Relevant bibliographies by topics / Microcrystal arthritis

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  1. Journal articles
  2. Dissertations / Theses

Academic literature on the topic 'Microcrystal arthritis'

Author: Grafiati
Published: 14 March 2023

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Journal articles on the topic "Microcrystal arthritis"

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Zamudio-Cuevas, Yessica, Javier Fernández-Torres, Gabriela Angélica Martínez-Nava, Karina Martínez-Flores, and Alberto López-Reyes. "Emergent nanotherapies in microcrystal-induced arthritis." International Immunopharmacology 61 (August 2018): 197–203. http://dx.doi.org/10.1016/j.intimp.2018.06.007.

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Patrón-Ordóñez, Gino, and María I. Anticona-Sayán. "Charcot neuroarthropathy: Differential diagnosis of monoarthritis of the knee in patients with type 2 diabetes mellitus." Revista de la Facultad de Medicina Humana 22, no. 4 (2022): 906–11. http://dx.doi.org/10.25176/rfmh.v22i4.5107.

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A 46-year-old man with long-standing, poorly controlled diabetes is hospitalized for right knee monoarthritis after trauma. His clinical evaluation found joint deformity, synovitis in the right knee and absence of multiple toes due to a history of amputation. With the radiological evaluation that showed bone destruction and the synovial fluid analysis that ruled out infectious causes and microcrystal arthritis, the diagnosis of Charcot neuroarthropathy of the knee was made. Conservative management was indicated with the use of a felt insole for unloading and the use of a walker, the case being
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Brune, Kay, Käthy Bucher, and Dieter Walz. "The avian microcrystal arthritis II. Central versus peripheral effects of sodium salicylate, acetaminophen and colchicine." Agents and Actions 43, no. 3-4 (1994): 211–17. http://dx.doi.org/10.1007/bf01986691.

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Ferreyra, Marine, Guillaume Coiffier, Jean-David Albert, Claire David, Aleth Perdriger, and Pascal Guggenbuhl. "Combining cytology and microcrystal detection in nonpurulent joint fluid benefits the diagnosis of septic arthritis." Joint Bone Spine 84, no. 1 (2017): 65–70. http://dx.doi.org/10.1016/j.jbspin.2016.04.002.

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Eliseev, M. S., and A. M. Novikova. "Clinical discussion: gout therapy in a comorbid patient." Meditsinskiy sovet = Medical Council, no. 11 (August 8, 2020): 154–62. http://dx.doi.org/10.21518/2079-701x-2020-11-154-162.

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The data accumulated to date suggests that it is extremely rare for a gout patient to have only his or her main disease and no accompanying pathology. One of the frequent situations is a combination with another microcrystal arthritis, a disease of calcium pyrophosphate crystals deposition. In addition, diseases of the kidneys (including chronic renal failure), cardiovascular system, gastrointestinal tract, as well as metabolic disorders directly associated with gout or indirectly related to taking medications necessary to control the disease, not only affect the quality and longevity of life
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Peral, M. L., I. Calabuig, A. Martín-Carratalá, M. Andrés, and E. Pascual. "THU0406 IDENTIFICATION OF INTRACELLULAR VACUOLES IN SYNOVIAL FLUID WITH CALCIUM PYROPHOSPHATE AND MONOSODIUM URATE CRYSTALS." Annals of the Rheumatic Diseases 79, Suppl 1 (2020): 440.1–441. http://dx.doi.org/10.1136/annrheumdis-2020-eular.3851.

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Background:Synovial fluid analysis using polarized microscopy is the gold standard for the diagnosis of crystal-related arthritis. In our experience, we have noted that, when calcium pyrophosphate (CPP) crystals are observed, they sometimes appear within intracellular vacuoles. However, this phenomenon is not seen in those samples containing monosodium urate (MSU) crystals. This finding has been scantly reported in the literature, but may be useful in clinical practice to ensure accurate crystal identification.Objectives:Our study aims to assess whether the presence of vacuoles contributes to
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Duse, Adina Octavia, Delia Berceanu Vaduva, Mirela Nicolov, et al. "Biostatistical Analysis and Possible Forecasting of Relationship Between Uric Acid and Specific Laboratory Tests in Cases of Gouty Arthritis." Revista de Chimie 68, no. 6 (2017): 1234–41. http://dx.doi.org/10.37358/rc.17.6.5648.

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Acute gouty arthritis represents an inflammatory response to microcrystals of monosodium urate that precipitate in joint tissues from supersaturated body fluids or are shed from preexisting articular deposits [1]. Gout is a metabolic disease characterized by recurrent episodes of arthritis associated with the presence of monosodium urate crystals in the tissue or synovial fluid during the attack.These forms of crystal-induced arthritis usually affect peripheral joints, including knee, ankle, wrist, and metacarpophalangeal and metatarsophalangeal joints. All of them may be associated with other
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Pouliot, Marc, Michael J. James, Shaun R. McColl, Paul H. Naccache, and Leslie G. Cleland. "Monosodium Urate Microcrystals Induce Cyclooxygenase-2 in Human Monocytes." Blood 91, no. 5 (1998): 1769–76. http://dx.doi.org/10.1182/blood.v91.5.1769.

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Abstract The formation and deposition of monosodium urate (MSU) microcrystals in articular and periarticular tissues is the causative agent of acute or chronic inflammatory responses known as gouty arthritis. Mononuclear phagocyte activation is involved in early triggering events of gout attacks. Because stimulated mononuclear phagocytes can constitute an important source of the inducible isoform of cyclooxygenase (COX-2), we evaluated the effects that proinflammatory microcrystals might have on COX-2 protein expression in crystal-stimulated monocytes. We found that MSU crystals, but not calci
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Pouliot, Marc, Michael J. James, Shaun R. McColl, Paul H. Naccache, and Leslie G. Cleland. "Monosodium Urate Microcrystals Induce Cyclooxygenase-2 in Human Monocytes." Blood 91, no. 5 (1998): 1769–76. http://dx.doi.org/10.1182/blood.v91.5.1769.1769_1769_1776.

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The formation and deposition of monosodium urate (MSU) microcrystals in articular and periarticular tissues is the causative agent of acute or chronic inflammatory responses known as gouty arthritis. Mononuclear phagocyte activation is involved in early triggering events of gout attacks. Because stimulated mononuclear phagocytes can constitute an important source of the inducible isoform of cyclooxygenase (COX-2), we evaluated the effects that proinflammatory microcrystals might have on COX-2 protein expression in crystal-stimulated monocytes. We found that MSU crystals, but not calcium pyroph
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10

Chemes, V., O. Abrahamovych, U. Abrahamovych, R. Ivanochko, and L. Kobak. "Calcium-phosphorus metabolism and markers of its regulation in patients with rheumatoid arthritis with violation of bone mineral density: character and diagnostic value." Lviv clinical bulletin 3-4, no. 39-40 (2023): 76–82. http://dx.doi.org/10.25040/lkv2022.03-04.076.

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Introduction. Patients with rheumatoid arthritis (RA) are twice as likely to have osteoporosis (OP) compared to the general population. The strength of bones depends on mineral substances, mainly represented by calcium phosphate microcrystals. The chief role in the regulation of calcium-phosphorus metabolism is played by vitamin D and parathormone (PTH). The aim of the study. To investigate markers of calcium-phosphorus metabolism in patients with rheumatoid arthritis accompanied by bone mineral density (BMD) disorders and to find out their diagnostic value. Materials and methods. 76 patients
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Dissertations / Theses on the topic "Microcrystal arthritis"

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Alì, Alessandra. "Studies on molecular aspects of inflammation : Involvement of purinergic P2X7R in collagen production in systemic sclerosis and chronic heart failure Neutrophil Extracellular Traps release in synovial fluid from microcrystal and non-microcrystal arthritis." Doctoral thesis, Università di Siena, 2019. http://hdl.handle.net/11365/1071012.

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Background: The purinergic P2X7 receptor (P2X7R) is a nucleotide-gated ionotropic channel chiefly involved in the inflammatory response triggered by release of ATP from damaged cells. It is largely expressed in inflammatory cells and plays a key role in promoting the release of pro-inflammatory cytokines such as IL-1β. Increasing evidence suggest that it is also expressed in fibroblasts and may play a pivotal role in the development of tissue fibrosis in different body districts. Therefore, we hypothesized a possible P2X7R involvement in the pathogenesis of two different diseases, Systemic scl
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