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Relevant bibliographies by topics / Molecular epidemiology

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Academic literature on the topic 'Molecular epidemiology'

Author: Grafiati

Published: 4 June 2021

Last updated: 7 September 2023

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Journal articles on the topic "Molecular epidemiology"

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Bauchet, Luc, and Quinn T. Ostrom. "Epidemiology and Molecular Epidemiology." Neurosurgery Clinics of North America 30, no. 1 (January 2019): 1–16. http://dx.doi.org/10.1016/j.nec.2018.08.010.

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Mathema, Barun, Natalia E. Kurepina, Pablo J. Bifani, and Barry N. Kreiswirth. "Molecular Epidemiology of Tuberculosis: Current Insights." Clinical Microbiology Reviews 19, no. 4 (October 2006): 658–85. http://dx.doi.org/10.1128/cmr.00061-05.

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SUMMARY Molecular epidemiologic studies of tuberculosis (TB) have focused largely on utilizing molecular techniques to address short- and long-term epidemiologic questions, such as in outbreak investigations and in assessing the global dissemination of strains, respectively. This is done primarily by examining the extent of genetic diversity of clinical strains of Mycobacterium tuberculosis. When molecular methods are used in conjunction with classical epidemiology, their utility for TB control has been realized. For instance, molecular epidemiologic studies have added much-needed accuracy and precision in describing transmission dynamics, and they have facilitated investigation of previously unresolved issues, such as estimates of recent-versus-reactive disease and the extent of exogenous reinfection. In addition, there is mounting evidence to suggest that specific strains of M. tuberculosis belonging to discrete phylogenetic clusters (lineages) may differ in virulence, pathogenesis, and epidemiologic characteristics, all of which may significantly impact TB control and vaccine development strategies. Here, we review the current methods, concepts, and applications of molecular approaches used to better understand the epidemiology of TB.

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Rundle, Andrew. "Molecular Epidemiology of Physical Activity and Cancer." Cancer Epidemiology, Biomarkers & Prevention 14, no. 1 (January 1, 2005): 227–36. http://dx.doi.org/10.1158/1055-9965.227.14.1.

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Abstract As in other areas of epidemiology, researchers studying physical activity and cancer have begun to include laboratory analyses of biological specimens in their studies. The incorporation of these “biomarkers” into epidemiology has been termed molecular epidemiology and is an approach primarily developed to study chemical carcinogens. Thus far, there has been no discussion in the field on how the established molecular epidemiologic framework might be adapted for research into physical activity, what methodologic needs exist, what the goals of such an approach might be, and what limitations exist. This article relates the literature on molecular epidemiology to the needs of physical activity research and tries to set research priorities for the field as it moves in this new direction. Although this approach will be very useful for investigating the mechanisms through which physical activity exerts effects, there are several challenges for physical activity epidemiologists in adapting molecular epidemiologic approaches. Primarily, there are currently no available biomarkers that might be considered measures of exposure or biologically effective dose. In addition, most available biomarkers of intermediate effects have been tested in training studies at activity levels much higher than those seen in population-based epidemiologic studies. Thus, it is not clear whether these biomarkers are valid at lower activity levels. Furthermore, the nature of the relationship between activity and many available biomarkers depends very much on the context of the activity. Addressing these issues should be a priority if we are to develop a molecular epidemiologic paradigm for studying physical activity.

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Wilcox, Alien J. "Molecular Epidemiology." Epidemiology 6, no. 5 (September 1995): 561–62. http://dx.doi.org/10.1097/00001648-199509000-00019.

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Boffetta, P. "Molecular epidemiology." Journal of Internal Medicine 248, no. 6 (December 2000): 447–54. http://dx.doi.org/10.1046/j.1365-2796.2000.00777.x.

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Boffetta, P. "Molecular epidemiology." Journal of Internal Medicine 249, S741 (February 2001): 129–36. http://dx.doi.org/10.1046/j.1365-2796.2001.00777.x.

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Boffetta, P. "Molecular epidemiology." Journal of Internal Medicine 248, no. 6 (July 7, 2008): 447–54. http://dx.doi.org/10.1111/j.1365-2796.2000.00777.x.

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Nishi, Akihiro, Ichiro Kawachi, Karestan C. Koenen, Kana Wu, Reiko Nishihara, and Shuji Ogino. "Lifecourse Epidemiology and Molecular Pathological Epidemiology." American Journal of Preventive Medicine 48, no. 1 (January 2015): 116–19. http://dx.doi.org/10.1016/j.amepre.2014.09.031.

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Najafzadeh, Mohammad Javad, Jiufeng Sun, Vania A. Vicente, Corne H. W. Klaassen, Alexandro Bonifaz, A. H. G. Gerrits van den Ende, Steph B. J. Menken, and G. Sybren de Hoog. "Molecular Epidemiology ofFonsecaeaSpecies." Emerging Infectious Diseases 17, no. 3 (March 2011): 464–69. http://dx.doi.org/10.3201/eid1703.100555.

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Brady, R. C. "Meningococcal Molecular Epidemiology." AAP Grand Rounds 34, no. 3 (September 1, 2015): 26. http://dx.doi.org/10.1542/gr.34-3-26.

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Dissertations / Theses on the topic "Molecular epidemiology"

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Petersson, Ramona. "Molecular epidemiology of tuberculosis." Stockholm : Umeå universitet, 2009. http://diss.kib.ki.se/2009/978-91-7409-456-5/.

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Kulaga, Sophie. "Molecular epidemiology of tuberculosis transmission." Thesis, McGill University, 2004. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=84278.

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We conducted a molecular epidemiologic study of tuberculosis (TB) transmission during the years 1996--98 on the Island of Montreal. By combining public health data on the 528 reported cases with IS6110 DNA fingerprints for 430, we detected an overall low frequency of transmission manifesting as secondary cases of active disease. We also identified an important sub-group of TB patients who harbour isolates with matching patterns. Depending on the matching criterion used we attributed between 6% (95% CI: 4, 9%) and 22% (95% CI: 18, 27%) of TB cases to recent transmission; the vast majority of active TB disease reflects infection acquired at an earlier time and/or a different place. However, Haitian-born TB patients yielded a disproportionately high frequency of isolates belonging to matching "clusters" (21%; 95% CI: 13, 32%), while, other foreign-born patients have disproportionately low numbers of clustered isolates (5%; 95% CI: 3, 9%).
The classical interpretation of such results is that there is more ongoing transmission within this immigrant sub-group. We explored an alternative hypothesis: that M. tuberculosis isolates from Haitian-born patients demonstrate reduced genetic diversity reflecting TB transmission patterns in their previously isolated country of origin---hence that a bacterial founder effect accounts for the higher frequency of matching fingerprints. Using a recently introduced measure of fingerprint similarity, genetic distance, we assessed the extent of pattern diversity. The median nearest genetic distance (NGD) was 130 months (inter-quartile range (IQR): 98--201 months) among the 47 distinct isolates from Haitian-born patients; among the non-Haitian foreign-born, the median NGD for the 191 distinct isolates was 128 months (IQR: 103--170 months). Hence the overall genetic heterogeneity of M. tuberculosis organisms among Haitian-born Montrealers was as great as that among a group of patients born in 70 other countries. Local transmission among the Haitian-born remains the most likely scenario.
We demonstrated that a continuous measure, such as genetic distance, may also permit researchers to address a challenge to the interpretation of M. tuberculosis molecular typing results: how to determine whether highly similar, non-identical fingerprint patterns in fact reflect underlying "matches." The distribution of NGD for isolates initially classified as identical (10--27 months), similar (15--108 months) and unique (40--244 months) suggested a possible cut-point of 40 months. Use of this cut-point labelled 19% of isolates as "clustered", suggesting that 14% of Montreal TB cases reflected transmission during the study period.

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Shrivastava, Jaya. "Molecular epidemiology of Schistosoma japonicum." Thesis, University of Oxford, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.414227.

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Taweenan, Weerapol. "Molecular epidemiology of Giardia duodenalis." Thesis, University of Liverpool, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.539914.

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Giardia duodenalis is a parasitic protozoan that affects the gastrointestinal tract, causing abdominal disorders of various animals and humans. To date, G. duodenalis has been genotypically divided into seven groups (assemblages), namely A to G, found in different host ranges. Whilst assemblages C to G are specific genotypes affecting restricted animal hosts, assemblages A and B parasitise both humans and a number of animal species, and have been considered as having zoonotic potential. The main objective of the current study was to investigate the molecular epidemiology of G. duodenalis in animals and humans in the UK. The current study also evaluated multilocus genotyping and determined the protein changes between assemblages A and B.

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Parkar, Unaiza. "Molecular epidemiology of Blastocystis infections." Thesis, Parkar, Unaiza (2016) Molecular epidemiology of Blastocystis infections. PhD thesis, Murdoch University, 2016. https://researchrepository.murdoch.edu.au/id/eprint/33832/.

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Blastocystis is an enteric protist and one of the most frequently reported parasitic infections in humans and a variety of animal hosts worldwide. The genus Blastocystis consists of numerous genetically distinct groups, referred to as subtypes (STs). Some STs are highly host specific, while others display moderate or low host specificity. Therefore, the aims of this study were to determine the prevalence amongst various animal hosts (captive, free-ranging and wild), genetic diversity and zoonotic potential of Blastocystis. As polyparasitism is considered to be the norm in wildlife, the final aim of this study was to develop a molecular-based diagnostic method for the simultaneous detection of Blastocystis, Cryptosporidium sp. and Giardia duodenalis in Australian native fauna. These aims were achieved by sampling captive animals and their keepers from the Perth Zoo. Also, animal samples were obtained from other zoos in Australia and Europe. Samples from free-ranging and wild non-human primates (NHPs) and Australian native fauna were also included in this study. All samples were screened for Blastocystis using Polymerase Chain Reaction (PCR), followed by phylogenetic analyses to characterise these isolates in order to determine the genetic diversity and zoonotic potential of isolates within the Blastocystis genus. Blastocystis was detected in 13 species of animals from the Perth Zoo. It was also detected in NHPs from Belgian zoos. All wild and free-ranging NHP and Australian wildlife populations also harboured Blastocystis. This study describes the first reports of Blastocystis in the elephant, giraffe, Javan lutung, quokka, southern hairy nosed wombat and western grey kangaroo. Similarly, 12 Blastocystis STs, including six novel STs (STs 11 – 13 and 18 – 20), were identified in humans and animal hosts sampled as part of this study. Blastocystis STs 1, 2, 18 and 19 were identified in captive NHPs. However, STs 2, 8 and 20 were identified in wild NHPs. Australian native animals at the Perth Zoo harboured STs 1, 12 and 13, whereas free-ranging animals from Karakamia Sanctuary (KS) and wild animals from the Upper Warren Region (UWR) harboured STs 1 – 4 and 7. Captive elephants and giraffes from Australian and European zoos harboured STs 11 and 12, respectively. Higher prevalence of Blastocystis amongst zoo keepers and sequence homology of isolates from zoo keepers and animals at the Perth Zoo provide evidence of the zoonotic potential of Blastocystis. High prevalence amongst zoo keepers may be due to close contact between the animals and the zoo keepers, and other tasks carried out by the zoo keepers, such as cleaning of enclosures. Similarly, some Blastocystis isolates from Australian wildlife were also homologous to human isolates, and it seems that these hosts are natural hosts for the zoonotic ST 4. Other parasites, such as strongyle nematodes and coccidia were detected using microscopy. Various species of Australian wildlife are known to harbour these and other parasites, including zoonotic parasites, such as Cryptosporidium sp. and Giardia duodenalis. As polyparasitism is considered to be the norm in wildlife, a multiplex PCR (mPCR) was developed to detect Blastocystis, Cryptosporidium and Giardia simultaneously from Australian wildlife. This mPCR was evaluated against other diagnostic methods routinely used for the detection of these parasites, such as microscopy and nested PCRs. The multiplex PCR showed comparative and/or greater sensitivity and specificity to routinely utilised nested PCRs. The major advantages of the multiplex PCR are that it is less labour intensive and is cost effective in comparison to the nested PCRs used to amplify each parasite. In conclusion, the host range and genetic diversity of Blastocystis is much greater than previously anticipated. Some STs and/or subgroups of STs appear to be highly host specific, while others display moderate or low host specificity. Also, some STs which have a broad host range may be zoonotic. This study also provides further insight into polyparasitism amongst Australian wildlife.

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Andrés, Vergés Cristina. "Los Picornavirus. De la levedad a la gravedad." Doctoral thesis, Universitat Autònoma de Barcelona, 2019. http://hdl.handle.net/10803/669845.

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Minetti, Corrado. "The epidemiology and molecular epidemiology of Giardiasis in North West England." Thesis, University of Liverpool, 2014. http://livrepository.liverpool.ac.uk/2006698/.

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Giardiasis, cause by the parasitic protozoan Giardia duodenalis, is one of the most common infectious gastrointestinal diseases in humans worldwide. However, its true population burden and epidemiology and in particular its zoonotic transmission potential are still poorly understood. Furthermore, G. duodenalis is not a uniform parasite but a complex of seven genetic assemblages or cryptic species (named A to G) that infect humans and a variety of domesticated and wild animals, and that can only be distinguished using molecular genotyping methods. Although there is some evidence that the two Giardia assemblages infecting humans (namely A and B) may differ in their virulence and major transmission routes, data are still scarce. In the UK, several studies suggested that giardiasis is considerably under-diagnosed and a few data are available on the genetic diversity of the parasite causing infection and disease in this country. We investigated the burden, clinical outcomes, risk factors and molecular diversity of giardiasis in North West England using both a descriptive and analytical approach. In Chapter 2, we analysed the self-reported clinical and exposure data collected over four years from clinical cases of giardiasis in Central Lancashire, as part of an enhanced surveillance program on the illness. The resulting average disease rate of 22.5 cases/100,000 population was high when compared to the available national figures. Giardiasis was particularly abundant in adults in their 30s and children under five, and the disease rate in males was significantly higher than in females. Furthermore, the clinical picture of the cases confirmed the high morbidity associated with this infection particularly in terms of the length of illness and severity of symptoms. Only 32% of the cases reported foreign travel during the exposure window. The results suggested the presence of a hidden burden of disease in adults and males, and indicated that local transmission of Giardia can be more common than expected. In Chapter 3, we performed a case-control study to determine the significant risk factors for symptomatic giardiasis in North West England, by recruiting clinical cases of Giardia and age and sex matched controls from Central and East Lancashire and Greater Manchester. The multivariable logistic regression analysis done on 118 cases and 226 controls revealed that overall travelling abroad (particularly to developing countries) was an important risk factor for the illness (OR 9.59). Following the exclusion of participants that reported foreign travel, four risk factors were significant for the acquisition of giardiasis: going to a swimming pool (OR 2.67), changing nappies (OR 3.38), suffering irritable bowel syndrome (OR 3.66) and drinking un-boiled water from the tap (OR 8.17). The results indicated the important role of swimming pools and contact with children in nappies for the transmission of the parasite. In Chapter 4, whole faecal DNA was extracted from the faecal samples of the cases part of the surveillance and case-control studies and the Giardia assemblages and sub-assemblages causing infection were determined using PCR amplification and DNA sequencing of up to four parasite genes (beta-giardin, glutamate dehydrogenase, triose-phosphate isomerase and small-subunit ribosomal RNA). The majority of infections (64%) were caused by assemblage B, followed by assemblage A (33%), whereas mixed-assemblage infections were rare (3%). The majority of the assemblage A isolates belonged to the sub-assemblage AII and showed completed identity with previously described isolates, and six multi-locus genotypes were identified. The level of genetic sub-structuring as revealed by phylogenetic analysis was significantly higher in assemblage B isolates compared with A isolates: a higher proportion of novel assemblage B sequences was detected compared to what was observed in assemblage A isolates. A high number of assemblage B sequences showed heterogeneous nucleotide positions that prevented the unambiguous assignment to a specific sub-assemblage. Up to 17 different assemblage B multi-locus genotypes were found. The molecular genotyping results showed that Giardia assemblage B was responsible for the majority of the clinical infections and confirmed the occurrence of a high diversity of parasite multi-locus genotypes. In Chapter 5, we integrated the epidemiological and the molecular data generated by the enhanced surveillance and case-control studies and we studied the clinico-epidemiological differences between cases infected with Giardia assemblage A or B. Our results showed a difference in the age prevalence between the two assemblages, with assemblage A being more common in older cases. Cases infected with assemblage B reported a series of symptoms more frequently than cases infected with assemblage A, as well as reporting a longer illness. Although the exposure profile of the cases largely overlapped between the two assemblages, two different types of exposures were reported more frequently in the two groups of cases: keeping a dog in assemblage A cases and the presence in the household of children and children at nursery in assemblage B cases. The results suggested that assemblage A could have a major zoonotic reservoir, whereas assemblage B could be transmitted more commonly via the human-to-human route.

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Punyapornwithaya, Veerasak. "Molecular epidemiology of mycoplasma mastitis outbreak." Pullman, Wash. : Washington State University, 2010. http://www.dissertations.wsu.edu/Dissertations/Spring2010/v_punyapornwithaya_042110.pdf.

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Ragonnet, Manon Lily. "Molecular Epidemiology of HIV in Canada." Thèse, Université d'Ottawa / University of Ottawa, 2011. http://hdl.handle.net/10393/20215.

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With over 35 million people currently infected, the World Health Organization considers HIV a global pandemic. HIV is characterized by a high mutation rate, which allows it to evade the host immune system and develop resistance to drugs. However, this extraordinary adaptive ability may also be the key to HIV’s demise. Through the field of phylodynamics, the evolutionary behavior of the virus is being studied in an attempt to control the epidemic. In this thesis, three papers are presented in which we analyze sequences generated through the Canadian HIV Strain and Drug Resistance Surveillance program. In chapter 2 we validate a classifier which distinguishes between recent and established infections based on the proportion of mixed bases observed in population-based pol sequences. Our results will help identify recent infections and improve incidence calculations. In chapter 3, we investigate immune-induced patterns in HIV that are shared by patients of the same ethnicity. An understanding of the forces shaping HIV evolution is instrumental to the development of a vaccine relevant to the Canadian epidemic. In chapter 4, we present preliminary results of a historical reconstruction of HIV across the provinces of Canada. This analysis will highlight strategies that have succeeded or failed in controlling the epidemic. Furthermore, our work will establish whether non-B subtypes of HIV are an increasing threat to Canadian public health. Overall, this thesis provides the first country-wide evolutionary and phylogenetic analysis of the HIV epidemic.

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Tu, Elise Biotechnology &amp Biomolecular Sciences Faculty of Science UNSW. "Molecular epidemiology and detection of norovirus." Publisher:University of New South Wales. Biotechnology & Biomolecular Sciences, 2008. http://handle.unsw.edu.au/1959.4/41562.

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Norovirus (NoV) is a major cause of infectious human viral gastroenteritis. Detection is important to understanding the epidemiology of NoV and the viral dynamics of NoV infection which is poorly understood. In 2006, a marked increase in gastroenteritis outbreaks occurred worldwide. During this period, a total of 231 stool samples were obtained from patients with acute gastroenteritis from Australia and New Zealand. A total of 186 isolates of NoV were detected and sequenced to determine the genotype and relatedness to known epidemic NoV GII.4 variants. Two GII.4 variants, 2006a and 2006b, were identified in 61.8% and 11.3%, in these cases, respectively. Thus, the increase in NoV gastroenteritis in 2006 was linked to the emergence of two novel co-circulating GII.4 variants, 2006a and 2006b. During an outbreak in an aged-care facility, stool samples were collected from the onset of illness to cessation of viral excretion. Here, viral shedding peaked in the acute stage of illness and continued for an average of 28.7 days. The viral decay rate was 0.76 per day. Prolonged asymptomatic shedding of NoV was detected in the elderly. A quality control for the assessment of molecular based viral assays for NoV and other RNA viruses is necessary to meet current testing requirements. Available controls only monitor the RNA and DNA amplification steps. An MS2 bacteriophage BioBallTM with 100 pfu was evaluated and applied as a multi-purpose phage control. It was assessed as a quality control, in comparison to MS2 phage stock, to validate MS2 phage assays. Furthermore, MS2 BioBallTM was used as a process control for the molecular detection of RNA viruses. It validated every performed step, determined if the assay worked and its sensitivity. Thus, MS2 BioBallTM offered uniformity, stability and reproducibility across molecular based viral detection systems. Overall, this thesis provided valuable insight into the molecular epidemiology of NoV in the southern hemisphere and nature of NoV infections in the elderly. The MS2 BioBallTM provides standardisation and quality control of viral RNA assays. Understanding the genetic diversity and viral dynamics of NoV will be crucial to developing effective intervention and treatment strategies, and ultimately lead to reduced viral gastroenteritis worldwide.

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Books on the topic "Molecular epidemiology"

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A, Thompson R. C., ed. Molecular epidemiology of infectious diseases. London: Arnold, 2000.

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1959-, Wild Chris, Vineis Paolo, and Garte Seymour J, eds. Molecular epidemiology of chronic diseases. Chichester, West Sussex, England: J. Wiley, 2008.

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Caugant, Dominique A., ed. Molecular Epidemiology of Microorganisms. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-999-4.

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A, Schulte Paul, and Perera Frederica P, eds. Molecular epidemiology: Principles and practice. London: Academic, 1998.

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Riley, Lee W. Molecular Epidemiology of Infectious Diseases. Washington, DC, USA: ASM Press, 2004. http://dx.doi.org/10.1128/9781555817688.

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Wild, Chris, Paolo Vineis, and Seymour Garte, eds. Molecular Epidemiology of Chronic Diseases. Chichester, UK: John Wiley & Sons, Ltd, 2008. http://dx.doi.org/10.1002/9780470725726.

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A, Schulte Paul, and Perera Frederica P, eds. Molecular epidemiology: Principles and practices. San Diego: Academic Press, 1993.

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Mata-Essayag, Sofia. The molecular epidemiology of candida. Manchester: University of Manchester, 1996.

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Mary, Carrington, and Hoelzel A. Rus, eds. Molecular epidemiology: A practical approach. New York: Oxford University Press, 2001.

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S, Hulka Barbara, Wilcosky Timothy C, and Griffith Jack D, eds. Biological markers in epidemiology. New York: Oxford University Press, 1990.

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Book chapters on the topic "Molecular epidemiology"

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Modrow, Susanne, Dietrich Falke, Uwe Truyen, and Hermann Schätzl. "Epidemiology." In Molecular Virology, 147–54. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-20718-1_11.

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Shields, P. G., A. Weston, H. Sugimura, E. D. Bowman, N. E. Caporaso, D. K. Manchester, G. E. Trivers, et al. "Molecular Epidemiology." In ACS Symposium Series, 186–206. Washington, DC: American Chemical Society, 1990. http://dx.doi.org/10.1021/bk-1990-0451.ch017.

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Torres, Salina M., Esther Erdei, Marianne Berwick, Giuseppe Matullo, and Paolo Vineis. "Molecular Epidemiology." In Handbook of Epidemiology, 1779–811. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-0-387-09834-0_28.

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Riley, Lee W. "Molecular Epidemiology." In Bacterial Infections of Humans, 69–89. Boston, MA: Springer US, 2009. http://dx.doi.org/10.1007/978-0-387-09843-2_3.

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Seitz, Amy E., and D. Rebecca Prevots. "Molecular Epidemiology." In Molecular Typing in Bacterial Infections, 3–13. Totowa, NJ: Humana Press, 2012. http://dx.doi.org/10.1007/978-1-62703-185-1_1.

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Trees, Eija, Paul A. Rota, Duncan MacCannell, and Peter Gerner-Smidt. "Molecular Epidemiology." In Manual of Clinical Microbiology, 131–60. Washington, DC, USA: ASM Press, 2015. http://dx.doi.org/10.1128/9781555817381.ch10.

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Cui, Miao, Fei Ye, and David Y. Zhang. "Molecular Microbiology Epidemiology." In Molecular Genetic Pathology, 813–22. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-4800-6_29.

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Breuer, Judith. "VZV Molecular Epidemiology." In Current Topics in Microbiology and Immunology, 15–42. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/82_2010_9.

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O’Malley, Padraic. "Epidemiology and Etiology." In Molecular Pathology Library, 13–26. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-64096-9_2.

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Smith, S. I., A. Seriki, and A. Ajayi. "Molecular Epidemiology of Salmonella." In Molecular Food Microbiology, 229–39. 3rd ed. First edition. | Boca Raton : Taylor & Francis, 2021. |: CRC Press, 2021. http://dx.doi.org/10.1201/9781351120388-16.

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Conference papers on the topic "Molecular epidemiology"

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Gonzalez, Frank J., and Harry V. Gelboin. "Cytochrome P450s and molecular epidemiology." In Environmental Sensing '92, edited by Tuan Vo-Dinh and Karl Cammann. SPIE, 1993. http://dx.doi.org/10.1117/12.140241.

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Khudyakov, Yury, Ion Mandoiu, Pavel Skums, and Alexander Zelikovsky. "Workshop on Computational Advances in Molecular Epidemiology." In BCB '19: 10th ACM International Conference on Bioinformatics, Computational Biology and Health Informatics. New York, NY, USA: ACM, 2019. http://dx.doi.org/10.1145/3307339.3343859.

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Schildkraut, Joellen M. "Abstract IA31: Molecular epidemiology of ovarian cancer." In Abstracts: Eighth AACR Conference on The Science of Health Disparities in Racial/Ethnic Minorities and the Medically Underserved; November 13-16, 2015; Atlanta, Georgia. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7755.disp15-ia31.

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Divi, Rao L., Mukesh Verma, and Anthony Dickherber. "Abstract 5571: Technologies for molecular epidemiology in cancer." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-5571.

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Farzan, A., R. Friendship, C. Poppe, L. Martin, C. Dewey, J. Gray, and Julie A. Funk. "Molecular epidemiology of Salmonella Typhimurium DT104 on Ontario swine farms." In Seventh International Symposium on the Epidemiology and Control of Foodborne Pathogens in Pork. Iowa State University, Digital Press, 2007. http://dx.doi.org/10.31274/safepork-180809-131.

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Chekanova, T. A., V. G. Akimkin, A. V. Kalenskaya, T. V. Tyrgina, and E. V. Tivanova. "FORMATION OF POPULATION IMMUNITY TO SARS-CoV-2 IN THE MOSCOW REGION." In Molecular Diagnostics and Biosafety. Federal Budget Institute of Science 'Central Research Institute for Epidemiology', 2020. http://dx.doi.org/10.36233/978-5-9900432-9-9-180.

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49 813 sera from persons without clinical symptoms of infectious diseases were tested for IgG to the nucleocapsid protein of SARS-CoV-2 at the Center for Molecular Diagnostics (Central Research Institute of Epidemiology) from May 22 to August 28, 2020. The dynamics of the incidence of seropositive persons was assessed by decades. The maximum seroprevalence was noted at the end of May. Seroprevalence indicators changed slightly from June 21 to August 28 (maximum values, about 17%, were noted from July 1 to July 20), which was consistent with the slowdown in the number of new cases of COVID-19 in the Moscow region.

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McCarthy, G., G. E. Gardiner, P. G. Lawlor, and M. Gutierrez. "Salmonella in Irish pig farms; prevalence, antibiotic resistance and molecular epidemiology." In Ninth International Conference on the Epidemiology and Control of Biological, Chemical and Physical Hazards in Pigs and Pork. Iowa State University, Digital Press, 2011. http://dx.doi.org/10.31274/safepork-180809-651.

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Chatterjee, SG, JS Kammerer, LS Cowan, TR Navin, and PK Moonan. "Molecular Epidemiology of the Beijing Genotype Family in the United States." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a2198.

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Suwanpimolkul, Gompol, Leah G. Jarlsberg, Dennis Osmond, Jennifer Grinsdale, Masae Kawamura, Philip C. Hopewell, and Midori Kato-Maeda. "Molecular Epidemiology Of Tuberculosis In Foreign-Born Persons In San Francisco." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a3250.

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Jerbi, Amira, Imene Fodha, Meriam Ben Hmida, Haifa Bennour, Imene Ataoui, Mouna Ben Hadj Fredj, and Abdelhalim Trabelsi. "Molecular epidemiology of respiratory syncytial virus in hospitalized children in Tunisia." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa1051.

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Reports on the topic "Molecular epidemiology"

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Trock, Bruce J. Molecular Epidemiology of Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, January 2005. http://dx.doi.org/10.21236/ada439125.

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Bowtell, David, Adele Green, Georgia Chenevix-Trench, Anna DeFazio, Dorota Gertig, David Purdie, Penelope Webb, and David Whiteman. Molecular Epidemiology of Ovarian Cancer. Fort Belvoir, VA: Defense Technical Information Center, September 2005. http://dx.doi.org/10.21236/ada444073.

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Bowtell, David, Adele Green, Georgia Chenevix-Trench, Anna deFazio, and Dorota Gertig. Molecular Epidemiology of Ovarian Cancer. Fort Belvoir, VA: Defense Technical Information Center, September 2002. http://dx.doi.org/10.21236/ada409447.

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Bowtell, David, Adele Green, Georgia Chenevix-Trench, Anna DeFazio, and Dorota Gertig. Molecular Epidemiology of Ovarian Cancer. Fort Belvoir, VA: Defense Technical Information Center, September 2004. http://dx.doi.org/10.21236/ada430282.

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Gabrielson, Edward W. Molecular Epidemiology of Breast Cancer in Korean Women. Fort Belvoir, VA: Defense Technical Information Center, August 2005. http://dx.doi.org/10.21236/ada443764.

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Gabrielson, Edward W. Molecular Epidemiology of Breast Cancer in Korean Women. Fort Belvoir, VA: Defense Technical Information Center, August 2002. http://dx.doi.org/10.21236/ada408692.

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Witzmann, Frank A. Protein Markers of Chemical Exposure and Molecular Epidemiology. Fort Belvoir, VA: Defense Technical Information Center, July 1999. http://dx.doi.org/10.21236/ada371936.

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Amon, Joseph J. The Molecular Epidemiology of Malaria in Western Kenya. Fort Belvoir, VA: Defense Technical Information Center, August 2002. http://dx.doi.org/10.21236/ad1012460.

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Erdman, Dean D., Wanhong Xu, Susan I. Gerber, Gregory C. Gray, and David Schnurr. Molecular Epidemiology of Adenovirus Type 7 in the United States, 1966-2000. Fort Belvoir, VA: Defense Technical Information Center, March 2002. http://dx.doi.org/10.21236/ada408854.

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Moysich, Kirsten, and Jo L. Freudenheim. The Molecular Epidemiology of Breast Cancer: Risk From Environmental Exposures and Genetic Susceptibility. Fort Belvoir, VA: Defense Technical Information Center, September 1997. http://dx.doi.org/10.21236/ada337489.

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