Relevant bibliographies by topics / Memory – Effect of glucose on

Academic literature on the topic 'Memory – Effect of glucose on'

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Published: 10 December 2022
Last updated: 28 January 2023

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Journal articles on the topic "Memory – Effect of glucose on"

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Smith, Michael A., Leigh M. Riby, J. Anke M. van Eekelen, and Jonathan K. Foster. "Glucose enhancement of human memory: A comprehensive research review of the glucose memory facilitation effect." Neuroscience & Biobehavioral Reviews 35, no. 3 (January 2011): 770–83. http://dx.doi.org/10.1016/j.neubiorev.2010.09.008.

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FOX, COSETTE, and SABRINA PAJOR. "The Effect of Sucrose and Stress on Male Participants' Memory." Michigan Academician 47, no. 2 (January 1, 2021): 162–72. http://dx.doi.org/10.7245/0026-2005-47.2.162.

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ABSTRACT Glucose has been shown to have a memory facilitating effect. The goal of this study is to test if sucrose, a carbohydrate consumed on a daily basis, would also enhance memory in male college students. Subjects were given either a sucrose (50 g) or a placebo drink (50.6 mg of saccharine). Subjects filled the Stress Indicator Questionnaire that measures five stress indicators: physical, sleep, behavioral, emotional, and personal habits. A slideshow of 52 IAPS pictures were then shown to the subjects followed by immediate and delayed recall tests and a recognition test. Even though we found no direct effect of sucrose on memory, the results showed that high fasting blood glucose level is associated with lower recognition memory. Furthermore, high sleep stress enhanced memory for immediate recall. On the other hand, high behavioral stress was detrimental for delayed recall and recognition. The differential effects of the different indicators of stress on memory is discussed in relation to changes in cortisol levels that may result in modulation of blood glucose levels which in turn can affect memory. The results of this study shed light on the effect of different types of stress and fasting glucose levels on memory.
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Gancheva, Silvia M., and Maria D. Zhelyazkova-Savova. "Vitamin K2 Improves Anxiety and Depression but not Cognition in Rats with Metabolic Syndrome: a Role of Blood Glucose?" Folia Medica 58, no. 4 (December 1, 2016): 264–72. http://dx.doi.org/10.1515/folmed-2016-0032.

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AbstractBackground:The metabolic syndrome is a socially important disorder of energy utilization and storage, recognized as a factor predisposing to the development of depression, anxiety and cognitive impairment in humans.Aim:In the present study we examined the effects of vitamin K2 on the behavior of rats with metabolic syndrome and looked for relationships with the effects on blood sugar.Materials and methods:Male Wistar rats were divided in four groups: a control group on a regular rat chow, a metabolic syndrome (MS) group fed a high-fat high-fructose diet, a control group treated with vitamin K2 and a MS group treated with vitamin K2. Vitamin K2 was given by gavage. At the end of the study (after 10 weeks) behavioral tests were performed and fasting blood glucose was measured. Anxiety was determined using the social interaction test and depression was assessed by the Porsolt test. Memory effects were estimated by the object recognition test. Correlations between fasting blood glucose and behavioral performance were analyzed.Results:The rats from the MS group had elevated blood glucose. They had anxiety, depression and memory deficit. Vitamin K2 normalized blood glucose, reduced anxiety and depression, but did not improve memory. Time of social interaction (inverse index of anxiety) and memory recognition were negatively correlated with blood glucose in the untreated rats but the immobility time (measure of depression) was not. When vitamin K2-treated rats were added, the correlation of blood glucose with the time of social interaction was kept, but the one with the recognition memory was lost. It might be that the anxiolytic effect of vitamin K2 in this setting is at least partly due to its effects on blood glucose, while the anti-depressant effect is glucose-independent.Conclusion:The present study demonstrated that vitamin K2 prevented the development of anxiety and depression, but did not improve the memory deficit caused by the dietary manipulation in an experimental model of metabolic syndrome. It might be that the anxiolytic effect of vitamin K2 is at least partly due to its effects on blood glucose, while the antidepressant effect is glucose-independent.
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Messier, C. "Effect of Glucose and Peripheral Glucose Regulation on Memory in the Elderly." Neurobiology of Aging 18, no. 3 (May 6, 1997): 297–304. http://dx.doi.org/10.1016/s0197-4580(97)80311-9.

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Messier, Claude, Alain Desrochers, and Michèle Gagnon. "Effect of glucose, glucose regulation, and word imagery value on human memory." Behavioral Neuroscience 113, no. 3 (1999): 431–38. http://dx.doi.org/10.1037/0735-7044.113.3.431.

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Meikle, Andrew, Leigh M. Riby, and Brian Stollery. "Memory processing and the glucose facilitation effect: The effects of stimulus difficulty and memory load." Nutritional Neuroscience 8, no. 4 (August 2005): 227–32. http://dx.doi.org/10.1080/10284150500193833.

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Riby, Leigh M., Jennifer McLaughlin, and Deborah M. Riby. "Lifestyle, glucose regulation and the cognitive effects of glucose load in middle-aged adults." British Journal of Nutrition 100, no. 5 (November 2008): 1128–34. http://dx.doi.org/10.1017/s0007114508971324.

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Interventions aimed at improving glucose regulatory mechanisms have been suggested as a possible source of cognitive enhancement in the elderly. In particular, previous research has identified episodic memory as a target for facilitation after either moderate increases in glycaemia (after a glucose drink) or after improvements in glucose regulation. The present study aimed to extend this research by examining the joint effects of glucose ingestion and glucose regulation on cognition. In addition, risk factors associated with the development of poor glucose regulation in middle-aged adults were considered. In a repeated measures design, thirty-three middle-aged adults (aged 35–55 years) performed a battery of memory and non-memory tasks after either 25 g or 50 g glucose or a sweetness matched placebo drink. To assess the impact of individual differences in glucose regulation, blood glucose measurements were taken on four occasions during testing. A lifestyle and diet questionnaire was also administered. Consistent with previous research, episodic memory ability benefited from glucose ingestion when task demands were high. Blood glucose concentration was also found to predict performance across a number of cognitive domains. Interestingly, the risk factors associated with poor glucose regulation were linked to dietary impacts traditionally associated with poor health, e.g. the consumption of high-sugar sweets and drinks. The research replicates earlier work suggesting that task demands are critical to the glucose facilitation effect. Importantly, the data demonstrate clear associations between elevated glycaemia and relatively poor cognitive performance, which may be partly due to the effect of dietary and lifestyle factors.
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Messier, Claude, and Norman M. White. "Memory improvement by glucose, fructose, and two glucose analogs: A possible effect on peripheral glucose transport." Behavioral and Neural Biology 48, no. 1 (July 1987): 104–27. http://dx.doi.org/10.1016/s0163-1047(87)90634-0.

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Wu, Lung-Yuan, Wang-Chuan Chen, Fan-Shiu Tsai, Chin-Chuan Tsai, Chi-Rei Wu, and Li-Wei Lin. "p-Hydroxybenzyl Alcohol, an Active Phenolic Ingredient of Gastrodia elata, Reverses the Cycloheximide-Induced Memory Deficit by Activating the Adrenal Gland in Rats." American Journal of Chinese Medicine 43, no. 08 (January 2015): 1593–604. http://dx.doi.org/10.1142/s0192415x15500901.

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The present study investigated the ameliorating effects of p-hydroxybenzyl alcohol (HBA), an active phenolic ingredient of Gastrodia elata, on cycloheximide (CXM)-induced impairment of passive avoidance response and clarified the role of adrenal glands on the effect of HBA in rats. An adrenalectomy (ADX) caused the memory deficit from 1 to 3 days after surgery. Administration of corticosterone (CORT) plus glucose completely recovered the memory deficit caused by ADX, and this effect was better than that of glucose or CORT alone. HBA ameliorated the memory deficit induced by CXM in sham and ADX rats, but ADX partially blocked it. Furthermore, plasma glucose, epinephrine and adrenal steroid levels of ADX rats significantly decreased. Sham rats who received HBA had an increase in plasma glucose and adrenal steroid levels. Therefore, we suggest that the reversal of CXM-induced memory deficit by HBA was partially dependent on adrenal glands through the increase of the levels of plasma adrenal steroids.
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Riby, Leigh M., Hazel McMurtrie, Jonathan Smallwood, Carrie Ballantyne, Andrew Meikle, and Emily Smith. "The facilitative effects of glucose ingestion on memory retrieval in younger and older adults: is task difficulty or task domain critical?" British Journal of Nutrition 95, no. 2 (February 2006): 414–20. http://dx.doi.org/10.1079/bjn20051649.

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The ingestion of a glucose-containing drink has been shown to improve cognitive performance, particularly memory functioning. However, it remains unclear as to the extent to which task domain and task difficulty moderate the glucose enhancement effect. The aim of this research was to determine whether boosts in performance are restricted to particular classes of memory (episodic v. semantic) or to tasks of considerable cognitive load. A repeated measures (25g glucose v saccharin), counterbalanced, double-blind design was used with younger and older adults. Participants performed a battery of episodic (e.g. paired associate learning) and semantic memory (e.g. category verification) tasks under low and high cognitive load. Electrophysiological measures (heart rate and galvanic skin response) of arousal and mental effort were also gathered. The results indicated that whilst glucose appeared to aid episodic remembering, cognitive load did not exaggerate the facilitative effect. For semantic memory, there was little evidence to suggest that glucose can boost semantic memory retrieval even when the load was manipulated. One exception was that glucose facilitated performance during the difficult category fluency task. Regardless, the present findings are consistent with the domain-specific account in which glucose acts primarily on the hippocampal region, which is known to support episodic memory. The possible contribution of the hippocampus in semantic memory processing is also discussed.
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Dissertations / Theses on the topic "Memory – Effect of glucose on"

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Messier, Claude. "Effect of glucose on memory : examination of possible mechanisms." Thesis, McGill University, 1986. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=74362.

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Previous research has shown that ingestion of sucrose or injection of glucose following a learning experience can improve an animal's memory for that experience. The present work was directed towards elucidating the mechanisms by which sucrose and glucose produce this effect. Memory was tested by determining the effects of post-training injections of various substances on a conditioned emotional response. Glucose itself exerted a dose-dependent bidirectional action on retention. This action was shown not to depend on particular blood glucose levels. Insulin did not improve retention at any of the doses tested. Fructose, a sugar that does not cross the blood-brain barrier produced a dose-response effect on retention similar to that of glucose suggesting that fructose and glucose may act through a common peripheral mechanism. The observation of a memory improvement following injections of either 2-deoxyglucose or 3-O-methylgucose, two non-metabolized glucose analogs, suggested that the effect of glucose on retention may be due to an action on glucose transport and not to any metabolic effects of glucose. Two peripheral organs were examined for their possible involvement in the memory-improving action of glucose. This action was shown not to be dependent on the adrenal medulla which has been implicated in the action of other mnemoactive treatments. Partial denervation of the liver produced a partial attenuation of the effect of glucose on retention. The results are discussed in terms of the action of reinforcers on endogenous physiological mechanisms that modulate memory consolidation.
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White, Lynn H. "Task-specific effects of glucose and stress on memory." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp03/NQ44628.pdf.

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Christian, Leonie Marie. "The effect of glucose on memory and aspects of cognitive function." Thesis, University of Bristol, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.541645.

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Hope, Christopher. "Glucose administration effects on sensorimotor function and declarative memory." Thesis, University of Surrey, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.580354.

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This thesis aimed to examine the effects of glucose drink administration on sensorimotor function (studies 1 - 3) and declarative memory (study 4). Glucose had no effect on a modified version of the Hick task in study 1. However in study 2 we observed that glucose slowed reaction times (RTs) during the initial performance of the Eriksen flanker task. One possible reason for this effect is that glucose only slows sensorimotor function when a response is weakly associated with a stimulus, such as at the beginning of task performance. In study 1 stimulus-response (S-R) associations may have been too strong to observe a glucose slowing effect. Here participants performed a greater number of training trials and stimuli were arguably mapped more directly to a response compared to study 2. In study 3 we tested the hypothesis that glucose slows sensorimotor function when S-R associations are weak. Here we used a letter version of the Eriksen flanker task and kept S-R association consistently low by changing the stimulus set to a novel pair of letters every 80 trials. We found that glucose constantly slowed RTs for the duration of this task, a result which is congruent with the hypothesis that glucose slows sensorimotor function when S-R associations are weak. In study 4 we focused on the effects of glucose administration on declarative memory function and sought to determine whether glucose affected the encoding of stimuli in a word recognition task. Here we used ERPs as an online measure of encoding processes. Our findings were that glucose enhanced recognition performance, replicating the well established effect that glucose- facilitates declarative memory. Furthermore, during encoding, glucose affected ERP components associated with early sensory processing, visual word-form generation, lexical/semantic access and long-term memory encoding/consolidation. Furthermore there was a correlation between recognition performance and the degree to which glucose amplified the N400 component, an ERP potential associated with lexical/semantic access. The results of this study therefore indicate that glucose modulates encoding processes and that these effects may, at least partially, underlie the glucose facilitation of declarative memory.
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Abrams, Darren G. "Dopaminergic system involvement in the memory, modulating effects of glucose." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape7/PQDD_0004/MQ45204.pdf.

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Catherine, Nicole. "The relationships among the effect of carbohydrates on blood glucose, appetite, food intake, mood and memory." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape4/PQDD_0015/MQ54155.pdf.

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Horne, Pamela. "The effects of glucose on the memory and attention of newborn human infants." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape4/PQDD_0033/MQ64372.pdf.

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Horne, Pamela. "The effects of glucose on the memory and attention of newborn human infants /." Thesis, McGill University, 1999. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=30668.

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The objective of this randomized, double-blind, placebo-controlled trial was to determine whether glucose enhanced memory for a repeated auditory stimulus in human newborns. Infants consumed water or glucose (2-g/kg) solution. Memory test phases were: Orientation (turning towards the stimulus); Habituation (not turning towards), indicating familiarity; Delay (100 seconds); Spontaneous Recovery (stimulus representation: not turning towards indicates remembering, while turning towards indicates forgetting), and Novelty (turning towards a different word confirms wakefulness). Decreased head-turning towards during Spontaneous Recovery indicates enhanced memory. Blood glucose levels were measured after testing.
"Glucose" infants had higher blood glucose levels than "water" infants (p < 0.001). "Glucose" infants had significantly decreased turns towards during Spontaneous Recovery compared to "water" infants (p = 0.008), indicating memory enhancement.
Therefore, glucose specifically enhances memory for a repeated auditory stimulus in newborn humans. Elevating blood glucose levels by approximately 2 mmol/L appears to be sufficient for memory enhancement in healthy newborns.
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Wilson, Hanna Jade. "The effects of glucose on memory, attention and speed of information processing in healthy, young adults /." Title page, table of contents and abstract only, 2003. http://web4.library.adelaide.edu.au/theses/09SSPS/09sspsw7462.pdf.

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Elliott, Jade M. "The impact of glucose and glucoregulation on memory." Thesis, Northumbria University, 2010. http://nrl.northumbria.ac.uk/3674/.

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The effect of glucose on memory has been investigated for in excess of 25 years, with some consensus generated amongst the literature indicating that glucose has a facilitating effect. However, the robustness of the glucose effect has been questioned, with a considerable body of evidence reporting no glucose facilitation of memory. It has been suggested that glucoregulatory control may be a key mediating factor of the glucose effect. Glucoregulatory control and cognitive functioning are intrinsically linked, with cognitive impairments a common feature in populations presenting with poor glucoregulatory control such as diabetics, Alzheimer‘s disease sufferers, schizophrenics and the elderly. Although again the evidence has proven contradictory, with evidence to suggest that both better and poorer glucoregulators are more / less susceptible to the glucose effects on cognition. Verbal declarative memory has been reported to be the most reliably enhanced aspect of memory to benefit from a glucose effect. However, it is not yet clear whether verbal declarative memory as a whole is being facilitated, or whether the different phases of memory (encoding, consolidation, retrieval etc.) are differentially targeted. Consequently the primary aim of this thesis was to evaluate the effect of glucoregulatory control and glucose, on the different phases of verbal declarative memory. This was achieved through the use of novel paradigms employed previously within the cognitive sciences literature. Chapter 2 addressed a secondary aim of this thesis; investigating the current gap in the literature pertaining to the effect of glucose administration on cognition in children. Chapter 3 investigated the types of recognition (recollection and familiarity) that were made subsequent to a glucose load, using the 'remember/know' paradigm. Chapter 4 investigated encoding efficiency during the item method directed forgetting paradigm, in which participants actively attempt to forget specific stimuli through cessation of encoding. In chapters 5 and 6 the potential mediation of inhibition processes was explored, with both semantically related (Retrieval Induced Forgetting paradigm) and orthographically similar but semantically unrelated stimuli (Memory Blocking Effect paradigm). The tentative evidence presented in this thesis indicates that glucoregulatory control may mediate the glucose facilitation effect during the encoding phase, with better regulators seemingly benefiting from greater encoding benefits than poorer following glucose. Glucose was not observed to influence inhibition processes, or types of recognitions made. However, better glucoregulators exhibited more efficient adaptive inhibition (overcoming inhibition of blocking items to continue searching the lexicon and increased inhibition of semantically related competing stimuli). Administration of glucose did not mediate cognition in children, with the exception of an impairment of performance on a challenging reaction time task following 20 g of glucose. Memory phases are seemingly differentially affected by glucose administration, with the effect mediated by glucoregulatory control. Utilising the paradigms employed here (or similar) to investigate a range of populations presenting with cognitive decline/glucoregulatory control, would further allow the glucose and glucoregulatory effects on the different phases of memory to be further disentangled.
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Books on the topic "Memory – Effect of glucose on"

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Kohl, M. Shape memory microactuators. Berlin: Springer, 2004.

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Tietze, Holger. Phasenübergänge mit Memory Effekt: Neutronenstreuung an der Shape Memory Legierung NiTi. Frankfurt am Main: Verlag für Akademische Schriften, 1985.

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1927-, Funakubo Hiroyasu, ed. Shape memory alloys. New York: Gordon and Breach Science Publishers, 1987.

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Shape-memory polymers. Heidelberg: Springer, 2010.

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Johnson, Shannan M. Music jingles: Their effect on the memory of advertisements. Sudbury, Ont: Laurentian University, Department of Psychology, 1995.

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Hjelmsäter, Emma Roos af. Children's memory reports: The effect of co-witness influence. [Gothenburg]: University of Gothenburg, 2010.

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Stephen, Colagiuri, and Brand Miller Janette 1952-, eds. The pocket guide to the glucose revolution and losing weight. London: Coronet, 2000.

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R, Squire Larry, and Butters Nelson, eds. Neuropsychology of memory. 2nd ed. New York: Guilford Press, 1992.

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N, Popov N. Razrabotka progressivnykh tekhnologiĭ na osnove materialov, obladai︠u︡shchikh ėffektom pami︠a︡ti formy. Sarov: RFI︠A︡T︠S︡-VNIIĖF, 2008.

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International, Symposium on Shape Memory Alloys (1986 Guilin China). Shape memory alloy' 86': Proceedings of the International Symposium on Shape Memory Alloys, September 6-9, 1986, Guilin, China. Beijing: China Academic Publishers, 1986.

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Book chapters on the topic "Memory – Effect of glucose on"

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Morgan, Michael M., MacDonald J. Christie, Thomas Steckler, Ben J. Harrison, Christos Pantelis, Christof Baltes, Thomas Mueggler, et al. "Memory-Storage Effect." In Encyclopedia of Psychopharmacology, 754. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-68706-1_1663.

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Baddeley, A. D., and G. Hitch. "The recency effect." In Exploring Working Memory, 80–98. Abingdon, Oxon ; New York, NY : Routledge, 2017. | Series: World library of psychologists: Routledge, 2017. http://dx.doi.org/10.4324/9781315111261-8.

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Spellacy, William N. "Oral Contraceptives Effect on Glucose Metabolism." In Clinical Perspectives in Obstetrics and Gynecology, 25–33. New York, NY: Springer New York, 1993. http://dx.doi.org/10.1007/978-1-4612-2730-4_3.

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Lüst, Dieter, and Ward Vleeshouwers. "The Gravitational Memory Effect." In SpringerBriefs in Physics, 105–6. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-10919-6_26.

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Gold, Paul E. "Plasma Glucose Regulation of Memory Storage Processes." In Cellular Mechanisms of Conditioning and Behavioral Plasticity, 329–41. Boston, MA: Springer US, 1988. http://dx.doi.org/10.1007/978-1-4757-9610-0_31.

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Chatham, John C. "The Effect of Diabetes on Glucose Metabolism." In The Heart in Diabetes, 189–214. Boston, MA: Springer US, 1996. http://dx.doi.org/10.1007/978-1-4613-1269-7_9.

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Huang, Wei Min, and Witold M. Sokolowski. "Effect of Cold Storage." In Cold Hibernated Elastic Memory Structure, 101–5. First edition. | Boca Raton, FL : CRC Press/Taylor & Francis Group, 2018.: CRC Press, 2018. http://dx.doi.org/10.1201/9780429425950-12.

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Getov, Vladimir. "Benchmarking the cache memory effect." In Lecture Notes in Computer Science, 233–46. Berlin, Heidelberg: Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/3-540-60902-4_27.

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Lee, Tanya, and Jean-Jacques Dugoua. "Nutritional Supplements and Their Effect on Glucose Control." In Advances in Experimental Medicine and Biology, 381–95. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-5441-0_27.

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Yamasaki, Kazuo. "Effect of Some Saponins on Glucose Transport System." In Advances in Experimental Medicine and Biology, 195–206. Boston, MA: Springer US, 1996. http://dx.doi.org/10.1007/978-1-4899-1367-8_18.

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Conference papers on the topic "Memory – Effect of glucose on"

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Siqueira, Fernando, Vanessa Siqueira, Lucas Falcão, Arthur Bezerra, and Carlos Silva. "THE INFLUENCE BETWEEN ALZHEIMER’S DISEASE AND HEALTHY EATING: A SYSTEMATIC REVIEW." In XIII Meeting of Researchers on Alzheimer's Disease and Related Disorders. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1980-5764.rpda062.

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Background: Alzheimer’s Disease (AD) is a neurodegenerative disease responsible for neuronal losses that affect mainly the cortex and hippocampus, which begin to shrink in size, damaging cognitive functions. This process affects cholinergic neurons, influencing acetylcholine (ACH) levels, a memoryrelated neurotransmitter. Glucose metabolism and low thiamine levels appear to be affected by AD. Consequently, diabetes becomes a disease associated with AD and the thiamine deficiency levels depress the use of glucose by the brain. Thus, nutrition may have a role in preventing dementia through the treatment and prevention. Objective: To summarize the knowledge about this topic by reviewing articles and analyzing if healthy eating influences the development of AD. Methods: Selection of articles from the Scielo database. Results: Inflammation contributes to the pathogenesis of AD. The effect observed in patients with adherence to the Mediterranean diet translates into a decrease in inflammatory markers at the plasma level. One of the symptoms, memory loss, can be prevented by a micronutrient named thiamine, a precursor of ACH, it is found in the leguminous. The Mediterranean diet has been shown to attribute a neuroprotective activity which goes with its anti-inflammatory effect. Conclusion: AD starts by its multifactorial etiology that consists of genotype and phenotype. Nutrition would be efficient as a preventive and a therapeutic alternative among other.
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Sabah, Al-ithawi. "Measurement of blood glucose level by Faraday effect." In 4TH ELECTRONIC AND GREEN MATERIALS INTERNATIONAL CONFERENCE 2018 (EGM 2018). Author(s), 2018. http://dx.doi.org/10.1063/1.5080822.

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Ke, H., T. Yang, and G. Li. "Effect of Blood Glucose Tolerance on Pulmonary Function." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a3247.

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Daniel Szöllosi, Zoltan Kovács, Evelin Várvölgyi, and Andras Fekete. "The Effect of Glucose on Electronic Taste Analyzer." In 2013 Kansas City, Missouri, July 21 - July 24, 2013. St. Joseph, MI: American Society of Agricultural and Biological Engineers, 2013. http://dx.doi.org/10.13031/aim.20131619422.

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Meng, Han. "Smart Continuous Glucose Monitoring Prediction with Grid Long Short-Term Memory." In ICCSIE2022: 7th International Conference on Cyber Security and Information Engineering. New York, NY, USA: ACM, 2022. http://dx.doi.org/10.1145/3558819.3565076.

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Rahman, Md Sazedur, Faisal Badal, Md Shahinur Alam, Mahib Tanvir, Sazzed Mahamud Khan, and Sajal Das. "Effect of PID Controller on Blood Glucose Concentration for Varying Plasma Insulin, Independent Glucose Flux, Renal Glucose Clearance and Gut Absorption Rate." In 2021 International Conference on Automation, Control and Mechatronics for Industry 4.0 (ACMI). IEEE, 2021. http://dx.doi.org/10.1109/acmi53878.2021.9528121.

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Wang, Yunjie, and Katherine Yanhang Zhang. "The Biomechanical Properties of Arterial Elastin With Glucose Effect." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14200.

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Elastin, as one of the major extracellular matrix (ECM) components, is essential to accommodate physiological deformation and provide elastic support for blood vessels. Elastin is a long-lived ECM protein and it can suffer from cumulative effects of exposure to chemical damage, which can greatly compromise its biomechanical properties. The mechanical properties of elastin are related to its microstructure and the chemical environment. Glucose is an important carbohydrate in human body. The effect of glucose on the mechanical properties of blood vessels is especially magnified in diabetic patients [1]. Glucose can directly condense with amino groups of proteins by nonenzymatic glycation, which is one of the main mechanisms of aging [2].
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King, Timothy W., and Gerard L. Cote. "Closed loop polarimetric glucose sensing using the pockels effect." In 1992 14th Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 1992. http://dx.doi.org/10.1109/iembs.1992.5760906.

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King and Cote. "Closed Loop Polarimetric Glucose Sensing Using The Pockels Effect." In Proceedings of the Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 1992. http://dx.doi.org/10.1109/iembs.1992.589588.

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Ziemys, Arturas, Alessandro Grattoni, Jaskaran Gill, and Mauro Ferrari. "Silica Nanochannel Surface Effect on Monosaccharide Transport." In ASME 2010 First Global Congress on NanoEngineering for Medicine and Biology. ASMEDC, 2010. http://dx.doi.org/10.1115/nemb2010-13216.

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The interface of silica nanochannel of 10 nm was studied by molecular modeling and experimental methods. Molecular Dynamics study on glucose solution revealed that 2–3 nm of interface solution to silica walls has reduced glucose diffusivity. That reduction affects the effective diffusivity of glucose in silica nanochannel. Experimental results show Fickian-like release of glucose through 13 nm nanochannel. Molecular modeling and experimental results suggest that glucose is not sufficiently confined to possess non-Fickian behavior.
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Reports on the topic "Memory – Effect of glucose on"

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Lahti, Janet. The effect of glucose on the food intake of goldthioglucose injected mice. Portland State University Library, January 2000. http://dx.doi.org/10.15760/etd.1571.

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Donahue, Katrina, Laura Young, John Buse, Mark Weaver, Maihan Vu, C. Madeline Mitchell, Tamara Blakeney, Kimberlea Grimm, Jennifer Rees, and Franklin Niblock. Effect of Glucose Monitoring on Patient and Provider Outcomes in Non-Insulin Treated Diabetes. Patient-Centered Outcomes Research Institute (PCORI), March 2018. http://dx.doi.org/10.25302/3.2018.ce.12114980.

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Baker, Linda. The effect of visual memory training on spelling achievement in the classroom. Portland State University Library, January 2000. http://dx.doi.org/10.15760/etd.5995.

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Han, Yiran, Zeyuan Lu, Meng Meng, Heran Wang, Pan Ting, Gao Tianjiao, and Mingjun Liu. Effect of Acupuncture on Glucose and Lipid Metabolism in Obese Type 2 Diabetes: A Systematic Review and Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, March 2021. http://dx.doi.org/10.37766/inplasy2021.3.0087.

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Han, Yiran, Zeyuan Lu, Meng Meng, Heran Wang, Pan Ting, Tianjiao Gao, and Mingjun Liu. Effect of Electroacupuncture on Glucose and Lipid Metabolism in Type 2 Diabetes: A protocol for Systematic Review and Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, August 2021. http://dx.doi.org/10.37766/inplasy2021.8.0008.

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Wang, Yudong, Guifen Fu, Xiang Li, Jiaxia Han, Jingfeng Chen, and Xiaohui Wei. Effect of different continuous glucose monitoring durations on glycemic control in Diabetes：A Systematic Review With Meta-analysis of Randomized Controlled Trials. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2021. http://dx.doi.org/10.37766/inplasy2021.11.0080.

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Ren, Bangjiaxin, and Ming Chen. Effect of Sodium‐glucose cotransport‐2 inhibitors on lowering blood pressure in patients with pre-hypertension and early hypertension: A meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, January 2021. http://dx.doi.org/10.37766/inplasy2021.2.0004.

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Battiato, James M., Thomas W. Stone, Miles J. Murdocca, Rebecca J. Bussjager, and Paul R. Cook. Free Space Optical Memory Based on Vertical Cavity Surface Emitting Lasers and Self-Electro-Optic Effect Devices. Fort Belvoir, VA: Defense Technical Information Center, April 1995. http://dx.doi.org/10.21236/ada297049.

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Vandermeer, R. A. Shape memory effect in uranium-niobium alloys below room temperature. Final report. [6. 2 to 7. 0 wt % Nb]. Office of Scientific and Technical Information (OSTI), March 1985. http://dx.doi.org/10.2172/5569116.

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Bennett, Alan B., Arthur Schaffer, and David Granot. Genetic and Biochemical Characterization of Fructose Accumulation: A Strategy to Improve Fruit Quality. United States Department of Agriculture, June 2000. http://dx.doi.org/10.32747/2000.7571353.bard.

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The goal of the research project was to evaluate the potential to genetically modify or engineer carbohydrate metabolism in tomato fruit to enhance levels of fructose, a sugar with nearly twice the sweetness value of other sugars. The specific research objectives to achieve that goal were to: 1. Establish the inheritance of a fructose-accumulating trait identified in F1 hybrids of an inferspecific cross between L. hirsutum XL. esculentum and identify linked molecular markers to facilitate its introgression into tomato cultivars. This objective was completed with the genetic data indicating a single major gene, termed Fgr (Fructose glucose ratio), that controlled the partitioning of hexose in the mature fruit. Molecular markers for the gene, were developed to aid introgression of this gene into cultivated tomato. In addition, a second major gene encoding fructokinase 2 (FK2) was found to be a determinant of the fructose to glucose ratio in fruit. The relationship between FK2 and Fgr is epistatic with a combined synergistic effect of the two hirsutum-derived genes on fructose/glucose ratios. 2. Characterize the metabolic and transport properties responsible for high fructose/glucose ratios in fructose-accumulating genotypes. The effect of both the Fgr and FK2 genes on the developmental accumulation of hexoses was studied in a wide range of genetic backgrounds. In all backgrounds the trait is a developmental one and that the increase in fructose to glucose ratio occurs at the breaker stage of fruit development. The following enzymes were assayed, none of which showed differences between genotypes, at either the breaker or ripe stage: invertase, sucrose synthase, FK1, FK2, hexokinase, PGI and PGM. The lack of effect of the FK2 gene on fructokinase activity is surprising and at present we have no explanation for the phenomenon. However, the hirsutum derived Fgr allele was associated with significantly lower levels of phosphorylated glucose, G1c-1-P and G1c-6-P and concomitantly higher levels of the phosphorylated fructose, Fru-6-P, in both the breaker and ripe stage. This suggests a significant role for the isomerase reaction. 3. Develop and implement molecular genetic strategies for the production of transgenic plants with altered levels of enzymes that potentially control fructose/glucose ratios in fruit. This objective focused on manipulating hexokinase and fructokinase expression in transgenic plants. Two highly divergent cDNA clones (Frk1 and Frk2), encoding fructokinase (EC 2.7.1.4), were isolated from tomato (Lycopersicon esculentum) and a potato fructokinase cDNA clone was obtained from Dr. Howard Davies. Following expression in yeast, each fructokinase was identified to code for one of the tomato or potato fructokinase isoforms Transgenic tomato plants were generated with the fructokinase cDNA clone in both sense and antisense orientations and the effect of the gene on tomato plants is currently being studied.
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