Relevant bibliographies by topics / Memory disorders

Academic literature on the topic 'Memory disorders'

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Published: 4 June 2021
Last updated: 29 July 2024

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Journal articles on the topic "Memory disorders"

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McNally, Richard J. "Memory and anxiety disorders." Philosophical Transactions of the Royal Society of London. Series B: Biological Sciences 352, no. 1362 (November 29, 1997): 1755–59. http://dx.doi.org/10.1098/rstb.1997.0158.

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Experimental psychopathologists have identified varying patterns in memory bias in people with depressive and anxiety disorders. Individuals suffering from depression tend to exhibit explicit memory deficits for positively-valanced material, and sometimes exhibit biases for retrieving negative self-relevant information as well. Most studies, however, provide scant evidence for implicit memory biases in depression. In contrast to depression, anxiety disorders are rarely associated with enhanced esplicit memory for threat-related information (with the exception of panic disorder). Evidence for implicit memory biases for threat in these syndromes is mixed. After providing an overview of findings on memory abnormalities in depressive and anxiety disorders, data from several new studies bearing on posttraumatic stress disorder (PTSD) in Vietnam combat veterans and in women with histories of childhood sexual abuse are presented. Involving directed forgetting, implicit memory and autobiographical cueing paradigms, these experiments point to a pattern of abnormalities linked to PTSD rather than to trauma per se.
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Miller, E. "Disorders of memory." Current Opinion in Psychiatry 1, no. 6 (November 1988): 740–44. http://dx.doi.org/10.1097/00001504-198811000-00016.

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Miller, Edgar. "Disorders of memory." Current Opinion in Psychiatry 4, no. 6 (December 1991): 902–4. http://dx.doi.org/10.1097/00001504-199112000-00017.

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Kopelman, M. D. "Disorders of memory." Brain 125, no. 10 (October 1, 2002): 2152–90. http://dx.doi.org/10.1093/brain/awf229.

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Hodges, John R., and Karalyn Patterson. "Semantic memory disorders." Trends in Cognitive Sciences 1, no. 2 (May 1997): 68–72. http://dx.doi.org/10.1016/s1364-6613(97)01022-x.

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Lucas, John A. "Disorders of Memory." Psychiatric Clinics of North America 28, no. 3 (September 2005): 581–97. http://dx.doi.org/10.1016/j.psc.2005.05.009.

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CAINE, ERIC D. "Clinical Disorders of Memory; Rehabilitation of Memory." American Journal of Psychiatry 145, no. 6 (June 1988): 760—a—761. http://dx.doi.org/10.1176/ajp.145.6.760-a.

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Ullman, Michael T., F. Sayako Earle, Matthew Walenski, and Karolina Janacsek. "The Neurocognition of Developmental Disorders of Language." Annual Review of Psychology 71, no. 1 (January 4, 2020): 389–417. http://dx.doi.org/10.1146/annurev-psych-122216-011555.

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Developmental disorders of language include developmental language disorder, dyslexia, and motor-speech disorders such as articulation disorder and stuttering. These disorders have generally been explained by accounts that focus on their behavioral rather than neural characteristics; their processing rather than learning impairments; and each disorder separately rather than together, despite their commonalities and comorbidities. Here we update and review a unifying neurocognitive account—the Procedural circuit Deficit Hypothesis (PDH). The PDH posits that abnormalities of brain structures underlying procedural memory (learning and memory that rely on the basal ganglia and associated circuitry) can explain numerous brain and behavioral characteristics across learning and processing, in multiple disorders, including both commonalities and differences. We describe procedural memory, examine its role in various aspects of language, and then present the PDH and relevant evidence across language-related disorders. The PDH has substantial explanatory power, and both basic research and translational implications.
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Delgadillo, Mia, Megan Frank, Aidan Boese, Tilman Schulte, and J. Kaci Fairchild. "Psychiatric Disorders and Mild Cognitive Impairment in Older Veterans With Subjective Memory Complaints." Innovation in Aging 4, Supplement_1 (December 1, 2020): 295. http://dx.doi.org/10.1093/geroni/igaa057.944.

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Abstract Psychiatric disorders pose a unique risk for Alzheimer’s disease (AD). Prior research indicates psychiatric disorders in MCI increase AD vulnerability. Less research has been done to understand how psychiatric disorders may affect the development of MCI. Understanding these potentially modifiable risk factors is important as they may represent a potential target of intervention for secondary prevention of AD. The present study examines the relationship between psychiatric disorders and amnestic MCI (aMCI) in a sample of Veterans with subjective memory complaints. The sample included 150 older adults with subjective memory complaints (90% male, age = 70.6±8.2). aMCI diagnosis was based upon performance on the delayed recall trials of the Rey Auditory Verbal Learning Test and Logical Memory II of the Wechsler Memory Scale-4th edition. Psychiatric disorders (e.g., Mood Disorders, Anxiety Disorders, and Substance Use Disorders) were assessed using the Mini Neuropsychiatric Interview for DSM-IV. Logistic regression modeling demonstrated that diagnosis of anxiety disorders, but not mood or substance use disorders, was significantly associated with aMCI status. Specifically, older adults with an anxiety disorder were less likely to have aMCI than those older adults without an anxiety disorder. Additional analyses revealed that within those with aMCI (n=107), persons with a psychiatric disorder were significantly younger than those without a psychiatric disorder by an average of 6 years. These findings support prior research on the complex relationship of anxiety and cognitive impairment as well as suggest that those with psychiatric disorders may be at risk for developing aMCI at younger ages.
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Fujii, Toshikatsu. "Memory and its disorders." Higher Brain Function Research 30, no. 1 (2010): 19–24. http://dx.doi.org/10.2496/hbfr.30.19.

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Dissertations / Theses on the topic "Memory disorders"

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Kenny, Lucy Margaret. "Memory processes in posttraumatic stress disorder." [New South Wales : University of New South Wales], 2006. http://www.library.unsw.edu.au/~thesis/adt-NUN/uploads/approved/adt-NUN20061110.142022/public/02whole.pdf.

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Westerberg, Helena. "Working memory : development, disorders and training /." Stockholm, 2004. http://diss.kib.ki.se/2004/91-7349-881-5/.

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Ostrovskaya, Irina. "Emotional memory and anxiety disorders: the impact of fear upon memory." Thesis, Boston University, 2003. https://hdl.handle.net/2144/27737.

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Boston University. University Professors Program Senior theses.
PLEASE NOTE: Boston University Libraries did not receive an Authorization To Manage form for this thesis. It is therefore not openly accessible, though it may be available by request. If you are the author or principal advisor of this work and would like to request open access for it, please contact us at open-help@bu.edu. Thank you.
2031-01-02
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Dewar, Bonnie-Kate Louise. "A memory aids clinic for the rehabilitation of acquired memory disorders." Thesis, King's College London (University of London), 2014. http://kclpure.kcl.ac.uk/portal/en/theses/a-memory-aids-clinic-for-the-rehabilitation-of-acquired-memory-disorders(dbd52b48-bd5f-40e9-80b3-b4a36572a9e2).html.

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Acquired memory impairment commonly occurs after acquired brain injury such as traumatic brain injury, stroke, seizure disorder and encephalitis and is one the defining features of progressive disorders, such as Alzheimer’s disease. There is a growing body of knowledge about the use of compensatory memory aids in the rehabilitation of memory disorders. This study investigated the effect of the systematic training of compensatory memory aids on everyday memory performance within a Memory Aids Clinic, a specialised outpatient clinic which supplied and trained the use of memory aids. A comparison was made between subjects with acquired memory disorders in a treatment group (n=63) and control group (n = 28) in a between subjects design. All subjects underwent a baseline session which was comprised of a neuropsychological assessment, clinical interview and goal setting session. Treatment subjects then underwent three training sessions, matching memory aids to goals, across a six week period. Training was based upon Sohlberg and Mateer’s (1989) application, acquisition and adaptation program. A follow-up session was conducted 12 weeks after the conclusion of training and included a review neuropsychological assessment and interview. Following the baseline session, control subjects were given written information about the management of memory problems and placed on an 18 week waiting list, prior to the three treatment sessions. The main outcome measure was everyday memory performance as assessed by a memory goal attainment diary. This was administered at baseline, the end of training and at the follow-up session. Outcome was also measured in terms of neuropsychological performance and performance on measures of mood, self-esteem, subjective memory performance, participation and carer strain. Generalisation was measured in terms of performance on a Problem Solving Inventory, a list of hypothetical memory scenarios which was developed for this study. The systematic training of memory aids in a Memory Aids Clinic was effective in improving everyday memory performance, but only across timefrom the end of training to follow-up. A significant difference in goal attainment was evident between the treatment and control group at follow-up but not at the end of training. Further analysis confirmed a significant treatment effect for participants with a non-progressive condition but not a progressive condition. This pattern is further illustrated by a series of case reports describing in detail the training content in the Memory Aids Clinic for both non-progressive and progressive subjects. In contrast to previous literature, there were no demographic or neuropsychological profile predictors of outcome in the current study. In addition, training in the Memory Aids Clinic did not impact upon the psychosocial measures of mood, self-esteem, participation, subjective memory function or carer strain. The treatment effect did generalise to the Problem Solving Inventory, notably for subjects with a progressive condition, suggesting adaptation of training to similar but diverse memory scenarios. Systematic training within the setting of a Memory Aids Clinic is beneficial for individuals with acquired memory problems as a consequence of a non-progressive condition. Improvement in everyday memory performance was attained with three training sessions and maintained across time. Training requires matching the memory aid to the client’s goals and scope remains for extension of training to encompass awareness of when to use the memory aid and acceptance of memory problems. Whilst the current results indicated that individuals with progressive memory disorders did not benefit from training in the Memory Aids Clinic, it is premature to suggest that this group cannot benefit from the use of memory aids. Additional training sessions may be required to facilitate generalisation into the home environment, to enhance the use of a significant other as co-therapist and to consolidate initial gains. A different and more general approach to using memory strategies including compensatory aids may be more beneficial in this group given more widespread and severe cognitive impairments. Finally, the results of the current study highlight the need for ecologically valid measures of outcome, including appropriate tools to determine self-efficacy and participation.
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Meeter, Martijn. "Long-term memory disorders measurement and modeling /." [S.l. : Amsterdam : s.n.] ; Universiteit van Amsterdam [Host], 2003. http://dare.uva.nl/document/69462.

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Kitchener, Erin Grace. "Selective disorders of memory in neurological patients." Thesis, University of Cambridge, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.627365.

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Airaksinen, Eija. "Cognitive functions in depression and anxiety disorders : findings from a population-based study /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-954-8/.

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Ricker, Timothy J. Cowan Nelson. "Cognitive load and time based forgetting." Diss., Columbia, Mo. : University of Missouri--Columbia, 2009. http://hdl.handle.net/10355/6470.

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Title from PDF of title page (University of Missouri--Columbia, viewed on Feb 18, 2010). The entire thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file; a non-technical public abstract appears in the public.pdf file. Thesis advisor: Dr. Nelson Cowan. Includes bibliographical references.
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Bakerink, Ronda Ann. "Semantic memory in Alzheimer's Disease." Thesis, University of British Columbia, 1988. http://hdl.handle.net/2429/27795.

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Alzheimer's Disease is characterized by a general decline in cognitive functioning. Although phonology are relatively unaffected, patients with Alzheimer's Disease have been reported to have deficits of semantic memory. Thirteen patients with dementia, five of whom had a confirmed diagnosis of dementia, participated in the study. The purpose of this investigation was to replicate a study performed by Mark Byrd (1984), using Alzheimer's Disease patients. Subjects were presented with category-word decision pairs, for which the task was to decide if the word was an exemplar of the category, and category-letter decision pairs for which the task was to generate an exemplar of the category beginning with the letter. The dependent variable was reaction time. Results indicated that Alzheimer's Disease patients and dementia patients had longer reaction times than a group of age-matched control subjects, and that the Alzheimer's Disease and dementia patients showed a pattern of responses similar to that of the control subjects. All groups showed longer reaction times for the generation trials than the decision trials. The results are consistent with the existence of a semantic memory deficit in Alzheimer's Disease, but other interpretations were discussed.
Medicine, Faculty of
Audiology and Speech Sciences, School of
Graduate
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Parameshwaran, Kodeeswaran Suppiramaniam Vishnu. "Prenatal nicotine exposure and molecular mechanisms of memory impairment." Auburn, Ala, 2008. http://repo.lib.auburn.edu/EtdRoot/2008/SUMMER/Pharmacal_Sciences/Dissertation/Parameshwaran_Kodeeswaran_21.pdf.

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Books on the topic "Memory disorders"

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Khan, Aman U. Clinical Disorders of Memory. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-5128-3.

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Völzke, Volker. Patients with Memory Disorders. Wiesbaden: Springer Fachmedien Wiesbaden, 2023. http://dx.doi.org/10.1007/978-3-658-39800-2.

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1934-, Baddeley Alan D., Wilson Barbara A. 1941-, and Watts Fraser N, eds. Handbook of memory disorders. Chichester: Wiley, 1995.

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Mayes, Andrew. Human organic memory disorders. Cambridge: Cambridge University Press, 1988.

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1934-, Baddeley Alan D., Wilson Barbara A. 1941-, and Watts Fraser N, eds. Handbook of memory disorders. Chichester: Wiley, 1996.

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Carol, Turkington, ed. The encyclopedia of memory and memory disorders. New York, NY: Facts on File, 1994.

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1936-, Yanagihara Takehiko, and Petersen Ronald C. 1946-, eds. Memory disorders: Research and clinical practice. New York: M. Dekker, 1991.

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1950-, Gilbert Susan, Nelson Aaron, Corliss Julie, and Harvard Medical School, eds. Improving memory: Understanding age-related memory loss. Boston, MA: Harvard Health Publications, 2008.

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1934-, Baddeley Alan D., Kopelman Michael D, and Wilson Barbara A, eds. The handbook of memory disorders. 2nd ed. New York: J. Wiley, 2002.

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1934-, Baddeley Alan D., Kopelman Michael D, and Wilson Barbara A. 1941-, eds. The essential handbook of memory disorders for clinicians. Chichester, West Sussex, England: J. Wiley, 2004.

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Book chapters on the topic "Memory disorders"

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Medved, Maria, and Jens Brockmeier. "Memory Disorders." In Encyclopedia of Critical Psychology, 1168–71. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-5583-7_184.

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Verfaellie, Mieke, and Margaret M. Keane. "Memory disorders." In APA handbook of neuropsychology, Volume 1: Neurobehavioral disorders and conditions: Accepted science and open questions (Vol. 1)., 205–24. Washington: American Psychological Association, 2023. http://dx.doi.org/10.1037/0000307-010.

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Damasceno, Benito. "Memory." In Research on Cognition Disorders, 33–45. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-57267-9_4.

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Lang, Peter J. "Anxiety and Memory." In Anxiety Disorders, 67–92. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-5254-9_5.

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Hutchins, Tiffany, Giacomo Vivanti, Natasa Mateljevic, Roger J. Jou, Frederick Shic, Lauren Cornew, Timothy P. L. Roberts, et al. "Memory." In Encyclopedia of Autism Spectrum Disorders, 1826–27. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-1698-3_350.

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Lickenbrock, Diane M. "Memory." In Encyclopedia of Autism Spectrum Disorders, 2854–55. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-319-91280-6_350.

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Stefanova, Elka, and Vladimir Kostic. "Isolated Memory Disorders." In International Neurology, 120–22. Oxford, UK: Wiley-Blackwell, 2010. http://dx.doi.org/10.1002/9781444317008.ch36.

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Evans, Jonathan J. "Disorders of Memory." In Clinical Neuropsychology, 141–63. Chichester, UK: John Wiley & Sons, Ltd, 2005. http://dx.doi.org/10.1002/0470013338.ch7.

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Groome, David. "Disorders of memory." In An Introduction to Cognitive Psychology, 177–205. 4th ed. London: Routledge, 2021. http://dx.doi.org/10.4324/9781351020862-7.

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Khan, Aman U. "Memory Assessment." In Clinical Disorders of Memory, 41–65. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-5128-3_2.

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Conference papers on the topic "Memory disorders"

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Staniloiu, Angelica, Sabine Borsutzky, and Hans Markowitsch. "Dissociative Memory Disorders and Immigration." In 9th Conference of the Australasian Society for Cognitive Science. Sydney: Macquarie Centre for Cognitive Science, 2010. http://dx.doi.org/10.5096/ascs200948.

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Vasina, Yuliya M. "The development of figurative memory in older preschool children with mental retardation through computer technology." In Специальное образование: методология, практика, исследования. Yaroslavl state pedagogical university named after К. D. Ushinsky, 2021. http://dx.doi.org/10.20323/978-5-00089-532-0-2021-79-83.

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The article discusses the use of computer technologies in the development of figurative memory in older preschool children with mental retardation. The features of memory development in children with developmental disorders are revealed. Criteria and diagnostic tools for identifying the level of development of figurative memory in children with this disorder are described. A series of computer training programs aimed at developing the basic properties of figurative memory are analyzed.
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GILBOA-SCHECHTMAN, EVA. "EXPLICIT AND IMPLICIT MEMORY BIASES IN ANXIETY DISORDERS." In Proceedings of the International School of Biocybernetics. WORLD SCIENTIFIC, 2002. http://dx.doi.org/10.1142/9789812776563_0041.

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Almeida, Eliane Borca, Geise Silva, Isabella Avolio, Camila Dias, Maíra Oliveira, Ricardo Nitrini, Sonia Brucki, and Eliane Miotto. "INVESTIGATION OF EPISODIC MEMORY DEFCITS IN PATIENTS WITH MILD COGNITIVE IMPAIRMENT (MCI)." In XIII Meeting of Researchers on Alzheimer's Disease and Related Disorders. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1980-5764.rpda043.

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Background: MCI can be classified as amnestic (aMCI) or non-amnestic (naMCI). Patients with aMCI are at increased risk of developing Alzheimer’s disease (AD). The clinical diagnosis encompasses episodic memory decline with preservation of activities of daily living, in addition to possible changes in other cognitive domains. Nevertheless, there is a lack of studies in the Brazilian population comparing the performance of aMCI on different episodic memory tests. Objectives: This study investigated episodic memory alterations in patients with aMCI and healthy controls (HC) through population-validated tests. Methods: We included 54 individuals, 36 aMCI and 18 HC. The neuropsychological protocol included estimated total IQ [vocabulary and matrix reasoning], Logical Memory (LM), Visual Reproduction (VR) and Rey Auditory Verbal Learning Test (RAVLT). Results: Significant differences were found between the groups in LM delayed recall (p=0.048); Visual Memory immediate recall (p=0.002); Visual Memory delayed recall (p=0.006); RAVLT immediate recall (p=0.19); RAVLT delayed recall (p=0.006) and RAVLT recognition (p=0.001). Conclusions: aMCI patients showed significant cognitive deficits in all episodic memory tests, except for the LM immediate recall. These findings corroborate the international literature and indicate the possibility of identifying differential cognitive alterations in the MCI.
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Macedo, Arthur Cassa, Luciano Inácio, Mariano Elisa De Paula França Resende, Antônio Lúcio Teixeira Júnior, Sarah Teixeira Camargos, Francisco Eduardo Costa Cardoso, Paulo Caramelli, and Leonardo Cruz De Souza. "EPISODIC MEMORY IMPAIRMENT IN PROGRESSIVE SUPRANUCLEAR PALSY (PSP): A NEUROIMAGING INVESTIGATION." In XIII Meeting of Researchers on Alzheimer's Disease and Related Disorders. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1980-5764.rpda016.

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Background: Progressive supranuclear palsy (PSP) has been classically considered a “subcortical dementia” with a frontal pattern of cognitive decline, but episodic memory dysfunction also occurs in most patients. However, it remains uncertain whether this is due to executive dysfunction or to the involvement of key brain areas responsible for memory processes. Objective: We aim to identify the specific brain regions underlying episodic memory impairment in PSP. Methods: In this cross-sectional study, we included 21 patients with PSP and 20 healthy controls matched for age, sex, and schooling. Participants underwent the Brief Cognitive Battery (BCB, including the Figures Test for episodic memory) and had brain MRI. Both standard exploratory voxel‐based morphometry and region of interest analyses were performed with FSL software. Results: Compared to controls, PSP patients performed worse (p < 0.001) on the BCB (delayed recall). Adjusting for both age and Frontal Assessment Battery scores, neuroimaging analyses of the correlation between delayed recall (5 minutes) and grey matter volumes yielded significant clusters on medial temporal structures, including the hippocampus, entorhinal cortex, and parahippocampal gyrus (FWE, p < 0.05). Conclusion: Our results suggest that atrophy of medial temporal structures may play a role in episodic memory impairment in PSP, indicating that amnesia in PSP is not due to executive dysfunction.
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Fernandes, Katarina, Henrique da Silva, Roberta Baradel, Raquel Fornari, Patrícia Vanzella, Katerina Lukasova, and Maria Carthery Goulart. "EFFICACY OF COGNITIVE TRAINING FOCUSED ON DIFFERENT MEMORY SUBSYSTEMS FOR COMMUNITY DWELLING ELDERLY INDIVIDUALS." In XIII Meeting of Researchers on Alzheimer's Disease and Related Disorders. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1980-5764.rpda105.

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Background: With the aging process, some memory subsystems as the episodic and working memory change. Cognitive training has been suggested for prevention of cognitive decline but previous studies have conflicting results. Objective: To verify the efficacy of two types of multidimensional cognitive training programs, one focusing on internal strategies to boost episodic memory (EMT) and another based on activities targeting speed processing and working memory (WMT). Methods: One hundred thirty six community dwelling older adults (aged 60 or over and living independently) took part in the study, 76 undertook EMT, 30 undertook WMT and 30 participated in a control program that focused on autobiographical memory (AMT). Training occurred during 8 weekly group sessions of 1,5h. All groups participated in lectures about cognition and aging during the training. They received activities to practice at home. A neuropsychological battery was applied before and after the interventions. Results: ANOVA of repeated measures pointed to significant higher scores in global cognition (ACER), their memory subtest, and in recall score of categorized noun pictures. The interaction showed that the episodic group improved more than the others in the recall of pictures (interaction between group and task p <0,01). Conclusion: The results demonstrated immediate gains after training in specific episodic memory trained skills.
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Malcorra, Bárbara, Natália Mota, Janaina Weissheimer, Lucas Schilling, Maximiliano Wilson, and Lilian Hübner. "REDUCED SPEECH CONNECTEDNESS IN ALZHEIMER’S DISEASE RELATES TO POORER SEMANTIC MEMORY." In XIII Meeting of Researchers on Alzheimer's Disease and Related Disorders. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1980-5764.rpda020.

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Background: Connected speech, an ecologically valid task, represents a valuable tool to support the detection of linguistic and cognitive impairment. Objective: First, to verify whether connected speech can differentiate oral narrative production between adults with Alzheimer’s disease (AD) and cognitively healthy older adults (HOA). Second, to verify whether specific speech graph attributes are associated with episodic, working, and semantic memory. Methods: 24 AD patients (age=72.75±8.13; education=4.25±3.30) and 48 HOA (age=69.85±7.06; education=5.08±3.10) produced an oral narrative task based on a sequence of pictures and performed episodic, working, and semantic memory tasks. Narratives were transcribed and each word was represented as a node and the temporal sequence was represented as directed edges. Graph connectedness was assessed by counting the number of edges (E), the number of nodes in the largest connected component (LCC) and the number of nodes in the largest strongly connected component (LSC). Results: AD patients produced less connected narratives than the HOA, with fewer edges (p=0.0035) and smaller LSC (p=0.0116). Semantic memory correlated moderately with LCC (Rho=0.59, p=0.002) exclusively in AD. Episodic memory correlated with LSC in HOA (Rho=0.47, p= <0.001). Conclusion: Word-graphs connectedness represents a practical tool to assess cognitive impairment in AD patients.
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Plácido, Talita, Pedro Rocha Filho, and Mário Silva Júnior. "PERFORMANCE IN COGNITIVE TESTS AND SUBJECTIVE PERCEPTION OF THE MEMORY IN INDIVIDUALS FROM RECIFE-PE." In XIII Meeting of Researchers on Alzheimer's Disease and Related Disorders. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1980-5764.rpda058.

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Introduction: Currently, there is a discussion about how subjective memory perception can predict performance in cognitive tests. Objective: To correlate the subjective perception of memory with performance in cognitive tests Mini-Mental State Examination (MMSE) and Montreal Cognitive Assessment (MoCA). Methods: Cross-sectional, descriptive study with 28 participants without cognitive complaints. People aged 40 years or more and at least four years of schooling were included. Participants were asked about their subjective perception of memory and then submitted to cognitive tests. Data were analyzed using SPSS software (v.23). Results: The population consisted of female individuals (89.3%), a mean age of 58.9 years (SD=±9.6), education of 11.9 years (SD=±4.4). As for the perception of memory, 53,5% of the individuals classified it as neutral, of which 7.1% had a score greater than or equal to 26 points in the MoCA, while 57.1% scored 24 or more in the MMSE. Also, 43,5% rated memory positively and, among them, 92.9% scored well on the MMSE, while only 28.7% had good performance on the MoCA. This self-assessment was correlated with MoCA performance (χ2=10.38; p=0.001). Conclusion: The subjective perception of memory was correlated with the performance of participants in the MoCA. Individuals with good perceptions had predominantly low performance on the tool.
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Jaeger, Antônio, Eduarda Carreira, Natália Gama, Paulo Caramelli, and Leonardo Souza. "CONTROLLED EPISODIC RETRIEVAL IN BEHAVIORAL VARIANT FRONTOTEMPORAL DEMENTIA AND ALZHEIMER DISEASE." In XIII Meeting of Researchers on Alzheimer's Disease and Related Disorders. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1980-5764.rpda012.

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Background: Recent studies showed that episodic memory is impaired in behavioral variant of Frontotemporal dementia (bvFTD), but in contrast to Alzheimer disease (AD) it is assumed to be caused primarily by deficits in executive control. Objective: Our goal was to probe this possibility by testing bvFTD and AD patients in a source memory task which manipulated executive control. Methods: We assessed 14 healthy controls (HC), 20 bvFTD patients, and 18 AD patients in a source memory task for spatial location in which objects were first seen at the left or right side of the screen, and at test in the center of the screen, when participants were asked to indicate in which side of the screen each object was studied. Importantly, at test, predictive arrow cues (66.7% valid/33.3% invalid) indicated the likely prior location of each object. Results: BvFTD and AD patients showed indistinguishable overall memory performances, although both showed significantly poorer performances than HC. Furthermore, although both HC and bvFTD participants had their memory judgments affected by cueing, showing poorer memory accuracy after invalid than after valid cues, AD patients showed equivalent performance for both cue types. Conclusion: The current findings support the notion that episodic memory is impaired in bvFTD, and suggests that such impairment can be as severe as in AD. The cause of this impairment, however, was not related to the executive dysfunctions manipulated in the current source memory task, but rather to further mechanisms in the bvFTD memory deficits.
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Resende, Elisa, Vivian Lara, Ana Luisa Santiago, Clarisse Friedlaender, Howard Rosen, Lea Grinberg, Lênio Lúcio Silva, et al. "HIPPOCAMPAL CONNECTIVITY IN ILLITERATE ADULTS FROM BRAZIL." In XIII Meeting of Researchers on Alzheimer's Disease and Related Disorders. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1980-5764.rpda036.

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Background: The role of hippocampal connectivity for good memory performance is well known in persons with high educational level. However, it is understudied the role of hippocampal connectivity in illiterate populations. Objectives: To determine whether the hippocampal connectivity correlate with episodic memory in illiterate adults. Methods: Thirty-nine illiterate adults underwent resting state functional MRI and an episodic memory test (Free and Cued Selective Reminding Test). We correlated the hippocampal connectivity at rest with the free recall scores. Analyzes were corrected for head motion and physiological BOLD signal. Results: Participants were most female (66%) and black (79%) and the mean age was 49 years-old (±13.9). The mean score on free recall was 27.2 (±10.7) out of 48 points. We found a significant correlation between both hippocampi and the posterior cingulate and ventral medial prefrontal cortex. However, we did not find an association between the hippocampal connectivity and the memory scores. Conclusions: The lack of association with memory scores might be associated with low brain reserve in this group of individuals.
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Reports on the topic "Memory disorders"

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Gresenz, Carole Roan, Jean M. Mitchell, Belicia Rodriguez, and Wilbert van der Klaauw. The Financial Consequences of Undiagnosed Memory Disorders. Federal Reserve Bank of New York, May 2024. http://dx.doi.org/10.59576/sr.1106.

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We examine the effect of undiagnosed memory disorders on credit outcomes using nationally representative credit reporting data merged with Medicare data. Years prior to eventual diagnosis, average credit scores begin to weaken and payment delinquency begins to increase, overall and for mortgage and credit card accounts specifically. Credit outcomes consistently deteriorate over the quarters leading up to diagnosis. The harmful financial effects of undiagnosed memory disorders exacerbate the already substantial financial pressure households face upon diagnosis of a memory disorder. Our findings substantiate the possible utility of credit reporting data for facilitating early identification of those at risk for memory disorders.
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Gupta, Shweta. The Science Behind Dreaming. Natur Library, October 2020. http://dx.doi.org/10.47496/nl.blog.11.

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The scientific study of dreams is oneirology, that seeks the relationship between dreaming and existing knowledge about the brain; understanding that how the brain functions during dreaming in accordance with memory shaping and mental disorders.
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Zhu, Qiqi, Jie Deng, Chong Xu, Meixi Yao, and Yu Zhu. Effects of physical activity on visuospatial working memory in healthy individuals: a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, August 2022. http://dx.doi.org/10.37766/inplasy2022.8.0053.

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Review question / Objective: P: Healthy individuals (including children, adolescents, adults, and seniors); I: Individuals who join various physical activities (including aerobic exercise, HIT, yoga, resistance training, Tai Chi, balance training, skill training, et al); C: Individuals who have no movement, do reading, or do same as normal activities; O: 1-Back Test, 2-Back Test, Trail Making Test-A, Trail Making Test-B, Digit Span Forward, Digit Span Backward; S: Randomized Controlled Trial (RCT). Condition being studied: Healthy individuals without any cognitive disorders.
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Luo, Minjing, Yilin Li, Yingqiao Wang, Jinghan Huang, Zhihan Liu, Yicheng Gao, Qianyun Chai, Yuting Feng, Jianping Liu, and Yutong Fei. The Fragility of Statistically Significant Findings from Depression Randomized Controlled Trials. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, April 2023. http://dx.doi.org/10.37766/inplasy2023.4.0086.

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Review question / Objective: The Fragility of Statistically Significant Findings from Depression Randomized Controlled Trials. Condition being studied: Depression is a mental disorder characterized by a range of symptoms, including loss of memory and sleep, decreased energy, feelings of guilt or low mood, disturbed appetite, poor concentration, and an increased risk of suicide. According to a systematic analysis of the Global Burden of Disease Study 2019, depression is recognized as the leading cause of disease burden for mental disorders, accounting for the largest proportion of disability-adjusted life years (DALYs) at 37.3%. The fragility index (FI), which is the minimum number of changes from events to non-events resulting in loss of statistical significance, has been suggested as a means to aid the interpretation of trial results, as the potential inadequacy about robustness of threshold P-value as a tool for reporting binary outcomes in clinical trials. In this systematic review, we want to calculate the FI of randomized controlled trials (RCTs) in depression.
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Carl, Merrigan, Shohat Dor, Sirote Chaviva, Lahini Yoav, Nisoli Cristiano, and Shokef Yair. Emergent Disorder and Mechanical Memory in Periodic Metamaterials. Office of Scientific and Technical Information (OSTI), July 2022. http://dx.doi.org/10.2172/2377305.

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The Centre for Attention Learning and Memory (CALM) Approach to Neurodevelopmental Research – MRC Cognition and Brain Sciences Unit University Of Cambridge. ACAMH, April 2021. http://dx.doi.org/10.13056/acamh.15509.

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Our thinking around neurodevelopmental disorders is undergoing a period of rapid change. The traditional approach, endorsed by classification systems such as the Diagnostic Statistical Manual, defines neurodevelopmental disorders such as autism and attention-deficit hyperactivity disorder (ADHD) as distinct categories.
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