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Books on the topic 'Megakaryocytes'

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Published: 4 June 2021
Last updated: 10 March 2023

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1

Gibbins, Jonathan M., and Martyn P. Mahaut-Smith. Platelets and Megakaryocytes. New Jersey: Humana Press, 2004. http://dx.doi.org/10.1385/1592597823.

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2

Gibbins, Jonathan M., and Martyn P. Mahaut-Smith. Platelets and Megakaryocytes. New Jersey: Humana Press, 2004. http://dx.doi.org/10.1385/1592597831.

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3

Gibbins, Jonathan M., and Martyn P. Mahaut-Smith, eds. Platelets and Megakaryocytes. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-61779-307-3.

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4

Gibbins, Jonathan M., and Martyn Mahaut-Smith, eds. Platelets and Megakaryocytes. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-8585-2.

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5

M, Gibbins Jonathan, and Mahaut-Smith Martyn P, eds. Platelets and megakaryocytes. Totowa, N.J: Humana Press, 2004.

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6

Harris, J. R., ed. Megakaryocytes, Platelets, Macrophages, and Eosinophils. Boston, MA: Springer US, 1991. http://dx.doi.org/10.1007/978-1-4757-9531-8.

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7

R, Harris James, ed. Megakaryocytes, platelets, macrophages, and eosinophils. New York: Plenum Press, 1991.

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8

W, Colman Robert, and Smith J. Bryan, eds. Methods for studying platelets and megakaryocytes. New York: Liss, 1987.

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9

F, Levine Richard, ed. Megakaryocyte development and function: Proceedings of an international conference held at the Marine Biological Laboratory, Woods Hole, Massachusetts, September 18-21, 1985. New York: Liss, 1986.

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10

A, Selivanov V., and Lanin V. N, eds. Reguli͡at͡sii͡a megakariot͡sitopoėza. Pushchino: Pushchinskiĭ nauch. t͡sentr AN SSSR, 1991.

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11

Janine, Breton-Gorius, ed. Molecular biology and differentiation of megakaryocytes: Proceedings of the Third International Conference on Megakaryocytes--Megakaryocytes--Cellular and Molecular Biology, held at Conseil Régional de Bourgogne, Dijon, France, July 23-27, 1989. New York: Wiley-Liss, 1990.

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12

J, Murphy Martin, and Kuter David J, eds. Thrombopoietin: From molecule to medicine. Miamisburg, Ohio: AlphaMed Press, 1998.

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13

I, Barnhart Marion, and Lusher Jeanne M. 1935-, eds. Factor VIII/vWF and platelet formation and function in health and disease: A tribute to Marion I. Barnhart. New York, N.Y: New York Academy of Sciences, 1987.

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14

Landesberg, Leonard John. Identification and expression of a novel homeobox gene in a megakaryocytic leukemia cell line. [New Haven, Conn: s.n.], 1994.

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15

Platelets and megakaryocytes. Totowa, NJ: Humana Press, 2004.

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16

Megakaryocytes and platelet disorders. London: Baillière Tindall, 1997.

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17

Breton-Gorius, Janin. Molecular Biology and Differentiation of Megakaryocytes: Proceedings of the Third International Conference on Megakaryocytes--Megakaryocytes--Cellular (Progress in Clinical & Biological Research). Wiley-Liss, 1990.

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18

Harris, J. Robin. Megakaryocytes, Platelets, Macrophages, and Eosinophils. Springer London, Limited, 2013.

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19

Harris, J. Robin. Megakaryocytes, Platelets, Macrophages, and Eosinophils. Springer, 2013.

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20

Colman, Robert W. Methods for Studying Platelets and Megakaryocytes. John Wiley & Sons Inc, 2000.

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21

Megakaryocyte development and function: Proceedings of an international conference held at the Marine Biological Laboratory, Woods Hole, Massachusetts, ... in clinical and biological research). Liss, 1986.

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22

Megakaryocytes and Platelet Disorders (Balliere's Clinical Haematology). Elsevier, 1997.

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23

Gibbins, Jonathan M., and Martyn P. Mahaut-Smith. Platelets and Megakaryocytes : Volume 1: Functional Assays. Humana Press, 2010.

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24

Platelets and Megakaryocytes Methods in Molecular Biology Hardcover. Humana Press, 2011.

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25

Gibbins, Jonathan M., and Martyn P. Mahaut-Smith. Platelets and Megakaryocytes : Volume 2: Perspectives and Techniques. Humana Press, 2010.

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26

Gibbins, Jonathan M., and Martyn P. Mahaut-Smith. Platelets and Megakaryocytes: Volume 3, Additional Protocols and Perspectives. Humana Press, 2016.

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27

Mahaut-Smith, Martyn, and Jonathan M. Gibbins. Platelets and Megakaryocytes: Volume 4, Advanced Protocols and Perspectives. Springer New York, 2019.

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28

GIBBINS, Jonathan, and Martyn MAHAUT-SMITH. Platelets and Megakaryocytes: Volume 4, Advanced Protocols and Perspectives. Springer New York, 2018.

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29

(Editor), Jonathan M. Gibbins, and Martyn P. Mahaut-Smith (Editor), eds. Platelets and Megakaryocytes: Volume 1: Functional Assays (Methods in Molecular Biology). Humana Press, 2004.

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30

Thrombopoietin: From Molecule to Medicine. AlphaMED Press, 1998.

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31

Menard, Michele. Granulopoiesis in megakaryocytes of normal cattle and cattle with the Chediak-Higashi syndrome. 1989.

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32

Platelets and Megakaryocytes: Volume 2: Perspectives and Techniques (Methods in Molecular Biology). Humana Press, 2004.

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33

Harris, J. Robin. Blood Cell Biochemistry, Volume 2: Megakaryocytes, Platelets, Macrophages, and Eosinophils (Blood Cell Biochemistry). Springer, 1991.

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34

Sobolewski, Stanislaw. A new function of megakaryocytes in malignancies: A clinical and experimental study concerning a possible phagocytic role.... Bradford, 1986.

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35

Levin, Jack. Megakaryocyte Development and Function. Wiley & Sons, Incorporated, John, 1986.

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36

Curry, Nicola, and Raza Alikhan. Normal platelet function. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0281.

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The platelet is a small (2–4 µm in diameter), discoid, anucleate cell that circulates in the blood. In health, it plays a vital role in haemostasis, and in disease it contributes to disorders of bleeding and thrombosis. Platelets are produced from the surface of megakaryocytes in the bone marrow, under tight homeostatic control regulated by the cytokine thrombopoietin. Platelets have a lifespan of approximately 7–10 days, and usually circulate in the blood stream in a quiescent state. Intact, undamaged vessel walls help to maintain platelets in this inactive state by releasing nitric oxide, which acts both to dilate the vessel wall and to inhibit platelet adhesion, activation, and aggregation. After trauma to the blood vessel wall, platelets are activated and, acting in concert with the endothelium and coagulation factors, form a stable clot. This chapter addresses platelet structure and function, and the response of platelets to vessel injury.
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37

Albert, Tyler J., and Erik R. Swenson. The blood cells and blood count. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0265.

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Blood is a dynamic fluid consisting of cellular and plasma components undergoing constant regeneration and recycling. Like most physiological systems, the concentrations of these components are tightly regulated within narrow limits under normal conditions. In the critically-ill population, however, haematological abnormalities frequently occur and are largely due to non-haematological single- or multiple-organ pathology. Haematopoiesis originates from the pluripotent stem cell, which undergoes replication, proliferation, and differentiation, giving rise to cells of the erythroid, myeloid, and lymphoid series, as well as megakaryocytes, the precursors to platelets. The haemostatic system is responsible for maintaining blood fluidity and, at the same time, prevents blood loss by initiating rapid, localized, and appropriate blood clotting at sites of vascular damage. This system is complex, comprising both cellular and plasma elements, i.e. platelets, coagulation and fibrinolytic cascades, the natural intrinsic and extrinsic pathways of anticoagulation, and the vascular endothelium. A rapid, reliable, and inexpensive method of examining haematological disorders is the peripheral blood smear, which allows practitioners to assess the functional status of the bone marrow during cytopenic states. Red blood cells, which are primarily concerned with oxygen and carbon dioxide transport, have a normal lifespan of only 120 days and require constant erythropoiesis. White blood cells represent a summation of several circulating cell types, each deriving from the hematopoietic stem cell, together forming the critical components of both the innate and adaptive immune systems. Platelets are integral to haemostasis, and also aid our inflammatory and immune responses, help maintain vascular integrity, and contribute to wound healing.
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38

Collins, Graham, and Chris Bunch. Myeloproliferative disorders. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0291.

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Myeloproliferative disorders (also called myeloproliferative neoplasms) can be defined as clonal haematopoietic disorders resulting in excess production of one or more blood cell lineage. The four main conditions are primary polycythaemia, which is characterized by excess red-cell production; essential thrombocythaemia, which is characterized by excess platelet production; chronic myeloid leukaemia, which is characterized by excess granulocyte production; and myelofibrosis, which is characterized by excess megakaryocyte proliferation, which results in a reactive fibroblast proliferation causing marrow fibrosis and failure. This chapter addresses the causes, diagnosis, and management of these myeloproliferative disorders.
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