Relevant bibliographies by topics / Mammary glands

Academic literature on the topic 'Mammary glands'

Author: Grafiati
Published: 4 June 2021
Last updated: 27 July 2024

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Journal articles on the topic "Mammary glands"

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Biswas, Swarajit Kumar, Saswati Banerjee, Ginger Wendolyn Baker, Chieh-Yin Kuo, and Indrajit Chowdhury. "The Mammary Gland: Basic Structure and Molecular Signaling during Development." International Journal of Molecular Sciences 23, no. 7 (March 31, 2022): 3883. http://dx.doi.org/10.3390/ijms23073883.

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The mammary gland is a compound, branched tubuloalveolar structure and a major characteristic of mammals. The mammary gland has evolved from epidermal apocrine glands, the skin glands as an accessory reproductive organ to support postnatal survival of offspring by producing milk as a source of nutrition. The mammary gland development begins during embryogenesis as a rudimentary structure that grows into an elementary branched ductal tree and is embedded in one end of a larger mammary fat pad at birth. At the onset of ovarian function at puberty, the rudimentary ductal system undergoes dramatic morphogenetic change with ductal elongation and branching. During pregnancy, the alveolar differentiation and tertiary branching are completed, and during lactation, the mature milk-producing glands eventually develop. The early stages of mammary development are hormonal independent, whereas during puberty and pregnancy, mammary gland development is hormonal dependent. We highlight the current understanding of molecular regulators involved during different stages of mammary gland development.
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Hambarova, G. H., H. E. Askerova, and M. S. Panakhova. "Normal decoding of the structure of the chest in the light of possibilities a new generation of high-frequency ultrasonographic sensors." HEALTH OF WOMAN, no. 3(129) (April 30, 2018): 123–27. http://dx.doi.org/10.15574/hw.2018.129.123.

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Over the years of the existence of echography in Japan and the English-speaking countries, a large number of atlases and a manual textbookы where the authors describe the echo anatomy of the mammary glands have been published. The rapid technological growth has led to the fact that the echo anatomy of the mammary glands described on the basis of equipment from the 80s-90s does not correspond to the capabilities of modern ultrasonic devices. There was a need to clarify and detail the echographic image of the mammary glands, taking into account the possibilities of a new generation of 10–12 MHz high-frequency, and also upon the use of the new Doppler techniques. The anatomical structures of the mammary glands are clearly differentiated using modern ultrasound equipment. The breast tissue is normally varied widely and depends on the ratio of fat, connective and glandular tissue. The USM allows visualizing the tomographic section of the image of a fragment of the mammary gland from the skin cover to the chest wall. Key words: breast glande, ultrasonography, dopрlerography, US-sensor.
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Tian, Lei, Shancheng Guo, Zhiye Zhao, Yuxu Chen, Chunmei Wang, Qingzhang Li, and Ye Li. "miR-30a-3p Regulates Autophagy in the Involution of Mice Mammary Glands." International Journal of Molecular Sciences 24, no. 18 (September 20, 2023): 14352. http://dx.doi.org/10.3390/ijms241814352.

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The mammary gland undergoes intensive remodeling during the lactation cycle, and the involution process of mammary gland contains extensive epithelial cells involved in the process of autophagy. Our studies of mice mammary glands suggest that miR-30a-3p expression was low during involution compared with its high expression in the mammary glands of lactating mice. Then, we revealed that miR-30a-3p negatively regulated autophagy by autophagy related 12 (Atg12) in mouse mammary gland epithelial cells (MMECs). Restoring ATG12, knocking down autophagy related 5 (Atg5), starvation, and Rapamycin were used to further confirm this conclusion. Overexpression of miR-30a-3p inhibited autophagy and altered mammary structure in the involution of the mammary glands of mice, which was indicative of alteration in mammary remodeling. Taken together, these results elucidated the molecular mechanisms of miR-30a-3p as a key induction mediator of autophagy by targeting Atg12 within the transition period between lactation and involution in mammary glands.
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Meador, Vincent P., and Billy L. Deyoe. "Effect of milk stasis on Brucella abortus infection of the mammary gland in goats." American Journal of Veterinary Research 52, no. 6 (June 1, 1991): 886–90. http://dx.doi.org/10.2460/ajvr.1991.52.06.886.

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SUMMARY To compare the effects of milk stasis and milk flow on Brucella abortus infection of the mammary gland under the same systemic conditions, primiparous goats (n = 5) were inoculated iv with B abortus on the day of parturition, and suckling by their neonates was restricted to one mammary gland. Goats were euthanatized and necropsied at 3 weeks after inoculation, and milk, mammary glands, and supramammary lymph nodes were evaluated by bacteriologic, histologic, and immunoenzymatic staining techniques. Nonnursed mammary glands had high titers of brucellae in milk, moderate interstitial mastitis, and brucellar antigen in macrophages located primarily in alveolar and ductal lumina. Brucellae often filled the macrophage cytoplasm. In contrast, nursed mammary glands had fewer brucellae in milk, minimal inflammatory changes, and no detectable brucellar antigen in histologic sections. Hyperplastic changes were only seen in supramammary lymph nodes draining nonnursed mammary glands; these contained more brucellae than lymph nodes draining nursed mammary glands. These studies show that milk stasis may be the sole cause of increased susceptibility of nonnursed mammary glands to B abortus infection.
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Payan-Carreira, Rita, and Ana C. Martins-Bessa. "Ultrasonographic assessment of the feline mammary gland." Journal of Feline Medicine and Surgery 10, no. 5 (October 2008): 466–71. http://dx.doi.org/10.1016/j.jfms.2008.03.006.

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The aim of this study is to characterise the feline mammary echotexture using B-mode ultrasonography, which is not routinely used to examine the feline mammary gland. Using a 5–9 MHz linear transducer the ultrasonographic appearance of non-stimulated and stimulated mammary glands was determined in 35 mature intact non-pregnant, pregnant and lactating queens aged from 16 months to 8 years. In intact non-pregnant queens, mammary glands are fairly underdeveloped and on the ultrasonograms they appear with a regular hypoechoic texture and generally show a thickness of less than 2.0 mm. The stimulated mammary tissue typically presents a more hyperechoic appearance compared to the non-stimulated gland and a fine granular echotexture. Maximum echogenicity of the mammary gland is reached during lactation. In late pregnancy, the mammary glands reach 6–9 mm in thickness. During lactation, the size of the glands depends on the existence of a suckling stimulus, with the suckled glands reaching about 11 mm in thickness. Ductal structures can only be imaged during late pregnancy and lactation. Ultrasonographic evaluation of the feline mammary gland can become a valuable diagnostic tool to characterise physiological changes and may further contribute to a better characterisation of diseased mammary tissue.
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Fang, Rendong, Jingchun Cui, Tengteng Cui, Haiyong Guo, Hisaya Ono, Chun-Ho Park, Masashi Okamura, Akio Nakane, and Dong-Liang Hu. "Staphylococcal Enterotoxin C Is an Important Virulence Factor for Mastitis." Toxins 11, no. 3 (March 2, 2019): 141. http://dx.doi.org/10.3390/toxins11030141.

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Staphylococcus aureus is an important bacterial pathogen causing bovine mastitis, but little is known about the virulence factor and the inflammatory responses in the mammary infection. Staphylococcal enterotoxin C (SEC) is the most frequent toxin produced by S. aureus, isolated from bovine mastitis. To investigate the pathogenic activity of SEC in the inflammation of the mammary gland and the immune responses in an animal model, mouse mammary glands were injected with SEC, and the clinical signs, inflammatory cell infiltration, and proinflammatory cytokine production in the mammary glands were assessed. SEC induced significant inflammatory reactions in the mammary gland, in a dose-dependent manner. SEC-injected mammary glands showed a severe inflammation with inflammatory cell infiltration and tissue damage. In addition, interleukin (IL)-1β and IL-6 production in the SEC-injected mammary glands were significantly higher than those in the PBS control glands. Furthermore, the SEC-induced inflammation and tissue damage in the mammary gland were specifically inhibited by anti-SEC antibody. These results indicated, for the first time, that SEC can directly cause inflammation, proinflammatory cytokine production, and tissue damage in mammary glands, suggesting that SEC might play an important role in the development of mastitis associated with S. aureus infection. This finding offers an opportunity to develop novel treatment strategies for reduction of mammary tissue damage in mastitis.
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Vitásek, R., and H. Dendisová. "Treatment of Feline Mammary Fibroepithelial Hyperplasia Following a Single Injection of Proligestone." Acta Veterinaria Brno 75, no. 2 (2006): 295–97. http://dx.doi.org/10.2754/avb200675020295.

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A case of fibroepithelial hyperplasia of the mammary gland in the cat is reported. A seven-monthold female cat was presented for diffuse enlargement of all of the mammary glands. The cat was treated with proligestone (Covinan®) for the suppression of estrus. Four weeks later all 8 mammary glands were asymmetrically enlarged. There was ulceration of one gland. Sonographic imaging of the affected mammary glands showed homogeneous and granular structures. The patient was treated with subcutaneous injections of 10 mg/kg aglépristone (Alizine®) on days 1, 2, 7, 14 and 21. Within six weeks the mammary glands had completely regressed. No side effects were observed. It is the first case of fully documented feline mammary fibroepithelial hyperplasia and its treatment in the Czech Republic.
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Palin, M. F., D. Beaudry, C. Roberge, and C. Farmer. "Expression levels of STAT5A and STAT5B in mammary parenchymal tissue from Upton-Meishan and Large White gilts." Canadian Journal of Animal Science 82, no. 4 (December 1, 2002): 507–18. http://dx.doi.org/10.4141/a01-091.

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The implication of STAT5A and STAT5B in mammary gland development and maintenance of lactation is well documented in rodents and humans. However, little is known regarding their roles in mammary gland development during gestation in pigs. We identified and analyzed the complete coding sequences of swine STAT5A and STAT5B and evaluated their mRNA levels in mammary glands of gestating gilts (day 110) in two different breeds, Upton-Meishan and Large White. Sequence analysis revealed a new APASA insertion in the STAT5A amino acid sequence that is in close proximity to residue Tyr 699 and whose phosporylation leads to the activation of target genes’ transcription. STAT5A mRNA levels were higher in Upton-Meishan than in Large White. In both breeds, STAT5B mRNA levels were higher than those of STAT5A , which is contrary to what was found in other mammals. A correlation between circulating IGF-I levels and STAT5B mRNA levels in the mammary gland was noticed in the Upton-Meishan breed only. STAT5B mRNA levels in mammary tissue of Large White gilts were highly correlated with extra-parenchymal tissue weight, parenchymal tissue weight, total parenchymal DNA, RNA and RNA/DNA ratio. In Upton-Meishan gilts, correlations were observed only between extra-parenchymal weight and STAT5A and STAT5B mRNA levels. These results indicate that there are significant differences in mRNA levels of STAT5A and STAT5B in the mammary glands of pregnant gilts when compared to other mammals, and between swine breeds. Key words: Mammary glands, signal transducers, pregnancy, kinases, pig, expression
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Conn, David Bruce, Cary A. Hefty, and Sarah Cross Owen. "Infection of Mammary Glands of Small Mammals in Eastern North America by Helminths." Animals 11, no. 11 (November 10, 2021): 3207. http://dx.doi.org/10.3390/ani11113207.

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To determine whether small mammals living in natural settings harbor helminth infections in their mammary glands, we conducted a survey of helminths infecting rodents and soricimorphs in three widespread locations in the eastern United States: states of New York, Tennessee, and Georgia. We examined all the primary organs in all hosts, and identified all helminths. We also excised the complete mammary glands within their subcutaneous fat pads, then stained and mounted each whole mammary gland set for microscopical examination. A total of 53 individual hosts were examined, including 32 Peromyscus spp., 11 Mus musculus, 5 Sigmodon hispidus, 4 Clethrionomys gapperi, and 1 Blarina carolinensis. Helminths collected included Heligmosomoides sp., Hymenolepisdiminuta, Hymenolepis nana, Pterygodermatites peromysci, Schistosomatium douthitti, Syphacia obvelata, Syphacia sigmodontis, and Trichostrongylus sigmodontis. Four S. hispidus were infected by T. sigmodontis in the small intestine; in all four, we also found nematode larvae in lactiferous duct lumen and lactogenic tissue of the mammary glands. We were unable to identify the species of nematode larvae, but the co-occurrence with T. sigmodontis in all cases may suggest an association. Future studies should seek to identify such larvae using molecular and other methods, and to determine the role of these mammary nematode larvae in the life cycle of the identified species. No other host species harbored helminths in the mammary glands. Overall, our results suggest that mammary infections in wild small mammals are not common, but warrant inclusion in future surveys.
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Kruk, O. Yu. "Clinic-anamnestic factors of the risk of hyperplastic diseases an endometrium, mammary glands and their combination at women of perimenopause age." HEALTH OF WOMAN, no. 6(142) (July 29, 2019): 71–73. http://dx.doi.org/10.15574/hw.2019.142.71.

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The objective: to improve well-timed diagnostics of hyperplastic diseases the endometrium and mammary glands at women of perimenopause age on the basis of studying of the main clinical risk factors. Materials and methods. Were surveyed 145 patients of perimenopause age, 15 from which (group of control) the women who didn’t have hyperplastic diseases of organs of genesial system (made of the contingent gynecological and mammology healthy). Results. Clinical-anamnestic risk factors of the isolated hyperplastic process an endometrium are: early menarche, long and plentiful menses, inflammatory diseases of genitalias and use of endometrial methods of contraception. Probability of development of the isolated hyperplasia of mammary glands define: the burdened family anamnesis on a cancer of a mammary gland, the menarche, lack of a lactemia or its duration less than 6 months, pyoinflammatory diseases of mammary glands is later. By risk factors of the combined hyperplastic process in mammary glands and an endometrium are defined: the burdened family anamnesis on a cancer of a mammary gland, the menarche, a long becoming of a menstrual cycle (over a year) is later, than a disease of a thyroid gland and cardiac vascular system, numerous abortions. All taped clinical-anamnestic data indicate disturbance of endocrine balance at patients, both with isolated, and with the combined hyperplastic diseases an endometrium and mammary glands. Conclusion. The received results needs to be considered when developing tactics of forecasting and early diagnostics of the combined pathology of uterus and mammary glands at women of perimenopause age. Key words: hyperplastic processes of uterus and mammary glands, risk factors, perimenopause age.
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Dissertations / Theses on the topic "Mammary glands"

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Wang, Mengdong. "Studies on IgA Induction in Intestine and Mammary Glands of Mammals." Kyoto University, 2015. http://hdl.handle.net/2433/199345.

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Kyoto University (京都大学)
0048
新制・課程博士
博士(農学)
甲第19021号
農博第2099号
新制||農||1030(附属図書館)
学位論文||H27||N4903(農学部図書室)
31972
京都大学大学院農学研究科応用生物科学専攻
(主査)教授 久米 新一, 教授 祝前 博明, 教授 廣岡 博之
学位規則第4条第1項該当
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Miao, Yu Rebecca. "The role of c-Myb in mammary gland development and tumourigenesis." Connect to thesis, 2009. http://repository.unimelb.edu.au/10187/7069.

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c-Myb/MYB is an established and key player in hematopoietic malignancies but more recently a strong case for c-Myb as an oncogene in breast cancer has emerged. c-Myb and its transcriptional target genes have direct bearing on tumour initiation and progression and thus this has opened new opportunities to the development of therapeutic approaches in a range of cancer types with the aim of treating cancer at its various stages. In this study, the requirement of c-Myb during mammary gland tumourigenesis is being examined. In addition a direct therapeutic approach to targeting c-Myb-driven gene grp78/GRP78 in the context of primary and metastatic breast cancer was assessed.
The first aim of this study is to examine the expression of c-Myb during normal mammary gland development. The expression of c-Myb is extensively characterised in a temporal and spatial fashion. Nuclear staining of c-Myb by immunohistochemisty was found to be most elaborately expressed in the ductal epithelium during early mammary gland development. Mouse mammary gland lacking c-myb showed disorganised ductal structure in virgin mice, but did not affect subsequent pregnancy and lactation.
To extend the view that c-Myb is involved in mammary tumourigenesis c-myb-transduced immortalised mammary epithelial cells and two mammary tumour prone transgenic mouse models were examined. NMuMG cells transduced with c-myb showed enhanced proliferation and reduced Annexin V staining consistent with the protection from apoptosis. This reduced apoptosis is consistent with, and perhaps contingent upon, the elevated expression observed for bcl-2 and grp78. The data assembled by expression studies raised the possibility that c-Myb is essential for the establishment of mammary gland tumor in both MMTV-Neu and MMTV-PyMT spontaneous mammary gland tumor models. Loss of c-Myb expression in these models significantly delayed and in most instances completely abolished the onset of mammary gland tumours in both models. Preliminary evidence also indicated that Stat3 phosphorylation may underpin the elevated c-Myb expression in mouse mammary tumour cells.
The focus of my thesis then shifted to examining ways to exploit elevated c-Myb target gene GRP78 expression on the cell surface of mammary tumour cells. To do this I employed a GRP78 binding pro-apoptotic chimera peptide that specifically binds to GRP78 where I examined its efficacy against primary and metastatic breast cancer models. My results demonstrated the anti-tumour activity of the GRP78-chimera peptide both in vitro and in vivo. More importantly, the peptide is also effective at prolonging disease-free survival in mice with established metastatic disease.
Evidence obtained within these studies suggests that c-Myb plays an important role in mammary gland development and tumourgenesis. Although it may be difficult to directly target c-Myb in malignant disease, alternative anti-tumoural therapy may be developed against c-Myb-regulated target genes that are also implicated in mammary tumours. Collectively my thesis studies have advanced our understanding of c-Myb in mammary cancer initiation, progression and as a direct or indirect therapeutic target.
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Aupperlee, Mark Douglas. "The regulation and function of progesterone receptor isoforms A and B in the normal mouse mammary gland." Diss., Connect to online resource - MSU authorized users, 2008.

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Thesis (Ph. D.)--Michigan State University. Cell and Molecular Biology Program, 2008.
Title from PDF t.p. (viewed on March 30, 2009) Includes bibliographical references (p. 174-178). Also issued in print.
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Raghuraman, Nandini. "Prepubertal bisphenol A exposure in the rat mammary gland mechanism of action for carcinogenesis /." Birmingham, Ala. : University of Alabama at Birmingham, 2007. https://www.mhsl.uab.edu/dt/2009r/raghuraman.pdf.

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Nemir, Mohamed. "Inhibition of osteopontin expression in mammary epithelial cells alters mammary gland morphogenesis." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape10/PQDD_0020/NQ44529.pdf.

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Smith, Diane H. "Insulin receptors in the mammary gland /." The Ohio State University, 1986. http://rave.ohiolink.edu/etdc/view?acc_num=osu148726802174716.

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Frend, Hayley Theresa. "Mammary gland hierarchy and its controlling mechanisms." Thesis, University of Cambridge, 2014. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.708178.

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Hughes, Katherine. "Inflammation and remodelling in mammary gland involution." Thesis, University of Cambridge, 2013. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.607688.

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Wang, Juexuan. "Characterization of BCL11 functions in the mouse mammary gland identifies two types of mammary stem cells." Thesis, University of Cambridge, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.610044.

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Enger, Benjamin David. "Intramammary infection in rapidly growing, non-lactating mammary glands." Diss., Virginia Tech, 2018. http://hdl.handle.net/10919/96306.

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Intramammary infections (IMI) are common in non-lactating heifer and dry cow mammary glands and occur during periods of appreciable mammary growth and development. The presence of these infections is expected to negatively impact mammary growth and development but has yet to be investigated. The works reported here investigated how IMI affects mammary tissue structure, cellularity, and the expression of integral mammogenic hormone receptors implicated in mammary growth. Non-pregnant non-lactating cows (n = 19) were administered estradiol and progesterone to stimulate mammary growth and 2 quarters of each cow were subsequently infused with either saline (n = 19) or Staphylococcus aureus (n = 19). Intramammary infusion of Staphylococcus aureus increased the number of immune cells present in gland secretions and also increased the proportion of neutrophils comprising these secretion somatic cells. Mammary tissues from quarters infused with Staphylococcus aureus contained more immune cells, less mammary epithelial tissue area, and greater tissue areas of intralobular stromal tissue than saline quarters. Staphylococcus aureus quarters also contained more apoptotic mammary epithelial cells and a lower proportion of apoptotic cells in the intralobular stroma compartment than saline infused quarters; this signified that Staphylococcus aureus quarters had less epithelial growth and experienced an expansion and/or lack of regression of stromal tissues. The number of cells expressing estrogen receptor α (ESR1) and progesterone receptor (PGR), as well as staining characteristics of ESR1 and PGR positive nuclei was also examined in these tissues. No appreciable differences were observed in any of the examined ESR1 and PGR measures between Staphylococcus aureus and saline mammary glands, but myoepithelial cells from Staphylococcus aureus glands had a greater nuclear staining area than saline quarters, indicating that these cells were affected by IMI. The results of these investigations indicate that IMI, in mammary glands that are concurrently stimulated to grow and develop, limits the growth of mammary epithelium and impairs regression of the stromal tissue, both of which are necessary for successful lactational performance.
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Books on the topic "Mammary glands"

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Jones, Thomas Carlyle, Ulrich Mohr, and Ronald Duncan Hunt, eds. Integument and Mammary Glands. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-83749-4.

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Carlyle, Jones Thomas, Mohr U, and Hunt Ronald Duncan, eds. Integument and mammary glands. Berlin: Springer-Verlag, 1989.

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Tavassoli, Fattaneh A. Tumors of the mammary gland. Washington, D.C: American Registry of Pathology in collaboration with the Armed Forces Institute of Pathology, 2009.

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A, Mol Jan, Clegg Roger A, and International Conference on the Biology of the Mammary Gland (1st : 1999 : Tours, France), eds. Biology of the mammary gland. New York: Kluwer Academic/Plenum Publishers, 2000.

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J, Wilde Colin, Peaker Malcolm, Knight Christopher H, and Hannah Symposium on Intercellular Signalling in the Mammary Gland (1994 : Ayr, Scotland), eds. Intercellular signalling in the mammary gland. New York: Plenum Press, 1995.

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Wilde, Colin J. Intercellular Signalling in the Mammary Gland. Boston, MA: Springer US, 1995.

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1934-, Neville Margaret C., and Daniel Charles W, eds. The Mammary gland: Development, regulation, and function. New York: Plenum Press, 1987.

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Jack, Martinet, and Houdebine Louis-Marie, eds. Biologie de la lactation. Paris: INSERM, 1993.

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O, Conneely, and Otto C, eds. Progestins and the mammary gland: From basic science to clinical applications. Berlin: Springer, 2008.

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Anderson, Lucia. Mammals and their milk. New York: Dodd, Mead, 1985.

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Book chapters on the topic "Mammary glands"

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Ashby, Karen L. "Mammary Glands." In Encyclopedia of Women’s Health, 748–50. Boston, MA: Springer US, 2004. http://dx.doi.org/10.1007/978-0-306-48113-0_250.

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Rutteman, G. R., and P. G. van Ooijen. "Mammary glands." In Medical History and Physical Examination in Companion Animals, 168–74. Dordrecht: Springer Netherlands, 1995. http://dx.doi.org/10.1007/978-94-011-0459-3_16.

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Rehm, Sabine. "Adenoacanthoma, Mammary Gland, Mouse." In Integument and Mammary Glands, 319–23. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-83749-4_44.

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Rehm, Sabine, Jerrold M. Ward, and Annabel G. Liebelt. "Mixed Adenocarcinoma, Mammary Gland, Mouse." In Integument and Mammary Glands, 323–28. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-83749-4_45.

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Delmain, Diane. "Disorders of the Mammary Glands." In Feline Reproduction, 143–52. GB: CABI, 2022. http://dx.doi.org/10.1079/9781789247107.0016.

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Imber, Michael J. "New Approaches and Diagnostic Techniques in Dermatopathology." In Integument and Mammary Glands, 3–14. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-83749-4_1.

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Kanno, Jun. "Melanocytic Tumors, Skin, Mouse." In Integument and Mammary Glands, 63–70. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-83749-4_10.

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Kanno, Jun. "Melanocytic Tumors, Skin, Hamster." In Integument and Mammary Glands, 70–75. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-83749-4_11.

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Zurcher, Chris, and P. J. M. Roholl. "Melanocytic Tumors, Rat." In Integument and Mammary Glands, 76–86. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-83749-4_12.

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Hart-Elcock, Laura, Stephen G. Lake, Robert E. Mueller, and Barry P. Stuart. "Fibroma, Dermis and Subcutis, Rat." In Integument and Mammary Glands, 87–89. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-83749-4_13.

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Conference papers on the topic "Mammary glands"

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Litvinenko, L. M., S. V. Klochkova, and A. A. Bakhmet. "MAMMARY GLANDS AND CENTERS OF DIGESTION." In MODERN PROBLEMS IN SYSTEMIC REGULATION OF PHYSIOLOGICAL FUNCTIONS. NPG Publishing, 2019. http://dx.doi.org/10.24108/5-2019-confnf-49.

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Yamamuro, Mika, Yoshiyuki Asai, Naomi Hashimoto, Nao Yasuda, Kenta Sakaguchi, Tatsuo Konishi, Koji Yamada, Yoshiaki Ozaki, Kazunari Ishii, and Yougbum Lee. "Deep learning-based segmentation of mammary gland region in digital mammograms of scattered mammary glands and fatty breasts." In Fifteenth International Workshop on Breast Imaging, edited by Chantal Van Ongeval, Nicholas Marshall, and Hilde Bosmans. SPIE, 2020. http://dx.doi.org/10.1117/12.2562180.

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Leffers, D., C. Idel, L. Tharun, S. Perner, and K.-L. Bruchhage. "(Mammary analog) Secretory Carcinoma (MASC) of the salivary glands." In 100 JAHRE DGHNO-KHC: WO KOMMEN WIR HER? WO STEHEN WIR? WO GEHEN WIR HIN? Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1728911.

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Ahmed, Irfan, and Condon Lau. "Detection of lithium in breast milk and the mammary glands." In Visualizing and Quantifying Drug Distribution in Tissue III, edited by Conor L. Evans and Kin Foong Chan. SPIE, 2019. http://dx.doi.org/10.1117/12.2507536.

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Tu, Shengxian, Su Zhang, Wei Yang, Xuesong Lu, and Yazhu Chen. "Automatic Segmentation of Rat Mammary Glands from Serial MRI Images." In 2007 IEEE/ICME International Conference on Complex Medical Engineering. IEEE, 2007. http://dx.doi.org/10.1109/iccme.2007.4381827.

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Polec, Anna, Jens Henrik Norum, Andreas Brech, and Therese Sorlie. "Abstract A05: Deep immunofluorescence imaging of solvent-cleared mouse mammary glands." In Abstracts: AACR Special Conference: Advances in Breast Cancer Research; October 7-10, 2017; Hollywood, CA. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1557-3125.advbc17-a05.

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Engelmann, Sophie Luca, Benedikt Hofauer, Barbara Wollenberg, Andreas Roth, and Julia Slotta-Huspenina. "Case series of five Mammary Analogue Secretory Carcinoma (MASC) of salivary glands and immunohistochemical characterization." In 94th Annual Meeting German Society of Oto-Rhino-Laryngology, Head and Neck Surgery e.V., Bonn. Georg Thieme Verlag, 2023. http://dx.doi.org/10.1055/s-0043-1767187.

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Barkova, Anna, Anatoliy Elesin, Igor Milshtein, and Mikhail Barashkin. "Thermovision diagnostics of the milking equipment impact on the state of mammary glands of cattle." In Proceedings of the International Scientific and Practical Conference “Digital agriculture - development strategy” (ISPC 2019). Paris, France: Atlantis Press, 2019. http://dx.doi.org/10.2991/ispc-19.2019.116.

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Cantrell, MA, ND Ebelt, and Berg CL Van Den. "PD08-01: JNK2 Regulates Mammary Lineage Differentiation in Tumors and Normal Glands through Notch1 and p53." In Abstracts: Thirty-Fourth Annual CTRC‐AACR San Antonio Breast Cancer Symposium‐‐ Dec 6‐10, 2011; San Antonio, TX. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/0008-5472.sabcs11-pd08-01.

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Yan, Leqin, K. Leslie Powell, and Michael C. MacLeod. "Abstract 1241: Tcf7, a mediator of Wnt/β-catenin signaling, is upregulated in mammary glands of BK5.ATF3 mice, without prior development of mammary tumors." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-1241.

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Reports on the topic "Mammary glands"

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Daniel, Charles W. Homebox Genes in Normal, Preneoplastic, and Neoplastic Mammary Glands. Fort Belvoir, VA: Defense Technical Information Center, December 1998. http://dx.doi.org/10.21236/ada370302.

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Daniel, Charles W. Homebox Genes in Normal, Preneoplastic, and Neoplastic Mammary Glands. Fort Belvoir, VA: Defense Technical Information Center, January 1998. http://dx.doi.org/10.21236/ada346064.

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Daniel, Charles W. Homeobox Genes in Normal, Preneoplastic, and Neoplastic Mammary Glands. Fort Belvoir, VA: Defense Technical Information Center, January 2000. http://dx.doi.org/10.21236/ada391550.

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Haddad, Bassem R. Characterization of Early Genomic Changes in Mammary Glands in High Risk Women. Fort Belvoir, VA: Defense Technical Information Center, July 2002. http://dx.doi.org/10.21236/ada409620.

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Dickson, Robert. Characterization of Early Genomic Changes in Mammary Glands of High Risk Women. Fort Belvoir, VA: Defense Technical Information Center, July 2000. http://dx.doi.org/10.21236/ada391200.

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Shpigel, Nahum Y., Ynte Schukken, and Ilan Rosenshine. Identification of genes involved in virulence of Escherichia coli mastitis by signature tagged mutagenesis. United States Department of Agriculture, January 2014. http://dx.doi.org/10.32747/2014.7699853.bard.

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Mastitis, an inflammatory response of the mammary tissue to invading pathogenic bacteria, is the largest health problem in the dairy industry and is responsible for multibillion dollar economic losses. E. coli are a leading cause of acute mastitis in dairy animals worldwide and certainly in Israel and North America. The species E. coli comprises a highly heterogeneous group of pathogens, some of which are commensal residents of the gut, infecting the mammary gland after contamination of the teat skin from the environment. As compared to other gut microflora, mammary pathogenic E. coli (MPEC) may have undergone evolutionary adaptations that improve their fitness for colonization of the unique and varied environmental niches found within the mammary gland. These niches include competing microbes already present or accompanying the new colonizer, soluble and cellular antimicrobials in milk, and the innate immune response elicited by mammary cells and recruited immune cells. However, to date, no specific virulence factors have been identified in E. coli isolates associated with mastitis. The original overall research objective of this application was to develop a genome-wide, transposon-tagged mutant collection of MPEC strain P4 and to use this technology to identify E. coli genes that are specifically involved in mammary virulence and pathogenicity. In the course of the project we decided to take an alternative genome-wide approach and to use whole genomes bioinformatics analysis. Using genome sequencing and analysis of six MPEC strains, our studies have shown that type VI secretion system (T6SS) gene clusters were present in all these strains. Furthermore, using unbiased screening of MPEC strains for reduced colonization, fitness and virulence in the murine mastitis model, we have identified in MPEC P4-NR a new pathogenicity island (PAI-1) encoding the core components of T6SS and its hallmark effectors Hcp, VgrG and Rhs. Next, we have shown that specific deletions of T6SS genes reduced colonization, fitness and virulence in lactating mouse mammary glands. Our long-term goal is to understand the molecular mechanisms of host-pathogen interactions in the mammary gland and to relate these mechanisms to disease processes and pathogenesis. We have been able to achieve our research objectives to identify E. coli genes that are specifically involved in mammary virulence and pathogenicity. The project elucidated a new basic concept in host pathogen interaction of MPEC, which for the best of our knowledge was never described or investigated before. This research will help us to shed new light on principles behind the infection strategy of MPEC. The new targets now enable prevalence and epidemiology studies of T6SS in field strains of MPEC which might unveil new geographic, management and ecological risk factors. These will contribute to development of new approaches to treat and prevent mastitis by MPEC and perhaps other mammary pathogens. The use of antibiotics in farm animals and specifically to treat mastitis is gradually precluded and thus new treatment and prevention strategies are needed. Effective mastitis vaccines are currently not available, structural components and effectors of T6SS might be new targets for the development of novel vaccines and therapeutics.
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Bradley, Allan. Tissue Specific Chromosome Deletions: An In Vivo Genetic Screen for Tumor Suppressor Genes in the Mammary Glands. Fort Belvoir, VA: Defense Technical Information Center, May 2001. http://dx.doi.org/10.21236/ada410501.

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Zhang, Hua, Zhu-ye Zhang, Wen-hong Li, Jia-xuan Ren, and Jian-lin Wu. Clinical efficacy of “Sancai therapy” for hyperplasia of the mammary glands: A systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, August 2023. http://dx.doi.org/10.37766/inplasy2023.8.0124.

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Ma, Dehui, Guochao Liu, Tianjiao Gao, Qi Zhang, and Mingjun Liu. Massage treatment of hyperplasia of mammary glands A protocol for a systematic review and meta-analysis: study protocol. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, October 2020. http://dx.doi.org/10.37766/inplasy2020.10.0066.

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Ma, Mengjie, Liuqiao Zhang, and Xiangli Wang. Effect of auricular point pressing therapy on hyperplasia of mammary glands A protocol for a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, January 2021. http://dx.doi.org/10.37766/inplasy2021.1.0028.

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