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Journal articles on the topic 'Microcirculation'

Author: Grafiati

Published: 4 June 2021

Last updated: 16 November 2024

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1

Puchinyan, D. M., and M. S. Sissakian. "Microcirculation state in patients with deforming coxarthrosis." Kazan medical journal 76, no. 1 (January 15, 1995): 52–54. http://dx.doi.org/10.17816/kazmj82726.

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The microcirculation state in 48 patients with unilateral and bilateral deforming coxarthrosis of IIII stages and in 34 healthy persons aged 26 to 63 is studied using biomicroscopy method of bulbar conjunctiva vessels. It is established that the pronounced microcirculating disorders depend on the disease gravity and pathologic process occurrence. The most constant signs of microhemo- circulation disorder are intravescular and vascular changes.

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2

Sorrentino, Elizabeth A., and Harvey N. Mayrovitz. "Microcirculation." Critical Care Nursing Quarterly 14, no. 3 (November 1991): 1–7. http://dx.doi.org/10.1097/00002727-199111000-00003.

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3

Piagnerelli, Michael, Can Ince, and Arnaldo Dubin. "Microcirculation." Critical Care Research and Practice 2012 (2012): 1–3. http://dx.doi.org/10.1155/2012/867176.

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4

KORTHUIS, RONALD J., and GEERT W. SCHMID-SCHÖNBEIN. "Microcirculation Supplement: Microcirculation and Chronic Venous Insufficiency." Microcirculation 7, s (January 2000): S1—S2. http://dx.doi.org/10.1080/mic.7.s.s1.s2.

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5

Korthuis, Ronald, and Geert Schmid-Schönbein. "Microcirculation Supplement: Microcirculation and Chronic Venous Insufficiency." Microcirculation 7, no. 6 (December 1, 2000): 1–2. http://dx.doi.org/10.1080/713774002.

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6

KORTHUIS, RONALD J., and GEERT W. SCHMID-SCHÖNBEIN. "Microcirculation Supplement: Microcirculation and Chronic Venous Insufficiency." Microcirculation 7, S1 (December 2000): S1—S2. http://dx.doi.org/10.1111/j.1549-8719.2000.tb00144.x.

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7

Lundborg, Göran. "Intraneural Microcirculation." Orthopedic Clinics of North America 19, no. 1 (January 1988): 1–12. http://dx.doi.org/10.1016/s0030-5898(20)30326-6.

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8

Zawieja, David C. "Lymphatic Microcirculation." Microcirculation 3, no. 2 (January 1996): 241–43. http://dx.doi.org/10.3109/10739689609148296.

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9

Heusch, Gerd. "Coronary Microcirculation." Circulation Journal 78, no. 8 (2014): 1830–31. http://dx.doi.org/10.1253/circj.cj-14-0539.

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10

TAMAKI, Toshiaki, and Masanori YOSHIZUMI. "Renal microcirculation." Folia Pharmacologica Japonica 113, no. 4 (1999): 261–67. http://dx.doi.org/10.1254/fpj.113.261.

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11

Komaru, Tatsuya, Hiroshi Kanatsuka, and Kunio Shirato. "Coronary microcirculation." Pharmacology & Therapeutics 86, no. 3 (June 2000): 217–61. http://dx.doi.org/10.1016/s0163-7258(00)00057-7.

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12

Garot, P., J. Garot, and M. C. Morice. "Microcirculation coronaire." EMC - Cardiologie 6, no. 3 (January 2011): 1–3. http://dx.doi.org/10.1016/s1166-4568(11)53108-0.

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13

Ocak, Işık, Atila Kara, and Can Ince. "Monitoring microcirculation." Best Practice & Research Clinical Anaesthesiology 30, no. 4 (December 2016): 407–18. http://dx.doi.org/10.1016/j.bpa.2016.10.008.

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14

Mayhan, WG, FM Faraci, GL Baumbach, and DD Heistad. "Cerebral Microcirculation." Physiology 3, no. 4 (August 1, 1988): 164–67. http://dx.doi.org/10.1152/physiologyonline.1988.3.4.164.

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Three concepts are summarized. First, cerebral microvascular pressure, which can be regulated independently from cerebral blood flow, may have important physiological effects. Second, the blood-brain barrier is more vulnerable to disruption in venules than in capillaries or arterioles during acute hypertension. Third, cerebral arterioles are protected during chronic hypertension by a surprising combination of vascular hypertrophy, remodeling of the vessel wall, and a paradoxical increase in distensibility.

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15

Lanzer, Peter, and Christos C. Zouboulis. "Media sclerosis Mönckeberg affects microcirculation." Cor et Vasa 60, no. 5 (October 1, 2018): e533-e535. http://dx.doi.org/10.1016/j.crvasa.2017.05.006.

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16

Richey, Rauchelle E., Holden W. Hemingway, Amy M. Moore, Albert H. Olivencia-Yurvati, and Steven A. Romero. "Acute heat exposure improves microvascular function in skeletal muscle of aged adults." American Journal of Physiology-Heart and Circulatory Physiology 322, no. 3 (March 1, 2022): H386—H393. http://dx.doi.org/10.1152/ajpheart.00645.2021.

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Acute heat exposure improves microvascular function in aged adults as assessed using reactive hyperemia. The cutaneous and skeletal muscle microcirculations are thought to contribute to this response, but this has never been confirmed due to the methodological challenges associated with differentiating blood flow between these vascular beds. Using the microdialysis technique to bypass the cutaneous circulation, we demonstrated that heat exposure improves endothelial-dependent and endothelial-independent vasodilation in the microcirculation of skeletal muscle in aged humans.

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17

Stanishevska, Tetana, Оksana Gorna, Daria Horban, and Olga Yusupova. "Features of blood`s microcirculation at physical loads." ScienceRise: Biological Science, no. 4(25) (December 30, 2020): 4–7. http://dx.doi.org/10.15587/2519-8025.2020.217693.

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This research deals with the study of blood microcirculation peculiarities.Materials and methods. 72 students of Bogdan Khmelnytsky Melitopol State Pedagogical University, aged 18–19, were examined. The experimental research consisted of the study of blood microcirculation functional state by means of Laser Doppler flowmetry (LDF) method. It helped to evaluate the state of tissue blood-circulation and to detect individual-typological peculiarities of blood microcirculation under the influence of physical activity (before and after exercise).Results. Three types of blood microcirculation were identified by using LDF-metry. The normoemic type of blood microcirculation, characterized by the superposition of oscillatory rhythms and reflected the balance of the mechanisms of regulation of microcirculation. The hyperemic type, characterized by a «monotonous» LDF-gram with a high parameter of microcirculation, which reflects the relative predominance of metabolic mechanisms in the regulation of microcirculation. The hypoemic type, characterized by a «monotonous» LDF-gram with a low parameter of the microcirculation parameter, which reflects the decrease of vasomotor mechanisms in the regulation of microcirculation. According to the LDF-metric data, the examined students under intensive physical activity have a significant increase in microcirculatory status: by 6 % of the microcirculation parameter, by 28 % of the mean square deviation and by 45 % of the initial value of the coefficient of variation.Conclusions. This dynamics of microcirculation shows that under the influence of physical exertion, a person creates significant functional reserves for the redistribution of blood flow and for more perfect intraorgan capillary blood flow. It was found, that in the process of physical activity, morpho-functional rearrangements of the human cardiovascular system occur. This reaction is formed by several components of blood microcirculation: blood flow in the transport direction, regulating blood supply in accordance with the needs of tissues and the exchange component of the histochemical barrier

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18

Wang, Hui, Hong Ding, Zi-Yan Wang, and Kun Zhang. "Research progress on microcirculatory disorders in septic shock: A narrative review." Medicine 103, no. 8 (February 23, 2024): e37273. http://dx.doi.org/10.1097/md.0000000000037273.

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Hemodynamic coherence plays a critical role in the outcomes of septic shock. Due to the potential negative consequences of microcirculatory disorders on organ failure and clinical outcomes, the maintenance of a balance between the macrocirculation and microcirculation is a topic of significant research focus. Although physical methods and specialized imaging techniques are used in clinical practice to assess microcirculation, the use of monitoring devices is not widespread. The integration of microcirculation research tools into clinical practice poses a significant challenge for the future. Consequently, this review aims to evaluate the impact of septic shock on the microcirculation, the methods used to monitor the microcirculation and highlight the importance of microcirculation in the treatment of critically ill patients. In addition, it proposes an evaluation framework that integrates microcirculation monitoring with macrocirculatory parameters. The optimal approach should encompass dynamic, multiparametric, individualized, and continuous monitoring of both the macrocirculation and microcirculation, particularly in cases of hemodynamic separation.

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19

Onoprienko, G. A., V. S. Zubikov, and I. G. Mikhailov. "Microcirculation and regeneration of long bones in extraosseous osteosynthesis by АО system." N.N. Priorov Journal of Traumatology and Orthopedics 3, no. 2 (June 15, 1996): 21–24. http://dx.doi.org/10.17816/vto64220.

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Three series of experiments in dogs were performed. In one series subtrochanteric osteotomy of the femur using compression osteosynthesis with Г-shape plate was carried out, in the other - osteotomy of the diaphysis of either the femur or tibia using osteosynthesis with straight plate, and in the third (control) series osteotomy was not performed and the plate was implanted under compression or without it. Bone tissue microcirculation was studied in the enlightened sections by authors method using Indian ink-gelatine mixture; morphologic examinations were carried out on eosin-hematoxylin stained specimens. Presice effect of osteogenesis induction in the place of plate contact with the metal fixative was observed and it was most pronounced around the screw thread, that was considered as a factor of the additional osteosynthesis fixation. During the formation of primary consolidation, the delay in angioarchitectonics retardation of the compact bone under the plate was noted. Microcirculatiry bed response was of universal addoptive pattern and showed the formation of extravascular microcirculation at early stages.

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20

Huiming, Gong, Wang Yuming, Yang Mingliang, Liu Changbin, Huang Qiuchen, and Li Jianjun. "Study on the characteristics of microcirculation in the site of pressure ulcer in patients with spinal cord injury." Science Progress 104, no. 3 (July 2021): 003685042110287. http://dx.doi.org/10.1177/00368504211028726.

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To investigate the characteristics of pressure ulcer microcirculation in SCI patients with pressure ulcer, and to provide evidence for the treatment of pressure ulcer in patients with SCI. Group 1 ( n = 12) SCI patients with pressure ulcer, 23 pressure ulcers were included. Group 2 ( n = 15) SCI patients without pressure ulcer and the control group ( n = 16) healthy adults. The application of laser Doppler perfusion imaging system (Moor FLPI) detector to the microcirculation perfusion of the sacrum area of the control group, the observation group 2 and the pressure ulcer site of the observation group 1, record the microcirculation perfusion (PU), The data of microcirculation perfusion (PU) were compared and analyzed. The correlation between microcirculation perfusion and healing time of pressure ulcer was analyzed. (1) The microcirculation perfusion was highest in the pressure ulcer center. (2) SCI patients and healthy adults had no significant difference of microcirculation perfusion at sacrococcygeal skin. (3) The lower the microcirculation perfusion of the pressure ulcer center, the longer the healing time of pressure ulcer. The healing time and the microcirculation perfusion of pressure ulcer center was negatively correlated. Microcirculation perfusion detection is a noninvasive and effective method for the determination of the scope of pressure ulcer, detection and direction judgment of pressure ulcer sinus tract, monitoring and guidance of pressure ulcer treatment, and prediction of the healing time of pressure ulcer.

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21

Sun, Zhengze, Yaxin Li, Rongjun Liu, Baikai Ma, Yifan Zhou, Hongyu Duan, Linbo Bian, Wenlong Li, and Hong Qi. "Progress of Bulbar Conjunctival Microcirculation Alterations in the Diagnosis of Ocular Diseases." Disease Markers 2022 (August 28, 2022): 1–6. http://dx.doi.org/10.1155/2022/4046809.

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Bulbar conjunctival microcirculation is a microvascular system distributed in the translucent bulbar conjunctiva near the corneal limbus. Multiple ocular diseases lead to bulbar conjunctival microcirculation alterations, which means that bulbar conjunctival microcirculation alterations would be potential screening and diagnostic indicators for these ocular diseases. In recent years, with the emergence and application of a variety of noninvasive observation devices for bulbar conjunctiva microcirculation and new image processing technologies, studies that explored the potential of bulbar conjunctival microcirculation alterations in the diagnosis of ocular diseases have been emerging. However, the potential of bulbar conjunctival microcirculation alterations as indicators for ocular diseases has not been exploited to full advantage. The observation devices, image processing methods, and algorithms are not unified. And large-scale research is needed to concrete bulbar conjunctival microcirculation alterations as indicators for ocular diseases. In this paper, we provide an update on the progress of bulbar conjunctival microcirculation alterations in the diagnosis of ocular diseases in recent five years (from January 2017 to March 2022). Relevant ocular diseases include contact lens wearing, dry eye, conjunctival malignant melanoma, conjunctival nevus, and diabetic retinopathy.

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22

Korkushko, O. V. "«Microcirculation» Experiment Influence of space flight factors on blood microcirculation and its rheological properties in human." Kosmìčna nauka ì tehnologìâ 6, no. 4 (July 30, 2000): 125. http://dx.doi.org/10.15407/knit2000.04.140.

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23

Padró, Teresa, Gemma Vilahur, and Lina Badimon. "Dyslipidemias and Microcirculation." Current Pharmaceutical Design 24, no. 25 (November 8, 2018): 2921–26. http://dx.doi.org/10.2174/1381612824666180702154129.

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Dyslipidemia is widely accepted as one of the major risk factors in cardiovascular disease mainly due to its contribution in the pathogenesis of atherosclerosis in medium-sized and large arteries. However, it has become increasingly accepted that high-cholesterol levels can also adversely affect the microvasculature prior to the development of overt atherosclerosis. Moreover, hypercholesterolemia has shown, in preclinical animal models, to exert detrimental effects beyond the vascular tree leading to larger infarcts and adverse cardiac remodeling post-myocardial infarction. At a functional level, hypercholesterolemia has shown to impair endotheliumdependent vasodilation because on defects on nitric oxide bioavailability. The pathogenic mechanisms underlying microvascular dysfunction involve an enhanced arginase activity, enhanced production of free radicals and the activation, recruitment and accumulation of leukocytes, primarily neutrophils, via their diffusion through postcapillary venules. In turn, recruited inflammatory cells and certain inflammatory mediators enhance platelet adhesion, overall inducing a proinflammatory and prothrombotic phenotype. Within the present review, we aim to discuss the existing evidence regarding the presence of dyslipidemia - particularly high low density lipoprotein-cholesterol levels - and the occurrence of microvascular dysfunction, the mechanism by which high cholesterol levels induce functional alterations in the microvascular bed and, finally comment on the impact of dislipidemia-induced microvascular dysfunction at the myocardial level.

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24

SUGII, Yasuhiko. "Visualization of Microcirculation." Journal of the Visualization Society of Japan 34, no. 134 (2014): 2–9. http://dx.doi.org/10.3154/jvs.34.2.

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25

Lionnet, François. "Drépanocytose et microcirculation." JMV-Journal de Médecine Vasculaire 47 (March 2022): S26. http://dx.doi.org/10.1016/j.jdmv.2022.01.133.

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26

Nam, Karam, and Yunseok Jeon. "Microcirculation during surgery." Anesthesia and Pain Medicine 17, no. 1 (January 31, 2022): 24–34. http://dx.doi.org/10.17085/apm.22127.

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Throughout the long history of surgery, there has been great advancement in the hemodynamic management of surgical patients. Traditionally, hemodynamic management has focused on macrocirculatory monitoring and intervention to maintain appropriate oxygen delivery. However, even after optimization of macro-hemodynamic parameters, microcirculatory dysfunction, which is related to higher postoperative complications, occurs in some patients. Although the clinical significance of microcirculatory dysfunction has been well reported, little is known about interventions to recover microcirculation and prevent microcirculatory dysfunction. This may be at least partly caused by the fact that the feasibility of monitoring tools to evaluate microcirculation is still insufficient for use in routine clinical practice. However, considering recent advancements in these research fields, with more popular use of microcirculation monitoring and more clinical trials, clinicians may better understand and manage microcirculation in surgical patients in the future. In this review, we describe currently available methods for microcirculatory evaluation. The current knowledge on the clinical relevance of microcirculatory alterations has been summarized based on previous studies in various clinical settings. In the latter part, pharmacological and clinical interventions to improve or restore microcirculation are also presented.

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27

Wade, J. "Stroke and Microcirculation." Journal of Neurology, Neurosurgery & Psychiatry 51, no. 9 (September 1, 1988): 1244. http://dx.doi.org/10.1136/jnnp.51.9.1244-a.

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28

Fallowfield, M. E., and M. G. Cook. "Microcirculation and prognosis." Melanoma Research 3, no. 1 (March 1993): 87. http://dx.doi.org/10.1097/00008390-199303000-00323.

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29

Gutterman, David D., Dawid S. Chabowski, Andrew O. Kadlec, Matthew J. Durand, Julie K. Freed, Karima Ait-Aissa, and Andreas M. Beyer. "The Human Microcirculation." Circulation Research 118, no. 1 (January 8, 2016): 157–72. http://dx.doi.org/10.1161/circresaha.115.305364.

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30

Zafrani, Lara, and Can Ince. "The Traumatic Microcirculation*." Critical Care Medicine 42, no. 6 (June 2014): 1556–57. http://dx.doi.org/10.1097/ccm.0000000000000273.

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31

Suzuki, Hidekazu, Masayuki Suzuki, Hiroyuki Imaeda, and Toshifumi Hibi. "Helicobacter pyloriand Microcirculation." Microcirculation 16, no. 7 (January 2009): 547–58. http://dx.doi.org/10.1080/10739680902949953.

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32

Tooke, J. E. "Microcirculation and diabetes." British Medical Bulletin 45, no. 1 (1989): 206–23. http://dx.doi.org/10.1093/oxfordjournals.bmb.a072313.

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33

Zimmerhackl, Bernd L., Channing R. Robertson, and Rex L. Jamison. "The medullary microcirculation." Kidney International 31, no. 2 (February 1987): 641–47. http://dx.doi.org/10.1038/ki.1987.46.

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34

Eriksson, Elof, Guenter Germann, and Aruna Mathur. "Microcirculation in Muscle." Annals of Plastic Surgery 17, no. 1 (July 1986): 13–16. http://dx.doi.org/10.1097/00000637-198607000-00004.

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35

Wilson, J. W., and S. J. Wilson. "The bronchial microcirculation." Clinical & Experimental Allergy Reviews 1, no. 2 (July 2001): 120–22. http://dx.doi.org/10.1046/j.1472-9725.2001.00021.x.

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36

Poole, David C. "Skeletal Muscle Microcirculation." Medicine & Science in Sports & Exercise 40, Supplement (May 2008): 38. http://dx.doi.org/10.1249/01.mss.0000320852.48150.8c.

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37

Lokmic, Zerina, and Geraldine M. Mitchell. "Engineering the Microcirculation." Tissue Engineering Part B: Reviews 14, no. 1 (March 2008): 87–103. http://dx.doi.org/10.1089/teb.2007.0299.

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38

Špiranec Spes, Katarina, Wen Chen, Lisa Krebes, Katharina Völker, Marco Abeßer, Petra Eder Negrin, Antonella Cellini, et al. "Heart-Microcirculation Connection." Hypertension 76, no. 5 (November 2020): 1637–48. http://dx.doi.org/10.1161/hypertensionaha.120.15772.

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Cardiac ANP (atrial natriuretic peptide) moderates arterial blood pressure. The mechanisms mediating its hypotensive effects are complex and involve inhibition of the renin-angiotensin-aldosterone system, increased natriuresis, endothelial permeability, and vasodilatation. The contribution of the direct vasodilating effects of ANP to blood pressure homeostasis is controversial because variable levels of the ANP receptor, GC-A (guanylyl cyclase-A), are expressed among vascular beds. Here, we show that ANP stimulates GC-A/cyclic GMP signaling in cultured microvascular pericytes and thereby the phosphorylation of the regulatory subunit of myosin phosphatase 1 by cGMP-dependent protein kinase I. Moreover, ANP prevents the calcium and contractile responses of pericytes to endothelin-1 as well as microvascular constrictions. In mice with conditional inactivation (knock-out) of GC-A in microcirculatory pericytes, such vasodilating effects of ANP on precapillary arterioles and capillaries were fully abolished. Concordantly, these mice have increased blood pressure despite preserved renal excretory function. Furthermore, acute intravascular volume expansion, which caused release of cardiac ANP, did not affect blood pressure of control mice but provoked hypertensive reactions in pericyte GC-A knock-out littermates. We conclude that GC-A/cGMP–dependent modulation of pericytes and microcirculatory tone contributes to the acute and chronic moderation of arterial blood pressure by ANP. Graphic Abstract A graphic abstract is available for this article.

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39

Ince, Can. "The Microcirculation Unveiled." American Journal of Respiratory and Critical Care Medicine 166, no. 1 (July 2002): 1–2. http://dx.doi.org/10.1164/rccm.2204033.

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40

Braverman, Irwin M. "The Cutaneous Microcirculation." Journal of Investigative Dermatology Symposium Proceedings 5, no. 1 (December 2000): 3–9. http://dx.doi.org/10.1046/j.1087-0024.2000.00010.x.

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41

Popel, A. S., A. R. Pries, and D. W. Slaaf. "Microcirculation Physiome Project." Journal of Vascular Research 36, no. 3 (1999): 253–55. http://dx.doi.org/10.1159/000025649.

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42

Keske, Michelle, K. A. Sjøberg, B. Kiens, E. A. Richter, S. M. Richards, and S. Rattigan. "Microcirculation and obesity." Obesity Research & Clinical Practice 5 (October 2011): 8. http://dx.doi.org/10.1016/j.orcp.2011.08.067.

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43

Casillas, J. M., B. Vergès, V. Dulieu, S. Vigier, and S. Febvre. "Microcirculation et artériopathie." Annales de Réadaptation et de Médecine Physique 42, no. 7 (September 1999): 424. http://dx.doi.org/10.1016/s0168-6054(99)85139-3.

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44

Girault, Véronique. "HTA et microcirculation." Archives des Maladies du Coeur et des Vaisseaux - Pratique 2005, no. 136 (February 2005): 28. http://dx.doi.org/10.1016/s1261-694x(05)88047-2.

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45

Vicaut, E., and N. Baudry. "Microcirculation et sepsis." Réanimation Urgences 3, no. 2 (January 1994): 63–68. http://dx.doi.org/10.1016/s1164-6756(05)80772-8.

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46

Vicaut, E., and N. Baudry. "Microcirculation et sepsis." Réanimation Urgences 8, no. 2 (May 1999): 129–31. http://dx.doi.org/10.1016/s1164-6756(99)80041-3.

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47

Chan-Ling, Tailoi, Mary E. Gerritsen, Caryl E. Hill, Paul Kubes, and Michael Perry. "Microcirculation down under." Trends in Pharmacological Sciences 23, no. 3 (March 2002): 106–8. http://dx.doi.org/10.1016/s0165-6147(00)01978-7.

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48

Popel, Aleksander S., and Paul C. Johnson. "MICROCIRCULATION AND HEMORHEOLOGY." Annual Review of Fluid Mechanics 37, no. 1 (January 2005): 43–69. http://dx.doi.org/10.1146/annurev.fluid.37.042604.133933.

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49

Daniel, S. E. "Stroke and Microcirculation." Journal of Clinical Pathology 42, no. 2 (February 1, 1989): 223. http://dx.doi.org/10.1136/jcp.42.2.223-a.

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50

Struijker-Boudier, Harry A. J., Bart F. J. Heijnen, Yan-Ping Liu, and Jan A. Staessen. "Phenotyping the Microcirculation." Hypertension 60, no. 2 (August 2012): 523–27. http://dx.doi.org/10.1161/hypertensionaha.111.188482.

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