Relevant bibliographies by topics / Mucosal immune system

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Mucosal immune system'

Author: Grafiati
Published: 4 June 2021
Last updated: 26 January 2023

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Journal articles on the topic "Mucosal immune system"

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Kim, Dong-Young, and Ji-Eun Lee. "Mucosal Immune System." Journal of Clinical Otolaryngology Head and Neck Surgery 21, no. 1 (May 2010): 3–12. http://dx.doi.org/10.35420/jcohns.2010.21.1.3.

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Gesualdo, Loreto, Vincenzo Di Leo, and Rosanna Coppo. "The mucosal immune system and IgA nephropathy." Seminars in Immunopathology 43, no. 5 (October 2021): 657–68. http://dx.doi.org/10.1007/s00281-021-00871-y.

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Abstract The precise pathogenesis of immunoglobulin A nephropathy (IgAN) is still not clearly established but emerging evidence confirms a pivotal role for mucosal immunity. This review focuses on the key role of mucosa-associated lymphoid tissue (MALT) in promoting the onset of the disease, underlying the relationship among microbiota, genetic factors, food antigen, infections, and mucosal immune response. Finally, we evaluate potential therapies targeting microbes and mucosa hyperresponsiveness in IgAN patients.
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Brown, T. A. "Immunity at Mucosal Surfaces." Advances in Dental Research 10, no. 1 (April 1996): 62–65. http://dx.doi.org/10.1177/08959374960100011201.

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The mucosae form a barrier between our bodies and a hostile external environment. Diseases and extrinsic factors which impair mucosal function may lead to serious consequences. The mucosal immune system is the primary mediator of specific immunity at mucosal surfaces. As such, it is responsible for maintaining homeostasis and for defense against both overt and opportunistic pathogens. For this reason, it is also the target of many new vaccine strategies for the induction of mucosal immunity. This brief review will examine the mucosal immune system, its role in maintaining the integrity of the mucosa, and some of the strategies aimed at enhancing specific immunity.
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Krejci, J., K. Nechvatalova, M. Blahutkova, and M. Faldyna. "The respiratory tract in pigs and its immune system: a review." Veterinární Medicína 58, No. 4 (May 7, 2013): 206–20. http://dx.doi.org/10.17221/6759-vetmed.

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The growing amount of information regarding mucosal immunology in animals resulted from a need to better understand the pathogenesis of diseases entering the body through mucosa surfaces, including the respiratory tract. The second reason for such studies is associated with a search for alternative ways of vaccine application, including delivery to the mucosa of the respiratory tract. This review provides a structural and functional description of the immune system of the pig respiratory tract.  
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MacDonald, Thomas T. "The mucosal immune system." Parasite Immunology 25, no. 5 (May 2003): 235–46. http://dx.doi.org/10.1046/j.1365-3024.2003.00632.x.

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Jecker, Peter, Andrew S. McWilliam, Wolf J. Mann, and Patrick G. Holt. "Dendritic Cell Influx Differs between the Subglottic and Glottic Mucosae during Acute Laryngotracheitis Induced by a Broad Spectrum of Stimuli." Annals of Otology, Rhinology & Laryngology 111, no. 7 (July 2002): 567–72. http://dx.doi.org/10.1177/000348940211100701.

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Clinically, the subglottic and glottic mucosae may react differently, eg, during acute laryngotracheitis. In healthy rats, we showed previously that the composition of the mucosal immune system of the larynx also differs between these areas. Neutrophils, lymphocytes, and dendritic cells (DCs) are part of this mucosal immune system. In particular, DCs occupy a key function. They migrate into inflamed mucosae during the early phase of the immune response, which is normally characterized by an influx of neutrophils. Thus, they help to overcome the time lag between the innate and the adaptive immune responses. In the present study, the influx of DCs, neutrophils, and T lymphocytes into the subglottic and glottic mucosae of rats was examined at different time points after challenge with a broad spectrum of stimuli such as dead Moraxella catarrhalis, viable Bordetella pertussis, viable Sendai virus, and the soluble protein ovalbumin. The number of DCs increased rapidly after the application of the antigens. This increase was as rapid as the increase in neutrophils. Depending on the kind of antigen, their number in the mucosa increased up to 1,000 cells per 0.1 mm2 (Sendai virus). The comparison of different mucosal areas shows that an overwhelming number of immunocompetent cells entered the subglottic mucosa, whereas only a few cells migrated into the adjacent glottic mucosa. In conclusion, after inhalation of different kinds of antigens, the subset of immunocompetent cells investigated in this study entered the laryngeal mucosa in high numbers. The number of DCs entering the laryngeal mucosa was higher than the numbers of the other immune cells investigated. This finding underlines their function as first-line sentinels of the mucosal immune system of the larynx. The observation that the number of cells entering the laryngeal mucosa is location-dependent indicates the ability of adjacent laryngeal regions to react differently. This is similar to the clinical observation of a selective subglottic reaction during acute laryngotracheitis.
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Lamm, Michael E. "The IgA Mucosal Immune System." American Journal of Kidney Diseases 12, no. 5 (November 1988): 384–87. http://dx.doi.org/10.1016/s0272-6386(88)80030-1.

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Chase, Christopher, and Radhey S. Kaushik. "Mucosal Immune System of Cattle." Veterinary Clinics of North America: Food Animal Practice 35, no. 3 (November 2019): 431–51. http://dx.doi.org/10.1016/j.cvfa.2019.08.006.

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James, Stephen P. "The Gastrointestinal Mucosal Immune System." Digestive Diseases 11, no. 3 (1993): 146–56. http://dx.doi.org/10.1159/000171407.

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McGhee, Jerry R., and Kohtaro Fujihashi. "Inside the Mucosal Immune System." PLoS Biology 10, no. 9 (September 25, 2012): e1001397. http://dx.doi.org/10.1371/journal.pbio.1001397.

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Dissertations / Theses on the topic "Mucosal immune system"

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Etling, Michele R. "THE AGING MUCOSAL IMMUNE SYSTEM IN THE INTERLEUKIN-10-DEFICIENT MOUSE." Case Western Reserve University School of Graduate Studies / OhioLINK, 2007. http://rave.ohiolink.edu/etdc/view?acc_num=case1184295867.

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Ewing, Patricia A., and n/a. "Developmental profiles of mucosal immunity in pre-school children." University of Canberra. Human & Biomedical Sciences, 2000. http://erl.canberra.edu.au./public/adt-AUC20060707.154930.

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Previous studies of the ontogeny of the mucosal immune system have shown a significant increase in salivary Immunoglobulin A levels occurring at about five years of age. This study has monitored a group of 3 and 4 year old children during one year of attendance at Pre-School to examine whether such an increase could be linked to increased antigenic exposure associated with moving into a school like environment. Saliva samples were collected at regular intervals and analysed for immunoglobulin and total protein levels. Daily health records were maintained for each child, and a detailed social and medical history was collected for each child at the beginning of the study. The elevated mucosal immune response observed in previous studies involving children in day care centres and attending school was not seen in this study. No significant difference was observed between children who had previously attended Pre-School or child care centres and those who were attending for the first time. However, a marked seasonal increase in mean salivary IgA during the winter months was observed and this increase correlated with an increase in respiratory infections. Hence, in studies of developmental aspects of mucosal immune response it is essential that modifiers such as season and infection be recorded.
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Blazek, Alisa D. "A Simulated Altitude Device can Improve Endurance Performance without Mucosal Immune System Compromise." The Ohio State University, 2010. http://rave.ohiolink.edu/etdc/view?acc_num=osu1267567607.

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Ucan, Uckun Sait. "T cells and cytokines in the lamina propria of the pig." Thesis, University of Bristol, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.389234.

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Christoforidou, Zoe. "The role of gut microbiota in driving the development of the mucosal immune system." Thesis, University of Bristol, 2014. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.689676.

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It is now accepted that gut microbiota are crucial for the development of the mucosal immune system. It has been suggested that differences in early life microbial colonisation may be associated with variable predisposition to allergic, auto immune, and inflammatory diseases. The exact underlying mechanisms are difficult to study in human infants, however, the similarities in physiology and nutritional requirements between pigs and humans suggest that piglets can be good models to elucidate the pathways involved. The aim of this PhD project was to investigate how differences in microbiota, early on in life, could affect immune regulation in telms of regulatory T cells (Tregs), a cell type fundamental for immune homeostasis. Manipulation of gut microbiota was attempted using different housing conditions [specific pathogen-free (SPF) facility and farm], birth environment (indoor and outdoor fmm) as well as with administration of antibiotics and specific microbiota inocula (with simple and complex composition). Furthermore, the effect of nutritional interventions, (inulin, starch and medium-chain triglycerides) with the potential to manipulate gut microbiota, was also studied. The effect of these manipulations on small intestinal Tregs was examined using fluorescent immunohistology. Furthermore, activation-induced Foxp3 expressions on gut and blood CD4 T cells was also investigated using flow cytometry. It was observed that indoor-born piglets were more susceptible to a reduction in Tregs when transfened to an SPF facility than outdoor bom piglets, but treatment with antibiotics reduced gut Tregs of outdoor-bom piglets at the level of those born indoor. On the other hand, colonisation of new-bom piglets with a complex microbiota inoculum reduced gut Tregs in comparison to the simple microbiota inoculum. However, none of the nutritional interventions had a significant effect on Tregs. Furthermore, no activation induced expression ofFoxp3 was observed in either gut or blood CD4 T cells of 5-month old piglets. The results of this Thesis suggest that both environment and direct manipulation of gut microbiota can affect levels of small intestinal Tregs, whereas the effect of nutrition is less clear. A more detailed analysis of small-intestinal microbiota is necessary to confirm that these observations are a result of differences in the microbiome between the groups or whether other possible factors are also involved.
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Thompson, Fiona Marie. "Activation of the mucosal immune system and growth of the small intestine at weaning /." Title page, abstract and contents only, 1994. http://web4.library.adelaide.edu.au/theses/09PH/09pht4677.pdf.

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Harris, Cecilia. "Effect of environmental factors on the development of the mucosal immune system in the piglet." Thesis, University of Bristol, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.419675.

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O’Meara, Connor Patrick. "The development of an effective vaccine against Chlamydia : utilisation of a non-toxic mucosal adjuvant to generate a protective mucosal response." Thesis, Queensland University of Technology, 2012. https://eprints.qut.edu.au/61614/1/Connor_O%27Meara_Thesis.pdf.

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Chlamydia is responsible for a wide range of diseases with enormous global economic and health burden. As the majority of chlamydial infections are asymptomatic, a vaccine has greatest potential to reduce infection and disease prevalence. Protective immunity against Chlamydia requires the induction of a mucosal immune response, ideally, at the multiple sites in the body where an infection can be established. Mucosal immunity is most effectively stimulated by targeting vaccination to the epithelium, which is best accomplished by direct vaccine application to mucosal surfaces rather than by injection. The efficacy of needle-free vaccines however is reliant on a powerful adjuvant to overcome mucosal tolerance. As very few adjuvants have proven able to elicit mucosal immunity without harmful side effects, there is a need to develop non-toxic adjuvants or safer ways to administered pre-existing toxic adjuvants. In the present study we investigated the novel non-toxic mucosal adjuvant CTA1-DD. The immunogenicity of CTA1-DD was compared to our "gold-standard" mucosal adjuvant combination of cholera toxin (CT) and cytosine-phosphate-guanosine oligodeoxynucleotide (CpG-ODN). We also utilised different needle-free immunisation routes, intranasal (IN), sublingual (SL) and transcutaneous (TC), to stimulate the induction of immunity at multiple mucosal surfaces in the body where Chlamydia are known to infect. Moreover, administering each adjuvant by different routes may also limit the toxicity of the CT/CpG adjuvant, currently restricted from use in humans. Mice were immunised with either adjuvant together with the chlamydial major outer membrane protein (MOMP) to evaluate vaccine safety and quantify the induction of antigen-specific mucosal immune responses. The level of protection against infection and disease was also assessed in vaccinated animals following a live genital or respiratory tract infectious challenge. The non-toxic CTA1-DD was found to be safe and immunogenic when delivered via the IN route in mice, inducing a comparable mucosal response and level of protective immunity against chlamydial challenge to its toxic CT/CpG counterpart administered by the same route. The utilisation of different routes of immunisation strongly influenced the distribution of antigen-specific responses to distant mucosal surfaces and also abrogated the toxicity of CT/CpG. The CT/CpG-adjuvanted vaccine was safe when administered by the SL and TC routes and conferred partial immunity against infection and pathology in both challenge models. This protection was attributed to the induction of antigen-specific pro-inflammatory cellular responses in the lymph nodes regional to the site of infection and rather than in the spleen. Development of non-toxic adjuvants and effective ways to reduce the side effects of toxic adjuvants has profound implications for vaccine development, particularly against mucosal pathogens like Chlamydia. Interestingly, we also identified two contrasting vaccines in both infection models capable of preventing infection or pathology exclusively. This indicated that the development of pathology following an infection of vaccinated animals was independent of bacterial load and was instead the result of immunopathology, potentially driven by the adaptive immune response generated following immunisation. While both vaccines expressed high levels of interleukin (IL)-17 cytokines, the pathology protected group displayed significantly reduced expression of corresponding IL-17 receptors and hence an inhibition of signalling. This indicated that the balance of IL-17-mediated responses defines the degree of protection against infection and tissue damage generated following vaccination. This study has enabled us to better understand the immune basis of pathology and protection, necessary to design more effective vaccines.
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Law, Yuet Ching. "Specific Compartmentalization of IgA ASCs in Mouse Salivary Glands via Differential Expression of Chemokines and Chemokine Receptors." BYU ScholarsArchive, 2008. https://scholarsarchive.byu.edu/etd/1952.

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The mucosal system, which forms a barrier between internal organ systems and the external environment, is frequently exposed to pathogenic microorganisms. Immunoglobulin A (IgA) antibody secreting cells (ASCs) localize in the lamina propria, and produce IgA antibodies which help protect mucosal tissues. The concept of a common mucosal immune system in which IgA ASCs have the ability to populate any mucosal site has been proposed (1, 2). However, recent research has suggested that IgA ASCs primed in different mucosal sites might possess different sets of chemokine receptors, and therefore migrate specifically to particular mucosal locations (3). In this study, the specific compartmentalization of IgA ASCs in two mouse salivary glands: sublingual gland (SLG), and submandibular gland (SMG) was studied. It was observed that SLG had 12 times more IgA ASCs per gram of gland than that of SMG (p<0.01). This suggested that IgA ASCs migrated to the two salivary glands with different efficiencies. Since the migration of lymphocytes is mediated by interactions between tissue specific chemokines and chemokine receptors, I hypothesized that the specific compartmentalization of IgA ASCs in the SLG and SMG was mediated by the differential expression of IgA ASC attracting chemokines. Quantitative PCR was used and showed that SLG expressed high levels of CCL28 and its receptor CCR10, which correlated to the distribution of IgA ASCs in the two salivary glands. In agreement with QPCR data, reduced levels of IgA ASCs were found in the SLG of CCR10 deficient mice when compared to wild type (WT) mice. Adoptive transfer of CCR10 deficient mice with WT spleen cells reconstituted the WT phenotype. It was therefore concluded that the interaction between CCL28 and CCR10 play an important role in mediating the migration of IgA ASCs into SLG. These results suggested that the accumulation of IgA ASC to distinct salivary glands is a highly selective process. These data also suggested that homing within mucosal sites is not common but rather a highly regulated process with specific subsets of cells homing to different tissues within the mucosal immune system.
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Zhang, Min Fen. "The role of milk transforming growth factor-[beta](TGF-[beta]) in the development of the infant gut and gut mucosal immune system." Title page, contents and abstract only, 2000. http://web4.library.adelaide.edu.au/theses/09PH/09phz51.pdf.

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In title, [beta] is represented by the Greek letter. Copies of author's previously published articles inserted. Errata pages pasted onto back end-paper. Bibliography: leaves 104-137. Studies milk TGF-[beta] and its receptors in the post-natal gut using a rat model to investigate a link between milk TGF-[beta] and the development of the infant gut and gut mucosal immune system. Finds maternal milk may be a major source of TGF-[beta] to the immature gut and may react with receptors on the cells of the mucosal immune system along the gastro-intestinal tract, modulating infant mucosal immune responses in the transition to the post-natal enteral feeding.
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Books on the topic "Mucosal immune system"

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Å, Hanson Lars, and Svanborg-Edén Catharina, eds. Mucosal immunobiology: Cellular-molecular interactions in the mucosal immune system. Basel: Karger, 1988.

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Mucosal immunity. San Diego, CA: Academic Press, 2010.

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Dayan, Nava, and Philip W. Wertz, eds. Innate Immune System of Skin and Oral Mucosa. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2011. http://dx.doi.org/10.1002/9781118025338.

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1951-, MacDonald Thomas T., ed. Ontogeny of the immune system of the gut. Boca Raton, Fla: CRC Press, 1990.

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Falk Symposium (133rd 2004 Berlin, Germany). Mechanisms of intestinal inflammation: Implications for therapeutic intervention in IBD : proceedings of Falk Symposium 133 (New Findings on Pathogenesis and Progress in Management of Inflammatory Bowel Diseases, Part I) held in Berlin, Germany, June 10-11, 2003. Dordrecht: Kluwer Academic Publishers, 2004.

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Derek, Chadwick, and Goode Jamie, eds. Inflammatory bowel disease: Crossroads of microbes, epithelium, and immune systems. Chichester, UK: J. Wiley, 2004.

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Innate immune system of skin and oral mucosa: Properties and impact in pharmaceutics, cosmetics, and personal care products. Hoboken, N.J: John Wiley & Sons, 2011.

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Hanson, Lars A. Nobel Symposium No 68, Mucosal Immunobiology: Cellular-Molecular Interactions in the Mucosal Immune System (Monographs in Allergy). S Karger Pub, 1988.

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Bourne, F. J. Mucosal Immune System: Proceedings of a Seminar in the EEC Programme of Coordination of Agricultural Research on Protection of the Young Animal Against Perinatal Diseases, Held at the University of Bristol, School of Veterinary Science, Langford, Nr. Bristol, United Kingdom on September 9-11 1980. Springer, 2012.

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Bourne, F. J. Mucosal Immune System: Proceedings of a Seminar in the Eec Programme of Coordination of Agricultural Research on Protection of the Young Animal Against Perinatal Diseases, Held at the University of Bristol, School of Veterinary Science, Langford, Nr. Bristol, United Kingdom on September 9–11 1980. Springer, 2011.

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Book chapters on the topic "Mucosal immune system"

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Georgiev, Vassil St. "Mucosal Immune System." In National Institute of Allergy and Infectious Diseases, NIH, 675–82. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-297-1_42.

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Agarwal, Shradha, and Lloyd Mayer. "The Mucosal Immune System." In Food Allergy, 1–15. Chichester, UK: John Wiley & Sons Ltd, 2014. http://dx.doi.org/10.1002/9781118744185.ch1.

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Roux, M. E., N. H. Slobodianik, P. Gauffin Cano, and G. Perdigón. "Mucosal Immune System and Malnutrition." In Gut Flora, Nutrition, Immunity and Health, 155–77. Oxford, UK: Blackwell Publishing Ltd, 2008. http://dx.doi.org/10.1002/9780470774595.ch7.

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Klimov, Vladimir V. "Skin and Mucosal Immune System." In From Basic to Clinical Immunology, 101–25. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-03323-1_2.

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Johansen, Finn-Eirik, Ranveig Braathen, Else Munthe, Hilde Schjerven, and Per Brandtzaeg. "Regulation of the Mucosal IgA System." In Mucosal Immune Defense: Immunoglobulin A, 111–43. Boston, MA: Springer US, 2007. http://dx.doi.org/10.1007/978-0-387-72232-0_5.

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Thrane, P. S., T. O. Rognum, and P. Brandtzaeg. "Ontogenesis of the human secretory immune system." In Advances in Mucosal Immunology, 455–58. Dordrecht: Springer Netherlands, 1990. http://dx.doi.org/10.1007/978-94-009-1848-1_130.

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Mestecky, Jiri. "Homeostasis of the Mucosal Immune System." In Advances in Experimental Medicine and Biology, 197–205. Boston, MA: Springer US, 2001. http://dx.doi.org/10.1007/978-1-4615-1371-1_26.

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Brandtzaeg, P. "Overview of the Mucosal Immune System." In Current Topics in Microbiology and Immunology, 13–25. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-74529-4_2.

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Taubman, M. A., T. D. Halazonetis, W. E. Stack, D. J. Smith, I. D. Mandel, and D. A. Sullivan. "Thyroid influence on the salivary secretory immune system." In Advances in Mucosal Immunology, 497–99. Dordrecht: Springer Netherlands, 1990. http://dx.doi.org/10.1007/978-94-009-1848-1_148.

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Sullivan, D. A., and L. E. Hann. "Androgen regulation of the ocular secretory immune system." In Advances in Mucosal Immunology, 250–54. Dordrecht: Springer Netherlands, 1990. http://dx.doi.org/10.1007/978-94-009-1848-1_70.

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Conference papers on the topic "Mucosal immune system"

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Polosukhin, Vasiliy V., Pierre P. Massion, Jae W. Lee, Scott H. Randell, and Timothy S. Blackwell. "TOBACCO SMOKE IMPAIRS THE BRONCHIAL MUCOSAL SECRETORY IgA IMMUNE SYSTEM THROUGH ALTERATION OF BRONCHIAL EPITHELIAL CELL DIFFERENTIATION." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a1424.

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Krishnakumar, D., and K. S. Jaganathan. "Development of nasal HPV vaccine formulations." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685403.

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Cervical cancer is the second most cancer in women worldwide with over 500000 new cases and 275000 deaths being registered every year. With nearly 73000 women dying every year, India now tops the world in cervical cancer deaths. India represents 26.4% of all women dying of cervical cancer globally. Cervical cancer estimated to be responsible for about 5% of human cancers worldwide. Currently available vaccines may not provide complete protection against all HPV types as the protection is primarily type specific. Furthermore, the available vaccines are delivered via intramuscular route and require three doses and require cold chain supply which increases the cost of vaccine. Therefore a single dose vaccine delivered via non-invasive route (nasal) that protects against multiple HPV types would be a cost effective and better alternative to the currently available HPV vaccines. The main objective of this study was to prepare HPV antigen loaded poly (lactic-co-glycolic acid) (PLGA) and Tri Methyl Chitosan (TMC) coated PLGA microparticles and compare their efficacy as nasal vaccine. The developed formulations were characterized for size, zeta potential, entrapment efficiency, mucin adsorption ability, in vitro and in vivo studies. PLGA microparticles demonstrated negative zeta potential whereas PLGA-TMC microparticles showed higher positive zeta potential. The protein loading efficiency was found as above 80%. Results indicated that PLGA-TMC microparticles demonstrated substantially higher mucin adsorption when compared to PLGA microparticles. HPV antigen encapsulated in PLGA-TMC particles elicited a significantly higher secretory (IgA) immune response compared to that encapsulated in PLGA particles. Present study demonstrates that PLGA-TMC microparticles with specific size range can be a better carrier adjuvant for nasal subunit vaccines. Surface modified PLGA microparticles proved great potential as a nasal delivery system for HPV infections where systemic and mucosal responses are necessary particularly in conditions after viral pathogens invade the host through the mucosal surface.
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Smith, Alana, Breia Reed, Joseph F. Pierre, Beverly Lyn-Cook, and Athena Starlard-Davenport. "Abstract B064: Investigation of the breast microbiome and mucosal immune system in African American and non-Hispanic White women with and without breast cancer: A pilot study." In Abstracts: Eleventh AACR Conference on The Science of Cancer Health Disparities in Racial/Ethnic Minorities and the Medically Underserved; November 2-5, 2018; New Orleans, LA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7755.disp18-b064.

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Boian, Ilie. "Impactul schimbărilor bruşte ale vremii din republica asupra stării de sănătate a populației." In Provocări şi tendinţe actuale în cercetarea componentelor naturale şi socio-economice ale ecosistemelor urbane şi rurale. Institute of Ecology and Geography, Republic of Moldova, 2020. http://dx.doi.org/10.53380/9789975891608.10.

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Along with the positive parts of the Moldovan climate - the warm period of the year, the mild winter, the abundance of light and heat - there are also some negative traits such as the high variability of the weather. The sudden changes in weather caused by the strong variation in the values of the main meteorological elements such as air temperature, precipitation, relative air humidity, nebulosity, and especially atmospheric pressure, adversely affect the health of a large number of people. When the body is virtually forced to suddenly pass from a temperature above 35°C to less than 17°C, it is instantly weakened. The immune system weakens and the body becomes vulnerable to viral infections. At the same time, the presence of nasal polyps, allergies, which affect the nasal mucosa and prolonged stress, increase the risk of developing viral infectionsThere are many people who accuse a state of acute fatigue, physical and mental asthenia, a state of continuous drowsiness and even depression, although they are properly resting.
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Boda, Z., J. Hársfalvi, K. Pecze, and K. Rak. "ACQUIRED HAEMOPHILIA DUE TO FACTOR VIII INHIBITOR WITH SEVERE HAEMORRHAGES IN A 46-YEAR-OLD WOMAN SUCCESSFULLY TREATED WITH CYCLOSPORIN A." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644845.

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A formerly healthy 46-year-old woman suffering from acquired haemophilia caused by factor VIII antibodies was admitted in an unconscious state following subarachnoid haemorrhage. Treatment with prothrombin complex concentrates (taken as a whole 100 000 U of PCC, home made and 10 OOOUofFEIBA, Immuno) , steroid (Prednison 50 mg/day) and cyclophosphamide (100 mg/day) was only partially successful: neurological state improved but the haemorrhagic tendency remained. Significant haematuria, and skin and mucosal bleeding characterized her clinical picture. In the meantime, signs of non-A non-B hepatitis were observed. After recovery treatment with Cyclosporin A (Sandimmun, Sandoz) was started (250 mg/day per os) together with small dose of Prednison (15 mg/day). No PCC was applied since that time and the partial thromboplastin times (PTT) became gradually shorter. Level of factor VIII inhibitor was 160 Bethesda unit prior and 9 unit after treatment, the duration of that was 60 days till now. Factor VIII coagulant activity (VIIIC) increased from value of less than 1 percent to 13.7 percent.Treatment of acquired haemophilia caused by factor VIII antibodies, particularly in cases with central nervous system bleeding, may be very difficult. History of our patient may indicate that patients resistent to substitution therapy, steroid and cytostatics may response well to Cyclosporin A. Therefore, its use is recommended.
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