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Journal articles on the topic 'Metabolic disorders'

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Author: Grafiati

Published: 4 June 2021

Last updated: 11 March 2023

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1

Wikiera, Beata, Agnieszka Zubkiewicz-Kucharska, Julita Nocoń-Bohusz, and Anna Noczyńska. "Metabolic disorders in polycystic ovary syndrome." Pediatric Endocrinology Diabetes and Metabolism 23, no. 4 (2017): 204–8. http://dx.doi.org/10.18544/pedm-23.04.0094.

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2

Dora, Esther. "Inherited Metabolic Disorders: A Prospective Study." Endocrinology and Disorders 2, no. 2 (February 15, 2018): 01. http://dx.doi.org/10.31579/2640-1045/020.

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3

Ding, Hui, Mengyuan Ouyang, Jinyi Wang, Minyao Xie, Yanyuan Huang, Fangzheng Yuan, Yunhan Jia, Jun Wang, Na Liu, and Ning Zhang. "Obsessive-Compulsive Disorder and Metabolic Disorders." Journal of Nervous & Mental Disease 210, no. 12 (December 2022): 951–59. http://dx.doi.org/10.1097/nmd.0000000000001594.

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4

Crushell, Ellen, and Mary King. "Metabolic disorders." Journal of Pediatric Neurology 08, no. 01 (July 30, 2015): 111–12. http://dx.doi.org/10.3233/jpn-2010-0370.

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5

&NA;. "METABOLIC DISORDERS." Journal of Pediatric Orthopaedics 5, no. 1 (January 1985): 122. http://dx.doi.org/10.1097/01241398-198501000-00036.

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6

&NA;. "METABOLIC DISORDERS." Journal of Pediatric Orthopaedics 6, no. 3 (May 1986): 386. http://dx.doi.org/10.1097/01241398-198605000-00034.

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7

&NA;. "METABOLIC DISORDERS." Journal of Pediatric Orthopaedics 6, no. 4 (July 1986): 514–15. http://dx.doi.org/10.1097/01241398-198607000-00036.

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8

&NA;. "METABOLIC DISORDERS." Journal of Pediatric Orthopaedics 7, no. 3 (May 1987): 374. http://dx.doi.org/10.1097/01241398-198705000-00037.

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9

Halliwell, Barry. "METABOLIC DISORDERS." Lancet 341, no. 8837 (January 1993): 92. http://dx.doi.org/10.1016/0140-6736(93)92566-c.

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10

Yu, Zhiping, and Valerie Muehleman. "Eating Disorders and Metabolic Diseases." International Journal of Environmental Research and Public Health 20, no. 3 (January 30, 2023): 2446. http://dx.doi.org/10.3390/ijerph20032446.

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Eating disorders are complex diseases with multifactorial causes. According to the Diagnostic and Statistical Manual of Mental Disorders text version (DSM-5-TR) and the WHO International Classification of Diseases and Related Health Problems (ICD-11), the major types of eating disorders include anorexia nervosa, bulimia nervosa, and binge eating disorder. The prevalence of eating disorders is alarmingly increasing globally. Moreover, the COVID-19 pandemic has led to more development and worsening of eating disorders. Patients with eating disorders exhibit high rates of psychiatric comorbidities and medical comorbidities such as obesity, diabetes, and metabolic syndrome. This paper aims to review and discuss the comorbidities of eating disorders with those metabolic diseases. Eating disorder treatment typically includes a combination of some or all approaches such as psychotherapy, nutrition education, and medications. Early detection and intervention are important for the treatment of eating disorders.

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11

Strashok, L. A., O. V. Buznytska, and О. М. Meshkova. "Indicators of lipid metabolism disorders in the blood serum of adolescents with metabolic syndrome." Ukrainian Biochemical Journal 92, no. 6 (December 24, 2020): 137–42. http://dx.doi.org/10.15407/ubj92.06.137.

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12

Davison, James E. "Eye involvement in inherited metabolic disorders." Therapeutic Advances in Ophthalmology 12 (January 2020): 251584142097910. http://dx.doi.org/10.1177/2515841420979109.

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Inherited metabolic disorders are a large group of rare disorders affecting normal biochemical pathways. Many metabolic disorders can present with symptoms affecting the eye, and eye disorders can evolve later in the natural history of an already diagnosed metabolic disorder. The ophthalmic involvement can be very varied affecting any part of the eye, including abnormalities of cornea, lens dislocation and cataracts, retina and the distal optic pathway, and extraocular muscles. Awareness of inherited metabolic disorders is important to facilitate early diagnosis and in some cases instigate early treatment if a patient presents with eye involvement suggestive of a metabolic disorder. Ophthalmological interventions are also an important component of the multisystem holistic approach to treating patients with metabolic disorders.

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13

Chambers, Jeanette K. "Metabolic Bone Disorders." Nursing Clinics of North America 22, no. 4 (December 1987): 861–72. http://dx.doi.org/10.1016/s0029-6465(22)01341-x.

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14

Hopkins, Maeve K., Lorraine Dugoff, and Jeffrey A. Kuller. "Inherited Metabolic Disorders." Obstetrical & Gynecological Survey 73, no. 6 (June 2018): 361–67. http://dx.doi.org/10.1097/ogx.0000000000000566.

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15

Boyer, Suzanne W., Lisa J. Barclay, and Lindsay C. Burrage. "Inherited Metabolic Disorders." Nutrition in Clinical Practice 30, no. 4 (June 16, 2015): 502–10. http://dx.doi.org/10.1177/0884533615586201.

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16

Di, Sha, Yitian Wang, Lin Han, Qi Bao, Zezheng Gao, Qing Wang, Yingying Yang, Linhua Zhao, and Xiaolin Tong. "The Intervention Effect of Traditional Chinese Medicine on the Intestinal Flora and Its Metabolites in Glycolipid Metabolic Disorders." Evidence-Based Complementary and Alternative Medicine 2019 (June 4, 2019): 1–13. http://dx.doi.org/10.1155/2019/2958920.

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Metabolic syndrome (MS), which includes metabolic disorders such as protein disorder, glucose disorder, lipid disorder, and carbohydrate disorder, has been growing rapidly around the world. Glycolipid disorders are a main type of metabolic syndrome and are characterized by abdominal obesity and abnormal metabolic disorders of lipid, glucose, and carbohydrate utilization, which can cause cardiovascular and cerebrovascular diseases. Glycolipid disorders are closely related to intestinal flora and its metabolites. However, studies about the biological mechanisms of the intestinal flora and its metabolites with glycolipid disorders have not been clear. When glycolipid disorders are treated with drugs, a challenging problem is side effects. Traditional Chinese medicine (TCM) and dietary supplements have fewer side effects to treat it. Numerous basic and clinical studies have confirmed that TCM decoctions, Chinese medicine monomers, or compounds can treat glycolipid disorders and reduce the incidence of cardiovascular disease. In this study, we reviewed the relationship between the intestinal flora and its metabolites in glycolipid metabolic disorders and the effect of TCM in treating glycolipid metabolic disorders through the intestinal flora and its metabolites. This review provides new perspectives and strategies for future glycolipid disorders research and treatment.

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17

Silver, Grace, and Saadet Mercimek-Andrews. "Inherited Metabolic Disorders Presenting with Ataxia." International Journal of Molecular Sciences 21, no. 15 (August 1, 2020): 5519. http://dx.doi.org/10.3390/ijms21155519.

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Ataxia is a common clinical feature in inherited metabolic disorders. There are more than 150 inherited metabolic disorders in patients presenting with ataxia in addition to global developmental delay, encephalopathy episodes, a history of developmental regression, coarse facial features, seizures, and other types of movement disorders. Seizures and a history of developmental regression especially are important clinical denominators to consider an underlying inherited metabolic disorder in a patient with ataxia. Some of the inherited metabolic disorders have disease specific treatments to improve outcomes or prevent early death. Early diagnosis and treatment affect positive neurodevelopmental outcomes, so it is important to think of inherited metabolic disorders in the differential diagnosis of ataxia.

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18

LeMieux, M., A. Al-Jawadi, S. Wang, and N. Moustaid-Moussa. "Metabolic Profiling in Nutrition and Metabolic Disorders." Advances in Nutrition: An International Review Journal 4, no. 5 (September 1, 2013): 548–50. http://dx.doi.org/10.3945/an.113.004267.

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19

Watkins, Susan. "Renal and Metabolic DisordersRenal and Metabolic Disorders." Nursing Standard 27, no. 37 (May 15, 2013): 30. http://dx.doi.org/10.7748/ns2013.05.27.37.30.s39.

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20

Valcheva, S., and Sp Todorov. "Dizziness systemic metabolic disorders." International Bulletin of Otorhinolaryngology 11, no. 4 (December 28, 2015): 5. http://dx.doi.org/10.14748/orl.v11i4.6853.

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21

Sharma, Santosh K., Gaurav Bhushan, and Sweta Chhangani. "Genetically inborn metabolic disorders." Indian Journal of Pharmaceutical and Biological Research 6, no. 01 (March 31, 2018): 48–57. http://dx.doi.org/10.30750/ijpbr.6.1.8.

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Metabolism is the process carried out in the cells of all living organisms converting the food we eat to chemical energy needed for sustaining life. It encompasses allbiochemical processes that occur within any living organism - including humans - to maintain life. These biochemical processes allow us to grow, reproduce, repair damage, and respond to our environment.

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22

Jeong, Hye Gyeong, and Hyuntae Park. "Metabolic Disorders in Menopause." Metabolites 12, no. 10 (October 8, 2022): 954. http://dx.doi.org/10.3390/metabo12100954.

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Menopause is an aging process and an important time equivalent to one-third of a woman’s lifetime. Menopause significantly increases the risk of cardiometabolic diseases, such as obesity, type 2 diabetes, cardiovascular diseases, non-alcoholic liver disease (NAFLD)/metabolic associated fatty liver disease (MFFLD), and metabolic syndrome (MetS). Women experience a variety of symptoms in the perimenopausal period, and these symptoms are distressing for most women. Many factors worsen a woman’s menopausal experience, and controlling these factors may be a strategy to improve postmenopausal women’s health. This review aimed to confirm the association between menopause and metabolic diseases (especially MetS), including pathophysiology, definition, prevalence, diagnosis, management, and prevention.

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23

Stachowiak, Grzegorz, Tomasz Pertyński, and Magdalena Pertyńska-Marczewska. "Metabolic disorders in menopause." Menopausal Review 1 (2015): 59–64. http://dx.doi.org/10.5114/pm.2015.50000.

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24

Ukkola, Olavi. "Ghrelin and Metabolic Disorders." Current Protein & Peptide Science 10, no. 1 (February 1, 2009): 2–7. http://dx.doi.org/10.2174/138920309787315220.

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25

ÖZSOYLU, SINASI, and AYTEKIN BESIM. "Rickets and Metabolic Disorders." Pediatrics 85, no. 4 (April 1, 1990): 624–25. http://dx.doi.org/10.1542/peds.85.4.624a.

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To the Editor.— The short report of Morrison and Kerr1 entitled "Growth Plate: Roentgenologic Findings in a Case of Tyrosinosis" deserves some comments. In addition to phenylketonuria and homocystinuria which were mentioned by the authors, roentgenologic bony changes were shown recently in a 5-month-old boy with mucolipidosis II (I-cell disease).2 Alkaline phosphatase was found to be elevated, and calcium and phosphorus levels were somewhat decreased in most of these cases. With the addition of roentgenologic findings one can be sure that all of these patients had rickets.

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26

MORRISON, STUART C., and DOUGLAS S. KERR. "Rickets and Metabolic Disorders." Pediatrics 85, no. 4 (April 1, 1990): 625. http://dx.doi.org/10.1542/peds.85.4.625.

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In Reply.— We have reviewed the letter from Drs Ozsoylu and Besim and have reviewed the metabolic data from our patient with tyrosinosis to determine if there was evidence of rickets in our patient. At the time that the published x-rays were obtained, the patient had normal serum calcium and phosphorus. However, a month earlier, when the x-rays appeared normal apart from probable osteopenia, the patient did have persistent hypophosphatemia, 1.5 to 1.8 mg/dL (normal 4.0 to 6.0 mg/dL), and elevated alkaline phosphatase, 956 IU/L (normal 50 to 380 IU/L).

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27

Hoffman, Robert S., and Lewis R. Goldfrank. "Ethanol-Associated Metabolic Disorders." Emergency Medicine Clinics of North America 7, no. 4 (November 1989): 943–61. http://dx.doi.org/10.1016/s0733-8627(20)30326-6.

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28

Navab, Mohamad, Nima Gharavi, and Andrew D. Watson. "Inflammation and metabolic disorders." Current Opinion in Clinical Nutrition and Metabolic Care 11, no. 4 (July 2008): 459–64. http://dx.doi.org/10.1097/mco.0b013e32830460c2.

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29

REECE, E. ALBERT. "METABOLIC DISORDERS IN PREGNANCY." Clinical Obstetrics and Gynecology 37, no. 1 (March 1994): 1–2. http://dx.doi.org/10.1097/00003081-199403000-00004.

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30

&NA;. "Metabolic Disorders in Pregnancy." Clinical Obstetrics and Gynecology 37, no. 1 (March 1994): 110–11. http://dx.doi.org/10.1097/00003081-199403000-00014.

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31

Muriithi, Angela. "Renal and Metabolic Disorders." Critical Care Medicine 42, no. 2 (February 2014): e175. http://dx.doi.org/10.1097/ccm.0000000000000152.

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32

Corley, K. T. T. "Metabolic disorders in foals." Equine Veterinary Education 24, no. 8 (February 20, 2012): 392–95. http://dx.doi.org/10.1111/j.2042-3292.2011.00376.x.

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33

Feldman, Robert G. "Metabolic disorders and neurotoxicology." Current Opinion in Neurology 12, no. 6 (December 1999): 723. http://dx.doi.org/10.1097/00019052-199912000-00011.

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34

Hotamisligil, Gökhan S. "Inflammation and metabolic disorders." Nature 444, no. 7121 (December 2006): 860–67. http://dx.doi.org/10.1038/nature05485.

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35

Cakir, Banu, Mehmet Teksam, Dilek Kosehan, Kayihan Akin, and Asli Koktener. "Neuroimaging in Metabolic Disorders." Journal of Neuroimaging 21, no. 3 (March 8, 2011): 209. http://dx.doi.org/10.1111/j.1552-6569.2011.00578.x.

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36

Markand, O. "EEG in metabolic disorders." Electroencephalography and Clinical Neurophysiology 103, no. 1 (July 1997): 33. http://dx.doi.org/10.1016/s0013-4694(97)88052-6.

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37

Rubash, J. Mark. "Metabolic Acid–Base Disorders." Veterinary Clinics of North America: Small Animal Practice 31, no. 6 (November 2001): 1323–54. http://dx.doi.org/10.1016/s0195-5616(01)50105-9.

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38

Yeoh, Cindy, Howard Teng, Jacob Jackson, Lee Hingula, Takeshi Irie, Aron Legler, Corrine Levine, Iris Chu, Casey Chai, and Luis Tollinche. "Metabolic Disorders and Anesthesia." Current Anesthesiology Reports 9, no. 3 (July 12, 2019): 340–59. http://dx.doi.org/10.1007/s40140-019-00345-w.

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39

Brown, G. K. "Metabolic disorders of embryogenesis." Journal of Inherited Metabolic Disease 17, no. 4 (1994): 448–58. http://dx.doi.org/10.1007/bf00711360.

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40

Morris, Andrew A. M., and Douglass M. Turnbull. "Metabolic disorders in children." Current Opinion in Neurology 7, no. 6 (December 1994): 535–41. http://dx.doi.org/10.1097/00019052-199412000-00011.

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41

Beal, M. Flint. "Metabolic disorders and neurotoxicology." Current Opinion in Neurology 8, no. 6 (December 1995): 467–68. http://dx.doi.org/10.1097/00019052-199512000-00013.

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42

&NA;. "Metabolic disorders and neurotoxicology." Current Opinion in Neurology 9, no. 6 (December 1996): B149. http://dx.doi.org/10.1097/00019052-199612000-00021.

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43

Arroyo, May, and James M. Crawford. "Hepatitic inherited metabolic disorders." Seminars in Diagnostic Pathology 23, no. 3-4 (August 2006): 182–89. http://dx.doi.org/10.1053/j.semdp.2006.11.005.

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44

Corbett, John, and Clayton Mathews. "Metabolic disorders and inflammation." Drug Discovery Today: Disease Mechanisms 3, no. 3 (September 2006): 365–66. http://dx.doi.org/10.1016/j.ddmec.2006.09.011.

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45

Reid, G. "Metabolic disorders of cattle." Medical Hypotheses 40, no. 5 (May 1993): 296–300. http://dx.doi.org/10.1016/0306-9877(93)90009-f.

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46

Pignatelli, Pasquale, Roberto Carnevale, and Danilo Menichelli. "Silybin and metabolic disorders." Internal and Emergency Medicine 14, no. 1 (October 17, 2018): 1–3. http://dx.doi.org/10.1007/s11739-018-1968-x.

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47

Owayes Muaffaq Hamed, Amjad Abdul-hadi Mohammed, and Raed Salem Alsaffar. "Genetic Metabolism Disorders in Newborn." International Journal for Research in Applied Sciences and Biotechnology 8, no. 1 (January 13, 2021): 77–81. http://dx.doi.org/10.31033/ijrasb.8.1.9.

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Babies with any type of metabolic disorders lack the ability to break down the food well, which may induce too little amino acids, phenylalanine and blood sugar to the body, there are numerous kinds of this disorders, most of babies with a genetic metabolic disease have many mutation in gene that coded an enzyme which results a deficiency in same enzyme are hundreds of these disorders and they were diagnosed by their symptoms and the treatment method. The treatment methods of the metabolic disorder depend on the specific type of disorders, inborn metabolic disease are some-time treated with dietary guidance, and other childcare choices, many hereditary metabolic disease are initially caused by gene mutations and that transferred from parents to offspring.

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48

Niwano, Yoshimi, Hidetsugu Kohzaki, Midori Shirato, Shunichi Shishido, and Keisuke Nakamura. "Putative Mechanisms Underlying the Beneficial Effects of Polyphenols in Murine Models of Metabolic Disorders in Relation to Gut Microbiota." Current Issues in Molecular Biology 44, no. 3 (March 18, 2022): 1353–75. http://dx.doi.org/10.3390/cimb44030091.

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The beneficial effects of polyphenols on metabolic disorders have been extensively reported. The interaction of these compounds with the gut microbiota has been the focus of recent studies. In this review, we explored the fundamental mechanisms underlying the beneficial effects of polyphenols in relation to the gut microbiota in murine models of metabolic disorders. We analyzed the effects of polyphenols on three murine models of metabolic disorders, namely, models of a high-fat diet (HFD)-induced metabolic disorder, dextran sulfate sodium (DSS)-induced colitis, and a metabolic disorder not associated with HFD or DSS. Regardless of the model, polyphenols ameliorated the effects of metabolic disorders by alleviating intestinal oxidative stress, improving inflammatory status, and improving intestinal barrier function, as well as by modulating gut microbiota, for example, by increasing the abundance of short-chain fatty acid-producing bacteria. Consequently, polyphenols reduce circulating lipopolysaccharide levels, thereby improving inflammatory status and alleviating oxidative imbalance at the lesion sites. In conclusion, polyphenols likely act by regulating intestinal functions, including the gut microbiota, and may be a safe and suitable therapeutic agent for various metabolic disorders.

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49

Mishra, Manish, Mignonette Relatado-Sotto, Raju Panta, Miguel Miyares, and Ranjan Solanki Trinity. "Association of Diabetes Mellitus and Thyroid Disorders: A Metabolic Prospective." Asian Pacific Journal of Health Sciences 4, no. 3 (September 30, 2017): 253–62. http://dx.doi.org/10.21276/apjhs.2017.4.3.39.

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50

Rajendran, Babu, Seetha Rami Reddy Mallampati, and Sheju Jonathan Jha J. "Acid based disorders in intensive care unit: a hospital-based study." International Journal of Advances in Medicine 6, no. 1 (January 23, 2019): 62. http://dx.doi.org/10.18203/2349-3933.ijam20190086.

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Background: Acid base disorders are common in the ICU patients and pose a great burden in the management of the underlying condition.Methods: Identifying the type of acid-base disorders in ICU patients using arterial blood gas analysis This was a retrospective case-controlled comparative study. 46 patients in intensive care unit of a reputed institution and comparing the type of acid-base disorder amongst infectious (10) and non-infectious (36) diseases.Results: Of the study population, 70% had mixed acid base disorders and 30% had simple type of acid base disorders. It was found that sepsis is associated with mixed type of acid-base disorders with most common being metabolic acidosis with respiratory alkalosis. Non-infectious diseases were mostly associated with metabolic alkalosis with respiratory acidosis. Analysis of individual acid base disorders revealed metabolic acidosis as the most common disturbance.Conclusions: These results projected the probability of acid bases disorders in various conditions and help in the efficient management. Mixed acid base disorders are the most common disturbances in the intensive care setup which is metabolic acidosis with respiratory alkalosis in infectious diseases and metabolic acidosis is the most common simple type of acid base disorder.

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