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Books on the topic 'Membrane transport systems'

Author: Grafiati
Published: 4 June 2021
Last updated: 13 February 2022

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1

Andreoli, Thomas E., Joseph F. Hoffman, Darrell D. Fanestil, and Stanley G. Schultz, eds. Membrane Transport Processes in Organized Systems. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4684-5404-8.

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2

Sharma, Kal Renganathan. Transport phenomena in biomedical engineering: Artificial organ design and development and tissue engineering. New York: McGraw-Hill, 2010.

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3

Sharma, Kal Renganathan. Transport phenomena in biomedical engineering: Artificial organ design and development, and tissue engineering. New York: McGraw-Hill, 2010.

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4

Mitochondrial medicine. New York: Humana Press, 2015.

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5

NATO Advanced Research Workshop on Molecular Biology of Mitochondrial Transport Systems (1992 Il Ciocco, Italy). Molecular biology of mitochondrial transport systems. Berlin: Springer-Verlag, 1994.

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6

Ito, Fumio. Comparative Aspects of Mechanoreceptor Systems. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992.

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7

1935-, Morré D. James, ed. Cell-free analysis of membrane traffic: Proceedings of a Conference on Cell-Free Analysis of Membrane Traffic, held at the European Molecular Biology Laboratory, Heidelberg, Federal Republic of Germany, October 1-4, 1986. New York: Liss, 1988.

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8

1935-, Andreoli Thomas E., ed. Membrane transport processes in organized systems. New York: Plenum Medical Book Co., 1987.

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9

Developmental Biology of Membrane Transport Systems. Elsevier, 1991. http://dx.doi.org/10.1016/s0070-2161(08)x6037-x.

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10

Benos, Dale J. Developmental Biology of Membrane Transport Systems (Current Topics in Membranes). Academic Press, 1991.

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11

Benos, Dale J. Developmental Biology of Membrane Transport Systems (Current Topics in Membranes). Academic Press, 1991.

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12

Ion and Molecule Transport in Membrane Systems. MDPI, 2021. http://dx.doi.org/10.3390/books978-3-0365-1360-7.

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13

(Editor), Hans Leo Kornberg, and P. J. F. Henderson (Editor), eds. Microbial Membrane Transport Systems (Royal Society Discussion Volumes). Cambridge University Press, 1991.

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14

Gheorghe, Benga, ed. Molecular basis of selected transport systems. Boca Raton, Fla: CRC Press, 1985.

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15

Transport Phenomena in Biomedical Engineering: Principles and Practices. Taylor & Francis Group, 2012.

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16

Peterson, Donald R., Robert J. Fisher, Joseph D. Bronzino, and Robert A. Peattie. Transport Phenomena in Biomedical Engineering: Principles and Practices. Taylor & Francis Group, 2012.

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17

L, Kornberg H., and Henderson P. J. F, eds. Microbial membrane transport systems: Proceedings of a Royal Society discussion meeting held on 22 and 23 February 1989. London: The Society, 1990.

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18

Andre, Herchuelz, and New York Academy of Sciences, eds. Sodium-calcium exchange and the plasma membrane Ca2+-ATPase in cell function: Fifth international conference. Boston, Mass: Blackwell Pub. on behalf of the New York Academy of Sciences, 2007.

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19

Bailey, Matthew A. An overview of tubular function. Edited by Robert Unwin. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0020.

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This chapter provides an overview of transport processes, describing both the membrane proteins that effect transepithelial solute flux and the systems that allow integrated regulation of electrolyte transport. The emphasis is on the physiological mechanisms but links to human diseases are made in order to illuminate fundamental principles of control. The key transport proteins and encoding genes are listed. First, the major transport pathways and regulatory features for each nephron segment are described. The focus here is on the transepithelial flux of sodium, potassium, and water. In the second part, other important aspects of renal homeostasis, including urine concentration and acid–base balance, are summarized.
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20

Surface Chemistry of Biological Systems. Springer, 1995.

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21

Herchulez, Mordecai P. Blaustein, Jonathan Lytton, and Kenneth D. Philipson. Sodium-Calcium Exchange and the Plasma Membrane Ca2+-ATPase in Cell Function: Fifth International Conference (Annals of the New York Academy of Sciences). Blackwell Publishing Limited, 2007.

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22

Sherwood, Dennis, and Paul Dalby. The bioenergetics of living cells. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198782957.003.0024.

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Living systems create order, and appear to break the Second Law. This chapter explains, and resolves, this apparent paradox, drawing on the concept of coupled reactions (as introduced in Chapters 13 and 16), as mediated by ‘energy currencies’ such as ATP and NADH. The chapter then examines the key energy-capturing systems in biological systems – glycolysis and the citric acid cycle, and also photosynthesis. Topics covered include how energy is captured in the conversion of glucose to pyruvate, the mitochondrial membrane, respiration, electron transport, ATP synthase, chloroplasts and thylakoids, photosystems I and II, and the light-independent reactions of photosynthesis.
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23

1914-, Bender Max, ed. Interfacial phenomena in biological systems. New York: M. Dekker, 1991.

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24

E, Barrett Kim, and Donowitz Mark 1943-, eds. Gastrointestinal transport: Molecular physiology. San Diego: Academic Press, 2001.

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25

Blank, M. Surface Chemistry of Biological Systems: Proceedings Of The American Chemical Society Symposium On Surface Chemistry Of Biological Systems Held In New ... in Experimental Medicine and Biology). Springer, 2012.

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26

Signaling At The Cell Surface In The Circulatory And Ventilatory Systems. Springer, 2011.

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27

Thiriet, Marc. Signaling at the Cell Surface in the Circulatory and Ventilatory Systems. Springer, 2011.

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28

(Editor), Kim E. Barrett, Mark Donowitz (Editor), Douglas M. Fambrough (Series Editor), and Dale J. Benos (Series Editor), eds. Gastrointestinal Transport (Current Topics in Membranes, Volume 50) (Current Topics in Membranes). Academic Press, 2000.

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29

Benga, Gheorghe. Water Transport in Biological Membranes: From Cells to Multicellular Barrier Systems. CRC Press, 1989.

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30

E, Vance Dennis, and Vance Jean E, eds. Biochemistry of lipids, lipoproteins, and membranes. Amsterdam: Elsevier, 1991.

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31

Cell-free analysis of membrane traffic: Proceedings of a Conference on Cell-Free Analysis of Membrane Traffic, held at the European Molecular Biology Laboratory, ... in clinical and biological research). Liss, 1988.

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32

Abhishek, Abhishek, and Michael Doherty. Pathophysiology of calcium pyrophosphate deposition. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0049.

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Calcium pyrophosphate (CPP) dihydrate crystals form extracellularly. Their formation requires sufficient extracellular inorganic pyrophosphate (ePPi), calcium, and pro-nucleating factors. As inorganic pyrophosphate (PPi) cannot cross cell membranes passively due to its large size, ePPi results either from hydrolysis of extracellular ATP by the enzyme ectonucleotide pyrophosphatase/phosphodiesterase 1 (also known as plasma cell membrane glycoprotein 1) or from the transcellular transport of PPi by ANKH. ePPi is hydrolyzed to phosphate (Pi) by tissue non-specific alkaline phosphatase. The level of extracellular PPi and Pi is tightly regulated by several interlinked feedback mechanisms and growth factors. The relative concentration of Pi and PPi determines whether CPP or hydroxyapatite crystal is formed, with low Pi/PPi ratio resulting in CPP crystal formation, while a high Pi/PPi ratio promotes basic calcium phosphate crystal formation. CPP crystals are deposited in the cartilage matrix (preferentially in the middle layer) or in areas of chondroid metaplasia. Hypertrophic chondrocytes and specific cartilage matrix changes (e.g. high levels of dermatan sulfate and S-100 protein) are related to CPP crystal deposition and growth. CPP crystals cause inflammation by engaging with the NALP3 inflammasome, and with other components of the innate immune system, and is marked with a prolonged neutrophilic inflitrate. The pathogenesis of resolution of CPP crystal-induced inflammation is not well understood.
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33

Barros, Rodrigo José Saraiva de, Tereza Cristina de Brito Azevedo, Carla de Castro Sant’Anna, Marianne Rodrigues Fernandes, Leticia Martins Lamarão, and Rommel Mario Rodríguez Burbano. Grupos sanguíneos e anticorpos anti-eritrocitários de importância transfusional. Brazil Publishing, 2020. http://dx.doi.org/10.31012/978-65-5861-112-7.

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Immunohematology is an area dedicated to the study of the interactions of the immune system and blood cells in transfusion practice. Blood transfusion is a therapeutic technique that has been widely used since the 17th century. The transfusion medicine aims to repair the pathological needs of blood components in the living organism, be it red blood cells, plasma, platelets, clotting factors, among others. Despite being a therapeutic means, transfusion of blood components can be considered at risk because it is a biological material and due to the transfusion immunological reactions that can be caused during or after the moment of transfusion. In the surface structure of red blood cells, numerous molecules of a protein, glycoprotein or glycolipid nature are found, which are also called membrane antigens that make up structures and perform transport functions, as receptors, as adhesion, enzymatic and / or complement regulatory molecules. The formation of these antigens occurs by an approximate amount of 39 genes involved in their production, of which 282 different antigens are organized in more than 30 blood group systems. This antigenic diversity is a major cause of the formation of irregular anti-erythrocyte antibodies. Therefore, with the increase in blood transfusions in surgeries, transplants and clinical treatment of cancer and other chronic diseases, a significant increase in the occurrence of alloimmunizations in polytransfused patients began to be observed. Such biological phenomena motivated us to carry out this study and the antigenic diversity motivated us to elaborate this small compendium where we also describe the main blood groups.
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