Relevant bibliographies by topics / Mice – Embryology

Academic literature on the topic 'Mice – Embryology'

Author: Grafiati
Published: 4 June 2021
Last updated: 29 January 2023

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Journal articles on the topic "Mice – Embryology"

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Salvadori, Maria Letícia Baptista, Thais Borges Lessa, Fabiele Baldino Russo, Renata Avancini Fernandes, José Roberto Kfoury, Patricia Cristina Baleeiro Beltrão Braga, and Maria Angélica Miglino. "Mice embryology: A microscopic overview." Microscopy Research and Technique 75, no. 10 (June 22, 2012): 1437–44. http://dx.doi.org/10.1002/jemt.22087.

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Martins, José Luiz, Maurício Macedo, and Edna Frasson de Souza Montero. "Anorectal Malformation: State of the Art in Translating Experimental Research to the Bedside." European Journal of Pediatric Surgery 29, no. 04 (August 2019): 368–70. http://dx.doi.org/10.1055/s-0039-1694743.

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AbstractThe embryology of anorectal malformation (ARM) is a controversial issue. The study in humans is difficult due to the scarcity of fetuses with this anomaly. Therefore, ARM animal models, naturally obtained or induced by drugs, have been employed to understand physiopathology and possible treatments. Pigs, rabbits, rats, and mice have been employed as animal models. Additionally, many drugs have been used with this purpose: Etretinate, Ethylenethiourea, and Adriamycin. The animal more frequently used is the rat because of good reproducibility, low cost, and easy handling. Pig is a good model, but it is expensive, and difficult to handling and lodging. Concerning the drugs, Adriamycin promotes a more severe ARM compared with Ethylenethiourea. The models of ARM are of value in the understanding of the embryologic development. Nowadays, researches are aimed at identifying the molecular mechanism of this process, providing the basis for the application of tissue engineering in future experiments with ARM.
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Helppi, Jussi, Ronald Naumann, and Oliver Zierau. "Phytoestrogen-containing diets offer benefits for mouse embryology but lead to fewer offspring being produced." Laboratory Animals 54, no. 6 (February 12, 2020): 536–45. http://dx.doi.org/10.1177/0023677219898486.

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One of the most commonly used protein sources in rodent diets is soy, which is naturally rich in phytoestrogens. Although phytoestrogens have shown potential health benefits in humans, they may also have the ability to disrupt reproduction. Consequently, there has been a tendency to try to exclude them from rodent diets. In the current study, we investigated whether phytoestrogen content in the mouse diet could affect reproduction in mice used as embryo donors. Donor mice (C57BL/6JOlaHsd) were maintained with three different diets: high phytoestrogen (ca. 400 mg/kg genistein), low phytoestrogen (ca. 10 mg/kg genistein) and standard breeding diet (ca. 120 mg/kg genistein). Mice fed a high phytoestrogen diet had a high yield of plugs, embryos, and injectable embryos, as well as producing good quality embryos. Results from donor mice fed a low phytoestrogen diet were consistently but only slightly inferior, whereas mice fed a standard diet performed the poorest. Interestingly, the largest number of born and weaned offspring were observed when recipient females received embryos from the standard diet group. Sperm yield and quality of stud males did not differ between the groups. We surmize that for experimental endpoints requiring fertilized embryos it may be more beneficial to feed mice a diet containing phytoestrogen, but if the goal is to produce transgenic mice, a diet high in phytoestrogen may be inadvisable. In conclusion, care should be taken when selecting a diet for experimental mouse colonies as phytoestrogen could influence the study outcome.
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Godin, Isabelle, and Ana Cumano. "Of birds and mice: hematopoietic stem cell development." International Journal of Developmental Biology 49, no. 2-3 (2005): 251–57. http://dx.doi.org/10.1387/ijdb.041945ig.

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Song, Haichen, Yu Xu, Wenchuan Chang, Junli Zhuang, and Xiaowei Wu. "Negative pressure wound therapy promotes wound healing by suppressing macrophage inflammation in diabetic ulcers." Regenerative Medicine 15, no. 12 (December 2020): 2341–49. http://dx.doi.org/10.2217/rme-2020-0050.

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Aim: This work aims to explore the biological role of negative pressure wound therapy (NPWT) in the treatment of diabetic ulcer. Materials & methods: Full-thickness skin defects were created in diabetic (db/db) and non diabetic (db/m) mice to create wound models. The mice were received NPWT or rapamycin injection. Mouse macrophage cells (Raw264.7) were treated with lipopolysaccharide to induce inflammatory response, and then received negative pressure treatment. We observed the wound healing of mice and examined gene and protein expression and CD68+ macrophage levels. Results: NPWT notably enhanced the wound closure ratio, and inhibited the LC3-II/LC3-I ratio and Beclin-1 expression in diabetes mellitus (DM) mice. NPWT decreased CD68+ macrophage levels in wound tissues of DM mice. The influence conferred by NPWT was abolished by rapamycin treatment. Negative pressure repressed the LC3-II/LC3-I ratio and the expression of Beclin-1, TNF-α, IL-6 and IL-1β in the Raw264.7 cells. Conclusion: NPWT promotes wound healing by suppressing autophagy and macrophage inflammation in DM.
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Nakajima, Mitsunari, Shigeki Yuasa, Masaya Ueno, Nobuyuki Takakura, Haruhiko Koseki, and Takuji Shirasawa. "Abnormal blood vessel development in mice lacking presenilin-1." Mechanisms of Development 120, no. 6 (June 2003): 657–67. http://dx.doi.org/10.1016/s0925-4773(03)00064-9.

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Anzai, Hiroko, Akihide Kamiya, Haruki Shirato, Takashi Takeuchi, and Atsushi Miyajima. "Impaired differentiation of fetal hepatocytes in homozygous jumonji mice." Mechanisms of Development 120, no. 7 (July 2003): 791–800. http://dx.doi.org/10.1016/s0925-4773(03)00071-6.

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Walker, Kenneth, Georgina Caruana, Sunder Sims-Lucas, Mai Sarraj, John Bertram, and Kaye Stenvers. "06-P014 High nephron number in betaglycan heterozygous mice." Mechanisms of Development 126 (August 2009): S124. http://dx.doi.org/10.1016/j.mod.2009.06.240.

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Simpson, Kaylene J. "MMTV-trBrca1 mice display strain-dependent abnormalities in vaginal development." International Journal of Developmental Biology 48, no. 7 (2004): 675–78. http://dx.doi.org/10.1387/ijdb.041849ks.

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Tamura, Shinobu, Yoshihiro Morikawa, Minoru Tanaka, Atsushi Miyajima, and Emiko Senba. "Developmental expression pattern of oncostatin M receptor β in mice." Mechanisms of Development 115, no. 1-2 (July 2002): 127–31. http://dx.doi.org/10.1016/s0925-4773(02)00081-3.

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Dissertations / Theses on the topic "Mice – Embryology"

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Poirier, Luc. "The degradation of the stem-loop binding protein at the late 2-cell stage of mouse embryogenesis /." Thesis, McGill University, 2003. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=80351.

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The efficient processing of replication-dependent histone mRNA requires the Stem-Loop Binding Protein (SLBP). SLBP is also involved in regulating histone mRNA half-life, their nucleocytoplasmic transport, and their translation. Unlike somatic cells, where SLBP protein accumulates only in S-phase, SLBP protein is present throughout the first two embryonic cell cycles in mice. We report here that in late 2-cell mouse embryos there is a substantial, proteasome-dependent decrease in SLBP throughout the cell. Based on chromosome morphology, the degradation of SLBP protein in late 2-cell embryos is most likely a late G2-phase event. The degradation of SLBP protein is not simply a zygotic clock event, but requires development to the late 2-cell stage. Furthermore, SLBP protein degradation in 2-cell mouse embryos requires cyclin-dependent kinase (Cdk) activity, DNA replication, and zygotic genome activation. A model for SLBP protein degradation is proposed based on observations made in both early mouse embryos and somatic cells.
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Joyce, Bradley. "Elucidating the molecular mechanisms underlying cell movements during early embryogenesis." Thesis, University of Oxford, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.589616.

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The anterior visceral endoderm (AVE) is a specialised subpopulation of the visceral endoderm (VE), a single layer simple epithelium that surrounds the extra-embryonic ectoderm and epiblast of the egg cylinder stage embryo. Initially induced at the distal tip of the egg cylinder, AVE cells undergo a stereotypic migration towards the prospective anterior, stopping at the interface between the underlying epiblast and extra-embryonic ectoderm (ExE). Previous research has shown that membrane enrichment of Dvl2 is present in the VE overlying the epiblast (Epi-VE). In this thesis I confirm the presence of planar cell polarity (pep) signalling in this region by assaying the subcellular localisation of additional core pep proteins Vangl2 and Daaml. I show that null embryos of the Nodal antagonist Lefty1 exhibit ectopic membrane enrichment of Dvl2 and a previously unreported AVE over-migration phenotype. Furthermore, using pharmacological inhibition of Nodal signalling I show that the TGF~ protein Nodal modulates pep signalling in the YE. Utilising DIe and confocal microscopy I perform detailed time-lapse analyses of the VE to quantify the dynamic cell behaviour and topology. Using this assay I show that wild-type embryos exhibit dynamic cell movement, which is regionally restricted to the Epi-VE. Analysis of Leftyl-/- and ROSA26lyn-Celsr-l mutants, both of which exhibit disrupted pep signalling and AVE over-migration phenotypes, indicates that normal VE dynamics and topology are disrupted. The results of this quantitation indicate that these mutants exhibit increased cell migration and neighbour exchange across the YE. These data show that regional restriction of movement is lost and results in the AVE over-migration phenotypes observed. Together these results show that regionally restricted pep signalling in the VE acts to modulate cell behaviour and topology, which in turn determines the regional restriction and normal end-point of AVE migration.
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Chan, Siu-yuen. "Effects of prostaglandins on peri-implantation development of mouse embryos /." [Hong Kong : University of Hong Kong], 1989. http://sunzi.lib.hku.hk/hkuto/record.jsp?B12730191.

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McClellan, Kelly Anne. "Murine oocyte loss occurs during fetal development." Thesis, McGill University, 2003. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=79047.

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Recently, the timing of oocyte loss during murine development has been brought into question as authors using mouse vasa homologue (MVH) as a germ cell marker did not observe a loss of oocytes during fetal life. Instead the major loss was observed in the days following birth, after chromosome pairing has occurred.
In this study the controversy was addressed by establishing a new and reliable method to quantify murine oocytes in meiotic prophase, as well as to determine the gestation age and meiotic prophase stage of oocyte loss. Earlier limitations were overcome through the use of Germ Cell Nuclear Antigen-1 (GCNA-1) antibody as a germ cell specific marker, and the novel addition of a cytospin centrifugation step to the method. Progress through meiotic prophase was examined in chromosome spread preparations where meiotic stages were assessed using an antiserum against synaptonemal complex (SC) proteins. Quantification was accomplished by counting the number of GCNA-1 immunoreactive cells in chromosome spread preparations and estimated in histological sections using the ratio estimation model. (Abstract shortened by UMI.)
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Fuku, Eiji. "Studies on the preservation of mammalian embryos in the supercooled state." Thesis, McGill University, 1991. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=60523.

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Firstly, exposure of compacted morulae (CM) and early blastocysts (EB) to methanol (M) or glycerol (G) for 1 h at room temperature followed by culture in standard culture medium showed that the embryos tolerated up to 12 to 24% methanol or 24 to 48% glycerol. Next, the effects of stage of embryo (CM vs EB), preservation temperature and concentration of 1:1 M:G on embryo survival were tested. EB survived longer than CM under all conditions. Increased concentrations of cryoprotectants (M and G) increased the survival of supercooled embryos, but survival was decreased with the storage temperature. Replacing G with propanediol (P) significantly increased blastocyst survival at lower temperatures.
Exposure for 1 h to $>$ 0.6 M of sucrose or trehalose at room temperature suppressed growth in culture, but dehydration in up to 0.4M sucrose before supercooling (in M:P) increased survival at $-$5 or $-$10$ sp circ$C, survival increasing with dehydration.
Finally, demi-embryos and intact embryos were cultured to the blastocyst stage, stored at $-$5$ sp circ$ for 48 h, then cultured for 24 h and transferred into pseudopregnant recipients.
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Chia, Gloryn Le Bin. "Investigating the role of Oct4 during lineage specification in the physiological context of mouse embryonic development." Thesis, University of Cambridge, 2013. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.607990.

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Yu, Hing-Sing. "Studies on the toxicity and teratogenicity of cadmium on mouse pre-embryos in vitro and in vivo with special reference to their subsequent development /." [Hong Kong] : University of Hong Kong, 1987. http://sunzi.lib.hku.hk/hkuto/record.jsp?B1221579X.

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Fu, Germaine 1976. "Mouse oocytes and embryos with or without the H10 gene : linker histone subtypes and development performance." Thesis, McGill University, 2000. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=33399.

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H1 histones are potentially significant to nuclear reprogramming during the oocyte-to-embryo transition. One characteristic distinguishing the H1 subtypes is that the somatic H1 histones are found primarily in dividing cells, whereas the H10 subtype is predominantly found in differentiated cells. The H1 complement in mouse oocytes and preimplantation embryos from wild-type and H10-/- animals was investigated.
Immunocytochemistry of wild-type cells demonstrated that H10 was predominant in oocytes while somatic H1 began accumulating in the 2-cell embryo. In H10-/- cells H10 was not detected, but, surprisingly, somatic H1 was detected beginning at the 1-cell stage. Radiolabeling of wild-type and H10-/- cells revealed that somatic H1 synthesis intensified after meiotic maturation, and therefore prior to its detection in embryos. The functional study found that loss of H10 impaired oogenesis but enhanced embryogenesis. The patterns of H1 immunodetection and synthesis are integrated, and the significance of H1 composition in development is discussed.
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McLay, David W. "Developmental regulation and molecular nature of an activity in murine oocytes that transfers histones onto sperm DNA." Thesis, McGill University, 2001. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=38235.

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At fertilization, the remodelling of the sperm nucleus into the male pronucleus is critical for normal development. Morphological and functional changes to the nucleus are underpinned by biochemical changes in the chromatin composition, most notably the removal of sperm specific protamines and assembly of histones onto the paternal DNA. This exchange is controlled by oocyte factors, as exemplified in Xenopus by nucleoplasmin. Though mammalian factors remain unidentified, a functional assay based on antibodies recognizing core histones has been developed to test the activity in oocytes that transfers histones onto sperm DNA, named histone transfer activity (HTA). The assay was applied to growing and maturing murine oocytes to determine when during oogenesis HTA develops, and to probe potential regulatory mechanisms. Fully-grown oocytes develop HTA upon maturation, in a protein-synthesis dependent manner. Large, growing oocytes also develop HTA upon entry into M-phase. Small growing meiotically incompetent oocytes, ones that do not spontaneously enter M-phase, do not develop HTA, though this can be overcome by culture of oocytes to meiotic competence, or by treatment with strontium to induce intracellular calcium oscillations. Taken together these findings form a model of how HTA develops throughout oogenesis. Finally, an attempt is made to identify a potential mammalian HTA factor. Transcripts for two remodelling factors, mNAP and Npm3, are identified in the murine oocyte, and injection of anti-sense oligonucleotides reveals that Npm3 plays a significant role in the deposition of histories and the remodelling of sperm chromatin at fertilization. Combined with the findings of the HTA assay, the data forms a testable model of how Npm3 may be regulated throughout oogenesis.
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Cheong, Wan-yee Ana, and 張韻怡. "A study on the embryotrophic action of the complement component-3 derivative (iC3b) in the preimplantation mouse embryo development." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2009. http://hub.hku.hk/bib/B44231994.

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Books on the topic "Mice – Embryology"

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Bürki, Kurt. Experimental embryology of the mouse. Basel: Karger, 1986.

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Gilmore, McKinnell Robert, ed. Cloning of frogs, mice, and other animals. Minneapolis: University of Minnesota Press, 1985.

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The mouse: Its reproduction and development. Oxford [England]: Oxford University Press, 1990.

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L, Bard Jonathan B., ed. The anatomical basis of mouse development. San Diego, CA: Academic Press, 1999.

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The house mouse: Atlas of embryonic development. New York: Springer-Verlag, 1989.

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Ormestad, Mattias. FoxF genes in embryonic development. Göteborg: Department of Cell and Molecular Biology, Göteborg University, 2006.

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service), ScienceDirect (Online, ed. Guide to techniques in mouse development: Mouse molecular genetics. Amsterdam: Elsevier /Academic Press, 2010.

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Kaufman, Matthew H. Histologic basis of mouse endocrine system development: A comparative analysis. Boca Raton: Taylor & Francis, 2010.

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Kaufman, Matthew H. Histologic basis of mouse endocrine system development: A comparative analysis. Boca Raton: Taylor & Francis, 2010.

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Homer, Hayden A. Mammalian oocyte regulation: Methods and protocols. New York: Humana Press, 2013.

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