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Relevant bibliographies by topics / Multiple sclerosis / Journal articles

Journal articles on the topic 'Multiple sclerosis'

To see the other types of publications on this topic, follow the link: Multiple sclerosis.

Author: Grafiati

Published: 4 June 2021

Last updated: 29 July 2024

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1

KAMALAK, HAKAN, and HICHAM NUAIMI. "MULTİPLE SCLEROSİS AND AMALGAM." Asian Pacific Journal of Health Sciences 1, no. 3 (July 2014): 146–48. http://dx.doi.org/10.21276/apjhs.2014.1.3.3.

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2

Artur Medina, Jose. "Multiple Sclerosis and Chlamydia." International Journal of Clinical Case Reports and Reviews 9, no. 1 (November 30, 2021): 01–02. http://dx.doi.org/10.31579/2690-4861/163.

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Although the epidemiological similarity between the two diseases is true, this thesis was not discussed extensively, perhaps because it implied that some children might have been victims of abuse, which sounds false and potentially unfair. We believe that transverse myelitis and MS are the result of an infectious disease, eventually sexually transmitted by chlamydia/gonococcus, which is caused by a subclinical bacterial urethritis/inflamatory pelvic disease (IPD) among adults. In children it is the same disease but caused by common uropathogens/enterobacteria. Both UTI and MS are much more common in girls than in boys [2, 3]. These UTIs would favor herpetic proliferation via toll-like receptors (TLRs), since the virus is endemic and always present, and it is not possible to eradicate it completely. Herpes viral load is counteracted by interferon alpha 1 (IFN alpha-1), present in different cell types, from macrophages to lymphocytes passing through endothelia and fibroblasts. Interferon alpha 1, when interacting with its specific receptors, produces in the intracellular the action of antiviral RNAse and the inhibition of viral protein synthesis

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3

Shepeleva, A. D., D. F. Shamardanov, and E. V. Ponomarenko. "PATHOPHYSIOLOGY OF MULTIPLE SCLEROSIS." European Journal of Natural History, no. 4 2022 (2022): 12–16. http://dx.doi.org/10.17513/ejnh.34281.

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4

ÖZTÜRK, Serra, Güneş AYTAÇ, Ferah KIZILAY, and Muzaffer SİNDEL. "Multiple Sclerosis." Akdeniz Medical Journal 3, no. 3 (September 28, 2017): 137–47. http://dx.doi.org/10.17954/amj.2017.86.

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5

Marenčák, Jozef. "Multiple sclerosis and male infertility." Urologie pro praxi 19, no. 4 (October 1, 2018): 181–84. http://dx.doi.org/10.36290/uro.2018.083.

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6

Medina, Jose Artur. "Multiple Sclerosis, Chlamydia and Gut Disbiosis." Journal of Clinical Research and Reports 9, no. 3 (November 20, 2021): 01–02. http://dx.doi.org/10.31579/2690-1919/208.

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I believe that multiple sclerosis progression could be stopped through antibiotics, dewormers, beta glucans and garlic extract and, by far the most difficult, changing the quality of the water ingested, or used for washing salads, in addition to increasing the consumption of probiotics. I believe that MS sclerosis is due to the unfortunate encounter of 3 diseases: intestinal dysbiosis, chlamydia infections and orthopedic problems. However, how would these three diseases cause demyelination of nerve fibers?

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7

Yamada, Shoko M., So Yamada, Hiroshi Nakaguchi, Mineko Murakami, Katsumi Hoya, Akira Matsuno, Kazuto Yamazaki, and Yasuo Ishida. "A Case of Tumefactive Multiple Sclerosis." Japanese Journal of Neurosurgery 21, no. 5 (2012): 427–32. http://dx.doi.org/10.7887/jcns.21.427.

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8

Kantorová, Ema. "Significance of multiple sclerosis early treatment." Neurologie pro praxi 20, no. 4 (September 1, 2019): 288–90. http://dx.doi.org/10.36290/neu.2019.155.

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9

La Mantia, L. "Neurodegenerazione, neuroprotezione e sclerosi multipla [Neurodegeneration, neuroprotection and multiple sclerosis]." Neurological Sciences 27, no. 4 (September 2006): 293. http://dx.doi.org/10.1007/s10072-006-0689-5.

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10

Antonis, Theofilidis. "Investigating memory storage difficulties in Multiple Sclerosis." Neuroscience and Neurological Surgery 9, no. 5 (November 24, 2021): 01–07. http://dx.doi.org/10.31579/2578-8868/185.

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Μemory by itself as a function, loses its capabilities with a normal deterioration. However, there are acquired conditions that negatively affect the functions of memory, resulting in dysfunction of its stages. Thus possible damage to the structures of the hemisphere that controls these processes disrupts the comprehension, organization and categorization of the material to be memorized. Patients with damage to these systems will have difficulty remembering because they have not adequately coded the material. Patients with Multiple Sclerosis report short-term memory difficulties in the sense that they have difficulty remembering details of recent conversations and events. Aim: To investigate the memory storage difficulties in Multiple Sclerosis. Materials and Methods: An international literature review was performed on Memory Disorders in Multiple Sclerosis. Conclusion: In patients with Multiple Sclerosis learning deficits are greatly aided by processing speed and working memory. It has been observed that slow mental processing makes it difficult for many patients with Multiple Sclerosis to capture an entire verbal message, especially if it is large, complex, delivered quickly and with external stimuli, such as a noisy environment

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11

ARYA, ASHWANI, SAHIL KUMAR, and RAKESH K. SINDHU. "Clinical Symptoms and Therapies for Multiple Sclerosis." JOURNAL OF PHARMACEUTICAL TECHNOLOGY, RESEARCH AND MANAGEMENT 3, no. 1 (May 2, 2015): 29–47. http://dx.doi.org/10.15415/jptrm.2015.31003.

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12

Kantorová, Ema, Babeta Hofericová, and Egon Kurča. "Epileptic seizures in patients with multiple sclerosis." Neurologie pro praxi 19, no. 3 (July 1, 2018): 187–92. http://dx.doi.org/10.36290/neu.2018.002.

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13

Donáth, Vladimír. "Cortical inflammation in pathogenesis of multiple sclerosis." Neurologie pro praxi 20, no. 4 (September 1, 2019): 312–15. http://dx.doi.org/10.36290/neu.2019.134.

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14

Lisý, Ľubomír. "The new trends in multiple sclerosis treatment." Neurologie pro praxi 21, no. 2 (June 2, 2020): 120–24. http://dx.doi.org/10.36290/neu.2020.059.

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15

Harrison, Daniel M. "Multiple Sclerosis." Annals of Internal Medicine 160, no. 7 (April 1, 2014): ITC4–1. http://dx.doi.org/10.7326/0003-4819-160-7-201404010-01004.

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16

Sutton, Lucy. "Multiple sclerosis." Nursing Standard 12, no. 16 (January 7, 1998): 48–56. http://dx.doi.org/10.7748/ns.12.16.48.s50.

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17

Gaby, Alan. "Multiple Sclerosis." Global Advances in Health and Medicine 2, no. 1 (January 2013): 50–56. http://dx.doi.org/10.7453/gahmj.2013.2.1.009.

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18

Kausar, Shahid A. "Multiple sclerosis." Clinical Medicine 20, no. 5 (September 2020): e138.3-e139. http://dx.doi.org/10.7861/clinmed.let.20.5.7.

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19

Eberhardie, Chris, and Chris Eberhardie. "Multiple Sclerosis." Nursing Standard 4, no. 52 (September 19, 1990): 42–43. http://dx.doi.org/10.7748/ns.4.52.42.s52.

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20

Wei, Fu-Chan. "Multiple sclerosis." Biomedical Journal 37, no. 2 (2014): 33. http://dx.doi.org/10.4103/2319-4170.130317.

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21

Olek, Michael J. "Multiple Sclerosis." Annals of Internal Medicine 174, no. 6 (June 2021): ITC81—ITC96. http://dx.doi.org/10.7326/aitc202106150.

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22

Zurawska, Anna E., Marcin P. Mycko, Igor Selmaj, Cedric S. Raine, and Krzysztof W. Selmaj. "Multiple Sclerosis." Neurology - Neuroimmunology Neuroinflammation 8, no. 5 (August 12, 2021): e1041. http://dx.doi.org/10.1212/nxi.0000000000001041.

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Background and ObjectivesTo investigate the total circular RNA (circRNA) profile in patients with relapsing-remitting multiple sclerosis (RRMS) and healthy controls (HCs).MethodsHybridization microarray was used to define the circRNA profile in peripheral blood mononuclear cells (PBMCs) from 20 untreated patients with RRMS (10 in relapse and 10 in remission) and 10 HCs. We analyzed close to 14,000 individual circRNAs per sample. The discovery set data were validated using quantitative reverse transcription-PCR with an independent cohort of 47 patients with RRMS (19 in relapse and 28 in remission) and 27 HCs.ResultsMicroarray analysis revealed 914 transcripts to be differentially expressed between patients with RRMS in relapse and HCs (p < 0.05). We validated 3 circRNAs from 5 showing highest levels of differential expression in the RRMS relapse vs HC group: hsa_circRNA_101348, hsa_circRNA_102611, and hsa_circRNA_104361. Their expression was significantly increased during relapse in RRMS (p = 0.0002, FC = 2.9; p = 0.01, FC = 1.6; and p = 0.001, FC = 1.5, respectively) and in patients showing gadolinium enhancement on brain MRI (hsa_circRNA_101348, p = 0.0039, FC = 2.4; hsa_circRNA_104361, p = 0.029, FC = 1.7). Bioinformatic analysis revealed 15 microRNAs interacting with these circRNAs in a complementary manner and led to the discovery and validation of 3 protein-coding RNAs upregulated in patients with RRMS during relapse. Two of these, AK2 and IKZF3, have previously been implicated in B-cell function.DiscussioncircRNAs display a distinct profile in PBMCs from patients with RRMS, and our results may implicate circRNA in the known disturbed B-cell activity in RRMS and thus represent a novel biomarker for monitoring relapse activity.

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23

Kim, Kwang-Kuk. "Multiple sclerosis." Journal of the Korean Medical Association 56, no. 8 (2013): 702. http://dx.doi.org/10.5124/jkma.2013.56.8.702.

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24

Kim, Kwang-Kuk. "Multiple Sclerosis." Journal of the Korean Medical Association 50, no. 3 (2007): 252. http://dx.doi.org/10.5124/jkma.2007.50.3.252.

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25

Kim, Woojun, and Ho Jin Kim. "Multiple Sclerosis." Journal of the Korean Medical Association 52, no. 7 (2009): 665. http://dx.doi.org/10.5124/jkma.2009.52.7.665.

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26

Ramagopalan, Sreeram, David Dyment, Rachel Farrell, and Noriko Isobe. "Multiple Sclerosis." Autoimmune Diseases 2011 (2011): 1. http://dx.doi.org/10.4061/2011/248758.

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27

Kim, J. K., F. G. Mastronardi, D. D. Wood, D. M. Lubman, R. Zand, and M. A. Moscarello. "Multiple Sclerosis." Molecular & Cellular Proteomics 2, no. 7 (June 25, 2003): 453–62. http://dx.doi.org/10.1074/mcp.m200050-mcp200.

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28

Reich, Daniel S., Claudia F. Lucchinetti, and Peter A. Calabresi. "Multiple Sclerosis." New England Journal of Medicine 378, no. 2 (January 11, 2018): 169–80. http://dx.doi.org/10.1056/nejmra1401483.

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29

Tsuda, Hiromasa. "Multiple sclerosis." Equilibrium Research 81, no. 4 (August 31, 2022): 173–83. http://dx.doi.org/10.3757/jser.81.173.

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30

Klocke, Shilpa, and Nicole Hahn. "Multiple sclerosis." Mental Health Clinician 9, no. 6 (November 1, 2019): 349–58. http://dx.doi.org/10.9740/mhc.2019.11.349.

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Abstract Multiple sclerosis is a chronic, unpredictable, and disabling disease. Significant advances have been made in recent years supporting an earlier, more accurate, diagnosis and have led to more than 15 disease-modifying therapies approved by the Food and Drug Administration for relapsing forms of multiple sclerosis. Disease-modifying therapies are now being classified into categories based on level of efficacy. Strategies to use disease-modifying therapies earlier and in a more customizable manner are also emerging. A clinical case study will be used throughout this pearl to review the disease-modifying therapies and use patient-specific factors to develop and provide recommendations on therapeutic strategies for individuals with relapsing forms of multiple sclerosis.

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31

Hafler, David A., Jacqueline M. Slavik, David E. Anderson, Kevin C. O'Connor, Philip De Jager, and Clare Baecher-Allan. "Multiple sclerosis." Immunological Reviews 204, no. 1 (April 2005): 208–31. http://dx.doi.org/10.1111/j.0105-2896.2005.00240.x.

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32

Noseworthy, John H., Claudia Lucchinetti, Moses Rodriguez, and Brian G. Weinshenker. "Multiple Sclerosis." New England Journal of Medicine 343, no. 13 (September 28, 2000): 938–52. http://dx.doi.org/10.1056/nejm200009283431307.

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33

Simon, Chantal. "Multiple Sclerosis." InnovAiT: Education and inspiration for general practice 2, no. 4 (April 2009): 205–11. http://dx.doi.org/10.1093/innovait/inp013.

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Multiple sclerosis is a chronic disabling neurological disease due to an autoimmune process of unknown cause. It is characterized by the formation of patches of demyelination (plaques) throughout the brain and spinal cord. There is no peripheral nerve involvement.

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34

Rühl, Geraldine, Anna G. Niedl, Atanas Patronov, Katherina Siewert, Stefan Pinkert, Maria Kalemanov, Manuel A. Friese, et al. "Multiple sclerosis." Neurology - Neuroimmunology Neuroinflammation 3, no. 4 (May 17, 2016): e241. http://dx.doi.org/10.1212/nxi.0000000000000241.

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35

Keegan, B. Mark, and John H. Noseworthy. "Multiple Sclerosis." Annual Review of Medicine 53, no. 1 (February 2002): 285–302. http://dx.doi.org/10.1146/annurev.med.53.082901.103909.

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36

Benedict, Ralph, and Julie Bobholz. "Multiple Sclerosis." Seminars in Neurology 27, no. 1 (February 2007): 078–85. http://dx.doi.org/10.1055/s-2006-956758.

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37

Hafler, David A. "Multiple sclerosis." Journal of Clinical Investigation 113, no. 6 (March 15, 2004): 788–94. http://dx.doi.org/10.1172/jci21357.

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38

Costello, Fiona E., and Jodie M. Burton. "Multiple Sclerosis." Journal of Neuro-Ophthalmology 38, no. 1 (March 2018): 81–84. http://dx.doi.org/10.1097/wno.0000000000000631.

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39

Pröbstel, Anne-Katrin, and Stephen L. Hauser. "Multiple Sclerosis." Journal of Neuro-Ophthalmology 38, no. 2 (June 2018): 251–58. http://dx.doi.org/10.1097/wno.0000000000000642.

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40

BEIRNE, DANIEL K., and F. REED MURTAGH. "Multiple Sclerosis." Contemporary Diagnostic Radiology 21, no. 1 (1998): 1–5. http://dx.doi.org/10.1097/00219246-199821010-00001.

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41

&NA;, &NA;. "MULTIPLE SCLEROSIS." Critical Care Nursing Quarterly 10, no. 1 (June 1987): 86. http://dx.doi.org/10.1097/00002727-198706000-00018.

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42

Mahdavian, Soheyla, Ukamaka Dike, Alsean Bryant, Carina Davison, Patty Ghazvini, and Angela Hill. "Multiple Sclerosis." Journal of Pharmacy Practice 23, no. 2 (February 18, 2010): 91–100. http://dx.doi.org/10.1177/0897190009360022.

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Multiple sclerosis, an autoimmune disorder causing the destruction of the impulse carrying myelin sheath of neurons, can be a debilitating and disabling disease. Symptomatic treatment has provided patients with relief through the use of antispasmodics, anticholinergics, and antidepressants, just to name a few, as well as disease treatment by decreasing progression of the illness by treating acute episodes through the use of corticosteroids, interferons, plasmapheresis, and other immunomodulators like glatiramer, mitoxantrone, and natalizumab. With medical advancements and the development of new treatments such as laquinimod, fampridine-SR, and several others, the future looks promising for those living with this illness.

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43

Capriotti, Teri, Julia Noel, and Simone Brissenden. "Multiple Sclerosis." Home Healthcare Now 36, no. 3 (2018): 169–80. http://dx.doi.org/10.1097/nhh.0000000000000666.

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44

Lustrin, E. S., M. J. Stallmeyer, J. H. Brown, and F. S. Chew. "Multiple sclerosis." American Journal of Roentgenology 164, no. 3 (March 1995): 672. http://dx.doi.org/10.2214/ajr.164.3.7863891.

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45

Varner, Christine A. "Multiple sclerosis." Nursing Made Incredibly Easy! 18, no. 2 (2020): 20–27. http://dx.doi.org/10.1097/01.nme.0000653172.64362.2e.

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46

&NA;. "Multiple sclerosis." Neurology Now 3, no. 2 (March 2007): 9. http://dx.doi.org/10.1097/01.nnn.0000267386.41653.e4.

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47

Cleary, Angela. "Multiple Sclerosis." Neurology Now 4, no. 6 (November 2008): 8. http://dx.doi.org/10.1097/01.nnn.0000343173.99193.37.

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48

Behrens, Uta M., and Deborah Schoenberger. "Multiple Sclerosis." Neurology Now 5, no. 1 (January 2009): 6. http://dx.doi.org/10.1097/01.nnn.0000345689.14193.53.

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49

Kunkel, Rachael. "Multiple Sclerosis." Neurology Now 5, no. 3 (May 2009): 6. http://dx.doi.org/10.1097/01.nnn.0000356881.40190.b2.

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50

Swainger, Corinne. "Multiple sclerosis." Nursing Management 8, no. 5 (September 2001): 22–26. http://dx.doi.org/10.7748/nm.8.5.22.s15.

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