Relevant bibliographies by topics / Muscle blood flow

Academic literature on the topic 'Muscle blood flow'

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Published: 4 June 2021
Last updated: 20 February 2023

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Journal articles on the topic "Muscle blood flow"

1

Hussain, Sabah N. A. "Regulation of ventilatory muscle blood flow." Journal of Applied Physiology 81, no. 4 (October 1, 1996): 1455–68. http://dx.doi.org/10.1152/jappl.1996.81.4.1455.

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Hussain, Sabah N. A. Regulation of ventilatory muscle blood flow. J. Appl. Physiol. 81(4): 1455–1468, 1996.—The ventilatory muscles perform various functions such as ventilation of the lungs, postural stabilization, and expulsive maneuvers (e.g., coughing). They are classified in functional terms as inspiratory muscles, which include the diaphragm, parasternal intercostal, external intercostal, scalene, and sternocleidomastoid muscles; and expiratory muscles, which include the abdominal muscles, internal intercostal, and triangularis sterni. The ventilatory muscles require high-energy phosphate compounds such as ATP to fuel the biochemical and physical processes of contraction and relaxation. Maintaining adequate intracellular concentrations of these compounds depends on adequate intracellular substrate levels and delivery of these substrates by arterial blood flow. In addition to the delivery of substrates, blood flow influences muscle function through the removal of metabolic by-products, which, if accumulated, could exert negative effects on several excitatory and contractile processes. Skeletal muscle substrate utilization is also dependent on the ability to extract substrates from arterial blood, which, in turn, is accomplished by increasing the total number of perfused capillaries. It follows that matching perfusion to metabolic demands is critical for the maintenance of normal muscle contractile function. In this article, I review the factors that influence ventilatory muscle blood flow. Major emphasis is placed on the diaphragm because a large number of published reports deal with diaphragmatic blood flow. The second reason for focusing on the diaphragm is because it is the largest and most important inspiratory muscle.
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Delp, Michael D., Changping Duan, Chester A. Ray, and R. B. Armstrong. "Rat hindlimb muscle blood flow during level and downhill locomotion." Journal of Applied Physiology 86, no. 2 (February 1, 1999): 564–68. http://dx.doi.org/10.1152/jappl.1999.86.2.564.

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During eccentrically biased exercise (e.g., downhill locomotion), whole body oxygen consumption and blood lactate concentrations are lower than during level locomotion. These general systemic measurements indicate that muscle metabolism is lower during downhill exercise. This study was designed to test the hypothesis that hindlimb muscle blood flow is correspondingly lower during downhill vs. level exercise. Muscle blood flow (determined by using radioactive microspheres) was measured in rats after 15 min of treadmill exercise at 15 m/min on the level (L, 0°) or downhill (D, −17°). Blood flow to ankle extensor muscles was either lower (e.g., white gastrocnemius muscle: D, 9 ± 2; L, 15 ± 1 ml ⋅ min−1 ⋅ 100 g−1) or not different (e.g., soleus muscle: D, 250 ± 35; L, 230 ± 21 ml ⋅ min−1 ⋅ 100 g−1) in downhill vs. level exercise. In contrast, blood flow to ankle flexor muscles was higher (e.g., extensor digitorum longus muscle: D, 53 ± 5; L, 31 ± 6 ml ⋅ min−1 ⋅ 100 g−1) during downhill vs. level exercise. When individual extensor and flexor muscle flows were summed, total flow to the leg was lower during downhill exercise (D, 3.24 ± 0.08; L, 3.47 ± 0.05 ml/min). These data indicate that muscle blood flow and metabolism are lower during eccentrically biased exercise but are not uniformly reduced in all active muscles; i.e., flows are equivalent in several ankle extensor muscles and higher in ankle flexor muscles.
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Elia, M., and A. Kurpad. "What is the Blood Flow to Resting Human Muscle?" Clinical Science 84, no. 5 (May 1, 1993): 559–63. http://dx.doi.org/10.1042/cs0840559.

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1. An investigation was carried out in five healthy lean adults to assess whether forearm and calf plethysmography largely reflect muscle blood flow as measured by 133Xe and whether there is substantial variability in the blood flow to muscles located at different sites in the body. 2. Blood flow to forearm and calf flexors and extensors, biceps, triceps and quadriceps was assessed using the 133Xe clearance technique. Blood flow to forearm skin and subcutaneous adipose tissue was also measured using the 133Xe clearance technique, whereas blood flow to the forearm and calf was measured using strain gauge plethysmography. 3. The mean blood flow to different muscles ranged from 1.4 ± 0.6 (gastrocnemius) to 1.8 ± 0.7 (forearm extensor) ml min−1 100 g−1 muscle (1.4 ± 0.6 and 1.9 ± 0.8 ml min−1 100 ml−1 muscle, respectively) but there were no significant differences between them. Forearm and calf blood flows (2.7 ± 0.3 and 3.0 ± 0.7 ml min−1 100 ml−1 limb tissue, respectively) were about 50% to more than 100% greater (P <0.025) than blood flow to the muscles within them (1.7 ± 0.5 and 1.4 ± 0.5ml min−1 100g−1 muscle, respectively, or 1.8 ± 0.6 and 1.5 ± 0.5 ml min−1 100 ml−1 muscle, respectively). In contrast, the blood flows to 100 g of forearm skin (9.1 ± 2.6 ml min−1 100 g−1) and adipose tissue (3.8 ± 1.1 ml min−1 100 g−1) were higher than the blood flow to 100 g of forearm (P <0.01 and not significant, respectively). 4. Although several possibilities can explain the discrepancy between muscle blood flow measured by 133Xe and blood flow to the distal limbs measured by plethysmography, the results suggest that non-muscular blood flow, especially that to skin, is substantially greater than muscular blood flow. Indeed, the overall blood flow to the forearm could be accounted for by summation of blood flows to individual constituent tissues, which were assumed to be present in proportions typical of lean subjects. The results have important implications in the use of arteriovenous catheterization studies for assessing flux of oxygen, carbon dioxide and metabolites across muscle.
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Armstrong, R. B., M. D. Delp, E. F. Goljan, and M. H. Laughlin. "Distribution of blood flow in muscles of miniature swine during exercise." Journal of Applied Physiology 62, no. 3 (March 1, 1987): 1285–98. http://dx.doi.org/10.1152/jappl.1987.62.3.1285.

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The purpose of this study was to determine how the distribution of blood flow within and among the skeletal muscles of miniature swine (22 +/- 1 kg body wt) varies as a function of treadmill speed. Radiolabeled microspheres were used to measure cardiac output (Q) and tissue blood flows in preexercise and at 3–5 min of treadmill exercise at 4.8, 8.0, 11.3, 14.5, and 17.7 km/h. All pigs (n = 8) attained maximal O2 consumption (VO2max) (60 +/- 4 ml X min-1 X kg-1) by the time they ran at 17.7 km/h. At VO2max, 87% of Q (9.9 +/- 0.5 l/min) was to skeletal muscle, which constituted 36 +/- 1% of body mass. Average total muscle blood flow at VO2max was 127 +/- 14 ml X min-1 X 100 g-1; average limb muscle flow was 135 +/- 17 ml X min-1 X 100 g-1. Within the limb muscles, blood flow was distributed so that the deep red parts of extensor muscles had flows about two times higher than the more superficial white portions of the same muscles; the highest muscle blood flows occurred in the elbow flexors (brachialis: 290 +/- 44 ml X min-1 X 100 g-1). Peak exercise blood flows in the limb muscles were proportional (P less than 0.05) to the succinate dehydrogenase activities (r = 0.84), capillary densities (r = 0.78), and populations of oxidative (slow-twitch oxidative + fast-twitch oxidative-glycolytic) fiber types (r = 0.93) in the muscles. Total muscle blood flow plotted as a function of exercise intensity did not peak until the pigs attained VO2max, although flows in some individual muscles showed a plateau in this relationship at submaximal exercise intensities. The data demonstrate that blood flow in skeletal muscles of miniature swine is distributed heterogeneously and varies in relation to fiber type composition and exercise intensity.
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Supinski, Gerald S. "Respiratory Muscle Blood Flow." Clinics in Chest Medicine 9, no. 2 (June 1988): 211–23. http://dx.doi.org/10.1016/s0272-5231(21)00500-1.

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Glenn, G. M., M. H. Laughlin, and R. B. Armstrong. "Muscle blood flow and fiber activity in partially curarized rats during exercise." Journal of Applied Physiology 63, no. 4 (October 1, 1987): 1450–56. http://dx.doi.org/10.1152/jappl.1987.63.4.1450.

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We previously reported that low doses of d-tubocurarine attenuated glycogen loss in red muscles of rats during treadmill walking but that the initial hyperemia in the muscles was normal. The present studies were performed to 1) determine with electromyography (EMG) whether red muscle fiber activity is reduced in walking, curarized rats and 2) study muscle blood flow and glycogen loss during running with different doses of curare (dose response). At 0.5 min of treadmill walking (15 m/min), integrated EMG in vastus intermedius (VI) muscle was reduced by an average of 18% in curarized (60 micrograms/kg) rats, although blood flow (measured with microspheres) was the same as in saline control rats. Comparison of blood flows and glycogen loss in quadriceps muscles at 1 min of treadmill running (30 m/min) with different curare doses (20–60 micrograms/kg) demonstrated that red muscle glycogen loss was inversely related to curare dose but that blood flows in the same muscles were unaffected by curare. These findings provide support for our previous conclusion that at the initiation of low to moderate treadmill exercise, red muscle blood flow is not proportional to the activity or metabolism of the muscle fibers.
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Armstrong, R. B., and M. H. Laughlin. "Rat muscle blood flows during high-speed locomotion." Journal of Applied Physiology 59, no. 4 (October 1, 1985): 1322–28. http://dx.doi.org/10.1152/jappl.1985.59.4.1322.

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We previously studied blood flow distribution within and among rat muscles as a function of speed from walking (15 m/min) through galloping (75 m/min) on a motor-driven treadmill. The results showed that muscle blood flows continued to increase as a function of speed through 75 m/min. The purpose of the present study was to have rats run up to maximal treadmill speeds to determine if blood flows in the muscles reach a plateau as a function of running speed over the animals' normal range of locomotory speeds. Muscle blood flows were measured with radiolabeled microspheres at 1 min of running at 75, 90, and 105 m/min in male Sprague-Dawley rats. The data indicate that even at these relatively high treadmill speeds there was still no clear evidence of a plateau in blood flow in most of the hindlimb muscles. Flows in most muscles continued to increase as a function of speed. These observed patterns of blood flow vs. running speed may have resulted from the rigorous selection of rats that were capable of performing the high-intensity exercise and thus only be representative of a highly specific population of animals. On the other hand, the data could be interpreted to indicate that the cardiovascular potential during exercise is considerably higher in laboratory rats than has normally been assumed and that inadequate blood flow delivery to the muscles does not serve as a major limitation to their locomotory performance.
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Laughlin, M. H. "Skeletal muscle blood flow capacity: role of muscle pump in exercise hyperemia." American Journal of Physiology-Heart and Circulatory Physiology 253, no. 5 (November 1, 1987): H993—H1004. http://dx.doi.org/10.1152/ajpheart.1987.253.5.h993.

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An appreciation for the potential of skeletal muscle vascular beds for blood flow (blood flow capacity) is required if one is to understand the limits of the cardiorespiratory system in exercise. To assess this potential, an index of blood flow capacity that can be objectively measured is required. One obvious index would be to measure maximal muscle blood flow (MBF). However, a unique value for maximal MBF cannot be measured, since once maximal vasodilation is attained MBF is a function of perfusion pressure. Another approach would be to measure maximal or peak vascular conductance. However, peak vascular conductance is different among skeletal muscles composed of different fiber types and is a function of perfusion pressure during peak vasodilation within muscle composed of a given fiber type. Also, muscle contraction can increase or decrease blood flow and/or the apparent peak vascular conductance depending on the experimental preparation and the type of muscle contraction. Blood flows and calculated values of conductance appear to be greater during rhythmic contractions (with the appropriate frequency and duration) than observed in resting muscle during what is called "maximal" vasodilation. Moreover, dynamic exercise in conscious subjects produces the greatest skeletal muscle blood flows. The purpose of this review is to consider the interaction of the determinants of muscle blood flow during locomotory exercise. Emphasis is directed toward the hypothesis that the "muscle pump" is an important determinant of perfusion of active skeletal muscle. It is concluded that, during normal dynamic exercise, MBF is determined by skeletal muscle vascular conductance, the perfusion pressure gradient, and the efficacy of the muscle pump.
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Klabunde, R. E., M. H. Laughlin, and R. B. Armstrong. "Systemic adenosine deaminase administration does not reduce active hyperemia in running rats." Journal of Applied Physiology 64, no. 1 (January 1, 1988): 108–14. http://dx.doi.org/10.1152/jappl.1988.64.1.108.

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The importance of adenosine in controlling the magnitude and distribution of blood flow among and within skeletal muscles in rats during slow locomotor exercise was tested by systemic infusion of adenosine deaminase (ADA). Blood flows were measured using labeled microspheres before exercise and at 0.5, 15, and 30 min of fast treadmill walking at 15 m/min. An initial infusion of ADA (1,000 U/kg) was given 30 min before the first blood flow measurement and a second injection (1,000 U/kg) was given 5 min into exercise. These infusions maintained ADA activity above 5 U/ml blood throughout the experimental period. This plasma concentration of ADA was shown to be sufficient to result in a 64% decrease in muscle adenosine levels during ischemic contraction. Blood flows were measured in all of the muscles of the hindlimb (28 samples) and in various nonmuscular tissues in ADA-treated and control rats. Preexercise blood flows were primarily directed to slow-twitch muscles and exercise blood flows were highest in muscles with fast-twitch oxidative fibers. ADA treatment did not reduce total muscle blood flow or exercise blood flows in any of the muscles at any time. These findings do not support the hypothesis that adenosine plays an essential role in controlling muscle blood flow in skeletal muscles during normal locomotor activity.
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Musch, T. I., D. B. Friedman, K. H. Pitetti, G. C. Haidet, J. Stray-Gundersen, J. H. Mitchell, and G. A. Ordway. "Regional distribution of blood flow of dogs during graded dynamic exercise." Journal of Applied Physiology 63, no. 6 (December 1, 1987): 2269–77. http://dx.doi.org/10.1152/jappl.1987.63.6.2269.

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The regional blood flow response to progressive treadmill exercise was measured with radioactive microspheres in 25 untrained mongrel dogs. Incremental increases in work intensity resulted in corresponding increases in blood flows to the gracilis, gastrocnemius, semimembranosus, and semitendinosus muscles of the hindlimb and to the heart. During maximal exercise, blood flow was greatest in the semimembranosus muscle and lowest in the semitendinosus muscle (342 and 134 ml–1.100 g tissue-1.min-1, respectively). Exercise produced a decrease in blood flow to the temporalis muscle, which was classified as nonlocomotive in function. Blood flows to the stomach, pancreas, and large intestine decreased at the lowest exercise work load and remained diminished throughout the continuum to maximal exercise. Blood flows to the small intestine and spleen were maintained during submaximal exercise but were reduced by 50% at maximal O2 consumption (VO2max). No changes in blood flows to the kidneys, adrenal glands, liver, and brain were found. These results demonstrate that 1) renal blood flow is maintained at resting levels during exercise in untrained dogs; 2) blood flow changes in the various organs of the splanchnic region of dogs during exercise are heterogeneous; and 3) blood flows to the working skeletal muscles of dogs progressively increase with increasing work loads up to VO2max.
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Dissertations / Theses on the topic "Muscle blood flow"

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Lutjemeier, Barbara June. "Control of muscle blood flow during dynamic exercise : muscle contraction / blood flow interactions." Diss., Manhattan, Kan. : Kansas State University, 2006. http://hdl.handle.net/2097/244.

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Monteiro, André Antonio. "Blood flow change in human masseter muscle elicited by voluntary isometric contraction." Stockholm : Kongl. Carolinska Medico Chirurgiska Institutet, 1990. http://catalog.hathitrust.org/api/volumes/oclc/21700760.html.

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Naamani, Randa. "The mechanical effects of muscle contractions of muscle blood flow /." Thesis, McGill University, 1990. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=60010.

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To determine whether muscle contractions can increase muscle blood flow independently from metabolic factors, we isolated the diaphragmatic vasculature of 16 anesthetized and mechanically ventilated dogs. Phrenic inflow (Qphr) was controlled with a constant pressure source and the pressure (Pa) was decreased in steps to obtain the pressure-flow relation (P-Q). The vasculture was maximally vasodilated and contractions occurred spontaneously (n = 6) or were induced by twitches (n = 12) or tetanic trains (n = 7). The P-Q relations with contractions were compared to those with vasodilatation alone. With spontaneous contractions, the pressure intercept decreased from 47.35 $ pm$ 17.44 to 33.77 $ pm$ 16.82 mmHg (p $<$ 0.05) and the slope remained unchanged so that at Pa = 100 mmHg, Qphr increased from 36.22 $ pm$ 34.85 to 43.91 $ pm$ 38.22 ml/min/100g (p $<$ 0.05). Flow increased slightly with twitches but not with trains. We also elicited twitches, 12/min and 60/min trains in vascularly isolated gastrocnemius muscles (n = 6) and found no change in flow. In conclusion, the muscle pump has only a small effect on muscle blood flow.
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Harper, Allison Jessica. "Adequacy of Muscle Blood Flow During Handgrip Exercise." University of Toledo / OhioLINK, 2009. http://rave.ohiolink.edu/etdc/view?acc_num=toledo1260580537.

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Hoy, Andrew James. "Nutritive and non-nutritive blood flow in skeletal muscle." Department of Biomedical Science - Faculty of Health & Behavioural Sciences, 2004. http://ro.uow.edu.au/theses/232.

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The vascular structure of mammalian skeletal muscle has been intensively investigated for the last seventy years. Since the early work of Pappenheimer and Barlow, the existence of a parallel dual vascular pathway has been suggested to explain the differences between total flow and clearance rates of a variety of infused substances. Direct microscopy studies using superficial muscles have shown that the microvascular arterioles have very frequent connections with the capillary modules of the associated connective tissue and adipose tissue within skeletal muscle. In more recent times, Clark and colleagues have identified two vascular pathways according to the opposing actions of two groups of vasoconstricting agents. While all increase perfusion pressure, Type A vasoconstrictors (low dose noradrenaline (Nad), vasopressin, angiotensin II) increase oxygen uptake but Type B vasoconstrictors (serotonin, high dose noradrenaline) decrease hindlimb oxygen consumption. The opposing effects on oxygen consumption are thought to arise from selective vasoconstriction of the mircrovasculature. Type A vasoconstrictors redirect blood into muscle tissue capillary beds (termed nutritive bed) whilst Type B vasoconstrictors redirect blood into the associated connective tissue, adipose and septum capillary beds (termed non-nutritive bed). Many of the previous studies are based on variations of an in situ rat, isolated perfused hindlimb model, having low vascular tone and often with insufficient oxygen carrying capacity to support active metabolism. In vivo, skeletal intramuscular blood redistribution during exercise occurs principally via the release of vasodilatory metabolites and the nervous system. This thesis used a novel in vivo model to test the hypothesis that nutritive and non nutritive blood flow distribution can still be observed under conditions of high vascular tone and oxygen delivery at rest and in metabolically active (contracting) muscle. Utilising the high vascular tone, it also tests the hypothesis that the vascular pathways can be differentiated using vasodilators. Male Wistar rats were anaesthetised with sodium pentobarbital (6mg.100g(superscript �1) i.p.). The right femoral artery was cannulated to supply blood to the left femoral artery (perfused) at a constant flow (basal 1ml.min(superscript �1), contraction 2ml.min(superscript �1) via a pump. Perfused hindlimb pressure was recorded distal to the pump and passive venous return occurred from the left femoral vein to the right external jugular vein. Systemic blood pressure was recorded from the left common carotid artery. Polyethylene cannulae were filled with heparinized 0.9% saline containing 6% w/v dextran70. The left sciatic nerve was isolated and stimulated (5Hz) to produce twitch contraction in the lower hindlimb muscle bundle and developed tension was recorded. Vasoactive drugs (2 constrictor, 8 dilator) were prepared with saline and 0.01% ascorbic acid, and injected into the arterial loop. Blood was sampled from the venous and arterial loops and oxygen consumption determined using the Fick equation. In the autoperfused rat hindlimb, the Type B vasoconstrictor increased perfusion pressure and caused a significant decrease in basal hindlimb oxygen consumption, however during muscle contraction this effect on oxygen consumption was diminished. The Type A vasoconstrictor had no significant effect on hindlimb oxygen consumption during significant increases in perfusion pressure. Eight vasodilators with a variety of mechanisms of action were screened at rest but none were observed to decreases hindlimb oxygen consumption in a fashion similar to Type B vasoconstrictors. Increases in oxygen availability at rest via increased nutritive flow by noradrenaline and vasodilator infusion had no effect upon basal metabolic rate. Therefore, during adequate oxygen delivery, increased availability has no effect upon metabolic demand. Isoprenaline and histamine significantly increased hindlimb oxygen consumption during the contraction protocol, whilst there was no significant effect observed at rest. It can be concluded that selective vasoconstriction metabolites can overcome exogenous vasoconstriction. These results confirm the possible existence of a dual vascular pathway however blood flow redistribution via vasodilation is likely determined by the locale of vasodilator release rather than differences in receptor distribution.
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Hughes, William Edward. "Dynamics of skeletal muscle blood flow and vasodilation with age." Diss., University of Iowa, 2018. https://ir.uiowa.edu/etd/6142.

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Aging is associated with attenuated blood flow and vasodilator responses during rhythmic exercise. Older adults also demonstrate attenuated blood flow and vasodilator responses following single skeletal muscle contractions (contraction-induced rapid onset vasodilation, ROV) within the forearm. These age-associated attenuations within the forearm have been demonstrated to be a result of endothelial and neural mechanisms. The objective of this research was to examine: 1) whether age-associated attenuations within the forearm are from mechanical factors; 2) whether age-associated attentions in ROV are present within the leg, as well as explore potential mechanisms for these age-associated attenuations in ROV; 3) examine whether aging is associated with a slower rate of adjustment in vasodilation (vasodilator kinetics) during rhythmic exercise preceding steady-state exercise; and 4) examine approaches to ameliorate age-related attenuations in blood flow and vasodilation within the leg across the entire exercise transient (onset to steady-state). The novel findings of this research are that 1) age-associated attenuations in ROV within the forearm are independent of mechanical factors; 2) older adults demonstrate attenuated ROV responses within the leg; 3) age-related attenuations in ROV within the leg are not explained by enhanced sympathetic adrenergic vasoconstriction; 4) older adults exhibit prolonged vasodilator kinetics preceding steady-state exercise; and 5) when examined in a cross-sectional design chronic exercise training improves ROV, vasodilator kinetics, as well as steady-state blood flow and vasodilator responses in older adults; 6) acute supplementation with dietary nitrate fails to exert any effect on blood flow and vasodilator responses during any domain of exercise. Collectively, this work establishes that aging is associated with reductions in blood flow and vasodilation across the entire exercise transient (onset to steady-state) within the leg, which is offset by chronic exercise training. Mechanistically, the current data suggests that mechanical and sympathetic factors do not explain age-related reductions in ROV in the arm and leg, respectively. Furthermore, acute supplementation of dietary nitrate does not impact leg blood flow and vasodilator responses in older adults during any domain of the exercise transient.
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Ferreira, Leonardo Franklin. "Dynamics of muscle blood flow, O[subscript2] uptake and muscle microvascular oxygenation during exercise." Diss., Manhattan, Kan. : Kansas State University, 2006. http://hdl.handle.net/2097/201.

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Bushell, Alison Jayne. "Protection of skeletal muscle against ischaemia and reperfusion induced damage." Thesis, University of Liverpool, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.365914.

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Stacy, Mitchel R. "The Effect of Eccentric Exercise-Induced Muscle Injury on Vascular Function and Muscle Blood Flow." University of Toledo / OhioLINK, 2011. http://rave.ohiolink.edu/etdc/view?acc_num=toledo1302229144.

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Sandberg, Margareta. "Acupuncture : the effects on muscle blood flow and aspects of treatment in the clinical context /." Linköping : Univ, 2004. http://www.bibl.liu.se/liupubl/disp/disp2004/med867s.pdf.

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Books on the topic "Muscle blood flow"

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Monteiro, Andre Antonio. Blood flow change in human masseter muscle elicited by voluntary isometric contraction. Stockholm: Karolinska Institutet, 1990.

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Margaliot, Zvi. Measurement of microvascular blood flow in skin and skeletal muscle using ultrasound contrast agents and a negative-bolus indicator-dilution technique. Ottawa: National Library of Canada, 1999.

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F, Nielsen Poul M., Miller Karol, and SpringerLink (Online service), eds. Computational Biomechanics for Medicine: Soft Tissues and the Musculoskeletal System. New York, NY: Springer Science+Business Media, LLC, 2011.

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Dawson, Judith Mary. Responses of the microcirculation in metabolically different skeletal muscles to increased or reduced blood flow. Birmingham: University of Birmingham, 1987.

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Cole, Mark Aaron. The effects of acute and prolonged low frequency electrical stimulation on blood flow and fatigue in the human triceps surae and tibialis anterior muscles. Birmingham: University of Birmingham, 1997.

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The relationship between skeletal muscle blood flow and blood lactate concentrations during exercise in rats. 1990.

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Carey, Michael Francis. Observations on the autoregulation of blood flow and capillary filtration in human skeletal muscle. 1995.

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The relationships among exercise blood lactate response, muscle blood flow, and oxidative adaptation to endurance training in the rat. 1992.

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The relationships among exercise blood lactate response, muscle blood flow, and oxidative adaptation to endurance training in the rat. 1992.

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Differential control of blood flow to muscles composed predominantly of different fiber types. 1991.

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Book chapters on the topic "Muscle blood flow"

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Lejemtel, Thierry H., and Stuart D. Katz. "Skeletal muscle blood flow." In Developments in Cardiovascular Medicine, 469–78. Dordrecht: Springer Netherlands, 1993. http://dx.doi.org/10.1007/978-94-011-1848-4_32.

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Tyml, Karel, Richard J. Roman, and Julian H. Lombard. "Blood Flow in Skeletal Muscle." In Laser-Doppler Blood Flowmetry, 215–26. Boston, MA: Springer US, 1990. http://dx.doi.org/10.1007/978-1-4757-2083-9_12.

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Fung, Y. C. "Blood Flow in Skeletal Muscle." In Biomechanics, 514–46. New York, NY: Springer New York, 1997. http://dx.doi.org/10.1007/978-1-4757-2696-1_8.

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Gao, Yuansheng. "Shear Stress, Myogenic Response, and Blood Flow Autoregulation." In Biology of Vascular Smooth Muscle, 173–87. Singapore: Springer Nature Singapore, 2022. http://dx.doi.org/10.1007/978-981-19-7122-8_10.

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Kooyman, Gerald L. "Blood and Muscle Metabolites: Clues to Flow." In Zoophysiology, 89–108. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-83602-2_8.

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Magder, Sheldon. "Respiratory Muscle Blood Flow and Heart–Lung Interactions." In Cardiopulmonary Monitoring, 219–33. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-73387-2_16.

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Gao, Yuansheng. "Shear Stress, Myogenic Response, and Blood Flow Autoregulation." In Biology of Vascular Smooth Muscle: Vasoconstriction and Dilatation, 127–36. Singapore: Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-10-4810-4_10.

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Schmid-Schönbein, Geert W., Thomas C. Skalak, and Donald W. Sutton. "Bioengineering Analysis of Blood Flow in Resting Skeletal Muscle." In Microvascular Mechanics, 65–99. New York, NY: Springer New York, 1989. http://dx.doi.org/10.1007/978-1-4612-3674-0_6.

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Laughlin, M. Harold. "Distribution of Skeletal Muscle Blood Flow During Locomotory Exercise." In Oxygen Transfer from Atmosphere to Tissues, 87–101. Boston, MA: Springer US, 1988. http://dx.doi.org/10.1007/978-1-4684-5481-9_8.

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Hamada, M., I. Nishio, Y. Kusuyama, M. Ura, H. Yoshikawa, and Y. Masuyama. "The Effect of Centrifugal Force on Glycosaminoglycans Synthesis and Cell Proliferation of Vascular Smooth Muscle Cells in Culture." In Role of Blood Flow in Atherogenesis, 217–22. Tokyo: Springer Japan, 1988. http://dx.doi.org/10.1007/978-4-431-68399-5_34.

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Conference papers on the topic "Muscle blood flow"

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Guo, Hao, Li Ke, Qiang Du, and Song Guo. "Muscle fatigue state classification based on blood flow bioimpedance." In 2022 15th International Congress on Image and Signal Processing, BioMedical Engineering and Informatics (CISP-BMEI). IEEE, 2022. http://dx.doi.org/10.1109/cisp-bmei56279.2022.9980152.

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Puttaswamy, Srinivasu Valagerahally, Gourav Bhattacharya, Shasidran Raj, Nikhil Bhalla, Chengkuo Lee, and James McLaughlin. "Effect of Functional Electrical Stimulation on Capillary Blood Flow to Muscle." In 2022 44th Annual International Conference of the IEEE Engineering in Medicine & Biology Society (EMBC). IEEE, 2022. http://dx.doi.org/10.1109/embc48229.2022.9871395.

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Chen, Cuiye, and Robert B. Roemer. "Simulation of Empirical Correlations Between Temperatures and Blood Perfusion During Heating Using a Temperature-Dependent Blood Perfusion Model." In ASME 2004 International Mechanical Engineering Congress and Exposition. ASMEDC, 2004. http://dx.doi.org/10.1115/imece2004-62061.

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This study applies a recently developed temperature-dependent blood perfusion model (TDBPM) coupled with a modified, one-dimensional Pennes bioheat transfer equation to predict the blood perfusion and temperature responses to step function microwave heating applied in the in vivo experiments performed by Sekins’ et al. [1] on human thigh muscle. The TDBPM model links the perfusion increase to the tissue temperature elevation based on physiological mechanisms underlying this temperature-blood-perfusion change phenomenon, i.e., a pharmacokinetic compartmental model. This physiology-based model avoids using ad hoc time delays between blood perfusion increases and tissue temperature elevations as done in previous efforts. It also includes a mechanism that produces the threshold temperature for blood flow increases that has been observed in vivo. In our recent study [2], the TDBPM model was used to simulate both the constant temperature water bath heating used in the in vivo experiments on rat leg muscle performed by Song et al. [3], and the step function microwave heating applied in the in vivo experiments on canine thigh muscle performed by Roemer et al. [4]. The blood perfusion rates predicted by the model are compared with those in vivo experimental data obtained in rat muscle and human muscle and good agreement was obtained. The TDBPM provides a possible explanation to the biochemical and biophysical origins of the relationships between temperature and blood flow that observed in rat muscle and human muscle. The physiology-based TDBPM is a simple, generic model of muscle blood flow responses of different animals to different heating conditions, which provides the type of fundamental information needed for the design of methods to thermally control blood flow in medical applications.
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Yu, Guoqiang, Katelyn Gurley, and Yu Shang. "DCS Measurement Can Be Gated Via Monitoring Muscle Movement to Derive Accurate Blood Flow in Exercising Muscle." In Biomedical Optics. Washington, D.C.: OSA, 2012. http://dx.doi.org/10.1364/biomed.2012.btu2a.5.

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Li, Zhe, Jinchao Feng, Zhonghua Sun, Pengyu Liu, Kebin Jia, Wesley Baker, and Arjun G. Yodh. "Optical Monitoring of Oxygen Saturation and Tissue Blood Flow in Skeletal Muscle." In Clinical and Translational Biophotonics. Washington, D.C.: OSA, 2018. http://dx.doi.org/10.1364/translational.2018.jth3a.61.

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Boushel, Robert, Kojiro Ide, Hasse Moller-Sorensen, Alvito Fernandes, Frank Pott, and Niels H. Secher. "NIRS and indocyanine-green-determined muscle blood flow during exercise in humans." In BiOS Europe '97, edited by David A. Benaron, Britton Chance, and Marco Ferrari. SPIE, 1998. http://dx.doi.org/10.1117/12.301045.

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Wang, Detian, Wesley B. Baker, Ashwin B. Parthasarathy, Liguo Zhu, Zeren Li, and Arjun Yodh. "Optical measurement of blood flow in exercising skeletal muscle: a pilot study." In Clinical and Preclinical Optical Diagnostics, edited by J. Quincy Brown and Ton G. van Leeuwen. SPIE, 2017. http://dx.doi.org/10.1117/12.2285447.

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Zhang, Jiabin, Nan Li, Feihong Dong, Jian An, Jue Zhang, and Yukun Luo. "Ultrasound diffraction attenuation microscopy in human quadriceps femoris muscle blood flow imaging." In 2019 IEEE International Ultrasonics Symposium (IUS). IEEE, 2019. http://dx.doi.org/10.1109/ultsym.2019.8925784.

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Queiroz de Oliveira, Sabrina, Barbarah Hemilly de Souza Valente, Dayane Azevedo Pereira, Letícia Pavoni dos Passos, Paloma Morgade Zaccaro, and Luciano Matos Chicayban. "Blood flow restriction in the postoperative of anterior cruciate ligament reconstruction." In 7th International Congress on Scientific Knowledge. Biológicas & Saúde, 2021. http://dx.doi.org/10.25242/8868113820212394.

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Blood flow restriction is a physical therapy technique that consists of promoting increased strength and muscle hypertrophy, similar to protocols with high loads. It can be used in the prevention and rehabilitation of injuries, health promotion and improvement in sports performance, as in the postoperative period of anterior cruciate ligament reconstruction, accelerating functional recovery. To identify the effects of blood flow restriction in patients undergoing anterior cruciate ligament reconstruction. Through a systematic review of the literature, randomized clinical trials were selected according to the highest PEDro score. The search involved the PEDro database, PubMed, using the following terms: bloodflow restriction, anterior cruciate ligament, anteriorcruciate ligament injury and BFR exercise, published between 2000 and 2021. Six RCTs were selected: three did not demonstrate benefits in relation to atrophy, strength, volume and activation of the quadriceps muscle during blood flow restriction, when used through isometric, concentric and eccentric exercises, with progressive loads or without loads. In two other RCTs, there were positive results in relation to the aforementioned parameters, where freeexercises were performed, with body resistance and sessions with occlusive stimuli and occlusion release. Finally, one of the RCTs showed equal improvement in both groups in quadriceps hypertrophy and strength; and regarding the use of flow restriction inone of the groups, there was a reduction in joint pain and effusion. Blood flow restriction has shown contradictory results in relation to atrophy, decreased pain, effusion and asymmetry of the quadriceps muscle in patients undergoing anterior cruciate ligament reconstruction.
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Patricia Traina Chacon Mikahil, Mara, and Gabriel Incerpi Agentilho. "EFFECTS OF AEROBIC TRAINING WITH BLOOD FLOW RESTRICTION IN CARDIORESPIRATORY AND MUSCLE FUNCTION." In XXIII Congresso de Iniciação Científica da Unicamp. Campinas - SP, Brazil: Galoá, 2015. http://dx.doi.org/10.19146/pibic-2015-37270.

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Reports on the topic "Muscle blood flow"

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Holdsworth, Clark, Steven Copp, Daniel Hirai, Scott Ferguson, Timothy Musch, and David Poole. Effects of dietary fish oil on exercising muscle blood flow in chronic heart failure rats. Peeref, June 2022. http://dx.doi.org/10.54985/peeref.2206p4902539.

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Li, Shuoqi, and Shazlin Shaharudin. Effects of blood flow restriction training on muscle strength and pain in patients with knee injuries: a meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, June 2020. http://dx.doi.org/10.37766/inplasy2020.6.0021.

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He, Xiaoyu, Guangzeng Fu, and Biyu Zhang. Effects Between High-Load Resistance Training Versus Low-Load Resistance Training Associated with Blood-Flow Restriction on Muscle Function in Healthy Adults: A Systematic Review and Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, October 2022. http://dx.doi.org/10.37766/inplasy2022.10.0009.

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Moridi, Mina, Parinaz Onikzeh, Aida Kazemi, and Hadi Zamanian. CABG versus myotomy in symptomatic myocardial bridge patients : A systematic Review and Meta-analysis protocol. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2021. http://dx.doi.org/10.37766/inplasy2021.11.0088.

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Review question / Objective: The aim of this study is to find which surgical intervention in myocardial bridge ( myotomy or CABG) is more effective in reducing adverse outcomes in symptomatic patients resistant to optimal medical therapy ? Condition being studied: Myocardial bridge : A myocardial bridge (MB) is a congenital heart defect in which a bridge of muscle fibers (myocardium) overlying a section of a coronary artery and the artery is squeezed and normal blood flow is disrupted. Most bridges don't seem to cause symptoms. However, some people can experience angina, or chest pain. In patients with symptoms, first line treatment is medication and if they have symptoms despite optimal medical treatment , invasive measures like CABG or myotomy should be taken.
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