Relevant bibliographies by topics / Melioidosis

Academic literature on the topic 'Melioidosis'

Author: Grafiati
Published: 4 June 2021
Last updated: 20 February 2023

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Journal articles on the topic "Melioidosis"

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Mohanty, Srujana, Saurav Sarkar, and Baijayantimala Mishra. "Melioidosis of the Head and Neck: A Case Series from Eastern India." Infectious Disease Reports 12, no. 3 (October 29, 2020): 36–45. http://dx.doi.org/10.3390/idr12030011.

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Melioidosis is an emerging entity in India. Though it is a potentially fatal disease, prognosis is excellent with early detection and appropriate management, especially of localized infections like abscesses of the head and neck area. We report nine cases of focal abscesses in the head and neck region due to Burkholderia pseudomallei, the causative agent of melioidosis, presenting to our hospital within a span of two-and-half years. Since melioidotic abscesses in the cervicofacial and head and neck region are likely to be confused with cold abscesses caused by Mycobacterium tuberculosis in a tuberculosis-endemic country like India, increased vigilance is necessary because of the widely divergent treatment modalities of the two disease entities.
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Mukhopadhyay, Chiranjay, VandanaKalwaje Eshwara, and VinodBhat Hattangadi. "Melioidosis." Journal of The Academy of Clinical Microbiologists 15, no. 1 (2013): 11. http://dx.doi.org/10.4103/0972-1282.116094.

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Chieng, Raymond. "Melioidosis." WikiJournal of Medicine 9, no. 1 (2022): 4. http://dx.doi.org/10.15347/wjm/2022.004.

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Anonymous, Anonymous. "Melioidosis." Journal of Special Operations Medicine 21, no. 4 (2021): 104. http://dx.doi.org/10.55460/wej5-a5ca.

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Borton, Dorothy. "Melioidosis." Nursing 52, no. 10 (October 2022): 29–34. http://dx.doi.org/10.1097/01.nurse.0000872460.50198.39.

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Annamalai, AnandK, and Kothandaramaraju Padmini. "Melioidosis." Indian Journal of Medical Research 149, no. 4 (2019): 561. http://dx.doi.org/10.4103/ijmr.ijmr_2018_17.

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Wiersinga, W. Joost, Bart J. Currie, and Sharon J. Peacock. "Melioidosis." New England Journal of Medicine 367, no. 11 (September 13, 2012): 1035–44. http://dx.doi.org/10.1056/nejmra1204699.

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Peacock, Sharon J. "Melioidosis." Current Opinion in Infectious Diseases 19, no. 5 (October 2006): 421–28. http://dx.doi.org/10.1097/01.qco.0000244046.31135.b3.

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Dance, David A. B. "Melioidosis." Current Opinion in Infectious Diseases 15, no. 2 (April 2002): 127–32. http://dx.doi.org/10.1097/00001432-200204000-00005.

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Haque, Nazia, Md Shafikul Bari, Md Moinul Hoque, Md Amirul Islam, Syada Monira Hoque, Santana Rani Sarkar, Md Rashedul Kabir, and Md Arifur Rahman. "Melioidosis." KYAMC Journal 4, no. 1 (April 21, 2017): 353–56. http://dx.doi.org/10.3329/kyamcj.v4i1.32262.

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Melioidosis is an important disease caused by a Gram-negative bacterium, Burkholderia pseudomallei. The true incidence of melioidosis is unknown for most countries of the world including Bangladesh. Due to its increasing incidence in many countries of the world it is an important issue now days. Due to variability of clinical features and limited availability of laboratory facilities the disease remains largely under-reported.Early and specific diagnosis is important to ensure a favourable outcome regarding this disease. In this paper history, transmission, sign symptoms, diagnosis and prevention of melioidosis are critically reviewed to know about something regarding this diseaseKYAMC Journal Vol. 4, No.-1, July 2013, Page 353-356
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Dissertations / Theses on the topic "Melioidosis"

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Brett, Paul James. "Immunotherapeutic strategies against melioidosis." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp02/NQ34659.pdf.

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Cheng, Allen Cheuk-Seng, and allencheng@ozemail com au. "MELIOIDOSIS: EPIDEMIOLOGY, PATHOPHYSIOLOGY AND MANAGEMENT." Flinders University. Medicine, 2005. http://catalogue.flinders.edu.au./local/adt/public/adt-SFU20051121.141305.

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In under a century, melioidosis, the infection due to Burkholderia pseudomallei, has emerged from Whitmore’s series of glanders-like infections amongst the morphia addicts in Burma to a major cause of mortality in northeastern Thailand and northern Australia. Also endemic in other parts of south-east Asia, melioidosis may have varied presentations ranging from severe, overwhelming infection to chronic, low grade disease. Observational evidence had suggested that granulocyte colony stimulating factor (G-CSF), a naturally occurring substance produced by the body in response to infection, may have been useful in reducing the high mortality associated with the more severe forms of this infection. Other observations linked the occurrence of this disease to various environmental factors, such as contamination of drinking water and the annual rainfall. This thesis explores and attempts to quantify these associations. There are three parts to this thesis. In the first part, I reviewed the epidemiology and management of patients with melioidosis. The use of G-CSF and meropenem was associated with a fall in mortality, although other factors may have at least partially contributed to this effect. In the second part, I progressed towards a clinical trial of G-CSF. There was no other evidence supporting the use of G-CSF in severe sepsis and ethical issues precluded a trial in Darwin. There was not evidence from laboratory models of G-CSF action in melioidosis to support the use of G-CSF in patients, although there remained some doubt regarding the applicability of such models to human disease. I examined clinical methods to identify patients at high risk of death from melioidosis. A simple scoring system based on clinical and laboratory parameters was developed and externally validated. However, clinical definitions of severe sepsis appeared to be better predictors of mortality. A clinical trial based on clinical definitions was commenced in Thailand. In the final part, I explored the question of whether different strains or B. pseudomallei or different environmental conditions caused different patterns of infection. There was no evidence that strain types of this bacterium determine the pattern or severity of disease, but weather conditions appeared to influence the distribution of disease in northern Australia.
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Limmathurotsakul, Direk. "Title: Determinants of recurrent melioidosis." Thesis, Open University, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.491474.

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Recurrent melioidosis represents relapse following failure to eradicate bacteria responsible for the primary infection or re-infection with a new strain. The first results chapter (chapter 3) evaluates the proportion of recurrent melioidosis due to relapse versus re-infection. Isolates from the same patient with an identical genotype were considered as relapse and those with a different genotype as re-infection. Three quarters of recurrent cases were due to relapse and one quarter were due to re-infection. There are two ways in which this approach could be confounded. First, 're-infection' could actually represent relapse if primary infection was caused by simultaneous infection with multiple B. pseudol1lallei strains, followed by chance selection of different strains from the two episodes for genotyping. The chance of this mistake occurring is based on the rate of polyclonal B. pseudol1lallei infection. Chapter 4 describes the rate of polyclonal infection in a large group of unselected patients in northeast Thailand, which was very low (2/133 cases, 1.5%). Second, 'relapse' could actually represent reinfection in the event that re-infection was caused by a B. pseudol1lallei strain that was by chance identical to the primary strain. The probability of this happening is based on the degree of genetic diversity of B. pseudomallci in the environment. Chapter 5 demonstrates that the population of B. pscudol1lallei in even a small sampling site is extremely diverse. Thus, it is unlikely that the assessment of the causes of recurrent melioidosis contained significant errors due to polyclonal infection or low genetic diversity of the organism. Chapter 6 examines specific risk factors of relapse and reinfection. Duration and choices of antibiotics used for the primary episode were major determinants of relapse. Chapter 7 compares the clinical manifestations of relapse and re-infection and develops a simple scoring index to predict relapse or re-infection in patients presenting with recurrent melioidosis.
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Tsai, Chi-Chun Wirongrong Chierakul. "Rheumatological manifestations in melioidosis patients /." Abstract, 2006. http://mulinet3.li.mahidol.ac.th/thesis/2549/cd387/4838796.pdf.

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Gerstenmaier, Jan Frank Wirongrong Chierakul. "Pulmonary manifestations in melioidosis patients /." Abstract, 2006. http://mulinet3.li.mahidol.ac.th/thesis/2549/cd387/4838790.pdf.

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Thematic Paper (M.Sc. (Clinical Tropical Medicine))--Mahidol University, 2006.
LICL has E-Thesis 0011 ; please contact computer services. LIRV has E-Thesis 0011 ; please contact circulation services.
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Leung, Beatrice Pui See. "Development of a DNA vaccine against melioidosis." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape2/PQDD_0018/MQ55225.pdf.

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Kinoshita, Reimi E. "Epidemiology of melioidosis in an oceanarium: a clinical, environmental and molecular study." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2003. http://hub.hku.hk/bib/B29757745.

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Virginio, Camila Gomes. "Aspectos fenotÃÂpicos de amostras de Burkhoderia pseudomallei isoladas de uma microepidemia do municÃÂpio de TejuÃÂuoca-Ce." Universidade Federal do CearÃ, 2005. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=768.

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CoordenaÃÃo de AperfeiÃoamento de Pessoal de NÃvel Superior
A Burkholderia pseudomallei à um bacilo Gram-negativo nÃo-fermentador, saprÃfita ambiental capaz de causar melioidose em homens e animais. A doenÃa à considerada endÃmica em diversos paÃses, entre os quais destacam-se TailÃndia e AustrÃlia. Em fevereiro de 2003, ocorreu no Brasil, o primeiro isolamento e identificaÃÃo da bactÃria em quatro crianÃas da localidade de TejuÃuoca, CearÃ. Este trabalho consiste na caracterizaÃÃo fenotÃpica de 3 amostras de B. pseudomallei originÃrias dos pacientes do municÃpio de TejuÃuoca, com o propÃsito de comparar os dados obtidos de tais amostras com os dados da literatura. Foram avaliados: morfologia das colÃnias em diferentes meios de cultivo, assimilaÃÃo de L-arabinose, testes bioquÃmicos manuais e em sistema semi-automatizado API 20NE, teste de sensibilidade antibacteriano e diagnÃstico por PCR, a partir de cultivos bacterianos. Nos resultados obtidos, foi observado o padrÃo morfolÃgico caracterÃstico de B. pseudomallei em Ãgar sangue, chocolate, Ashdown, Mac Conkey, CLED e tripticase soja. As amostras 1 e 3 foram classificadas como mucÃides e a amostra 2 como rugosa. As trÃs amostras apresentaram os padrÃes fenotÃpicos caracterÃsticos de B. pseudomallei tanto nos testes bioquÃmicos manuais: motilidade, crescimento à 42ÂC, oxidase positiva e resistÃncia à polimixina B, como no Kit DiagnÃstico API 20NE. Neste Ãltimo, houve diferenÃa na esculina entre as amostras, o que nÃo interferiu no resultado final de identificaÃÃo, quando a leitura foi realizada com 48h. Todas as trÃs amostras foram incapazes de assimilar o carboidrato L-arabinose, quando testadas em meio sais mÃnimo e API 20NE, caracterÃstica de amostras virulentas de B. pseudomallei e utilizada tambÃm para diferenciar esta espÃcie da B. thailandensis, que à capaz de assimilar este carboidrato. O padrÃo de sensibilidade resultante do TSA em disco difusÃo apresentado pelas trÃs amostras foi o caracterÃstico da espÃcie B. pseudomallei. Os isolados foram resistentes à gentamicina, cefalotina, ciprofloxacina (1 amostra apresentou resistÃncia intermediÃria) e sulfa-trimetoprim; 2 amostras apresentaram sensibilidade intermediÃria à ceftriaxona. Todas as trÃs amostras foram sensÃveis à piperacilina-tazobactam, ticarcilina-Ãcido clavulÃnico, ceftazidima, imipenem, tetraciclina e cloranfenicol. Com o protocolo fenol-clorofÃrmio modificado de extraÃÃo de DNA, a PCR apresentou banda de 718 pb, o que confirmou o diagnÃstico da bactÃria tambÃm por mÃtodo molecular. O estudo confirma a presenÃa da B. pseudomallei em territÃrio brasileiro, com fenotipagem semelhante à descrita na literatura internacional.
Burkholderia pseudomallei is a Gram-negative non-fermentative bacilli, environmental saprophyte able to cause melioidosis on men and animals. The disease is considered endemic in several countries, especially in Thailand and Australia. The bacteria was isolated and identified for the first time in Brazil, february 2003, from four children that lived in a place called TejuÃuoca, CearÃ. This work consists of the phenotypic characterization of 3 strains of B. pseudomallei isolated from the patients from TejuÃuoca. The main aim of this study is to compare the data from these samples with the ones from the literature. It was assessed: the colonies morphology in different culture mediums, assimilation of L-arabinose, manual and semi-automatized biochemical tests in API 20NE, antibacterial sensitivity test and diagnosis by PCR, from bacterial cultures. In the obtained results, it was observed the morphological pattern of B. pseudomallei in blood agar, chocolate, Ashdown, Mac Conkey, CLED and trypticase soy agar. The strains 1 and 3 were classified as mucoid and the strain 2 as wrinkled. The three strains had shown the usual phenotypic patterns of B. pseudomallei as much in biochemical manual tests: motility, growth at 42ÂC, positive oxidase and resistance to polimixin B, as in the API 20NE Diagnosis Kit. In this last one, there was difference in the esculin test among the strains, when the reading was carried out with 48 hours, which did not change on the final identification. All of the three strains were unable to metabolize the carbohydrate L-arabinose, when tested in minimum medium salts and API 20NE, which is a characteristic of virulent strains of B. pseudomallei and is also used to differ this species from B. thailandensis, that is able to use the carbohydrate. The three isolates had shown a poor sensitivity pattern from disk diffusion on TSA, which were resistant to gentamicin, cefalotin, ciprofloxacin (one strain presented intermediate resistance) and sulfa-trimethoprim; two strains presented intermediate sensitivity to ceftriaxone. All of them were sensitive to piperacilin-tazobactam, ticarcilin-clavulanate, ceftazidime, imipenem, tetracycline and chloramphenicol. A modified extraction protocol based on phenol-chloroform was used to obtain DNA and later to test it by PCR, which had shown a 718 bp product, what also confirmed the diagnosis of the organisms by molecular method. The study confirms the presence of B. pseudomallei in Brazil with similar phenotype to that described in the international literature.
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Sangiambut, Sutha. "Cloning, expression and characterisation of potential therapeutic targets of Burkholderia pseudomallei." Thesis, Imperial College London, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.271682.

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Davies, Clare. "Molecular characterisation of Burkholderia pseudomallei." Thesis, University of Plymouth, 2001. http://hdl.handle.net/10026.1/2281.

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A programme of research was carried out to attempt the molecular characterisation of the human and animal pathogen, Burkholderia pseudomallei, the causative agent of melioidosis and the newly described avirulent species, B.thailandensis for comparative purposes. Melioidosis is still little understood, and so the clinical approach to the prevention and control of melioidosis must ultimately rest upon the basic understanding of the causative organism, particularly the pathogenic properties of B.pseudomallei. A range of B.pseudomallei and B.thailandensis isolates were cultured and the extracellular products were isolated and concentrated and an initial study conducted to identify potential target molecules for cloning. Those isolates tested were shown to have somewhat differing ECP profiles when analysed with SDS-PAGE and antigenic profiles when subject to irnmunoblotting using convalescent human serum although isolates within and between species shared a number of common bands. The ECPs were also tested for a range of activities and it was established that both species had proteolytic and phospholipase activities neither had a haemolytic activity and only isolates of B.pseudomallei had a hexosaminidase activity a putative pathogenicity determinant. Genomic DNA of B.pseudomallei was used to construct genomic libraries in a range of E. coli host vector systems. A λGTII genomic library was screened with antisera for the presence of B.pseudomallei antigens and a number of natural and synthetic substrates for the presence of haemolytic and proteolytic components. Screening yielded one stable immunopositive clone with a novel positive reaction in the form of a "halo" of reaction around the plaque. The 5 kbp cloned fragment was subcloned into a plasmid vector, and the resulting recombinant molecule, pBPGT2 was DNA sequenced and found to contain a putative pilin gene. Attempts were made to determine the size of the recombinant antigen and to further express the pilin gene product all of which were unsuccessful. A southern blot procedure confirmed the fidelity of the cloning procedure proving that the fragment was from the host organism, B.pseudomallei. A further southern blot procedure tested for the presence of the pilin sequence in a range of B.pseudomallei and B.thailandensis isolates proving the presence of the gene in only isolates of B.pseudomallei. PCR primers were designed to amplify the DNA encoding the active site of the ADP-ribosylating toxin (ET A) of Pseudomonas aeruginosa and a PCR reaction was carried out on a number of B.pseudomallei and B.thailandensis isolates. The reaction yielded a 500 bp product in only B.pseudomallei isolates and DNA sequencing of the product revealed no obvious homology to ETA of P.aeruginosa but was used as a probe to isolate a larger fragment of DNA which was found to encode a number of interesting putative genes. These included one with homology to a porin similar to that of the pathogen Neisseria gonorrhoea, with a role in virulence. During attempts to digest the genomic DNA of B.pseudomallei isolate 4845 with the restriction enzyme Sau3A two 12 kbp bands of DNA were resistant to the endonuclease activity. Attempts were made to clone these bands into a range of plasmid vectors with two clones containing deleted products. DNA sequencing proved inadequate with only a small amount of sequence information obtained. However, towards the final stages of the research project sequence information from the B.pseudomallei genome sequencing project facilitated the recognition of a 38 kbp fragment containing the sequence information from one of the clones, which encodes an alkaline protease and a putative haemagglutinin and is postulated to be a Pathogenicity Island encoding secreted virulence factors. The sequencing project also facilitated the isolation of two putative hexosaminidase genes postulated to be responsible for the activities observed when testing the B.pseudomallei isolates concentrated ECPs. Future studies for the putative genes identified and other components of B.pseudomallei are discussed.
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Books on the topic "Melioidosis"

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Parker, James N., and Philip M. Parker. The official patient's sourcebook on melioidosis. Edited by Icon Group International Inc and NetLibrary Inc. San Diego, Calif: Icon Health Publications, 2002.

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Jones, Michael. Application of enzyme linked immunosorbent assays for the diagnosis of melioidosis. Salford: University of Salford, 1991.

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Human Melioidosis. Singapore University Press, 2002.

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Berger, Stephen, and Inc Gideon Informatics. Melioidosis and Glanders: Global Status. Gideon Informatics, Incorporated, 2019.

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Berger, Stephen, and Inc Gideon Informatics. Melioidosis and Glanders: Global Status. Gideon Informatics, Incorporated, 2022.

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Berger, Stephen, and Inc Gideon Informatics. Melioidosis and Glanders: Global Status. Gideon Informatics, Incorporated, 2021.

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Global Burden and Challenges of Melioidosis. MDPI, 2019. http://dx.doi.org/10.3390/books978-3-03897-743-8.

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Torres, Alfredo G., ed. Recent progress in Melioidosis and Glanders. Frontiers Media SA, 2013. http://dx.doi.org/10.3389/978-2-88919-081-2.

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Mohapatra, Prasanta Raghab. Clinical Melioidosis: A Practical Guide to Diagnosis and Management. Taylor & Francis Group, 2023.

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Mohapatra, Prasanta Raghab. Clinical Melioidosis: A Practical Guide to Diagnosis and Management. Taylor & Francis Group, 2023.

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Book chapters on the topic "Melioidosis"

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Tolaney, Pooja, and Larry I. Lutwick. "Melioidosis." In Beyond Anthrax, 145–58. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-326-4_7.

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Eltorai, Ibrahim M. "Melioidosis." In Rare Diseases and Syndromes of the Spinal Cord, 265–67. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-45147-3_81.

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Enenkel, Sabine, and Wolfgang Stille. "Melioidosis." In Antibiotics in the Tropics, 282–83. Berlin, Heidelberg: Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-73276-8_31.

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Sprague, Lisa D., and Mandy C. Elschner. "Burkholderia pseudomallei: Melioidosis." In BSL3 and BSL4 Agents, 303–5. Weinheim, Germany: Wiley-VCH Verlag GmbH & Co. KGaA, 2012. http://dx.doi.org/10.1002/9783527645114.ch18.

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Sprague, Lisa D., and Mandy C. Elschner. "Burkholderia pseudomallei: Melioidosis." In BSL3 and BSL4 Agents, 47–56. Weinheim, Germany: Wiley-VCH Verlag GmbH & Co. KGaA, 2012. http://dx.doi.org/10.1002/9783527645114.ch4.

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Buisson, Yves, Sayaphet Rattanavong, Valy Keoluangkhot, Khamsing Vongphayloth, Loungnilanh Manivanh, Rattanaphone Phetsouvanh, Alain Pierret, et al. "Melioidosis in Laos." In Socio-Ecological Dimensions of Infectious Diseases in Southeast Asia, 89–104. Singapore: Springer Singapore, 2015. http://dx.doi.org/10.1007/978-981-287-527-3_7.

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Vedros, Neylan A. "Melioidosis and Glanders." In Laboratory Diagnosis of Infectious Diseases, 366–74. New York, NY: Springer New York, 1988. http://dx.doi.org/10.1007/978-1-4612-3898-0_38.

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Simpson, Andrew J. H. "Management of Melioidosis." In Management of Multiple Drug-Resistant Infections, 209–22. Totowa, NJ: Humana Press, 2004. http://dx.doi.org/10.1007/978-1-59259-738-3_12.

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Hussein, Mansour F. "Melioidosis (Whitmore Disease; Burkholderia pseudomallei Infection)." In Infectious Diseases of Dromedary Camels, 159–61. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-79389-0_25.

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Warawa, Jonathan M., and Frank C. Gherardini. "Modeling of Acute Respiratory Melioidosis and Glanders." In National Institute of Allergy and Infectious Diseases, NIH, 117–20. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60761-512-5_13.

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Conference papers on the topic "Melioidosis"

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Domthong, Pornanan, Uthumporn Domthong, and Surasak Faisatjathum. "Scoring system predicting mortality in melioidosis." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa2910.

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Velasco, Alvin Brian C., Christine Agatha Untalan, and Julie Christie Visperas. "Melioidosis Presenting As Severe Community Acquired Pneumonia." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a4711.

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Cortes Lopez, R., and P. H. Eshaghian. "A Case of Melioidosis in a Patient with Cystic Fibrosis." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6466.

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West, T. E., W. Chierakul, D. Limmathurotsakul, V. Wuthiekanun, N. Chantratita, Thomas R. Hawn, Sharon J. Peacock, and Shawn J. Skerrett. "Toll-Like Receptor Pathway Gene Variants And Susceptibility To Melioidosis." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a1803.

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Leong, Tracy, Parm Naidoo, Beena Kumar, Daniel Stefanski, Ian Woolley, and Michael Farmer. "Acute Mediastinal Melioidosis: Diagnosis By Endobronchial Ultrasound-Guided Transbronchial Needle Aspiration." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a4568.

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Nguyen, Ho Lam, Huyen Duong-Thanh, Hoa Le, and Nhat-Thinh Le-Phu. "Clinical characteristic differences between pulmonary melioidosis and pulmonary tuberculosis with cavitary lesion." In ERS International Congress 2021 abstracts. European Respiratory Society, 2021. http://dx.doi.org/10.1183/13993003.congress-2021.pa1890.

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Coston, T. D., R. Phunpang, A. Yarasai, A. Dulsuk, T. Yimthin, E. Thiansukhon, S. Chaisuksant, et al. "Morbidity and Mortality Following Melioidosis Pneumonia: Results from a Multicenter Cohort Study." In American Thoracic Society 2022 International Conference, May 13-18, 2022 - San Francisco, CA. American Thoracic Society, 2022. http://dx.doi.org/10.1164/ajrccm-conference.2022.205.1_meetingabstracts.a3108.

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Onofrey, L., R. Phunpang, A. Yarasai, A. Dulsuk, T. Yimthin, E. Thiansukhon, S. Chaisuksant, et al. "ICU Utilization and Outcomes in Patients with Critical Illness Due to Melioidosis." In American Thoracic Society 2022 International Conference, May 13-18, 2022 - San Francisco, CA. American Thoracic Society, 2022. http://dx.doi.org/10.1164/ajrccm-conference.2022.205.1_meetingabstracts.a5153.

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Kusumawati, R. Lia, Mirzan Hasibuan, Afrinayanti W. Siregar, and Tryna Tania. "Role Selective and Nonselective Media for Isolation of Burkholderia Species from Patients with Suspected Melioidosis." In International Conference of Science, Technology, Engineering, Environmental and Ramification Researches. SCITEPRESS - Science and Technology Publications, 2018. http://dx.doi.org/10.5220/0010079305820585.

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Rufino Leão, Bruna, Bruna Aurora Nunes Cavalcante Castro, Maria das Graças Barbosa Sousa, Kharen Assunção Bezerra Galdino, and Yarla Catarina Antão de Alencar. "OSTEOMYELITIS IN SACROILIAC AND SEPSIS BY Burkholderia pseudomallei: MELIOIDOSIS IN PATIENT WITH SYSTEMIC LUPUS ERYTHEMATOSUS – CASE REPORT." In SBR 2021 Congresso Brasileiro de Reumatologia. Sociedade Brasileira de Reumatologia, 2021. http://dx.doi.org/10.47660/cbr.2021.1952.

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