and Henrique R. Martins. "Development of a portable labor-contraction monitor based on mechanomyography." In Simpósio Brasileiro de Computação Aplicada à Saúde. Sociedade Brasileira de Computação (SBC), 2022. http://dx.doi.org/10.5753/sbcas_estendido.2022.222422.

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Mechanomyography is a technique that measures the mechanical signal observed from the muscle surface during the contraction. It is expected to be a promising tool to evaluate uterine dynamics even though the uterus contraction is known to be involuntary. This study aimed to describe the development of a portable device based on mechanomyography to monitor uterine contraction. To assess the system was performed isometric contractions of the rectus femoris muscle with varying loads. The acquired data showed that the system could identify each contraction's onset, offset, and amplitude. The sensors and the system architecture proved to be a promising tool for the desired application.
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Milićević, Bogdan, Miloš Ivanović, Boban Stojanović, and Nenad Filipović. "HUXLEY SURROGATE MODEL FOR TWITCH MUSCLE CONTRACTION." In 1st INTERNATIONAL Conference on Chemo and BioInformatics. Institute for Information Technologies, University of Kragujevac,, 2021. http://dx.doi.org/10.46793/iccbi21.239m.

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Biophysical muscle models, often called Huxley-type models, are based on the underlying physiology of muscles, making them suitable for modeling non-uniform and unsteady contractions. This kind of model can be computationally intensive, which makes the usage of large-scale simulations difficult. To enable more efficient usage of the Huxley muscle model, we created a data-driven surrogate model, which behaves similarly to the original Huxley muscle model, but it requires significantly less computational power. From several numerical simulations, we acquired a lot of data and trained deep neural networks so that the behavior of the neural network resembles the behavior of the Huxley model. Since muscle models are history-dependent we used time series as an input and we trained a recurrent neural network to produce stress and instantaneous stiffness. The real challenge was to get the neural network to predict these values precisely enough for the numerical simulation to work properly and produce accurate results. In our work, we showed results obtained with the original Huxley model and surrogate Huxley model for several muscle twitch contractions. Based on similarities between the surrogate model and the original model we can conclude that the surrogate has the potential to replace the original model within numerical simulations.
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Palladino, Joseph L. "Modeling Mouse Soleus Muscle Contraction." In 2020 42nd Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC) in conjunction with the 43rd Annual Conference of the Canadian Medical and Biological Engineering Society. IEEE, 2020. http://dx.doi.org/10.1109/embc44109.2020.9176436.

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Palladino, Joseph L. "Canine Smooth Muscle Contraction Model." In 2022 44th Annual International Conference of the IEEE Engineering in Medicine & Biology Society (EMBC). IEEE, 2022. http://dx.doi.org/10.1109/embc48229.2022.9871599.

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Sandler, Reuben, and Stephen N. Robinovitch. "Impact Severity During a Fall Is Decreased by Lower Extremity Muscle Contractions During Descent." In ASME 1998 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 1998. http://dx.doi.org/10.1115/imece1998-0084.

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Abstract Falls are a major cause of injury in the elderly, including the vast majority of hip and wrist fractures, and a considerable portion of vertebral fractures. In the event of a fall, one’s risk for such an injury depends on bone strength, and the configuration and kinetic energy (KE) of the body at the instant it contacts the ground. Consideration of acts such as sitting and squatting suggests that a major determinant of KE is the amount of energy “absorbed” (or negative work performed) during the descent phase of the fall by eccentrically-contracting lower extremity muscles. In the present study, we used a computational model of backwards falling to address the following questions: (1) what degree of impact energy attenuation might be achieved through muscle contraction during the descent phase of falling? and (2) what is the relative importance of muscle contractions at the hip, knee, and ankle in attenuating impact energy?
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Patel, Harshil, Gerald O’Neill, and Panagiotis Artemiadis. "Regulation of 3D Human Arm Impedance Through Muscle Co-Contraction." In ASME 2013 Dynamic Systems and Control Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/dscc2013-3942.

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Humans have the inherent ability of performing highly dexterous and skillful tasks with their arms, involving maintenance of posture, movement, and interaction with the environment. The latter requires the human to control the dynamic characteristics of the upper limb musculoskeletal system. These characteristics are quantitatively represented by inertia, damping, and stiffness, which are measures of mechanical impedance. Many previous studies have shown that arm posture is a dominant factor in determining the end point impedance on a horizontal (transverse) plane. This paper presents the characterization of the end point impedance of the human arm in three-dimensional space. Moreover, it models the regulation of the arm impedance with respect to various levels of muscle co-contraction. The characterization is made by route of experimental trials where human subjects maintained arm posture while their arms were perturbed by a robot arm. Furthermore, the subjects were asked to control the level of their arm muscles’ co-contraction, using visual feedback of their muscles’ activation, in order to investigate the effect of this muscle co-contraction on the arm impedance. The results of this study show a very interesting, anisotropic increase of arm stiffness due to muscle co-contraction. These results could lead to very useful conclusions about the human’s arm biomechanics, as well as many implications for human motor control-specifically the control of arm impedance through muscle co-contraction.
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Nagel, Vincent, Sarah Chu, Jack Forney, Lyle Kosinski, and Vimal Viswanathan. "Design and Control of an Assistive Bionic Joint for Leg Muscle Rehabilitation." In ASME 2017 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2017. http://dx.doi.org/10.1115/imece2017-71143.

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This project aims to create an electronically powered and controlled knee brace to aid stroke victims with partial paralysis with their leg muscle rehabilitation process. The newly designed assistive bionic joint takes the functionality of the existing assistive knee braces to the next level by incorporating a control algorithm that uses sensor signals gathered from the patient’s leg muscles. Electromyography (EMG) is used for gathering impulse signals from electrodes placed on key muscles as inputs for the device. The action of each major leg muscle is replicated using a set of fluidic muscles that mimic the functionality of the actual leg muscles. A microcontroller is used to interpret sensor data and adjust the contraction length of the muscles, thereby providing the wearer with augmented strength and mobility. Initial testing of a proof-of-concept prototype has led to finite control over muscle contraction length based on sensor data and has a response time of 280ms from full extension to contraction. Further testing of the brace assembly, fluidic muscles and control system is conducted and the results indicate a 600ms response time due to a step input. This personalized, powered brace has many implications for the enrichment of muscle rehabilitation such as higher patient morale, more muscle activity, and shortened recovery times.
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Kamarudin, Nor H., Siti A. Ahmad, Mohd K. Hassan, Rosnah M. Yusoff, and Siti Z. Dawal. "Muscle contraction analysis during lifting task." In 2014 IEEE Conference on Biomedical Engineering and Sciences (IECBES). IEEE, 2014. http://dx.doi.org/10.1109/iecbes.2014.7047540.

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Jaramillo, Paola, Adam Shoemaker, Alexander Leonessa, and Robert W. Grange. "Skeletal Muscle Contraction in Feedback Control." In ASME 2012 5th Annual Dynamic Systems and Control Conference joint with the JSME 2012 11th Motion and Vibration Conference. ASME, 2012. http://dx.doi.org/10.1115/dscc2012-movic2012-8592.

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Jansen, Sepp R., Marieke van Ziel, Hoeke A. Baarsma, and Reinoud Gosens. "²-catenin Regulates Airway Smooth Muscle Contraction." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a5292.

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Reports on the topic "Muscle contraction"

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Westerlind, Kim. Muscle Contraction Arrests Tumor Growth. Fort Belvoir, VA: Defense Technical Information Center, September 2006. http://dx.doi.org/10.21236/ada572645.

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Jalil, Yorschua, and Ruvistay Gutierrez. Myokines secretion and their role in critically ill patients. A scoping review protocol. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, September 2021. http://dx.doi.org/10.37766/inplasy2021.9.0048.

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Review question / Objective: 1-How and by which means stimulated muscle from critically ill patients can liberate myokines?, 2-Which are the main characteristics of the critically ill population studied and if some of these influenced myokine´s secretion?, 5-Can myokines exert local or distant effects in critically ill patients?, 5-Which are the potential effects of myokines in critically ill patients? Eligibility criteria: Participants and context: We will include primary studies (randomized or non-randomized trials, observational studies, case series or case report) that consider hospitalized critically ill adult patients (18 years or older) in risk for developing some degree of neuromuscular disorders such as ICU-AW, diaphragmatic dysfunction, or muscle weakness, therefore the specific setting will be critical care. Concept: This review will be focused on studies regarding the secretion or measure of myokines or similar (exerkines, cytokines or interleukin) by any mean of muscle activation or muscle contraction such as physical activity, exercise or NMES, among others. The latter strategies must be understood as any mean by which muscle, and there for myocytes, are stimulated as result of muscle contraction, regardless of the frequency, intensity, time of application and muscle to be stimulated (upper limb, lower limb, thoracic or abdominal muscles). We also will consider myokine´s effects, local or systemic, over different tissues in terms of their structure or function, such as myocytes function, skeletal muscle mass and strength, degree of muscle wasting or myopathies, among others.
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Comeaux, James A., James R. Jauchem, David D. Cox, Carrie C. Crane, and John A. D'Andrea. Muscle Contraction During Electro-Muscular Incapacitation: A Comparison Between Square-Wave Pulses and the Taser (registered trademark) X26 Electronic Control Device. Fort Belvoir, VA: Defense Technical Information Center, February 2009. http://dx.doi.org/10.21236/ada597215.

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Holdsworth, Clark, Steven Copp, Tadakatsu Inagaki, Daniel Hirai, Scott Ferguson, Gabrielle Sims, Michael White, David Poole, and Timothy Musch. Chronic (-)-epicatechin administration does not affect contracting skeletal muscle microvascular oxygenation. Peeref, May 2022. http://dx.doi.org/10.54985/peeref.2206p3750191.

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Hosseini, Jeanette M. Trinitrobenzenesulfonic Acid Colitis Induces Changes in the Contractile Response of Circular Smooth Muscle in the Distal Colon. Fort Belvoir, VA: Defense Technical Information Center, February 1996. http://dx.doi.org/10.21236/ad1011516.

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Rafaeli, Ada, and Russell Jurenka. Molecular Characterization of PBAN G-protein Coupled Receptors in Moth Pest Species: Design of Antagonists. United States Department of Agriculture, December 2012. http://dx.doi.org/10.32747/2012.7593390.bard.

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The proposed research was directed at determining the activation/binding domains and gene regulation of the PBAN-R’s thereby providing information for the design and screening of potential PBAN-R-blockers and to indicate possible ways of preventing the process from proceeding to its completion. Our specific aims included: (1) The identification of the PBAN-R binding domain by a combination of: (a) in silico modeling studies for identifying specific amino-acid side chains that are likely to be involved in binding PBAN with the receptor and; (b) bioassays to verify the modeling studies using mutant receptors, cell lines and pheromone glands (at tissue and organism levels) against selected, designed compounds to confirm if compounds are agonists or antagonists. (2) The elucidation ofthemolecular regulationmechanisms of PBAN-R by:(a) age-dependence of gene expression; (b) the effect of hormones and; (c) PBAN-R characterization in male hair-pencil complexes. Background to the topic Insects have several closely related G protein-coupled receptors (GPCRs) belonging to the pyrokinin/PBAN family, one with the ligand pheromone biosynthesis activating neuropeptide or pyrokinin-2 and another with diapause hormone or pyrokinin-1 as a ligand. We were unable to identify the diapause hormone receptor from Helicoverpa zea despite considerable effort. A third, related receptor is activated by a product of the capa gene, periviscerokinins. The pyrokinin/PBAN family of GPCRs and their ligands has been identified in various insects, such as Drosophila, several moth species, mosquitoes, Triboliumcastaneum, Apis mellifera, Nasoniavitripennis, and Acyrthosiphon pisum. Physiological functions of pyrokinin peptides include muscle contraction, whereas PBAN regulates pheromone production in moths plus other functions indicating the pleiotropic nature of these ligands. Based on the alignment of annotated genomic sequences, the primary and secondary structures of the pyrokinin/PBAN family of receptors have similarity with the corresponding structures of the capa or periviscerokinin receptors of insects and the neuromedin U receptors found in vertebrates. Major conclusions, solutions, achievements Evolutionary trace analysisof receptor extracellular domains exhibited several class-specific amino acid residues, which could indicate putative domains for activation of these receptors by ligand recognition and binding. Through site-directed point mutations, the 3rd extracellular domain of PBAN-R was shown to be critical for ligand selection. We identified three receptors that belong to the PBAN family of GPCRs and a partial sequence for the periviscerokinin receptor from the European corn borer, Ostrinianubilalis. Functional expression studies confirmed that only the C-variant of the PBAN-R is active. We identified a non-peptide agonist that will activate the PBAN-receptor from H. zea. We determined that there is transcriptional control of the PBAN-R in two moth species during the development of the pupa to adult, and we demonstrated that this transcriptional regulation is independent of juvenile hormone biosynthesis. This transcriptional control also occurs in male hair-pencil gland complexes of both moth species indicating a regulatory role for PBAN in males. Ultimate confirmation for PBAN's function in the male tissue was revealed through knockdown of the PBAN-R using RNAi-mediated gene-silencing. Implications, both scientific and agricultural The identification of a non-peptide agonist can be exploited in the future for the design of additional compounds that will activate the receptor and to elucidate the binding properties of this receptor. The increase in expression levels of the PBAN-R transcript was delineated to occur at a critical period of 5 hours post-eclosion and its regulation can now be studied. The mysterious role of PBAN in the males was elucidated by using a combination of physiological, biochemical and molecular genetics techniques.
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