Relevant bibliographies by topics / MicroRNA-200

Academic literature on the topic 'MicroRNA-200'

Author: Grafiati
Published: 10 December 2022
Last updated: 28 January 2023

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Journal articles on the topic "MicroRNA-200"

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Liu, Xiaodong, Jianhua Zhang, Botao Xie, Hao Li, Jihong Shen, and Jianheng Chen. "MicroRNA-200 Family Profile." American Journal of Therapeutics 23, no. 2 (2016): e388-e397. http://dx.doi.org/10.1097/mjt.0000000000000361.

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Tian, Yin, Qiong Pan, Yangyang Shang, Rong Zhu, Jun Ye, Yun Liu, Xiaoli Zhong, et al. "MicroRNA-200 (miR-200) Cluster Regulation by Achaete Scute-like 2 (Ascl2)." Journal of Biological Chemistry 289, no. 52 (November 4, 2014): 36101–15. http://dx.doi.org/10.1074/jbc.m114.598383.

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Shi, Min, Yulan Mu, Hui Zhang, Ming Liu, Jipeng Wan, Xiaoyan Qin, and Changzhong Li. "MicroRNA-200 and microRNA-30 family as prognostic molecular signatures in ovarian cancer." Medicine 97, no. 32 (August 2018): e11505. http://dx.doi.org/10.1097/md.0000000000011505.

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Hsieh, Pei-Ling, Chun-Chung Huang, and Cheng-Chia Yu. "Emerging Role of MicroRNA-200 Family in Dentistry." Non-Coding RNA 7, no. 2 (June 11, 2021): 35. http://dx.doi.org/10.3390/ncrna7020035.

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MicroRNAs (miRNAs) are endogenous non-coding RNAs ~22 nucleotides in length, which have been shown to participate in various biological processes. As one of the most researched miRNAs, the miR-200 family has been found to regulate several factors that are associated with the epithelial to mesenchymal transition (EMT) and cancer stem cells (CSCs) behavior. In this review, we briefly summarize the background of the miR-200 family and their implication in various dental diseases. We focus on the expression changes, biological functions, and clinical significance of the miR-200 family in oral cancer; periodontitis; oral potentially malignant disorder; gingival overgrowth; and other periodontal diseases. Additionally, we discuss the use of the miR-200 family as molecular biomarkers for diagnosis, prognostic, and therapeutic application.
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Senfter, Daniel, Sibylle Madlener, Georg Krupitza, and Robert M. Mader. "The microRNA-200 family: still much to discover." Biomolecular Concepts 7, no. 5-6 (December 1, 2016): 311–19. http://dx.doi.org/10.1515/bmc-2016-0020.

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AbstractIn the last decade, microRNAs (miRs or miRNAs) became of great interest in cancer research due to their multifunctional and active regulation in a variety of vital cellular processes. In this review, we discuss the miR-200 family, which is composed of five members (miR-141, miR-200a/200b/200c and miR-429). Although being among the best investigated miRNAs in the field, there are still many open issues. Here, we describe the potential role of miR-200 as prognostic and/or predictive biomarker, its influence on motility and cell migration as well as its role in epithelial to mesenchymal transition (EMT) and metastasis formation in different tumour types. Recent studies also demonstrated the influence of miR-200 on drug resistance and described a correlation between miR-200 expression levels and overall survival of patients. Despite intense research in this field, the full role of the miR-200 family in cancer progression and metastasis is not completely understood and seems to differ between different tumour types and different cellular backgrounds. To elucidate these differences further, a finer characterisation of the role of the individual miRNA-200 family members is currently under investigation.
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Shinozaki, Aya, Takashi Sakatani, Tetsuo Ushiku, Rumi Hino, Maya Isogai, Shunpei Ishikawa, Hiroshi Uozaki, Kenzo Takada, and Masashi Fukayama. "Downregulation of MicroRNA-200 in EBV-Associated Gastric Carcinoma." Cancer Research 70, no. 11 (May 18, 2010): 4719–27. http://dx.doi.org/10.1158/0008-5472.can-09-4620.

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Snowdon, Jaime, Xiao Zhang, Tim Childs, Victor A. Tron, and Harriet Feilotter. "The MicroRNA-200 Family Is Upregulated in Endometrial Carcinoma." PLoS ONE 6, no. 8 (August 29, 2011): e22828. http://dx.doi.org/10.1371/journal.pone.0022828.

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Castilla, María Ángeles, Juan Díaz-Martín, David Sarrió, Laura Romero-Pérez, María Ángeles López-García, Begoña Vieites, Michele Biscuola, Susana Ramiro-Fuentes, Clare M. Isacke, and José Palacios. "MicroRNA-200 Family Modulation in Distinct Breast Cancer Phenotypes." PLoS ONE 7, no. 10 (October 24, 2012): e47709. http://dx.doi.org/10.1371/journal.pone.0047709.

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Paterson, Emily L., Natasha Kolesnikoff, Philip A. Gregory, Andrew G. Bert, Yeesim Khew-Goodall, and Gregory J. Goodall. "The microRNA-200 Family Regulates Epithelial to Mesenchymal Transition." Scientific World JOURNAL 8 (2008): 901–4. http://dx.doi.org/10.1100/tsw.2008.115.

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Škrha, Pavel, Aleš Hořínek, Eva Pazourková, Jan Hajer, Přemysl Frič, Jan Škrha, and Michal Anděl. "Serum microRNA-196 and microRNA-200 in pancreatic ductal adenocarcinoma of patients with diabetes mellitus." Pancreatology 16, no. 5 (September 2016): 839–43. http://dx.doi.org/10.1016/j.pan.2016.05.005.

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Dissertations / Theses on the topic "MicroRNA-200"

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Backlund, Kristina. "microRNA-200 Family Expression Level Changes in Stimulated THP-1 Cells Following NLRP3 Inflammasome Activation." Thesis, Högskolan i Skövde, Institutionen för biovetenskap, 2020. http://urn.kb.se/resolve?urn=urn:nbn:se:his:diva-18917.

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Innate immunity is the immune systems rapid responses to infection after being attacked by a pathogen. Inflammatory responses are activated by the detection of pathogen-associated molecular patterns and danger-associated molecular patterns through pattern recognition receptors on inflammatory cells. NLRs are activated by intracellular PAMPs which warn cells of damage and have a major role in initiating the innate inflammatory responses as well as the development of infectious and inflammatory diseases. NLRP3 is a very large multiprotein complex and is the most studied inflammasome. The NLRP3 Inflammasome follows a two-signal model for activation, signal one forms the NLRP3 complex and signal two activates the inflammasome. NLRP3 initiates an inflammatory form of cell death called pyroptosis and triggers the release of pro-inflammatory cytokines IL-1β and IL-18. The miR-200 family has five members, miR-200a, miR-200b and miR-429 located on chromosome 1 and miR-200c and miR-141 located on chromosome 12. In this study, THP-1 cells were differentiated with PMA then stimulated with LPS and ATP. Various time samples were collected and isolated to obtain miRNA. Two-step RT-qPCR was then performed to quantitively monitor the changes in miRNA-200 family expression levels. The purpose of this study was to observe how miRNA-200 family expression levels change in stimulated THP-1 cells as the NLRP3 inflammasome is activated. This became a pilot study as all biological replicates could not be analyzed, miR-200 family is showing a potential response to the activation of the NLRP3 inflammasome and they should be investigated further.
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"The roles of microRNA-200 family in ovarian cancer development." 2013. http://library.cuhk.edu.hk/record=b5884525.

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Choi, Pui Wah.
Thesis (Ph.D.)--Chinese University of Hong Kong, 2013.
Includes bibliographical references (leaves 202-232).
Electronic reproduction. Hong Kong : Chinese University of Hong Kong, [2012] System requirements: Adobe Acrobat Reader. Available via World Wide Web.
Abstracts also in Chinese.
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Lim, Yat Yuen. "The miR-200 family is controlled by epigenetic-based mechanisms and mediates transition between non-stem and stem-like cell phenotypes." Thesis, 2012. http://hdl.handle.net/2440/79825.

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MicroRNAs (miRNAs) are ~22 nucleotide (nt) single-stranded non-coding RNAs which are important regulators of gene expression in many biological processes including controlling cellular phenotype. The epithelial to mesenchymal transition (EMT) and the reverse process termed mesenchymal to epithelial transition (MET) are key programs that control the transition of cells between stem-like and non-stem phenotypes which are collectively termed epithelial plasticity. The miR-200 family is a key regulator of EMT however its role in controlling the transition between stem-like and non-stem phenotypes has not been well characterized. I utilized immortalized human mammary epithelial cells (HMLE) to investigate the function and regulation of the miR-200s during their conversion from a non-stem to a stem-like phenotype. HMLE cells were found to spontaneously convert from a non-stem to a stem-like phenotype. Isolation and comparison of the miR-200 expression between the spontaneously derived stem- like cells (sl-HMLE) and non-stem HMLE cells (nsl-HMLE) showed that the spontaneous conversion to a stem-like phenotype was accompanied by the loss of miR-200 expression. Likewise, miR-200 expression was also found to be down-regulated in prospective breast cancer stem cells (bCSCs) from metastatic pleural or ascites effusions and SUM159PT breast cancer cell line compared to non-CSC cells. This phenotypic change from a non-stem to a stem-like phenotype was directly controlled by the miR-200s as restoration of its expression partially converted the sl-HMLE cells to a non-stem phenotype with decrease stem-like properties and induction of an MET-like phenotype, although restoration of the miR-200 expression in SUM159PT prospective bCSCs did not have this effect. Next, using bioinformatic approaches and cell-based assays, I aimed to identify new miR-200 targets that are responsible for regulating the stem-like properties in both sl-HMLE cells and SUM159PT prospective bCSCs. Although the predicted genes (WNT5A, PKCα and PKCε) were not direct miR-200 targets, preliminary data suggest those genes may be involve in the survival or anoikis-resistance of stem-like cells and bCSCs. Investigation of the mechanism(s) controlling miR-200 expression revealed both DNA methylation and histone modifications were significantly altered in the stem-like and non-stem phenotypes. In particular, in the stem-like phenotype, the miR-200b/a/429 cluster was silenced primarily through polycomb group-mediated silencing whereas the miR-200c/141 cluster was repressed by DNA methylation. Furthermore, slight increase in EZH2 expression was observed in the stem-like phenotype and this might potentially contribute to the polycomb group-mediated silencing of the miR-200b/a/429 cluster. Lastly, preliminary co-immunoprecipitation results suggest that the targeting of polycomb group proteins to the miR-200b/a/429 promoter is not dependent on the ZEB1 transcription factor which is a repressor of the miR-200 transcription. Collectively, these results indicate that the miR-200 family plays a critical role in the transition between stem-like and non-stem phenotypes and that distinct epigenetic-based mechanisms regulate each miR-200 gene in this process. Therefore, combination of chemotherapy with therapies targeted against the miR-200 family members and epigenetic modifications would be beneficial towards treatment of breast cancer.
Thesis (Ph.D.) -- University of Adelaide, School of Medicine, 2012
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Zhang, Xin. "The role of microRNAs in the p53 tumor suppressor pathway." Doctoral thesis, 2010. http://hdl.handle.net/11858/00-1735-0000-0006-B5D7-9.

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Yalcin, Abdullah. "Expression analysis of Drosophila melanogaster microRNAs." Doctoral thesis, 2007. http://hdl.handle.net/11858/00-1735-0000-0006-AC44-B.

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Conference papers on the topic "MicroRNA-200"

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Ungewiss, Christin, Jonathon D. Roybal, David H. Peng, and Don L. Gibbons. "Abstract C56: microRNA-200 control of tumor cell cytoskeletal changes and invasion." In Abstracts: AACR Special Conference on Tumor Invasion and Metastasis - January 20-23, 2013; San Diego, CA. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.tim2013-c56.

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Enokida, Hideki, Hirofumi Yoshino, Takeshi Yamasaki, Hideo Hidaka, Takeshi Chiyomaru, Nijiro Nohata, Naohiko Seki, and Masayuki Nakagawa. "Abstract 2284: MIR-200 family as EMT related microRNA in renal cell carcinoma." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-2284.

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Porseva, Valentina, Andrey Spirichev, Lydia Pankrаsheva, Polina Vyshnyakova, and Petr Masliukov. "ANALYSIS OF MICRORNA (miR-200) EXPRESSION IN HYPOTHALAMIC NUCLEI IN YOUNG AND OLD MALE RATS." In XVIII INTERNATIONAL INTERDISCIPLINARY CONGRESS NEUROSCIENCE FOR MEDICINE AND PSYCHOLOGY. LCC MAKS Press, 2022. http://dx.doi.org/10.29003/m2896.sudak.ns2022-18/276-277.

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Choi, Pui Wah, Junzheng Yang, Wing Ping Fong, Ross S. Berkowitz, William R. Welch, Gregory J. Goodall, and Shu Wing Ng. "Abstract A23: The dual roles of microRNA-200: From inclusion cyst formation to cell migration." In Abstracts: AACR Special Conference: Advances in Ovarian Cancer Research: Exploiting Vulnerabilities; October 17-20, 2015; Orlando, FL. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1557-3265.ovca15-a23.

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Chen, Limo, Sangeeta Goswami, Xiaohui Yi, Lauren Byers, Lixia Diao, Jonathon Roybal, Christin Ungewiss, et al. "Abstract B67: A big role in tumor immune microenvironment of a small non-coding RNA: microRNA-200." In Abstracts: AACR Special Conference: Tumor Immunology and Immunotherapy: A New Chapter; December 1-4, 2014; Orlando, FL. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/2326-6074.tumimm14-b67.

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Ungewiss, Christin, Jonathon D. Roybal, David H. Peng, and Don L. Gibbons. "Abstract B44: The microRNA-200/Zeb1 axis regulates ECM-dependent Src signaling, cytoskeletal changes and cancer cell invasion." In Abstracts: AACR Special Conference on Cellular Heterogeneity in the Tumor Microenvironment; February 26 — March 1, 2014; San Diego, CA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.chtme14-b44.

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Roy, SS, VK Gonugunta, AM Bandyopadhyay, M. Rao, G. Goodall, L. Sun, RR Tekmal, and RK Vadlamudi. "Abstract P5-04-04: Significance of PELP1/HDAC2/microRNA-200 regulatory network in EMT and metastasis of breast cancer." In Abstracts: Thirty-Fifth Annual CTRC‐AACR San Antonio Breast Cancer Symposium‐‐ Dec 4‐8, 2012; San Antonio, TX. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/0008-5472.sabcs12-p5-04-04.

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Song, Fengju, and Kexin Chen. "Abstract 1474: Integrated microRNA network analyses identify a poor prognosis subtype of gastric cancer characterized by the miR-200 family." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-1474.

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Gotoh, Masahiro, Eri Arai, Akio Matsuda, Hiroyuki Fujimoto, Kenji Matsumoto, and Yae Kanai. "Abstract 1065: Reduced expression of microRNA-200 family associated with aggressiveness and poorer patient outcome of clear cell renal cell carcinomas." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-1065.

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Roybal, Jonathon D., David H. Peng, Christin Ungewiss, Pijus K. Mandal, John S. McMurray, Lauren A. Byers, and Don L. Gibbons. "Abstract C57: The role of the microRNA-200 family in the regulation of tumor cell signaling and metastasis in lung cancer." In Abstracts: AACR Special Conference on Tumor Invasion and Metastasis - January 20-23, 2013; San Diego, CA. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.tim2013-c57.

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