Relevant bibliographies by topics / Mood disorders/epidemiology

Academic literature on the topic 'Mood disorders/epidemiology'

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Published: 10 December 2022
Last updated: 28 January 2023

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Journal articles on the topic "Mood disorders/epidemiology"

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Merikangas, Kathleen Ries, and Nancy C. P. Low. "The epidemiology of mood disorders." Current Psychiatry Reports 6, no. 6 (December 2004): 411–21. http://dx.doi.org/10.1007/s11920-004-0004-1.

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Marneros, Andreas. "Mood disorders: epidemiology and natural history." Psychiatry 5, no. 4 (April 2006): 119–22. http://dx.doi.org/10.1383/psyt.2006.5.4.119.

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Marneros, Andreas. "Mood disorders: epidemiology and natural history." Psychiatry 8, no. 2 (February 2009): 52–55. http://dx.doi.org/10.1016/j.mppsy.2008.10.022.

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Flynn, Heather A. "Epidemiology and Phenomenology of Postpartum Mood Disorders." Psychiatric Annals 35, no. 7 (July 1, 2005): 544–51. http://dx.doi.org/10.3928/0048-5713-20050701-12.

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Feld, Judith, Uriel Halbreich, and Sandhya Karkun. "The Association of Perimenopausal Mood Disorders with Other Reproductive-Related Disorders." CNS Spectrums 10, no. 6 (June 2005): 461–70. http://dx.doi.org/10.1017/s1092852900023154.

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AbstractData regarding the increased incidence of psychiatric illness during midlife in women are still conflicting. However, there is a growing consensus that certain groups of women may in fact be at higher risk for mood symptoms and psychiatric disorders during the perimenopausal transition. Mood symptoms during the perimenopause may be related to mood disorders during other periods of hormonal fluctuation throughout a woman's reproductive lifecycle. Elucidating these associations may advance the understanding of mood disorders during the perimenopausal transition. The epidemiology and treatment of perimenopausal mood symptoms compared with the epidemiology and treatment of mood disorders during the late luteal phase of the menstrual cycle, pregnancy, and postpartum. Common risk factors associated with mood disorders during these periods of hormonal changes or instability include poor lifestyle habits, a history of hormonally related mood disorders, stress and negative life events, ethnicity, and comorbidity. Reproductive-related mood disorders also are subject to an improvement in symptoms in response to treatment with selective serotonin reuptake inhibitors. As the morbidity associated with mood disorders during midlife may be quite significant, and as life expectancy continues to increase, recognition, prevention, and treatment of perimenopausal affective illness is becoming increasingly essential.
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McCracken, James T. "The Epidemiology of Child and Adolescent Mood Disorders." Child and Adolescent Psychiatric Clinics of North America 1, no. 1 (July 1992): 53–72. http://dx.doi.org/10.1016/s1056-4993(18)30611-4.

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Huang, Deborah L., Indraneil Bardhan, Joosun Shin, Jordan F. Karp, and Mijung Park. "Chronic Pain and Mood Disorders in Asian Americans." Asian/Pacific Island Nursing Journal 5, no. 4 (March 24, 2021): 217–26. http://dx.doi.org/10.31372/20200504.1115.

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Purpose: Pain and mood disorder frequently coexist. Yet, for Asian Americans (AAs), scant information about pain and mood disorder is available. Our aims were to compare (1) the rates of pain and mood disorders and (2) the magnitude of associations between pain and mood disorders between AAs and European Americans (EAs), and across different Asian subgroups. Methods: An analytical data was constructed from the Collaborative Psychiatric Epidemiology Studies (CPES), a representative sample of community-residing U.S. adults (n = 9,871). Pain morbidity was assessed by self-report. Mood disorders, including major depression and anxiety disorders, were assessed using the diagnostic interview. Analysis included descriptive statistics and multivariate logistic regression modeling. All analyses were weighted to approximate the U.S. populations, and controlled for sociodemographic and immigration characteristics. Results: Greater proportion of EAs, compared to AAs, endorsed lifetime pain (56.8% vs. 35.8%). Having life pain disorders elevated the likelihood of lifetime mood disorder by more than 2-folds (weight adjusted odds ratio (WAOR): 2.12, 95% CI: 1.77, 2.55). Having pain disorders over the past 12 months elevated the likelihood of mood disorder in the same time period by more than 3-folds (WAOR: 3.29, 95% CI: 2.02, 5.37) among AAs. The magnitude of the association between pain and psychiatric morbidity were greater in Vietnamese Americans compared to other AAs and EAs. Discussion: The conventional belief that rates of pain and mood disorders are greater in AAs than EAs may need to be further examined. Vietnamese Americans may be particularly vulnerable for experience of comorbid pain and mood disorders.
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Souery, D., and J. Mendlewicz. "Molecular genetic findings in mood disorders." Acta Neuropsychiatrica 11, no. 2 (June 1999): 67–70. http://dx.doi.org/10.1017/s092427080003619x.

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Traditional methods used to asses genetic effects, such as twins, adoption and family studies, have demonstrated the role genetic vulnerability factors in the etiology of major psychiatric diseases such as affective disorders and schizophrenia. It remains however impossible, using these methods, to specify the genetic variables involved and the exact mode of transmission of these diseases. New genetic approaches in psychiatry include the use of DNA markers in sophisticated strategies to examine families and populations. Genetic linkage (in families) and allelic association (in unrelated subjects) are the most frequent techniques applied searching for genes in psychiatric diseases. Advances in these methods have permitted their application to complex diseases in which the mode of genetic transmission is unknown. Affective disorders and, in particular, bipolar affective disorder (BPAD) have been examined in many molecular genetic studies which have covered a large part of the genome, specific hypotheses such as mutations have also, been studied. Most recent studies indicate that several chromosomal regions may be involved in the aetiology of affective disorders. Large multi-centre and multi-disciplinary projects are currently underway in Europe and in the US and hopefully will improve our understanding of the genetic factors involved in affective disorders. In parallel to these new developments in molecular genetics, the classical genetic epidemiology, represented by twin, adoption and family studies, have been improved, providing validated models to test the gene-environment interactions.
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Faravelli, Carlo, Benedetta Guerrini Degl'Innocenti, Leandro Aiazzi, Guya Incerpi, and Stefano Pallanti. "Epidemiology of mood disorders: a community survey in Florence." Journal of Affective Disorders 20, no. 2 (October 1990): 135–41. http://dx.doi.org/10.1016/0165-0327(90)90127-t.

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Thomson, Michael, and Verinder Sharma. "Between a rock-a-bye and a hard place: mood disorders during the peripartum period." CNS Spectrums 22, S1 (December 2017): 49–64. http://dx.doi.org/10.1017/s1092852917000852.

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Mood disorders including major depressive disorder and bipolar disorder are common during and after pregnancy. Timely identification and appropriate management of mood episodes is essential to maximize maternal well-being and minimize adverse outcomes. Failure to do so results in maternal suffering and impaired child bonding, and has the potential for devastating outcomes including suicide and infanticide. Women are routinely screened for unipolar depression during or after pregnancy but not for bipolar disorder, in spite of the fact that childbirth is associated with a major risk for onset or exacerbation of bipolar disorder. Delays in detection as well as misdiagnosis of bipolar disorder as major depressive disorder may put women at risk of many adverse consequences, including symptom exacerbation, psychiatric hospitalization, and suicide. A thorough psychiatric assessment is necessary to establish diagnosis, to address safety issues, and to formulate a treatment plan. Treatment of mood disorders during pregnancy is complicated by the potential risks of fetal exposure to psychotropic medications, and the use of these medications during the postpartum period may result in infant medication exposure through breastmilk. These risks of psychotropic medication exposure must be weighed against the risk of untreated mood disorders. This review will discuss the pathophysiology, epidemiology, diagnosis, and treatment of mood disorders during pregnancy and the postpartum period. Screening tools that can be used in the primary care and obstetrics settings to assist in identifying women with peripartum mood disorders will also be discussed.
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Dissertations / Theses on the topic "Mood disorders/epidemiology"

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Merikangas, Kathleen R., Rajni L. Mehta, Beth E. Molnar, Ellen E. Walters, Joel D. Swendsen, Sergio Aguilar-Gaziola, Rob Bijl, et al. "Comorbidity of substance use disorders with mood and anxiety disorders: Results of the international consortium in psychiatric epidemiology." Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2013. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-109936.

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This article reports the results of a cross-national investigation of patterns of comorbidity between substance use and psychiatric disorders in six studies participating in the International Consortium in Psychiatric Epidemiology. In general, there was a strong association between mood and anxiety disorders as well as conduct and antisocial personality disorder with substance disorders at all sites. The results also suggest that there is a continuum in the magnitude of comorbidity as a function of the spectrum of substance use category (use, problems, dependence), as well as a direct relationship between the number of comorbid disorders and increasing levels of severity of substance use disorders (which was particularly pronounced for drugs). Finally, whereas there was no specific temporal pattern of onset for mood disorders in relation to substance disorders, the onset of anxiety disorders was more likely to precede that of substance disorders in all countries. These results illustrate the contribution of cross-national data to understanding the patterns and risk factors for psychopathology and substance use disorders.
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Merikangas, Kathleen R., Rajni L. Mehta, Beth E. Molnar, Ellen E. Walters, Joel D. Swendsen, Sergio Aguilar-Gaziola, Rob Bijl, et al. "Comorbidity of substance use disorders with mood and anxiety disorders: Results of the international consortium in psychiatric epidemiology." Technische Universität Dresden, 1998. https://tud.qucosa.de/id/qucosa%3A26799.

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This article reports the results of a cross-national investigation of patterns of comorbidity between substance use and psychiatric disorders in six studies participating in the International Consortium in Psychiatric Epidemiology. In general, there was a strong association between mood and anxiety disorders as well as conduct and antisocial personality disorder with substance disorders at all sites. The results also suggest that there is a continuum in the magnitude of comorbidity as a function of the spectrum of substance use category (use, problems, dependence), as well as a direct relationship between the number of comorbid disorders and increasing levels of severity of substance use disorders (which was particularly pronounced for drugs). Finally, whereas there was no specific temporal pattern of onset for mood disorders in relation to substance disorders, the onset of anxiety disorders was more likely to precede that of substance disorders in all countries. These results illustrate the contribution of cross-national data to understanding the patterns and risk factors for psychopathology and substance use disorders.
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Kierulf, Jacqueline C. "The association of chronic physical illness and eating attitudes in school-aged children: A secondary analysis based on a community survey of the epidemiology and risk factors for eating and mood disorders in children." Thesis, University of Ottawa (Canada), 1994. http://hdl.handle.net/10393/6686.

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Children with chronic physical illness have twice the risk of psychosocial maladjustment as healthy children. One specific aspect of mental functioning is abnormal eating attitudes, hypothesized to be an early stage of an eating disorder. To date, there has been no study of the association of a variety of physical chronic illnesses with a specific psychiatric syndrome in a diagnostically heterogeneous population, comparing chronically ill children with healthy children. This study examined the association between chronic illness and eating attitudes in a population of school children from Western Quebec. A checklist of chronic medical conditions was added to the parent questionnaire in order to determine presence of chronic illness. There were two objectives of this thesis. The first objective was to determine whether chronic illness was associated with eating attitudes in children as measured by the Eating Attitudes Test. The second study objective was to determine whether there was a meaningful group difference in othcr psychometric scores of depression, family functioning and stress between children with a chronic illness and children without a chronic illness. (Abstract shortened by UMI.)
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Krieger, Fernanda Valle. "Refinando o diagnóstico de Transtorno de Oposição e Desafio na infância e adolescência: validação e caracterização da dimensão irritável." Universidade de São Paulo, 2015. http://www.teses.usp.br/teses/disponiveis/5/5142/tde-09062015-163129/.

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O Transtorno de Oposição e Desafio (TOD) é definido por um padrão recorrente de comportamento desafiante, desobediente e hostil com início na infância e adolescência e caracteriza-se por uma alta taxa de comorbidades. Estudos longitudinais apontam o TOD na infância como um dos principais preditores de psicopatologia na idade adulta. Uma possível explicação para a grande heterogeneidade de comorbidades e trajetórias longitudinais é de que o diagnóstico de TOD abrange distintas dimensões de sintomas, cada qual com seu desfecho. O primeiro objetivo desta tese foi a validação das distintas dimensões do TOD em uma amostra comunitária Brasileira composta de 2512 sujeitos. Através de análise fatorial confirmatória, demonstramos que o modelo que melhor representa a heterogeneidade do TOD é composto por três dimensões: a dimensão \"argumentative/defiant\" que está associada com transtorno de déficit de atenção/hiperatividade (TDAH); a dimensão \"vindictiveness\" que possui associação com transtorno de conduta (TC); e a dimensão \"angry/irritable mood\" onde predominam as associações com transtornos depressivos e de ansiedade. O objetivo seguinte foi investigar o papel da dimensão irritável na classificação nosológica dos transtornos mentais na infância e adolescência. A apresentação da irritabilidade é um aspecto crucial: irritabilidade crônica caracterizada por baixa tolerância à frustração e frequentes explosões de raiva, que é distinta da apresentação episódica, associada ao diagnóstico Transtorno de Humor Bipolar (TB). \"Severe mood dysregulation\", \"disruptive mood dysregulation disorder\", ou dimensão irritável do TOD são formas distintas de classificar o fenótipo de irritabilidade crônica. Entretanto, independente da classificação utilizada, a alta taxa de comorbidades é invariavelmente o denominador comum em estudos sobre irritabilidade. Neste sentido, examinamos o impacto da irritabilidade como uma dimensão subjacente a vários transtornos. Para tanto, avaliamos o impacto da dimensão irritável do TOD através de vários cenários: indivíduos sem diagnóstico, indivíduos com TDAH e sujeitos com transtornos emocionais. Esta 9 investigação foi realizada em duas amostras, uma brasileira constituída por 2.512 sujeitos e uma amostra britânica composta de 7.977 sujeitos. Os resultados demonstram que a irritabilidade está associada ao aumento do prejuízo funcional independente do diagnóstico comórbido concomitante. Seguindo esta linha, investigamos a influência genética na etiologia da irritabilidade. Para tanto, criamos um escore poligênico que incluiu polimorfismos associados à baixa tolerância à frustração, raiva, agressividade reativa e labilidade emocional. O escore poligênico foi altamente preditivo dos níveis de irritabilidade em 350 sujeitos da amostra brasileira. A associação foi específica para irritabilidade e não foi significativa para TDAH, TOD ou medidas contínuas de sintomas. Além disso, a influência genética se manteve mesmo quando fatores ambientais foram incluídos no modelo estatístico. Por fim, quando testada em diferentes ambientes, a influência genética na etiologia da irritabilidade foi mais importante em ambientes de alto risco, sugerindo uma correlação gene-ambiente (rGE). Concluindo, nossos resultados sugerem que a irritabilidade se caracteriza como um traço dimensional subjacente a inúmeros transtornos na infância e adolescência e agregando impacto e prejuízo funcional. Neste sentido, o constructo da irritabilidade se enquadra no conceito do \"Research Domain Criteria\" (RDoC) que propõe o entendimento dos transtornos mentais através de dimensões subjacentes aos diagnósticos clínicos
The Oppositional Defiant Disorder (ODD) is defined as a pattern of disobedient, hostile and defiant behavior beginning in childhood or adolescence and often accompanied by a wide range of comorbidities. Longitudinal studies support ODD as a predictor of psychopathology in adulthood. A potential explanation for such heterogeneity of comorbidities and longitudinal trajectories is that ODD diagnosis encompasses distinct clusters of symptoms, each with its outcome. The first aim of this work was the validation of ODD dimensions in a Brazilian community sample of 2512 subjects. Confirmatory factorial analysis showed that the best model for ODD comprised three dimensions: an \"argumentative/defiant\" dimension, which associates with attention deficit/hyperactivity disorder (ADHD); a \"vindictiveness\" dimension, which associates with conduct disorder (CD); and an \"angry/irritable\" dimension where emotional disorders such as depression and anxiety are the most common associations. The next step was the investigation of the role of the irritable dimension of oppositionality in diagnostic classifications of childhood mental disorders. The pattern of irritability is a crucial point: its chronic presentation as easy annoyance and frequent temper outbursts should be differentiated from the episodic course of irritability associated with the specific diagnosis of Bipolar Disorder (BD). \"Severe mood dysregulation\", \"disruptive mood dysregulation disorder\", and the irritable dimension of oppositionality are different ways to classify the chronic irritability phenotype. However, regardless of the classification, the high rate of comorbidities is invariably the common denominator in studies of irritability. Therefore, we examined the impact of irritability as a dimension cutting across multiple settings: individuals without any diagnosis, subjects with ADHD, and also those with emotional disorders. For that we used two samples, one from Brazil, with 2.512 subjects, and one from the UK, with 7.977 individuals. Results showed that irritability associates with increased functional impairment regardless of concurrent comorbid status. We then investigated the genetic influence on the etiology of irritability. A polygenic score was generated encompassing polymorphisms previously associated with anger, emotional lability and reactive aggression. The polygenic score significantly predicted irritability in 350 subjects in the brazilian sample, yet failed to predict ADHD, ODD, CD and continuous measures of symptoms. Moreover, the association between the polygenic score and irritability remained significant even after taking into account environmental factors. Finally, when stratified across diverse levels of environmental risk, genetic influence upon the etiology of irritability appears to be stronger in high-risk environments. Taken together, our results suggest that irritability is characterized as a dimensional trait that underlies multiple disorders, adding functional impairment. Thus, the construct of irritability fits well within the concept of Research Domain Criteria (RDoC) that suggests that mental disorders should be understood through dimensions underlying diagnostic categories
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Comaskey, Brenda. "Maternal mood and anxiety disorders and child school readiness: a Manitoba population-based study." 2015. http://hdl.handle.net/1993/30399.

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Title: Maternal Mood and Anxiety Disorders and Child School Readiness: A Manitoba Population-Based Study Objective: This study examined the relationship between several features of maternal Mood/Anxiety – timing, recurrence/persistence and severity – and child development on five key areas of school readiness at kindergarten, controlling for health, demographic and socio-economic factors. Methods: Using administrative data to further understand these relationships at a population level and over time, 18,331mother-child pairs were linked using a unique identifier. Maternal Mood/Anxiety was defined using the number of physician visits for mood and anxiety disorders and the number of prescriptions filled for antidepressants or sedatives/hypnotics. The outcome measures were child scores on five domains of the Early Development Instrument, a population-level tool for determining readiness for school at an aggregate level. Structural equation modeling was used to examine the relationship between maternal Mood/Anxiety and child outcomes for individual time periods, Mood/Anxiety recurrence/persistence and Mood/Anxiety severity. Infant health at birth, the family/socio-economic environment, child age and child gender were also entered into the models. Stratified analysis was undertaken to determine whether SES moderates the relationship between Mood/Anxiety and child outcomes. Results: Maternal Mood/Anxiety had a modest significant negative association with EDI scores, particularly for social, emotional and physical development. Prenatal and recurrent Mood/Anxiety were associated with lower scores on all five outcome domains. The influence of maternal Mood/Anxiety was mediated by the family environment, which had a strong, significant association with the outcomes, particularly for language and cognitive development. Infant health at birth was significantly associated with child outcomes, particularly for physical health. Socio-economic status (SES) was a moderator of the relationship between Mood/Anxiety and child outcomes. Conclusion: Using administrative data to define maternal mood and anxiety disorders is a sensitive measure to detect differences in five key areas of child development, particularly for social, emotional and physical outcomes. Family-related variables had a much stronger relationship with child outcomes than mood and anxiety disorders. Findings from this study can be used to design, implement and evaluate high quality clinical, program and policy interventions to support mothers and families and the healthy development of their children.
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McCabe, Delia. "The neurological impact of specific nutrients on female stress: a two-phase, sequential, mixed methods study." Thesis, 2019. http://hdl.handle.net/2440/122611.

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A growing body of literature suggests that the intake of specific nutrients impact mental health, and recent findings support a causative relationship between dietary patterns and affective disorders, whilst also providing evidence of a robust relationship between affective disorders and chronic stress. There is however a paucity of evidence regarding the relationship between chronic stress and nutrition. This thesis reports on research aimed to provide insight into the relationship between chronic stress and the intake of specific dietary nutrients among women. A systematic review examined the current evidence regarding nutrient intake and stress levels. A cross-sectional survey followed, in which a sample of women provided information regarding their perceived stress levels and nutrient intake via diet and dietary supplements. A participatory action research project concluded the study, wherein the lived experience of stress and the role of diet and/or dietary supplementation to manage stress was investigated. The systematic review revealed that there was insufficient evidence to support the intake of specific dietary nutrients either via diet or supplementation to manage stress level. After adjustment, data from the cross-sectional survey revealed a further lack of evidence to support a robust relationship between stress and specific nutrients consumed via the diet or supplementation. The lived experience of stress and the use of specific nutrients to manage stress among a group of women in the PAR project was influenced by relationships and confusion and scepticism regarding the usefulness of nutrients to impact mental wellbeing, while early life stress and thinking patterns were further factors influencing the experience of stress. Evidence regarding the neurobiological efficacy of nutrient intervention is required to support decision-making by clinicians and policy makers in light of the well-established role that chronic stress plays in the development of affective disorders among women globally. This study provides a first step towards the development of a knowledge base regarding the role that specific nutrients play in the stress response among women, although further research is warranted due to the far reaching neurobiological effects of chronic stress.
Thesis (Ph.D.) -- University of Adelaide, The Joanna Briggs Institute, 2019
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Books on the topic "Mood disorders/epidemiology"

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J, Power Michael, ed. Mood disorders: A handbook of science and practice. Chichester, England: John Wiley & Sons, 2004.

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A, Cory Gerald, ed. The evolutionary epidemiology of mania and depression: A theoretical and empirical interpretation of mood disorders. Lewiston: Edwin Mellen Press, 2008.

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Stress and adaptation in the context of culture: Depression in a Southern Black community. Albany, N.Y: State University of New York Press, 1991.

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Robinson, Elise B., Benjamin M. Neale, and Mark J. Daly. Diagnosis and Epidemiology of Pediatric Psychiatric Disorders. Edited by Dennis S. Charney, Eric J. Nestler, Pamela Sklar, and Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0058.

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Pediatric psychiatric conditions are rising in estimated prevalence, and these disorders place an enormous burden on parents, educators, and the health care system. This rise in prevalence likely contains elements of diagnostic changes, greater awareness of these disorders, and true changes in incidence. It has been estimated that there is nearly a 50% lifetime childhood prevalence of one or more mood, anxiety, or behavioral disorders (excluding eating and substance abuse disorders) and that more than 20% of children meet the definition of severe impairment. This chapter focuses on epidemiology, heritability, and implied genetic architecture in representative pediatric neuropsychiatric conditions. We consider five major diagnostic categories and highlight major diagnosis within each, specifically, intellectual disability, pervasive developmental disorders (autism spectrum disorder [ASD]), hyperactive and inattentive behavior (attention deficit/hyperactivity disorder [ADHD]), obsessive compulsive disorder (OCD) and tic disorders (TD) (which includes Tourette Syndrome [TS] and other chronic tic disorders).
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Black, Donald W. Epidemiology and Phenomenology of Compulsive Buying Disorder. Edited by Jon E. Grant and Marc N. Potenza. Oxford University Press, 2012. http://dx.doi.org/10.1093/oxfordhb/9780195389715.013.0072.

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Compulsive buying disorder (CBD) is defined as excessive shopping cognitions and buying behavior that leads to distress or impairment. This chapter presents an overview of its definition and recognition, clinical symptoms, epidemiology, natural history, and both cultural and family factors. Compulsive buying disorder is found worldwide and has a lifetime prevalence of 5.8% in the U.S. general population. The disorder has a female preponderance, has an onset in the late teen years or early 20s, appears to be chronic or recurrent, and occurs mainly in women. Subjects with CBD report a preoccupation with shopping, prepurchase tension or anxiety, and a sense of relief following a purchase. Compulsive buying disorder is associated with significant psychiatric comorbidity, particularly mood and anxiety disorders, substance use disorders, eating disorders, other disorders of impulse control, and Axis II disorders, although there is no special “shopping” personality. The disorder tends to run in families, and these families are filled with mood and substance use disorders.
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Reis Merikangas, Kathleen, and Rebecca Hommer. Psychiatric Epidemiology: Concepts and Findings. Edited by Thomas H. Ollendick, Susan W. White, and Bradley A. White. Oxford University Press, 2018. http://dx.doi.org/10.1093/oxfordhb/9780190634841.013.5.

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This chapter provides background on the discipline of epidemiology and its contribution to our understanding of childhood mental disorders. A summary is provided of data on the prevalence and correlates of mental disorders in children and adolescents in community surveys. Anxiety disorders are the most frequent conditions in children, followed by behavior disorders, mood disorders, and substance use disorders. About half of youth with one disorder also meet criteria for a second disorder, demonstrating the pervasive nature of comorbidity even in childhood. Less than half of youth with current mental disorders receive mental health specialty treatment. However, those with the most severe disorders tend to receive mental health services. Current issues that are now being identified in the field of child psychiatric epidemiology includes refinement of classification and assessment; integration of child and adult psychiatric epidemiology; and evaluation of both mental and medical disorders in children.
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Calamari, John E., Heather M. Chik, Noelle K. Pontarelli, and Brandon L. DeJong. Phenomenology and Epidemiology of Obsessive Compulsive Disorder. Edited by Gail Steketee. Oxford University Press, 2012. http://dx.doi.org/10.1093/oxfordhb/9780195376210.013.0016.

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Obsessive compulsive disorder (OCD) is a complex, often debilitating syndrome that significantly diminishes quality of life. Although the exact prevalence of OCD is unclear, estimates suggest that it is a common form of psychopathology in the West and throughout the world. A challenge to researchers and clinicians is the significant heterogeneity of OCD. Initial heterogeneity research points to important subtypes of the disorder. Elucidation of disorder heterogeneity might advance etiologic theory and treatment research, and suggest where OCD or OCD-like conditions should be placed in a comprehensive psychiatric disorder nosology. OCD more often occurs with other psychiatric disorders, and evaluation of OCD comorbidity will help clarify this condition’s relation to anxiety disorders, mood disorders, and conditions posited to be part of a broad OCD spectrum. Despite significant advancements, much work remains before we can fully understand obsessional disorders and the relation of OCD to commonly experienced negative intrusive thoughts.
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Larocca, Nicholas G. Cognitive Impairment and Mood Disturbances. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199341016.003.0018.

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This chapter presents a comprehensive review of two of the most prevalent symptoms in persons with multiple sclerosis. While cognitive impairment and mood disorders may affect at least half of the MS population, and can have a significant effect on function and quality of life, they are often under recognized and under treated. The epidemiology and most common clinical manifestations of cognitive dysfunction and mood disorders are presented, along with a detailed discussion of screening and assessment tools. Pharmacologic and behavioral treatment interventions are reviewed, with analyses of their comparative efficacy.
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Schneck, Christopher. Treating Depression and Bipolar Disorder in Integrated Care Settings. Edited by Robert E. Feinstein, Joseph V. Connelly, and Marilyn S. Feinstein. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190276201.003.0012.

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Primary care clinics are the de facto treatment settings for patients with major depression and bipolar disorder. Primary care patients with mood disorders are more difficult to assess and treat than patients without such disorders, often have comorbid medical and psychiatric conditions, and require greater practice resources for optimal management. Because current treatment of mood disorder patients in primary care settings is often minimally adequate, changes in overall management strategies are needed to improve outcomes. This chapter describes pathways by which primary care providers can implement an integrated care and collaborative model likely to improve patient outcomes. It describes the epidemiology and costs of mood disorders, as well as basic pharmacologic and psychosocial approaches useful in primary care settings. Depressed patients who are refractory to treatment and patients with bipolar disorder are more complicated to manage and almost always require collaboration with a behavioral health specialist and a consulting psychiatrist.
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Genetic analysis of complex traits: Proceedings of Genetic Analysis Workshop 5, held at Chantilly, France, September 2-5, 1987. Liss, 1989.

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Book chapters on the topic "Mood disorders/epidemiology"

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Jacobi, Frank, Simone Rosi, Carlo Faravelli, Renee Goodwin, Saena Arbabzadeh-Bouchez, and Jean-Pierre Lépine. "The Epidemiology of Mood Disorders." In Mood Disorders, 1–34. Chichester, UK: John Wiley & Sons, Ltd, 2005. http://dx.doi.org/10.1002/0470094281.ch1.

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Meltzer-Brody, Samantha, and David Rubinow. "An Overview of Perinatal Mood and Anxiety Disorders: Epidemiology and Etiology." In Women's Mood Disorders, 5–16. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-71497-0_2.

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Jauhar, Sameer, and Jonathan Cavanagh. "Classification and Epidemiology of Bipolar Disorder." In The Wiley-Blackwell Handbook of Mood Disorders, 289–309. Chichester, UK: John Wiley & Sons Ltd, 2013. http://dx.doi.org/10.1002/9781118316153.ch11.

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Bebbington, Paul. "The Classification and Epidemiology of Unipolar Depression." In The Wiley-Blackwell Handbook of Mood Disorders, 1–37. Chichester, UK: John Wiley & Sons Ltd, 2013. http://dx.doi.org/10.1002/9781118316153.ch1.

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Poznanski, Elva O., and Hartmut B. Mokros. "Phenomenology and Epidemiology of Mood Disorders in Children and Adolescents." In Handbook of Depression in Children and Adolescents, 19–39. Boston, MA: Springer US, 1994. http://dx.doi.org/10.1007/978-1-4899-1510-8_2.

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6

Kessing, Lars Vedel. "Epidemiology of mood disorders." In New Oxford Textbook of Psychiatry, edited by John R. Geddes, Nancy C. Andreasen, and Guy M. Goodwin, 691–99. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198713005.003.0067.

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Unipolar depressive disorder and bipolar disorder are common mental diseases that impose a very high societal burden in terms of morbidity, mortality, lost productivity, and costs. The 1-year prevalence of bipolar disorder is 1–2%, while the 1-year prevalence of depressive disorder is 3–6%. Mean age at onset is in the early to mid twenties for bipolar disorder and around 30 years of age for depressive disorder. The risk of recurrence of episodes after a first depression or manic episode is high, especially for bipolar disorder, and approximately 1% of patients convert from depressive disorder to bipolar disorder per year. Comorbidity with anxiety disorders, alcohol and drug dependence, and personality disorders is high. Both disorders are associated with long-term deficits in social, functional, and cognitive outcomes, and life expectancy is decreased by approximately 10 years, emphasizing the need for future studies on early intervention.
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Joyce, Peter R. "Epidemiology of mood disorders." In New Oxford Textbook of Psychiatry, 645–50. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199696758.003.0086.

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The Global Burden of Disease, which is a comprehensive assessment of mortality and disability from diseases and injuries in 1990 and projected to 2020, highlights the importance of mood disorders for the world. Using the measure of disability-adjusted life years, it was determined that unipolar major depression was the fourth leading cause of disease burden in the world. It was also projected that, in the year 2020, unipolar major depression would be the second leading cause of disease burden in the world. Disabilityadjusted life years is based on both mortality and disability. If one looks at disability alone, then unipolar major depression was the leading cause of disability in the world in 1990, and bipolar disorder was the sixth leading cause. Across the world, 10.7 per cent of disability can be attributed to unipolar major depression and, in developed countries, unipolar major depression contributes to nearly 20 per cent of disease burden in women aged from 15 to 44 years. This chapter addresses bipolar disorders and depressive disorders, covering diagnostic issues, prevalence, comorbidity, use of health services, and risk factors for both types of disorder.
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Cowen, Philip, Paul Harrison, and Tom Burns. "Mood disorders." In Shorter Oxford Textbook of Psychiatry, 205–53. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199605613.003.0010.

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Chapter 10 discusses mood disorders, including clinical features, transcultural features, classification and differential diagnosis, epidemiology and aetiology, course and prognosis, acute treatment of depression and mania, longer-term treatment strategies, assessment and management of depressive disorders, assessment of mania, and management of manic patients.
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Brent, David, and Boris Birmaher. "Paediatric mood disorders." In New Oxford Textbook of Psychiatry, 1669–80. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199696758.003.0218.

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In this chapter, we describe the nosology and epidemiology of paediatric unipolar and bipolar disorders, risk factors and predictors of course, and the evidence base for pharmacological and psychosocial treatments. We conclude this chapter by suggesting areas for future research.
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McKnight, Rebecca, Jonathan Price, and John Geddes. "Mood disorders." In Psychiatry. Oxford University Press, 2019. http://dx.doi.org/10.1093/oso/9780198754008.003.0029.

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Variations in mood are part of normal experience; we all have our ‘good’ and ‘bad’ days and different ways of managing these. Sadness is a natural re­sponse to loss, adversity, stress, or other negative life experiences and is not necessarily abnormal. The main difference between normal sadness and a mood disorder is that normal sadness is usually a temporary state strongly relating to the person’s current situation, whereas mood disorder is a more persistent pervasive change in mood which affects social and occupational functioning. Primary mood (or ‘affective’) disorders are very common, and are also seen in most other psychiatric disorders or co-morbid to a physical illness. The distribution of mood variation in the general population is probably continuous, producing a spec­trum of severity (see Fig. 21.1). As with all psychiatric disorders, classification is descriptive and based on clinical characteristics. The most useful current approach to classification is based on the clinical course. Fundamental elements of this approach include: … ● classifying an illness as a single episode, recurrent, or persistent; ● distinguishing between people who have only low mood (unipolar depression) and those who also have elated mood (bipolar disorder); ● classifying episodes of illness according to severity: depressive episodes are mild, moderate, or severe; elated mood is hypomanic or manic (Table 21.1). … The classification includes two categories for less severe and more chronic illnesses: … ● Dysthymia: chronic mildly low mood which lasts at least several years but does not meet criteria for a recurrent depressive disorder. ● Cyclothymia: chronic instability of mood with periods of mild depressive and elation, none of which are severe enough to meet criteria for bipolar disorder or recurrent depressive disorder. It is often seen in relatives of those who have bipolar disorder, and some patients may eventually meet criteria for bipolar disorder themselves. The prevalence of mood disorders is hard to accurately ascertain, as many patients with low mood do not seek professional help. This is especially common in men. However, data from research studies (which tend to use structured diagnostic criteria) and large national sur­veys (self- report) give very similar results, outlined in Table 21.2. Bipolar disorder epidemiology is well cap­tured, as patients tend to seek help and the diagnostic criteria are well defined.
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