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Relevant bibliographies by topics / Meiosis; Meiotic

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Academic literature on the topic 'Meiosis; Meiotic'

Author: Grafiati

Published: 4 June 2021

Last updated: 2 February 2022

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Journal articles on the topic "Meiosis; Meiotic"

1

Hasenkampf, C. A., A. A. Taylor, N. U. Siddiqui, and C. D. Riggs. "meiotin-1 gene expression in normal anthers and in anthers exhibiting prematurely condensed chromosomes." Genome 43, no. 4 (August 1, 2000): 604–12. http://dx.doi.org/10.1139/g00-021.

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We have cloned and sequenced the promoter of a meiotin-1 gene, and have determined the precise temporal and spatial pattern of meiotin-1 gene expression. The expression of the meiotin-1 gene is controlled in two increments. The meiotin-1 gene is not expressed in any of the vegetative tissues examined. Early in microsporogenesis, low levels of meiotin-1 RNA can be detected. At the onset of meiosis, there is a dramatic increase in meiotin-1 RNA levels in both tapetal and meiotic cells. However, while meiotin-1 RNA is observed in both the nucleus and cytoplasm of meiotic cells, it is found only in the nucleus of the tapetal cells. We have also examined the expression of the meiotin-1 gene in aberrant meiotic nuclei that prematurely condense their chromosomes; these nuclei have reduced levels of the meiotin-1 protein. The aberrant nuclei have only the basal level of meiotin-1 RNA; they do not exhibit the transcriptional induction seen for normal cells at the onset of meiosis. Implications for the function of meiotin-1 in regulating chromatin condensation, and in coordinating meiotic and tapetal cell activities are discussed.Key words: anther development, chromatin, meiosis, meiotin-1, promoter.

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Goldway, M., A. Sherman, D. Zenvirth, T. Arbel, and G. Simchen. "A short chromosomal region with major roles in yeast chromosome III meiotic disjunction, recombination and double strand breaks." Genetics 133, no. 2 (February 1, 1993): 159–69. http://dx.doi.org/10.1093/genetics/133.2.159.

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Abstract A multicopy plasmid was isolated from a yeast genomic library, whose presence resulted in a twofold increase in meiotic nondisjunction of chromosome III. The plasmid contains a 7.5-kb insert from the middle of the right arm of chromosome III, including the gene THR4. Using chromosomal fragments derived from chromosome III, we determined that the cloned region caused a significant, specific, cis-acting increase in chromosome III nondisjunction in the first meiotic division. The plasmid containing this segment exhibited high spontaneous meiotic integration into chromosome III (in 2.4% of the normal meiotic divisions) and a sixfold increase (15.5%) in integration in nondisjunctant meioses. Genetic analysis of the cloned region revealed that it contains a "hot spot" for meiotic recombination. In DNA of rad50S mutant cells, a strong meiosis-induced double strand break (DSB) signal was detected in this region. We discuss the possible relationships between meiosis-induced DSBs, recombination and chromosome disjunction, and propose that recombinational hot spots may be "pairing sites" for homologous chromosomes in meiosis.

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Tsuchiya, Dai, Claire Gonzalez, and Soni Lacefield. "The spindle checkpoint protein Mad2 regulates APC/C activity during prometaphase and metaphase of meiosis I in Saccharomyces cerevisiae." Molecular Biology of the Cell 22, no. 16 (August 15, 2011): 2848–61. http://dx.doi.org/10.1091/mbc.e11-04-0378.

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In many eukaryotes, disruption of the spindle checkpoint protein Mad2 results in an increase in meiosis I nondisjunction, suggesting that Mad2 has a conserved role in ensuring faithful chromosome segregation in meiosis. To characterize the meiotic function of Mad2, we analyzed individual budding yeast cells undergoing meiosis. We find that Mad2 sets the duration of meiosis I by regulating the activity of APCCdc20. In the absence of Mad2, most cells undergo both meiotic divisions, but securin, a substrate of the APC/C, is degraded prematurely, and prometaphase I/metaphase I is accelerated. Some mad2Δ cells have a misregulation of meiotic cell cycle events and undergo a single aberrant division in which sister chromatids separate. In these cells, both APCCdc20 and APCAma1 are prematurely active, and meiosis I and meiosis II events occur in a single meiotic division. We show that Mad2 indirectly regulates APCAma1 activity by decreasing APCCdc20 activity. We propose that Mad2 is an important meiotic cell cycle regulator that ensures the timely degradation of APC/C substrates and the proper orchestration of the meiotic divisions.

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Cooper, Katrina F., and Randy Strich. "Saccharomyces cerevisiae C-Type Cyclin Ume3p/Srb11p Is Required for Efficient Induction and Execution of Meiotic Development." Eukaryotic Cell 1, no. 1 (February 2002): 66–74. http://dx.doi.org/10.1128/ec.01.1.66-74.2002.

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ABSTRACT The yeast C-type cyclin Ume3p/Srb11p and its cyclin-dependent kinase partner Ume5p/Srb10p repress the transcription of several genes required for meiotic recombination or meiosis I nuclear division. To relieve this repression, Srb11p is destroyed early in meiosis, prior to the first meiotic division. This report identifies two roles for Srb11p in regulating meiotic development. First, SRB11 is required for the normal exit from the mitotic cell cycle prior to meiotic induction. Specifically, mutants lacking SRB11 (srb11Δ) uncouple bud growth from chromosome segregation, producing small buds with nuclei. The bud growth defect is most likely due to the failure of srb11Δ mutants to reestablish polarized actin fibers at the bud tip following exposure to sporulation medium. Second, Srb11p is required for the efficient execution of meiosis I. srb11Δ mutants either exhibited a delay in performing meiosis I and meiosis II or skipped meiosis I entirely. This meiotic defect is not due to the activation of the recombination or spindle assembly checkpoint pathways. However, the expression of several meiotic genes is delayed and reduced in the mutant strains. These results suggest a positive role for Srb10-Srb11p in regulating the transcription program. This model is supported by the finding that overexpression of the meiotic inducer IME2 partially restored the ability of srb11 mutants to perform meiosis I. In conclusion, these findings indicate that Srb11p is required for both entry into and execution of the meiotic program, thus describing multiple roles for a C-type cyclin in the regulation of a developmental pathway.

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Page, A. W., and T. L. Orr-Weaver. "The Drosophila genes grauzone and cortex are necessary for proper female meiosis." Journal of Cell Science 109, no. 7 (July 1, 1996): 1707–15. http://dx.doi.org/10.1242/jcs.109.7.1707.

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In Drosophila, normal female meiosis arrests at metaphase I. After meiotic arrest is released by egg activation, the two meiotic divisions are rapidly completed, even in unfertilized eggs. Since little is known about the regulation of the meiotic cell cycle after the meiotic arrest, we screened for mutants that arrest in meiosis. Here we describe the phenotype of eggs laid by sterile mothers mutant for either grauzone or cortex. These eggs arrest in metaphase of meiosis II, and although they can enter into an aberrant anaphase II, they never exit meiosis. Prolonged sister-chromatid cohesion is not the cause of this arrest, since a premature release of sister cohesion does not rescue the meiotic arrest of cortex eggs. Aberrant chromosome segregation at meiosis I was the earliest observable defect, suggesting that grauzone and cortex are first required immediately after egg activation. The cortical microtubules are also defective, remaining in a pre-activated state in activated mutant eggs. The mutations had no observable effect on either male meiosis or mitosis. We believe these genes will provide insight into the developmental regulation of meiosis in a genetically tractable organism.

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Nelms, Brad, and Virginia Walbot. "Defining the developmental program leading to meiosis in maize." Science 364, no. 6435 (April 4, 2019): 52–56. http://dx.doi.org/10.1126/science.aav6428.

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In multicellular organisms, the entry into meiosis is a complex process characterized by increasing meiotic specialization. Using single-cell RNA sequencing, we reconstructed the developmental program into maize male meiosis. A smooth continuum of expression stages before meiosis was followed by a two-step transcriptome reorganization in leptotene, during which 26.7% of transcripts changed in abundance by twofold or more. Analysis of cell-cycle gene expression indicated that nearly all pregerminal cells proliferate, eliminating a stem-cell model to generate meiotic cells. Mutants defective in somatic differentiation or meiotic commitment expressed transcripts normally present in early meiosis after a delay; thus, the germinal transcriptional program is cell autonomous and can proceed despite meiotic failure.

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Nag, D. K., M. P. Koonce, and J. Axelrod. "SSP1, a gene necessary for proper completion of meiotic divisions and spore formation in Saccharomyces cerevisiae." Molecular and Cellular Biology 17, no. 12 (December 1997): 7029–39. http://dx.doi.org/10.1128/mcb.17.12.7029.

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During meiosis, a diploid cell undergoes two rounds of nuclear division following one round of DNA replication to produce four haploid gametes. In yeast, haploid meiotic products are packaged into spores. To gain new insights into meiotic development and spore formation, we followed differential expression of genes in meiotic versus vegetatively growing cells in the yeast Saccharomyces cerevisiae. Our results indicate that there are at least five different classes of transcripts representing genes expressed at different stages of the sporulation program. Here we describe one of these differentially expressed genes, SSP1, which plays an essential role in meiosis and spore formation. SSP1 is expressed midway through meiosis, and homozygous ssp1 diploid cells fail to sporulate. In the ssp1 mutant, meiotic recombination is normal but viability declines rapidly. Both meiotic divisions occur at the normal time; however, the fraction of cells completing meiosis is significantly reduced, and nuclei become fragmented soon after meiosis II. The ssp1 defect does not appear to be related to a microtubule-cytoskeletal-dependent event and is independent of two rounds of chromosome segregation. The data suggest that Ssp1 is likely to function in a pathway that controls meiotic nuclear divisions and coordinates meiosis and spore formation.

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Li, Qianyan, Sara Hariri, and JoAnne Engebrecht. "Meiotic Double-Strand Break Processing and Crossover Patterning Are Regulated in a Sex-Specific Manner by BRCA1–BARD1 in Caenorhabditis elegans." Genetics 216, no. 2 (August 12, 2020): 359–79. http://dx.doi.org/10.1534/genetics.120.303292.

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Meiosis is regulated in a sex-specific manner to produce two distinct gametes, sperm and oocytes, for sexual reproduction. To determine how meiotic recombination is regulated in spermatogenesis, we analyzed the meiotic phenotypes of mutants in the tumor suppressor E3 ubiquitin ligase BRC-1-BRD-1 complex in Caenorhabditis elegans male meiosis. Unlike in mammals, this complex is not required for meiotic sex chromosome inactivation, the process whereby hemizygous sex chromosomes are transcriptionally silenced. Interestingly, brc-1 and brd-1 mutants show meiotic recombination phenotypes that are largely opposing to those previously reported for female meiosis. Fewer meiotic recombination intermediates marked by the recombinase RAD-51 were observed in brc-1 and brd-1 mutants, and the reduction in RAD-51 foci could be suppressed by mutation of nonhomologous-end-joining proteins. Analysis of GFP::RPA-1 revealed fewer foci in the brc-1brd-1 mutant and concentration of BRC-1-BRD-1 to sites of meiotic recombination was dependent on DNA end resection, suggesting that the complex regulates the processing of meiotic double-strand breaks to promote repair by homologous recombination. Further, BRC-1-BRD-1 is important to promote progeny viability when male meiosis is perturbed by mutations that block the pairing and synapsis of different chromosome pairs, although the complex is not required to stabilize the RAD-51 filament as in female meiosis under the same conditions. Analyses of crossover designation and formation revealed that BRC-1-BRD-1 inhibits supernumerary COs when meiosis is perturbed. Together, our findings suggest that BRC-1-BRD-1 regulates different aspects of meiotic recombination in male and female meiosis.

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Loidl, Josef. "Tetrahymena meiosis: Simple yet ingenious." PLOS Genetics 17, no. 7 (July 15, 2021): e1009627. http://dx.doi.org/10.1371/journal.pgen.1009627.

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The presence of meiosis, which is a conserved component of sexual reproduction, across organisms from all eukaryotic kingdoms, strongly argues that sex is a primordial feature of eukaryotes. However, extant meiotic structures and processes can vary considerably between organisms. The ciliated protist Tetrahymena thermophila, which diverged from animals, plants, and fungi early in evolution, provides one example of a rather unconventional meiosis. Tetrahymena has a simpler meiosis compared with most other organisms: It lacks both a synaptonemal complex (SC) and specialized meiotic machinery for chromosome cohesion and has a reduced capacity to regulate meiotic recombination. Despite this, it also features several unique mechanisms, including elongation of the nucleus to twice the cell length to promote homologous pairing and prevent recombination between sister chromatids. Comparison of the meiotic programs of Tetrahymena and higher multicellular organisms may reveal how extant meiosis evolved from proto-meiosis.

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Mukherjee, Kaustav, Bruce Futcher, and Janet Leatherwood. "mmi1 and rep2 mRNAs are novel RNA targets of the Mei2 RNA-binding protein during early meiosis in Schizosaccharomyces pombe." Open Biology 8, no. 9 (September 2018): 180110. http://dx.doi.org/10.1098/rsob.180110.

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The RNA-binding protein Mei2 is crucial for meiosis in Schizosaccharomyces pombe. In mei2 mutants, pre-meiotic S-phase is blocked, along with meiosis. Mei2 binds a long non-coding RNA (lncRNA) called meiRNA, which is a ‘sponge RNA’ for the meiotic inhibitor protein Mmi1. The interaction between Mei2, meiRNA and Mmi1 protein is essential for meiosis. But mei2 mutants have stronger and different phenotypes than meiRNA mutants, since mei2Δ arrests before pre-meiotic S, while the meiRNA mutant arrests after pre-meiotic S but before meiosis. This suggests Mei2 may bind additional RNAs. To identify novel RNA targets of Mei2, which might explain how Mei2 regulates pre-meiotic S, we used RNA immunoprecipitation and cross-linking immunoprecipitation. In addition to meiRNA, we found the mRNAs for mmi1 (which encodes Mmi1) and for the S-phase transcription factor rep2 . There were also three other RNAs of uncertain relevance. We suggest that at meiotic initiation, Mei2 may sequester rep2 mRNA to help allow pre-meiotic S, and then may bind both meiRNA and mmi1 mRNA to inactivate Mmi1 at two levels, the protein level (as previously known), and also the mRNA level, allowing meiosis. We call Mei2–meiRNA a ‘double sponge’ (i.e. binding both an mRNA and its encoded protein).

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Dissertations / Theses on the topic "Meiosis; Meiotic"

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Canales, C. "Characterisation of extra sporogenous cells (ESP) : an avbidopsis gene required for another development." Thesis, University of Oxford, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.365861.

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Pasternak, Michał. "RNAi screen for meiotic genes in mammals reveals BTG4 as a novel regulator of meiosis." Thesis, University of Cambridge, 2016. https://www.repository.cam.ac.uk/handle/1810/283984.

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Widger, Alexander David. "Ablating ATR in mouse meiosis and its consequences for synapsis, recombination and meiotic surveillance mechanisms." Thesis, University College London (University of London), 2018. http://discovery.ucl.ac.uk/10043772/.

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Meiosis is a fundamental part in the life cycle of sexual species. It denotes a specialised cell division that halves chromosome numbers to generate haploid gametes for reproduction. Cells unable to competently progress through meiotic prophase activate cell surveillance mechanisms causing their elimination. Given the importance of DNA damage kinases like ATR in facilitating mitotic cell surveillance mechanisms, I characterized Atr-deficient spermatocytes to determine the importance of ATR for mammalian meiosis. I found that ATR ensures efficient chromosome synapsis, and that that is partially independent of meiotic recombination. In addition, ATR has three distinct roles in meiotic recombination. Firstly, during nucleolytic processing, it acts to regulate SPO11-oligonucleotide size when ATM is deleted. Secondly, it is required for accurate RAD51 and DMC1 recruitment to DSBs. Thirdly, it regulates the timing of DNA DSB repair on both unsynapsed and synapsed chromosomes. Finally I found that the loss of ATR is unable to rescue meiotic arrest in multiple meiotic mutants, including mice deficient for the other DNA damage PIKKs ATM and DNA-PK. My findings reveal multiple roles for ATR in male mouse meiosis.

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Malik, Shehre-Banoo. "The early evolution of meiotic genes." Diss., University of Iowa, 2007. http://ir.uiowa.edu/etd/275/.

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Ye, Jinpei. "Signalling pathways controlling meiosis in porcine oocytes." Thesis, University of Nottingham, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.273192.

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Wolf, Peter G. [Verfasser], and Olaf [Akademischer Betreuer] Stemmann. "Meiosis made simple : Mechanisms of meiotic chromosome dynamics elucidated in somatic cells / Peter G. Wolf ; Betreuer: Olaf Stemmann." Bayreuth : Universität Bayreuth, 2017. http://d-nb.info/113220092X/34.

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Fazio, Cynthia Marie. "The influence of meiotic onset on and the role of apoptosis in oocyte death during the meiotic prophase /." Thesis, McGill University, 2005. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=97951.

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Loss of germ cells that entered meiosis at different developmental stages was compared. Mice were injected with BrdU at 13.3, 14.3 or 15.3 days post coitum (dpc) and sacrificed either 3 days after BrdU injection or 4 days post partum (dpp). BrdU-labeled germ cells were detected in ovarian sections through double immunofluorescent staining for BrdU and either GCNA-1 or MVH as a germ cell marker. The results show that the loss of germ cells that entered meiosis at 13.3 or 15.3 dpc was excessive compared to the loss of total germ cells. Such preferential elimination was not found for germ cells that entered meiosis at 14.3 dpc. We conclude that oocyte loss during meiotic prophase is influenced by the timing of meiotic onset.
The mechanism of germ cell loss during ovarian development was tested by the presence of markers for apoptosis. Mouse ovaries were isolated at 12.5 dpc, 18.5 dpc and 2 dpp and cultured with doxorubicin (DXR) to induce cell death. Ovarian histological sections were double immunofluorescent stained for GCNA-1 and cleaved caspase-3 or PARP-1. The results suggest that caspase-3 is not activated in germ cells throughout ovarian development whereas PARP-1 is activated in germ cells at 12.5 dpc and 2 dpp but not at 18.5 dpc. Thus, no evidence has yet been provided to support the hypothesis that oocyte death during the meiotic prophase is mediated by apooptosis.

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Ferguson, Kyle Akira. "Meiotic defects in infertile men." Thesis, University of British Columbia, 2008. http://hdl.handle.net/2429/1228.

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While the introduction of intracytoplasmic sperm injection (ICSI) has revolutionized the treatment of male infertility, concerns have been raised regarding the risk of chromosomal abnormalities in pregnancies derived from ICSI. Studies on sperm from infertile men have suggested that this population may produce higher rates of aneuploid sperm. Thus, we hypothesized that defects in early meiotic events may contribute to both male infertility and the production of aneuploid sperm. We used immunofluorescent techniques to observe the synapsis and recombination of chromosomes during meiosis, and fluorescent in-situ hybridization (FISH) to assess sperm aneuploidy. We analyzed testicular tissue from thirty-one men (10 fertile and 21 infertile men). We observed that ~36% (5/14) of men with impaired spermatogenesis displayed reduced genome-wide recombination. When all men were pooled, we observed an inverse correlation between the frequency of sex chromosome recombination and XY disomy in the sperm. We combined immunofluorescent and FISH techniques to study recombination patterns on chromosomes 13, 18 and 21 in fifteen men (5 fertile and 10 infertile men). Four of the infertile men displayed altered recombination distributions on at least one of the chromosome arms studied. Finally, we examined early meiotic events in two biopsies from an azoospermic t(8;13) carrier. While global recombination rates were not altered, recombination frequencies were reduced specifically on the rearranged chromosomes. Asynapsed quadrivalents were observed in 90% and 87% of pachytene nuclei from the first and second biopsies, respectively, and were frequently associated with the sex chromosomes. BRCA1 and γH2AX, two proteins implicated in meiotic sex chromosome inactivation, localized along asynapsed regions regardless of whether or not they were associated with the sex chromosomes, suggesting that regions of autosomal chromosomes that fail to synapse undergo transcriptional silencing in humans. In summary, we observed that a subset of infertile men display alterations in the number and position of meiotic crossovers, which may contribute to both infertility and an increased risk of sperm aneuploidy. The fidelity of synapsis is also a critical factor in determining the outcome of gametogenesis in humans, as the transcriptional inactivation of asynapsed regions may silence meiotic genes, leading to meiotic arrest and infertility.

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Cooper, Timothy J. "Investigating the spatial regulation of meiotic recombination in S. cerevisiae." Thesis, University of Sussex, 2018. http://sro.sussex.ac.uk/id/eprint/74309/.

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Koehn, Demelza Rae Malone Robert E. "Analysis of meiotic recombination initiation in Saccharomyces cerevisiae." Iowa City : University of Iowa, 2009. http://ir.uiowa.edu/etd/303.

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Books on the topic "Meiosis; Meiotic"

1

John, Bernard. Meiosis. Cambridge [England]: Cambridge University Press, 1990.

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Meiosis. Dordrecht: Humana Press, 2009.

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John, B. Meiosis. Cambridge: Cambridge University Press, 1990.

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Stuart, David T., ed. Meiosis. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-6340-9.

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Keeney, Scott, ed. Meiosis. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60761-103-5.

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Keeney, Scott, ed. Meiosis. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-527-5.

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Pradillo, Mónica, and Stefan Heckmann, eds. Plant Meiosis. New York, NY: Springer New York, 2020. http://dx.doi.org/10.1007/978-1-4939-9818-0.

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Pawlowski, Wojciech P., Mathilde Grelon, and Susan Armstrong, eds. Plant Meiosis. Totowa, NJ: Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-333-6.

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Egel, Richard, and Dirk-Henner Lankenau, eds. Recombination and Meiosis. Berlin, Heidelberg: Springer Berlin Heidelberg, 2008. http://dx.doi.org/10.1007/978-3-540-75373-5.

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Egel, Richard, and Dirk-Henner Lankenau, eds. Recombination and Meiosis. Berlin, Heidelberg: Springer Berlin Heidelberg, 2008. http://dx.doi.org/10.1007/978-3-540-68984-3.

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Book chapters on the topic "Meiosis; Meiotic"

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Muyt, A. De, R. Mercier, C. Mézard, and M. Grelon. "Meiotic Recombination and Crossovers in Plants." In Meiosis, 14–25. Basel: KARGER, 2008. http://dx.doi.org/10.1159/000166616.

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Pryce, D. W., and R. J. McFarlane. "The Meiotic Recombination Hotspots of Schizosaccharomyces pombe." In Meiosis, 1–13. Basel: KARGER, 2008. http://dx.doi.org/10.1159/000166614.

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Khil, P. P., and R. D. Camerini-Otero. "Variation in Patterns of Human Meiotic Recombination." In Meiosis, 117–27. Basel: KARGER, 2008. http://dx.doi.org/10.1159/000166623.

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Alsheimer, M. "The Dance Floor of Meiosis: Evolutionary Conservation of Nuclear Envelope Attachment and Dynamics of Meiotic Telomeres." In Meiosis, 81–93. Basel: KARGER, 2008. http://dx.doi.org/10.1159/000166621.

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Mogessie, Binyam. "Visualization and Functional Analysis of Spindle Actin and Chromosome Segregation in Mammalian Oocytes." In Methods in Molecular Biology, 267–95. New York, NY: Springer US, 2019. http://dx.doi.org/10.1007/978-1-0716-0219-5_17.

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Abstract Chromosome segregation is conserved throughout eukaryotes. In most systems, it is solely driven by a spindle machinery that is assembled from microtubules. We have recently discovered that actin filaments that are embedded inside meiotic spindles (spindle actin) are needed for accurate chromosome segregation in mammalian oocytes. To understand the function of spindle actin in oocyte meiosis, we have developed high-resolution and super-resolution live and immunofluorescence microscopy assays that are described in this chapter.

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Loidl, Josef, Pawel Pasierbek, and Ann M. Rose. "Conservation and Variability of Meiotic Processes — Lessons from the Unconventional Meiosis of C. elegans." In Chromosomes Today, 93–101. Dordrecht: Springer Netherlands, 2004. http://dx.doi.org/10.1007/978-94-017-1033-6_10.

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Martinez-Perez, E. "Meiosis in Cereal Crops: the Grasses are Back." In Meiosis, 26–42. Basel: KARGER, 2008. http://dx.doi.org/10.1159/000166617.

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Zetka, M. "Homologue Pairing, Recombination and Segregation in Caenorhabditis elegans." In Meiosis, 43–55. Basel: KARGER, 2008. http://dx.doi.org/10.1159/000166618.

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McKee, B. D. "Homolog Pairing and Segregation in Drosophila Meiosis." In Meiosis, 56–68. Basel: KARGER, 2008. http://dx.doi.org/10.1159/000166619.

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Yang, F., and P. J. Wang. "The Mammalian Synaptonemal Complex: A Scaffold and Beyond." In Meiosis, 69–80. Basel: KARGER, 2008. http://dx.doi.org/10.1159/000166620.

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Conference papers on the topic "Meiosis; Meiotic"

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Andronic, Larisa. "Impactul destabilizator al infecțiilor virale asupra microsporogenezei la plantele gazdă." In International Scientific Symposium "Plant Protection – Achievements and Prospects". Institute of Genetics, Physiology and Plant Protection, Republic of Moldova, 2020. http://dx.doi.org/10.53040/9789975347204.61.

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The pathogenicity reactions described in the sensitive genotypes of tomatoes and barley include specifically changes in the processes of meiotic division, with repercussions in the offspring of infected plants. The percentage of aberrant pollen mother cells (PMCs) in the offspring is at the level of control plants, while the percentage of aberrations per PMC and the frequency of meiotic conjugation are significantly higher. The consequences in meiotic division in virus free progenies reflect the destabilizing transgenerational effect of viral infection on microsporogenesis processes.

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Wiriyasermkul, Nattavut, Veera Boobjing, and Pisit Chanvarasuth. "A Meiosis Genetic Algorithm." In 2010 Seventh International Conference on Information Technology: New Generations. IEEE, 2010. http://dx.doi.org/10.1109/itng.2010.152.

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Booker, William, and Dean Frederick Hougen. "Meiotic Inheritance and Gene Dominance in Synthetic Sympatric Speciation." In 2018 IEEE Congress on Evolutionary Computation (CEC). IEEE, 2018. http://dx.doi.org/10.1109/cec.2018.8477761.

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Stauffer, Weston T., Liangyu Zhang, and Abby Dernburg. "Diffusion through a liquid crystalline compartment regulates meiotic recombination." In Biophysics, Biology and Biophotonics IV: the Crossroads, edited by Adam Wax and Vadim Backman. SPIE, 2019. http://dx.doi.org/10.1117/12.2513378.

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Bugao Li, Xiaohong Guo, and Li Zhang. "Characterization of a fertility candidate gene Ccdc79 in meiotic germ cells." In 2011 International Conference on Remote Sensing, Environment and Transportation Engineering (RSETE). IEEE, 2011. http://dx.doi.org/10.1109/rsete.2011.5966088.

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Escandon, Julia, Scott Lindsey, Mark S. Eller, and James M. Grichnik. "Abstract 3010: Meiotic cohesin REC8 associates with chromosome instability in melanoma." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-3010.

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Rivera, Maricruz, Qiulian Wu, Petra Hamerlik, Anita Hjelmeland, Shideng Bao, and Jeremy Rich. "Abstract B44: Acquisition of meiotic DNA repair regulators maintain genome stability in glioblastoma." In Abstracts: AACR Special Conference: Advances in Brain Cancer Research; May 27-30, 2015; Washington, DC. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.brain15-b44.

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8

"Chromatin and cytoskeleton reorganization in meiosis of wheat-rye substitution line (3R3B)." In Plant Genetics, Genomics, Bioinformatics, and Biotechnology. Institute of Cytology and Genetics, Siberian Branch of the Russian Academy of Sciences, 2019. http://dx.doi.org/10.18699/plantgen2019-215.

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"3D-microscopy of prophase nucleus in the meiosis I of wheat-rye amphihaploids." In Plant Genetics, Genomics, Bioinformatics, and Biotechnology. Institute of Cytology and Genetics, Siberian Branch of the Russian Academy of Sciences, 2019. http://dx.doi.org/10.18699/plantgen2019-106.

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Wu, Min, Chee Keong Kwoh, Teresa M. Przytycka, Jing Li, and Jie Zheng. "Integration of genomic and epigenomic features to predict meiotic recombination hotspots in human and mouse." In the ACM Conference. New York, New York, USA: ACM Press, 2012. http://dx.doi.org/10.1145/2382936.2382974.

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Reports on the topic "Meiosis; Meiotic"

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Gregory p. Copenhaver. Regulation of Meiotic Recombination. Office of Scientific and Technical Information (OSTI), November 2011. http://dx.doi.org/10.2172/1028811.

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Durham, Mary. Demonstrating Meiosis Using Manipulatable Chromosomes and Cells. Genetics Society of America Peer-Reviewed Education Portal (GSA PREP), May 2015. http://dx.doi.org/10.1534/gsaprep.2015.002.

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Dray, Eloise, Myun Hwa Dunlop, Liisa Kauppi, Joseph San San Filippo, Claudia Wiese, Miaw-Sheue Tsai, Sead Begovic, et al. Molecular Basis for Enhancement of the Meiotic DMCI Recombinase by RAD51AP1. Office of Scientific and Technical Information (OSTI), November 2010. http://dx.doi.org/10.2172/1011037.

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Hood-DeGrenier, Jennifer. Active Learning Workshops for Teaching Key Topics in Introductory Cell and Molecular Biology: Structure of DNA/RNA, Structure of Proteins, and Cell Division via Mitosis and Meiosis. Genetics Society of America Peer-Reviewed Education Portal (GSA PREP), December 2015. http://dx.doi.org/10.1534/gsaprep.2015.004.

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