Log in

Relevant bibliographies by topics / Muller cells / Journal articles

Journal articles on the topic 'Muller cells'

To see the other types of publications on this topic, follow the link: Muller cells.

Author: Grafiati

Published: 4 June 2021

Last updated: 20 February 2023

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the top 50 journal articles for your research on the topic 'Muller cells.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Browse journal articles on a wide variety of disciplines and organise your bibliography correctly.

1

Kosaka, Yudai, and Tetsuhiko Ohba. "3P174 Study on membrane microfluidity of living cells using Muller Matrix microscopy(12. Cell biology,Poster)." Seibutsu Butsuri 53, supplement1-2 (2013): S240. http://dx.doi.org/10.2142/biophys.53.s240_5.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Newman, EA. "Membrane physiology of retinal glial (Muller) cells." Journal of Neuroscience 5, no. 8 (August 1, 1985): 2225–39. http://dx.doi.org/10.1523/jneurosci.05-08-02225.1985.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Ripps, H., R. P. Malchow, and H. Oian. "GABA-mediated currents of skate Muller cells." Experimental Eye Research 55 (September 1992): 17. http://dx.doi.org/10.1016/0014-4835(92)90273-u.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Bringmann, Andreas. "Role of Muller cells in retinal degenerations." Frontiers in Bioscience 6, no. 1 (2001): e77. http://dx.doi.org/10.2741/bringman.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

Linser, Paul J. "Comparative immunochemistry of elasmobranch retina muller cells and horizontal cells." Journal of Experimental Zoology 256, S5 (1990): 88–96. http://dx.doi.org/10.1002/jez.1402560513.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Newman, Eric A. "Physiological properties and possible functions of muller cells." Neuroscience Research Supplements 4 (January 1986): S209—S220. http://dx.doi.org/10.1016/s0921-8696(86)80020-2.

Full text

APA, Harvard, Vancouver, ISO, and other styles

7

Newman, Eric A. "Physiological properties and possible functions of Muller cells." Neuroscience Research 4 (January 1986): S209—S220. http://dx.doi.org/10.1016/0168-0102(86)90084-2.

Full text

APA, Harvard, Vancouver, ISO, and other styles

8

Attwell, David, Marek Szatkowski, Muriel Bouvier, and Alessandra Amato. "Ion movements accompanying glutamate uptake into Muller cells." Experimental Eye Research 55 (September 1992): 17. http://dx.doi.org/10.1016/0014-4835(92)90271-s.

Full text

APA, Harvard, Vancouver, ISO, and other styles

9

Puro, Donald G. "Calcium-permeable ion channels in human Muller cells." Experimental Eye Research 55 (September 1992): 18. http://dx.doi.org/10.1016/0014-4835(92)90275-w.

Full text

APA, Harvard, Vancouver, ISO, and other styles

10

Puro, Donald G. "Stretch-activated channels in human retinal muller cells." Glia 4, no. 5 (1991): 456–60. http://dx.doi.org/10.1002/glia.440040505.

Full text

APA, Harvard, Vancouver, ISO, and other styles

11

Das, S. R., N. Bhardwaj, H. Kjeldbye, and P. Gouras. "Muller cells of chicken retina synthesize 11-cis-retinol." Biochemical Journal 285, no. 3 (August 1, 1992): 907–13. http://dx.doi.org/10.1042/bj2850907.

Full text

Abstract:

The amounts of endogenous retinyl palmitate, retinol and retinaldehyde were measured in the neural retina and retinal pigment epithelium (RPE) of predominantly cone (chicken), rod (rat) and more mixed (cat, human) retinae. The ratio of 11-cis to all-trans isomers of retinyl palmitate and retinol in the neural retina and the RPE increases progressively with the increase in diurnality of the species from rat to chicken. The membrane fractions of both chicken and bovine RPE enzymically isomerize all-trans retinol to 11-cis-retinol. Chicken neural retina membranes enzymically form 11-cis-retinol and all-trans-retinyl palmitate from all-trans-retinol. Light and electron microscopy revealed no contamination of chicken neural retina by RPE. Muller cells from chicken retina were isolated, cultured and characterized by immunocytochemical localization of cellular retinaldehyde-binding protein. Cultured chicken Muller cells form all-trans-retinyl palmitate, 11-cis-retinol and 11-cis-retinyl palmitate from all-trans-retinol and release most of the 11-cis-retinol into the medium. The results indicate that chicken neural retina and Muller cells in particular synthesize 11-cis-retinoids from all-trans-retinol.

APA, Harvard, Vancouver, ISO, and other styles

12

Newman, EA. "Inward-rectifying potassium channels in retinal glial (Muller) cells." Journal of Neuroscience 13, no. 8 (August 1, 1993): 3333–45. http://dx.doi.org/10.1523/jneurosci.13-08-03333.1993.

Full text

APA, Harvard, Vancouver, ISO, and other styles

13

Henshel, Diane S., and Robert F. Miller. "Catecholamine effects on dissociated tiger salamander Muller (glial) cells." Brain Research 575, no. 2 (March 1992): 208–14. http://dx.doi.org/10.1016/0006-8993(92)90081-j.

Full text

APA, Harvard, Vancouver, ISO, and other styles

14

Yao, Jing, Xinghuai Sun, Yang Wang, and Ling Wang. "Muller glia induce retinal progenitor cells to differentiate into retinal ganglion cells." NeuroReport 17, no. 12 (August 2006): 1263–67. http://dx.doi.org/10.1097/01.wnr.0000227991.23046.b7.

Full text

APA, Harvard, Vancouver, ISO, and other styles

15

Schlosshauer, B., D. Grauer, D. Dutting, and J. Vanselow. "Expression of a novel Muller glia specific antigen during development and after optic nerve lesion." Development 111, no. 3 (March 1, 1991): 789–99. http://dx.doi.org/10.1242/dev.111.3.789.

Full text

Abstract:

To generate monoclonal antibodies, immunogen fractions were purified from embryonic chick retinae by temperature-induced detergent-phase separation employing Triton X-114. Under reducing conditions, the monoclonal antibody (mAb) 2M6 identifies a protein doublet at 40 and 46 × 10(3) Mr, which appears to form disulfide-coupled multimers. The 2M6 antigen is regulated developmentally during retinal histogenesis and its expression correlates with Muller glial cell differentiation. Isolated glial endfeet and retinal glial cells in vitro were found to be 2M6-positive, identified with the aid of the general glia marker mAb R5. mAb 2M6 does not bind to any other glial cell type in the CNS as judged from immunohistochemical data. Cell-type specificity was further substantiated by employing retinal explant and single cell cultures on laminin in conjunction with two novel neuron-specific monoclonal antibodies. MAb 2M6 does not bind either to neurites or to neuronal cell bodies. Incubation of retinal cells in vitro with bromodeoxyuridine (BrdU) and subsequent immunodouble labelling with mAb 2M6 and anti-BrdU reveal that mitotic Muller cells can also express the 2M6 antigen. To investigate whether Muller cell differentiation depends on interactions with earlier differentiating ganglion cells, transections of early embryonic optic nerves in vivo were performed. This operation eliminates ganglion cells. Muller cell development and 2M6 antigen expression were not affected, suggesting a ganglion-cell-independent differentiation process. If, however, the optic nerve of juvenile chicken was crushed to induce a transient degeneration/regeneration process in the retina, a significant increase of 2M6 immunoreactivity became evident. These data are in line with the hypothesis that Muller glial cells, in contrast to other distinct glial cell types, might facilitate neural regeneration.

APA, Harvard, Vancouver, ISO, and other styles

16

&NA;. "Muller glia induce retinal progenitor cells to differentiate into retinal ganglion cells: Retraction." NeuroReport 21, no. 13 (September 2010): 907. http://dx.doi.org/10.1097/wnr.0b013e32833e9b7a.

Full text

APA, Harvard, Vancouver, ISO, and other styles

17

Russell, William, Robert D. Hollifield, Adam J. West, Miles R. Stanford, and G. Astrid Limb. "Expression of haematopoietic cell markers by retinal pigment epithelial cells and Muller cells." Biochemical Society Transactions 25, no. 2 (May 1, 1997): 251S. http://dx.doi.org/10.1042/bst025251s.

Full text

APA, Harvard, Vancouver, ISO, and other styles

18

Claudepierre, T., C. Dalloz, D. Mornet, K. Matsumura, J. Sahel, and A. Rendon. "Characterization of the intermolecular associations of the dystrophin-associated glycoprotein complex in retinal Muller glial cells." Journal of Cell Science 113, no. 19 (October 1, 2000): 3409–17. http://dx.doi.org/10.1242/jcs.113.19.3409.

Full text

Abstract:

The abnormal retinal neurotransmission observed in Duchenne muscular dystrophy patients has been attributed to altered expression of C-terminal products of the dystrophin gene in this tissue. Muller glial cells from rat retina express dystrophin protein Dp71, utrophin and the members of the dystrophin-associated glycoprotein complex (DGC), namely beta-dystroglycan, delta- and gamma-sarcoglycans and alpha1-syntrophin. The DGC could function in muscle as a link between the cystoskeleton and the extracellular matrix, as well as a signaling complex. However, other than in muscle the composition and intermolecular associations among members of the DGC are still unknown. Here we demonstrate that Dp71 and/or utrophin from rat retinal Muller glial cells form a complex with beta-dystroglycan, delta-sarcoglycan and alpha1-syntrophin. We also show that beta-dystroglycan is associated with alpha-dystrobrevin-1 and PSD-93 and that anti-PSD antibodies coimmunoprecipitated alpha-syntrophin with PSD-93. By overlay experiments we also found that Dp71and/or utrophin and alpha-dystroglycan from Muller cells could bind to actin and laminin, respectively. These results indicate that the DGC could have both structural and signaling functions in retina. On the basis of our accumulated evidence, we propose a hypothetical model for the molecular organization of the dystrophin-associated glycoprotein complex in retinal Muller glial cells, which would be helpful for understanding its function in the central nervous system.

APA, Harvard, Vancouver, ISO, and other styles

19

Franze, K., J. Grosche, S. N. Skatchkov, S. Schinkinger, C. Foja, D. Schild, O. Uckermann, K. Travis, A. Reichenbach, and J. Guck. "Muller cells are living optical fibers in the vertebrate retina." Proceedings of the National Academy of Sciences 104, no. 20 (May 7, 2007): 8287–92. http://dx.doi.org/10.1073/pnas.0611180104.

Full text

APA, Harvard, Vancouver, ISO, and other styles

20

Matteucci, Andrea, Lucia Gaddini, Gianfranco Macchia, Monica Varano, Tamara C. Petrucci, Pompeo Macioce, Fiorella Malchiodi-Albedi, and Marina Ceccarini. "Developmental expression of dysbindin in Muller cells of rat retina." Experimental Eye Research 116 (November 2013): 1–8. http://dx.doi.org/10.1016/j.exer.2013.08.006.

Full text

APA, Harvard, Vancouver, ISO, and other styles

21

López-Colomé, A. M., and M. Romo-de-Vivar. "Maturation-induced changes in L-aspartate receptors from muller cells." Neurochemistry International 21 (January 1992): B19. http://dx.doi.org/10.1016/0197-0186(92)92010-2.

Full text

APA, Harvard, Vancouver, ISO, and other styles

22

Xu, Guo-Tong. "EPO attenuates inflammatory cytokines by Muller cells in diabetic retinopathy." Frontiers in Bioscience E3, no. 1 (2011): 201–11. http://dx.doi.org/10.2741/e234.

Full text

APA, Harvard, Vancouver, ISO, and other styles

23

Hatakeyama, J., K. Tomita, T. Inoue, and R. Kageyama. "Roles of homeobox and bHLH genes in specification of a retinal cell type." Development 128, no. 8 (April 15, 2001): 1313–22. http://dx.doi.org/10.1242/dev.128.8.1313.

Full text

Abstract:

Previous analysis of mutant mice has revealed that the bHLH genes Mash1 and Math3, and the homeobox gene Chx10 are essential for generation of bipolar cells, the interneurons present in the inner nuclear layer of the retina. Thus, a combination of the bHLH and homeobox genes should be important for bipolar cell genesis, but the exact functions of each gene remain largely unknown. We have found that in Mash1-Math3 double-mutant retina, which exhibits a complete loss of bipolar cells, Chx10 expression did not disappear but remained in Muller glial cells, suggesting that Chx10 expression per se is compatible with gliogenesis. In agreement with this, misexpression of Chx10 alone with retrovirus in the retinal explant cultures induced generation of the inner nuclear layer cells, including Muller glia, but few of them were mature bipolar cells. Misexpression of Mash1 or Math3 alone did not promote bipolar cell genesis either, but inhibited Muller gliogenesis. In contrast, misexpression of Mash1 or Math3 together with Chx10 increased the population of mature bipolar cells and decreased that of Muller glia. Thus, the homeobox gene provides the inner nuclear layer-specific identity while the bHLH genes regulate the neuronal versus glial fate determination, and these two classes of genes together specify the bipolar cell fate. Moreover, Mash1 and Math3 promoted the bipolar cell fate, but not the other inner nuclear layer-specific neuronal subtypes in the presence of Chx10, raising the possibility that the bHLH genes may be involved in neuronal subtype specification, in addition to simply making the neuronal versus glial fate choice.

APA, Harvard, Vancouver, ISO, and other styles

24

Hojo, M., T. Ohtsuka, N. Hashimoto, G. Gradwohl, F. Guillemot, and R. Kageyama. "Glial cell fate specification modulated by the bHLH gene Hes5 in mouse retina." Development 127, no. 12 (June 15, 2000): 2515–22. http://dx.doi.org/10.1242/dev.127.12.2515.

Full text

Abstract:

Neurons and glial cells differentiate from common precursors. Whereas the gene glial cells missing (gcm) determines the glial fate in Drosophila, current data about the expression patterns suggest that, in mammals, gcm homologues are unlikely to regulate gliogenesis. Here, we found that, in mouse retina, the bHLH gene Hes5 was specifically expressed by differentiating Muller glial cells and that misexpression of Hes5 with recombinant retrovirus significantly increased the population of glial cells at the expense of neurons. Conversely, Hes5-deficient retina showed 30–40% decrease of Muller glial cell number without affecting cell survival. These results indicate that Hes5 modulates glial cell fate specification in mouse retina.

APA, Harvard, Vancouver, ISO, and other styles

25

Neroev, Vladimir Vladimirovich, Marina Vladimirovna Zueva, Pavel Alexandrovich Bichkov, Irina Vladimirovna Tsapenko, Ol'ga Ivanovna Sarygina, Pavel Andreevich Ilyukhin, and Natalija Alekseevna Semenova. "ERG assessment of the functional activity of the retina in following the surgical closure of idiopathic macular holes." Ophthalmology journal 6, no. 4 (December 15, 2013): 21–27. http://dx.doi.org/10.17816/ov2013421-27.

Full text

Abstract:

Twenty patients with stage 3 or 4 idiopathic macular holes were evaluated with standard cone ERG, photopic flicker ERG at 8,3-30-Hz, to study the function of cone photoreceptors, bipolar cells, and Muller cells after the surgical correction of their macular hole. The correlations between the ERG parameters and the microperimetry and optical coherent tomography data were evaluated. IMH has been associated with the general reduction in the function of the cone photoreceptors and bipolar cells and with the sharp increase in the activity of Muller cells and their functional relationships with some bipolar cells. The nature of the changes in photopic standard and flicker ERGs demonstrated the dependence of the central retinal function on the dynamics of cone bipolar cells’ recovery. In the early period after the surgery of IMH, a sharp reduction in the flicker ERG at 24 Hz was found, to be followed by a progressive increase, which indicated a pronounced reduction in the bipolar cell function with the formation of the IMH and its substantial decrease after the vitreoretinal surgery. A significant increase in the glial index for flicker ERG at 24 Hz may be associated with a compensatory overreaction of retinal Muller cells in the preoperative and in the recovery period after the closure of IMH.

APA, Harvard, Vancouver, ISO, and other styles

26

KLJAVIN, I., and T. REH. "Muller cells are a prefered substrate for the neurite extension by rod photoreceptor cells." Cell Biology International Reports 14 (September 1990): 231. http://dx.doi.org/10.1016/0309-1651(90)91025-y.

Full text

APA, Harvard, Vancouver, ISO, and other styles

27

Luo, Yan. "EPO reduces reactive gliosis and stimulates neurotrophin expression in Muller cells." Frontiers in Bioscience E3, no. 1 (2009): 1541. http://dx.doi.org/10.2741/355.

Full text

APA, Harvard, Vancouver, ISO, and other styles

28

Newman, EA. "Sodium-bicarbonate cotransport in retinal Muller (glial) cells of the salamander." Journal of Neuroscience 11, no. 12 (December 1, 1991): 3972–83. http://dx.doi.org/10.1523/jneurosci.11-12-03972.1991.

Full text

APA, Harvard, Vancouver, ISO, and other styles

29

Zhong, Yong. "EPO reduces reactive gliosis and stimulates neurotrophin expression in Muller cells." Frontiers in Bioscience E3, no. 4 (2011): 1541–55. http://dx.doi.org/10.2741/e355.

Full text

APA, Harvard, Vancouver, ISO, and other styles

30

Kubrusly, Regina Celia Cussa, Maria Cristina Caldas da Cunha, Ricardo Augusto de Melo Reis, Heline Soares, Ana Lúcia Marques Ventura, Eleonora Kurtenbach, Maria Cristina Fialho de Mello, and Fernando Garcia de Mello. "Expression of functional receptors and transmitter enzymes in cultured Muller cells." Brain Research 1038, no. 2 (March 2005): 141–49. http://dx.doi.org/10.1016/j.brainres.2005.01.031.

Full text

APA, Harvard, Vancouver, ISO, and other styles

31

Currie, S. N., and R. C. Carlsen. "Functional significance and neural basis of larval lamprey startle behaviour." Journal of Experimental Biology 133, no. 1 (November 1, 1987): 121–35. http://dx.doi.org/10.1242/jeb.133.1.121.

Full text

Abstract:

1. The vibration-evoked startle response mediates rapid withdrawal in burrowed larval lampreys (ammocoetes). Ammocoetes withdraw in response to vibration by contracting pre-existing lateral bends in the trunk and tail, thus pulling their heads deeper into the burrow. 2. The motor effects of an ammocoete startle response are dependent on pre-existing posture. Areas of lateral body curvature contract more and exhibit larger electromyogram (EMG) amplitudes on their inner sides than on their outer sides. 3. Both of the anterior Mth and posterior Mth' (Mauthner) cells and both of the B1 and B2 (bulbar) Muller cells fired action potentials in response to vibration of the otic capsules. Both B3 and B4 Muller cells were inhibited by vibration, while M (mesencephalic) and I1 (isthmic) Muller cells were inhibited by ipsilateral vibration and excited by contralateral vibration. 4. Simultaneous action potentials in both of the anterior Mth cells were appropriate and sufficient for initiating the startle response EMG in a semi-intact preparation. 5. This study demonstrates a Mauthner-initiated startle response which activates musculature on both sides of the body to produce a rapid withdrawal movement and is thus adapted to the eel-like form and burrowed lifestyle of larval lampreys.

APA, Harvard, Vancouver, ISO, and other styles

32

Kljavin, IJ, and TA Reh. "Muller cells are a preferred substrate for in vitro neurite extension by rod photoreceptor cells." Journal of Neuroscience 11, no. 10 (October 1, 1991): 2985–94. http://dx.doi.org/10.1523/jneurosci.11-10-02985.1991.

Full text

APA, Harvard, Vancouver, ISO, and other styles

33

SEKI, T. "PACAP Stimulates the Release of Interleukin-6 in Cultured Rat Muller Cells." Annals of the New York Academy of Sciences 1070, no. 1 (July 1, 2006): 535–39. http://dx.doi.org/10.1196/annals.1317.043.

Full text

APA, Harvard, Vancouver, ISO, and other styles

34

Moscona, A. A., L. Fox, J. Smith, and L. Degenstein. "Antiserum to lens antigens immunostains Muller glia cells in the neural retina." Proceedings of the National Academy of Sciences 82, no. 16 (August 1, 1985): 5570–73. http://dx.doi.org/10.1073/pnas.82.16.5570.

Full text

APA, Harvard, Vancouver, ISO, and other styles

35

Mascarelli, F., J. Tassin, and Y. Courtois. "Effect of FGFs on Adult Bovine Muller Cells: Proliferation, Binding and Internalization." Growth Factors 4, no. 2 (January 1991): 81–95. http://dx.doi.org/10.3109/08977199109000260.

Full text

APA, Harvard, Vancouver, ISO, and other styles

36

Karwoski, C., H. Lu, and E. Newman. "Spatial buffering of light-evoked potassium increases by retinal Muller (glial) cells." Science 244, no. 4904 (May 5, 1989): 578–80. http://dx.doi.org/10.1126/science.2785716.

Full text

APA, Harvard, Vancouver, ISO, and other styles

37

Harstad, H. Kr, and A. Ringvold. "Scanning and transmission electron microscopy of Muller cells isolated from rabbit retina." Graefe's Archive for Clinical and Experimental Ophthalmology 223, no. 1 (March 1985): 29–34. http://dx.doi.org/10.1007/bf02150570.

Full text

APA, Harvard, Vancouver, ISO, and other styles

38

Xu, X., and C. J. Karwoski. "Current source density analysis of retinal field potentials. II. Pharmacological analysis of the b-wave and M-wave." Journal of Neurophysiology 72, no. 1 (July 1, 1994): 96–105. http://dx.doi.org/10.1152/jn.1994.72.1.96.

Full text

Abstract:

1. The actions of two pharmacological agents, barium ions (Ba2+) and picrotoxin (PTX), were examined on components of the electroretinogram (ERG) in frog retina. Depth profiles of light-evoked field potentials were recorded, and current source densities (CSDs) were computed from these. 2. Ba2+ abolished the M-wave, slow PIII, and the c-wave, but only decreased b-wave amplitude down to approximately 65% of control amplitude. 3. Ba2+ abolished a slow current sink in the inner plexiform layer (IPL) and the source at the inner limiting membrane (ILM). This IPL sink/ILM source appears to generate the M-wave. 4. Ba2+ decreased the current sink at the outer plexiform layer (OPL) to approximately 70% of control amplitude, and it increased an IPL source. This Ba(2+)-resistant OPL sink/IPL source appears to generate a significant portion of the b-wave. The Ba(2+)-sensitive portion of the b-wave might be generated by Muller cells. 5. PTX enhanced retinal field potentials, particularly the M-wave in the proximal retina. This enhanced M-wave was shown to originate from an enhanced IPL sink/ILM source. 6. Our results suggest that the M-wave originates from Muller cells, through the spatial buffering of the light-evoked increase in [K+]o of the proximal retina. A portion of the b-wave may also originate from Muller cells, but a stronger direct contribution from depolarizing bipolar cells is suggested.

APA, Harvard, Vancouver, ISO, and other styles

39

Szczesny, Piotr J., and Don Claugher. "High-Resolution SEM and Freeze Fracture Studies of Human Retina." Proceedings, annual meeting, Electron Microscopy Society of America 48, no. 3 (August 12, 1990): 146–47. http://dx.doi.org/10.1017/s0424820100158273.

Full text

Abstract:

The fine structure of human retinae were investigated with use of TEM and High Resolution SEM combined with freeze fracture and osmium maceration technique paying partic ularattention to the morphology of photo receptor cells. Gluteraldehyde fixed retin al samples from 8 individuals of different age groups, were examined. Fixation time varied from 0 to 3 hours post mortem.Results showed good corelation between the TEM and HRSEM Fig 1-2. HRSEM enabled a detailed study of a several plasma membrane domains of rods and cones. It was possible to examine the inner limiting membrane formed by podocytes of Muller cells Fig 3, and also the cell junctions formed by rods, cones and Muller cells at the level of the outerlimiting membrane, Fig 4. HRSEM demonstrated particularly well the area of the photorecept or connecting cilium and the proximal portion of the outer segment in photoreceptors.

APA, Harvard, Vancouver, ISO, and other styles

40

Kim, Minho, Soonil Kwon, Sohee Jeon, Byung Ju Jung, and Kyu Seop Kim. "Sphingosine-1-phosphate expression in human epiretinal membranes." PLOS ONE 17, no. 8 (August 31, 2022): e0273674. http://dx.doi.org/10.1371/journal.pone.0273674.

Full text

Abstract:

The abnormal posterior vitreous detachment (PVD) is speculated as an important mechanism of the development of the epiretinal membrane (ERM). However, there is only limited information about the molecular mechanism. Sphingosine-1-phosphate (S1P) is a mediator of the mechanosensitive response in several cell types that may have a role in the pathogenesis of ERM during abnormal PVD. Therefore, we evaluated the expression of S1P in the human ERM and the role of S1P in cultured human Muller glial cells. Among 24 ERM specimens, seven specimens (29.2%) exhibited S1P expression. Patients with secondary ERM or ellipsoid zone defects, which suggest abnormal PVD presented a significantly higher S1P+ cell density (secondary ERM: 128.20 ± 135.61 and 9.68 ± 36.01 cells, p = 0.002; EZ defects: 87.56 ± 117.79 vs 2.80 ± 8.85, p = 0.036). The addition of S1P increased the migrative ability and expression of N-cadherin and α-SMA in human Muller glial cells, suggesting S1P is a potential causative molecule for the development of ERM during abnormal PVD.

APA, Harvard, Vancouver, ISO, and other styles

41

LOPEZ-COLOME, ANA MARIA, and ANA GADEA. "Regulation of Glycine Transport in Cultured Muller Cells by Ca2+/Calmodulin-Dependent Enzymes." Annals of the New York Academy of Sciences 868, no. 1 MOLECULAR AND (April 1999): 685–88. http://dx.doi.org/10.1111/j.1749-6632.1999.tb11346.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

42

Poitry-Yamate, CL, S. Poitry, and M. Tsacopoulos. "Lactate released by Muller glial cells is metabolized by photoreceptors from mammalian retina." Journal of Neuroscience 15, no. 7 (July 1, 1995): 5179–91. http://dx.doi.org/10.1523/jneurosci.15-07-05179.1995.

Full text

APA, Harvard, Vancouver, ISO, and other styles

43

Liepe, BA, C. Stone, J. Koistinaho, and DR Copenhagen. "Nitric oxide synthase in Muller cells and neurons of salamander and fish retina." Journal of Neuroscience 14, no. 12 (December 1, 1994): 7641–54. http://dx.doi.org/10.1523/jneurosci.14-12-07641.1994.

Full text

APA, Harvard, Vancouver, ISO, and other styles

44

Archer, Simon N., Poonam Ahuja, Romeo Caffe, Catherine Mikol, Russell G. Foster, Theo van Veen, and Malcolm von Schantz. "Absence of phosphoglucose isomerase-1 in retinal photoreceptor, pigment epithelium and Muller cells." European Journal of Neuroscience 19, no. 11 (June 2004): 2923–30. http://dx.doi.org/10.1111/j.0953-816x.2004.03417.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

45

Costa de Andrade, Gabriel, Christian Wertheimer, Kirsten Eibl, Armin Wolf, Anselm Kampik, Eduardo Buchele Rodrigues, Michel Eid Farah, and Christos Haritoglou. "Viability of Primary Human Pigment Epithelium Cells and Muller-Glia Cells after Intravitreal Ziv-Aflibercept and Aflibercept." Ophthalmologica 236, no. 4 (2016): 223–27. http://dx.doi.org/10.1159/000452677.

Full text

APA, Harvard, Vancouver, ISO, and other styles

46

Ivkovic, Sanja, Irena Jovanovic-Macura, Tijana Antonijevic, Selma Kanazir, and Domingos Henrique. "Different levels of epidermal growth factor signaling modifies the differentiation of specific cell types in mouse postnatal retina." Archives of Biological Sciences 71, no. 4 (2019): 711–19. http://dx.doi.org/10.2298/abs190617054i.

Full text

Abstract:

Epidermal growth factor (EGF) signaling has been implicated in the regulation of the differentiation and proliferation of retinal progenitors. We assessed how different levels of EGF signaling, achieved either by increasing receptor expression or via addition of the exogenous ligand, or an increase in both, can affect the differentiation of progenitors in the first week of postnatal retinal development in the model system of retinal explants (REs). Proliferating progenitor cells in REs were infected with either the control CLV3/ESR-related peptide family (CLE)-green fluorescent protein (GFP)- or with EGF receptor (EGFR)-GFP-expressing retrovirus, and grown in the control medium or in the presence of exogenous EGF (10 ng/mL). The differentiation of infected cells into Muller glia (Sox9+), rod photoreceptors (rhodopsin+) and horizontal cells (calbindin+) was analyzed. In all the examined conditions, infected cells differentiated into Muller glia and rod photoreceptors that normally develop postnatally. Horizontal cells finished their development during the embryonic stages and progenitors infected with control-GFP virus did not differentiate into GFP+/calbindin- in either control or EGFsupplemented medium, however, cells infected with EGFR-GFP differentiated into horizontal cells (GFP+/calbindin+) in both culture conditions. These results imply that altering the levels of EGFR and/or the amount of the EGF ligand can overcome progenitor competence restriction.

APA, Harvard, Vancouver, ISO, and other styles

47

Di Polo, A., L. J. Aigner, R. J. Dunn, G. M. Bray, and A. J. Aguayo. "Prolonged delivery of brain-derived neurotrophic factor by adenovirus-infected Muller cells temporarily rescues injured retinal ganglion cells." Proceedings of the National Academy of Sciences 95, no. 7 (March 31, 1998): 3978–83. http://dx.doi.org/10.1073/pnas.95.7.3978.

Full text

APA, Harvard, Vancouver, ISO, and other styles

48

Sauter, Monica M., and Curtis R. Brandt. "Knockdown of TRIM5α or TRIM11 increases lentiviral vector transduction efficiency of human Muller cells." Experimental Eye Research 204 (March 2021): 108436. http://dx.doi.org/10.1016/j.exer.2021.108436.

Full text

APA, Harvard, Vancouver, ISO, and other styles

49

ZUEVA, M., V. NEROEV, I. TSAPENKO, P. BYCHKOV, and O. SARYGINA. "Functional activity of retinal neurons and Muller cells in idiopathic full-thickness macular holes." Acta Ophthalmologica 90 (August 6, 2012): 0. http://dx.doi.org/10.1111/j.1755-3768.2012.f068.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

50

Muniz, Alberto, Elia T. Villazana-Espinoza, Bridget Thackeray, and Andrew T. C. Tsin. "11-cis-Acyl-CoA:RetinolO-Acyltransferase Activity in the Primary Culture of Chicken Muller Cells†." Biochemistry 45, no. 40 (October 2006): 12265–73. http://dx.doi.org/10.1021/bi060928p.

Full text

APA, Harvard, Vancouver, ISO, and other styles

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!