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Journal articles on the topic 'Metabolic processes'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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1

Zemskov, Andrei M., Tatiyana A. Berezhnova, Veronika A. Zemskova, Kseniya S. Dyadina, Yana V. Kulintsova, and Anton V. Larin. "Immune-metabolic genesis of pathological processes." Research Results in Pharmacology 5, no. 4 (December 12, 2019): 19–31. http://dx.doi.org/10.3897/rrpharmacology.5.38386.

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This article deals with metabolic-immune processes at rest and under stress conditions, which, in turn, results in the development of immune-dependent and immune-associated disorders. The article analyzes study results and conclusions of various literature sources and experimental data in healthy individuals and patients suffering from non-specific inflammatory lung diseases; purulent-inflammatory diseases and their combinations, primary and secondary progressive multiple sclerosis in the acute stage and remission. Research studies investigated the impact of the type, stage, combination of diseases on the parameters of the immunologic and metabolic statuses, as well as their correlations. The authors also analyzed metabolic effects of immunomodulators. Based on the analysis of the literature and own clinical and experimental data, the authors identified the ability of metabolic factors to regulate immunological processes. A correlative analysis of examination results of the patients with various diseases helped detect the unity of the immune-metabolic mechanisms of pathology. The data on the therapeutic effect of various modulators through differentiated biochemical chains and vice versa – the metabolic effect through immunological mechanisms –were analyzed in the study. Thus, one can testify that there is the phenomenon of a mediated effect of some immunocorrectors on the reactivity through metabolic chains. The fact that a number of modulators and metabolics can simultaneously affect the biochemical and immunological parameters of patients proved the above phenomenon. There was revealed a significant correlation interaction of the immune-metabolic parameters with various types of purulent-inflammatory diseases, which proves the formation of a single mechanism of pathology.

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2

Shipman, Jason, Jeffrey Guy, and Naji N. Abumrad. "Repair of metabolic processes." Critical Care Medicine 31, Supplement (August 2003): S512—S517. http://dx.doi.org/10.1097/01.ccm.0000081547.31084.23.

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3

Gutfreund, H. "Control of metabolic processes." FEBS Letters 284, no. 1 (June 17, 1991): 133. http://dx.doi.org/10.1016/0014-5793(91)80780-7.

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4

Sanchez, Sergio, and Arnold L. Demain. "Metabolic regulation of fermentation processes." Enzyme and Microbial Technology 31, no. 7 (December 2002): 895–906. http://dx.doi.org/10.1016/s0141-0229(02)00172-2.

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5

McHugh, Jessica. "Targeting autoimmune-specific metabolic processes." Nature Reviews Rheumatology 14, no. 12 (November 12, 2018): 686. http://dx.doi.org/10.1038/s41584-018-0126-1.

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6

Татарчук, Т. Ф., Н. Ю. Педаченко, and З. Б. Хомінська. "Metabolic syndrome and hyperproliferative endometrial processes." Reproductive Endocrinology, no. 16 (July 11, 2014): 61. http://dx.doi.org/10.18370/2309-4117.2014.16.61-69.

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7

Heinrich, Reinhart, and Christine Reder. "Metabolic control analysis of relaxation processes." Journal of Theoretical Biology 151, no. 3 (August 1991): 343–50. http://dx.doi.org/10.1016/s0022-5193(05)80383-2.

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8

Segraves, Daniel. "Data City: Urban Metabolic Decision Processes." Architectural Design 83, no. 4 (July 2013): 120–23. http://dx.doi.org/10.1002/ad.1628.

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9

Žurauskienė, Justina, Paul Kirk, Thomas Thorne, John Pinney, and Michael Stumpf. "Derivative processes for modelling metabolic fluxes." Bioinformatics 30, no. 13 (February 26, 2014): 1892–98. http://dx.doi.org/10.1093/bioinformatics/btu069.

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10

Iwatani, Shintaro, Yohei Yamada, and Yoshihiro Usuda. "Metabolic flux analysis in biotechnology processes." Biotechnology Letters 30, no. 5 (January 26, 2008): 791–99. http://dx.doi.org/10.1007/s10529-008-9633-5.

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11

Bobik, Thomas A. "Polyhedral organelles compartmenting bacterial metabolic processes." Applied Microbiology and Biotechnology 70, no. 5 (March 9, 2006): 517–25. http://dx.doi.org/10.1007/s00253-005-0295-0.

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12

Grytsay, V. I., and I. V. Musatenko. "Self-organization and fractality in a metabolic processes of the Krebs cycle." Ukrainian Biochemical Journal 85, no. 5 (October 28, 2013): 191–200. http://dx.doi.org/10.15407/ubj85.05.191.

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13

Naz, Sarwat, Shun Kishimoto, James B. Mitchell, and Murali C. Krishna. "Imaging Metabolic Processes to Predict Radiation Responses." Seminars in Radiation Oncology 29, no. 1 (January 2019): 81–89. http://dx.doi.org/10.1016/j.semradonc.2018.10.004.

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14

Brisson, Diane, Marie-Claude Vohl, Julie St-Pierre, Thomas J. Hudson, and Daniel Gaudet. "Glycerol: a neglected variable in metabolic processes?" BioEssays 23, no. 6 (2001): 534–42. http://dx.doi.org/10.1002/bies.1073.

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15

Sharma, Santosh K., Gaurav Bhushan, and Sweta Chhangani. "Genetically inborn metabolic disorders." Indian Journal of Pharmaceutical and Biological Research 6, no. 01 (March 31, 2018): 48–57. http://dx.doi.org/10.30750/ijpbr.6.1.8.

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Metabolism is the process carried out in the cells of all living organisms converting the food we eat to chemical energy needed for sustaining life. It encompasses allbiochemical processes that occur within any living organism - including humans - to maintain life. These biochemical processes allow us to grow, reproduce, repair damage, and respond to our environment.

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16

Dvorak, JA, and CP Mudd. "Microcalorimetric studies of metabolic processes in parasitic protozoa." Parasitology International 47 (August 1998): 223. http://dx.doi.org/10.1016/s1383-5769(98)80597-7.

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17

Udristioiu, Aurelian, and Manole Cojocaru. "Autophagic processes of normal and malignant metabolic pathways." African Journal of Biological Sciences 01, no. 03 (July 8, 2019): 01. http://dx.doi.org/10.33472/afjbs.1.3.2019.1-13.

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18

Jones, Ralph E., Ralph E. Beeman, and Joseph M. Suflita. "Anaerobic metabolic processes in the deep terrestrial subsurface." Geomicrobiology Journal 7, no. 1-2 (January 1989): 117–30. http://dx.doi.org/10.1080/01490458909377854.

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19

Geyer, Tihamér. "Modeling metabolic processes between molecular and systems biology." Current Opinion in Structural Biology 23, no. 2 (April 2013): 218–23. http://dx.doi.org/10.1016/j.sbi.2012.12.001.

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20

OZAKI, Koichi. "Metabolic processes of visual pigments in the retina." Seibutsu Butsuri 31, no. 4 (1991): 15–20. http://dx.doi.org/10.2142/biophys.31.4_15.

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21

Janda, S., and A. Kotyk. "Effects of suspension density on microbial metabolic processes." Folia Microbiologica 30, no. 6 (November 1985): 465–73. http://dx.doi.org/10.1007/bf02927608.

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22

Smith, Robert B., Claire Canton, Nathan S. Lawrence, Callum Livingstone, and James Davis. "Molecular anchors—mimicking metabolic processes in thiol analysis." New J. Chem. 30, no. 12 (2006): 1718–24. http://dx.doi.org/10.1039/b611471g.

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23

Thorgersen, Michael P., and Diana M. Downs. "Cobalt Targets Multiple Metabolic Processes in Salmonella enterica." Journal of Bacteriology 189, no. 21 (August 24, 2007): 7774–81. http://dx.doi.org/10.1128/jb.00962-07.

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ABSTRACT Cobalt is essential for growth of Salmonella enterica and other organisms, yet this metal can be toxic when present in excess. Wild-type Salmonella exhibits several metabolic defects when grown in the presence of cobalt, some of which generate visible growth consequences. Work herein identifies sulfur assimilation, iron homeostasis, and Fe-S cluster metabolism as targets for cobalt toxicity. In each case it is proposed that cobalt exerts its effect by one of two mechanisms: direct competition with iron or indirectly through a mechanism that involves the status of reduced thiols in the cell. Cobalt toxicity results in decreased siroheme production, increased expression of the Fur regulon, and decreased activity of Fe-S cluster proteins. The consequences of reduced sulfite reductase activity in particular are exacerbated by the need for glutathione in cobalt resistance. Significantly, independent metabolic perturbations could be detected at cobalt concentrations below those required to generate a detectable growth defect.

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24

Litvinova, Larisa Sergeevna, Maria Alexandrovna Vasilenko, Pavel Anatol'evich Zatolokin, Natalya Nikolaevna Aksenova, Nikolay Sergeevich Fattakhov, Igor Zinov'evich Vaysbeyn, Natalya Ivanovna Mironyuk, and Elena Vital'evna Kirienkova. "Adipokines in metabolic processes regulating during obesity treatment." Diabetes mellitus 17, no. 3 (June 14, 2014): 51–59. http://dx.doi.org/10.14341/dm2014351-59.

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Bariatric surgery serves as a model for the assessment of the relationship between body mass index (BMI) reduction and changes in adipokine production and for exploring the endocrine function of the pancreas in patients who do not have the proximal part of the small intestine. Aim. of the study was to assess the biochemical parameters and plasma levels of adipokines [adiponectin, adipsin, leptin, plasminogen activator inhibitor (PAI-1), resistin and visfatin], insulin, C-peptide, ghrelin and incretins [glucose insulinotropic polypeptide (GIP) and glucagon-like peptide-1 (GLP-1)] in patients with morbid obesity after surgery (gastric bypass) and therapeutic correction. Materials and methods. A total of 75 patients (34 men and 41 women; age range: 24?67 years) diagnosed as obese were divided into two groups according to the treatment they received. Biochemical analysis was performed to estimate carbohydrate and lipid metabolism rates and plasma levels of adipokines (adiponectin, adipsin, leptin, PAI-1, resistin, visfatin), insulin, C-peptide, ghrelin and incretins (GIP and GLP-1) using the flow fluorometry. Results. Surgical treatment of obesity resulted in a significant decrease in BMI (from 45.67?9.87 to 32.45?5.35 kg/m2, p

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25

Batstone, DJ, T. Hülsen, and A. Oehmen. "Metabolic modelling of mixed culture anaerobic microbial processes." Current Opinion in Biotechnology 57 (June 2019): 137–44. http://dx.doi.org/10.1016/j.copbio.2019.03.014.

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26

Berg, Hermann, Günter Horn, Hans-Egon Jacob, Ursula Fiedler, Uta Luthardt, and Dieter Tresselt. "Polarographic modelling of metabolic processes of cancerostatic anthracyclines." Bioelectrochemistry and Bioenergetics 16, no. 1 (August 1986): 135–48. http://dx.doi.org/10.1016/0302-4598(86)80052-6.

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27

Thorne, Steve. "Modeling the role of gravitation in metabolic processes." Communicative & Integrative Biology 14, no. 1 (January 1, 2021): 115–35. http://dx.doi.org/10.1080/19420889.2021.1914913.

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28

Hargreaves, Mark. "Muscle glycogen and metabolic regulation." Proceedings of the Nutrition Society 63, no. 2 (May 2004): 217–20. http://dx.doi.org/10.1079/pns2004344.

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Muscle glycogen is an important fuel for contracting skeletal muscle during prolonged strenuous exercise, and glycogen depletion has been implicated in muscle fatigue. It is also apparent that glycogen availability can exert important effects on a range of metabolic and cellular processes. These processes include carbohydrate, fat and protein metabolism during exercise, post-exercise glycogen resynthesis, excitation–contraction coupling, insulin action and gene transcription. For example, low muscle glycogen is associated with reduced muscle glycogenolysis, increased glucose and NEFA uptake and protein degradation, accelerated glycogen resynthesis, impaired excitation–contraction coupling, enhanced insulin action and potentiation of the exercise-induced increases in transcription of metabolic genes. Future studies should identify the mechanisms underlying, and the functional importance of, the association between glycogen availability and these processes.

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29

McGinity, Christopher L., Erika M. Palmieri, Veena Somasundaram, Dibyangana D. Bhattacharyya, Lisa A. Ridnour, Robert Y. S. Cheng, Aideen E. Ryan, et al. "Nitric Oxide Modulates Metabolic Processes in the Tumor Immune Microenvironment." International Journal of Molecular Sciences 22, no. 13 (June 30, 2021): 7068. http://dx.doi.org/10.3390/ijms22137068.

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The metabolic requirements and functions of cancer and normal tissues are vastly different. Due to the rapid growth of cancer cells in the tumor microenvironment, distorted vasculature is commonly observed, which creates harsh environments that require rigorous and constantly evolving cellular adaption. A common hallmark of aggressive and therapeutically resistant tumors is hypoxia and hypoxia-induced stress markers. However, recent studies have identified alterations in a wide spectrum of metabolic pathways that dictate tumor behavior and response to therapy. Accordingly, it is becoming clear that metabolic processes are not uniform throughout the tumor microenvironment. Metabolic processes differ and are cell type specific where various factors promote metabolic heterogeneity within the tumor microenvironment. Furthermore, within the tumor, these metabolically distinct cell types can organize to form cellular neighborhoods that serve to establish a pro-tumor milieu in which distant and spatially distinct cellular neighborhoods can communicate via signaling metabolites from stroma, immune and tumor cells. In this review, we will discuss how biochemical interactions of various metabolic pathways influence cancer and immune microenvironments, as well as associated mechanisms that lead to good or poor clinical outcomes.

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30

Kravchenko, N. O., L. V. Kovalenko, O. P. Rudenko, and V. S. Boiko. "Status of metabolic processes in horses during spring period." Veterinary Medicine: inter-departmental subject scientific collection, no. 105 (August 7, 2019): 88–91. http://dx.doi.org/10.36016/vm-2019-105-17.

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The purpose of these studies was to determine status of metabolic processes in clinically healthy horses of sport breeds in spring period. Blood samples for biochemical studies were collected from 12 clinically healthy 7–9 month-old stallions of Ukrainian horse breed at Dnipropetrovsk region equestrian club. Protein (albumin, globulin, urea and creatinine) and mineral (common calcium and inorganic phosphorus) metabolic statuses, level of glucose, vitamins A and E and acid, as well as activity of hepatospecific enzymes (ALT, AST and AP) were determined using common techniques. It has been found that common protein level was within the limits of physiologic norm, although the level of albumins was decreased at the average rate of 12.0%, and the level of β- and γ-globulins was increased at the average rates of 5.2 and 11.3% respectively. AST activity was decreased at the rate of 38.0% regarding to physiological norm. Thereby, urea and creatinine concentrations were within the referent levels. Hyperglycaemia was observed in 50.0% of tested animals with maximal excess at the rate of 44.0%. Also, decreasing of common calcium and inorganic phosphorus levels was determined at the rate of 16.0 and 58.6%, vitamins А and Е — at the rate of 64.0 and 48,6% respectively, in comparison to lower level of physiological norm. The average index of acid capacity reached maximal referent levels. At the same time, it was increased in 33.3% of animals. Therefore, detected changes in biochemical indices in horse blood evidence that various metabolic disorders progress in clinically healthy stallions at spring and may furtherly lead to the appearance of metabolic syndrome

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31

Aretz, Jonas, Tobias Thüte, Sebastian Scholz, Klaudia Kersting, Thomas Noll, and Heino Büntemeyer. "Understanding cell behavior in cultivation processes - A metabolic approach." BMC Proceedings 7, Suppl 6 (2013): P90. http://dx.doi.org/10.1186/1753-6561-7-s6-p90.

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32

Link, H., and D. Weuster-Botz. "Metabolic control analysis of cells in fed-batch processes." New Biotechnology 25 (September 2009): S339. http://dx.doi.org/10.1016/j.nbt.2009.06.821.

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33

Frassetto, L. A., and A. Sebastian. "Aging, metabolic acidosis and renal failure: Interactive accelerating processes." Medical Hypotheses 124 (March 2019): 95–97. http://dx.doi.org/10.1016/j.mehy.2019.02.015.

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34

MÖLLERSTRÖM, J., and A. SOLLBERGER. "The 24-hour Rhythm of Metabolic Processes in Diabetes." Acta Medica Scandinavica 160, no. 1 (April 24, 2009): 25–46. http://dx.doi.org/10.1111/j.0954-6820.1958.tb10324.x.

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35

Gillis, C. N. "Pharmacological Aspects of Metabolic Processes in the Pulmonary Microcirculation." Annual Review of Pharmacology and Toxicology 26, no. 1 (April 1986): 183–200. http://dx.doi.org/10.1146/annurev.pa.26.040186.001151.

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36

Lindon, John C., Jeremy K. Nicholson, Elaine Holmes, and Jeremy R. Everett. "Metabonomics: Metabolic processes studied by NMR spectroscopy of biofluids." Concepts in Magnetic Resonance 12, no. 5 (2000): 289–320. http://dx.doi.org/10.1002/1099-0534(2000)12:5<289::aid-cmr3>3.0.co;2-w.

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37

Lukina, M. M., M. V. Shirmanova, T. F. Sergeeva, and E. V. Zagaynova. "Metabolic Imaging in the Study of Oncological Processes (Review)." Sovremennye tehnologii v medicine 8, no. 4 (December 2016): 113–26. http://dx.doi.org/10.17691/stm2016.8.4.16.

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38

Finoshin, Alexander D., Kim I. Adameyko, Kirill V. Mikhailov, Oksana I. Kravchuk, Anton A. Georgiev, Nicolay G. Gornostaev, Igor A. Kosevich, et al. "Iron metabolic pathways in the processes of sponge plasticity." PLOS ONE 15, no. 2 (February 21, 2020): e0228722. http://dx.doi.org/10.1371/journal.pone.0228722.

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39

Glazier, Douglas S. "Is metabolic rate a universal ‘pacemaker’ for biological processes?" Biological Reviews 90, no. 2 (May 23, 2014): 377–407. http://dx.doi.org/10.1111/brv.12115.

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40

Santi, Lucélia, Walter O. Beys-da-Silva, Markus Berger, Diego Calzolari, Jorge A. Guimarães, James J. Moresco, and John R. Yates. "Proteomic Profile ofCryptococcus neoformansBiofilm Reveals Changes in Metabolic Processes." Journal of Proteome Research 13, no. 3 (January 27, 2014): 1545–59. http://dx.doi.org/10.1021/pr401075f.

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41

Almeida, João R. M., Magnus Bertilsson, Marie F. Gorwa-Grauslund, Steven Gorsich, and Gunnar Lidén. "Metabolic effects of furaldehydes and impacts on biotechnological processes." Applied Microbiology and Biotechnology 82, no. 4 (March 2009): 625–38. http://dx.doi.org/10.1007/s00253-009-1875-1.

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42

Krokker, Lilla, Gábor Nyírő, Lilla Reiniger, Ottó Darvasi, Nikolette Szücs, Sándor Czirják, Miklós Tóth, Péter Igaz, Attila Patócs, and Henriett Butz. "Differentially Expressed miRNAs Influence Metabolic Processes in Pituitary Oncocytoma." Neurochemical Research 44, no. 10 (April 3, 2019): 2360–71. http://dx.doi.org/10.1007/s11064-019-02789-2.

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43

Shakespeare, P. G. "Disturbances in energy-yielding metabolic processes after burn injury." Burns 14, no. 4 (August 1988): 333. http://dx.doi.org/10.1016/0305-4179(88)90078-2.

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44

Cserjan-Puschmann, M., W. Kramer, E. Duerrschmid, G. Striedner, and K. Bayer. "Metabolic approaches for the optimisation of recombinant fermentation processes." Applied Microbiology and Biotechnology 53, no. 1 (December 12, 1999): 43–50. http://dx.doi.org/10.1007/s002530051612.

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45

van Bilsen, M., Ger J. van der Vusse, and Robert S. Reneman. "Transcriptional regulation of metabolic processes: implications for cardiac metabolism." Pfl�gers Archiv European Journal of Physiology 437, no. 1 (November 19, 1998): 2–14. http://dx.doi.org/10.1007/s004240050739.

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46

Lineva, O. I., F. N. Gilmiyarova, N. V. Spiridonova, and N. A. Krasnova. "Metabolic processes during physiologic pregnancy in unfavourable ecologic conditions." Kazan medical journal 79, no. 2 (March 25, 1998): 98–102. http://dx.doi.org/10.17816/kazmj63742.

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The peculiarities of metabolic and oxidation reduction processes as well as energoplastic metabolism during physiologic course of gestation in unfavourable ecologie conditions are studied. It is established that in ecologically unfavourable region during physiologic pregnancy general degydrogenase activity of blood plasma increases, cell membranes are packed, general protein level increases. The decrease of general degydrogenase activity of blood plasma with the increase of pregnancy length, rarefaction of membranes, the reduction of relation coefficient of malate dehydrogenase and lactate degydrogenase activity, the increase of combined quantity relationship of reduced and oxidated equivalents can be considered to be biomarkers of physiologic pregnancy transfer to pathologic.

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47

Tukhvatullina, L. M., O. V. Chechulina, and T. N. Mansurova. "Metabolic processes during physiologic pregnancy in unfavourable ecologic conditions." Kazan medical journal 79, no. 2 (March 25, 1998): 103–8. http://dx.doi.org/10.17816/kazmj63743.

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The peculiarities of metabolic and oxidation reduction processes as well as energoplastic metabolism during physiologic course of gestation in unfavourable cologie conditions are studied. It is established that in ecologically unfavourable region during physiologic pregnancy general degydrogenase activity of blood plasma increases, cell membranes are packed, general protein level increases. The decrease of general degydrogenase activity of blood plasma with the increase of pregnancy length, rarefaction of membranes, the reduction of relation coefficient of malate dehydrogenase and lactate degydrogenase activity, the increase of combined quantity relationship of reduced and oxidated equivalents can be considered to be biomarkers of physiologic pregnancy transfer to pathologic.

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48

Tashtemirova, Irodakhon Makhkambaevna. "State Of Purine Exchange And Microalbuminuria In Patients With Metabolic Syndrome." American Journal of Medical Sciences and Pharmaceutical Research 03, no. 01 (January 17, 2021): 46–54. http://dx.doi.org/10.37547/tajmspr/volume03issue01-08.

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The aim of the given work was study interactions of impairments sympa-thetic – adrenal systems functional condition and processes of peroxidal oxida-tion of lipids in woman with metabolic syndrome. 107women at the age of 25-49 were observation. They were randomized into 3 groups: I (control) – 15 healthy persons, II – 43 patients with arterial hypertension, III – 49 women with arterial hypertension in combination with metabolic syndrome. The results of carried investigations showed that activation of sympathetic adrenal system and processes of peroxidal oxidation of lipids took place in metabolic syn-drome. Marked lowering of sympathetic – adrenal system key ferment catechol-amins (MAO monoaminooxidaze) desamidization activity and considerable ac-tivation of peroxidal oxidation of lipid products which have great significance in revealing the mechanism of metabolic syndrome development were observed in metabolic syndrome. This results in the prolonged toxic influence of catechola-mins on myocardium.

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49

Mishra, Prashant, and David C. Chan. "Metabolic regulation of mitochondrial dynamics." Journal of Cell Biology 212, no. 4 (February 8, 2016): 379–87. http://dx.doi.org/10.1083/jcb.201511036.

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Mitochondria are renowned for their central bioenergetic role in eukaryotic cells, where they act as powerhouses to generate adenosine triphosphate from oxidation of nutrients. At the same time, these organelles are highly dynamic and undergo fusion, fission, transport, and degradation. Each of these dynamic processes is critical for maintaining a healthy mitochondrial population. Given the central metabolic function of mitochondria, it is not surprising that mitochondrial dynamics and bioenergetics reciprocally influence each other. We review the dynamic properties of mitochondria, with an emphasis on how these processes respond to cellular signaling events and how they affect metabolism.

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50

Kubo, F., Shinichiro Okazaki, and Isao Ujike. "Development of Microbial Metabolic Processes to Repair Concrete Joint Leakage." Advanced Materials Research 845 (December 2013): 158–62. http://dx.doi.org/10.4028/www.scientific.net/amr.845.158.

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This study proposes methods and examines the applicability of material generated from the metabolic processes of microorganisms to repair the leakage of concrete joints. Yeast was selected as the microorganism used for grout. The combination of yeast and a nutrition source with the largest amount of calcium carbonate extraction was found using the metabolic process of yeast. Calcium carbonate presence was determined by X-ray analysis. Water permeability of the concrete joint repaired with grout made from the largest yeast extraction was tested. From the results of the water permeability test, it is shown that grout made from the metabolic processes of yeast is capable of repairing a concrete joint.

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