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Books on the topic 'Mechanisms'

Author: Grafiati

Published: 4 June 2021

Last updated: 6 September 2023

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1

Caldwell, Raymond. Mechanisms. Tonbridge: Hands On Publishing, 1997.

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2

Rich, Steve. Mechanisms. Cheltenham: Thornes, 1991.

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3

CCA Wattis Institute for Contemporary Arts and Wiener Secession, eds. Mechanisms. San Francisco: CCA Wattis Institute for Contemporary Arts, 2017.

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4

Lenarčič, Jadran. Robot Mechanisms. Dordrecht: Springer Netherlands, 2013.

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5

Lobontiu, Nicolae. Compliant Mechanisms. Second edition. | Boca Raton : CRC Press, 2021.: CRC Press, 2020. http://dx.doi.org/10.1201/9780429184666.

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Lenarcic, Jadran, Tadej Bajd, and Michael M. Stanišić. Robot Mechanisms. Dordrecht: Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-4522-3.

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7

Mori, Nozomu, and Inhee Mook-Jung, eds. Aging Mechanisms. Tokyo: Springer Japan, 2015. http://dx.doi.org/10.1007/978-4-431-55763-0.

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Bruckner, Reinhard. Organic Mechanisms. Edited by Michael Harmata. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-03651-4.

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Calzavarini, Fabrizio, and Marco Viola, eds. Neural Mechanisms. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-54092-0.

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10

Koloc, Zdeněk. Cam mechanisms. Amsterdam: Elsevier, 1993.

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11

Hypertension mechanisms. Orlando: Grune & Stratton, 1987.

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12

Ingle, M. R. Genetic mechanisms. Oxford: Basil Blackwell, 1986.

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13

D, Lapointe Jacques Ph, and Brakier-Gingras L, eds. Translation mechanisms. Georgetown, Tex: Landes Bioscience/Eurekah.com, 2003.

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14

Materials & mechanisms. Tonbridge): Hands On Publishing, 1998.

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15

(Project), BIOTOL, Open Universiteit (Heerlen Netherlands), and University of Greenwich, eds. Defence mechanisms. Oxford: Butterworth-Heinemann, 1993.

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16

Brusco, Sandro. Merger mechanisms. Milan: Fondazione Eni Enrico Mattei, 2004.

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17

Sinclair-Desgagne, Bernard. "Unavoidable mechanisms". Fontainbleau: INSEAD, 1986.

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18

I, Danilov V. Social choice mechanisms. Berlin: Springer, 2002.

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19

Mathura, Sanya. Lubrication Degradation Mechanisms. First edition. | Boca Raton : CRC Press, 2021. |: CRC Press, 2020. http://dx.doi.org/10.1201/9781003102274.

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20

Glennan, Stuart. Mechanisms. Edited by Helen Beebee, Christopher Hitchcock, and Peter Menzies. Oxford University Press, 2010. http://dx.doi.org/10.1093/oxfordhb/9780199279739.003.0016.

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This article explores three issues surrounding the adequacy of the mechanical approach to causation. First, it considers whether the appeal to laws or invariant generalizations in characterizing interactions between parts of mechanisms either makes the mechanical theory circular or reduces it to a regularity, counterfactual, or manipulability theory. Second, the article discusses Machamer, Darden, and Craver's argument that the proper understanding of the causal productivity of mechanisms requires the recognition of the novel metaphysical category of activities. Third, it discusses the relationship between mechanical theories and process theories.

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21

Kirschenbaum, Matthew G. Mechanisms. The MIT Press, 2007. http://dx.doi.org/10.7551/mitpress/7393.001.0001.

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22

Mechanisms. Elsevier, 2022. http://dx.doi.org/10.1016/c2021-0-02621-3.

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23

Glennan, Stuart. Mechanisms. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780198779711.003.0002.

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This chapter introduces the account of minimal mechanism, according to which a mechanism consists of a set of parts or entities whose activities and interactions are organized so as to be responsible for some phenomenon. The concepts appealed to in this account—phenomena, entities, activities, organization—are elaborated, and the relation of minimal mechanism to other accounts is explored. Mechanisms are shown to be compounds that are organized in two dimensions, the horizontal dimension of causal dependence and a vertical or part-whole dimension called mechanistic constitution. I argue that almost all natural and social phenomena depend upon mechanisms so characterized, and that this fact leads to a new mechanical ontology, which alters the way we should think about traditional metaphysical categories like substances, processes, properties, dispositions, causes, and laws of nature.

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24

Mechanisms. New York: Plenum Press, 1985.

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25

Mechanisms. London: Gatsby Technical Education Project, 1997.

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26

Chandler, Martin. Mechanisms. Cassell, 1991.

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27

Mechanisms. [Leamington Spa]: [Scholastic], 1996.

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28

Mechanisms. Continuum Intl Pub Group (Sd), 1990.

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29

(Editor), N. B. Finter, and R. M. Friedman (Editor), eds. Mechanisms Produc & Action: (Mechanisms of Production & Action). Elsevier Science & Technology, 1985.

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30

Organic Reaction Mechanisms, 1983 (Organic Reaction Mechanisms). John Wiley & Sons Inc, 1986.

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31

(Editor), A. C. Knipe, and Bill Watts (Editor), eds. Organic Reaction Mechanisms , Organic Reaction Mechanisms, 1997. Wiley-Interscience, 2001.

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32

Persaud, T. V. N. Teratogenic Mechanisms. Springer, 2012.

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33

Robot Mechanisms. Springer, 2012.

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34

Mansfeld, Florian B. Corrosion Mechanisms. Taylor & Francis Group, 2020.

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35

Hughes, Alis, and Lesley Jones. Pathogenic Mechanisms. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199929146.003.0013.

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Huntington’s disease (HD) pathogenesis is complex. In the two decades since the gene and its mutation were discovered, there has been extensive exploration of how the expanded CAG repeat in HTT leads to neurodegeneration in HD. This chapter focuses on the mechanisms that potentially contribute to the dysfunction and death of cells in HD. These include repeat instability and RNA toxicity and the production, processing, modification, and degradation of mutant huntingtin. The effects of mutant HTT on cellular processes such as transcription, transport, neurotransmission, and protein clearance are also described. The interdependence and individual importance of these mechanisms in disease etiology remains to be clarified; however, consideration of each could be important for the development of therapeutic interventions in HD.

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36

Monaco, Claudia, and Giuseppina Caligiuri. Molecular mechanisms. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755777.003.0014.

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The development of the atherosclerotic plaque relies on specific cognate interactions between ligands and receptors with the ability to regulate cell recruitment, inflammatory signalling, and the production of powerful inflammatory and bioactive lipid mediators. This chapter describes how signalling is engaged by cell-cell surface interactions when the endothelium interacts with platelets and leukocytes enhancing leukocyte recruitment during atherogenesis. It also exemplifies intracellular signalling pathways induced by the activation of innate immune receptors, the most potent activators of inflammation in physiology and disease. Differences are highlighted in innate signalling pathways in metabolic diseases such as atherosclerosis compared to canonical immunological responses. Finally, the key lipid mediators whose production can affect endothelial function, inflammation, and atherosclerosis development are summarized. This Chapter will take you through these fundamental steps in the development of the atherosclerotic plaque by summarizing very recent knowledge in the field and highlighting recent or ongoing clinical trials that may enrich our ability to target cardiovascular disease in the future.

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37

Bechtel, William. Mental Mechanisms. Psychology Press, 2007. http://dx.doi.org/10.4324/9780203810095.

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38

Keele), NATO Advanced Research Workshop on Mechanics of Hearing (1988 University of. Cochlear mechanisms. Plenum in cooperation with NATO Scientific Affairs Division, 1989.

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39

Wolstenholme, G. E. W., and Maeve O'Connor. Adrenergic Mechanisms. Wiley & Sons, Incorporated, John, 2009.

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40

Hedström, Peter, and Richard Swedberg, eds. Social Mechanisms. Cambridge University Press, 1998. http://dx.doi.org/10.1017/cbo9780511663901.

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41

Montgomery, Erwin B. Pathophysiological Mechanisms. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190259600.003.0008.

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Pathophysiology is central to neuroscience and psychiatry for a number of reasons. Indeed, nearly every inference to normal function of the nervous system is derived from notions of pathophysiology. Understanding DBS’s mechanisms of action—a therapeutic mechanism, particularly—greatly depends on the prior informing conception of pathophysiology. Several current theories of pathophysiology are critically reviewed such as the GPi Rate, high beta oscillations, excessive bursting and hypersynchronization theories. A novel theory offered that introduces the concept of the basal ganglia-thalamic-cortical system as a network of loosely coupled nonlinear polysynaptic reentrant oscillators.

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42

Lobontiu, Nicolae. Compliant Mechanisms. CRC Press, 2002. http://dx.doi.org/10.1201/9781420040272.

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43

Johnson, Samuel G. B., and Woo-kyoung Ahn. Causal Mechanisms. Edited by Michael R. Waldmann. Oxford University Press, 2017. http://dx.doi.org/10.1093/oxfordhb/9780199399550.013.12.

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This chapter reviews empirical and theoretical results concerning knowledge of causal mechanisms—beliefs about how and why events are causally linked. First, it reviews the effects of mechanism knowledge, showing that mechanism knowledge can override other cues to causality (including covariation evidence and temporal cues) and structural constraints (the Markov condition), and that mechanisms play a key role in various forms of inductive inference. Second, it examines several theories of how mechanisms are mentally represented—as associations, forces or powers, icons, abstract placeholders, networks, or schemas—and the empirical evidence bearing on each theory. Finally, it describes ways that people acquire mechanism knowledge, discussing the contributions from statistical induction, testimony, reasoning, and perception. For each of these topics, it highlights key open questions for future research.

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44

Piccinini, Gualtiero. Neurocognitive Mechanisms. Oxford University Press, 2020. http://dx.doi.org/10.1093/oso/9780198866282.001.0001.

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This book provides the foundations for a neurocomputational explanation of cognition based on contemporary cognitive neuroscience. An ontologically egalitarian account of composition and realization, according to which all levels are equally real, is defended. Multiple realizability and mechanisms are explicated in light of this ontologically egalitarian framework. A goal-contribution account of teleological functions is defended, and so is a mechanistic version of functionalism. This provides the foundation for a mechanistic account of computation, which in turn clarifies the ways in which the computational theory of cognition is a multilevel mechanistic theory supported by contemporary cognitive neuroscience. The book argues that cognition is computational at least in a generic sense. The computational theory of cognition is defended from standard objections yet a priori arguments for the computational theory of cognition are rebutted. The book contends that the typical vehicles of neural computations are representations and that, contrary to the received view, neural representations are observable and manipulable in the laboratory. The book also contends that neural computations are neither digital nor analog; instead, neural computations are sui generis. The book concludes by investigating the relation between computation and consciousness, suggesting that consciousness may have a functional yet not wholly computational nature.

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45

van, José. Platform Mechanisms. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190889760.003.0003.

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The second chapter discusses how platforms introduce new mechanisms to social interaction through the mutual articulation of platform technologies, economic models, and user practices. The mechanism of “datafication” refers to the ability of networked platforms to render into data many aspects of the world that have never been quantified before. Datafication revolves around the capturing and circulation of data. “Commodification” concerns the transformation of online and offline objects, activities, emotions, and ideas into tradable commodities. It involves the development of multisided markets and new business models. Finally, the mechanism of “selection” is about the curation of most relevant topics, terms, actors, objects, offers, services, etc. It takes shape through personalization, trends and reputations, and moderation practices. Understanding the platform society requires a thorough analysis of the ecosystem’s mechanisms and the constantly evolving techno-commercial and sociocultural practices through which they take shape.

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46

Corrosion mechanisms. New York: M. Dekker, 1987.

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47

Market mechanisms. [London]: Peat Marwick, 1992.

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48

Abraham, Wickliffe. Memory Mechanisms. Psychology Press, 2014. http://dx.doi.org/10.4324/9781315807980.

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49

Simple mechanisms. Alfreton: Technology Teaching Systems, 1995.

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50

Staff, CIBA Foundation Symposium, and G. E. W. Wolstenholme. Adrenergic Mechanisms. Wiley & Sons, Limited, John, 2008.

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