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Journal articles on the topic 'Muschio'

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Author: Grafiati

Published: 11 March 2023

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1

Duente, Tim, Justin Schulte, Max Pfeiffer, and Michael Rohs. "MuscleIO." Proceedings of the ACM on Interactive, Mobile, Wearable and Ubiquitous Technologies 2, no. 2 (July 5, 2018): 1–21. http://dx.doi.org/10.1145/3214267.

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2

Kaiser, Herbert. ",,Satanische Blendung“ Ernst Barlachs apokalyptischer Humor in seinem nachgelassenen Roman Der gestohlene Mond." Literatur für Leser 37, no. 3 (January 1, 2014): 141–60. http://dx.doi.org/10.3726/90068_141.

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Nachdem Adolf Muschg Barlachs Roman zu seinem ,,Jahrhundertbuch“ erklärt und damit die Familientradition seiner Würdigung erneuert hatte1, die Walter Muschg2 und sein Schüler Heinz Schweizer schon um 1950 begründeten3, blieb die Rezeptionsgeschichte dieser letzten literarischen Arbeit Barlachs insgesamt doch mehr als verhalten. Das hat Gründe: Es gibt eine fast überbordende Literatur zu ihm als darstellendem Künstler; immerhin noch eine – mit der Zeit arg schrumpfende – Auseinandersetzung mit seinen Dramen. Seine Prosaschriften bleiben aber im Schatten dieser Wahrnehmung, nicht nur weil das Bild von Barlach, dem Plastiker und Zeichner, so dominiert, sondern auch, weil er als Erzähler den Leser mit Gedanken, Vorstellungen und Bildern, mit sprachlichen Eigenwilligkeiten konfrontiert, für die es in der zeitverwandten Literatur der 30er und 50er Jahre kaum Anknüpfungsmöglichkeiten gibt. Speziell für diesen Roman kommt dessen katastrophisches Überlebens- und Editionsschicksal hinzu. Er konnte, nach kriegsbedingter Verschüttung und Beschädigung, erst 10 Jahre nach Barlachs Tod veröffentlicht werden4. Aber bald nach dem Krieg bestimmte die Gruppe 47 das öffentliche literarische Bewusstsein. Die verdienstvollen Publikationen seiner Dramen, Briefe und Prosaschriften5, auch des Gestohlenen Monds, konnten nur den Dramen eine begrenzte Aufmerksamkeit verschaffen.

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3

Kita, Shunbun, Hitoshi Nishizawa, Yosuke Okuno, Masaki Tanaka, Atsutaka Yasui, Morihiro Matsuda, Yukio Yamada, and Iichiro Shimomura. "Competitive binding of musclin to natriuretic peptide receptor 3 with atrial natriuretic peptide." Journal of Endocrinology 201, no. 2 (February 20, 2009): 287–95. http://dx.doi.org/10.1677/joe-08-0551.

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Musclin is a novel skeletal muscle-derived secretory factor that was isolated by our group. Musclin contains a region homologous to natriuretic peptides (NPs). This study investigated the interaction between musclin and NP receptors (NPRs). Musclin specifically bound to NPR3, but not to NPR1 or NPR2. Musclin and atrial natriuretic peptide (ANP) competed for binding to NPR3. We conducted binding assays using various synthetic musclin peptides and mutant musclin proteins. The first NP-homologous region in musclin (88LDRL91) and the second homologous region (117MDRI120) were responsible cooperatively for high-affinity binding to NPR3. The first NP-homologous region was more importantly associated with binding to NPR3, than the second homologous region. The competitive nature of musclin with ANP for the natriuretic clearance receptor NPR3 was also confirmed in vivo. We conclude that musclin binds to NPR3 competitively with ANP and may affect ANP concentrations in a local or systemic manner.

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4

Lin, Jia-Wei, Cheng-Chia Tsai, Li-Jen Chen, Ho-Shan Niu, Chen Kuei Chang, and Chiang-Shan Niu. "Characterization of Musclin as a New Target for Treatment of Hypertension." BioMed Research International 2014 (2014): 1–7. http://dx.doi.org/10.1155/2014/354348.

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Musclin is a novel skeletal muscle-derived factor found in the signal sequence trap of mouse skeletal muscle cDNAs. Recently, it has been demonstrated that musclin is involved in the pathogenesis of spontaneously hypertensive rats (SHRs). However, it is known as a genetic hypertension model. In the present study, we aim to investigate the role of musclin in another animal model of hypertension and characterize the direct effect of musclin on vascular contraction. The results show that expression of musclin was increased in arterial tissues isolated from DOCA-salt induced hypertensive rats or the normal rats received repeated vasoconstriction with phenylephrine. Additionally, direct incubation with phenylephrine did not modify the expression of musclin in thein vitrostudies. Also, the direct effect of musclin on the increase of intracellular calcium was observed in a concentration-dependent manner. These results provide the evidence to support that musclin is involved in hypertension. Thus, musclin is suitable to be considered as a novel target for treatment of hypertension.

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5

Nishizawa, Hitoshi, Morihiro Matsuda, Yukio Yamada, Kenichiro Kawai, Emi Suzuki, Makoto Makishima, Toshio Kitamura, and Iichiro Shimomura. "Musclin, a Novel Skeletal Muscle-derived Secretory Factor." Journal of Biological Chemistry 279, no. 19 (March 24, 2004): 19391–95. http://dx.doi.org/10.1074/jbc.c400066200.

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Skeletal muscle is involved in the homeostasis of glucose and lipid metabolism. We hypothesized that the skeletal muscle produces and secretes bioactive factor(s), similar to adipocytokines secreted by fat tissue. Here, we report the identification of a novel secretory factor, musclin, by signal sequence trap of mouse skeletal muscle cDNAs. Musclin cDNA encoded 130 amino acids, including NH2-terminal 30-amino acid signal sequence. Musclin protein contained a region homologous to natriuretic peptide family, and KKKR, a putative serine protease cleavage site, similar to the natriuretic peptide family. Full-length musclin protein and KKKR-dependent cleaved form were secreted in media of musclin cDNA-transfected mammalian cell cultures. Musclin mRNA was expressed almost exclusively in the skeletal muscle of mice. Musclin mRNA levels in skeletal muscle were markedly low in fasted, increased upon re-feeding, and were low in streptozotocin-treated insulin-deficient mice. Musclin mRNA expression was induced at late stage in the differentiation of C2C12 myocytes. In myocytes, insulin increased, while epinephrine, isoproterenol, and forskolin reduced musclin mRNA, all of which are known to increase the cellular content of cyclic AMP, a counter-regulator to insulin. Pathologically, overexpression of musclin mRNA was noted in the muscles of obese insulin-resistant KKAy mice. Functionally, recombinant musclin significantly attenuated insulin-stimulated glucose uptake and glycogen synthesis in myocytes. In conclusion, we identified musclin, a novel skeletal muscle-derived secretory factor. Musclin expression level is tightly regulated by nutritional changes and its physiological role could be linked to glucose metabolism.

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6

Clark, Audra, Ryan M. Huebinger, Deborah L. Carlson, Steven E. Wolf, and Juquan Song. "Serum Level of Musclin Is Elevated Following Severe Burn." Journal of Burn Care & Research 40, no. 5 (June 11, 2019): 535–40. http://dx.doi.org/10.1093/jbcr/irz101.

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Abstract Muscle wasting induced by severe burn worsens clinical outcomes is associated with hyperglycemia. A novel muscle-specific secretory factor, musclin, was reported to regulate glucose metabolism with a homologous sequence of natriuretic peptides. The purpose of the study was to investigate musclin expression in response to burn injury in both human and animal models. Serum was collected from 13 adult burn patients and circulating levels of musclin protein were measured via elisa. The cytokine profile was measured by Bio-Plex multiple immunoassay. Following the clinical study, we used a burn rat model with 40% TBSA to study the time course of musclin expression till day 14. Rat serum and muscle tissue sample were harvested. Finally, an in vitro study was applied to investigate whether the muscle cell C2C12 myoblast expressed musclin under 10% burn serum stimulation. Pearson analysis showed that there was a significant positive correlation of musclin expression to total body surface area of burn in patients (P &= .038). Musclin expression was significantly positively correlated with IL-4, IL-7, IL-12, and IL-13 in burn patients’ serum (P < .05). In the animal study, we found that the musclin level evaluated at 6 hours and 1 day in burn rat serum (P < .05). In vitro, musclin mRNA expression significantly increased with burn serum stimulation at 24 hours (P < .05). In conclusion, serum level of musclin elevated both in human patients and burn animals; musclin was correlated with the severity of burn injury as well as with an elevated cytokine profile in patients; burn serum-stimulated musclin expression in vitro further identified the resource of musclin expression after burn.

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7

Chen, Wen-Jia, Yue Liu, Yu-Bin Sui, Bo Zhang, Xiao-Hui Zhang, and Xin-Hua Yin. "Increased circulating levels of musclin in newly diagnosed type 2 diabetic patients." Diabetes and Vascular Disease Research 14, no. 2 (February 10, 2017): 116–21. http://dx.doi.org/10.1177/1479164116675493.

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Background: Musclin is a newly identified skeletal muscle–derived secretory factor, which has been recently characterized as a stimulator that induces insulin resistance in mice. However, the pathophysiological role of musclin in humans remains poorly understood. The aim of this study was to explore the potential correlations between musclin plasma levels and various metabolic parameters in patients with type 2 diabetes mellitus. Materials and methods: In this hospital-based study, plasma samples were collected from the enrolled individuals, including 38 newly diagnosed, treatment-naive type 2 diabetes mellitus patients and 41 age- and gender-matched control subjects. Plasma musclin levels were examined by radioimmunoassay. Results: Compared with the control group, musclin plasma levels were significantly higher in untreated type 2 diabetes mellitus patients. Musclin levels in the plasma of newly diagnosed type 2 diabetes mellitus patients were positively correlated with fasting plasma glucose, haemoglobin A1c, serum insulin, triglycerides and homeostasis model assessment of insulin resistance. Furthermore, multivariate logistic regression analysis showed that the level of musclin was associated with the presence of type 2 diabetes mellitus. Receiver operating characteristic curve analysis yielded an area under the curve for musclin of 0.718 in type 2 diabetes mellitus. Conclusion: The circulating concentration of musclin was significantly increased in type 2 diabetes mellitus patients. Our results suggest that musclin has a strong relationship with insulin resistance in type 2 diabetes mellitus.

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8

Guo, Qian, Hailong Hu, Xiaohuan Liu, DaQian Yang, Yao Yin, Boya Zhang, Hongjuan He, et al. "C/EBPβ mediates palmitate-induced musclin expression via the regulation of PERK/ATF4 pathways in myotubes." American Journal of Physiology-Endocrinology and Metabolism 316, no. 6 (June 1, 2019): E1081—E1092. http://dx.doi.org/10.1152/ajpendo.00478.2018.

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Musclin is a muscle-secreted cytokine that disrupts glucose uptake and glycogen synthesis in type 2 diabetes. The purpose of this study was to investigate the mechanisms responsible for the regulation of musclin gene expression in response to treatment with palmitate. RNA sequencing results showed that biological processes activated by palmitate are mainly enriched in endoplasmic reticulum (ER) stress. The protein kinase RNA-like ER kinase (PERK) signaling pathway is involved in the regulation of musclin expression induced by palmitate. Chromatin immunoprecipitation data showed that activating transcription factor 4 (ATF4)—downstream of PERK—bound to the promoter of the C/EBPβ gene. Notably, C/EBPβ also contains a binding site in the region −94~−52 of the musclin gene promoter. Knockdown or knockout of PERK and ATF4 using short hairpin RNA or CRISPR-Cas9 decreased the expression of C/EBPβ and musclin induced by palmitate. Furthermore, knockdown and knockout of C/EBPβ alleviated the high expression of musclin in response to treatment with palmitate. Moreover, CRISPR-Cas9 knockout of the region −94~−52 in which C/EBPβ binds to the promoter of musclin abrogated the induction of high musclin expression caused by palmitate. Collectively, these findings suggest that treatment with palmitate activates the PERK/ATF4 signaling pathway, which in turn increases the expression of C/EBPβ. C/EBPβ binds directly to the promoter of the musclin gene and upregulates its expression.

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9

Subbotina, Ekaterina, Ana Sierra, Zhiyong Zhu, Zhan Gao, Siva Rama Krishna Koganti, Santiago Reyes, Elizabeth Stepniak, et al. "Musclin is an activity-stimulated myokine that enhances physical endurance." Proceedings of the National Academy of Sciences 112, no. 52 (December 14, 2015): 16042–47. http://dx.doi.org/10.1073/pnas.1514250112.

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Exercise remains the most effective way to promote physical and metabolic wellbeing, but molecular mechanisms underlying exercise tolerance and its plasticity are only partially understood. In this study we identify musclin—a peptide with high homology to natriuretic peptides (NP)—as an exercise-responsive myokine that acts to enhance exercise capacity in mice. We use human primary myoblast culture and in vivo murine models to establish that the activity-related production of musclin is driven by Ca2+-dependent activation of Akt1 and the release of musclin-encoding gene (Ostn) transcription from forkhead box O1 transcription factor inhibition. Disruption of Ostn and elimination of musclin secretion in mice results in reduced exercise tolerance that can be rescued by treatment with recombinant musclin. Reduced exercise capacity in mice with disrupted musclin signaling is associated with a trend toward lower levels of plasma atrial NP (ANP) and significantly smaller levels of cyclic guanosine monophosphate (cGMP) and peroxisome proliferator-activated receptor gamma coactivator 1-α in skeletal muscles after exposure to exercise. Furthermore, in agreement with the established musclin ability to interact with NP clearance receptors, but not with NP guanyl cyclase-coupled signaling receptors, we demonstrate that musclin enhances cGMP production in cultured myoblasts only when applied together with ANP. Elimination of the activity-related musclin-dependent boost of ANP/cGMP signaling results in significantly lower maximum aerobic capacity, mitochondrial protein content, respiratory complex protein expression, and succinate dehydrogenase activity in skeletal muscles. Together, these data indicate that musclin enhances physical endurance by promoting mitochondrial biogenesis.

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10

Tsechkovski, Mark S. "Musculo-skeletal diseases." Acta Orthopaedica Scandinavica 69, sup281 (January 1998): 61–62. http://dx.doi.org/10.1080/17453674.1998.11744796.

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11

Richard, Olivier. "Katharina Simon-Muscheid." Revue d’Alsace, no. 139 (October 1, 2013): 535–36. http://dx.doi.org/10.4000/alsace.1975.

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12

Drapé, Jean-Luc. "Echographie musculo-squelettique." Journal de Radiologie 89, no. 10 (October 2008): 1358. http://dx.doi.org/10.1016/s0221-0363(08)76086-4.

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13

Finlayson, D. "Musculo-Skeletal Issue." Scottish Medical Journal 54, no. 1 (February 2009): 3. http://dx.doi.org/10.1258/rsmsmj.54.1.3.

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14

Drapé, Jean-Luc, Gérard Morvan, and Christophe Courthaliac. "Echographie musculo-squelettique." Journal de Radiologie 90, no. 10 (October 2009): 1325. http://dx.doi.org/10.1016/s0221-0363(09)75268-0.

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15

Merchant, M. "Muscoid Fly Parisitoids." Bulletin of the Entomological Society of America 34, no. 2 (June 1, 1988): 100. http://dx.doi.org/10.1093/besa/34.2.100.

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16

Re Cecconi, Andrea D., Mara Forti, Michela Chiappa, Zhiyong Zhu, Leonid V. Zingman, Luigi Cervo, Luca Beltrame, Sergio Marchini, and Rosanna Piccirillo. "Musclin, A Myokine Induced by Aerobic Exercise, Retards Muscle Atrophy During Cancer Cachexia in Mice." Cancers 11, no. 10 (October 12, 2019): 1541. http://dx.doi.org/10.3390/cancers11101541.

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Physical activity improves the prognosis of cancer patients, partly by contrasting the associated muscle wasting (cachexia), through still unknown mechanisms. We asked whether aerobic exercise causes secretion by skeletal muscles of proteins (myokines) that may contrast cachexia. Media conditioned by peroxisome proliferator-activated receptor γ coactivator 1α (PGC1α)-expressing myotubes, reproducing some metabolic adaptations of aerobic exercise, as increased mitochondrial biogenesis and oxidative phosphorylation, restrained constitutively active Forkhead box-containing subfamily O3 (caFoxO3)-induced proteolysis. Microarray analysis identified amphiregulin (AREG), natriuretic peptide precursor B (NppB), musclin and fibroblast growth factor 18 (FGF18) as myokines highly induced by PGC1α. Notably, only musclin tended to be low in muscle of mice with a rare human renal carcinoma; it was reduced in plasma and in muscles of C26-bearing mice and in atrophying myotubes, where PGC1α expression is impaired. Therefore, we electroporated the Tibialis Anterior (TA) of C26-bearing mice with musclin or (its receptor) natriuretic peptide receptor 3 (Npr3)-encoding plasmids and found a preserved fiber area, as a result of restrained proteolysis. Musclin knockout (KO) mice lose more muscle tissue during growth of two distinct cachexia-causing tumors. Running protected C26-bearing mice from cachexia, not changing tumor growth, and rescued the C26-induced downregulation of musclin in muscles and plasma. Musclin expression did not change in overloaded plantaris of mice, recapitulating partially muscle adaptations to anaerobic exercise. Musclin might, therefore, be beneficial to cancer patients who cannot exercise and are at risk of cachexia and may help to explain how aerobic exercise alleviates cancer-induced muscle wasting.

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17

Fabian, Hans J., and Manfred Dierks. "Adolf Muschg." World Literature Today 64, no. 1 (1990): 105. http://dx.doi.org/10.2307/40145884.

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18

Burkhard, Marianne, and Renate Voris. "Adolf Muschg." German Quarterly 59, no. 4 (1986): 670. http://dx.doi.org/10.2307/406551.

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19

Smoodin, Eric. "Review: Hollywood's New Deal by Giuliana Muscio." Film Quarterly 53, no. 1 (1999): 57–58. http://dx.doi.org/10.2307/3697223.

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20

Dia, M. G., G. Miceli, and F. M. Raimondo. "Check-list dei Muschi noti in Sicilia." Webbia 41, no. 1 (January 1987): 61–123. http://dx.doi.org/10.1080/00837792.1987.10670402.

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21

Sánchez, Yeliana L., Manuela Yepes-Calderón, Luis Valbuena, Andrés F. Milán, María C. Trillos-Almanza, Sergio Granados, Miguel Peña, et al. "Musclin Is Related to Insulin Resistance and Body Composition, but Not to Body Mass Index or Cardiorespiratory Capacity in Adults." Endocrinology and Metabolism 36, no. 5 (October 31, 2021): 1055–68. http://dx.doi.org/10.3803/enm.2021.1104.

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Background: We studied whether musclin function in humans is related to glycemic control, body composition, and cardiorespiratory capacity.Methods: A cross-sectional study was performed in sedentary adults with or without metabolic syndrome (MS). Serum musclin was measured by enzyme-linked immunosorbent assay. Insulin resistance (IR) was evaluated by the homeostatic model assessment (HOMA-IR). Body composition was determined by dual-energy X-ray absorptiometry and muscle composition by measuring carnosine in the thigh, a surrogate of fiber types, through proton magnetic resonance spectroscopy. Cardiorespiratory capacity was assessed through direct ergospirometry.Results: The control (n=29) and MS (n=61) groups were comparable in age (51.5±6.5 years old vs. 50.7±6.1 years old), sex (72.4% vs. 70.5% women), total lean mass (58.5%±7.4% vs. 57.3%±6.8%), and peak oxygen consumption (VOpeak) (31.0±5.8 mL O2./kg.min vs. 29.2±6.3 mL O2/kg.min). Individuals with MS had higher body mass index (BMI) (30.6±4.0 kg/m2 vs. 27.4± 3.6 kg/m2), HOMA-IR (3.5 [95% confidence interval, CI, 2.9 to 4.6] vs. 1.7 [95% CI, 1.1 to 2.0]), and musclin (206.7 pg/mL [95% CI, 122.7 to 387.8] vs. 111.1 pg/mL [95% CI, 63.2 to 218.5]) values than controls (P˂0.05). Musclin showed a significant relationship with HOMA-IR (β=0.23; 95% CI, 0.12 to 0.33; P˂0.01), but not with VOpeak, in multiple linear regression models adjusted for age, sex, fat mass, lean mass, and physical activity. Musclin was significantly associated with insulin, glycemia, visceral fat, and regional muscle mass, but not with BMI, VCO2peak, maximum heart rate, maximum time of work, or carnosine.Conclusion: In humans, musclin positively correlates with insulinemia, IR, and a body composition profile with high visceral adiposity and lean mass, but low body fat percentage. Musclin is not related to BMI or cardiorespiratory capacity.

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Freitas, Betânia de Jesus e. Silva de Almendra, Larissa Coimbra Mesquita, Nayanna de Jesus Vilhena Teive, and Samilys Ramos de Souza. "Antropometria Clássica e Músculo Adutor do Polegar na Determinação do Prognóstico Nutricional em Pacientes Oncológicos." Revista Brasileira de Cancerologia 56, no. 4 (December 31, 2010): 415–22. http://dx.doi.org/10.32635/2176-9745.rbc.2010v56n4.1462.

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A avaliação nutricional e amplamente reconhecida por sua importância no acompanhamento hospitalar; direcionamento da conduta medica em todo processo de hospitalização; investigação para corrigir deficiências nutricionais; e ferramenta para fins prognósticos. A apatia induzida pela desnutrição provoca redução das atividades diárias resultando em diminuição progressiva da espessura do musculo adutor do polegar. O objetivo do trabalho foi estabelecer o prognostico nutricional, por meio da antropometria clássica e espessura do musculo adutor do polegar, em pacientes oncológicos. O estudo realizado foi transversal de campo do tipo quantitativo. Foram avaliados 82 pacientes oncológicos, adultos e idosos, internados no Hospital São Marcos em Teresina (PI). Os pacientes foram submetidos a avaliação nutricional pela antropometria clássica (%PP, CB, CMB, AMBc e PCT) e a medida da espessura do musculo adutor do polegar. O estado nutricional dos pacientes caracterizou-se por perda grave de peso (56,1%), preservação de massa muscular e prevalência de perda grave de massa gorda (25,93%). A media da espessura do musculo adutor do polegar para o total da amostra foi de 13 ± 3,2 mm. O musculo adutor do polegar não se associou ao tempo de internação e mortalidade. O %PP foi o parâmetro que melhor se correlacionou com o tempo de internação (p=0,007). Os demais parâmetros não se associaram a nenhum dos indicadores de morbi-mortalidade estudados. A espessura do musculo adutor do polegar não se revelou um bom indicador prognostico antropométrico, podendo essa afirmação ser atribuída a presença de um menor comprometimento do estado nutricional observada na maioria dos pacientes e por permanecerem menos tempo no ambiente hospitalar.

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23

Ahern, M. J., M. Soden, D. Schultz, and M. Clark. "The musculo-skeletal examination." Australian and New Zealand Journal of Medicine 21, no. 3 (June 1991): 303–6. http://dx.doi.org/10.1111/j.1445-5994.1991.tb04694.x.

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24

Savouroux, Nicolas. "Le domaine musculo-squelettique." Kinésithérapie, la Revue 11, no. 119 (November 2011): 18. http://dx.doi.org/10.1016/s1779-0123(11)75209-3.

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25

Colaianni, G., T. Mongelli, S. Colucci, S. Cinti, and M. Grano. "Irisina, osso e muscolo." L'Endocrinologo 17, no. 4 (August 2016): 199–202. http://dx.doi.org/10.1007/s40619-016-0222-x.

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26

Camargo, Olavo Pires de, and André Mathias Baptista. "Neoplasias musculo-esqueléticas malignas." Revista de Medicina 86, no. 2 (June 20, 2007): 71. http://dx.doi.org/10.11606/issn.1679-9836.v86i2p71-81.

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27

Uribe Alvarado, Maria Victoria, and Ennio Palomino Salazar. "Pausas activas en la educación virtual: una solución económica y de grandes resultados." Ciencia Latina Revista Científica Multidisciplinar 6, no. 6 (December 10, 2022): 5434–54. http://dx.doi.org/10.37811/cl_rcm.v6i6.3820.

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Las clases desde casa tiene aspectos positivos, sin embargo hay riesgos, como las posturas inadecuadas frente a un ordenador, los largos períodos de tiempo sentado, la utilización de mobiliario inadecuado, la falta de actividad física, entre otros, que podrían llevarnos a desarrollar trastornos musculo esqueléticos, por lo que consideramos importante dar a conocer e implementar en los estudiantes programas de “Pausas Activas” que buscan disminuir los dichas patologías musculo esqueléticas, así como, mejorar su rendimiento académico a aquellos que las incorporen a su rutina diaria. El presente estudio busca conocer la influencia de las pausas activas en la sintomatología musculo esquelética de los estudiantes universitarios durante el desarrollo de sus clases virtuales por Covid 19, realizando un programa de pausas activas donde se aplicará un pre test y pos test del cuestionario Nórdico Kourinka, para poder evidenciar los efectos en la sintomatología musculo esquelética y posteriormente cuando el retorno a clases se produzca sigan empleando las pausas activas en su diario quehacer

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Chavez-Rivaldi, Cristhian, Francisco Perrotta, Edgar García, and Carolina Saldivar. "Schwannoma of the psoas muscle." Cirugía paraguaya 45, no. 2 (August 30, 2021): 54–55. http://dx.doi.org/10.18004/sopaci.2021.agosto.54.

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Chavez-Rivaldi, Cristhian, Francisco Perrotta, Edgar García, and Carolina Saldivar. "Schwannoma of the psoas muscle." Cirugía paraguaya 45, no. 2 (August 30, 2021): 54–55. http://dx.doi.org/10.18004/sopaci.2021.agosto.54.

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30

Bourne, IHJ. "Economic Aspects of Tender Spot Injection Therapy." Acupuncture in Medicine 14, no. 2 (November 1996): 114–16. http://dx.doi.org/10.1136/aim.14.2.114.

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One partner of a four man general practice in Essex used injection therapy to tender spots for musculo-skeletal pain. In a financial audit it was found that the prescription costs for this doctor were significantly less than the average for the other partners. It is speculated that the savings were due to effective elimination of musculo-skeletal pain by tender spot injection, thus reducing the need for analgesic and non-steroidal anti-inflammatory medication in these patients. If one doctor in every group practice nationwide were to adopt a similar, effective technique for treating musculo-skeletal pain, extrapolation of the savings to this practice suggest a national saving in excess of £95 million per year.

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31

Okada, Tatsuya, Takashi Imamura, Takanori Miyoshi, Kazuhiko Terashima, Yoshifumi Yasuda, and Takuo Suzuki. "Muscle Strength Estimation Using Musculo-Skeletal Model for Upper Limb Rehabilitation." Journal of Robotics and Mechatronics 20, no. 6 (December 20, 2008): 863–71. http://dx.doi.org/10.20965/jrm.2008.p0863.

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This article proposes a new muscle strength estimation method for upper limbs. It uses an inverse model of a musculo-skeletal model based on the muscle force applied on joints as well as the arrangement of the muscles. We did basic analyses of the parameters of the shape of a living body from magnetic resonance imaging (MRI) images and electromyography (EMG) in order to express isometric motion using the musculo-skeletal model. The model shows high reproducibility of a living body's isometric motion. We propose two easy methods of estimating muscle strength without optimization calculations and using a model which is the inverse of the musculo-skeletal model: a method based on output force distribution and a method based on joint torque distribution. We have validated them via experiments.

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32

Ashton, Jonathan. "Integrated musculo-skeletal service (IMSK)?" Morecambe Bay Medical Journal 7, no. 6 (April 1, 2016): 143. http://dx.doi.org/10.48037/mbmj.v7i6.138.

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33

Rydholm, Anders. "European Musculo-skeletal Oncology Society." Acta Orthopaedica Scandinavica 65, sup256 (January 1994): 107–11. http://dx.doi.org/10.3109/17453679409155645.

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Winkelmann, W. "European Musculo-Skeletal Oncology Society." Acta Orthopaedica Scandinavica 68, sup277 (January 1997): 53–84. http://dx.doi.org/10.1080/17453674.1997.11744776.

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Vanel, Daniel, Gilles Missenard, and Bernard Tomeno. "European Musculo-Skeletal Oncology Society." Acta Orthopaedica Scandinavica 70, sup289 (January 1999): 43–68. http://dx.doi.org/10.1080/17453674.1999.11744837.

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Lidgren, Lars. "Evidence based musculo-skeletal treatment." Acta Orthopaedica Scandinavica 73, sup305 (January 2002): 3. http://dx.doi.org/10.1080/000164702760379468.

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Silman, A. J. "MUSCULO–SKELETAL DISORDERS IN CHILDHOOD." British Medical Bulletin 42, no. 2 (1986): 196–99. http://dx.doi.org/10.1093/oxfordjournals.bmb.a072122.

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Pollen, Alex A., and Arnold R. Kriegstein. "Primate Neurons Flex Their Musclin." Neuron 92, no. 4 (November 2016): 681–83. http://dx.doi.org/10.1016/j.neuron.2016.11.024.

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Bourgeois, Pierre. "Progrès dans l’imagerie musculo-squelettique." Revue du Rhumatisme 74 (November 2007): 79–83. http://dx.doi.org/10.1016/s1169-8330(07)80616-8.

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George, Anthony Joseph. "MUSCULO – SKE Wii TAL MEDICINE." Injury 43, no. 3 (March 2012): 390–91. http://dx.doi.org/10.1016/j.injury.2010.11.028.

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Rodriguez-Merchan, E. Carlos. "Musculo-skeletal manifestations of haemophilia." Blood Reviews 30, no. 5 (September 2016): 401–9. http://dx.doi.org/10.1016/j.blre.2016.04.008.

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Descatha, A. "Trouble musculo-squelettique de l’épaule." Archives des Maladies Professionnelles et de l'Environnement 74, no. 3 (June 2013): 322–24. http://dx.doi.org/10.1016/j.admp.2013.02.006.

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Perry, J. David. "Electrodiagnosis in musculo-skeletal disease." Best Practice & Research Clinical Rheumatology 19, no. 3 (June 2005): 453–66. http://dx.doi.org/10.1016/j.berh.2005.01.007.

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Döhler, Diana, and Wolfgang Binder. "Von der Muschel zum Autolack." Nachrichten aus der Chemie 64, no. 9 (August 30, 2016): 836–41. http://dx.doi.org/10.1002/nadc.20164046036.

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45

Landau, Kurt, Regina Brauchler, Marianela Diaz-Meyer, Johannes Kiesel, Andreas Lenz, Herwig Meschke, and Angelika Presl. "Occupational stress factors and musculo-skeletal disease in patients at a rehabilitation center." Occupational Ergonomics 10, no. 4 (September 26, 2012): 139–53. http://dx.doi.org/10.3233/oer-2012-0198.

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Disorders of the musculo-skeletal system are one of the main occupational diseases occurring in industrial countries and the number of reported cases is rapidly increasing, especially among the older age groups. Musculo-skeletal diseases can be influenced by a number of stress factors arising during physical work. The aim of this study is to identify the physical work stress factors, to which persons already suffering from musculo-skeletal diseases (patients at a rehabilitation center) have been exposed in their work, and, firstly, to examine whether these data can be used to predict the probability of future musculo-skeletal disorders in workers occupying this type of job, secondly, to determine how these factors interact with each other in the development of these disorders. Trained specialists in occupational medicine using a program called Medical Job-oriented Rehabilitation (MJOR), collected data on 6668 patients by means of a standardized checklist called Bavaria Rehabilitation Assessment Method (BRA). Analysis of the recursive binary partitioning trees revealed that 19 predictor variables corresponding to age and gender, plus repetitive operations, rotation in sedentary position, degree of hand force used and forced head/neck postures were good predictors for the disorders of the hand-/arm system, the cervico-brachial syndrome and the impingement syndrome of shoulder.

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46

Valle, Ramón. "Fisicoculturismo estructural funcional. Una forma diferente de concebir, practicar el culturismo y el acondicionamiento físico." Revista Arbitrada Interdisciplinaria Koinonía 4, no. 8 (July 20, 2019): 190. http://dx.doi.org/10.35381/r.k.v4i8.276.

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Recibido: 25 de enero del 2019Aprobado: 01 de marzo del 2019La investigación tuvo por objetivo diseñar una propuesta sobre fisicoculturismo estructural funcional como alternativa de concebir y practicar el culturismo y el acondicionamiento físico. El deportista asume una nueva concepción de entrenar en un enfoque que procura trabajar sobre la funcionalidad del musculo, ejercitarlo para que este cumpla la función anatómica natural correspondiente. Se trabajó metodológicamente desde la perspectiva de la investigación holística. El fisicoculturismo funcional se rige por los principios: 1. Función muscular similar. 2. Orden muscular. 3. Prioridad muscular. Entre las principales conclusiones se tiene que el principal objetivo que se persigue es trabajar el musculo desde el principio de su funcionalidad, ejercitándolo según la función correspondiente, por ejemlo tensión o extensión, así se podrá realizar una biomecánica armónica, permitiendo proveer al musculo de un movimiento natural acompañado de la ejercitación estimulante de su atrofia estética.

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Kaissi, Ali Al, Elisabeth Zwettler, Rudolf Ganger, Simone Schreiner, Klaus Klaushofer, and Franz Grill. "Musculo-Skeletal Abnormalities in Patients with Marfan Syndrome." Clinical Medicine Insights: Arthritis and Musculoskeletal Disorders 6 (January 2013): CMAMD.S10279. http://dx.doi.org/10.4137/cmamd.s10279.

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Background A leptosomic body type is tall and thin with long hands. Marfanoid features may be familial in nature or pathological, as occurs in congenital contractual arachnodactyly (Beal's syndrome) and Shprintzen-Goldberg syndrome mimicking some of the changes of Marfan syndrome, although not accompanied by luxation of lens and dissecting aneurysm of aorta. Methods In this article we collected eight patients who were consistent with the diagnosis of Marfan syndrome via phenotypic and genotypic characterization. Results Our patients manifested a constellation of variable presentations of musculo-skeletal abnormalities ranging from developmental dysplasia of the hip, protrusio acetabuli, leg length inequality, patellar instability, scoliosis, to early onset osteoarthritis. Each abnormality has been treated accordingly. Conclusion This is the first paper which includes the diagnosis and the management of the associated musculo-skeletal abnormalities in patients with Marfan syndrome, stressing that patients with Marfan syndrome are exhibiting great variability in the natural history and the severity of musculo-skeletal abnormalities.

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Van Rooy, J. "BRYACEAE (MUSCI)." Bothalia 16, no. 1 (July 22, 1986): 42–44. http://dx.doi.org/10.4102/abc.v16i1.1061.

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Winderlich, M. E. "INFLUENCE OF HORMONES (HORMONAL STATUS) ON THE MUSCLE-LIGGING APPARATUS INPREGNANT WOMEN." Journal of Volgograd State Medical University 80, no. 4 (December 30, 2021): 8–11. http://dx.doi.org/10.19163/1994-9480-2021-4(80)-8-11.

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e. Imbalance of hormones in a woman's body provokes a number of disorders of the body's functions, which affects her emotional and physical well-being. Purpose – to study the current state of the problem of changes in the musculo-ligamentous apparatus in pregnant women against the background of changes in their hormonal status. Material and methods. A systematic analysis of the main hormonal changes occurring in a woman's body during childbearing, and the results of research by Russian and foreign experts in this area. Results and discussion. It has been established that to date, in Russian and foreign practice, the number of studies that show the correlation between changes in the hormonal background of a pregnant woman and her musculo-ligamentous apparatus is still insignificant and is extremely fragmented. Conclusion. It is concluded that it is necessary to expand the boundaries of experimental studies and conduct a detailed analysis of clinical cases of the onset and exacerbation of musculo-ligamentous pathologies in a situation of a change in the hormonal status of a woman against the background of her pregnancy.

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Molinari-Novoa, Eduardo Antonio. "Further Nomenclatural Changes Regarding Peruvian Endemics." Polish Botanical Journal 61, no. 2 (December 1, 2016): 275–77. http://dx.doi.org/10.1515/pbj-2016-0021.

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Abstract The nomenclature of eight species of Peruvian endemic plants from six genera (Arenaria L., Calceolaria L., Erigeron L., Pycnophyllum J. Rémy, Pycnophyllopsis Skottsb., Schizotrichia Benth.) is reviewed, and the corresponding changes are proposed. Additionally, lectotypes for Pycnophyllum peruvianum Muschl. and P. macrophyllum Muschl. are selected, and conservation assessments are provided for all species.

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