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Dissertations / Theses on the topic 'Murine SIM'

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Author: Grafiati

Published: 10 December 2022

Last updated: 28 January 2023

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1

Kukuk, Laura [Verfasser], and Bernd [Gutachter] König. "High-resolution structure of the SAM domain homodimer of the murine adapter protein SLY1 / Laura Kukuk ; Gutachter: Bernd König." Düsseldorf : Universitäts- und Landesbibliothek der Heinrich-Heine-Universität Düsseldorf, 2018. http://d-nb.info/1164763040/34.

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2

Langton, Simne. "The generation and characterization of CYP26A1(-/-) murine embryonic stem cells /." Access full-text from WCMC, 2007. http://proquest.umi.com/pqdweb?did=1436351451&sid=25&Fmt=2&clientId=8424&RQT=309&VName=PQD.

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3

Lawson, Devon Ann. "Identification and functional characterization of murine prostate epithelial stem cells." Diss., Restricted to subscribing institutions, 2008. http://proquest.umi.com/pqdweb?did=1666132121&sid=8&Fmt=2&clientId=1564&RQT=309&VName=PQD.

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4

Rodrigues, Andréa Mendonça. "Nova proposta de modelo murino de asma aguda: utilização de protocolo curto sem adjuvante com sensibilização a ovalbumina." Pontifícia Universidade Católica do Rio Grande do Sul, 2011. http://hdl.handle.net/10923/4722.

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Introduction: Some limitations have been raised over the murine models with ovalbumin (OVA) sensitization in asthma research. However, this model is still widely used and the acute OVA protocol in mice still plays a role in pre-clinical investigation. The use of adjuvant and long sensitization periods are some of the limitations raised. Aims: We have tested whether a shorter period of subcutaneous sensitization with OVA, with no adjuvant, induces a similar eosinophilic pulmonary response in mice, when compared with previous well-established control protocols. Methods: Adult female BALB/c mice were used and divided into groups, according to the number of OVA sensitizations (once or twice, OVA: 20 μg) and the number (two or three times) /dosage(40 μg and 100 μg) of intranasal OVA challenge. The shorter protocol (10 days-length) consisted of one subcutaneous OVA sensitization and three OVA challenges (100 μg). Total (TCC) and differential cell counts from bronchoalveolar lavage (BAL), eosinophil peroxidase (EPO) from lung tissue and histopathology (HE) of the lungs were performed 24 hours after the last OVA challenge. Results: Cell counts from BAL, EPO from lung tissue and histological lung abnormalities were not different between the groups studied. The shorter protocol induced a similar allergic lung response to OVA, when compared with the positive control, the same occurring with the other groups. Conclusion: We concluded that the use of one subcutaneous OVA sensitization elicit a strong allergic pulmonary response, free of adjuvant, in a 10-day-length protocol. Our findings suggest that this protocol may be used a first-line pre-clinical test, reducing cost and time of experiments, and avoiding the use of artificial adjuvants.
Introdução: Várias limitações têm sido levantadas em estudos de asma utilizando modelos murinos agudos sensibilizados com ovalbumina (OVA), mas este modelo é ainda amplamente usado, ainda mantendo sua importância em estudos pré-clínicos. Algumas limitações encontradas são o uso de adjuvante e os longos períodos de sensibilização. Objetivos: Testar se a sensibilização com OVA em um período curto, sem adjuvante, induziria uma resposta pulmonar eosinofílica em camundongos similar aos protocolos já previamente estabelecidos.Métodos: Fêmeas adultas de camundongos BALB/c foram utilizadas e divididas em grupos de acordo com o número de sensibilizações com OVA (uma ou duas vezes, OVA: 20 μg) e o número(duas ou três vezes)/dosagem(40 μg e 100 μg) de desafios intranasais. O protocolo mais curto (10 dias) consistiu de uma sensibilização subcutânea e três desafios com OVA (100 μg). Contagem total (CTC) e diferencial de células no lavado broncoalveolar (LBA), ensaio da peroxidase eosinofílica (EPO) do tecido pulmonar e histopatologia (HE) dos pulmões foram realizados 24 horas após o último desafio com OVA. Resultados: Contagem celular do LBA, EPO do tecido pulmonar e alterações inflamatórias da histologia pulmonar não foram diferentes entre os grupos estudados. O protocolo mais curto induziu uma resposta eosinofílica pulmonar à OVA semelhante ao grupo controle, ocorrendo o mesmo com os outros grupos. Conclusão: O uso de sensibilização subcutânea com OVA, sem adjuvante, resulta em uma significativa resposta pulmonar alérgica, permitindo sua utilização em protocolos de duração mais curta. Nossos achados sugerem que este protocolo pode ser utilizado como teste pré-clínico de primeira linha para pesquisa de novos fármacos, reduzindo custo, tempo e uso de adjuvante.

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5

Aguettaz, Elizabeth. "Effets de l'étirement axial sur des cardiomyocytes murins déficients en dystrophine : dérégulation calcique et canaux TRPs." Thesis, Poitiers, 2015. http://www.theses.fr/2015POIT2263/document.

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La dystrophie musculaire de Duchenne (DMD) est la conséquence de la perte de la dystrophine, protéine sous membranaire indispensable au maintien mécanique et fonctionnel du sarcolemme. Cette déficience augmenterait les influx cationiques par des microruptures de la membrane ou par la dérégulation de canaux tels que les canaux activés par l'étirement (SACs: Stretch-activated channel). Dans ce travail, les effets d'une stimulation mécanique ont été explorés sur des cardiomyocytes dans le contexte pathologique de la cardiomyopathie dilatée associée à la DMD. L'utilisation de fibres de carbone a permis de réaliser un étirement axial similaire aux conditions physiologiques de remplissage ventriculaire. Dans ces conditions, l'exploration de la topographie membranaire par la microscopie de conductance ionique à balayage n'a montré aucune évolution de la surface ni de lésion du sarcolemmel dans les conditions d'étirement. L'étude s'est donc focalisée sur l'activité de candidats moléculaires des SACs et plus particulièrement ceux appartenant à la famille des TRPs (Transient Receptor Potential) dans le dérèglement de l'homéostasie calcique induite par l'étirement. Les influx cationiques évalués par la technique d'extinction de fluorescence et l'étude de la concentration intracellulaire de Ca2+ ([Ca2+]i) grâce à la sonde Fluo8 montrent une implication des canaux TRPV2 et TRPCs. Les premiers semblent responsables d'une entrée cationique et d'une augmentation de [Ca2+]i importante dans les cardiomyocytes mdx. Les seconds, bien que responsables d'un influx, ne participeraient pas à l'augmentation de [Ca2+]i. Ces résultats révèlent que les canaux TRPV2 pourraient jouer un rôle important dans la dérégulation calcique observée dans les cardiomyocytes déficients en dystrophine
Duchenne muscular dystrophy (DMD) is the consequence of the loss of dystrophin, a subsarcolemmal protein essential for mechanical and functional maintenances of the sarcolemma. This deficiency could increase cationic influxes by membrane microruptures or by dysregulation of channels such as stretch-activated channels (SACs). In this work, the effects of a mechanical stretch were explored on cardiomyocytes in the pathological context of dilated cardiomyopathy associated with DMD. Using carbon fibers, an homogenous axial stretch was performed to mimic physiological conditions of ventricular filling. In these conditions, exploration of membrane topography using the scanning ion conductance microscopy did not show any surface evolution or sarcolemma disruption in stretch condition. The study was thus focused on activity and identification of molecular candidates for SACs, especially the TRPs (Transient Receptor Potential) channels in the stretch-induced. Ca2+ homeostasis dysregulation. Cationic influxes assessed by Mn2+-quenching and assessment of the intracellular Ca2+ concentration ([Ca2+]i) using fluo-8 fluorescence demonstrated an involvement of TRPV2 and TRPCs channels. The first ones seem to be responsible for cationic entry and [Ca2+]i increase in mdx cardiomyocytes. The latter, though responsible for an influx, do not contribute to [Ca2+]i increase. These findings reveal that TRPV2 channels could play an important role in calcium dysregulation observed in dystrophin-deficient cardiomyocytes

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6

Rodrigues, Andr?a Mendon?a. "Nova proposta de modelo murino de asma aguda : utiliza??o de protocolo curto sem adjuvante com sensibiliza??o a ovalbumina." Pontif?cia Universidade Cat?lica do Rio Grande do Sul, 2011. http://tede2.pucrs.br/tede2/handle/tede/1360.

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Introdu??o: V?rias limita??es t?m sido levantadas em estudos de asma utilizando modelos murinos agudos sensibilizados com ovalbumina (OVA), mas este modelo ? ainda amplamente usado, ainda mantendo sua import?ncia em estudos pr?-cl?nicos. Algumas limita??es encontradas s?o o uso de adjuvante e os longos per?odos de sensibiliza??o. Objetivos: Testar se a sensibiliza??o com OVA em um per?odo curto, sem adjuvante, induziria uma resposta pulmonar eosinof?lica em camundongos similar aos protocolos j? previamente estabelecidos. M?todos: F?meas adultas de camundongos BALB/c foram utilizadas e divididas em grupos de acordo com o n?mero de sensibiliza??es com OVA (uma ou duas vezes, OVA: 20 μg) e o n?mero(duas ou tr?s vezes)/dosagem(40 μg e 100 μg) de desafios intranasais. O protocolo mais curto (10 dias) consistiu de uma sensibiliza??o subcut?nea e tr?s desafios com OVA (100 μg). Contagem total (CTC) e diferencial de c?lulas no lavado broncoalveolar (LBA), ensaio da peroxidase eosinof?lica (EPO) do tecido pulmonar e histopatologia (HE) dos pulm?es foram realizados 24 horas ap?s o ?ltimo desafio com OVA. Resultados: Contagem celular do LBA, EPO do tecido pulmonar e altera??es inflamat?rias da histologia pulmonar n?o foram diferentes entre os grupos estudados. O protocolo mais curto induziu uma resposta eosinof?lica pulmonar ? OVA semelhante ao grupo controle, ocorrendo o mesmo com os outros grupos. Conclus?o: O uso de sensibiliza??o subcut?nea com OVA, sem adjuvante, resulta em uma significativa resposta pulmonar al?rgica, permitindo sua utiliza??o em protocolos de dura??o mais curta. Nossos achados sugerem que este protocolo pode ser utilizado como teste pr?-cl?nico de primeira linha para pesquisa de novos f?rmacos, reduzindo custo, tempo e uso de adjuvante.

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7

Rajput, Sandeep. "Murine Aldo-Keto Reductases: Identification of AKR1B8 as an ortholog of human AKR1B10." Available to subscribers only, 2009. http://proquest.umi.com/pqdweb?did=1966541891&sid=6&Fmt=2&clientId=1509&RQT=309&VName=PQD.

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Thesis (M.S.)--Southern Illinois University Carbondale, 2009.
"Department of Molecular Biology, Microbiology and Biochemistry." Includes bibliographical references (p. 31-37). Also available online.

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8

Alvarez, Espitia Jorge. "Mechanisms of brain barrier disruption and leukocyte extravasation in murine neurocysticercosis : a dissertation /." San Antonio : UTHSC, 2006. http://proquest.umi.com/pqdweb?did=1390336361&sid=1&Fmt=2&clientId=70986&RQT=309&VName=PQD.

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9

Tian, Hua. "Visualisation and profiling of lipids in single biological cells using time-of-flight secondary ion mass spectrometry." Thesis, University of Manchester, 2012. https://www.research.manchester.ac.uk/portal/en/theses/visualisation-and-profiling-of-lipids-in-single-biological-cells-using-timeofflight-secondary-ion-mass-spectrometry(c36313be-4ffd-4809-b5c9-8fbe1f720bd1).html.

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Imaging Time-of-Flight secondary ion mass spectrometry (ToF-SIMS) has been developed to perform 2D imaging and depth profiling of biological systems with micron or submicron scale lateral resolution, which can be attributed to the advent of polyatomic ion beam particularly C60+ and new concept of ToF-SIMS instrument, the J105 3D Chemical Imager (J105). These recent advances in ToF-SIMS have opened a new dimension for biological analysis. In this study, 2D and 3D imaging have been performed on two biological systems, Xenopus laevis (X. laevis) zygote/embryo and murine embryonic fibroblasts NIH 3T3 BXB-ER cells to explore the capability of ToF-SIMS to handle the biological samples with extreme topography and high resolution depth profiling of microdomains, which still represent major challenges for the ToF-SIMS. The study on X. laevis embryo explored the capability of ToF-SIMS to handle spherical samples (approx. 1-1.2 mm in diameter), identify lipid species in mixtures of lipid extraction from the zygotes and image of an intact embryo in 2D/3D during dynamic biological events, e.g., fertilisation and early embryo development. For the first time the J105 and conventional BioToF-SIMS instrument were employed for the study of developmental biology. The major classes of lipid were identified through multiple lipid assay in a single analytical run using ToF-SIMS. Topography effects of the embryo were assessed through imaging a single intact zygote/embryo that revealed secondary ions loss at the edge of the single cell. However, the topography effects on the mass resolution could be minimised using the J105. Moreover, in situ lipid profiling of the zygote revealed different lipid compositions and intensities on the membrane of the animal and vegetal hemispheres. Furthermore, high resolution imaging and depth profiling that performed on a single intact cell in a time course study visualised the egg-sperm fusion sites on the membrane of the zygote 10 min post-insemination and lipids arrangement on the membrane of the embryo through the early development stages. Subcellular signalling upon the fertilisation was also spatially located on the serial cryosections of a single zygote. With the NIH 3T3 BXB-ER cells, the study firstly adopted a finely focused C60+ beam to track morphological changes and rearrangement of subcellular organelle mitochondria (0.5-2 µm) in response to the activation of Raf/ERK (extracellular signal regulated kinase) pathway using the J105. The SIMS images of the unlabelled cells showed the shifting of membrane distribution and nuclei shrinking following Raf/ERK activation. The mitochondria fluorescence probe within the cells were located 3-dimensionally using confocal microscopy and ToF-SIMS, which revealed the distribution pattern of condensing in the two sides of the nuclei following the Raf/ERK activation. Coupled with scanning electron microscopy (SEM), the three imaging modes showed good agreement in cellular morphological changes and subcellular mitochondrial rearrangement without or following Raf/ERK activation, demonstrating an integrated approaching to study the biological processes at subcellular dimension.

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10

McPherson, Jr Michael Gene. "Microbial interactions, B cell immunoregulation , and positron emission detection in murine models of intestinal inflammation." Diss., Restricted to subscribing institutions, 2008. http://proquest.umi.com/pqdweb?did=1580937001&sid=1&Fmt=2&clientId=48051&RQT=309&VName=PQD.

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11

Puntambekar, Shweta Satish. "Molecular analysis of microglial activation and macrophage recruitment in murine models of neuroinflammation." Diss., [Riverside, Calif.] : University of California, Riverside, 2010. http://proquest.umi.com/pqdweb?index=0&did=2019822741&SrchMode=2&sid=2&Fmt=2&VInst=PROD&VType=PQD&RQT=309&VName=PQD&TS=1274114350&clientId=48051.

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Thesis (Ph. D.)--University of California, Riverside, 2010.
Includes abstract. Available via ProQuest Digital Dissertations. Title from first page of PDF file (viewed May 17, 2010). Includes bibliographical references. Also issued in print.

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12

Chen, Qian. "Validation and localization of restricted gene expression in the developing prostate -- with emphasis on PAX gene expression profiling and functional studies during murine prostate development." Access to citation, abstract and download form provided by ProQuest Information and Learning Company; downloadable PDF file, 150 p, 2010. http://proquest.umi.com/pqdweb?did=1992440861&sid=7&Fmt=2&clientId=8331&RQT=309&VName=PQD.

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13

Herbreteau, Vincent. "Géographie de zoonoses en Thaïlande : de la distribution des rongeurs, vecteurs et hôtes, au risque de transmission." Phd thesis, Université de Nanterre - Paris X, 2007. http://tel.archives-ouvertes.fr/tel-00376326.

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Longtemps considérés en Thaïlande comme simple gibier mais souvent destructeurs des cultures, les rats et les souris (Murinae) se sont révélés d'importants vecteurs de germes pathogènes pour l'Homme, depuis l'émergence soudaine de la leptospirose en 1996. Ils sont aussi responsables de la transmission du typhus des broussailles et probablement d'hantaviroses dont l'incidence reste suspectée. Cette thèse a pour objectif d'analyser la géographie de ces zoonoses afin d'en mesurer le risque de transmission à l'Homme.
Un important travail de terrain a permis de collecter et d'étudier les rongeurs murins dans différents milieux représentatifs de leur diversité. Parallèlement, une enquête conduite dans la province de Phrae a montré la variabilité du système de soins et des comportements de santé. Un Système d'Information Géographique « Rongeurs et santé » centralise l'intégralité des données sur l'ensemble du territoire pour une analyse spatio-temporelle.
Cette recherche a permis de mettre à jour la description et la distribution par télédétection des principaux rongeurs murins thaïlandais ainsi que leur implication dans la transmission de germes pathogènes. La géographie de ces zoonoses reflète des différences de niveau de vie : l'exposition de l'Homme à ces maladies résulte de la chasse et de la consommation de rongeurs mais aussi d'un accès et d'un recours aux soins limités, traduisant ainsi la pauvreté des populations touchées.
Ce travail offre une approche critique des méthodes alliant les outils de la géomatique, l'analyse spatiale et la télédétection, pour l'étude des zoonoses.

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14

Woods, Susan Lesley. "Cross-talk between SIM and other bHLH/PAS factors and the search for SIM2 target genes." Thesis, 2004. http://hdl.handle.net/2440/65465.

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This project aimed to investigate how the murine Simgle minded (mSIM) proteins interact with other active bHLH/PAS transcription factors and provide the first study to identify direct target genes of mSIM2. We show that the murine SIM (mSIM) factors are constitutively nuclear proteins in cells and that the presence of an mSIM protein outcompetes the dioxin receptor (DR) for aryl hydrocarbon nuclear translocator 1 (ARNT1) binding, resulting in attenuation of transcription of a reporter gene induced by a constitutively active form of the DR. In contrast, the hypoxia inducible factor-1a (HIF-1a) and mSIM proteins appear equally competent to sequester ARNT1. Expression of either mSIM protein repressed, but did not totally prevent, the hypoxic activation of a reporter containing hypoxic response element (HRE) sequences from the erythropoietin (EPO) enhancer and endogenous hypoxic target genes in 293T cells. This repression occurs through competition of the HIF-1a and theSIM proteins for binding to ARNT1 but also for DNA binding sites in the HRE-reporter plasmid. In contrast to HIF-1a, which rapidly accumulates in low oxygen conditions, SIM protein levels decrease in hypoxia, most likely as a result of general translational inhibition in hypoxia stressed cells. Cross-talk between the mSIM and HIF-a or DR proteins may occur to enable the cell to adapt to multiple environmental and developmental signals and is particularly relevant in pathological states such as Down’s Syndrome (DS) or tumour types recently recognized to contain elevated levels of a short isoform of human SIM2 (hSIM2). Microarray experiments identified a large number of transcripts that are differentially regulated by mSIM2 and one of thes predominantly expressed in muscle, Myomesin2 (Myom2), was shown to potentially be the first identified direct target gene of mSIM2/ARNT1. Bacterially expressed and partially purified N-terminal portions of mSIM2/ARNT1 bound a 40bp probe derived from the Myom2 promoter sequence but not an identical probe with the 5’-AACGTG-3’ site mutated, and mutation of the putative mSIM2/ARNT1 binding site in the Myom2 promoter led to a decreased reporter induction with the mSIM2AD chimeric protein, in which the mSIM2 repression region is replaced with the activation domain from the DR. Evidence that mSIM2 regulates the transcription of endogenous Myom2 awaits the generation of a specific anti-mSIM2 antibody to allow examination of the Myom2 promoter occupation by mSIM2/ARNT1 using chromatin immunoprecipitation and the effect of mSim2 knockdown on Myom2 transcript levels. The transcriptional activity of the mSIM proteins appears to be dependent on the promoter context and cell type examined, as surprisingly we have observed the mSIM1/ARNT1 dimer activating transcription of the HRE-reporter, whilst the mSIM2/ARNT1 dimer does not activate the Myom2-reporter in 293T cells, but does activate it in 293 cells. Both SIM1 and SIM2 were previously described as harbouring repression regions in their C-termini, however, when dimerised with ARNT1, activation is sometimes observed which is dependent on the presence of the ARNT1 transactivation domain.
Thesis (Ph.D.) -- University of Adelaide, School of Molecular and Biomedical Science, 2004

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15

Kim, Young [Verfasser]. "Der Effekt von murinem IL-12 und CD137L im Mausmodell bei der Behandlung des multiplen Myeloms / Young-Sik Kim." 2008. http://d-nb.info/989636267/34.

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16

Gameiro, Sofia Ribeiro. "Pulmonary delivery of liposome-based vaccines application to a murine model of Schistosoma mansoni infection /." 2008. http://proquest.umi.com/pqdweb?did=1546799171&sid=10&Fmt=2&clientId=39334&RQT=309&VName=PQD.

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Thesis (Ph.D.)--State University of New York at Buffalo, 2008.
Title from PDF title page (viewed on Dec. 3, 2008) Available through UMI ProQuest Digital Dissertations. Thesis adviser: Straubinger, Robert M. Includes bibliographical references.

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Keepers, Tiffany Rae. "Renal inflammation in a shiga toxin plus lipopolysaccharide induced murine model of hemolytic uremic syndrome." 2007. http://proquest.umi.com/pqdweb?did=1801471441&sid=4&Fmt=2&clientId=3507&RQT=309&VName=PQD.

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Psotka, Mitchell Adam. "The pathophysiology of renal failure in a shiga toxin plus lipopolysaccharide induced murine model of hemolytic uremic syndrome." 2008. http://proquest.umi.com/pqdweb?did=1805440271&sid=3&Fmt=2&clientId=3507&RQT=309&VName=PQD.

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Muralidharan, Ranjani. "Antifungal activity of salivary mucin-derived peptide, MUC7 12-mer, in an in-vivo murine model of oral candidiasis." 2005. http://proquest.umi.com/pqdweb?did=1027490561&sid=11&Fmt=2&clientId=39334&RQT=309&VName=PQD.

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Thesis (M.S.)--State University of New York at Buffalo, 2005.
Title from PDF title page (viewed on May 11, 2006) Available through UMI ProQuest Digital Dissertations. Thesis adviser: Baier, Robert E., Bobek, Libuse A. Includes bibliographical references.

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Beaudoin, Frédéric. "Analyse de la réponse immune contre Chlamydia pneumoniae à l'aide d'anticorps monoclonaux murins /." 2001. http://proquest.umi.com/pqdweb?did=766651801&sid=58&Fmt=2&clientId=9268&RQT=309&VName=PQD.

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21

Laplante, Alain. "Expression des protéines de stress au cours de la guérison de plaies cutanées murines /." 1998. http://proquest.umi.com/pqdweb?did=734140501&sid=29&Fmt=2&clientId=9268&RQT=309&VName=PQD.

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22

Seaborn, Tommy. "Caractérisation d'un modèle d'explant pulmonaire murin pour l'étude des mécanismes biomoléculaires induits par l'occlusion trachéale fœtale /." 2001. http://proquest.umi.com/pqdweb?did=766398141&sid=31&Fmt=2&clientId=9268&RQT=309&VName=PQD.

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23

Morency, Vincent. "Prévention de la dégénérescence de motoneurones dans une modèle murin transgénique de sclérose latérale amyotrophique à l'aide du "ciliary neurotrophic factor" (CNTF) /." 2004. http://proquest.umi.com/pqdweb?did=845769681&sid=4&Fmt=2&clientId=9268&RQT=309&VName=PQD.

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Racine, Claudia. "Étude de l'expression de la protéine hsp-72 par le macrophage alvéolaire dans l'alvéolite allergique extrinsèque chez l'humain et parallèlement dans un modèle murin /." 1997. http://proquest.umi.com/pqdweb?did=738288491&sid=6&Fmt=2&clientId=9268&RQT=309&VName=PQD.

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