Relevant bibliographies by topics / Metabolic depression

Academic literature on the topic 'Metabolic depression'

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Published: 4 June 2021
Last updated: 5 February 2022

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Journal articles on the topic "Metabolic depression"

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Vogelzangs, Nicole, Aartjan T. F. Beekman, Ingrid G. Boelhouwer, Stefania Bandinelli, Yuri Milaneschi, Luigi Ferrucci, and Brenda W. J. H. Penninx. "Metabolic Depression." Journal of Clinical Psychiatry 72, no. 05 (April 5, 2011): 598–604. http://dx.doi.org/10.4088/jcp.10m06559.

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Lamers, F., Y. Milaneschi, P. de Jonge, E. J. Giltay, and B. W. J. H. Penninx. "Metabolic and inflammatory markers: associations with individual depressive symptoms." Psychological Medicine 48, no. 7 (September 11, 2017): 1102–10. http://dx.doi.org/10.1017/s0033291717002483.

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AbstractBackgroundLiterature has shown that obesity, metabolic syndrome and inflammation are associated with depression, however, evidence suggests that these associations are specific to atypical depression. Which of the atypical symptoms are driving associations with obesity-related outcomes and inflammation is unknown. We evaluated associations between individual symptoms of depression (both atypical and non-atypical) and body mass index (BMI), metabolic syndrome components and inflammatory markers.MethodsWe included 808 persons with a current diagnosis of depression participating in the Netherlands Study of Depression and Anxiety (67% female, mean age 41.6 years). Depressive symptoms were derived from the Composite International Diagnostic Interview and the Inventory of Depressive Symptomatology. Univariable and multivariable regression analyses adjusting for sex, age, educational level, depression severity, current smoking, physical activity, anti-inflammatory medication use, and statin use were performed.ResultsIncreased appetite was positively associated with BMI, number of metabolic syndrome components, waist circumference, C-reactive protein and tumor necrosis factor-α. Decreased appetite was negatively associated with BMI and waist circumference. Psychomotor retardation was positively associated with BMI, high-density lipoprotein cholesterol and triglycerides, and insomnia with number of metabolic syndrome components.ConclusionIncreased appetite – in the context of a depressive episode – was the only symptom that was associated with both metabolic as well as inflammatory markers, and could be a key feature of an immuno-metabolic form of depression. This immuno-metabolic depression should be considered in clinical trials evaluating effectiveness of compounds targeting metabolic and inflammatory pathways or lifestyle interventions.
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Fauziyati, Ana, Agus Siswanto, Luthfan Budi Purnomo, and Hemi Sinorita. "Type of psychosocial stressor as risk factor of depressive symptom in metabolic syndrome." Bangladesh Journal of Medical Science 15, no. 2 (August 10, 2016): 262–68. http://dx.doi.org/10.3329/bjms.v15i2.28867.

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Metabolic syndrome and depression are two major diseases over the world, which are increasing in prevalence over time. Depression is a major mental health burden over the world. In long time, depression can lead to metabolic syndrome, while metabolic syndrome is a risk factor for developing depression. Metabolic syndrome is a major risk factor for developing cardiovascular disease. Chronic stress induced by psychosocial stressor leads to the development of both metabolic syndrome and depression. Further research is important to identify which type of psychosocial stressor is the risk factor for depressive symptom in patients with metabolic syndrome. The objective of this study is to identify the type of psychosocial stressor which could be the risk factor for depressive symptom. The study design was case control. The case group consisted of metabolic syndrome patients with depressive symptom, while the control group consisted of metabolic syndrome patients without depressive symptom. Metabolic syndrome was diagnosed based on International Diabetes Federation (IDF) criteria. Depressive symptom was measured by Beck Depression Inventory (BDI). Psychosocial stressors were measured by Stressful Life Events (SLE) questionnaire. Dependent variable was depressive symptom, while independent variables were type of psychosocial stressors (finance, work, social relationship, health and housing). Analysis methods that used in this study were independent t test, Pearson/Spearman correlation analysis, chi square and logistic regression. There were 54 patients in this study, consisted of 24 in case group and 30 in control group. There was no significant difference in most basic characteristics between two groups. There was significant difference of SLE score between two groups. Chi square analysis showed that housing, finance, health, social relationship, and work stressors were risk factors for developing depressive symptom in metabolic syndrome (OR 24.5 (p 0.001); 9.7 (p 0.039); 8.4 (p 0.016); 5.4 (p 0.004); 3.9 (p 0.001), respectively). Demographic factor which also influenced depressive symptom was salary less than 1 million per month (OR 45, p 0.004). According to logistic regression analysis, psychosocial stressors which most influenced the depressive symptom were finance and housing. In conclusion, this study showed that housing, finance, health, social relationship and work stressors were risk factors for developing depressive symptom in metabolic syndrome.Bangladesh Journal of Medical Science Vol.15(2) 2016 p.262-268
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Heiskanen, Tuula H., Leo K. Niskanen, Jukka J. Hintikka, Heli T. Koivumaa-Honkanen, Kirsi M. Honkalampi, Kaisa M. Haatainen, and Heimo T. Viinamäki. "Metabolic Syndrome and Depression." Journal of Clinical Psychiatry 67, no. 09 (September 15, 2006): 1422–27. http://dx.doi.org/10.4088/jcp.v67n0913.

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Heuser, I. "Metabolic syndrome and depression." Journal of Affective Disorders 107 (March 2008): S24—S25. http://dx.doi.org/10.1016/j.jad.2007.12.158.

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Gary, Tiffany L., Kesha Baptiste-Roberts, Rosa M. Crum, Lisa A. Cooper, Daniel E. Ford, and Frederick L. Brancati. "Changes in Depressive Symptoms and Metabolic Control over 3 Years among African Americans with Type 2 Diabetes." International Journal of Psychiatry in Medicine 35, no. 4 (December 2005): 377–82. http://dx.doi.org/10.2190/bq22-4hu0-p6ek-l7yx.

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Objective: It is established that individuals with diabetes have high rates of depression, but the longitudinal relationship between depression and glycemic control has not been well examined, particularly among African Americans. The objective of this study was to evaluate the longitudinal relationship between depressive symptoms and metabolic control. Method: We conducted an earlier cross-sectional study that demonstrated marginal and significant associations between depressive symptoms (using the Center for Epidemiologic Studies Depression Scale [CES-D]) and metabolic control (HbA1c, lipids, blood pressure) among 183 African Americans with type 2 diabetes. In this report, we present data on these individuals, followed for three years, and examine the relationship between change in depressive symptoms and change in metabolic control over that time period. Results: Results showed that that there were no statistically significant associations between baseline or change in depressive symptoms and metabolic control over three years. Limited statistical power may explain this negative finding. Conclusions: This study provides insight into the relationship between depression and metabolic control. Prospective observational studies are needed to further evaluate this relationship.
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Flores-Ramos, Mónica, Martín Armando Burrola-Suárez, Rodrigo Guiza-Zayas, J. Miguel Enciso-Araujo, Dannia Islas-Preciado, and Erika Estrada Camarena. "Evaluation of hormonal and metabolic factors related to depression in reproductive age women." Salud mental 43, no. 1 (January 30, 2020): 35–41. http://dx.doi.org/10.17711/sm.0185-3325.2020.006.

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Introduction. Major depressive disorder (MDD) is a prevalent disease affecting women more than men worldwide. Various factors are involved in the genesis of depression, including hormones such as testosterone and certain metabolic factors Objective. To evaluate hormone levels and metabolic variables in women with major depression and healthy controls. Method. A cross-sectional, comparative analytical study was conducted in 40 participants, 23 patients with an MDD diagnosis and 17 controls, all of women in reproductive age between the ages of 18 and 45. Sociodemographic variables, hormonal profile, and metabolic variables were assessed and the 17-item Hamilton Depression Scale was used to evaluate depressive symptoms. Results. No statistically significant differences were observed between the groups in the hormonal and metabolic variables explored. Nevertheless, it was observed that the lower the testosterone levels and the higher the serum glucose levels, the more intense depressive symptoms were. Discussion and conclusion. Testosterone is associated with a lower depressive symptoms score on the Hamilton Depression scale, suggesting a potential antidepressant effect, whereas high glucose levels are associated with a higher score on this scale. We believe that the measurement of hormonal and metabolic variables in women can contribute to a better understanding of the pathophysiology of depression.
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Marijnissen, R. M., N. Vogelzangs, M. E. Mulder, R. H. S. van den Brink, H. C. Comijs, and R. C. Oude Voshaar. "Metabolic dysregulation and late-life depression: a prospective study." Psychological Medicine 47, no. 6 (December 12, 2016): 1041–52. http://dx.doi.org/10.1017/s0033291716003196.

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BackgroundDepression is associated with the metabolic syndrome (MS). We examined whether metabolic dysregulation predicted the 2-year course of clinical depression.MethodA total of 285 older persons (⩾60 years) suffering from depressive disorder according to DSM-IV-TR criteria was followed up for 2 years. Severity of depression was assessed with the Inventory of Depressive Symptomatology (IDS) at 6-month intervals. Metabolic syndrome was defined according the National Cholesterol Education Programme (NCEP-ATP III). We applied logistic regression and linear mixed models adjusted for age, sex, years of education, smoking, alcohol use, physical activity, somatic co-morbidity, cognitive functioning and drug use (antidepressants, anti-inflammatory drugs) and severity of depression at baseline.ResultsMS predicted non-remission at 2 years (odds ratioper component = 1.26, 95% confidence interval 1.00–1.58), p = 0.047), which was driven by the waist circumference and HDL cholesterol. MS was not associated with IDS sum score. Subsequent analyses on its subscales, however, identified an association with the somatic symptom subscale score over time (interaction time × somatic subscale, p = 0.005), driven by higher waist circumference and elevated fasting glucose level.ConclusionsMetabolic dysregulation predicts a poor course of late-life depression. This finding supports the concept of ‘metabolic depression’, recently proposed on population-based findings of a protracted course of depressive symptoms in the presence of metabolic dysregulation. Our findings seem to be driven by abdominal obesity (as indicated by the waist circumference) and HDL cholesterol dysregulation.
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John, George, Mona Asghari, Vipin VP, and Valsamma Eapen. "Depression and Metabolic Syndrome: Two Sides of the Same Coin." Journal of Biomedical and Clinical Research 12, no. 1 (July 1, 2019): 3–9. http://dx.doi.org/10.2478/jbcr-2019-0001.

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Summary This aim of this review was to examine the relationship between metabolic syndrome (MetS) and depression, which is complex and multifaceted with many inter-related factors includinggenetics, lifestylefactors, environmentalfactorsand other psychological factors at play. There is some evidence to suggest that depression may lead to the development of cardiovascular disease through its association with MetS. It has also been suggested that depressive symptoms may be a consequence rather than the cause of the MetS, as obesity and dyslipidemia have been shown as predictive of depressive symptoms. Thus, the relationship between MetS and depression seems to be a two-way street and bi-directional just as the two sides of the same coin.
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Olvera, Rene L., Douglas E. Williamson, Susan P. Fisher-Hoch, Kristina P. Vatcheva, and Joseph B. McCormick. "Depression, Obesity, and Metabolic Syndrome." Journal of Clinical Psychiatry 76, no. 10 (October 21, 2015): e1300-e1305. http://dx.doi.org/10.4088/jcp.14m09118.

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Dissertations / Theses on the topic "Metabolic depression"

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Muir, Timothy J. "Mechanisms and phylogenetic breadth of urea-induced hypometabolism." Oxford, Ohio : Miami University, 2009. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=miami1247688904.

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Tweedy, Maureen P. "Metabolic Syndrome and Psychosocial Factors." Thesis, University of North Texas, 2009. https://digital.library.unt.edu/ark:/67531/metadc11005/.

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Metabolic syndrome is a constellation of risk factors, including abdominal obesity, hypertriglyceridemia, low HDL cholesterol, high blood pressure, and high fasting glucose, that commonly cluster together and can result in cardiovascular disease. The prevalence of metabolic syndrome and the components that comprise the syndrome vary by age and by racial/ethnic group. In addition, previous research has indicated that the risk factors contributing to metabolic syndrome may be exacerbated by exposure to perceived stress. This study utilized data from the 2002, 2004, and 2006 Health and Retirement Study (HRS) and National Health and Nutrition Examination Survey (NHANES) data sets. It was hypothesized that depression and anxiety (conceptualized as stress in this study) increase the risk of presenting with metabolic syndrome while social support decreases the risk of metabolic syndrome. While results of cross-sectional analysis do not indicate a significant relationship between depression and metabolic syndrome (t = -.84, ns), longitudinal analysis does indicate a significant relationship between depression and metabolic syndrome over time (t = -5.20, p <.001). However, anxiety is not significantly related to metabolic syndrome when the relationship is examined through cross-sectional analysis (t = -1.51, ns) and longitudinal analysis (χ² = 13.83, ns). Similarly, social support is not significantly related to metabolic syndrome when examined in cross-sectional (χ² = .63, ns) and longitudinal (t = 1.53, ns) analysis. Although level of stress is not significantly related to metabolic syndrome as a whole, there is a significant relationship between stress and both triglyceride level (t = -2.94, p = .003) and blood glucose level (t = -3.26, p = .001).
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Tweedy, Maureen P. Guarnaccia Charles Anthony. "Metabolic syndrome and psychosocial factors." [Denton, Tex.] : University of North Texas, 2009. http://digital.library.unt.edu/permalink/meta-dc-11005.

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Bishop, T. "Living at a snail's pace : the cellular basis of metabolic depression." Thesis, University of Cambridge, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.596665.

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The garden snail Helix aspersa depresses its metabolic rate in response to desiccation (termed aestivation), as well as in response to reduced oxygen tension (termed oxygen conformation). This depression persists in cells isolated from the hepatopancreas of snails, thereby providing a good model system for analysing the cellular basis of metabolic depression. Isolated hepatopancreas cells were used to assess the contributions of various cellular processes (non-mitochondrial and mitochondrial respiration, and, within mitochondrial respiration, substrate oxidation and respiration to drive proton leak and ATP turnover) to metabolic depression seen during oxygen conformation and aestivation. Non-mitochondrial respiration accounts for a large proportion (~50%) of metabolic rate at physiological oxygen tensions, and it is solely responsible for the oxygen conforming behaviour of the cells. Both non-mitochondrial and mitochondrial respiration, however, decrease in aestivation. This decrease in mitochondrial respiration is not caused by differences in mitochondrial volume or inner membrane surface density but is associated with other intrinsic changes in the mitochondria (decrease in the activities of the mitochondrial enzymes citrate synthase and cytochrome c oxidase). Within mitochondrial respiration, the activity of substrate oxidation and probably ATP turnover, but not the activity of proton leak, decrease during aestivation. At least 75% of the total response of mitochondrial respiration to aestivation is due to primary changes in the kinetics of substrate oxidation, with only 25% or less of the response occurring through primary effects on ATP turnover. The primary change in the activity of substrate oxidation resulted in a lower mitochondrial membrane potential in hepatopancreas cells from aestivating compared to active snails, leading to secondary decreases in respiration to drive ATP turnover and proton leak.
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Wang, Yijie, and 王怡洁. "Associations among type A and type D personalities, metabolicsyndrome, and anxiety/depression." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2012. http://hub.hku.hk/bib/B47849496.

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Background Metabolic syndrome refers to a cluster of metabolic dysfunctions denoting a high risk of developing cardiovascular disease (CVD) and diabetes. The key risk factors include insulin resistance and obesity. In recent decades psychological factors, including Type A personality, Type D personality, anxiety, and depression, have been found to be additional risk factors of metabolic syndrome. As people‘s behaviours and personalities are often influenced by cultural values, it would seem to be necessary to examine the associations between Type A and Type D personalities and metabolic syndrome within the context of culture. This study specifically examines the issue in the context of Chinese culture. In addition, people with Type A personality who tend to feel impatience or time urgency, anger or hostility, and competitiveness, were reported to be positively associated with anxiety. People with Type D personality who would easily have negative affectivity and social inhibition were reported to be positively associated with anxiety as well as depression. Therefore, anxiety or depression might have an effect on the associations between Type A and Type D personalities. However, to my best knowledge, no previous studies have examined the associations among Type A and Type D personalities, metabolic syndrome, and anxiety/depression. Objective This study examines associations among Type A and Type D personalities, metabolic syndrome, and anxiety/depression. It includes: 1) validating the Chinese versions of the Type A personality scale (Adolescent/Adult Type A Behaviour Scale, AATABS-3) and Type D personality scale (DS-14); 2) assessing the associations between Type A and Type D personalities with metabolic syndrome; 3) investigating the associations between anxiety/depression and metabolic syndrome; 4) measuring the association between Type A personality and anxiety; 5) testing the association between Type D personality and anxiety, as well as depression; and 6) determining the associations among Type A and Type D personalities, metabolic syndrome, and anxiety/depression. Methods A cross-sectional questionnaire survey was conducted on a random community sample of 741 adults in Hong Kong recruited by cluster sampling. Participants meeting the screening criteria of metabolic syndrome were offered further physical examination for confirming the diagnosis. Results For the Chinese version AATABS-3 scale (revised-AATABS-3), exploratory factor analysis (EFA) produced an 11-item 3-factor solution. The three factors were: 1) impatience/time urgency; 2) hostility/anger; and 3) competitiveness. The revised-AATABS-3 scale showed satisfactory internal consistency (Cronbach‘s alpha = 0.74). For the Chinese version DS-14 scale (revised-DS-14), EFA provided a 10-item 2-factor solution. The two factors were: negative affectivity and social inhibition. The revised-DS-14 scale showed good internal consistency (Cronbach‘s alpha = 0.90). Gender differences appeared in the associations between Type A and Type D personalities and metabolic syndrome. In the total population and female participants, there were no significant associations between Type A personality and metabolic syndrome. However, male participants with Type A personality were positively associated with metabolic syndrome. In the total participants, there were no significant associations between Type D personality and metabolic syndrome. However, female participants with Type D personality were positively associated with metabolic syndrome; whereas male participants with Type D personality were negatively associated with metabolic syndrome. Anxiety affected the association between Type A personality and metabolic syndrome in males only, whereas it affected the association between Type D personality and metabolic syndrome both in females and males. Depression affected the association between Type D personality and metabolic syndrome both in females and males. Conclusion and Discussion The revised-AATABS-3 and revised-DS-14 scales showed satisfactory psychometric properties. They might be more convenient and acceptable than the original scales for measuring Type A and Type D personalities in future research. Since EFA was a preliminary method for scale validation, further validation studies for these two scales are needed, for examples, on concurrent and discriminant validity. Gender differences occurred in the associations between Type A and Type D personalities and metabolic syndrome. The key findings were: *Type A personality was a risk factor of metabolic syndrome in male participants. *Type D personality was a risk factor of metabolic syndrome in female participants, but it exhibited a protective effect for preventing metabolic syndrome in male participants. *Anxiety played a protective effect in the associations between Type A and Type D personalities and metabolic syndrome in male participants. *Depression had a protective effect on Type D personality for developing metabolic syndrome in female participants, and it reduced the protective effect of Type D personality for preventing metabolic syndrome in male participants. The results of this study may give directions to future studies pursuing further investigations on metabolic syndrome, particularly in regard to personality traits, lifestyles, mental health issues, and coping strategies in cultural and social contexts, as well as gender differences related to the endocrine system.
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Silveira, Lilian Cristina da. "Reorganização estrutural e metabólica do tecido cardíaco associada à dormência e jejum sazonal em lagartos teiú Tupinambis merianae." Universidade de São Paulo, 2011. http://www.teses.usp.br/teses/disponiveis/41/41135/tde-13052011-100148/.

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O coração é um órgão notável por sua flexibilidade estrutural e metabólica em resposta a variações de demanda. Na dormência sazonal, a interrupção da alimentação, associada à inatividade física e à acentuada redução da frequência cardíaca, ocasiona uma inibição da demanda sobre a função do órgão e, provavelmente, uma reorganização estrutural e metabólica do tecido cardíaco. Estes aspectos foram investigados ao longo do ciclo anual de atividades em lagartos teiú Tupinambis merianae, com o objetivo de examinar as alterações de capacidade funcional cardíaca dadas por ajustes da massa, estrutura e composição do tecido, por regulação do fluxo de substratos energéticos em vias de produção de energia e por mudanças da composição de ácidos graxos dos fosfolipídios das membranas. Grupos de animais jovens foram mortos em diferentes fases do primeiro ciclo anual e após 20 dias de jejum na fase ativa e o ventrículo cardíaco foi removido e pesado. Um fragmento da parede ventricular foi retirado, transferido para fixador e utilizado posteriormente para a confecção de cortes histológicos de 10 μm de espessura que foram analisados utilizando-se método estereológico. O restante do tecido ventricular foi congelado em N2 líquido e conservado em freezer -80 ºC. Os teores de água, proteína total e solúvel e lipídio total foram medidos por meio de ensaios padrão; as atividades máximas de enzimas foram medidas por espectrofotometria em condições saturantes de substratos e cofatores; e o perfil de ácidos graxos dos lipídios neutros e polares foi determinado por cromatografia gasosa. No início do outono, a massa ventricular relativa é 0,16% e aumenta 31% até o final desta fase, quando o miocárdio esponjoso possui aspecto denso e poucos espaços lacunares que ocupam cerca de 8% da área total do corte. Este arranjo é mantido na dormência, quando a massa ventricular relativa aumenta 29% em relação ao final do outono, e no início do despertar, quando a massa ventricular relativa diminui para valores semelhantes aos do final do outono. Após a retomada da alimentação, a massa ventricular relativa volta a exibir uma porcentagem comparável a da dormência, juntamente com um pequeno aumento da área de lacunas no miocárdio esponjoso. Na primavera, a massa ventricular relativa é de 0,24% e o miocárdio esponjoso possui aspecto extremamente reticulado, com 29% da área total do corte ocupada por espaços lacunares. Animais ativos submetidos a jejum apresentam redução de 19% da massa ventricular relativa em relação a animais alimentados. A densidade numérica de cardiomiócitos na camada esponjosa é 37% menor na dormência em relação à atividade de primavera, resultando em um volume calculado de um cardiomiócito nesta fase 52% maior em relação à atividade de primavera. A análise do teor de água, proteínas totais e solúveis não indica variação ao longo do ciclo anual, com exceção de uma tendência ao aumento do teor de água na dormência e de uma tendência à redução do teor de proteínas solúveis após o despertar e ingestão de água e no grupo de animais ativos submetidos ao jejum. Na atividade de outono e dormência de inverno a concentração de proteínas miofibrilares é reduzida em relação à atividade de primavera e aumenta no início do despertar após a ingestão de água. A concentração de lipídios totais é menor na dormência e despertar em relação à atividade de outono e no grupo de animais submetidos a jejum em relação a animais alimentados. As enzimas glicolíticas PK e LDH não variam ao longo do ciclo anual, enquanto a CS, indicadora da capacidade aeróbia, exibe forte tendência ao aumento na dormência, e a HOAD, enzima da β-oxidação lipídica, encontra-se inibida na dormência e no despertar em relação ao outono. Em contraste, com exceção da LDH que também não varia, a PK e a CS diminuem, enquanto que a HOAD é mantida constante após jejum na fase ativa. As variações do perfil de ácidos graxos da fração lipídica neutra sugerem que ácidos graxos insaturados são preferencialmente mobilizados das reservas do miocárdio durante a dormência e início do despertar, enquanto que no jejum durante a fase ativa as diferentes classes de ácidos graxos são equitativamente mobilizadas. A composição de ácidos graxos da fração lipídica polar exibe uma notável constância ao longo do ciclo anual, sugerindo que os ajustes à dormência sazonal não afetam de modo abrangente os fosfolipídios do tecido cardíaco e, portanto, não sugerem um papel preponderante de mudanças da composição lipídica das membranas na regulação metabólica sazonal nos teiús. Além disso, o contraste em relação às alterações observadas em mamíferos hibernantes sugere que, nestes, os ajustes seriam mais relacionados com a adaptação às baixas temperaturas corpóreas típicas da hibernação. A análise de regressão indica uma variação do conteúdo dos ácidos graxos C18:1n-9, C22:5n-6 e C22:6n-3 em função da massa corpórea dos jovens teiús e as mudanças do padrão alométrico sugerem uma relação entre o conteúdo destes ácidos graxos e as diferenças de taxa metabólica em animais de diferentes massas corpóreas, observadas em determinadas fases do ciclo anual de atividades e após o jejum durante a fase ativa.
The structural and metabolic flexibility of cardiac response to a variable physiological demand is notable. During seasonal dormancy, interruption of feeding together with inactivity and reduced heart beating, cause a large decrease of demand which probably brings about structural and metabolic heart tissue reorganization. These aspects were studied during the annual cycle in young tegu lizards Tupinambis merianae to investigate the hypothesis of seasonal changes of the heart capacities given by adjustments of tissue mass, structure and composition, by regulation of flux of substrates in the pathways of energy production, and by changes in the composition of fatty acids of tissue membranes. Groups of animals were killed in selected phases during the first year cycle of young tegus and after a 20 days fasting period during spring activity. Heart ventricle was removed and weighed and a tissue sample was collected and transfered to fixative solution, being used to obtain tissue slices of 10μm width for histological analysis with stereological tools. The remaining tissue was cut and split into aliquots, frozen in liquid N2 and stored at -80ºC. Later, the aliquots were used to assess the content of water, total and soluble proteins, and total lipids, by standard assays, the maximum activity of enzymes by spectrophotometry, and neutral and polar fatty acids profiles by gas chromatography. In early fall, the relative mass ventricle is 0.16%, and 31% increased in late fall, when the spongy myocardium appears dense and with few lacunar spaces which area corresponds to 8% of slice total área. During dormancy, the ventricle mass increases further 29%, decreasing to values of late fall during early arousal. After food intake, mass ventricle is again increased together with a small increase of the lacunar spaces, which appear highly expanded later in spring (29% of the total area), when tissue mass is 0,24% increased in relation to early fall. Unlike dormancy, fasting during spring caused a decrease of 19% of the ventricle mass. The cardiomyocytes density in the spongy layer is 37% decreased during dormancy while estimated cell volume is 52% increased, in relation to spring activity. There was no seasonal changes in the content of water and proteins in the groups analysed, except to a tendency to increase in the water content during dormancy, and to decrease in the soluble proteins in early arousal and in fasted animals. Myofibrillar protein is lower during fall and dormancy in relation to spring, increasing soon in early arousal after water intake. Total lipids decrease in the tissue during dormancy in relation to late fall by similar proportion than after fasting during spring. The glycolytic enzymes PK e LDH are unchanged during the year cycle, whereas the mitochondrial CS shows a tendency to increase, and HOAD, a β-oxidation enzyme, is decreased during dormancy and early arousal, in relation to fall. Unlike, PK and CS are decreased, while HOAD is unchanged after a period of fasting during spring. Fatty acids (FA) profiles of neutral lipids suggest that unsaturated FA are preferentially mobilized during dormancy and arousal, whereas all FA would be equally used during spring fasting. FA of polar lipids are remarkably constant during the year, suggesting that membrane FA in the heart tissue are not generally affected by season, and thus, results do not support a predominant role played by compositional changes of membranes in metabolic depression in the tegu. In addition, the otherwise distinct findings with hibernating mammals suggest that changes of FA composition in these animals would be an adaptation to the low body temperature of torpor, rather than mechanism of metabolic inhibition. Regression analysis indicate significant relationships of C18:1n-9, C22:5n-6, and C22:6n-3 contents as a function of body mass in young tegus, and changes in the allometric patterns are consistent with a putative relationship between these FA levels and the scaling of mass specific metabolic rates of young tegus during the year cycle.
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Hoopes, Lisa Ann. "Metabolic and thermoregulatory capabilities of juvenile steller sea lions, Eumetopias jubatus." [College Station, Tex. : Texas A&M University, 2007. http://hdl.handle.net/1969.1/ETD-TAMU-1390.

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Olvera, Anna E. "Diabetes in Latinas : depression, metabolic control and the roles of acculturation and social support." Access to abstract only; dissertation is embargoed until after 12/20/2006, 2005. http://www4.utsouthwestern.edu/library/ETD/etdDetails.cfm?etdID=135.

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Sharovsky, Lilian Lopes. "Análise de sintomas depressivos e ansiosos nas variáveis clínicas da síndrome metabólica." Universidade de São Paulo, 2010. http://www.teses.usp.br/teses/disponiveis/5/5131/tde-22062010-131123/.

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Introdução: A Síndrome Metabólica (SM) representa uma complexa interação entre aspectos genéticos e ambientais associados à adiposidade central, diabetes tipo 2, hipertensão arterial e dislipidemia, contribuindo para o aumento da morbimortalidade por eventos cardiovasculares e elevando concomitantemente os custos com os problemas de saúde que provocam. Os sintomas depressivos e ansiosos tem sido associados à SM. Uma das explicações possíveis para isso se dá através da ativação do eixo hipotálamo-hipófise-adrenal (HHA). Objetivos O objetivo do presente estudo é explorar a associação da intensidade dos sintomas depressivos e ansiosos com as variáveis clínicas da SM e com o cortisol salivar. Métodos: Estudo de corte transversal com seleção consecutiva de 136 pacientes ambulatoriais (idade=55,37±7,62), sendo 69 mulheres, realizado em hospital especializado em Cardiologia. Todos os pacientes possuíam diagnóstico médico de SM, de acordo com o National Cholesterol Education Program Revised (NCEP-ATPIII-R). Numa primeira visita, responderam a um questionário sociodemográfico e os sintomas depressivos foram acessados pelo Inventário de Depressão de BECK (BDI) enquanto os sintomas ansiosos pelo Hamilton Anxiety Rating Scale (HARS). Na segunda visita, uma semana depois, foram colhidas amostras de saliva para dosagem de cortisol, às 07:00h da manhã. Resultados: Verificou-se positiva e significante correlação entre BDI, glicemia e gênero (p<0,001). O mesmo foi observado entre HARS, glicemia e gênero (p<0,001). A correlação entre BDI e HARS também foi positiva. Observou-se uma correlação negativa entre HARS e idade: os de idade inferior a 50 anos apresentaram valores maiores que aqueles acima de 60 anos. Não verificou-se correlação entre BDI e cortisol salivar (p=0,56), entre HARS e cortisol salivar (p=0,39) Conclusão: A intensidade dos sintomas depressivos e ansiosos esteve associada à glicemia e ao gênero feminino, em pacientes portadores de SM. O cortisol salivar não se associou, de maneira significante, aos sintomas depressivos e nem aos ansiosos
Objective: Metabolic Syndrome (MetS) has been associated to depression sintomatology by means of the hypothalamus-pituitary-adrenal axle activation and consequent alterations of the cortisol levels. The objective of this study is to explore the association of depressive and anxiety symptoms severity with MetS clinical variables and salivary cortisol. Methods: We studied 136 consecutive ambulatory patients (aged= 55,37±7,62), n= 69 women, who presented MetS criteria according to the National Cholesterol Education Program Revised (NCEP-ATPIII-R). At the first visit they all completed a social-demographic questionnaire.Then, we assessed depressive symptoms by Beck Depression Inventory (BDI) and anxiety symptoms by Hamilton Anxiety Rating Scale (HARS). Salivary cortisol was assessed at 7:00h A.M. Results: We observed that the higher the intensity of the depressive symptoms by the BDI, the higher the glycemia value (p=0,01) in the female group (p 0,001). The same correlation was observed between HARS and glycemia (p=0,001) and HARS and genre (p=0,02). No correlation between depressive and anxiety symptoms with the other MetS clinical variable was found. The correlation was also positive between HARS and BDI. No correlation between depressive and anxiety symptoms with salivary cortisol was found. Furthermore, there was no association between cortisol and MS clinical variables. Conclusions: In the studied population, a presence of a higher intensity of depressive and anxiety symptoms was verified in the female group. We observed that, in this group, both the BDI and HARS value was explained by an increased fasting glucose. There was neither a correlation between depressive and anxiety symptoms and salivary cortisol
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Kemp, David E. "Use of Insulin Sensitizers as a Novel Treatment for Major Depressive Disorder: A Pilot Study of Pioglitazone for Major Depression Accompanied by Abdominal Obesity." Case Western Reserve University School of Graduate Studies / OhioLINK, 2010. http://rave.ohiolink.edu/etdc/view?acc_num=case1275567223.

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Books on the topic "Metabolic depression"

1

Farooqui, Akhlaq A. Metabolic syndrome: An important risk factor for stroke, Alzheimer disease, and depression. New York, NY: Springer, 2013.

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Jay, Lombard, and Monte Tom, eds. Freedom from disease: The breakthrough approach to preventing cancer, heart disease, alzheimer's, and depression by controlling insulin. New York: St. Martin's Griffin, 2009.

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Jay, Lombard, and Monte Tom, eds. Freedom from disease: The breakthrough approach to preventing cancer, heart disease, Alzheimer's, and depression by controlling insulin. New York: St. Martin's, 2008.

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Osher, Susan. One-carbon metabolism in adults with major depression. Ottawa: National Library of Canada, 1999.

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Jones, Shirley Anne. Tetrahydrobiopterin metabolism in depression and senile dementia of Alzheimer type. Birmingham: Aston University. Department of Pharmaceutical Sciences, 1988.

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1935-, Takada Akikazu, and Curzon G. 1928-, eds. Serotonin in the central nervous system and periphery: Proceedings of the Symposium on Serotonin in the Central Nervous System and Periphery, April 1-2, 1995, Nagoya, Japan. Amsterdam: Elsevier, 1995.

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Gissey, Lidia Castagneto, James R. Casella Mariolo, Geltrude Mingrone, and Francesco Rubino. Metabolic surgery and depression. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198789284.003.0012.

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The incidence of obesity is rising worldwide and so are its comorbidities: type-2 diabetes mellitus (T2DM), dyslipidaemia, hypertension, cardiovascular disease, sleep apnoea, and depression. Bariatric/metabolic surgery has established itself over the past several years as an effective treatment not only for morbid obesity but also for its associated morbidities. The effects of bariatric/metabolic surgery on depression are controversial, with some studies showing improvement and others demonstrating a worsening. However, a major drawback of these studies is that they do not compare patients with the same baseline psychiatric disorders. In fact, mild to severe depressive symptoms are observed in most candidates for bariatric/metabolic surgery. Preoperative evaluation of the patient’s mental state would enable identification of the appropriate interventions, enhancing long-term compliance and weight maintenance. It could also leverage psychological support in case the patient’s disorder relapses postoperatively. Preoperative evaluation should detect potential psychological contraindications to surgery, such as severe eating disorders.
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Bertocci, Michele A., and Mary L. Phillips. Neuroimaging of Depression. Edited by Dennis S. Charney, Eric J. Nestler, Pamela Sklar, and Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0025.

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This chapter illustrates the historical progression, methodological approaches, and current neurobiological understanding of depression, the first leading cause of mental and behavioral disorder disability in the United States. We describe and position, in relation to depressive symptoms, the complex abnormalities that depressed adults and youth show concerning neural function during tasks and at rest, structural abnormalities, as well as key neurotransmitter, neuroreceptor, and metabolic abnormalities that have been examined in the literature. We also describe newer findings and methods such as differentiating between unipolar and bipolar depression and applying machine learning to individual prediction. Finally, we provide suggestions for future study.
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Farooqui, Akhlaq A. Metabolic Syndrome: An Important Risk Factor for Stroke, Alzheimer Disease, and Depression. Springer, 2013.

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Farooqui, Akhlaq A. A. Metabolic Syndrome: An Important Risk Factor for Stroke, Alzheimer Disease, and Depression. Springer, 2015.

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Book chapters on the topic "Metabolic depression"

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Farooqui, Akhlaq A. "Metabolic Syndrome as a Risk Factor for Depression." In Metabolic Syndrome, 343–78. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-7318-3_9.

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Withers, Philip C., and Christine E. Cooper. "Metabolic Depression: A Historical Perspective." In Aestivation, 1–23. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-642-02421-4_1.

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Harvey, Brian H. "Major Depression and Metabolic Encephalopathy: Syndromes More Alike Than Not?" In Metabolic Encephalopathy, 349–69. New York, NY: Springer New York, 2008. http://dx.doi.org/10.1007/978-0-387-79112-8_18.

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Storey, Kenneth B., and Janet M. Storey. "Oxygen Limitation and Metabolic Rate Depression." In Functional Metabolism, 415–42. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2005. http://dx.doi.org/10.1002/047167558x.ch15.

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Brand, Martin D., Tammie Bishop, Robert G. Boutilier, and Julie St-Pierre. "Mitochondrial Proton Conductance, Standard Metabolic Rate and Metabolic Depression." In Life in the Cold, 413–30. Berlin, Heidelberg: Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-662-04162-8_44.

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Rosenblat, Joshua D., Ron Kakar, and Roger S. McIntyre. "Metabolic-Inflammation Aspects of Depression and Cardiovascular Disease." In Cardiovascular Diseases and Depression, 211–33. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-32480-7_14.

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Leonard, Brian E. "Depression, the Metabolic Syndrome and Neurodegeneration." In Current Topics in Neurotoxicity, 229–41. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-13602-8_11.

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N. Das, Undurti. "Neurochemical Linkage Among Metabolic Syndrome, Alzheimer Disease, and Depression." In Metabolic Syndrome and Neurological Disorders, 197–217. Chichester, UK: John Wiley & Sons Ltd, 2013. http://dx.doi.org/10.1002/9781118395318.ch11.

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Outhred, Tim, Claire McAulay, Danielle Gessler, and Gin S. Malhi. "Monitoring for Metabolic Dysfunction and Cardiovascular Disease in Bipolar Disorder: A Shared Illness Process Approach." In Cardiovascular Diseases and Depression, 333–50. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-32480-7_19.

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Goethe, John W., Bonnie L. Szarek, and Charles F. Caley. "Metabolic Syndrome in Psychiatric Inpatients Treated for Depression." In Modern Trends in Pharmacopsychiatry, 90–104. Basel: KARGER, 2009. http://dx.doi.org/10.1159/000189777.

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Conference papers on the topic "Metabolic depression"

1

NIE, ZHI, TAO YANG, YASHU LIU, QINGYANG LI, VAIBHAV A. NARAYAN, GAYLE WITTENBERG, and JIEPING YE. "MELANCHOLIC DEPRESSION PREDICTION BY IDENTIFYING REPRESENTATIVE FEATURES IN METABOLIC AND MICROARRAY PROFILES WITH MISSING VALUES." In Proceedings of the Pacific Symposium. WORLD SCIENTIFIC, 2014. http://dx.doi.org/10.1142/9789814644730_0043.

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Zöllner, R., M. Bopp, P. Dietsche, H. Rekate, B. Dietsche, A. Krug, B. Hanewald, O. Steinsträter, J. Sommer, and M. Zavorotnyy. "Structural and metabolic changes in the anterior cingulate cortex (ACC) after treatment with repetitive transcranial magnetic stimulation (rTMS) in patients with treatment-resistant unipolar depression (TRD)." In Abstracts of the 30th Symposium of the AGNP. Georg Thieme Verlag KG, 2017. http://dx.doi.org/10.1055/s-0037-1606408.

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Khoshnevis, Sepideh, Daniel W. Hensley, and Kenneth R. Diller. "Measurement and Analysis of Cutaneous Perfusion Depression During Cryotherapy." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53853.

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Localized cooling is commonly used after orthopedic surgery and in sports medicine to reduce bleeding, inflammation, metabolism, muscle spasm, pain, and swelling following musculoskeletal trauma and injury. The therapeutic application of cold therapy has a long history dating from the time of Hippocrates and has been widely documented in the literature1–3. Nonetheless, there remains to the present time considerable controversy over the appropriate protocol for application of cryotherapy. One extreme camp advocates continuous use of cryotherapy to a treatment site with no break in cooling for days or even weeks4–5, whereas other practitioners recommend a maximum application duration of 20 to 30 minutes followed by a cessation period of about 2 hours6–7. Although continuous cooling appears to be tolerated by many patients, there has been a large number of reported incidences in which continuous application of cryotherapy device led directly to extensive tissue necrosis and/or nerve injury in the treatment area, sometimes with dire medical consequences6,8.
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Chovancova, Olga, Jan Rabcan, Jozef Kostolny, and Denisa Macekova. "Human Reliability Evaluation through Analysis of Depression Prediction based on Metabolomic Data." In 2019 10th International Conference on Dependable Systems, Services and Technologies (DESSERT). IEEE, 2019. http://dx.doi.org/10.1109/dessert.2019.8770036.

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Xu, Jiale, Yingying Tang, C. Cecilio Baro, Xiaoliu Zhang, Ziyu Meng, and Yao Li. "Left fimbria atrophy is associated with hippocampal metabolism in female major depressive disorder patients." In 2018 40th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2018. http://dx.doi.org/10.1109/embc.2018.8512472.

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