Books: 'Mildura' – Grafiati

1

Paddle steamers to cornucopia: The Renmark-Mildura experiment of 1887. Renmark [S. Aust.]: Murray Pioneer, 1986.

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2

ASVO Viticulture Seminar (1999 Tanunda, S. Aust., etc.). Modern viticulture: Meeting market specifications : Barossa Convention Centre, Tanunda, 15 July 1999, Cowra Civic Centre, 25 August 1999, Mildura Arts Centre, Mildura, 3 September 1999, Technology Park Function Centre, Perth, 16 September 1999. Adelaide: Australian Society of Viticulture and Oenology, 2000.

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3

Cane, Georgina. Telecommunications support for professional development programs for rural library networks: Report of a study undertaken for Mildura Rural City Council Library Service & Office of Local Government. Victoria, Australia: Office of Local Government, Dept. of Infrastructure, 1998.

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4

Paasilinna, Arto. Milda makter. Stockholm: Brombergs, 2008.

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5

Maines, Barbara. Punishment: The milder the better. Marlborough: Lame Duck Enterprises, 1991.

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6

Dietlein, Damian L. Christian Dietlein & Milda Stemmler descendants. [Richardton, ND (Assumption Abbey, P.O. Box A, Richardton 58652)]: D.L. Dietlein, 1991.

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7

McCartney, James. The milder day, or, a promise kept. Carrickfergus: Macson Books, 1994.

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8

Granath, Gunilla. Milda makter!: Utvecklingssamtal och loggböcker som disciplineringstekniker. Göteborg: Göteborgs universitet, 2008.

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9

Alan, Marshall. People in pieces: Multiple personality in milder forms & greater numbers. Highland City, Fla: Rainbow Books, 1993.

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10

Lee, Stewart. Stewart Lee!: The 'If you prefer a milder comedian, please ask for one' EP. London: Faber, 2012.

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11

Varley, E. A. The last of the prince bishops: William Van Mildert and the High Church movement of the early nineteenth century. Cambridge [England]: Cambridge University Press, 1992.

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12

Burger, Klaus. "Denen von milder Gunst des Schicksals kein wohlgeordnetes Familienleben vergönnt war": Kleinkinderbewahranstalten und Kindergärten in Freiburg 1848-1945. Freiburg im Breisgau: Lambertus, 1998.

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13

Programme national de lutte contre le paludisme (Cameroon) and National Institute of Statistics (Cameroon), eds. Enquête post-campagne sur l'utilisation des moustiquaires imprégnées à longue durée d'action: MILDA 2013 : rapport principal. Yaoundé: Ministère de la santé publique, Programme national de lutte contre le paludisme, 2013.

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14

Psychologische Psychotherapie nach einem "Schleudertrauma": Theorie und Praxis : eine Falldarstellung zum besseren Verständnis des therapeutischen Geschehens nach milder traumatischer Hirnschädigung. Frankfurt am Main: P. Lang, 1999.

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15

"A Prison with a milder name": The Shipmeadow House of Industry 1766-1800 : an account based on the minutes of weekly and quarterly meetings of the directors and guardians. (Beccles): (E.A. Goodwyn), 1987.

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16

Goodwyn, E. A. "A prison with a milder name": The Shipmeadow House of Industry, 1766-1800 : an account based on the minutes of weekly and quarterly meetings of the directors and guardians. [Beccles]: [s.n.], 1987.

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17

Agar, Jason. Walking on water?: Some recommendations on the role of the Personal Business Adviser : a paper presented at the one day workshop on the role and characteristics of the Personal Business Advisers in the emerging Business Links, held at Van Mildert College, Durham, 28 July 1994. Durham: Durham University Business School, 1994.

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18

Parsons, Ronald. Where The Mallee Meets The Murray : A Centenary History Of The Shire Of Mildura. Mildura Shire Council, 1990.

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19

Taylor, Jane. The Milder Garden. J.M.Dent & Sons Ltd, 1990.

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20

Modick, Klaus. Milder Rausch. Essays und Portraits. Eichborn, 2000.

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21

Arnold-Forster, W. Shrubs for the Milder Counties. Alison Hodge, 2000.

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22

Morse, Milda Aileen. Milda Has No Regrets:: 81 Years and Counting... PublishAmerica, 2007.

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23

Varley, Elizabeth. Last of the Prince Bishops, Episcopate of William Van Mildert, 1826-36. Durham,Dean & Chapter of, 1986.

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24

Chavez-Badiola, Alejandro, and N. Allahbadia Gautam. Textbook of Minimal Stimulation IVF � Milder, Mildest or Back to Nature. Jaypee Brothers Medical Publishers (P) Ltd, 2011. http://dx.doi.org/10.5005/jp/books/12251.

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25

Varley, E. A. The Last of the Prince Bishops: William Van Mildert and the High Church Movement of the Early Nineteenth Century. Cambridge University Press, 2002.

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26

Hilary, Constable, Brown Ron 1934-, Sunderland Polytechnic, British Educational Research Associates, Strengthening the Inquiry Networks (Conference), Researching In-Service Education (Seminar), and Supporting the Inquiry Network (Conference), eds. Researching inset: A selection of papers from the B.E.R.A. 'Supporting the Inquiry Network' Conference at Van Mildert College, Durham, September 1988. [s.l.]: [s.n.], 1998.

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27

The Worcks of the Daniel W. Mildert. Worcks of the Rev. Daniel Waterland... to Which Is Prefixed a Review of the Authors Life and Writings, by William Van Mildert. HardPress, 2020.

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28

Autism: Perspectives on progress : a collection of papers from the conference held at Van Mildert College, University of Durham, April 5th - 7th, 2000. The Autism Research Unit and Autism North Ltd, 2000.

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29

University of Sunderland. Autism Research Unit., ed. From research into therapy: A collection of papers from the conference held at Van Mildert College, University of Durham, April 7th-9th, 1999. Sunderland: Autism Research Unit, University of Sunderland, 1999.

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30

Brusse, Esther, Pascal Laforêt, and Ans T. van der Ploeg. Danon Disease. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0056.

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Danon disease, like Pompe disease, is a muscle disorder caused by a primary defect in lysosomal proteins. Danon disease (OMIM #300257) is an X-linked dominant disorder, with males being more severely affected than female carriers. In males, mean disease onset is in their early teens and in females in their late twenties. Clinical hallmarks are a severe cardiomyopathy, muscle weakness, and mild mental retardation. Retinal, liver, and pulmonary disease may also occur. Milder, sometimes isolated cardiac phenotypes without mental retardation are also described. Regular cardiac evaluation, even in asymptomatic patients, is obligatory.

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31

Psychobiology of autism: Current research and practice : a collection of papers from the conference held at Van Mildert College, University of Durham, April 15th-17th, 1998. [Sunderland]: Autism Research Unit and Autism North Ltd, 1998.

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32

Ghaemi, S. Nassir. Understanding Mania and Depression. Edited by K. W. M. Fulford, Martin Davies, Richard G. T. Gipps, George Graham, John Z. Sadler, Giovanni Stanghellini, and Tim Thornton. Oxford University Press, 2013. http://dx.doi.org/10.1093/oxfordhb/9780199579563.013.0047.

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Mania is a mental state centrally characterized by psychomotor activation, creativity, and lack of insight. It is biologically based, and primarily characterized by psychomotor activation, with mood changes being secondary and epiphenomenal. Mania may be diagnostically central to all mood illnesses, including depressive conditions. Mania enhances creativity, and in its milder forms, such as cyclothymic or hyperthymic temperaments, has many positive aspects, reflected in social benefits and historical achievements. Lack of insight impairs the self-understanding of the person with manic symptoms, and is an important clinical problem. Psychoanalytic and postmodernist approaches fail to appreciate these important features of mania.

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33

Brugha, Traolach S. Awareness of autism in adulthood. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198796343.003.0002.

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In its milder forms, autism is widespread, but often not considered as an explanation for odd, egocentric, rigid, and/or potentially socially problematic behaviour. The reader is encouraged in this chapter to consider how autism might present in the world in which (s)he works and lives. A series of case vignettes are then used (in this chapter), drawn from ordinary life settings (work, college, neighbourhood, family, etc.) to illustrate and inform awareness, and possible consideration of autism that has been missed earlier in the person’s life. Questions are posed following each case vignette that the reader is asked to try to answer. Care recommendations then follow.

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34

Linnebo, Øystein. On the Permissibility of Impredicative Comprehension. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198792161.003.0009.

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Which comprehension axioms of higher-order logic are acceptable? That is, under what conditions does a formula define a concept or circumscribe some objects? It is well known that unrestricted higher-order comprehension is incompatible with unrestricted reification of higher-order entities. In search of a response to this conflict, an argument against all forms of impredicative comprehension is formulated; for example, when defining a concept, we may not quantify over a totality to which this concept belongs. Although this predicativist argument is ultimately rejected, a careful analysis of it points the way to some milder logical restrictions, which suffice to resolve the conflict.

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35

Conference, Remote Sensing Society. Remote sensing and national mapping: Proceedings of the Fifth Annual Conference of the Remote Sensing Society, held at Van Mildert College, University of Durham, 18th-20th December, 1978. Remote Sensing Society, 1988.

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36

Fox, Susan H. Could It Possibly Be … ? Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190607555.003.0027.

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There is increasing recognition of adult presentations of metabolic diseases typically associated with children. These phenotypes are often milder and potentially mistaken for more common adult-onset disorders, therefore requiring a high index of suspicion. The most common adult-onset clinical features involve focal dystonias and parkinsonism. Making a diagnosis of dopa-responsive dystonia in an adult typically involves a therapeutic trial of low-dose levodopa. Genetic testing may be useful to confirm dopa-responsive dystonia and rule out other causes of dystonia and tremor. The neurological examination should be performed carefully to ensure that subtle dystonia is not missed. A trial of levodopa may be warranted to ensure that late-onset dopa-responsive dystonia is not missed.

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37

Walsh, Richard A. Always Worth a Second Look. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190607555.003.0031.

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The serotonin syndrome is a toxic syndrome resulting from excessive stimulation of central 5-HT1A and 5-HT2A receptors. This is most commonly an iatrogenic syndrome, which in its most severe form can be fatal. It is more common for milder forms to present, and there is increasing recognition of serotoninergic drugs that can give rise to serotonin syndrome when used in combination. It is essential for physicians to be familiar with the clinical features of serotonin toxicity and similar syndromes discussed in this chapter that are marked by altered awareness, autonomic instability, changes in muscle tone, and pyrexia. Withdrawal of the drug(s) believed to be responsible and supportive care are the primary therapeutic steps.

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38

Shorter, Edward, and Max Fink. Eugen Bleuler. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190881191.003.0005.

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Eugen Bleuler, professor of psychiatry in Zurich, renamed Kraepelin’s dementia praecox as “schizophrenia” in 1908. He retained catatonia as a subtype. Bleuler’s dementia praecox was a much milder and broader condition than the downhill course toward dementia that Kraepelin had described: it could strike at any moment in life, not just in youth, and often ended in partial recovery. Ultimately, Kraepelin’s and Bleuler’s efforts gave rise to an immense “schizophrenia” literature, an industry that continues today, an outpouring that can be compared only to the enormous “hysteria” industry that existed before DSM-III abolished the diagnosis in 1980. The effect is striking: one moment the profession believes implicitly in a disease so huge as to dominate the literature; the next moment the disease no longer exists.

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39

Peabody, Sue. The Revolution. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190233884.003.0005.

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Slave labor in eighteenth-century Isle Bourbon was shaped by the cultivation of staple crops, unlike the proto-industrial forms of labor found in the sugar plantations of the Atlantic world, and may have been milder, though periodic cyclones brought famine to slaves and their masters alike. On the eve of the French Revolution, following the death of Charles Routier, Madeleine’s mistress filed manumission papers, freeing her. As a result of the slave revolt in Saint-Domingue (Haiti), France issued the 1794 Decree of 16 Pluviôse abolishing slavery throughout the colonies. Although Madeleine should have been considered free twice over, the widow Routier declared Madeleine her slave on her 1796 census, a moment when Madeleine—like many free people of color in France’s empire—faced potential or actual re-enslavement.

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40

Murphy, Elaine, Yann Nadjar, and Christine Vianey-Saban. Fatty Acid Oxidation, Electron Transfer and Riboflavin Metabolism Defects. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0008.

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The fatty acid oxidation disorders are a group of autosomally recessively inherited disorders of energy metabolism that may present with life-threatening hypoketotic hypoglycemia, encephalopathy and hepatic dysfunction, muscle symptoms, and/or cardiomyopathy. Milder phenotypes may present in adulthood, causing exercise intolerance, episodic rhabdomyolysis, and neuropathy. Specific investigations include acylcarnitine profiling, urine organic acid analysis, fibroblast or leucocyte studies of fatty acid oxidation flux/enzyme activity, and genetic testing. Management varies depending on the condition but includes avoidance of precipitants such as fasting, fever, and intense exercise, a high-carbohydrate, low-fat diet, and supplementation with carnitine or riboflavin. Inborn errors of riboflavin transport mainly present with Brown-Vialetto-Van Laere syndrome. Some patients respond dramatically to riboflavin supplementation; therefore it has to be tried in all suspected patients.

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41

Oladeji, Bibilola D., and Kevin R. Robertson. Neuropsychological Evaluation for Persons with HIV and AIDS. Edited by Mary Ann Cohen, Jack M. Gorman, Jeffrey M. Jacobson, Paul Volberding, and Scott Letendre. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199392742.003.0012.

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With recent developments in the classification and definitions for HIV-associated neurocognitive disorders (HAND), neuropsychological assessments have become central to the diagnostic process. The pattern and manifestation of neurocognitive dysfunction associated with HIV have changed since the introduction of effective antiretroviral medications and their increasingly widespread use. Prior to the introduction of antiretroviral therapy, severe cognitive disorder presenting as HIV-associated dementia (HAD) was a common manifestation of HIV-associated neurocognitive disorders. In more recent times, however, milder forms of neurocognitive impairment have become more common, often being asymptomatic and difficult to recognize without formal neuropsychological testing. Hence, recognizing individuals at most risk for cognitive decline through neuropsychological testing will offer opportunities for developing targeted interventions that could delay disease progression and improve individual functioning and quality of life.

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42

Zachar, Peter, and Richard J. McNally. Vagueness, the sorites paradox, and posttraumatic stress disorder. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198722373.003.0009.

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This chapter explores the vagueness inherent in the conceptual structure of posttraumatic stress disorder (PTSD). Although psychopathologists have developed precise diagnostic criteria for PTSD, concepts such as traumatic, severe, and impaired generate borderline cases. As in the sorites paradox, where difficult to distinguish but successively smaller piles of sand may be called heaps, in PTSD similar but successively milder traumatic events may produce PTSD symptoms. The vagueness that bedevils PTSD is of two sorts: the degree vagueness manifested in gradual transitions between subtraumatic and traumatic stressors; and vagueness between normal and abnormal reactions. Also discussed here is an alternative causal systems approach in which the symptoms of PTSD are causally related parts of PTSD. Such mereological structures produce combinatorial vagueness in which there are borderline cases between PTSD and other psychiatric syndromes.

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43

de Koning, Tom J. Serine Deficiency. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0023.

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Serine deficiency is a rare cause of intractable seizures and severe psychomotor retardation in infants and young children. However, in recent years it has become clear that serine deficiency in adolescents and adults can give rise to milder forms of seizure disorders and mild mental retardation or to a phenotype with severe progressive polyneuropathy. Serine deficiency can be diagnosed on the basis of low values of serine in plasma and CSF using routine amino acid analysis. However, with the introduction of next generation sequencing in clinical diagnostics the majority of patients are diagnosed through molecular testing of the three genes of the synthesis pathway. L-serine therapy is highly effective in the treatment of seizures and improvement of wellbeing and daily activities. Early diagnosis and timely treatment are important to prevent irreversible damage to the central or peripheral nervous system.

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44

Yager, Joel, Philip S. Mehler, Eileen D. Yager, and Alison R. Yager. Integrated Care for Binge Eating and Other Eating Disorders. Edited by Robert E. Feinstein, Joseph V. Connelly, and Marilyn S. Feinstein. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190276201.003.0015.

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Binge eating disorder, bulimia nervosa, and anorexia nervosa, particularly milder cases, often go unrecognized and untreated in primary care practice settings. Eating disorders are frequently associated with shame, and masked by other prominent physical and psychiatric conditions that demand attention. Among adults, binge eating disorder, the most prevalent of the eating disorders, occurs with increasing frequency, in tandem with higher rates and degrees of obesity, across all age ranges, and in both genders. Bulimia nervosa and anorexia nervosa are more common in females, and although they are most often seen in pediatric and adolescent medicine practices cases, they are also seen in adult practice populations. This chapter describes pathways by which primary care practices can implement integrated and collaborative care treatment programs, likely to benefit large numbers of patients, and effectively coordinate with specialist levels of care, as necessary.

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45

Ganança, Licínia, David A. Kahn, and Maria A. Oquendo. Mood Disorders. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199326075.003.0003.

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This chapter discusses the mood disorders. Major depressive disorder is characterized by neurovegetative changes, anhedonia, and suicidal ideation. Persistent depressive disorder is a milder form of depression, lasting for at least 2 years, with little or no remission during that time... Psychotic features can occur in both depressive and manic episodes. Premenstrual dysphoric disorder is diagnosed through use of a prospective daily symptom ratings log showing a cyclical pattern over at least 2 consecutive months. Patients with mood episodes with mixed features have a high risk of suicide. Some patients with bipolar disorder and major depressive disorder may develop catatonic features characterized by marked psychomotor disturbance. Selective serotonin reuptake inhibitors (SSRIs) are the usual first-line medication treatment for patients with major depressive disorder. For patients with bipolar disorder the mainstays of somatic therapy are lithium and the anticonvulsants valproate and carbamazepine.

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46

Newman, Chris, Christina D. Buesching, and David W. Macdonald. Meline mastery of meteorological mayhem: the effects of climate changeability on European badger population dynamics. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198759805.003.0021.

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Adaptation to climatic conditions is a major ecological and evolutionary driver. Long-term study of European badger population dynamics in Oxfordshire reveals that rainfall and temperature patterns affect food (principally earthworm) availability, energy expended in thermoregulation, and activity patterns, with badgers able to seek refuge in their setts. Cubs prove especially vulnerable to harsh weather conditions, where drought and food shortages exacerbate the severity of pandemic juvenile coccidial parasite infections. Crucially, weather variability, rather than just warming trends, stresses badgers, by destabilising their bioclimatic niche. Summer droughts cause mortality, even driving genetic selection; and while milder winters generally benefit badgers, less time spent in torpor leads to more road casualties. Similar effects also operate over a wide spatial scale in Ireland, impacting regional badger densities and bodyweights. That even an adaptable, generalist musteloid is so variously susceptible to weather conditions highlights how climate change places many species and ecosystems at risk.

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47

Monani, Umrao R., and Darryl C. De Vivo. Spinal Muscular Atrophy. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0033.

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Spinal muscular atrophy (SMA) is a common, inherited, pediatric motor neuron disorder caused by insufficient SMN protein. As of yet, there is no good treatment for the disease. SMA has an incidence of ~1 in 10,000 newborns carrier frequency of 1 in 50, making it the most common inherited cause of infant mortality. Patients with severe SMA, or Werdnig-Hoffman disease, typically manifest weakness during the first 6 months of life. Such patients are so debilitated that they never sit independently, frequently succumbing to the disease before age 2 years. A much milder form of SMA, Kugelberg-Welander disease, with onset after 18 months of age, often during childhood and characterized by prolonged ambulation and a normal life expectancy, was described in 1956. In 1995 mutations in a novel gene, Survival of Motor Neuron 1 (SMN1), were determined to be the specific cause of SMA.

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48

Abrahams, Sharon, and Christopher Crockford. Cognitive and behavioural dysfunction in ALS and its assessment. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757726.003.0008.

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Cognitive and behavioural dysfunction in amyotrophic lateral sclerosis (ALS) occurs in up to half of patients with a spectrum from ALS with no cognitive or behavioural impairment to ALS with frontotemporal dementia (FTD). ~ 15% have a full blown ALS-FTD syndrome, while ~ 35% show milder and specific deficits on verbal fluency, executive and language functions and social cognition. Patients may show a behavioural syndrome that ranges from mild specific difficulties to changes that fulfil diagnostic criteria for behavioural variant-FTD. Apathy is the most prevalent symptom, but disinhibition, perseveration, loss of sympathy/empathy, and change in eating behaviour are also described. The importance of assessment is increasingly recognized. A distinction is made between brief assessment tools useful within ALS clinics and more extensive neuropsychological assessment by a qualified clinical neuropsychologist. Newly developed assessments specifically designed for ALS are available and will make valuable contribution to clinical care.

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49

Buchanan, Elaine, and Chris Lavy. Low back pain. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0064.

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Low back pain (LBP) is pain arising from the structures of the lumbar spine, including joints, discs, connective tissue, and nerves. Symptoms include pain and, muscle tightness or stiffness, with or without referral of pain to the legs. Most (95%) of LBP is managed in primary care; the rest is managed in secondary care. LBP affects nearly everyone at some point in their life and has an annual prevalence of around 40%. It is less common in children but from age 16 onwards the point prevalence for all age groups is around 25%. Many experience milder persisting symptoms interspersed with exacerbations, and 7% of adults have persisting LBP, which restricts function. This chapter discusses LBP, including definitions, differential diagnosis, context, approach to diagnosis, specific clues to the diagnosis, key diagnostic tests, treatment and therapy, prognosis, and how to handle uncertainty in the diagnosis of the symptom.

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50

Briddon, Anthony. Approach to the Patient with Hyperhomocysteinemia. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0079.

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Hyperhomocysteinaemia (HHC) may occur as a result of a variety of inherited and acquired conditions ranging from mild and benign to severe and life threatening, and there is a higher probability that they will first manifest during early adulthood rather than infancy, with acquired forms commonly presenting into old age. Milder forms of HHC may exist without homocystinuria, and screening tests relying on the presence of homocystine in the urine will give a false negative result. Methylcobalamin is an essential cofactor for methionine synthase, a key enzyme in the homocysteine remethylation pathway: consequently, investigation for inherited abnormalities of intracellular B12 metabolism forms an integral part of the biochemical investigation of HHC. Additionally, there are acquired forms of HHC, notably those involving vitamin B12 deficiency and malabsorption. Mild to moderate HHC has also been associated with alcohol abuse, excessive caffeine intake, hypothyroidism, and poor renal function.

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Books on the topic 'Mildura'

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