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Books on the topic 'Metabolic processes'

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Published: 4 June 2021
Last updated: 1 February 2022

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1

NATO Advanced Research Workshop on Control of Metabolic Processes (1989 Lucca, Italy). Control of metabolic processes. New York: Plenum Press, 1990.

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2

Cornish-Bowden, Athel, and María Luz Cárdenas, eds. Control of Metabolic Processes. Boston, MA: Springer US, 1990. http://dx.doi.org/10.1007/978-1-4757-9856-2.

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3

Nutrient timing: Metabolic optimization for health, performance, and recovery. Boca Raton, FL: CRC Press, 2012.

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4

Colombo, Michael J. Nutrient enrichment, phytoplankton algal growth, and estimated rates of instream metabolic processes in the Quinebaug River basin, Connecticut, 2000-2001. Reston, Va: U.S. Dept. of the Interior, U.S. Geological Survey, 2004.

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5

L, Kon O., International Union of Biochemistry. Committee on Symposia., and Satellite Symposium on Molecular and Protein Engineering (1986 : Singapore), eds. Integration and control of metabolic processes: Pure and applied aspects : the proceedings of the Fourth Federation of Asian and Oceanian Biochemists Congress and Satellite Symposium on Molecular and Protein Engineering, held in Singapore during November 30-December 5, 1986. Cambridge [Cambridgeshire]: Cambridge University Press, 1987.

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6

Engin, Atilla, and Ayse Basak Engin, eds. Tryptophan Metabolism: Implications for Biological Processes, Health and Disease. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-15630-9.

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7

M, Altura Burton, Durlach Jean, and Seelig Mildred S. 1920-, eds. Magnesium in cellular processes and medicine. Basel: Karger, 1987.

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8

1935-, Mellinger Jean, Truchot J. P. 1937-, Lahlou B. 1936-, and Association des physiologistes (France), eds. Animal nutrition and transport processes. Basel: Karger, 1990.

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9

Bli͡umenfelʹd, L. A. Biophysical thermodynamics of intracellular processes: Molecular machines of the living cell. New York: Springer-Verlag, 1994.

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10

MAP kinase signaling protocols. 2nd ed. New York, N.Y: Humana Press, 2010.

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11

E, Adams Gerald, and North Atlantic Treaty Organization. Scientific Affairs Division., eds. Selective activation of drugs by redox processes. New York: Plenum Press, 1990.

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12

Dicke, Marcel. The ecology of plant secondary metabolites: From genes to global processes. New York: Cambridge University Press, 2012.

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13

Whittaker, Dean. The effect of phosphorus on-phosophorus metabolism, metals accumulation and floc properties in activated sludge processes. Ottawa: National Library of Canada, 2002.

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14

The obesity reality: A comprehensive approach to a growing problem. Lanham, Md: Rowman & Littlefield Publishers, 2012.

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15

Regulatory networks in stem cells. New York, NY: Humana Press, 2009.

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16

Claude, Bernard. Memoir on the pancreas and on the role of pancreatic juice in digestive processes: Particularly in the digestion of neutral fat. London: Academic Press, 1985.

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17

Memoir on the pancreas and on the role of pancreatic juice in digestive processes, particularly in the digestion of neutral fat. London: Academic, 1985.

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18

Cornish-Bowden, Athel, and Maria Luz Cárdenas. Control of Metabolic Processes. Springer, 2014.

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19

Cornish-Bowden, Athel, and Maria Luz Cárdenas. Control of Metabolic Processes. Springer, 2013.

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20

Kalia, Vipin Chandra, and Adesh Kumar Saini. Metabolic Engineering for Bioactive Compounds: Strategies and Processes. Springer, 2017.

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21

Kalia, Vipin Chandra, and Adesh Kumar Saini. Metabolic Engineering for Bioactive Compounds: Strategies and Processes. Springer, 2018.

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22

(Editor), Athel Cornish-Bowden, and Maria Luz Cárdenas (Editor), eds. Control of Metabolic Processes (Nato Science Series: A:). Springer, 1990.

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23

Kalia, Vipin Chandra, and Adesh Kumar Saini. Metabolic Engineering for Bioactive Compounds: Strategies and Processes. Springer, 2017.

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24

Lilleker, James B., and Mark E. Roberts. Metabolic myopathies. Edited by Hector Chinoy and Robert Cooper. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198754121.003.0005.

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Metabolic myopathies are caused by defects in the metabolic processes of energy storage and utilization, and can present with exercise intolerance, fatigue, muscle pain, and weakness. Metabolic myopathies are rare and can be difficult to diagnose. However, the clinical presentation can be similar to, and thus mimic, both the idiopathic inflammatory myopathies and other genetic muscle disorders including the muscular dystrophies. Careful enquiry about the nature and timing of muscle pain, as well as identification of other clinical ‘red-flags’, can highlight the possibility of a metabolic myopathy. The possibility of metabolic myopathy or muscular dystrophy mimicking myositis should be considered early in ‘treatment-resistant myositis’ or ‘seronegative myositis’. The diagnosis of metabolic myopathies depends on a multidisciplinary team, an awareness of the increasing availability of enzyme activity testing and the utility of expanding genetic technologies. In some cases, dietary manipulation and enzyme replacement therapies are useful treatments.
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25

(Editor), Max M. Häggblom, and Ingeborg D. Bossert (Editor), eds. Dehalogenation: Microbial Processes and Environmental Applications. Springer, 2003.

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26

1962-, Häggblom Max M., and Bossert Ingeborg D. 1954-, eds. Dehalogenation: Microbial processes and environmental applications. Boston: Kluwer Academic Pub., 2003.

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27

Haas, Paul. An Introduction To The Chemistry Of Plant Products - Vol II: Metabolic Processes. Obscure Press, 2007.

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28

Arleevskaya, Marina I., Rustam Aminov, Wesley H. Brooks, Gayane Manukyan, and Yves Renaudineau, eds. Shaping of Human Immune System and Metabolic Processes by Viruses and Microorganisms. Frontiers Media SA, 2019. http://dx.doi.org/10.3389/978-2-88945-941-4.

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29

Clinical Disorders of Membrane Transport Processes. Springer, 2012.

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30

1935-, Andreoli Thomas E., ed. Clinical disorders of membrane transport processes. New York: Plenum Medical Book Co., 1987.

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31

Kon, Oi Lian. Integration and Control of Metabolic Processes: Pure and Applied Aspects (Icsu Press Symposium Series, No 7). Cambridge University Press, 1987.

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32

Curran, Christine Perdan. Metabolic Processes And Energy Transfers: An Anthology Of Current Thought (Contemporary Discourse in the Field of Biology). Rosen Central, 2005.

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33

Gropler, Robert J. Imaging of Myocardial Metabolism. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199392094.003.0025.

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Abnormalities in myocardial substrate metabolism play a key role in the pathogenesis of a host of cardiac disease processes. The importance is highlighted by the routine clinical use of positron emission tomography (PET) using 18F-fluorodeoxyglucose (FDG) to measure myocardial glucose metabolism to detect viable tissue in patients with ischemic cardiomyopathy and the development of novel therapies designed to modulate myocardial metabolism. Our understanding of myocardial metabolism has benefitted from the application of advanced molecular biologic techniques and the development of elegant genetic models to of myocardial metabolic disease resulting in a greater appreciation for the pleiotropic actions of cellular metabolism. In parallel, there have been significant advances in radionuclide-based metabolic imaging techniques in terms instrumentation design, radiopharmaceutical development and small animal imaging. These advances have further ensconced radionuclide metabolic imaging techniques as tools to further our understanding of various forms of cardiovascular disease and potentially improve the care of the cardiac patient. In this chapter several of key advances in metabolic imaging will be described, their potential new clinical applications are reviewed and contribution to cardiovascular research highlighted.
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34

Clarke, Andrew. Metabolism. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780199551668.003.0008.

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Metabolism is driven by redox reactions, in which part of the difference in potential energy between the electron donor and acceptor is used by the organism for its life processes (with the remainder being dissipated as heat). The key process is intermediary metabolism, by which the energy stored in reserves (glycogen, starch, lipid, protein) is transferred to ATP. In aerobic respiration the electrons released from reserves are passed to oxygen, which is thereby reduced to water. Not all ATP regeneration involves oxygen as the final electron acceptor, and not all oxygen is used for ATP regeneration, but oxygen consumption is often the simplest and most practical way to measure the rate of intermediary metabolism and the errors in doing so are believed to be small. The costs of existence, as estimated by resting metabolism, represent only a part (~ 25%) of the daily energy expenditure of organisms. The costs of the organism’s ecology (growth, reproduction, movement and so on) are additional to existence costs. Resting metabolic rate increases with cell temperature, indicating that it costs more energy to maintain a warm cell than it does a cool or cold cell. The temperature sensitivity of resting metabolism is highly conserved across organisms.
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35

Veech, Richard L., and M. Todd King. Alzheimer’s Disease. Edited by Detlev Boison. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190497996.003.0026.

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Deficits in cerebral glucose utilization in Alzheimer’s disease (AD) arise decades before cognitive impairment and accumulation of amyloid plaques and neurofibrillary tangles in brain. Addressing this metabolic deficit has greater potential in treating AD than targeting later disease processes – an approach that has failed consistently in the clinic. Cerebral glucose utilization requires numerous enzymes, many of which have been shown to decline in AD. Perhaps the most important is pyruvate dehydrogenase (PDH), which links glycolysis with the Krebs cycle and aerobic metabolism, and whose activity is greatly suppressed in AD. The unique metabolism of ketone bodies allows them to bypass the block at pyruvate dehydrogenase and restore brain metabolism. Recent studies in mouse genetic models of AD and in a human Alzheimer’s patient showed the potential of ketones in maintaining brain energetics and function. Oral ketone bodies might be a promising avenue for treatment of Alzheimer’s disease.
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36

Gluckman, Sir Peter, Mark Hanson, Chong Yap Seng, and Anne Bardsley. Magnesium in pregnancy and breastfeeding. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780198722700.003.0021.

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Magnesium in important for a wide variety of physiological processes; prominent among them is the control of muscle contractions, cardiac function, and carbohydrate metabolism. In pregnancy, low magnesium status is associated with hypertension, pre-eclampsia, gestational diabetes, leg cramping, and preterm labour. Magnesium has been used extensively in obstetrics to reduce uterine contractility in threatened preterm labour. Although current evidence does not support a requirement for magnesium supplementation for most pregnant women, those who are overweight, have risk factors for hypertensive or metabolic disorders or malabsorption syndromes, or who are carrying multiple fetuses, should pay particular attention to their diets and should be monitored for signs of magnesium deficiency. Consuming a magnesium-rich diet throughout pregnancy is recommended.
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37

Rossor, Martin. Coma. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569381.003.0749.

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Many pathological processes can be responsible for stupor or coma, for example, head injury, tumour, vascular and inflammatory lesions, and most commonly toxic and metabolic states which usually lead to unconsciousness primarily through their effect upon the brainstem. In the series of Plum and Posner (1980) of 500 cases of stupor or coma, initially of unknown aetiology, 101 proved to be due to supratentorial lesions probably producing their effects by indirect action upon the brainstem, 65 to subtentorial lesions, and 326 to diffuse or metabolic brain.
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38

Narayana, Shalini, Babak Saboury, Andrew B. Newberg, Andrew C. Papanicolaou, and Abass Alavi. Positron Emission Tomography. Edited by Andrew C. Papanicolaou. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199764228.013.8.

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Positron emission tomography (PET) is an imaging method that utilizes compounds labeled with positron-emitting radioisotopes as molecular probes to evaluate different neurophysiological processes quantitatively and noninvasively. This chapter provides a background regarding positron emission, radiotracer chemistry, and detector and scanner instrumentation, as well as analytical methods for evaluating basic brain physiology, such as cerebral blood flow and oxygen and glucose metabolism. The methodological aspects of PET imaging, such as patient preparation and optimal scanning parameters, are discussed. Examples of application of blood flow and metabolic imaging in both research and clinical scenarios for the evaluation of normal neurophysiology are provided. Recent advances in PET imaging, including PET-CT and PET-MRI, are also described. Finally, the unique strengths of PET imaging are highlighted.
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39

Keshav, Satish, and Alexandra Kent. Unintentional weight loss. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0080.

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Body weight is determined by the combination of metabolic rate, calorie intake, and activity levels. Natural weight loss is usually due to declining muscle mass, with the redistribution of muscle mass in the extremities, leading to greater truncal fat stores. Unintentional weight loss refers to weight loss that is not voluntary, and can reflect serious underlying pathology. It can be caused by inadequate nutritional intake, increased metabolism, malabsorption, or a combination of these factors. Weight loss of 5% of body weight over 6–12 months should be investigated. Cachexia is a complex syndrome in which loss of body mass (fat and protein) cannot be reversed nutritionally, that is, is due to underlying disease processes inducing catabolism, rather than to inadequate nutritional intake.
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40

Gluckman, Sir Peter, Mark Hanson, Chong Yap Seng, and Anne Bardsley. The importance of nutrition and lifestyle to healthy development. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780198722700.003.0001.

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Good fetal and infant nutrition, whether derived from the mother via the placenta during gestation or via breast milk after birth, consists of the macronutrients protein, carbohydrates, and fats, all of which are needed for building the fundamental components of the body, and micronutrients such as vitamins and trace elements, which are essential structural components and cofactors in metabolic processes. Understanding the concept of a ‘balanced diet’ and the implications of maternal body composition is critical for pregnant and breastfeeding women to ensure that their metabolic adaptation to pregnancy and lactation is appropriate and that their offspring gets the required nutrients in the appropriate amount and proportion to ensure optimal development. An unbalanced diet, or over- or under-nutrition, can increase the risks of low birthweight and gestational diabetes and result in unfavourable metabolic adjustments by the fetus.
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41

D’Mello, Ajay. Mitochondrial Disease. Edited by Kirk Lalwani, Ira Todd Cohen, Ellen Y. Choi, and Vidya T. Raman. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190685157.003.0047.

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Mitochondrial disease is now considered to be an important cause for a diverse range of neurological, muscular, cardiac and endocrine disorders. Initially thought to be a rare group of disorders, it is now increasingly common for children with mitochondrial disease to present for a surgical procedure. While the mitochondrial respiratory chain is the essential finally common pathway for aerobic metabolism, mitochondria also play a role in a several important cellular processes. A variety of anesthetic techniques have been successfully used for this group of patients. However, the possibility of complications due to inhibition of mitochondrial function by anesthetic agents and surgical stress is a worry for the physician managing these patients. Anesthetic management focuses on disease symptoms at presentation, maintaining normoglycemia, while preventing further metabolic stress and complications that worsen lactic acidosis.
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42

Rho, Jong M. Overview. Edited by Jong M. Rho. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190497996.003.0011.

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After nearly a century of clinical use, the ketogenic diet is firmly established as an efficacious treatment for medically intractable epilepsy. Intriguingly, there is growing experimental evidence that the ketogenic diet and its metabolites also render neuroprotective and potentially disease-modifying effects. Hence, dietary and metabolic therapies have been attempted in a variety of neurological disorders other than epilepsy, including brain cancer, cognitive disorders, autism, neurotrauma, pain, and multiple sclerosis. This section, “Ketogenic Diet: Emerging Clinical Applications and Future Potential,” explores the current preclinical and clinical evidence for metabolism-based treatments designed to counter the myriad disease processes seen in many neurological conditions. Specific attention has been given to the effects of the ketogenic diet in malignant brain cancer, autism spectrum disorder, Alzheimer’s disease, traumatic brain and spinal cord injury, pain, and multiple sclerosis.
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43

Dupré, John, and Daniel J. Nicholson. A Manifesto for a Processual Philosophy of Biology. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198779636.003.0001.

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This chapter argues that scientific and philosophical progress in our understanding of the living world requires that we abandon a metaphysics of things in favour of one centred on processes. We identify three main empirical motivations for adopting a process ontology in biology: metabolic turnover, life cycles, and ecological interdependence. We show how taking a processual stance in the philosophy of biology enables us to ground existing critiques of essentialism, reductionism, and mechanicism, all of which have traditionally been associated with substance ontology. We illustrate the consequences of embracing an ontology of processes in biology by considering some of its implications for physiology, genetics, evolution, and medicine. And we attempt to locate the subsequent chapters of the book in relation to the position we defend.
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44

Dean, R. T. Cellular Degradative Processes. Springer, 2014.

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45

Systems Microbiology: Current Topics and Applications. Caister Academic Press, 2012.

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46

DiFrisco, James. Biological Processes. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198779636.003.0004.

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This chapter investigates the identity and persistence conditions for processes as a task of biological process ontology. It argues that the problem of intrinsic variation in evolution, development, and metabolism motivates viewing biological individuals as processes rather than as substances. Different criteria of identity for processes are then evaluated, including causal and spatio-temporal relations. The chapter ultimately settles on the view that processes are individuated by causal cohesion and are identical if they share the same cohesive properties and spatio-temporal region. The persistence of processes is interpreted on the model of perdurance, as a form of causal continuity or genidentity.
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47

Photosynthesis Ii Photosynthetic Carbon Metabolism And Related Processes. Springer, 2012.

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48

Maquet, Pierre, and Julien Fanielle. Neuroimaging in normal sleep and sleep disorders. Edited by Sudhansu Chokroverty, Luigi Ferini-Strambi, and Christopher Kennard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199682003.003.0011.

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Functional neuroimaging techniques include methods that probe various aspects of brain function and help derive models of brain organization in health and disease. These techniques can be grouped in two categories. Some are mainly based on electromagnetic signals (electroencephalography, magnetoencephalography), recording brain activity using a large number of sensors with exquisite temporal resolution (usually of the order of a kilohertz) but allowing only indirect characterization of three-dimensional brain activity by resorting to mathematical models. The second type includes different techniques (PET scan, SPECT, MRI, optical imaging) that typically assess metabolic or hemodynamic parameters, with millimeter spatial resolution and usually from the entire brain volume. However, temporal resolution is usually low because it is primarily driven by metabolic processes that unfold in several seconds or minutes. This chapter focuses on this second type, covering the contribution of brain imaging to understanding NREM sleep and REM sleep and also sleep disorders.
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49

O’Callaghan, Chris A. Renal function. Edited by Rutger Ploeg. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0126.

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The kidneys play a central role in homeostasis by maintaining extracellular fluid composition and volume. They do this by continuous filtration of plasma in the renal glomeruli and then subsequent modification of the filtered fluid as it passes along the nephron. The filtration process excludes large molecules, but most small molecules and ions are freely filtered. The filtrate that is produced in the glomeruli has a similar composition to plasma with respect to small molecules and ions. Most of the water and solutes are reabsorbed along the tubules and this process requires high levels of metabolic activity. In addition, a range of compounds and ions are secreted into the tubules along the nephron. Renal function is central to homeostasis and an appreciation of normal renal physiology is essential to understand the role of the kidney in a wide variety of disease processes.
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50

Engin, Ayse Basak, and Atilla Engin. Tryptophan Metabolism: Implications for Biological Processes, Health and Disease. Humana, 2015.

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