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Books on the topic 'Major depressive disorder'

Author: Grafiati
Published: 4 June 2021
Last updated: 9 March 2023

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1

Kim, Yong-Ku, ed. Major Depressive Disorder. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-33-6044-0.

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2

McIntyre, Roger S., and Danielle S. Cha, eds. Cognitive Impairment in Major Depressive Disorder. Cambridge: Cambridge University Press, 2016. http://dx.doi.org/10.1017/cbo9781139860567.

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3

Association, American Psychiatric. Practice guideline for major depressive disorder in adults. Washington: American Psychiatric Association, 1993.

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4

American Psychiatric Association. Practice guideline for major depressive disorder in adults. Washington, DC: The Association, 1993.

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5

Kim, Yong-Ku, and Meysam Amidfar, eds. Translational Research Methods for Major Depressive Disorder. New York, NY: Springer US, 2022. http://dx.doi.org/10.1007/978-1-0716-2083-0.

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6

Association, American Psychiatric, ed. Treatment works: Major depressive disorder : a patient and family guide. Washington, DC: American Psychiatric Association, 2000.

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7

Alexopoulos, George, Siegfried Kasper, Hans-Jürgen Möller, and Carmen Moreno. Guide to Assessment Scales in Major Depressive Disorder. Cham: Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-04627-3.

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8

Tolman, Anton O. Major depressive disorder: The latest assessment and treatment strategies. Kansas City, MO: Compact Clinicals, 1995.

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9

Understanding and managing the pieces of major depressive disorder. Carlsbard, Calif: NEI Press, 2009.

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10

American Psychiatric Association. Practice guideline for the treatment of patients with major depressive disorder. Washington, D.C: The Association, 2000.

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11

Disorder, American Psychiatric Association Work Group on Major Depressive. Practice guideline for the treatment of patients with major depressive disorder, (revision). Washington, D.C: American Psychiatric Association, 2000.

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12

Greden, John F. Treatment resistant depression: A roadmap for effective care. Washington, DC: American Psychiatric Pub., 2011.

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13

R, Bancroft Pauline, and Ardley Leli B, eds. Major depression in women. New York: Nova Biomedical Books, 2008.

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14

Depression sourcebook: Basic consumer health information about the symptoms, causes, and types of depression, including major depression, dysthymia, atypical depression, bipolar disorder, depression during and after pregnancy, premenstrual dysphoric disorder, schizoaffective disorder, and seasonal affective disorder; along with facts about depression and chronic illness, treatment-resistant depression and suicide, mental health medications, therapies, and treatments, tips for improving self-esteem, resilience, and quality of life while living with depression ... 3rd ed. Detroit, MI: Omnigraphics, Inc., 2012.

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15

Antidepressants therapy and risk of suicide among patients with major depressive disorders. Hauppauge, N.Y: Nova Biomedical Books, 2011.

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16

Hepner, Kimberly A. Measuring the quality of care for psychological health conditions in the military health system: Candidate quality measures for posttraumatic stress disorder and major depressive disorder. Santa Monica, CA: RAND, 2015.

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17

Major Depressive Disorder. Oxford University Press, Incorporated, 2015.

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18

Major Depressive Disorder. Elsevier, 2020. http://dx.doi.org/10.1016/c2017-0-01421-0.

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19

McIntyre, Roger S., Yena Lee, Carola Rong, and Mehala Subramaniapillai. Major Depressive Disorder. Elsevier - Health Sciences Division, 2019.

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20

Pierce, Simon. Major Depressive Disorder. Greenhaven Publishing LLC, 2017.

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21

Major Depressive Disorder. Lucent Books, 2017.

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22

Jeon, Sang Won, Hwa Young Lee, Anna C. Phillips, and Xiaobin Zhang. Major Depressive Disorder. DI Press, 2022.

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23

Cognition In Major Depressive Disorder. Oxford University Press, 2013.

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24

Soczynska, Joanna K., Roger S. McIntyre, and Danielle S. Cha. Cognition in Major Depressive Disorder. Oxford University Press, 2014.

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25

Cognition in Major Depressive Disorder. Oxford University Press, Incorporated, 2014.

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26

Craighead, W. Edward, Benjamin N. Johnson, Sean Carey, and Boadie W. Dunlop. Psychosocial Treatments for Major Depressive Disorder. Oxford University Press, 2015. http://dx.doi.org/10.1093/med:psych/9780199342211.003.0013.

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Behavior therapy, cognitive-behavioral therapy, and interpersonal psychotherapy have each been shown by at least two randomized controlled trials, as well as by numerous meta-analytic reports, to be effective psychosocial interventions for patients meeting criteria for major depressive disorder. All three psychosocial treatments have yielded substantial reductions in scores on the two major depression rating scales, significant decreases in percentage of patients meeting depression criteria at posttreatment, and substantial maintenance of effects well after treatment has ended. The data for outcomes of psychosocial and pharmacological interventions for major depressive episodes suggest that the two treatment modes are equally efficacious.
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27

Radenkovi?, Milena ?uki?, and Milena Cukic Radenkovic. Novel Approaches in Treating Major Depressive Disorder (Depression). Nova Science Publishers, Incorporated, 2019.

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28

Milena Čukić Radenković. Novel Approaches in Treating Major Depressive Disorder (Depression). Nova Science Publishers, Incorporated, 2019.

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29

Baune, Bernhard T. Cognitive Dimensions of Major Depressive Disorder. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780198835554.001.0001.

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Cognitive Dimensions of Major Depressive Disorder (MDD) examines the key clinical and pathophysiological characteristics and treatment options of MDD. The volume emphasizes that while the traditional model of depression implicates mood as the primary symptom cluster, a more recently published conceptual understanding of depression has been extended to consider cognitive function as more than just a symptom. It furthers our understanding of the central role of the cognitive dimension for the pathophysiology, diagnosis, and treatment of MDD. It reviews the key cognitive dimensions of depression comprising impaired cognitive and emotional processes of cognitive function, emotion processing, and social cognitive processing. It focuses on the cognitive and emotional dimensions of depression and offers extended and novel diagnostic and treatment approaches ranging from pharmacological to psychological interventions targeting those dimensions of depression.
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30

Baune, Bernhard T., and Catherine Harmer, eds. Cognitive Dimensions of Major Depressive Disorder. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780198810940.001.0001.

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The lifetime prevalence of 15% for major depressive disorder (MDD) within the general population is among the highest among all mental disorders. MDD is also one of the leading causes of disability and has been estimated to affect 300 million people worldwide. Clinical, functional, and biological correlates of MDD are frequently investigated almost exclusively based on research that defines depression as a categorical disorder assessed by established diagnostic instruments. Given the phenotypic and biological heterogeneity of depression, a refocus of the clinical phenotype of depression is required and widely recommended. Cognitive dimensions of depression have long been implicated in the nature of depression as a disorder that is characterized by typically impaired cognitive and emotional processes. The systems of cognitive function, emotion processing, and social cognitive processing are regarded as comprehensively describing large parts of the clinical symptoms as well as the pathophysiology of the brain-based disorder of depression. The focus on the above cognitive and emotional dimensions of depression offers promising extended and novel diagnostic and treatment approaches ranging from pharmacological to psychological interventions targeting those dimensions of depression. This book aims to provide an improved understanding of the characteristics of the dimensional approach of depression, focusing on the cognitive, emotional, and social cognitive processes.
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31

Carvalho, André F., Gilberto S. Alves, Cristiano A. Köhler, and Roger S. McIntyre. Cognitive Enhancement in Major Depressive Disorder. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190214401.003.0010.

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Major depressive disorder (MDD) is a chronic and disabling illness often associated with elevated rates of non-recovery and substantial psychosocial burden. Cognitive impairment is a common residual manifestations of MDD. Overactivation of the hypothalamic–pituitary–adrenal axis, along with immune–inflammatory imbalances, a decrease in neurotrophin signaling, and an increase in oxidative and nitrosative stress, leads to neuroprogression and cognitive deterioration in MDD. “Cognitive remission” has been proposed as a novel treatment target for MDD. Cognitive remediation therapy has provided encouraging results for the management of cognitive deficits in MDD. The effects of standard antidepressant drugs on MDD-related cognitive dysfunction are often suboptimal, which calls for the development of novel agents with the potential to target cognitive impairments in MDD. The incorporation of biobehavioral strategies (e.g., exercise) and multimodal treatment approaches (e.g., cognitive training, antidepressant therapy, and neuromodulation) is more likely to generate therapeutic benefit.
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32

Baune, Bernhard. Cognitive Dimensions of Major Depressive Disorder. Oxford University Press, 2021.

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33

Baune, Bernhard T., and Catherine Harmer. Cognitive Dimensions of Major Depressive Disorder. Oxford University Press, 2019.

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34

Kim, Yong-Ku, and Meysam Amidfar. Translational Research Methods for Major Depressive Disorder. Springer, 2022.

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35

Kim, Yong-Ku, ed. Major Depressive Disorder - Cognitive and Neurobiological Mechanisms. InTech, 2015. http://dx.doi.org/10.5772/58711.

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36

Narziss und Echo: Psychobiologisches Modell der Depression. Wien, Austria: Springer, 2009.

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37

Loo, Colleen. TMS in the treatment of major depressive disorder. Edited by Charles M. Epstein, Eric M. Wassermann, and Ulf Ziemann. Oxford University Press, 2012. http://dx.doi.org/10.1093/oxfordhb/9780198568926.013.0040.

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There is a growing body of literature on animal experiments using behavioural paradigms or investigations of neurobiological mechanisms to assess the antidepressant effects of repetitive (r)TMS. This article summarizes the findings from these studies. The evidence from these studies is supportive of antidepressant effects for rTMS. This evidence has provided several plausible hypotheses for the antidepressant effects of rTMS. Several studies have suggested that rTMS may have neuroprotective effects. There are many variations in the way rTMS can be given as a clinical treatment, involving choices over treatment site, stimulation parameters and treatment course. Clinical trials to date have found rTMS to be safe when given within recommended parameter guidelines. Further research is needed to understand the role of rTMS with respect to other established antidepressant treatments.
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38

EEG-Based Experiment Design for Major Depressive Disorder. Elsevier, 2019. http://dx.doi.org/10.1016/c2018-0-01657-6.

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39

Alexopoulos, George, Siegfried Kasper, Carmen Moreno, and Hans-Jürgen Möller. Guide to Assessment Scales in Major Depressive Disorder. Adis, 2014.

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40

Kim, Yong-Ku. Major Depressive Disorder: Rethinking and Understanding Recent Discoveries. Springer, 2022.

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41

Alexopoulos, George, Siegfried Kasper, Carmen Moreno, and Hans-Jürgen Möller. Guide to Assessment Scales in Major Depressive Disorder. Springer, 2014.

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42

Kim, Yong-Ku. Major Depressive Disorder: Current Research and Management Approaches. Springer Singapore Pte. Limited, 2021.

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43

DeMarco, Daryl V. Major Depression and the Woke Government: The Causes and Cures of Major Depressive Disorder. Tellwell Talent, 2022.

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44

Chen, Michael C., and Ian H. Gotlib. Molecular Foundations of the Symptoms of Major Depressive Disorder. Edited by Turhan Canli. Oxford University Press, 2013. http://dx.doi.org/10.1093/oxfordhb/9780199753888.013.002.

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Major Depressive Disorder (MDD) is a prevalent and costly disorder with a broad range of cognitive, affective, and behavioral symptoms. Despite the absence of a clear final common molecular pathway in depression, many molecular systems have been implicated in MDD. In particular, disruptions in molecular systems like serotonin, dopamine, glutamate, and other neurotransmitters, as well as in stress hormones, cytokines, neurotrophins, and neuropeptides, may contribute to MDD. To link the symptoms of MDD with molecular dysfunction, this article examines these molecules in the context of three symptom clusters of MDD: cognitive/affective symptoms, volitional/behavioral symptoms, and homeostatic/vegetative symptoms. It examines how these molecules and their receptor, transport, and regulatory systems contribute to MDD and to the development of specific symptom clusters. It presents two possible frameworks of molecular dysfunction in MDD that encompass the interactions between vulnerability phenotypes and biochemical perturbations that may lead to the heterogeneous symptoms of this disorder.
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45

Kim, Yong-Ku. Major Depressive Disorder: Risk Factors, Characteristics and Treatment Options. Nova Science Publishers, Incorporated, 2017.

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46

Mineka, Susan, Deepika Anand, and Jennifer A. Sumner. Important Issues in Understanding Comorbidity Between Generalized Anxiety Disorder and Major Depressive Disorder. Edited by C. Steven Richards and Michael W. O'Hara. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199797004.013.031.

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The comorbidity of anxiety and mood disorders has been of great interest to psychopathology researchers for the past 25 years. One topic––the comorbidity of generalized anxiety disorder (GAD) and major depressive disorder (MDD)––has received considerable attention, in part because it has raised fundamental nosological issues regarding whether GAD should continue to be categorized as an anxiety disorder or whether it should be recategorized as a mood disorder. We review the logic for reclassifying GAD with the mood disorders as well as what we believe to be even more compelling reasons for why it should be retained as an anxiety disorder. In doing so, we review three different kinds of comorbidity—cross-sectional, cumulative (lifetime), and sequential. We also discuss overlaps and distinctions in what is known about the etiology of GAD and MDD and how their somewhat different cognitive and affective profiles bear on these issues of classification. Finally, we briefly discuss what some of the treatment implications may be for individuals with comorbid GAD and MDD.
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47

McIntyre, Roger S., and Danielle S. Cha. Cognitive Impairment in Major Depressive Disorder: Clinical Relevance, Biological Substrates, and Treatment Opportunities. Cambridge University Press, 2016.

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48

McIntyre, Roger S., and Danielle S. Cha. Cognitive Impairment in Major Depressive Disorder: Clinical Relevance, Biological Substrates, and Treatment Opportunities. Cambridge University Press, 2016.

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49

McIntyre, Roger S., and Danielle S. Cha. Cognitive Impairment in Major Depressive Disorder: Clinical Relevance, Biological Substrates, and Treatment Opportunities. Cambridge University Press, 2016.

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50

Barton, Casey P. Major Depressive Disorder in Veterans: Antidepressant Use and Suicide Prevention. Nova Science Publishers, Incorporated, 2015.

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