Books on the topic 'Metabolic pathways analysis'

To see the other types of publications on this topic, follow the link: Metabolic pathways analysis.

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the top 19 books for your research on the topic 'Metabolic pathways analysis.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Browse books on a wide variety of disciplines and organise your bibliography correctly.

1

service), ScienceDirect (Online, ed. RNA turnover in eukaryotes: Nucleases, pathways and analysis of mRNA decay. San Diego, Calif: Academic, 2008.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
2

Bacterial Cellular Metabolic Systems Metabolic Regulation Of A Cell System With 13cmetabolic Flux Analysis. Woodhead Publishing, 2012.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
3

Systems Biology: Constraint-Based Reconstruction and Analysis. Cambridge University Press, 2015.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
4

Palsson, Bernhard O. Systems Biology: Constraint-Based Reconstruction and Analysis. Cambridge University Press, 2015.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
5

Lamari, Foudil, and Jean-Marie Saudubray. Disorders of Complex Lipids Synthesis and Remodeling. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0066.

Full text
Abstract:
Defective lipid catabolic pathways are involved in numerous inherited metabolic diseases such as lysosomal storage diseases and peroxisome biogenesis disorders. We recently described a new classification of a rapidly growing group of inherited metabolic disorders involving biosynthesis and remodeling of complex lipids including phospholipids and sphingolipids. The remarkable progress achieved over the last decade in high throughput gene sequencing and in lipid analysis technologies have enabled the description of more than 40 diseases linked to defects in enzymes involved in these pathways. Some of these defects present in infancy or childhood but most of them are diagnosed in adolescence or adulthood. In this review we focus on those with adult presentation.
APA, Harvard, Vancouver, ISO, and other styles
6

Voit, Eberhard O., and Néstor V. Torres. Pathway Analysis and Optimization in Metabolic Engineering. Cambridge University Press, 2002.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
7

Voit, Eberhard O., and Néstor V. Torres. Pathway Analysis and Optimization in Metabolic Engineering. Cambridge University Press, 2004.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
8

Pathway Analysis and Optimization in Metabolic Engineering. Cambridge University Press, 2002.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
9

Voit, Eberhard O., and Néstor V. Torres. Pathway Analysis and Optimization in Metabolic Engineering. Cambridge University Press, 2002.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
10

Voit, Eberhard O., and Néstor V. Torres. Pathway Analysis and Optimization in Metabolic Engineering. Cambridge University Press, 2002.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
11

Voit, Eberhard O., and Néstor V. Torres. Pathway Analysis and Optimization in Metabolic Engineering. Cambridge University Press, 2011.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
12

Voit, Eberhard O., and Néstor V. Torres. Pathway Analysis and Optimization in Metabolic Engineering. Cambridge University Press, 2009.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
13

Challacombe, Jean F. Metabolic Pathway Engineering: Analysis and Applications in the Life Sciences. Jenny Stanford Publishing, 2021.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
14

Challacombe, Jean F. Metabolic Pathway Engineering: Analysis and Applications in the Life Sciences. Jenny Stanford Publishing, 2021.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
15

Metabolic Pathway Engineering: Analysis and Applications in the Life Sciences. Jenny Stanford Publishing, 2021.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
16

Challacombe, Jean F. Metabolic Pathway Engineering: Analysis and Applications in the Life Sciences. Jenny Stanford Publishing, 2021.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
17

Kiledjian, Megerditch, and Lynne E. Maquat. RNA Turnover in Eukaryotes: Analysis of Specialized and Quality Control RNA Decay Pathways. Elsevier Science & Technology Books, 2011.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
18

von Caemmerer, S. Biochemical Models of Leaf Photosynthesis. CSIRO Publishing, 2000. http://dx.doi.org/10.1071/9780643103405.

Full text
Abstract:
Increasing concerns of global climate change have stimulated research interests in all aspects of carbon exchange. This has restored interest in leaf photosynthetic models to predict and assess changes in photosynthetic CO2 assimilation in different environments. This is a comprehensive presentation of the most widely used models of steady-state photosynthesis by an author who is a world authority. Treatments of CO3, CO4 and intermediate pathways of photosynthesis in relation to environment have been update to include work on antisense transgenic plants. It will be a standard reference for the formal analysis of photosynthetic metabolism in vivo by advanced students and researchers.
APA, Harvard, Vancouver, ISO, and other styles
19

Goligorsky, Michael S., Julien Maizel, Radovan Vasko, May M. Rabadi, and Brian B. Ratliff. Pathophysiology of acute kidney injury. Edited by Norbert Lameire. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0221.

Full text
Abstract:
In the intricate maze of proposed mechanisms, modifiers, modulators, and sensitizers for acute kidney injury (AKI) and diverse causes inducing it, this chapter focuses on several common and undisputable strands which do exist.Structurally, the loss of the brush border, desquamation of tubular epithelial cells, and obstruction of the tubular lumen are commonly observed, albeit to various degrees. These morphologic hallmarks of AKI are accompanied by functional defects, most consistently reflected in the decreased glomerular filtration rate and variable degree of reduction in renal blood flow, accompanied by changes in the microcirculation. Although all renal resident cells participate in AKI, the brunt falls on the epithelial and endothelial cells, the fact that underlies the development of tubular epithelial and vascular compromise.This chapter further summarizes the involvement of several cell organelles in AKI: mitochondrial involvement in perturbed energy metabolism, lysosomal involvement in degradation of misfolded proteins and damaged organelles, and peroxisomal involvement in the regulation of oxidative stress and metabolism, all of which become defective. Common molecular pathways are engaged in cellular stress response and their roles in cell death or survival. The diverse families of nephrotoxic medications and the respective mechanisms they induce AKI are discussed. The mechanisms of action of some nephrotoxins are analysed, and also of the preventive therapies of ischaemic or pharmacologic pre-conditioning.An emerging concept of the systemic inflammatory response triggered by AKI, which can potentially aggravate the local injury or tend to facilitate the repair of the kidney, is presented. Rational therapeutic strategies should be based on these well-established pathophysiological hallmarks of AKI.
APA, Harvard, Vancouver, ISO, and other styles
We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!

To the bibliography