Relevant bibliographies by topics / Minamata disease

Academic literature on the topic 'Minamata disease'

Author: Grafiati
Published: 10 December 2022
Last updated: 29 January 2023

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Journal articles on the topic "Minamata disease"

1

MURATA, Katsuyuki. "Minamata Disease." TRENDS IN THE SCIENCES 24, no. 10 (October 1, 2019): 10_14–10_17. http://dx.doi.org/10.5363/tits.24.10_14.

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Taylor, D. "Minamata disease." Environmental Science & Technology 29, no. 2 (February 1995): 81A. http://dx.doi.org/10.1021/es00002a701.

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Eto, Komyo. "Minamata disease." Neuropathology 20, s1 (September 2000): 14–19. http://dx.doi.org/10.1046/j.1440-1789.2000.00295.x.

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Semionov, Alexandre. "Minamata Disease—Review." World Journal of Neuroscience 08, no. 02 (2018): 178–84. http://dx.doi.org/10.4236/wjns.2018.82016.

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Furuta, Shigeru, Kengo Nishimoto, Masahiko Egawa, Masaru Ohyama, and Hiroyuki Moriyama. "Olfactory Dysfunction in Patients with Minamata Disease." American Journal of Rhinology 8, no. 5 (September 1994): 259–64. http://dx.doi.org/10.2500/105065894782537280.

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We studied the olfactory functions of organic mercury poisoning (Minamata disease) by using both the smell identification test and the olfactory detection threshold test (with phenyl ethyl alcohol). The subjects were 19 patients with Minamata disease who were treated in Meisuien, Minamata, Kumamoto, Japan and include cases that developed the disease in utero. The mean age was 78.7 ± 14.3 years old. Both smell identification and olfactory detection tests in the majority of patients decreased significantly compared with those of healthy elder subjects. A few cases showed normal olfactory identification and detection functions. The olfactory identification function deceased with advancing age. Correlation existed between detection threshold and background factors such as age and duration after documented Minamata disease was found. No significant relationship was identified between olfactory function and the typical symptoms of Minamata disease.
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Takaoka, Shigeru. "Minamata Disease at Present." Epidemiology 22 (January 2011): S100. http://dx.doi.org/10.1097/01.ede.0000391973.26704.bc.

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7

Ishihara, Nobuo. "Reconsideration of Minamata disease." Trace Elements and Electrolytes 31, no. 04 (April 1, 2014): 86–88. http://dx.doi.org/10.5414/tex01331.

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Smoczyński, Kamil. "Portrety choroby z Minamaty i Niigaty w japońskim filmie dokumentalnym." Studia Filmoznawcze 42 (August 1, 2022): 87–102. http://dx.doi.org/10.19195/0860-116x.42.6.

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The article aims to introduce the history of Japanese documentaries that show the diseases of Minamata and Niigata. To do this, the author initially provides basic information about the origins and symptoms of these diseases. Then the figure of Noriaki Tsuchimoto, a pioneer of portraying Minamata in film, is introduced. The productions Minamata: The Victims and Their World (Minamata: Kanja-san to sono sekai, Japan, 1971) and Shiranui Sea (Shiranuikai, Japan, 1975) are described, which are distinguished by their insightful look into the lives of the people affected by the disease. Another director described in the article is Makoto Satō, whose method the author describes as creative volunteering. Life on the Agano River (Aga ni ikiru, Japan, 1992) focuses on the daily routines of Niigata inhabitants, whose lives were greatly influenced by Satō. Then he created Memories of the Agano River (Aga no kioku, Japan, 2005), which is a documentary experiment with the camera as a medium of memory. The last depicted director is Kazuo Kara, whose Minamata Mandala (Japan, 2020) differs significantly from his earlier works. This production, as stated by the author of the article, aspires to be the last one about the Minamata disease. However, as long as the last patients are fighting for their dignity, the story is not finished.
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Takizawa, Y. "Understanding minamata disease and strategies to prevent further environmental contamination by methylmercury." Water Science and Technology 42, no. 7-8 (October 1, 2000): 139–46. http://dx.doi.org/10.2166/wst.2000.0562.

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Minamata disease is a neurological disorder caused by methylmercury poisoning which originated from the discharge of wastewater containing methylmercury from chemical plants in Japan. Residents in the area who consumed large amounts of fish and other seafoods suffered from the disease. The main symptoms consist of sensory disturbance, ataxia, restriction of visual field and hearing impairment. Various measures have been taken to deal with Minamata disease, including environmental pollution control, treatment for patients, and promotion of research activities. Through the compensation law, 2,952 persons have been certified as Minamata disease patients, and a total of approximately 144 billion yen had been paid in compensation by the responsible companies as of March, 1999. Meanwhile, people who were not certified as patients have filed suits against the Japanese Government and local government challenging the diagnostic criteria. The Japanese Government, patients and their supporters reached an agreement in 1996, and Minamata disease legal issues were finally resolved, 40 years after the outbreak. The Minamata experience has left us with an invaluable understanding of the importance of taking thoroughgoing measures to prevent health damage from environmental pollution.
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ISHIHARA, Nobuo. "Bibliographical Study of Minamata Disease." Nippon Eiseigaku Zasshi (Japanese Journal of Hygiene) 56, no. 4 (2002): 649–54. http://dx.doi.org/10.1265/jjh.56.649.

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Dissertations / Theses on the topic "Minamata disease"

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Gilbertson, Michael. "Injury to health : a forensic audit of the Great Lakes Water Quality Agreement (1972-2005) with special reference to congenital Minamata disease." Thesis, University of Stirling, 2006. http://hdl.handle.net/1893/249.

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The objective of this research was to examine whether the United States and Canada have successfully implemented their Great Lakes Water Quality Agreement and to identify the factors determining the continuation of any injury to human health from pollution of the boundary waters. The Agreement was first negotiated in 1972 as part of the legitimation of the social unrest of the 1960s and gave special responsibilities to the International Joint Commission to advise the Parties of problems of water quality. It has been subject to periodic review and occasional renegotiation and amendment. Specifically, the Agreement was renegotiated in 1978 to address the health effects from the imperceptible exposures to persistent toxic substances. Though extensive scientific evidence of continuing injury to health from persistent toxic substances has been available, there has been a consistent pattern of deliberate failure by the authorities to report the injury and to implement many of the remedial provisions contained in the Agreement. The thesis claims that the failure of the International Joint Commission to advise the Parties of the new information about the injury to health and the failure of the Parties to act upon the information when it was obtained from other sources constituted dereliction of duty. While synthesis of the science linking the pollutant-induced injury to specific causal agents was necessary to provide an empirical measure of the failure to implement the Agreement, consideration of the social, economic and political aspects was needed to provide a sufficient explanation for the failure of the International Joint Commission to inform and of the authorities to act. There have been active attempts to use diversionary reframing of the Agreement, based on a multi-causal ecosystem theory proposed by fisheries ecologists, to attenuate the risk message and transform the Great Lakes Water Quality Agreement into a more inclusive and less focused agreement on restoring ecosystem integrity. This has been welcomed by industry and governments as a means to remove the focus from addressing the unresolved dangers of persistent toxic substances through costly remedial actions. The International Joint Commission undermined its credibility when it recommended ‘sunsetting’ the use of chlorine in chemical manufacturing. The Parties failed to use a precautionary approach to prevent the commercial introduction of new persistent toxic substances, such as the brominated flame retardants. Since the 1980s, the economic politics of the two nations have been profoundly influenced by neo-liberalism and one of the consequences has been the removal of environmental health as a priority from the respective political agenda. Advisory bodies seem to have been captured not only by the prevailing neo-liberalism but also by corporate interests and these factors seem to underlie the reluctance to report the injury to health from exposures to persistent toxic substances. Though there were many different health endpoints affected by exposures to water pollutants in the Great Lakes, the thesis concentrated on the evidence of neuro-teratogenic effects. The adequacy of the implementation of the Agreement during the past thirty-three years was tested by using Health Canada data on cerebral palsy hospitalisation to evaluate whether there were indications of previously undetected outbreaks of congenital Minamata disease in human populations in Canadian Great Lakes communities potentially exposed to methyl mercury from natural sources or from historic industrial uses of mercury. The uncertainties in the apparent association that was found were reduced by the application of Hill’s guidelines. While these findings indicated both the need for further multi-disciplinary research to locate and diagnose the victims and for a precautionary approach to the consumption of Great Lakes fish, they also indicated that, for more than three decades, health authorities have not diligently implemented the Agreement. The inclusion of the social, economic and political considerations in the forensic audit has revealed the dangers inherent in any renegotiation of the Great Lakes Water Quality Agreement.
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Books on the topic "Minamata disease"

1

Ishimure, Michiko. Paradise in thesea of sorrow: Our Minamata disease. (Kyoto): Yamaguchi Publishing House, 1990.

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2

Mishima, Akio. Bitter sea: The human cost of Minamata disease. Tokyo: Kosei Pub. Co., 1992.

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3

Seisakukyoku, Japan Kankyōshō Sōgō Kankyō. Lessons from Minamata disease and mercury management in Japan. Tokyo: Environmental Health and Safety Division Environmental Health Department Ministry of the Environment, 2013.

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4

Livia, Monnet, ed. Paradise in the sea of sorrow: Our Minamata disease. [Kyoto]: Yamaguchi Pub. House, 1990.

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5

Livia, Monnet, ed. Paradise in the sea of sorrow: Our Minamata disease. Ann Arbor, Mich: Center for Japanese Studies, University of Michigan, 2003.

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6

Niigata minamatabyō mondai: Kagai to higai no shakaigaku = Environmental sociology of minamata disease. Tōkyō: Tōshindō, 2006.

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7

Niigata Minamatabyō mondai no juyō to kokufuku: Niigata Minamata : sociolgical study of the sufferes from Niigata Minamata disease, processes of accepting and overcoming the problem. Tōkyō: Tōshindō, 2002.

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8

Takeuchi, Tadao. The pathology of Minamata disease: A tragic story of water pollution / Tadao Takeuchi and Komyo Eto, with editorial collaboration of H. Nakayama and A. Sumiyoshi. Fukuoka, Japan: Kyushu University Press, Inc., 1999.

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9

Minamata jiken: Kuwabara Shisei shashinshū = The Minamata disaster : documentary photographer Kuwabara Shisei. Tōkyō-to Shinjuku-ku: Fujiwara Shoten, 2013.

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10

Tsurumi, Kazuko. New lives: Some case studies in Minamata. Tokyo, Japan: Sophia University, 1988.

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Book chapters on the topic "Minamata disease"

1

Murata, Katsuyuki, and Kanae Karita. "Minamata Disease." In Current Topics in Environmental Health and Preventive Medicine, 9–19. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-16-6249-2_2.

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2

Nabi, Shabnum. "Methylmercury and Minamata Disease." In Toxic Effects of Mercury, 187–99. New Delhi: Springer India, 2014. http://dx.doi.org/10.1007/978-81-322-1922-4_25.

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3

Hachiya, N. "Minamata disease and methylmercury exposure." In A handbook of environmental toxicology: human disorders and ecotoxicology, 353–70. Wallingford: CABI, 2020. http://dx.doi.org/10.1079/9781786394675.0353.

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4

Igata, Akihiro. "Epidemiological and Clinical Features of Minamata Disease." In Advances in Mercury Toxicology, 439–57. Boston, MA: Springer US, 1991. http://dx.doi.org/10.1007/978-1-4757-9071-9_28.

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5

Hachiya, Noriyuki. "Epidemiological Update of Methylmercury and Minamata Disease." In Methylmercury and Neurotoxicity, 1–11. Boston, MA: Springer US, 2012. http://dx.doi.org/10.1007/978-1-4614-2383-6_1.

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6

Sugiyama, Shigeo. "Minamata Disease: Interaction Between Government, Scientists, and Media." In Lessons From Fukushima, 125–40. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-15353-7_6.

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7

Matsuda, Hiroyuki, and Sin’ya Ueno. "How to Determine the Relief Target for Minamata Disease." In Ecological Risk Management, 13–29. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-33-6934-4_2.

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Otsuka, Tadashi. "Lessons of Court Decisions on Minamata Disease and Future Actions." In Current Topics in Environmental Health and Preventive Medicine, 121–26. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-16-6249-2_15.

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9

Onuki, Motoharu. "Role in Framing in Sustainability Science — The Case of Minamata Disease." In Science for Sustainable Societies, 119–31. Singapore: Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-9061-6_6.

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10

Okuda, T., Y. Korogi, M. Takahashi, Y. Sakamoto, T. Hirai, S. Hamatake, R. Murakami, and T. Okajima. "MRI in Minamata disease (organic mercury poisoning): correlation with visual field abnormalities." In Proceedings of the XV Symposium Neuroradiologicum, 206–7. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-642-79434-6_99.

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