Log in

Relevant bibliographies by topics / Murine Mycobacterium tuberculosis infection / Books

Books on the topic 'Murine Mycobacterium tuberculosis infection'

To see the other types of publications on this topic, follow the link: Murine Mycobacterium tuberculosis infection.

Author: Grafiati

Published: 10 December 2022

Last updated: 28 January 2023

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the top 25 books for your research on the topic 'Murine Mycobacterium tuberculosis infection.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Browse books on a wide variety of disciplines and organise your bibliography correctly.

1

O, Thoen Charles, and Steele James H, eds. Mycobacterium bovis infection in animals and humans. Ames, Iowa: Iowa State University Press, 1995.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

2

O, Thoen Charles, Steele James H, and Gilsdorf Michael J, eds. Mycobacterium bovis infection in animals and humans. 2nd ed. Ames, Iowa: Blackwell Pub., 2006.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

3

Venketaraman, Vishwanath, ed. Understanding the Host Immune Response Against Mycobacterium tuberculosis Infection. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-97367-8.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Hasnain, Seyed Ehtesham, Nasreen Z. Ehtesham, and Sonam Grover, eds. Mycobacterium Tuberculosis: Molecular Infection Biology, Pathogenesis, Diagnostics and New Interventions. Singapore: Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-32-9413-4.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

Hingley-Wilson, Suzanne Maria. The detection and identification of macrophage genes induced by intracellular infection with mycobacterium tuberculosis. Birmingham: University of Birmingham, 1998.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

6

Alli, Oyebode Armstrong Terry. Development of a RAP-PCR strategy to study mycobacterium tuberculosis gene expression during intracellular infection of macrophages. Birmingham: University of Birmingham, 1998.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

7

Steele, James H., Charles O. Thoen, and Michael J. Gilsdorf. Mycobacterium Bovis Infection in Animals and Humans. Wiley & Sons, Incorporated, John, 2008.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

8

Understanding the Host Immune Response Against Mycobacterium tuberculosis Infection. Springer, 2018.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

9

Understanding the Host Immune Response Against Mycobacterium tuberculosis Infection. Springer, 2018.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

10

Steele, James H., Charles O. Thoen, and Michael J. Gilsdorf. Mycobacterium Bovis Infection in Animals and Humans. Wiley & Sons, Incorporated, John, 2010.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

11

Aston, Stephen, Geraint Davies, and Nick Beeching. Mycobacterial infection other than tuberculosis. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0311.

Full text

Abstract:

Mycobacteria are aerobic bacilli with a lipid-rich cell wall and are widespread both in the environment and in animals. Many species within the genus cause disease in humans, most notably those of the Mycobacterium tuberculosis complex, which cause tuberculosis, and Mycobacterium leprae, the causative agent of leprosy. Several other species, termed non-tuberculous mycobacteria, can cause chronic cutaneous, pulmonary, and disseminated infections. This chapter will briefly review infection with non-tuberculous mycobacteria and Mycobacterium leprae.

APA, Harvard, Vancouver, ISO, and other styles

12

Mycobacterium tuberculosis Pathogenesis, Infection Prevention and Treatment. MDPI, 2020. http://dx.doi.org/10.3390/books978-3-03936-659-0.

Full text

APA, Harvard, Vancouver, ISO, and other styles

13

Mycobacterium Tuberculosis: Molecular Infection Biology, Pathogenesis, Diagnostics and New Interventions. Springer Singapore Pte. Limited, 2020.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

14

Cuervo, Lilian María Mederos. Infection for Mycobacterium tuberculosis and Nontuberculous Mycobacteria in the HIV. INTECH Open Access Publisher, 2011.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

15

Hasnain, Seyed Ehtesham, Nasreen Z. Ehtesham, and Sonam Grover. Mycobacterium Tuberculosis: Molecular Infection Biology, Pathogenesis, Diagnostics and New Interventions. Springer, 2019.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

16

Haldar, Pranabashis. Tuberculosis. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0130.

Full text

Abstract:

Tuberculosis (TB) is an infectious disease caused by the bacterial organism Mycobacterium tuberculosis. In this context, reference to the word ‘disease’ is important, as TB implies Mycobacterium tuberculosis infection associated with symptoms. Approximately 10% of Mycobacterium tuberculosis infection is manifest as disease. In the large majority of cases, Mycobacterium tuberculosis infection is latent and defined by evidence of a measurable and significant cell-mediated immune response to mycobacterial antigens, in the absence of clinical or radiological evidence of disease. TB may be clinically classified further according to the site of disease. Miliary TB refers to systemic disease that may affect multiple organs.

APA, Harvard, Vancouver, ISO, and other styles

17

Joint Commission on Accreditation of Healthcare Organizations., ed. Tuberculosis: Arresting everyone's enemy. Oakbrook Terrace, IL: Joint Commission on Accreditation of Healthcare Organizations, 1996.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

18

Garnett, Ben. Behavioural aspects of bovine tuberculosis (Mycobacterium bovis) transmission and infection in badgers (Meles meles). 2003.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

19

A, Jensen Paul, Centers for Disease Control and Prevention (U.S.), and National Center for HIV, STD, and TB Prevention (U.S.), eds. Guidelines for preventing the transmission of Mycobacterium tuberculosis in health-care settings, 2005. Atlanta, GA: U.S. Dept. of Health and Human Services, Public Health Service, Centers for Disease Control and Prevention, 2005.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

20

David, Schlossberg, ed. Tuberculosis and nontuberculous mycobacterial infections. 5th ed. New York: McGraw-Hill, Medical Pub. Division, 2005.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

21

Hansoti, Bhakti. Pulmonary Tuberculosis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0028.

Full text

Abstract:

Mycobacterium tuberculosis (TB) is most commonly known for its manifestations in the lungs; symptoms include fever and chest pain (retrosternal pain and/or dull intracapsular pain). In the reactivation stage of TB, typical symptoms may include cough, weight loss, fatigue, fever, night sweats, chest pain, dyspnea, and/or hemoptysis. Symptoms may remain undiagnosed for several years. Poverty, HIV, and drug resistance are major contributors to the resurging global TB epidemic. Two kinds of tests are used to detect TB: the tuberculin skin test or a TB blood test. These tests only tell you if a person has been infected with the bacteria. The do not differentiate between latent TB infection and active TB. This distinction clinically suspected when the clinical picture of active TB matches with initial investigations (such as acid-fast bacilli stains, chest x-ray, or CT) and is definitively confirmed by the growth of M. tuberculosis in a clinical specimen.

APA, Harvard, Vancouver, ISO, and other styles

22

Heyns, Chris. Tuberculosis and parasitic infestations involving the urogenital system. Edited by Rob Pickard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0006.

Full text

Abstract:

Urogenital tuberculosis is caused by Mycobacterium tuberculosis, which evokes a granulomatous tissue reaction leading to caseous necrosis, fibrosis, and eventual calcification. It most commonly presents as cystitis with sterile pyuria but can show many other symptoms and signs requiring a high index of suspicion to make the diagnosis. Schistosomiasis (Bilharzia) affecting the urinary tract is caused by the flatworm Schistosoma haematobium. Humans are infested by contact with fresh water harbouring the intermediate snail host. Echinococcosis (hydatid disease), is caused by the tapeworm Echinococcus granulosis or multilocularis. Human infection results from close contact with the parasite host (usually dogs and sheep). Filariasis, caused by the roundworm Wuchereria bancrofti, is transmitted by mosquito bite

APA, Harvard, Vancouver, ISO, and other styles

23

B, Heifets Leonid, and Cynamon Michael H, eds. Drug susceptibility in the chemotherapy of mycobacterial infections. Boca Raton, Fla: CRC Press, 1991.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

24

Nageshwaran, Sathiji, Heather C. Wilson, Anthony Dickenson, and David Ledingham. Neurological infections. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199664368.003.0015.

Full text

Abstract:

This chapter reviews the clinical features and management of meningitis (community-acquired bacterial meningitis and chronic meningitis), Mycobacterium tuberculosis, encephalitis and myelitis, Lyme disease, brain abscess and parameningeal infection, neurological infections in the immunocompromised, fungal infection, parasitic infection, and bacterial neurotoxins.

APA, Harvard, Vancouver, ISO, and other styles

25

Eastwood, John, Cathy Corbishley, and John Grange. Mycobacterial infections. Edited by Vivekanand Jha. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0196.

Full text

Abstract:

The genus Mycobacterium contains over a hundred species including the M. tuberculosis complex and M. leprae, the causative agents of, respectively, tuberculosis and leprosy. The many other species are environmental saprophytes, present particularly in free and piped water sources, and some species are causes of opportunist disease in humans, especially in those who are immune compromised.The genitourinary tract is a common site of both primary and post-primary tuberculosis. In most cases of renal tuberculosis there are gross lesions consisting of caseating granulomas from which tubercle bacilli enter the urinary tract, often with the development of secondary lesions in the ureters, bladder, epididymis, and testis. Tuberculous interstitial nephritis is a less common condition with an insidious course and may result in renal failure. The urine is often negative for tubercle bacilli, emphasizing the need for biopsy in those with renal insufficiency.The risk of developing pulmonary or disseminated tuberculosis after infection is greatly enhanced by any form of immune compromise including renal failure and post-renal transplant immunosuppression.

APA, Harvard, Vancouver, ISO, and other styles

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!