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Relevant bibliographies by topics / Methicillin

Academic literature on the topic 'Methicillin'

Author: Grafiati

Published: 4 June 2021

Last updated: 20 February 2023

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Journal articles on the topic "Methicillin"

1

Shah, Manish D., and Adam M. Klein. "Methicillin-resistant and methicillin-sensitiveStaphylococcus aureuslaryngitis." Laryngoscope 122, no. 11 (September 10, 2012): 2497–502. http://dx.doi.org/10.1002/lary.23537.

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Oche, D. A., U. Abdulrahim, A. S. Oheagbulem, and B. O. Olayinka. "Isolation of Biofilm Producing Methicillin-Resistant Staphylococcus aureus from Hospitalized Orthopaedic Patients in Kano State, Nigeria." Nigerian Journal of Basic and Applied Sciences 28, no. 1 (April 23, 2021): 66–74. http://dx.doi.org/10.4314/njbas.v28i1.9.

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Biofilm formation and resistance to methicillin are among the factors that makes Staphylococcus aureus a very important human pathogen in both health-care and community settings. This study investigated methicillin-resistance among biofilm-producing S. aureus isolated from 49 orthopaedic in-patients within a 3 months period. Wound swabs, nasal swabs, bed swabs and urine samples were collected from each patient. The samples were cultured and screened for presence of S. aureus while the micro-titre plate method was used to detect biofilm producing isolates. PCR technique was finally used to detect the presence of mecA gene in methicilin resistant S. aureus (MRSA) isolates. Findings reveal 14.8% of bacterial isolates were Staphylococcus aureus of which 96.4% were biofilm-producers. However, strong biofilm producers constitute 11.1%. The mecA gene was detected in 15.8% of the MRSA isolates. Therefore, MRSA among biofilm-producing S. aureus is a potential threat primarily to the community of National Orthopaedic Hospital Dala and a major public health challenge. Keywords: Biofilm, Methicillin-resistance Staphylococcus aureus (MRSA), mecA gene, Orthopaedic patient

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Young, T. "Methicillin-resistantStaphylococcusaureus." Journal of Wound Care 5, no. 10 (November 2, 1996): 475–77. http://dx.doi.org/10.12968/jowc.1996.5.10.475.

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Ash, Caroline. "Menacing Methicillin." Science 329, no. 5997 (September 9, 2010): 1261.1–1261. http://dx.doi.org/10.1126/science.329.5997.1261-a.

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Boyle-Vavra, Susan, Shouhui Yin, Dae Sun Jo, Christopher P. Montgomery, and Robert S. Daum. "VraT/YvqF Is Required for Methicillin Resistance and Activation of the VraSR Regulon in Staphylococcus aureus." Antimicrobial Agents and Chemotherapy 57, no. 1 (October 15, 2012): 83–95. http://dx.doi.org/10.1128/aac.01651-12.

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ABSTRACTStaphylococcus aureusinfections caused by strains that are resistant to all forms of penicillin, so-called methicillin-resistantS. aureus(MRSA) strains, have become common. One strategy to counter MRSA infections is to use compounds that resensitize MRSA to methicillin.S. aureusresponds to diverse classes of cell wall-inhibitory antibiotics, like methicillin, using the two-component regulatory system VraSR (vra) to up- or downregulate a set of genes (the cell wall stimulon) that presumably facilitates resistance to these antibiotics. Accordingly, VraS and VraR mutations decrease resistance to methicillin, vancomycin, and daptomycin cell wall antimicrobials.vraSandvraRare encoded together on a transcript downstream of two other genes, which we callvraUandvraT(previously calledyvqF). By producing nonpolar deletions invraUandvraTin a USA300 MRSA clinical isolate, we demonstrate thatvraTis essential for optimal expression of methicillin resistancein vitro, whereasvraUis not required for this phenotype. The deletion ofvraTalso improved the outcomes of oxacillin therapy in mouse models of lung and skin infection. SincevraTexpressed intransdid not complement avraoperon deletion, we conclude that VraT does not inactivate the antimicrobial. Genome-wide transcriptional microarray experiments reveal that VraT facilitates resistance by playing a necessary regulatory role in the VraSR-mediated cell wall stimulon. Our data prove that VraTSR comprise a novel three-component regulatory system required to facilitate resistance to cell wall agents inS. aureus. We also provide the firstin vivoproof of principle for using VraT as a sole target to resensitize MRSA to β-lactams.

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Nicolson, Kirsty, Gary Evans, and Paul W. O'Toole. "Potentiation of methicillin activity against methicillin-resistantStaphylococcus aureusby diterpenes." FEMS Microbiology Letters 179, no. 2 (October 1999): 233–39. http://dx.doi.org/10.1111/j.1574-6968.1999.tb08733.x.

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Hill, E. E., W. E. Peetermans, S. Vanderschueren, P. Claus, M. C. Herregods, and P. Herijgers. "Methicillin-resistant versus methicillin-sensitive Staphylococcus aureus infective endocarditis." European Journal of Clinical Microbiology & Infectious Diseases 27, no. 6 (January 26, 2008): 445–50. http://dx.doi.org/10.1007/s10096-007-0458-2.

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Gelfand, Michael S., and Kerry O. Cleveland. "Reversion From Methicillin Susceptibility to Methicillin Resistance inStaphylococcus aureus." Journal of Infectious Diseases 213, no. 10 (March 8, 2016): 1671.1–1671. http://dx.doi.org/10.1093/infdis/jiw051.

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Chambers, H. F. "Methicillin-resistant staphylococci." Clinical Microbiology Reviews 1, no. 2 (April 1988): 173–86. http://dx.doi.org/10.1128/cmr.1.2.173.

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Strains of staphylococci resistant to methicillin were identified immediately after introduction of this drug. Methicillin-resistant strains have unusual properties, the most notable of which is extreme variability in expression of the resistance trait. The conditions associated with this heterogeneous expression of resistance are described. Methicillin resistance is associated with production of a unique penicillin-binding protein (PBP), 2a, which is bound and inactivated only at high concentrations of beta-lactam antibiotics. PBP2a appears to be encoded by the mec determinant, which also is unique to methicillin-resistant strains. The relationships between PBP2a and expression of resistance and implications for the mechanism of resistance are discussed. The heterogeneous expression of methicillin resistance by staphylococci poses problems in the detection of resistant strains. Experience with several susceptibility test methods is reviewed and guidelines for performance of these tests are given. Treatment of infections caused by methicillin-resistant staphylococci is discussed. Vancomycin is the treatment of choice. Alternatives have been few because methicillin-resistant strains often are resistant to multiple antibiotics in addition to beta-lactam antibiotics. New agents which are active against methicillin-resistant staphylococci are becoming available, and their potential role in treatment is discussed.

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Chambers, H. F. "Methicillin-resistant staphylococci." Clinical Microbiology Reviews 1, no. 2 (1988): 173–86. http://dx.doi.org/10.1128/cmr.1.2.173-186.1988.

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Dissertations / Theses on the topic "Methicillin"

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Al-Thani, Roda. "Interstrain comparison of some methicillin-resistant and methicillin-sensitive strains of staphylococcus aureus." Thesis, University of Newcastle Upon Tyne, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.246109.

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Velasco, Valeria. "Detection and Molecular Typing of Methicillin-Susceptible Staphylococcus Aureus (MSSA) and Methicillin-Resistant Staphylococcus Aureus (MRSA)." Diss., North Dakota State University, 2015. http://hdl.handle.net/10365/24928.

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Methicillin-resistant (MRSA) and multidrug-resistant (MDR) Staphylococcus aureus, and the serotype (ST) 398 have been associated with human and livestock infections, being also detected in retail meat. The aim of this study was to determine the prevalence and molecular types of S. aureus strains from animals, retail raw meat, deli meat, and humans, determining the genetic similarity between the strains. A two-step selective enrichment followed by selective plating were used to isolate S. aureus from animals (n=167), retail raw meat (n=145), and deli meat (n=46). In addition, S. aureus from healthy people (n=550) was isolated by culture method. Positive isolates and MRSA isolates from clinical cases (n=108) were subjected to multiplex PCR (16S rRNA, mecA, and PVL genes), molecular typing and antimicrobial susceptibility testing. In addition, a real-time PCR assay was developed in order to decrease the time of detection of target genes of S. aureus in animal and meat samples, comparing the results with the standard culture/PCR method. The prevalence of S. aureus was 34.7% in animals, 47.6% in meat, and 13.0% in deli meat. The mecA gene was detected in S. aureus isolated from five pork meat samples and exhibited penicillin resistance. The ST398 was found in sheep, pigs, and pork meat. The S. aureus nasal carriage in healthy people was 7.6%. A total of 105 MRSA strains (97.2%) from clinical cases harbored the mecA gene and 11 (10.2%) the PVL gene. The rate of MDR was 70% in humans. A genetic similarity between strains from animals and meat, and from humans and meat was observed. Total agreement between the culture/PCR method and real-time PCR for detection of S. aureus was 68.9 to 97.8% (k=0.68-0.88), and the mecA gene, 86.7 to 98.7% (k=0-0.49). Therefore, the real-time PCR assay may be recommended as a rapid method for the detection of S. aureus, with confirmation of MRSA using the standard culture method. The presence of emerging S. aureus strains in the meat production chain and the genetic similarity between strains of different origin, suggests the contamination of meat, and a potential risk of transmission to humans.
Dean?s Office, College of Agriculture, Food Systems and Natural Resources, North Dakota State University

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Blank, Tiana. "Comparative Mid-term Outcomes of Pediatric Hematogenous Methicillin-resistant Staphylococcus aureus and Methicillin-susceptible Staphylococcus aureus osteomyelitis." Thesis, The University of Arizona, 2018. http://hdl.handle.net/10150/626843.

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Weiß, Susanne. "Mortalität und Morbidität von chronischen Dialysepatienten bei Besiedlung mit Methicillin-sensiblem Staphylococcus aureus sowie Methicillin-resistentem Staphylococcus aureus." Doctoral thesis, Universitätsbibliothek Leipzig, 2016. http://nbn-resolving.de/urn:nbn:de:bsz:15-qucosa-197315.

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Systemische Infektionen mit S. aureus (MSSA und MRSA) und Infektionen des Gefäßzugangs bei HD-Patienten sind eine der wichtigsten Ursachen für Morbidität und Mortalität in dieser speziellen Population. Infektionsrisikos stellen die zunehmende Verwendung von Fremdkörpern, wie Katheter und Graft als Gefäßzugänge, sowie die intensivmedizinische Behandlung bei älteren und multimorbiden Patienten dar. Unter den bakteriell bedingten Infektionen bleiben Staphylokokken der am häufigsten nachgewiesene Stamm. Mit dem zunehmenden Gebrauch von Vancomycin zur Behandlung von MSSA-Infektionen hat das Vorkommen von MRSA zugenommen. Dies macht die Entwicklung von alternativen Antibiotikaregimen nötig, die eine Selektion von MRSA-Spezies verhindern. Unter dieser Überlegung wurde auf die Behandlung mit Vancomycin bei Zugangs-bezogenen Infektionen verzichtet. Es wurde im Jahr 2000 durch ein Standardregime bestehend aus Flucloxacillin und Rifampicin ersetzt. Mithilfe eines Screeningprogramms wurde nach MSSA- (n=88) und MRSA- (n=1) Kolonisationen gesucht. Dies gelang mit Hilfe von Querschnitts-Screenings und Indikations-Screeninguntersuchungen bei Aufnahme über den Zeitraum von 2000 bis 2010. Eine Besiedlung mit MRSA wurde bei nur einem Patienten während des 10-Jahres-Screenings registriert. Die gefundenen MSSA-Kolonisationen bei HD-Patienten beeinflussten die Morbidität und Mortalität nicht. Die Anzahl an HD-Patienten mit MSSA-Kolonisation nahm während des Beobachtungszeitraums von zehn Jahren ab Behandlungen mit dem Vancomycin-freien Regime waren generell erfolgreich und resultierten in einem Rückgang der klinischen und laborativen Infektionsmarker und/oder negativen Blutkulturen. Es konnte gezeigt werden, dass mit dem Gebrauch von vancomycinfreien Antibiotikaregimen ein erfolgreiches Management von Staphylokokkus-assoziierten Zugangsinfektionen bei HD-Patienten möglich ist.

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Gaisford, Wendy Caron. "Aspects of methicillin resistance in Staphylococcus epidermis." Thesis, University of Cambridge, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.302972.

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劉昌志 and Cheong-chi Lau. "Epidemiology of community-associated methicillin resistant staphylococcus aureus (CA-MRSA) infection in Hong Kong, 2007: a descriptive and analytical study." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2008. http://hub.hku.hk/bib/B41710484.

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Lau, Cheong-chi. "Epidemiology of community-associated methicillin resistant staphylococcus aureus (CA-MRSA) infection in Hong Kong, 2007 a descriptive and analytical study /." Click to view the E-thesis via HKUTO, 2008. http://sunzi.lib.hku.hk/hkuto/record/B41710484.

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Alshami, Issam Abdulla. "The pathogenesis of methicillin resistant staphylococcus aureus infection." Thesis, University of Manchester, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.595299.

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Outbreaks with strains of methicillin resistant Staphylococcus aureus (MRSA) began in a London hospital in 1982 and continue to be associated with significant morbidity and mortality. These particular strains, termed epidemic methicillin resistant S. aureus (EMRSA), are recognised by their phage type and antibiogram. This study examined the ability of isolates representing EMRSA strains recovered from different outbreaks in England and Wales (EMRSA 1-16) to grow in escalating levels of vancomycin. A series of the EMRSA strains were subjected to passage-selection in vancomycin. Six strains became vancomycin resistant, three became vancomycin intermediate, and seven remained susceptible, which confirmed that EMRSA strains were able to develop resistance to vancomycin in vitro. The vancomycin MICs for the vancomycin-resistant derivatives ranged from 24 -32 ug/ml, were associated with decreased lysostaphin susceptibilities and increased cell wall thickness. The MICs of teicoplanin was also increased. Five of the six vancomycin-resistant derivatives became susceptible to methicillin with instability of the S. aureus mecA gene. There were no significant changes to other antimicrobial agents susceptibility, including Linezolid. During this study an attempt was made to elucidate the development of resistance to vancomycin by studying the heterogeneous phenotype of resistance. Population analysis profiles for the vancomycin-resistant derivatives demonstrated that four of them expressed a heterogeneous subpopulation. Bacteriophage typing failed to type vancomycin-resistant clones, while Pulsed-field gel electrophoresis (PFGE) gave good discrimination. In conclusion, we found that, although vancomycin is usually bactericidal against S. aureus, resistance can be common amongst MRSA isolates. Decreased susceptibility to vancomycin appeared to be a selective or inducible process that increased during persistent sublethal exposure to vancomycin. Thus, if this effect occurs in vivo, then not only is vancomycin therapy of serious EMRSA infection compromised, but the propensity to develop resistance may remain undetected by standard laboratory sensitivity testing.

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Sinclair, Grant Richard. "Characterisation of epidemic methicillin resistant Staphylococcus aureus clones." Thesis, Glasgow Caledonian University, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.499572.

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Estuary is a unique area of important economic, cultural and environ- mental value. It is also a region with very complicated hydrodynamic mechanisms, partly due to the interaction of freshwater and seawater. Much research e®ort has been invested in improving the application of estuary and preventing estuarine environment from any undue damage. With the rapid development of computational resources, mathematical model has become a popular approach used for the investigation of es- tuarine hydrodynamics. The aim of this research study is to set up the numerical models which can be used for investigating the saline-wedge purging process, astronomical tidal circulation and typhoon-induced storm surge in the estuarine regions. Two mathematical models have been developed for this purpose. One objective of this project is to set up a two-dimensional model for exploring the °ushing process of trapped saltwater subject to upstream freshwater turbulent °ow. Most numerical simulations currently ap- plied are based on single-phase models, which are not suitable for the two-phase °ow before the mixture of saltwater and freshwater. The multiphase Eulerian model, a part of commercial code FLUENT6.2, has been applied for the ¯rst time to study this complex mixing inter- action in estuary. The distinguishing characteristic of this model is to treat saltwater and freshwater as two single miscible phases instead of a mixture phase with density variation, and the advantage of using a multiphase approach over a single-phase model is that it can e±ciently and accurately treat both the free water surface and relatively high density excess between two °uids simultaneously. The other objective of this project is to develop a three-dimensional model based on the FVCOM open source code, with the aim to better understand the estuarine hydrodynamics with or without the presence of typhoon. It is found that the original FVCOM code can not re- produce an accurate tidal hydrodynamics in estuary, mainly due to the inaccurate calculation of bottom friction in shallow water. To overcome this di±culty, an improved simulation of the bed friction has been in- corporated into the existing code for estuarine tide. This model has also been developed by including air-pressure gradient term to study the hydrodynamic response to cyclonic typhoon. To include the e®ect of typhoon (wind stress and pressure de¯cit), a symmetrical cyclone model is adopted. However, the typhoon-induced wind ¯eld has been predicted poorly when the typhoon enters the near-shore region. This is because the typhoon quickly loses its symmetrical property in the near-shore region. To overcome this di±culty, an asymmetrical cyclone model is derived on the basis of characteristic isobar. The accuracy of open sea boundary for storm surge model has also been improved by using large scale model. The numerical models have been compared with laboratory experiments or ¯eld observations. The comparison results show a good agreement numerical simulations and physical measurements. It is anticipated that the models developed in this research can make signi¯cant contribution in estuarine application and protection.

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Shubin, P., and E. Nazbar. "The problem of proliferation methicillin-resistant S. aureus." Thesis, Sumy state university, 2017. http://essuir.sumdu.edu.ua/handle/123456789/54026.

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Staphylococci are natural inhabitant of human and animal skin but sometimes they can cause infections affecting many organs (endocarditis, toxic shock syndrome, sepsis, pneumonia and arthritis). Most staphylococci are responsible for skin infections such as boil, carbuncle, and furuncle and some cause food poisoning resulting in severe vomiting and diarrhea. Staphylococci also cause mastitis in cow and also cause joint infection leading to edema and arthritis. An emerging problem in treating S. aureus infections is the increasing resistance against antibiotics.

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Books on the topic "Methicillin"

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Maurice, Rapin Colloquia (4th 1994 Les Baux-de-Provence France). Methicillin resistant staphylococci. Paris: Flammarion médecine-sciences, 1995.

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Wolseley, Nicola Anne. Effects of mupirocin on methicillin sensitive and methicillin resistant staphylococci. [s.l: The Author], 1991.

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Methicillin-resistant staphylococcus aureus (MRSA) protocols. New York: Springer, 2014.

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Yinduo, Ji. Methicillin-Resistant Staphylococcus aureus (MRSA) Protocols. New Jersey: Humana Press, 2007. http://dx.doi.org/10.1385/1597454680.

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McKane, Ann Marie. Study of methicillin resistant staphyloccus aureus. [S.l: The Author], 1995.

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Ji, Yinduo, ed. Methicillin-Resistant Staphylococcus Aureus (MRSA) Protocols. Totowa, NJ: Humana Press, 2014. http://dx.doi.org/10.1007/978-1-62703-664-1.

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Ji, Yinduo, ed. Methicillin-Resistant Staphylococcus aureus (MRSA) Protocols. Totowa, NJ: Humana Press, 2007. http://dx.doi.org/10.1007/978-1-59745-468-1.

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Ji, Yinduo, ed. Methicillin-Resistant Staphylococcus Aureus (MRSA) Protocols. New York, NY: Springer US, 2020. http://dx.doi.org/10.1007/978-1-4939-9849-4.

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Sheehan, Ray. Evaluation of mannitol salt agar with methicillin as a screening for methicillin-resistant Staphylococcus aureus. [S.l: The Author], 1993.

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Higgins, Frances. Detection of methicillin resistance in staphylococcus species. [S.l: The Author], 1995.

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Book chapters on the topic "Methicillin"

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Wood, Sara. "Methicillin-Resistant Staphylococcus aureus." In Vulvar Disease, 301–2. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-61621-6_46.

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Weese, Scott. "Methicillin-Resistant Staphylococcal Infections." In Complications in Small Animal Surgery, 39–44. Chichester, UK: John Wiley & Sons, Ltd, 2017. http://dx.doi.org/10.1002/9781119421344.ch7.

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Bensard, Denis D., Philip F. Stahel, Jorge Cerdá, Babak Sarani, Sajid Shahul, Daniel Talmor, Peter M. Hammer, et al. "Methicillin-Resistant Staphylococcus aureus (MRSA)." In Encyclopedia of Intensive Care Medicine, 1394. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-00418-6_3203.

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Riendeau, Debra. "Community-Associated, Methicillin-ResistantStaphylococcus Aureus(MRSA)." In Clinical Case Studies in Home Health Care, 377–85. West Sussex UK: John Wiley & Sons, Inc., 2013. http://dx.doi.org/10.1002/9781118785744.ch36.

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Crawford, Susan E., Susan Boyle-Vavra, and Robert S. Daum. "Community-Associated Methicillin-Resistant Staphylococcus aureus." In Emerging Infections 7, 153–79. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555815585.ch9.

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Miller, Loren Gregory, and Samantha J. Eells. "Community-Associated Methicillin-Resistant Staphylococcus aureus." In Emerging Infections 8, 229–56. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555815592.ch12.

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Bryskier, André. "Agents against Methicillin-Resistant Staphylococcus aureus." In Antimicrobial Agents, 1183–238. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555815929.ch49.

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Boyce, John M. "Methicillin-Resistant Staphylococcus Aureus: Is Control Necessary?" In Infection Control in the ICU Environment, 57–65. Boston, MA: Springer US, 2001. http://dx.doi.org/10.1007/978-1-4615-0781-9_5.

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Bonten, M. J. M., and M. C. J. Bootsma. "Nosocomial Transmission: Methicillin-Resistant Staphylococcus aureus (MRSA)." In Modern Infectious Disease Epidemiology, 395–407. New York, NY: Springer New York, 2009. http://dx.doi.org/10.1007/978-0-387-93835-6_22.

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Tacconelli, Evelind. "Risk Assessment for Methicillin-Resistant Staphylococcus aureus." In Antibiotic Policies: Fighting Resistance, 223–36. Boston, MA: Springer US, 2008. http://dx.doi.org/10.1007/978-0-387-70841-6_14.

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Conference papers on the topic "Methicillin"

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Fetsch, A., U. Roesler, B. Kraushaar, and A. Friese. "Methicillin-sensitive and Methicillin-resistant Staphylococcus aureus in pigs: (co-) colonization dynamics and clonal diversity." In Fourth International Symposium on the Epidemiology and Control of Salmonella and Other Food Borne Pathogens in Pork. Iowa State University, Digital Press, 2015. http://dx.doi.org/10.31274/safepork-180809-256.

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Dai, Y. L., Z. C. Fan, L. P. Zhang, X. Y. Xu, and Z. L. Zhang. "Improved Random Forest Algorithm to Classify Methicillin-Resistant and Methicillin-Susceptible Staphylococcus Aureus on Mass Spectra." In the 9th International Conference. New York, New York, USA: ACM Press, 2017. http://dx.doi.org/10.1145/3093293.3093300.

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Herathge, N. D. S., J. T. George, and D. A. Rowley. "Differential antimicrobial activities of Human Beta-Defensins against Methicillin Resistant (MRSA) and Methicillin sensitive (MSSA) Staphylococcus aureus." In Proceedings of the International Conference on Antimicrobial Research (ICAR2010). WORLD SCIENTIFIC, 2011. http://dx.doi.org/10.1142/9789814354868_0003.

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Akella, S., B. Bhandari, and G. Abraham. "Community Associated Methicillin-Resistant Staphylococcus Aureus (MRSA) Meningitis." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6593.

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Bai, J., Z. C. Fan, L. P. Zhang, X. Y. Xu, and Z. L. Zhang. "Classification of Methicillin-Resistant and Methicillin-Susceptible Staphylococcus Aureus Using an Improved Genetic Algorithm for Feature Selection Based on Mass Spectra." In the 9th International Conference. New York, New York, USA: ACM Press, 2017. http://dx.doi.org/10.1145/3093293.3093299.

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Garcês, Andreia, Augusto Silva, Ricardo Lopes, Filipe Sampaio, Daniela Duque, and Paula Brilhante-Simões. "Methicillin-Resistant Staphylococcus aureus (MRSA) and methicillin-resistant Staphylococcus pseudintermedius (MRSP) in Skin Infections from Company Animals in Portugal (2013–2021)." In The 2nd International Electronic Conference on Antibiotics—Drugs for Superbugs: Antibiotic Discovery, Modes of Action and Mechanisms of Resistance. Basel Switzerland: MDPI, 2022. http://dx.doi.org/10.3390/eca2022-12689.

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Bhattarai, B., J. Lamichhane, A. Mandal, P. B. Datar, O. Mukhtar, O. Alhafdh, D. Enriquez, J. B. K. Quist, and F. Schmidt. "Widespread Thrombosis in Disseminated Methicillin Resistant Staphylococcus Aureus Infection." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6579.

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Leanse, Leon G., Xueping Sharon Goh, Ji-Xin Cheng, David C. Hooper, and Tianhong Dai. "Dual-wavelength photo-killing of methicillin resistant staphylococcus aureus." In Photonic Diagnosis, Monitoring, Prevention, and Treatment of Infections and Inflammatory Diseases 2021, edited by Tianhong Dai, Mei X. Wu, and Jürgen Popp. SPIE, 2021. http://dx.doi.org/10.1117/12.2577202.

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Shui-Yee Wong, Yizhen Hai, Vincent CC Cheng, Kwok-Yung Yuen, and Kwok-Leung Tsui. "Prognostication of Methicillin-resistant Staphylococcus Aureus (MRSA) patient survival." In 2011 Prognostics and System Health Management Conference (PHM-2011 Shenzhen). IEEE, 2011. http://dx.doi.org/10.1109/phm.2011.5939586.

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Wang, Yucheng, Tianhong Dai, and Ying Gu. "Antimicrobial blue light inactivation of Methicillin-resistant Staphylococcus aureus." In SPIE/COS Photonics Asia, edited by Qingming Luo, Xingde Li, Ying Gu, and Yuguo Tang. SPIE, 2016. http://dx.doi.org/10.1117/12.2246533.

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Reports on the topic "Methicillin"

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Spencer, Jessica, and Uzo Chukwuma. Methicillin-Resistant Staphylococcus aureus (MRSA) Infections in the Department of Defense (DOD): Annual Summary 2013. Fort Belvoir, VA: Defense Technical Information Center, January 2015. http://dx.doi.org/10.21236/ada612614.

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Ju, Mohan, Yueying Huang, Xiaofeng Xu, Yiyi Qian, Yingmin Bi, Shuang Liu, Xiangyu Li, Shuaiyue Dong, Jinyi Yuan, and Dongfang Lin. Predictors of mortality in patients with Methicillin-resistant Staphylococcus aureus bloodstream infection: A meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, February 2021. http://dx.doi.org/10.37766/inplasy2021.2.0082.

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Neyra, Joan M. Nasal Colonization with Methicillin-Resistant Staphylococcus aureus in Military Personnel in a Developing Country - Development of a Skin and Soft Tissue Infection Surveillance System in the Peruvian Air Force. Fort Belvoir, VA: Defense Technical Information Center, March 2015. http://dx.doi.org/10.21236/ad1012737.

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Health hazard evaluation report: HETA-2009-0098-3103, evaluation of methicillin-resistant Staphylococcus aureus (MRSA) cases among employees at a workholding manufacturing facility, Positrol Inc., Cincinnati, Ohio. U.S. Department of Health and Human Services, Public Health Service, Centers for Disease Control and Prevention, National Institute for Occupational Safety and Health, March 2010. http://dx.doi.org/10.26616/nioshheta200900983103.

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