Relevant bibliographies by topics / Morphogenesis

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Morphogenesis'

Author: Grafiati
Published: 4 June 2021
Last updated: 28 September 2024

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Journal articles on the topic "Morphogenesis"

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Harold, F. M. "From morphogenes to morphogenesis." Microbiology 141, no. 11 (November 1, 1995): 2765–78. http://dx.doi.org/10.1099/13500872-141-11-2765.

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Yang, Tsung-Lin, Ya-Chuan Hsiao, and Tai-Horng Young. "COMPARISON OF PLGA, PCL, AND CHITOSAN IN SALIVARY GLAND BRANCHING MORPHOGENESIS." Biomedical Engineering: Applications, Basis and Communications 20, no. 05 (October 2008): 287–96. http://dx.doi.org/10.4015/s1016237208000908.

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Branching morphogenesis is a fundamental morphogenetic process in generating glandular tissues. Although the mechanism of branching morphogenesis has been well-explored in the salivary gland development, its interaction with different biodegradable materials has never been investigated. For the purpose of salivary gland regeneration, recapitulation of morphogenetic processes on biodegradable materials might be requisite. Toward this aim, biodegradable biomaterials including poly-lactic-co-glycolic acid (PLGA), poly-epsilon-caprolactone (PCL), and chitosan were examined in the submandibular gland (SMG) culture systems to elucidate their possible impact on salivary morphogenesis. It was found that when SMG explants were cultured on PLGA and PCL, the explants failed to form well-developed branching phenotypes with limited cell migration (5.6 ± 8.8 μm; 10.0 ± 14.1 μm) and decreasing cell viability (56.9% ± 12.5%; 50.3% ± 8.1%). On the contrary, explants cultured on chitosan showed well-developed branches, which were superior in number to those on the control substrata, without any alteration of the morphogenetic phenotypes. Furthermore, the increased cell migration (267.8 ± 45.2 μm) and explants viability (146.8% ± 18.4%) along with the greater deposition of type III collagen, altogether account for better SMG morphogenesis on chitosan. According to the results, it was found that branching morphogenesis of SMG was affected by different biodegradable materials. Chitosan might be an appropriate biodegradable material for salivary morphogenesis, and has applicable potential in the regeneration of salivary tissue.
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Archer, Charles W., Paul Rooney, and Christopher P. Cottrill. "Cartilage morphogenesis in vitro." Development 90, no. 1 (December 1, 1985): 33–48. http://dx.doi.org/10.1242/dev.90.1.33.

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The morphogenetic capacity of prechondrogenic mesenchyme from two developmentally distinct sources was investigated in high density micromass cultures. We confirmed an earlier report (Weiss & Moscona, 1958) that scleral mesenchyme formed cartilage sheets whilst limb bud mesenchyme formed distinct cartilage nodules. It was thus suggested by these authors that this morphogenesis was tissue type specific. However, by varying cell density at inoculation (which controls cell configuration) and by varying the relative amount of prechondrogenic mesenchyme present in cultures we found that dramatic changes in morphogenesis could be brought about. Viewed in these terms we suggest that cartilage morphogenesis in vitro is dependent on cell configuration and the presence of non-chondrogenic cell types and hence is not necessarily a function of an intrinsic morphogenetic potential of the constituent cells.
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Haiping, Zhao, Chu Wenhui, Liu Zhen, and Li Chunyi. "Deer antler: a unique model for studying mammalian organ morphogenesis." Animal Production Science 56, no. 6 (2016): 946. http://dx.doi.org/10.1071/an14902.

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It is now widely accepted that organ morphogenesis in the lower animals, such as amphibians, is encoded by bioelectricity. Whether this finding applies to mammals is not known, a situation which is at least partially caused by the lack of suitable models. Deer antlers are complex mammalian organs, and their morphogenetic information resides in a primordium, the periosteum overlying the frontal crest of a prepubertal deer. The present paper reviews (1) the influence of morphogenetic information on antler formation and regeneration, and proposes that antlers are an appropriate organ for studying mammalian organ morphogenesis and (2) the storage, duplication and transferring pathways of morphogenetic information for deer antlers, and outlines a preliminary idea about how to understand the morphogenesis of mammalian organs through an involvement of bioelectricity. We believe that findings made using the deer antler model will benefit human health and wellbeing.
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Röper, Katja. "Microtubules enter centre stage for morphogenesis." Philosophical Transactions of the Royal Society B: Biological Sciences 375, no. 1809 (August 24, 2020): 20190557. http://dx.doi.org/10.1098/rstb.2019.0557.

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Cell shape changes are key to observable changes at the tissue level during morphogenesis and organ formation. The major driver of cell shape changes in turn is the actin cytoskeleton, both in the form of protrusive linear or branched dynamic networks and in the form of contractile actomyosin. Over the last 20 years, actomyosin has emerged as the major cytoskeletal system that deforms cells in epithelial sheets during morphogenesis. By contrast, the second major cytoskeletal system, microtubules, have so far mostly been assumed to serve ‘house-keeping' functions, such as directed transport or cell division, during morphogenetic events. Here, I will reflect on a subset of studies over the last 10 years that have clearly shown a major direct role for the microtubule cytoskeleton in epithelial morphogenesis, suggesting that our focus will need to be widened to give more attention and credit to this cytoskeletal system in playing an active morphogenetic role. This article is part of a discussion meeting issue ‘Contemporary morphogenesis'.
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Shi, Zhixin, David Christian, and Hei Leung. "Interactions Between Spore Morphogenetic Mutations Affect Cell Types, Sporulation, and Pathogenesis in Magnaporthe grisea." Molecular Plant-Microbe Interactions® 11, no. 3 (March 1998): 199–207. http://dx.doi.org/10.1094/mpmi.1998.11.3.199.

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We have previously defined four single-gene mutations, con1, con2, con4, and con7, that control various stages of spore morphogenesis in the rice blast fungus. To delineate the developmental pathway of spore morphogenesis, we investigated the interactions among these morphogenetic genes by generating strains with double mutations via transformation-mediated gene disruption. Plasmids containing portions of the inactivated CON4 and CON7 genes were introduced into strains harboring single mutation to produce double mutants. Interaction between con1 and con4 resulted in reduced vegetative growth and suppression of sporulation. Interaction between con1 and con7 suppressed spore production but not vegetative growth. The con2/con4 and con4/con7 double mutants produced hybrid spore types with characteristic features of both parental mutants. The con2/con7 mutant produced con2 type spores, indicating that con2 is epistatic to con7 in spore morphogenesis. The epistatic relationship, however, reversed when traits related to pathogenesis were considered. Double mutants harboring the con7 mutation could not form appressoria or colonize plant tissue, indicating that con7 is epistatic to con2 and con4 in appressorium formation and pathogenesis. Thus, morphogenetic genes interact at multiple levels leading to different epistatic relationships in the pathways of spore morphogenesis, appressorium formation, and pathogenesis.
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Goldgaber, Deborah. "Morphogenesis." Philosophy Today 63, no. 4 (2019): 999–1012. http://dx.doi.org/10.5840/philtoday2020124306.

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This article explores the ways new materialism centers the problem of morphogenesis—and de-centers language and culture—in philosophical accounts of corporeality. Attention to organic structures gives insight into the entanglement of nature and culture obscured by tendencies to think matter as lacking agential features. I suggest, in conclusion, that new materialism may operate with a notion of “entanglement” or “intra-activity” that is too productive. New materialisms may require a more pliable set of distinctions to capture the relations between morphogenetic forces.
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Blake, Joshua, and Norman D. Rosenblum. "Renal branching morphogenesis: Morphogenetic and signaling mechanisms." Seminars in Cell & Developmental Biology 36 (December 2014): 2–12. http://dx.doi.org/10.1016/j.semcdb.2014.07.011.

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Reddi, A. H. "Cartilage-derived morphogenetic proteins and cartilage morphogenesis." Microscopy Research and Technique 43, no. 2 (October 15, 1998): 131–36. http://dx.doi.org/10.1002/(sici)1097-0029(19981015)43:2<131::aid-jemt6>3.0.co;2-c.

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Jaslove, Jacob M., and Celeste M. Nelson. "Smooth muscle: a stiff sculptor of epithelial shapes." Philosophical Transactions of the Royal Society B: Biological Sciences 373, no. 1759 (September 24, 2018): 20170318. http://dx.doi.org/10.1098/rstb.2017.0318.

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Smooth muscle is increasingly recognized as a key mechanical sculptor of epithelia during embryonic development. Smooth muscle is a mesenchymal tissue that surrounds the epithelia of organs including the gut, blood vessels, lungs, bladder, ureter, uterus, oviduct and epididymis. Smooth muscle is stiffer than its adjacent epithelium and often serves its morphogenetic function by physically constraining the growth of a proliferating epithelial layer. This constraint leads to mechanical instabilities and epithelial morphogenesis through buckling. Smooth muscle stiffness alone, without smooth muscle cell shortening, seems to be sufficient to drive epithelial morphogenesis. Fully understanding the development of organs that use smooth muscle stiffness as a driver of morphogenesis requires investigating how smooth muscle develops, a key aspect of which is distinguishing smooth muscle-like tissues from one another in vivo and in culture. This necessitates a comprehensive appreciation of the genetic, anatomical and functional markers that are used to distinguish the different subtypes of smooth muscle (for example, vascular versus visceral) from similar cell types (including myofibroblasts and myoepithelial cells). Here, we review how smooth muscle acts as a mechanical driver of morphogenesis and discuss ways of identifying smooth muscle, which is critical for understanding these morphogenetic events. This article is part of the Theo Murphy meeting issue ‘Mechanics of Development’.
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Dissertations / Theses on the topic "Morphogenesis"

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Thomas, Nicole. "Bone morphogenetic proteins and hair and wool follicle morphogenesis." Title page, contents and abstract only, 2002. http://web4.library.adelaide.edu.au/theses/09PH/09pht4592.pdf.

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Bibliography: leaves 119-135. A thesis which describes a study to establish the relative roles that the bone morphogenetic proteins BMP-2 and BMP-4 play in initiating hair and derived wool follicles by first establishing their expression patterns by in situ hybridisation and then manipulating them in vitro.
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COSBITT, NICOLE. "MORPHOGENESIS: BUILDING AS A NATIVE PLANT." University of Cincinnati / OhioLINK, 2007. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1179327038.

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Cocho, Bermejo Ana. "EmDeplo morphogenesis." Doctoral thesis, Universitat Politècnica de Catalunya, 2012. http://hdl.handle.net/10803/97040.

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These thesis will argue about the importance of Dynamic Parametric Architecture versus Static Parametric Architecture. Describing the architectonical and the algorithmic context within which the Emergency Deployable System emerged, it will be discussed the importance of adaptability as the missing concept of parametric architecture. Developing the concept of Human Oriented Parametric architecture, it will be discussed the need of implementing time as the lost parameter in current design techniques. Using the media-tic building, of the Spanish architect Enrique Ruiz Geli, as an example of a current design that tries to implement new technologies and parametric ideas in its design process, it will be explained the idea of the need of the building of working with the environment, not defending against it as the basis for a good adaptability. Geli¿s design is currently using 104 Arduino chips for an individual control and performance of the 104 ETFE pillows of the building façade. Through the creation of a virtual stimulus-reaction model of the system façade versus a model in which machine learning have been implemented for a better environmental performance, the efficacy on insulation of both models will be compared. Morphogenetic processes idea will be discussed through also the principle of an adaptable membrane, as the thought solution for future architecture design processes improvement. A model implementing a unique Arduino on the façade, will control the performance of the façade patterns, through, an Artificial Neural Network that will decide the kind of scenario the building is in, activating a Genetic Algorithm that will optimize insulation performance of the ETFE pillows. The final virtual model will be able to obtained the goal proposed, for this thesis, a homogeneous temperature in all the spaces of the building of 22ºC. The maximum thermal optimization obtained, nevertheless, appears if the opening of the pillows is free within and interval of 0 and 1 m thickness. The constrains of the opening of the ETFE pillows to three positions, will be demonstrated more effective than a just stimulus-reaction behaviour, but also, much less effective that an unconstrained façade system. The EmDeplo System will work with a Global behaviour, pattern performance of the façade, but also with a local behaviour for each pillow, giving the option of individual sun shading control. Machine learning implementation will give the façade the possibility to learn from the efficacy of its decisions through time, eliminating the need of an on-off behaviour for defending against the environment. Instead it will work with it, adapting to it, and evolving with its variabilities.
Esta tesis debatira la importancia de la Arquitectura Parametrica Dinamica versus la Arquitectura Parametrica Estatica.Describiento el actual escenario arquitectonico y algoritmico dentro del cual el "Emergency Deployable System" emergio,sera debatida la importancia de la adaptabilidad como el concepto perdido actualmente el la arquitectura parametrica. Desarrollando el concepto de "Human Oriented Parametric Architecture", sera discutida la necesidad de implementar el tiempo como el parametro perdido en los actuales procedimientos de diseño. Empleando el edificio Media-Tic, del arquitecto español Enrique Ruiz Geli, como un ejemplo de un diseño actual que intenta implementar nuevas tecnologias e ideas parametricas en su proceso de diseño,sera explicada la idea de la necesidad del edificio de trabajar con el entorno, no de defenderse contra el, como base para una buena adaptabilidad. El diseño de Geli usa 104 chipsArduino para un control y comportamiento individual de los 104 cojines de ETFe en su fachada. A traves de la creacion de un sistema virtual estimulo-reaccion de la fachada versus un modelo virtual en el cual el aprendizaje de maquinas ha sido implementado para un mejor comportamiento ambiental, la eficacia de ambos modelos sera compararda. La idea de los procesos Morfogeneticos sera discutida a traves del pricipio de una mebrana adaptable, como la solucion propuesta para la mejora futura del proceso de diseño arquitectonico. Un modelo implementando un unico Arduino en la fachada, controlara el comportamiento de los patrones de la fachada a traves de una Red Neuronal Artificial que decidira el tipo de escenario de entorno en que el edificio se encuentra, activando un Algoritmo Genetico que optimizara el aislamiento termico de os cojines ETFE. La maqueta virtual final sera capaz de llegar a la meta propuesta para esta tesis doctoral, una temperatura homogenea en todos los espacios del edificio de 22ºC. La maxima optimizacion termica obtenida, sin embargo, aparece si la posibilidad de apertura de los cojines ETFE es libre en un intervalo entre o y 1m de espesor. La reduccion de las posibilidades de apertura de los cojines a tres posiciones unicamente, sera demostrada como una posibilidad mejoor que el comportamiento estimulo-reaccion, sin embargo, mucho menos efectivo que una posibilidad de apertura de rango libre. El sistema EmDeplo funcionara con un comportamiento global, la variabilidad del patron de la fachada, pero tambien con un comportamiento local, el de cada cojin ETFE, dando la opcion de un control de sombra-sol individual. El aprendizaje de maquinas implementado dara a la fachada la posibilidad de aprender de la eficacia de sus decisiones a lo largo del tiempo, eliminando la necisidad de un comportamiento on-off para defenderse del entorno. En lugar de esta defensa, trabajara con el, adaptamdose a el, y evolucionando con su variabilidad.
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Saygun, Yakup. "Computational Stochastic Morphogenesis." Thesis, Uppsala universitet, Avdelningen för beräkningsvetenskap, 2015. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-257096.

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Self-organizing patterns arise in a variety of ways in nature, the complex patterning observed on animal coats is such an example. It is already known that the mechanisms responsible for pattern formation starts at the developmental stage of an embryo. However, the actual process determining cell fate has been, and still is, unknown. The mathematical interest for pattern formation emerged from the theories formulated by the mathematician and computer scientist Alan Turing in 1952. He attempted to explain the mechanisms behind morphogenesis and how the process of spatial cell differentiation from homogeneous cells lead to organisms with different complexities and shapes. Turing formulated a mathematical theory and proposed a reaction-diffusion system where morphogens, a postulated chemically active substance, moderated the whole mechanism. He concluded that this process was stable as long as diffusion was neglected; otherwise this would lead to a diffusion-driven instability, which is the fundamental part of pattern formation. The mathematical theory describing this process consists of solving partial differential equations and Turing considered deterministic reaction-diffusion systems.   This thesis will start with introducing the reader to the problem and then gradually build up the mathematical theory needed to get an understanding of the stochastic reaction-diffusion systems that is the focus of the thesis. This study will to a large extent simulate stochastic systems using numerical computations and in order to be computationally feasible a compartment-based model will be used. Noise is an inherent part of such systems, so the study will also discuss the effects of noise and morphogen kinetics on different geometries with boundaries of different complexities from one-dimensional cases up to three-dimensions.
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Nelsen, Brian. "Morphogenesis a theory of places /." This title; PDF viewer required. Home page for entire collection, 2010. http://archives.udmercy.edu:8080/dspace/handle/10429/9.

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Briggs, Laura Joanne. "Flagellar morphogenesis in Trypanosoma brucei." Thesis, University of Oxford, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.427895.

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Hooley, Clare Verity. "Morphogenesis of Drosophila renal tubules." Thesis, University of Cambridge, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604211.

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The renal or Malpighian tubules (MpTs) are the major excretory organs of insects. Drosophila has 4 MpTs, an anterior and posterior pair. During embryogenesis these MpTs undergo a morphogenetic programme such that cell rearrangement by convergent extension causes the MpTs to elongate so the short fat tubules become longer thin tubules. In addition directed migration or pathfinding occurs that the MpTs take up a specific location within the body cavity. In this study an analysis of wild type development demonstrates that several aspects of normal MpT morphogenesis are invariant whilst other aspects are less defined. The shape of an organ is a consequence of both extrinsic factors and intrinsic properties. I demonstrate that external tissues and other regulators are controlling morphogenesis of the MpTs through alterations in the cytoskeleton. I find that when the visceral mesoderm is disrupted genetically the MpTs have an aberrant morphology; the visceral mesoderm acts as an extrinsic cue for normal MpT pathfinding. The visceral mesoderm secretes Decapentaplegic (Dpp) and my analysis suggests that Dpp is an attractant controlling the normal MpT migratory behaviour. Downstream activators of the dpp signalling pathway are present in the MpTs and ectopic expression of Dpp in tissues near the extending MpTs affects their morphogenesis. The convergent extension process itself may be intrinsic to the MpTs and can be disrupted when signalling via the Rho family of small GTPases is perturbed. In order to identify novel genes involved in MpT morphogenesis I have analysed lines from a mutagenesis screen previously performed to select for MpT defects. One locus was mapped and characterised as an allele of D-Cbl, previously shown to be in inhibitor of the EGF pathway. I present evidence to show that when the regulation of EGF signalling is disrupted subtle defects on MpT morphology are observed, thus revealing a requirement for the activity of this pathway during MpT morphogenesis.
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Farzaneh, Ali. "Computational morphogenesis of city tissues." Thesis, Open University, 2017. http://oro.open.ac.uk/49302/.

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Scientific discoveries of the 20th century had a profound impact not only on the study of the natural sciences but disciplines worldwide. The studies were rooted in understanding the complex process of organisation, development and evolution in natural systems before attempting to emulate the behaviours in artificial systems, leading to the emergence of new disciplines such as systems theory, complexity science, genetics, developmental and evolutionary biology. The discoveries had a profound impact in understanding the nature of cities as they develop over time. Once considered top-down models in equilibrium, the dynamic qualities of cities could be explained through the study of dynamic complex systems, exhibiting non-deterministic characteristics that over time emerge as organised structures. These characteristics are not exclusive to cities alone; they are inherent to all complex systems. The understanding of cities as complex systems has stimulated a body of research through mathematical and scientific modelling in understanding the behaviour of cities over time. The studies have been strongly focused on the analytical performance of city morphologies and less on the relational qualities of how systems interact to produce functioning spatial configurations. With the rapid rate of urbanisation and the emergence of new cities around the world, the approach to the design of cities remains rooted in static, top-down models. The implications of such models have led to high energy consumption, lack of integration and poor performance. It is a contradiction to consider cities as complex systems but design them as simple systems. The thesis explores principles of complex systems through the study of biological morphogenesis (the formation and development of organisms over time) for their implementation in formalising a design model for the formation, development and evolution of cities. The central contribution of the thesis lies in the computational modelling of cities in three main areas. The first is the co-evolution of networks and block systems towards the generation of differentiated spatial morphologies. Network systems are generated by coupling multi-agent systems and branching systems from the mathematics of natural systems, and the block systems are generated through procedural subdivision and volumetric modelling. The process involves substantial computational coding and the integration of knowledge from outside disciplines including biology, genetics, complexity theory and mathematics. The second is the development of a unified computational model combining morphological, topological and analytical modelling. The integration of the models is contingent on the writing of classes including graph theory, centrality measures and environmental calculations - all classes were written in C#. The third area is the evolutionary modelling of urban systems. The process utilised the open-source evolutionary solver Octopus in evolving solutions. The advantage lay in the populace-based nature of the model in generating differentiated phenotypes - or geometries - as a response to multiple-objectives. The model has been designed to enable the integration of systems of different types. Analytical data can be used as input to influence the model on the types of decisions it can make. The model has also been designed to enable the exploration of multiple design objectives at varying spatial and time scales. A significant part of the design model takes advantage of open source software including the open source language C#. The software have been extensively modified by hard coding. The model is mutable so that others may add new classes and procedures in the future.
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Bunt, Stephanie Marie. "Renal tubule morphogenesis in Drosophila." Thesis, University of Cambridge, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.612231.

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Rauzi, Matteo. "Mapping Subcellular Forces Controlling Morphogenesis." Thesis, Aix-Marseille 2, 2010. http://www.theses.fr/2010AIX22025.

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Pendant le développement d’un embryon, le remodelage des tissus amène un changement de forme: les tissu peuvent s’allonger, s’envaginer, s’étirer etc. Différentes voies de signalisation règlent ces comportements dans le temps et l’espace à travers le contrôle du cytosquelette d’actine et des tensions produites par ce dernier en interaction avec le moteur moléculaire Myosine-II.Quelle est la distribution des forces subcellulaires qui contrôlent la morphogenèse des tissus? Quelle est la nature des forces engendrée set pour finir, quelle est l’origine de ces forces? Voici les questions pour lesquelles ma thèse cherche des réponses. J’utilise l’élongation de la bande germinale de l’embryon de Drosophile comme système modèle afin d’investiguer la mécanique de la morphogenèse des tissus
During embryonic development tissue remodeling leads to shape changes: for example, tissues can elongate, invaginate, and stretch. Distinct signaling pathways regulate in space and time these behaviors through the control ofthe actin cytoskeleton and of myosin-based tension required for cell shapechange. What is the spatiotemporal pattern of subcellular forcesthat orient tissue morphogenesis? What is the nature of the generated forces and finally, what lies at the origin of such forces? These are the main questions that my thesis tries to illuminate. I use the germbandelongation of the Drosophila embryo as a model system to investigate themechanics of tissue morphogenesis
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Books on the topic "Morphogenesis"

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Bourgine, Paul, and Annick Lesne, eds. Morphogenesis. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-13174-5.

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Turing, Alan Mathison. Morphogenesis. Amsterdam: North-Holland, 1992.

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Schwalm, Fritz E. Insect morphogenesis. Basel: Karger, 1988.

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Pismen, Len. Morphogenesis Deconstructed. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-36814-2.

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Davies, Jamie A. Branching Morphogenesis. Boston, MA: Springer US, 2006. http://dx.doi.org/10.1007/0-387-30873-3.

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Nelson, Celeste M., ed. Tissue Morphogenesis. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-1164-6.

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Ribatti, Domenico, ed. Vascular Morphogenesis. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-1462-3.

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Ribatti, Domenico, ed. Vascular Morphogenesis. New York, NY: Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-0916-3.

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Ebrahimkhani, Mo R., and Joshua Hislop, eds. Programmed Morphogenesis. New York, NY: Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-1174-6.

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Archer, Margaret S., ed. Social Morphogenesis. Dordrecht: Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-6128-5.

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Book chapters on the topic "Morphogenesis"

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Morgan, Michael M., MacDonald J. Christie, Thomas Steckler, Ben J. Harrison, Christos Pantelis, Christof Baltes, Thomas Mueggler, et al. "Morphogenesis." In Encyclopedia of Psychopharmacology, 799. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-68706-1_4376.

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Taber, Larry A. "Morphogenesis." In Continuum Modeling in Mechanobiology, 401–517. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-43209-6_8.

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Hangay, George, Susan V. Gruner, F. W. Howard, John L. Capinera, Eugene J. Gerberg, Susan E. Halbert, John B. Heppner, et al. "Morphogenesis." In Encyclopedia of Entomology, 2466. Dordrecht: Springer Netherlands, 2008. http://dx.doi.org/10.1007/978-1-4020-6359-6_4693.

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Bosshard, Hans Heinrich. "Morphogenesis." In Dendrophysica, 185–209. Basel: Birkhäuser Basel, 1990. http://dx.doi.org/10.1007/978-3-0348-7131-0_7.

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Pismen, Len. "Morphogenesis." In Active Matter Within and Around Us, 171–99. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-68421-1_8.

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Perego, Auro Michele. "Morphogenesis." In Unstable Nature, 59–68. Boca Raton: CRC Press, 2023. http://dx.doi.org/10.1201/9781003462040-9.

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Twyman, R. M. "Morphogenesis." In BIOS Instant Notes in Developmental Biology, 38–44. London: Taylor & Francis, 2023. http://dx.doi.org/10.1201/9781003416371-8.

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Lesne, Annick, and Paul Bourgine. "Introduction." In Morphogenesis, 1–13. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-13174-5_1.

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Douady, Stéphane. "Phyllotaxis, or How Plants Do Maths When they Grow." In Morphogenesis, 189–98. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-13174-5_10.

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Marchand, Didier. "The Logic of Forms in the Light of Developmental Biology and Palaeontology." In Morphogenesis, 199–209. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-13174-5_11.

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Conference papers on the topic "Morphogenesis"

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Trusiak, Maciej, Piotr Arcab, Mikołaj Rogalski, Piotr Rogujski, and Luiza Stanaszek. "Multiplexed label-free high-throughput holographic lensless method for live cell migration sensing." In Computational Optical Sensing and Imaging, CTu1B.3. Washington, D.C.: Optica Publishing Group, 2024. http://dx.doi.org/10.1364/cosi.2024.ctu1b.3.

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Cell migration plays crucial role in regeneration, morphogenesis and cancer metastasis. We present a novel hardware-software method for multiplexed (3-cameras) holographic lensless label-free full-culture live-cell quantitative migration sensing with single-cell sensitivity and sub-cellular motion precision.
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"Morphogenesis." In 27th Solvay Conference on Physics. WORLD SCIENTIFIC, 2020. http://dx.doi.org/10.1142/9789813239258_0005.

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Sabin, Jenny E., and Peter Lloyd Jones. "Branching morphogenesis." In ACM SIGGRAPH 2008 art gallery. New York, New York, USA: ACM Press, 2008. http://dx.doi.org/10.1145/1400385.1400391.

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Cho, Hye Min, Maru Garcia, and Maura Palacios Mejia. "Morphogenesis I." In SIGGRAPH '22: Special Interest Group on Computer Graphics and Interactive Techniques Conference. New York, NY, USA: ACM, 2022. http://dx.doi.org/10.1145/3532837.3534949.

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MOROZOVA, NADYA, and ROBERT PENNER. "GEOMETRY OF MORPHOGENESIS." In International Symposium on Mathematical and Computational Biology. WORLD SCIENTIFIC, 2015. http://dx.doi.org/10.1142/9789814667944_0022.

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Zahadat, Payam, Daniel Nicolas Hofstadler, and Thomas Schmickl. "Vascular morphogenesis controller." In GECCO '17: Genetic and Evolutionary Computation Conference. New York, NY, USA: ACM, 2017. http://dx.doi.org/10.1145/3071178.3071247.

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Pappas, Vasileios, Dinesh C. Verma, and Pietro Lio. "Morphogenesis in computer networks." In 2010 IEEE Sarnoff Symposium. IEEE, 2010. http://dx.doi.org/10.1109/sarnof.2010.5469776.

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Bhattacharyya, Arnab. "Morphogenesis as an amorphous computation." In the 3rd conference. New York, New York, USA: ACM Press, 2006. http://dx.doi.org/10.1145/1128022.1128032.

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Shalygo, Yuri. "The Kinetic Basis of Morphogenesis." In European Conference on Artificial Life 2015. The MIT Press, 2015. http://dx.doi.org/10.7551/978-0-262-33027-5-ch027.

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Farzaneh, Ali. "Computational Morphogenesis of Architectural Objects." In eCAADe 2012 : Digital Physicality. eCAADe, 2012. http://dx.doi.org/10.52842/conf.ecaade.2012.2.593.

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Reports on the topic "Morphogenesis"

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Heineike, Benjamin M. Modeling Morphogenesis with Reaction-Diffusion Equations Using Galerkin Spectral Methods. Fort Belvoir, VA: Defense Technical Information Center, May 2002. http://dx.doi.org/10.21236/ada403766.

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Weaver, Valerie M. Adhesion-Linked Protein Tyrosine Phosphatases, Morphogenesis and Breast Cancer Progression. Fort Belvoir, VA: Defense Technical Information Center, July 2004. http://dx.doi.org/10.21236/ada435265.

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Liu, Yi-Hsin. Msx2 Plays a Central Role in Regulating Branching Morphogenesis During Mammary Development. Fort Belvoir, VA: Defense Technical Information Center, October 2001. http://dx.doi.org/10.21236/ada398933.

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Liu, Yi-Hsin. Msx2 Plays a central Role in Regulating Branching Morphogenesis During Mammary Development. Fort Belvoir, VA: Defense Technical Information Center, October 2002. http://dx.doi.org/10.21236/ada412700.

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Patch, Jared R. The Central Role of the Matrix Protein in Nipah Virus Assembly and Morphogenesis. Fort Belvoir, VA: Defense Technical Information Center, February 2007. http://dx.doi.org/10.21236/ad1014034.

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Bednarek, Sebastian, Y. Role of AtCDC48 & the AtCDC48 Regulatory Protein Family, PUX, in Plant Cell Morphogenesis. Office of Scientific and Technical Information (OSTI), November 2009. http://dx.doi.org/10.2172/977066.

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Timko, Michael P. NADPH: Protochlorophyllide Oxidoreductase-Structure, Catalytic Function, and Role in Prolamellar Body Formation and Morphogenesis. Office of Scientific and Technical Information (OSTI), February 2013. http://dx.doi.org/10.2172/1061451.

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Chamovitz, Daniel, and Xing-Wang Deng. Morphogenesis and Light Signal Transduction in Plants: The p27 Subunit of the COP9-Complex. United States Department of Agriculture, 1997. http://dx.doi.org/10.32747/1997.7580666.bard.

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Plants monitor environmental signals and modulate their growth and development in a manner optimal for the prevailing light conditions. The mechanisms by which plants transduce light signals and integrate them with other environmental and developmental signals to regulate plant pattern development are beginning to be unraveled. A large body of knowledge has accumulated regarding the roles of specific photoreceptors in perceiving light signals, and about the downstream developmental responses responding to light (Batschauer, 1999; Chamovitz and Deng, 1996; Deng and Quail, 1999). Still, little is know about the molecular mechanisms connecting the photoreceptors to development, and how these developmental pathways are integrated with additional developmental regulatory pathways to modulate growth. The multi-subunit protein complex COP9 signalosome (previously referred to as the "COP9 complex") has a central role in mediating the light control of plant development, and in general developmental regulation. Arabidopsis mutants that lack this complex develop photomorphogenically even in the absence of light signals (reviewed in Chamovitz and Deng 1996, 1997). Various genetic studies have indicated that the COP9 signalosome acts at the nexus of upstream signals transduced from the individual photoreceptors, and specific downstream signaling pathways. Thus the COP9 signalosome was hypothesized to be a master repressor of photomorphogenesis, and that light acts to abrogate this repression. However, the COP9 signalosome has roles beyond the regulation of photomorphogenesis as all mutants lacking this complex die following early seedling development, and an essentially identical complex has also been detected in animal systems (Chamovitz and Deng, 1995; Seeger et al., 1998; Wei et al., 1998). Our long term objective is to determine the role of the COP9 signalosome in controlling plant development. In this research project we showed that this complex contains at least eight subunits (Karniol et al., 1998; Serino et al., 1999) and that the 27 kD subunit is encoded by the FUS5 locus (Karniol et al., 1999). The FUS5 subunit also has a role extraneous to the COP9 signalosome, and differential kinase activity has been implicated in regulating FUSS and the COP9 signalosome (Karniol et al., 1999). We have also shown that the COP9 signalosome may work together with the translational-regulator eIF3. Our study of the COP9 signalosome is one of the exciting examples of plant science leading the way to discoveries in basic animal science (Chamovitz and Deng, 1995; Karniol and Chamovitz, 2000; Wei and Deng, 1999).
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Dickman, Martin B., and Oded Yarden. Involvement of the PKA and MAPK signal transduction pathways in sclerotial morphogenesis in Sclerotinia sclerotiorum. United States Department of Agriculture, September 2007. http://dx.doi.org/10.32747/2007.7695861.bard.

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The long-term goals of our research are to understand the regulation of sclerotial development and pathogenicity in S. sclerotiorum. The focus in this project is on the elucidation of the signaling events and environmental cues involved in the regulation of these processes, utilizing and continuously developing tools our research groups have established and/or adapted for analysis of S. sclerotiorum. Our stated specific objectives were to: 1. Follow activities and function of S. sclerotiorumPKA. 2. Identify and functionally evaluate effectors of the S. sclerotiorumERK-likeMAPK signaling pathway. 3. Perform structural and functional analysis of genes whose expression is altered under conditions affecting either PKA and/or MAPK. As can be seen below, we have not only met most of the listed goals, but have also expanded our research. We have been working both together and in parallel in order to advance our goals. We have jointly shown how an ERK-likeMAPK is required sclerotia formation. We have analyzed, in parallel, the involvement of PKA in sclerotiogenesis and, interestingly, have reached some overlapping results but each group has provided a slightly different interpretation to the picture obtained. It will be interesting to see how this aspect of the analysis progresses, as we jointly tackle the yet unresloved issues. We have also made progress on the analysis of ser/thr phosphatases (specifically – calcineurin, which has been reported to interact with PKA) and PP2A in S. sclerotiorum as well as the S. sclerotiorum rasgene, which we have cloned and shown induces SMK1, the ERK-like kinase responsible for sclerotia formation. In addition to the time and efforts invested towards reaching the specific goals mentioned, both PIs are actively involved in a major international effort to sequence and annotate the entire S. sclerotiorum genome. Though time consuming (and perhaps requiring divergence of some time and resources from the original workplan), we have given this topic a very high priority to this effort as the long term implications of the success of this venture are enormous.
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Dickman, Martin B., and Oded Yarden. Phosphorylative Transduction of Developmental and Pathogenicity-Related Cues in Sclerotinia Sclerotiorum. United States Department of Agriculture, April 2004. http://dx.doi.org/10.32747/2004.7586472.bard.

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Sclerotinia sclerotiorum (Lib.) de Bary is among the world's most successful and omnivorous fungal plant pathogens. Included in the more than 400 species of plants reported as hosts to this fungus are canola, alfalfa, soybean, sunflower, dry bean, and potato. The general inability to develop resistant germplasm with these economically important crops to this pathogen has focused attention on the need for a more detailed examination of the pathogenic determinants involved in disease development. This proposal involved experiments that examined the involvement of protein phosphorylation during morphogenesis (hyphal elongation and sclerotia formation) and pathogenesis (oxalic acid). Data obtained from our laboratories during the course of this project substantiates the fact that kinases and phosphatases are involved and important for these processes. A mechanistic understanding of the successful strategy(ies) used by S . sclerotiorum in infecting and proliferating in host plants and this linkage to fungal development will provide targets and/or novel approaches with which to design resistant crop plants including interference with fungal pathogenic development. The original objectives of this grant included: I. Clone the cyclic AMP-dependent protein kinase A (PKA) catalytic subunit gene from S.sclerotiorum and determine its role in fungal pathogenicity, OA production (OA) and/or morphogenesis (sclerotia formation). II. Clone and characterize the catalytic and regulatory subunits of the protein phosphatase PP2A holoenzyme complex and determine their role in fungal pathogenicity and/or morphogenesis as well as linkage with PKA-regulation of OA production and sclerotia formation. III. Clone and characterize the adenylate cyclase-encoding gene from S . sclerotiorum and detennine its relationship to the PKA/PP2A-regulated pathway. IV. Analyze the expression patterns of the above-mentioned genes and their products during pathogenesis and determine their linkage with infection and fungal growth.
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